Relevant bibliographies by topics / Dysfunctional protest

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Dysfunctional protest'

Author: Grafiati
Published: 7 June 2025
Last updated: 2 August 2025

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Journal articles on the topic "Dysfunctional protest"

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Mistygacz, Michał, and Anna Materska-Sosnowska. "Dysfunkcjonalność protestów wyborczych w Polsce w świetle standardu uczciwych wyborów." Przegląd Prawa Konstytucyjnego 75, no. 5 (2023): 191–203. http://dx.doi.org/10.15804/ppk.2023.05.14.

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Fair elections are one of the foundations of electoral axiology in a democratic state ruled by law. Every citizen has the right to protest, and the justification of the citizen’s complaint is the public interest. An election protest is a legal measure to control the held General elections and its purpose is to protect the public interest in the electoral process. The standard of fair elections is not only a context for legal solutions strictly derived from the Constitution or the Electoral Code. The juridical architecture of electoral protests in Poland generally creates an effective mechanism
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Eneni, Roberts Ofrey, and bada Oluchi Vincent Ogbumg. "Remediating youth restiveness using social entrepreneurship education." GPH-International Journal of Educational Research 7, no. 11 (2025): 90–108. https://doi.org/10.5281/zenodo.14621293.

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Youth restiveness has been on the increase in almost every community in Nigeria. This has contributed to massive loss of lives and properties, which constitute a major threat to security of the state and its corporate existence. The aim of this research was to examine the relationship between Social Entrepreneurship (SE) andYouth Restiveness (YR). The study followed the pragmatic philosophical paradigm combining both the inductive and the deductive methods of scientific inquiry in a cross-sectional survey. A systematic random sampling was adopted to collect datafrom308 youths that werebetween
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POOLE, STEVE. "‘A lasting and salutary warning’: Incendiarism, Rural Order and England's Last Scene of Crime Execution." Rural History 19, no. 2 (2008): 163–77. http://dx.doi.org/10.1017/s0956793308002471.

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AbstractAgricultural incendiarism was a perennial factor in social relations in some areas of nineteenth-century rural England and is often understood by historians as an expression of ‘covert’ social protest. However, such categorisation risks oversimplifying what may be diverse and locally specific factors. In 1830, the high sheriff of Somerset presided over England's last ever scene of crime execution at the Somerset village of Kenn following the conviction of three labourers for incendiarism. This requires explanation. Crime scene executions were not only anachronistic and rare, but unfash
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Snyder-Young, Dani. "The Promise and Pitfalls of the Real." Theatre Survey 57, no. 3 (2016): 424–31. http://dx.doi.org/10.1017/s0040557416000442.

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Audience members sit at tables and desks in an interactive classroom, an immersive performance space designed to evoke a K–12 classroom. Blackboards are covered with homework assignments and test reminders, posters with test-taking tips and motivational quotes such as “For success, attitude is as important as ability.” Collaboraction's production of Forgotten Future: The Education Project begins as an interracial, intergenerational ensemble of actors enters the space chanting and waving signs reading “Support our Schools: Don't Close Them” and “Save our Schools” in protest of Chicago's dysfunc
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Pustovoyt, Yurii. "PROTEST IN THE CITIES OF SIBERIA: THE EXPERIENCE OF MEASURING FACTORS OF MOBILIZATION ACTIVITY." Вестник Пермского университета. Политология 14, no. 3 (2020): 122–30. http://dx.doi.org/10.17072/2218-1067-2020-3-122-130.

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The paper considers a set of conditions that stimulate mobilization activity. Based on the J. Goldstone’s fourth-generation revolution model, the author proposes such stimuli: the effectiveness of government, the dissatisfaction and the elites, and the protesters' notion of the power. The author reviews protesting regions’ capitals with these factors based on the assessment of mobilization. The preliminary results show that the higher the level of urban prosperity and of formal competition, the higher the mobilization activity and the ability of protest communities to establish control over re
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Neshev, Petar. "Domestic Violence, Family Relationships and Problem Behavior in Childhood and Adolescent Age." Педагогически форум 11, no. 1 (2023): 31–40. http://dx.doi.org/10.15547/pf.2023.004.

