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Journal articles on the topic 'Dyslexia – genetics'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Pennington, Bruce F. "The Genetics of Dyslexia." Journal of Child Psychology and Psychiatry 31, no. 2 (1990): 193–201. http://dx.doi.org/10.1111/j.1469-7610.1990.tb01561.x.

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2

GRIFFIN, JOHN R. "Genetics of Dyseidetic Dyslexia." Optometry and Vision Science 69, no. 2 (1992): 148–51. http://dx.doi.org/10.1097/00006324-199202000-00008.

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3

Scerri, Thomas S., and Gerd Schulte-Körne. "Genetics of developmental dyslexia." European Child & Adolescent Psychiatry 19, no. 3 (2009): 179–97. http://dx.doi.org/10.1007/s00787-009-0081-0.

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4

Williams, Julie, and Michael C. O'Donovan. "The genetics of developmental dyslexia." European Journal of Human Genetics 14, no. 6 (2006): 681–89. http://dx.doi.org/10.1038/sj.ejhg.5201575.

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5

Pennington, Bruce F. "Using genetics to understand dyslexia." Annals of Dyslexia 39, no. 1 (1989): 81–93. http://dx.doi.org/10.1007/bf02656902.

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6

Bishop, D. V. M. "The interface between genetics and psychology: lessons from developmental dyslexia." Proceedings of the Royal Society B: Biological Sciences 282, no. 1806 (2015): 20143139. http://dx.doi.org/10.1098/rspb.2014.3139.

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Developmental dyslexia runs in families, and twin studies have confirmed that there is a substantial genetic contribution to poor reading. The way in which discoveries in molecular genetics are reported can be misleading, encouraging us to think that there are specific genes that might be used to screen for disorder. However, dyslexia is not a classic Mendelian disorder that is caused by a mutation in a single gene. Rather, like many other common disorders, it appears to involve combined effects of many genes and environmental factors, each of which has a small influence, possibly supplemented

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7

Wilcke, Arndt, Carolin Ligges, Jana Burkhardt, et al. "Imaging genetics of FOXP2 in dyslexia." European Journal of Human Genetics 20, no. 2 (2011): 224–29. http://dx.doi.org/10.1038/ejhg.2011.160.

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8

Schumacher, J., P. Hoffmann, C. Schmal, G. Schulte-Korne, and M. M. Nothen. "Genetics of dyslexia: the evolving landscape." Journal of Medical Genetics 44, no. 5 (2007): 289–97. http://dx.doi.org/10.1136/jmg.2006.046516.

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9

Carrion-Castillo, Amaia, Barbara Franke, and Simon E. Fisher. "Molecular Genetics of Dyslexia: An Overview." Dyslexia 19, no. 4 (2013): 214–40. http://dx.doi.org/10.1002/dys.1464.

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10

Shastry, Barkur S. "Developmental dyslexia: an update." Journal of Human Genetics 52, no. 2 (2006): 104–9. http://dx.doi.org/10.1007/s10038-006-0088-z.

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11

Wood, Frank B., and Elena L. Grigorenko. "Emerging Issues in the Genetics of Dyslexia." Journal of Learning Disabilities 34, no. 6 (2001): 503–11. http://dx.doi.org/10.1177/002221940103400603.

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12

Kere, Juha. "Molecular genetics and molecular biology of dyslexia." Wiley Interdisciplinary Reviews: Cognitive Science 2, no. 4 (2011): 441–48. http://dx.doi.org/10.1002/wcs.138.

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13

Wilcke, Arndt, Carolin Ligges Jana Burkhardt, Michael Alexander, et al. "Erratum: Imaging genetics of FOXP2 in dyslexia." European Journal of Human Genetics 20, no. 6 (2012): 714. http://dx.doi.org/10.1038/ejhg.2012.31.

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14

Degrandi, Roberta, Flavia Prodam, Giulia Genoni, et al. "The Prevalence of Thyroid Autoimmunity in Children with Developmental Dyslexia." BioMed Research International 2021 (February 8, 2021): 1–5. http://dx.doi.org/10.1155/2021/7656843.

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Background and Objectives. An association between developmental dyslexia and autoimmune disorders has been postulated. We aimed to evaluate the prevalence of thyroid autoimmunity in pediatric subjects with developmental dyslexia. Methods. We enrolled pediatric subjects with developmental dyslexia and, as a control group, healthy age- and sex-matched subjects without developmental dyslexia. Thyroid function was evaluated in subjects with developmental dyslexia measuring serum concentrations of thyroid-stimulating hormone (TSH), free triiodothyronine (fT3), and free thyroxine (fT4). Thyroid auto

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15

Woollams, Anna M. "Connectionist neuropsychology: uncovering ultimate causes of acquired dyslexia." Philosophical Transactions of the Royal Society B: Biological Sciences 369, no. 1634 (2014): 20120398. http://dx.doi.org/10.1098/rstb.2012.0398.

