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Journal articles on the topic 'Dysplasie osseuse'

Author: Grafiati

Published: 4 June 2021

Last updated: 8 February 2022

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1

Ouadnouni, Y., M. Smahi, M. Bouchikh, A. Achir, Y. Msougar, M. Lakranbi, M. Caidi, L. Harrak, A. S. Alaziz, and A. Benosman. "121 Dysplasie fibreuse osseuse." Revue des Maladies Respiratoires 24 (January 2007): 49. http://dx.doi.org/10.1016/s0761-8425(07)72497-x.

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2

Benazzou, S., M. Boulaadas, A. El Ayoubi, N. Nazih, L. Essakalli, and M. Kzadri. "Dysplasie cémento-osseuse floride des maxillaires." Revue de Stomatologie et de Chirurgie Maxillo-faciale 112, no. 3 (June 2011): 174–76. http://dx.doi.org/10.1016/j.stomax.2011.01.004.

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3

Couturier, A., O. Aumaître, T. Mom, L. Gilain, and M. André. "La dysplasie fibreuse osseuse cranio-faciale." La Revue de Médecine Interne 37, no. 12 (December 2016): 834–39. http://dx.doi.org/10.1016/j.revmed.2016.02.008.

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4

André, J. L., P. Bordigoni, M. J. Krier, N. Cheurfa, and B. Leheup. "Syndrome néphrotique et dysplasie spondyloépiphysaire :Dysplasie immuno-osseuse de Schimke." Archives de Pédiatrie 5, no. 7 (July 1998): 824–25. http://dx.doi.org/10.1016/s0929-693x(98)80104-8.

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5

Maccotta, M., and L. Radoï. "Dysplasie osseuse floride : gestion d’un cas symptomatique." Revue de Stomatologie, de Chirurgie Maxillo-faciale et de Chirurgie Orale 117, no. 6 (December 2016): 425–28. http://dx.doi.org/10.1016/j.revsto.2016.10.004.

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6

Ardizzone, M., P. Khorshidi, T. Weitten, L. Sparsa, J. Durckel, J. L. Kuntz, and N. Afif. "Un cas rare de dysplasie osseuse: la Mélorhéostose." Revue du Rhumatisme 74, no. 10-11 (November 2007): 1173–74. http://dx.doi.org/10.1016/j.rhum.2007.10.352.

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7

Alexandre, Christian. "La maladie osseuse de Paget et la dysplasie fibreuse." Revue du Rhumatisme 70, no. 8 (September 2003): 631–32. http://dx.doi.org/10.1016/s1169-8330(03)00284-9.

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8

Bouriche, K., D. Senouci, W. Mazari, and F. Guellai. "P-304 – Chimke immuno-osseuse dysplasie: à propos d'un cas." Archives de Pédiatrie 22, no. 5 (May 2015): 310–11. http://dx.doi.org/10.1016/s0929-693x(15)30484-x.

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9

Couturier, A., O. Aumaître, L. Gilain, B. Jean, T. Mom, and M. André. "Dysplasie fibreuse osseuse cranio-faciale : à propos de 10 cas." Annales françaises d'Oto-rhino-laryngologie et de Pathologie Cervico-faciale 134, no. 4 (September 2017): 224–30. http://dx.doi.org/10.1016/j.aforl.2016.09.002.

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10

Hamdi, H., W. Hasni, T. Ben Alaya, and M. Ben Khelifa. "O 17-2 Dysplasie cémento-osseuse des maxillaires : du diagnostic au traitement." Revue de Stomatologie et de Chirurgie Maxillo-faciale 106, no. 4 (September 2005): 63–64. http://dx.doi.org/10.1016/s0035-1768(05)86019-x.

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11

Boulate, G., C. Cormier, G. Assie, L. Guignat, L. Groussin, X. Bertagna, and J. Bertherat. "Déminéralisation osseuse dans l’hypercortisolisme secondaire à la dysplasie micronodulaire pigmentée des surrénales (PPNAD)." Annales d'Endocrinologie 77, no. 4 (September 2016): 426–27. http://dx.doi.org/10.1016/j.ando.2016.07.963.