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The article examines the causes of domestic violence and the relationship between family relationships and problematic behavior in childhood and adolescence. The influence of different types of parenting models on child development is presented. The symptoms of the dysfunctional family are indicated. Domestic violence and faulty parenting patterns can lead children and adolescents to traumatic experiences and sufferers should be given support and professional help. The psychological specificity of different types of family relationships has been revealed. In family relationships with prevailin
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Adeyemi, Amos ADEGBOYEGA. "Protest, Democracy and Social Development in Nigeria: Reflections on Contemporary Nigerian Writings." International Journal of Academic Research in Business, Arts & Science ( IJARBAS.COM ) 1, no. 4 (2020): 51–71. https://doi.org/10.5281/zenodo.3598132.

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Literary productions from Nigeria since inception have envisioned a utopian society. Driven by protest to foster sociopolitical development, the literary artist from the days of Chinua Achebe in <em>Things Fall Apart</em>** engaged in and has remained relevant to emerging trends of discourses in Nigeria. While forebear nationalists protested against colonial incursions and the attending destabilization of the hitherto organized Nigerian society, contemporary postcolonial writers contend with realities militating against the nation&rsquo;s fragile democracy. Their creativities beyond entertainm
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Gabsi, Zouhir. "Rap and Mizoued Music: Claiming a Space for Dissent and Protest in Post-Arab Spring Tunisia." Sociological Research Online 25, no. 4 (2020): 626–43. http://dx.doi.org/10.1177/1360780419898494.

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The Arab Spring in Tunisia has brought with it positive changes, such as freedom of expression and democracy. However, Tunisians have found that these applauded achievements have not improved their living conditions. After Ben Ali was ousted in 2010, the decline of Tunisia’s economy was exacerbated by internal and external factors such as global recession, a dysfunctional liberal economy, internal political infighting, and corruption. To ventilate their frustration and dismay with the government and the overall socio-economic situation in Tunisia, some Tunisians turn to the music of rap and Mi
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Hemann, Michael T., Karl Lenhard Rudolph, Margaret A. Strong, Ronald A. DePinho, Lynda Chin, and Carol W. Greider. "Telomere Dysfunction Triggers Developmentally Regulated Germ Cell Apoptosis." Molecular Biology of the Cell 12, no. 7 (2001): 2023–30. http://dx.doi.org/10.1091/mbc.12.7.2023.

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Telomere dysfunction results in fertility defects in a number of organisms. Although data from fission yeast and Caenorhabditis elegans suggests that telomere dysfunction manifests itself primarily as defects in proper meiotic chromosome segregation, it is unclear how mammalian telomere dysfunction results in germ cell death. To investigate the specific effects of telomere dysfunction on mammalian germ cell development, we examined the meiotic progression and germ cell apoptosis in late generation telomerase null mice. Our results indicate that chromosome asynapsis and missegregation are not t
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Scanavino, Marco de Tubino. "Sexual Dysfunctions of HIV-Positive Men: Associated Factors, Pathophysiology Issues, and Clinical Management." Advances in Urology 2011 (2011): 1–10. http://dx.doi.org/10.1155/2011/854792.

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Sexual dysfunctions in HIV-positive men are associated with an increase in risky sexual behavior and decreased adherence to antiretroviral drug regimens. Because of these important public health issues, we reviewed the literature on the pathophysiology, associated factors and clinical management of sexual dysfunction in HIV-positive men. The goal was to investigate the current research on these issues. Literature searches were performed in June 2011 on PubMed, Web of Science, and PsycInfo databases with the keywords “AIDS” and “sexual dysfunction” and “HIV” and “sexual dysfunction”, resulting
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Dissertations / Theses on the topic "Dysfunctional protest"

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Faggian, Alessia. "Glucotoxicity, Protein Glycation and Adipose Tissue Dysfunction." Doctoral thesis, Università degli studi di Padova, 2016. http://hdl.handle.net/11577/3425305.