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Acquired dyslexia offers a unique window on to the nature of the cognitive and neural architecture supporting skilled reading. This paper provides an integrative overview of recent empirical and computational work on acquired dyslexia within the context of the primary systems framework as implemented in connectionist neuropsychological models. This view proposes that damage to general visual, phonological or semantic processing abilities are the root causes of different forms of acquired dyslexia. Recent case-series behavioural evidence concerning pure alexia, phonological dyslexia and surface

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16

Müller, Bent, Johannes Boltze, Ivonne Czepezauer, Volker Hesse, Arndt Wilcke, and Holger Kirsten. "Dyslexia risk variant rs600753 is linked with dyslexia-specific differential allelic expression of DYX1C1." Genetics and Molecular Biology 41, no. 1 (2018): 41–49. http://dx.doi.org/10.1590/1678-4685-gmb-2017-0165.

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17

OVERY, KATIE. "Dyslexia and Music." Annals of the New York Academy of Sciences 999, no. 1 (2003): 497–505. http://dx.doi.org/10.1196/annals.1284.060.

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18

Carrion-Castillo, Amaia, Sara B. Estruch, Ben Maassen, Barbara Franke, Clyde Francks, and Simon E. Fisher. "Whole-genome sequencing identifies functional noncoding variation in SEMA3C that cosegregates with dyslexia in a multigenerational family." Human Genetics 140, no. 8 (2021): 1183–200. http://dx.doi.org/10.1007/s00439-021-02289-w.

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AbstractDyslexia is a common heritable developmental disorder involving impaired reading abilities. Its genetic underpinnings are thought to be complex and heterogeneous, involving common and rare genetic variation. Multigenerational families segregating apparent monogenic forms of language-related disorders can provide useful entrypoints into biological pathways. In the present study, we performed a genome-wide linkage scan in a three-generational family in which dyslexia affects 14 of its 30 members and seems to be transmitted with an autosomal dominant pattern of inheritance. We identified

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19

Rello, Luz, Ricardo Baeza-Yates, Abdullah Ali, Jeffrey P. Bigham, and Miquel Serra. "Predicting risk of dyslexia with an online gamified test." PLOS ONE 15, no. 12 (2020): e0241687. http://dx.doi.org/10.1371/journal.pone.0241687.

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Dyslexia is a specific learning disorder related to school failure. Detection is both crucial and challenging, especially in languages with transparent orthographies, such as Spanish. To make detecting dyslexia easier, we designed an online gamified test and a predictive machine learning model. In a study with more than 3,600 participants, our model correctly detected over 80% of the participants with dyslexia. To check the robustness of the method we tested our method using a new data set with over 1,300 participants with age customized tests in a different environment -a tablet instead of a

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20

Marino, Cecilia, Haiying Meng, Sara Mascheretti, et al. "DCDC2 genetic variants and susceptibility to developmental dyslexia." Psychiatric Genetics 22, no. 1 (2012): 25–30. http://dx.doi.org/10.1097/ypg.0b013e32834acdb2.

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21

Schulte-Körne, Gerd, Kerstin U. Ludwig, Jennifer el Sharkawy, Markus M. Nöthen, Bertram Müller-Myhsok, and Per Hoffmann. "Genetics and Neuroscience in Dyslexia: Perspectives for Education and Remediation." Mind, Brain, and Education 1, no. 4 (2007): 162–72. http://dx.doi.org/10.1111/j.1751-228x.2007.00017.x.

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22

Nopola-Hemmi, J. "Two translocations of chromosome 15q associated with dyslexia." Journal of Medical Genetics 37, no. 10 (2000): 771–75. http://dx.doi.org/10.1136/jmg.37.10.771.

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23

Schulte-Körne, G., A. Ziegler, W. Deimel, et al. "Interrelationship and Familiality of Dyslexia Related Quantitative Measures." Annals of Human Genetics 71, no. 2 (2007): 160–75. http://dx.doi.org/10.1111/j.1469-1809.2006.00312.x.

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24

LLINÁS, RODOLFO. "Is Dyslexia a Dyschronia?" Annals of the New York Academy of Sciences 682, no. 1 Temporal Info (1993): 48–56. http://dx.doi.org/10.1111/j.1749-6632.1993.tb22958.x.

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25

Folia, Vasiliki, Julia Uddén, Christian Forkstam, Martin Ingvar, Peter Hagoort, and Karl Magnus Petersson. "Implicit Learning and Dyslexia." Annals of the New York Academy of Sciences 1145, no. 1 (2008): 132–50. http://dx.doi.org/10.1196/annals.1416.012.