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12

Belhimer, F., and S. Fedala. "Métastases osseuses isolées d’un phéochromocytome chez une patiente atteinte de neurofibromatose : difficultés diagnostiques en présence de lésions de dysplasie osseuse." Annales d'Endocrinologie 79, no. 4 (September 2018): 396. http://dx.doi.org/10.1016/j.ando.2018.06.649.

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13

Khonsari, R. H., P. Corre, J. Bouguila, and B. Piot. "Évolution à long terme de la dysplasie cémento-osseuse : une récidive controlatérale avant radiothérapie." Revue de Stomatologie et de Chirurgie Maxillo-faciale 112, no. 1 (February 2011): 51–53. http://dx.doi.org/10.1016/j.stomax.2009.02.006.

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14

Massereau, Eugénie, Ugo Ordioni, Maud Guivarc’h, Guillaume Royer, and Jean-Hugues Catherine. "Dysplasie osseuse floride mandibulaire : un cas de découverte fortuite et revue de la littérature." Médecine Buccale Chirurgie Buccale 21, no. 2 (April 2015): 101–4. http://dx.doi.org/10.1051/mbcb/2015012.

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15

Couturier, A., A. Mania, A. Bardy, N. Saroul, T. Mom, O. Aumaître, and M. Andre. "Une cause rare de céphalées et de troubles neurosensoriels : la dysplasie fibreuse osseuse cranio-faciale." La Revue de Médecine Interne 35 (December 2014): A178—A179. http://dx.doi.org/10.1016/j.revmed.2014.10.316.

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16

Benbellal, Amina, Hanène Belabbassi, Sarrah Ait Ziane, and Houria Kaced. "Management of type IV A mucopolysaccharidosis or Marquio A syndrome (A case report)." Batna Journal of Medical Sciences (BJMS) 6, no. 2 (December 30, 2019): 121–24. http://dx.doi.org/10.48087/bjmscr.2019.6209.

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La maladie de Morquio A, ou mucopolysaccharidose de type IV A (Morquio A syndrome, MPS IVA), est une maladie génétique rare ; multisystémique, et extrêmement invalidante. Elle est liée à un déficit enzymatique en N-acétylgalactosamine-6-sulfate sulfatase (GALNS), enzyme lysosomale responsable de la dégradation du kératane sulfate (KS) et de la chondroïtine-6-sulfate (C6S), éléments présents principalement dans le cartilage et la cornée. Cette maladie métabolique se manifeste principalement par une atteinte osseuse constante, sous forme d’une dysplasie spondylo-épi-métaphysaire progressive, et des complications ophtalmologiques, auditives et cardiaques plus modérées d’apparition tardive. Nous relatons le cas d’un enfant âgé de 8 ans qui présente cette pathologie, en étayant ses caractéristiques cliniques, et ses modalités thérapeutiques pluridisciplinaires.

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17

Ndiaye, A. R., J. Magnin, I. Imbert, J. Samy, B. Havet, J. P. Leberre, and D. Lechevalier. "Le Syndrome de Mac Cune et Albright, une dysplasie fibreuse osseuse associée à une puberté précoce." La Revue de Médecine Interne 27 (December 2006): S372. http://dx.doi.org/10.1016/j.revmed.2006.10.206.

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18

Berete-Coulibaly, R., C. Vignal Clermont, A. Russo, O. Galatoire, F. Mann, and S. Morax. "243-1 Dysplasie osseuse fibreuse orbito-cérébro-faciale dans le syndrome de Turner : une exophtalmie bilatérale révélatrice." Journal Français d'Ophtalmologie 28 (March 2005): 217. http://dx.doi.org/10.1016/s0181-5512(05)74639-8.

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19

Boudriga, H., M. Ben Fredj, A. Ezzine, S. Mensi, S. Ajmi, K. Chatti, and M. Guezguez. "Scintigraphie osseuse au 99mTc-HMDP et dysplasie fibreuse polyostotique dans le cadre du syndrome de McCune–Albright : à propos d’un cas." Annales d'Endocrinologie 78, no. 4 (September 2017): 377. http://dx.doi.org/10.1016/j.ando.2017.07.763.