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Introduction: Glycation is a non-enzymatic reaction between a sugar and a free-amino group contained in molecules such as proteins, amino acids, DNA, RNA, and lipids. In the initial phase, the carbonyl group of the reducing carbohydrate condenses with the free-amino group on the biomolecule to form a reversible glycosylamine, which is then converted to a more stable Amadori product. Once formed, these products can with time undergo to dehydration, cyclization, oxidation, and rearrangements forming a polymorphic group of compounds collectively referred as Advanced Glycation End-products (AGEs).
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Dutt, Tina. "The effect of activated protein C on cardiac cells." Thesis, University of Liverpool, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.631571.

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Activated Protein C (APC) is an endogenous anticoagulant that regulates thrombin generation. Its physiological importance is reflected by the thrombotic phenotype of PC deficient individuals and those with APC resistance due to mutation at the Factor V cleavage site. At a molecular level, thrombin binding to the receptor thrombomodulin (TM) converts endothelial protein C receptor (EPCR) bound PC into APC. APC has a negative feedback role in curbing thrombin generation by inactivating coagulation factors V and VIII. Clinically, acquired PC deficiency occurs in conditions such as severe sepsis a
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Adams, Gregory Nicholas. "Prolylcarboxypeptidase protects from vascular dysfunction and promotes vascular repair." Case Western Reserve University School of Graduate Studies / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=case1346973249.

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Gianni, Davide. "The role of unfolded protein deposits in cardiac dysfunction." Thesis, Imperial College London, 2011. http://hdl.handle.net/10044/1/7029.

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In this study we investigated the role of unfolded proteins as a toxic insult for cardiomyocytes in idiopathic dilated cardiomyopathy (DCM). We first confirmed the presence of amyloid fibers in DCM cardiomyocytes by histological and ultrastructural analysis, showing their preferentially intracellular distribution. These molecular species seem to coexist with low-complexity β-folded precursors (oligomers) which in our experiments could promote increase of systolic Ca2+ in normal cardiomyocytes and alterations of contractility. Our results suggest that these molecular species trigger the overexp
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Marrerios, Rita [Verfasser]. "Protein homeostasis dysfunction in chronic brain disorders / Rita Marrerios." Düsseldorf : Universitäts- und Landesbibliothek der Heinrich-Heine-Universität Düsseldorf, 2020. http://d-nb.info/1219299618/34.

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Del, Dotto Valentina <1987&gt. "OPA1 isoforms and protein domains in the rescue of mitochondrial dysfunctions." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2015. http://amsdottorato.unibo.it/7033/1/Del_Dotto_Valentina_tesi.pdf.

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Mutations in OPA1 gene have been identified in the majority of patients with Dominant Optic Atrophy (DOA), a blinding disease, and the syndromic form DOA-plus. OPA1 protein is a mitochondrial GTPase involved in various mitochondrial functions, present in humans in eight isoforms, resulting from alternative splicing and proteolytic processing. In this study we have investigated the specific role of each isoform through expression in OPA-/- MEFs, by evaluating their ability to improve the defective mitochondrial phenotypes. All isoforms were able to rescue the energetic efficiency, mitochondrial
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Del, Dotto Valentina <1987&gt. "OPA1 isoforms and protein domains in the rescue of mitochondrial dysfunctions." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2015. http://amsdottorato.unibo.it/7033/.

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Mutations in OPA1 gene have been identified in the majority of patients with Dominant Optic Atrophy (DOA), a blinding disease, and the syndromic form DOA-plus. OPA1 protein is a mitochondrial GTPase involved in various mitochondrial functions, present in humans in eight isoforms, resulting from alternative splicing and proteolytic processing. In this study we have investigated the specific role of each isoform through expression in OPA-/- MEFs, by evaluating their ability to improve the defective mitochondrial phenotypes. All isoforms were able to rescue the energetic efficiency, mitochondrial
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Chang, Alex Hongsheng. "Intracellular inhibition of immune dysfunction induced by HIV-1 Nef protein." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0017/NQ56524.pdf.