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26

Eden, Guinevere F., and Chandan J. Vaidya. "ADHD and Developmental Dyslexia." Annals of the New York Academy of Sciences 1145, no. 1 (2008): 316–27. http://dx.doi.org/10.1196/annals.1416.022.

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27

Snowling, Maggie, and Jules Davidoff. "Visual deficits in dyslexia?" Current Biology 2, no. 4 (1992): 196–97. http://dx.doi.org/10.1016/0960-9822(92)90525-f.

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28

Wolff, Peter H., and Ilze Melngailis. "Family patterns of developmental dyslexia: Clinical findings." American Journal of Medical Genetics 54, no. 2 (1994): 122–31. http://dx.doi.org/10.1002/ajmg.1320540207.

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29

Velayos-Baeza, Antonio, Claudio Toma, Stephanie da Roza, Silvia Paracchini, and Anthony P. Monaco. "Alternative splicing in the dyslexia-associated gene KIAA0319." Mammalian Genome 18, no. 9 (2007): 627–34. http://dx.doi.org/10.1007/s00335-007-9051-3.

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30

Pedroli, Elisa, Patrizia Padula, Andrea Guala, Maria Teresa Meardi, Giuseppe Riva, and Giovanni Albani. "A Psychometric Tool for a Virtual Reality Rehabilitation Approach for Dyslexia." Computational and Mathematical Methods in Medicine 2017 (2017): 1–6. http://dx.doi.org/10.1155/2017/7048676.

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Dyslexia is a chronic problem that affects the life of subjects and often influences their life choices. The standard rehabilitation methods all use a classic paper and pencil training format but these exercises are boring and demanding for children who may have difficulty in completing the treatments. It is important to develop a new rehabilitation program that would help children in a funny and engaging way. A Wii-based game was developed to demonstrate that a short treatment with an action video game, rather than phonological or orthographic training, may improve the reading abilities in dy

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31

Liu, Lingfei, Huaiting Gu, Fang Hou, et al. "Dyslexia associated functional variants in Europeans are not associated with dyslexia in Chinese." American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 180, no. 7 (2019): 488–95. http://dx.doi.org/10.1002/ajmg.b.32750.

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32

Ramachandra, NB, and P. Saviour. "Modes of genetic transmission of dyslexia in south Indian families." Indian Journal of Human Genetics 11, no. 3 (2005): 135. http://dx.doi.org/10.4103/0971-6866.19532.

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33

Nopola-Hemmi, J. "A dominant gene for developmental dyslexia on chromosome 3." Journal of Medical Genetics 38, no. 10 (2001): 658–64. http://dx.doi.org/10.1136/jmg.38.10.658.

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34

Grigorenko, Elena L., Frank B. Wood, Lina Golovyan, Marianne Meyer, Christina Romano, and David Pauls. "Continuing the search for dyslexia genes on 6p." American Journal of Medical Genetics 118B, no. 1 (2003): 89–98. http://dx.doi.org/10.1002/ajmg.b.10032.

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35

Eicher, John D., and Jeffrey R. Gruen. "Imaging-genetics in dyslexia: Connecting risk genetic variants to brain neuroimaging and ultimately to reading impairments." Molecular Genetics and Metabolism 110, no. 3 (2013): 201–12. http://dx.doi.org/10.1016/j.ymgme.2013.07.001.

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36

Walsh, Vincent. "Dyslexia: Reading between the laminae." Current Biology 5, no. 11 (1995): 1216–17. http://dx.doi.org/10.1016/s0960-9822(95)00240-5.

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37

STEIN, JOHN. "Dyslexia?Impaired Temporal Information Processing?" Annals of the New York Academy of Sciences 682, no. 1 Temporal Info (1993): 83–86. http://dx.doi.org/10.1111/j.1749-6632.1993.tb22961.x.

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38

FAWCETT, A. J., A. K. CHATTOPADHYAY, R. H. KANDLER, J. A. JARRATT, R. I. NICOLSON, and M. PROCTOR. "Event-related Potentials and Dyslexia." Annals of the New York Academy of Sciences 682, no. 1 Temporal Info (1993): 342–45. http://dx.doi.org/10.1111/j.1749-6632.1993.tb22988.x.

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39

MARTOS, FRANCISCO J., and ANTONIO MARMOLEJO. "Visible Persistence in Developmental Dyslexia." Annals of the New York Academy of Sciences 682, no. 1 Temporal Info (1993): 383–86. http://dx.doi.org/10.1111/j.1749-6632.1993.tb23000.x.