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Low, Darryl Weijun. "Synaptic vesicle protein 2A-dependent function and dysfunction at the presynapse." Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/31320.

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Neurotransmission is essential for neuronal communication. At the presynapse, synaptic vesicles (SVs) undergo exocytosis to release neurotransmitter in response to incoming action potentials, and endocytosis to maintain the supply of SVs needed for further rounds of exocytosis. A key event during SV endocytosis is the efficient sorting and localisation of SV proteins at the plasma membrane. This ensures that nascent SVs that are formed have the correct molecular composition to participate in subsequent exocytic events. The sorting of SV proteins at the plasma membrane is usually facilitated by
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Betters, Jenna Leigh Jones. "Trolox supplementation during mechanical ventilation attenuates contractile dysfunction and protein degradation." [Gainesville, Fla.] : University of Florida, 2004. http://purl.fcla.edu/fcla/etd/UFE0004290.

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Books on the topic "Dysfunctional protest"

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Moseley, Mason W. Protest State. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190694005.001.0001.

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In the midst of an unprecedented era of democratic governments and economic prosperity, why are a record number of Latin American citizens choosing to participate in protests? This book argues that increasingly engaged citizenries, forged by economic progress and technological advances throughout the region, combined with dysfunctional political institutions have fueled more contentious modes of participation in Latin America, as citizens’ demands for government responsiveness have overwhelmed many regimes’ institutional capacity to provide it. Where weak institutions and active citizenries co
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Brickman, Cristina. Can We Inject to Protect. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199938568.003.0415.

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These case studies illustrate infections encountered in hospitals among patients with compromised immune systems. As a result of immunocompromise, the patients are vulnerable to common and uncommon infections. These cases are carefully chosen to reflect the most frequently encountered infections in the patient population, with an emphasis on illustrations and lucid presentations to explain state-of-the-art approaches in diagnosis and treatment. Common and uncommon presentations of infections are presented while the rare ones are not emphasized. The cases are written and edited by clinicians an
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McGregor, Lynnette. Determining the association beteween protease inhibitors and sexual dysfunction in HIV/AIDS patients. 2001.

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Keshav, Satish, and Palak Trivedi. Genetic liver disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0214.

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This chapter discusses three of the major inherited forms of liver disease (all autosomal recessive): hereditary haemochromatosis, Wilson’s disease, and alpha-1-antitrypsin deficiency. Hereditary haemochromatosis is characterized by excessive absorption of dietary iron, with a pathological increase in total body iron that accumulates in tissues and organs, disrupting their function. Wilson’s disease (hepatolenticular degeneration) is an autosomal recessive genetic disorder in which copper accumulates in tissues. Alpha-1-antitrypsin deficiency is characterized by reduced circulating levels of a
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Rosenbaum, Richard B. Systemic Lupus Erythematosus. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0192.

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The neurological manifestations of systemic lupus erythematosus are protean: headache, affective disorders, cognitive dysfunction, seizures, strokes, psychosis, acute confusional states, myelopathies, chorea, mimics of demyelinating disease, meningitis, polyneuropathy, mononeuropathy or mononeuritis multiplex, cranial neuropathies, autonomic dysfunction, Guillain-Barre syndrome, or myasthenia gravis make an incomplete list. Each neurological manifestation needs to be analyzed separately to understand pathogenesis, possible relation to primary lupus-related inflammation and vasculopathy, and op
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Snell, Jamey, and Thomas J. Mancuso. Cystic Fibrosis. Edited by Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi, and Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0023.

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Cystic fibrosis (CF) is an inherited, autosomal recessive, multisystem disease. Dysfunction of the cystic fibrosis transmembrane conductance regulator protein (CFTR) in epithelial cells is the primary defect in CF. Defects in CFTR are the cause for lung disease, exocrine pancreatic insufficiency and failure, male infertility, and liver disease. CF can present with a variety of respiratory and gastrointestinal signs, including meconium ileus in the newborn period, hypernatremic dehydration, pulmonary insufficiency, nasal polyps, and insulin-dependent diabetes mellitus. As affected children grow
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Saleem, Moin A., and Corinne Antignac. Molecular basis of nephrotic syndrome. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0327_update_001.

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Nephrotic syndrome is broadly a disorder of the glomerular filtration barrier, but in practice the site of dysfunction in the great majority of pathologies is in the podocyte. Genetic causes of nephrotic syndrome provide the strongest proof of this. Almost all the genetic associations with nephrotic syndrome are podocyte proteins. Some basement membrane protein mutations associated with nephrotic syndrome may act through signalling to podocytes, or by causing severe disruption to their environment.
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Gray, Doug, Carole Proctor, and Tom Kirkwood. Biological aspects of human ageing. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0001.

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At the molecular and cellular levels human ageing is characterized by the accumulation of unrepaired random damage, and an accompanying loss of function. A major source of damage is oxidative stress caused by the generation of reactive oxygen species as a by-product of respiration. DNA and proteins are both susceptible to damage but whereas DNA damage repair systems exist, faulty proteins are generally removed by protein degradation systems. During ageing these systems become less efficient and the subsequent accumulation of damaged protein promotes protein aggregation, a process which is espe
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Kotagal, Vikas, and Praveen Dayalu. Parkinson Syndromes. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0005.

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Progressive supranuclear palsy (PSP) is a neurodegenerative condition characterized by axial motor features, oculomotor abnormalities, and cognitive dysfunction. PSP is characterized by progressive tau deposition with neuronal loss in cortical and subcortical regions. The underlying etiology of PSP may reflect complex gene-environment interactions, though genetic heterogeneity in the microtubule-associated protein tau (MAPT) gene can confer increased risk. Clinical care of patients with PSP focuses on minimizing motor and non-motor morbidity using available symptomatic therapies.
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Dionisi-Vici, Carlo, Diego Martinelli, Enrico Bertini, and Claude Bachmann. HHH Syndrome. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0020.

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Hyperornithinemia-hyperammonemia-homocitrullinuria (HHH) syndrome is an autosomal recessive disorder of the urea cycle characterized by impaired transport of ornithine across the inner mitochondrial membrane. As seen in other urea cycle defects, in the acute phase the disease is characterized by intermittent episodes of hyperammonemia accompanied by vomiting, lethargy, and coma, with or without signs of acute liver failure. The disease course is characterized by a pyramidal tract dysfunction associated with myoclonic seizures and cerebellar symptoms. Most patients reaching adulthood manifest v
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Book chapters on the topic "Dysfunctional protest"

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Hauck, Amy K., Dalay H. Olson, Joel S. Burrill, and David A. Bernlohr. "Adipose Carbonylation and Mitochondrial Dysfunction." In Protein Carbonylation. John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781119374947.ch12.

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Edwards, Yvonne H., and Dallas M. Swallow. "Mutation and protein dysfunction." In Protein Dysfunction in Human Genetic Disease. Garland Science, 2024. http://dx.doi.org/10.1201/9781003579953-1.

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Kaufman, Randal J., and Steven W. Pipe. "Factor VIII and haemophilia A." In Protein Dysfunction in Human Genetic Disease. Garland Science, 2024. http://dx.doi.org/10.1201/9781003579953-5.

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Bhattacharya, Shomi, Mai Al-Maghtheh, and Chris Inglehearn. "Rhodopsin mutations in photoreceptor degeneration: implications for protein dysfunction." In Protein Dysfunction in Human Genetic Disease. Garland Science, 2024. http://dx.doi.org/10.1201/9781003579953-10.

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Calandra, Sebastiano, and Stefano Bertolini. "The Iow-density Lipoprotein (LDL) receptor and familial hypercholesterolaemia." In Protein Dysfunction in Human Genetic Disease. Garland Science, 2024. http://dx.doi.org/10.1201/9781003579953-7.

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Mahuran, Don J. "GM2 gangliosidosis and structure–function relationships in β-hexosaminidase." In Protein Dysfunction in Human Genetic Disease. Garland Science, 2024. http://dx.doi.org/10.1201/9781003579953-6.