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40

Schumacher, Johannes, Heidi Anthoni, Faten Dahdouh, et al. "Strong Genetic Evidence of DCDC2 as a Susceptibility Gene for Dyslexia." American Journal of Human Genetics 78, no. 1 (2006): 52–62. http://dx.doi.org/10.1086/498992.

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41

Nishiyama, Kumiko V., Yoko Satta, and Jun Gojobori. "Do Genes Associated with Dyslexia of Chinese Characters Evolve Neutrally?" Genes 11, no. 6 (2020): 658. http://dx.doi.org/10.3390/genes11060658.

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Dyslexia, or reading disability, is found to have a genetic basis, and several related genes have been reported. We investigated whether natural selection has acted on single nucleotide polymorphisms (SNPs) that were reported to be associated with risk/non-risk for the reading disability of Chinese characters. We applied recently developed 2D SFS-based statistics to SNP data of East Asian populations to examine whether there is any sign of selective sweep. While neutrality was not rejected for most SNPs, significant signs of selection were detected for two linkage disequilibrium (LD) regions c

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42

Conway, Aisling, Nuala Brady, and Karuna Misra. "Holistic word processing in dyslexia." PLOS ONE 12, no. 11 (2017): e0187326. http://dx.doi.org/10.1371/journal.pone.0187326.

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43

Hall, Colby, and Sharon Vaughn. "Current Research Informing the Conceptualization, Identification, and Treatment of Dyslexia Across Orthographies: An Introduction to the Special Series." Learning Disability Quarterly 44, no. 3 (2021): 140–44. http://dx.doi.org/10.1177/0731948720929010.

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This introduction to the special series summarizes evidence for the genetic and brain bases for dyslexia and cognitive–behavioral indicators (including ones that can be measured even before the onset of reading instruction) that attest to meaningful differences between children with dyslexia and their non-dyslexic peers. Authors review controversies that have surrounded approaches to dyslexia identification and treatment during the last few decades. Finally, they introduce the findings of the articles in the special series and discuss potential implications for dyslexia identification and trea

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44

PESTALOZZI, D. "Ophthalmologic Aspects of Dyslexia: The Influence of Full Prismatic Correction of Heterophoria on Dyslexic Symptoms." Annals of the New York Academy of Sciences 682, no. 1 Temporal Info (1993): 397–99. http://dx.doi.org/10.1111/j.1749-6632.1993.tb23004.x.

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45

Bender, Bruce G., Mary H. Puck, James A. Salbenblatt, and Arthur Robinson. "Dyslexia in 47,XXXY boys identified at birth." Behavior Genetics 16, no. 3 (1986): 343–54. http://dx.doi.org/10.1007/bf01071315.

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46

Wolff, Peter H., Ilze Melngailis, Mateo Obregon, and Martha Bedrosian. "Family patterns of developmental dyslexia, part II: Behavioral phenotypes." American Journal of Medical Genetics 60, no. 6 (1995): 494–505. http://dx.doi.org/10.1002/ajmg.1320600604.

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47

Hsu, Li, Ellen M. Wijsman, Virginia W. Berninger, Jennifer B. Thomson, and Wendy H. Raskind. "Familial aggregation of dyslexia phenotypes. II: Paired correlated measures." American Journal of Medical Genetics 114, no. 4 (2002): 471–78. http://dx.doi.org/10.1002/ajmg.10523.

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48

Petryshen, T. L. "Supportive evidence for the DYX3 dyslexia susceptibility gene in Canadian families." Journal of Medical Genetics 39, no. 2 (2002): 125–26. http://dx.doi.org/10.1136/jmg.39.2.125.

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49

Ozer, O., Z. Yilmaz, E. Simsek, M. Derbent, S. Guner, and F. Sahin. "Two Patients with X Chromosome Duplication: dupXp and dupXq." Balkan Journal of Medical Genetics 12, no. 2 (2009): 59–63. http://dx.doi.org/10.2478/v10034-010-0008-6.

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Two Patients with X Chromosome Duplication: dupXp and dupXqStructural abnormalities of the X chromosome may lead to different phenotypes, depending on the chromosome region affected. We report phenotypic findings of two patients who had X chromosome duplications. One had a menstrual irregularity, a low hairline, cubitus valgus and suffered from dyslexia. The other had multiple congenital anomalies, severe mental-motor retardation and intractable epilepsy. The karyotypes were 46,X,dup(X) (p11.3p21) and 46,X,dup(X)(q13q25) respectively.

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50

DENCKLA, MARTHA BRIDGE. "A Neurologist's Overview of Developmental Dyslexia." Annals of the New York Academy of Sciences 682, no. 1 Temporal Info (1993): 23–26. http://dx.doi.org/10.1111/j.1749-6632.1993.tb22956.x.

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