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Kilimann, Manfred W. "Glycogen storage disease due to phosphorylase kinase deficiency." In Protein Dysfunction in Human Genetic Disease. Garland Science, 2024. http://dx.doi.org/10.1201/9781003579953-4.

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Tiemann, Frank, Kiran Musunuru, and Philip W. Hinds. "The retinoblastoma tumour suppressor protein and cancer." In Protein Dysfunction in Human Genetic Disease. Garland Science, 2024. http://dx.doi.org/10.1201/9781003579953-9.

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Petersen, Robert B., Piero Parchi, Sabina Capellari, and Pierluigi Gambetti. "Fatal familial insomnia, Creutzfeldt-Jakob disease and the prion protein." In Protein Dysfunction in Human Genetic Disease. Garland Science, 2024. http://dx.doi.org/10.1201/9781003579953-13.

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Clegg, J. B. "Haemoglobin." In Protein Dysfunction in Human Genetic Disease. Garland Science, 2024. http://dx.doi.org/10.1201/9781003579953-2.

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Conference papers on the topic "Dysfunctional protest"

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Protasi, Feliciano. "Regular physical activity protects skeletal muscle fibers from age-related dysfunctional remodeling of sarcotubular system and mitochondria." In 2nd World conference on Ageing and Gerontology. United Research Forum, 2025. https://doi.org/10.51219/urforum.2025.feliciano-protasi.

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Liu, Zhi-Ping, Yong Wang, Tieqiao Wen, Xiang-Sun Zhang, Weiming Xia, and Luonan Chen. "Dynamically dysfunctional protein interactions in the development of Alzheimer's disease." In 2009 IEEE International Conference on Systems, Man and Cybernetics - SMC. IEEE, 2009. http://dx.doi.org/10.1109/icsmc.2009.5346814.

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Donaldson, V. H., and M. D. B. H. Mitchell. "INTERACTIONS OF DYSFUNCTIONAL Cl-INHIBITORS FROM PATIENTS WITH TYPE II HEREDITARY ANGIONEUROTIC EDEMA (HANE) WITH ACTIVATED HAGEMAN FACTOR (FACTOR XIIa)." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643302.

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Type II HANE is characterized by a deficiency of Cl-inhibitor (Cl-INH) activity in serum which is associated with a dysfunctional inhibitor protein having a normal or increased quantity o|_the antigenic properties of normal serum Cl-inhibitor. Dysfunctional Cl-INH proteins were purified from members_of eight different kindred with Type II HANE and compared to normal Cl-inhibitor with respect to their inhibitory activity directed against the amidolytic and clot-promoting properties of purified activated Hageman factor. All but one dysfunctional Cl-inhibitor blocked the amidolytic activity of el
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Singh, H., J. Safi, L. L. Price, P. LaCamera, and J. R. Tedrow. "Arterial Catheters May Protect Against Renal Dysfunction in Shock." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6705.

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Ali Mohammed, Imtithal. "Protective Role of Cranberry Extract Against Zovirax-Induced Spleen Dysfunction in Adult Female Wistar Rats." In XI. International Scientific Congress of Pure, Applied and Technological Sciences (MINAR Congress). Rimar Academy, 2024. https://doi.org/10.47832/minarcongress11-8.

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This research explored a possible protective function of antioxidants, such as cranberries, against zoviraxinduced spleen dysfunction in adult female Wistar rats. A collection of 24(adult female Wistar rats) was haphazardly appointed to four equal sets, each including six animals. They received the following treatment for (0–22–44) days per day. The first group, known as group (C), was obtained in (tap water), and served as a control. The second set (A1) obtained orally 150 mg/kg B.W. of cranberry alone. The third group (A2) received zovirax (450mg/kg B.W.) during the trial to elicit spleen to
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Figueroa, Dannielle S., and Alisa Morss Clyne. "A Cell Stretching System to Measure Endothelial Cell Cyclic Strain Response on Spatially Defined Basement Membrane." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206848.

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A principal concern in diabetes is the increased risk of vascular disease associated with unregulated blood glucose. Endothelial cells that line the vasculature are dysfunctional in hyperglycemia. The high glucose environment promotes basement membrane protein glycation and enhanced cross-linking, which can increase stiffness and may alter endothelial cell response to strain.
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Buu, MyMy C., John D. Mark, and David N. Cornfield. "Surfactant Protein Dysfunction Masquerading As Asthma In A Toddler." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a3752.

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Aman, Jurjan, Jan van Bezu, Amin Damansfshan, Victor W. van Hinsbergh, and Geerten P. van Nieuw Amerongen. "The Anticancer Drug Imatinib Protects Against Endothelial Barrier Dysfunction." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4184.

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Usatyuk, Peter, and Viswanathan Natarajan. "Mitochondrial Sod Protects Hyperoxia-Induced Dysfunction Of Lung Endothelium." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a5966.

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Figueroa, Dannielle Solomon, and Alisa Morss Clyne. "Basement Membrane Collagen Glycation Prevents Endothelial Cell Response to Strain due to Altered Focal Adhesion Formation." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53406.

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Vascular morbidity and mortality are primary complications of diabetes and have been correlated with unregulated blood glucose control. Endothelial cells that line the vasculature are known to be dysfunctional in hyperglycemia (1). Furthermore, the high glucose environment promotes basement membrane protein glycation and enhanced cross-linking. This can increase matrix stiffness, decrease matrix degradation, and potentially alter the spatial distribution of cell-matrix binding sites.
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Reports on the topic "Dysfunctional protest"

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Neodo, Anna, Fiona Augsburger, Jan Waskowski, Joerg C. Schefold, and Thibaud Spinetti. Monocytic HLA-DR expression and clinical outcomes in adult ICU patients with sepsis – a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.11.0119.

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Review question / Objective: The scope of this review was defined using PICOTS framework where 1) population: adult critically ill patients with sepsis or septic shock; 2) index prognostic factor: cell surface protein expression of mHLA-DR in blood; 3) comparative factor: none; 4) outcomes to be predicted: mortality, secondary infections, length of stay, and organ dysfunction score (sequential organ failure assessment [SOFA], multiple organ dysfunction score [MODS], logistic organ dysfunction score [LODS]), composite outcomes where component endpoints consist of at least one of the outcomes st
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Patumraj, Suthiluk, and Sheepsumon Viboolvorakul. Protective effects of exercise training against vascular and neuronal dysfunction in aging brain. Department of Physiology, Faculty of Medicine, Chulalongkorn University, 2019. https://doi.org/10.58837/chula.res.2019.11.

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During advancing age, reduction of microvessels in the brain contributes insufficiency tissue perfusion. Mounting evidence indicates that microvascular deterioration in aged brain relates to oxidative stress. Nuclear factor erythroid-related factor 2 (Nrf2) plays an important role in cellular antioxidant defense. Regular physical exercise is well known to have beneficial effect to brain health, including promoted blood flow and augmented angiogenesis, in aging individuals. However, the underlying mechanism of regular physical exercise in improvement of brain microvascular density during advanc
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Czosnek, Henryk Hanokh, Dani Zamir, Robert L. Gilbertson, and Lucas J. William. Resistance to Tomato Yellow Leaf Curl Virus by Combining Expression of a Natural Tolerance Gene and a Dysfunctional Movement Protein in a Single Cultivar. United States Department of Agriculture, 2000. http://dx.doi.org/10.32747/2000.7573079.bard.

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Background The tomato yellow leaf curl disease (TYLCV) has been a major deterrent to tomato production in Israel for the last 20 years. This whitefly-transmitted viral disease has been found in the Caribbean Island in the early 1990s, probably as an import from the Middle East. In the late 1990s, the virus has spread to the US and is now conspicuous in Florida and Georgia. Objectives Because of the urgency facing the TYLCV epidemics, there was a compelling need to mobilize scientists to develop tomato variety resistant to TYLCV. The major goal was to identify the virus movement protein (MP) an
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Block, Michelle L. The Role of Protein Radicals in Chronic Neuroimmune Dysfunction and Neuropathology in Response to a Multiple-Hit Model of Gulf War Exposure. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada613307.

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