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20

Djigo, M. S., B. Ndong, E. Bathily, and O. Diop. "Contribution de la scintigraphie osseuse TEMP : à propos d’un cas de dysplasie fibreuse humérale au service de médecine nucléaire de l’hôpital général de Dakar." Médecine Nucléaire 45, no. 4 (July 2021): 230–31. http://dx.doi.org/10.1016/j.mednuc.2021.06.125.

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21

Onor, Gabriel I., Sercan Yalcin, Scott G. Kaar, J. Lee Pace, Paolo Ferrua, and Lutul D. Farrow. "The Evaluation of Trochlear Osseous Morphology: An Epidemiologic Study." Orthopaedic Journal of Sports Medicine 9, no. 4 (April 1, 2021): 232596712199454. http://dx.doi.org/10.1177/2325967121994548.

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Background: The femoral trochlea is considered the most significant osseous factor affecting stability in the patellofemoral joint. The true prevalence of trochlear dysplasia in the general population is largely unknown. Purpose/Hypothesis: To investigate the prevalence of trochlear dysplasia in the general population. Our hypothesis was that, while trochlear dysplasia is not uncommon, there is a low prevalence of severe dysplasia in the general population. Study Design: Descriptive epidemiology study. Methods: Five observers were asked to evaluate 692 skeletally mature femoral specimens from 359 skeletons for trochlear dysplasia at 2 time points. We further subclassified the dysplastic trochlea in 62 femora with the highest rated degree of dysplasia. Results: Sex ( P = .11) and race ( P = .2) had no effect on the severity of dysplasia. Interobserver reliability was excellent (0.906 and 0.904), and intraobserver reliability was good to excellent (0.686 to 0.808). The percentages of trochlea graded as normal, mildly dysplastic, moderately dysplastic, and severely dysplastic were 61.5%, 21.4%, 12.7%, and 4.4%, respectively, in the first evaluation, and 58.5%, 23.7%, 12.7%, and 5.1% in the second evaluation. Of the 62 trochlea with the highest scores for dysplasia, 36 had trochlear dysplasia without a supratrochlear spur, 8 had trochlear dysplasia with medial femoral condyle hypoplasia, and 18 had trochlear dysplasia with a supratrochlear spur. Conclusion: Observers with differing degrees of clinical experience had similar opinions on the degree of trochlear dysplasia. Also, our cohort showed that moderate to severe dysplasia is not uncommon, as it is present in approximately 17% of knees in our cohort. Our findings also suggest that clinicians are speaking the same language when identifying and describing trochlear dysplasia on gross inspection.

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22

Onor, Gabriel, Bonnie Mason, and Lutul Farrow. "THE EVALUATION OF SUPRATROCHLEAR OSSEOUS MORPHOLOGY: AN EPIDEMIOLOGIC STUDY." Orthopaedic Journal of Sports Medicine 7, no. 3_suppl (March 1, 2019): 2325967119S0009. http://dx.doi.org/10.1177/2325967119s00095.

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Introduction: Patellar instability can impede activities of daily life. The morphology of the femoral trochlea has significant involvement in maintaining patellofemoral joint stability. Trochlear dysplasia, or a shallow trochlear groove, has been implicated as a risk factor for recurrent patellar dislocation. A supratrochlear spur in the setting of existing trochlear dysplasia can generate further patellar instability. Trochleoplasty has been popularized by some surgeons for the treatment of recurrent patellar instability in the setting of significant trochlear dysplasia. Despite recommendation of trochleoplasty in treatment of patellar instability in the setting of trochlear dysplasia, much is unknown about the prevalence of trochlear dysplasia and supratrochlear spurs in the general population. The purpose of this study is to investigate the prevalence of supratrochlear spurs and trochlear dysplasia in the population and investigate the relationship between supratrochlear spurs and dysplastic femoral trochleae. Methods: This study was performed at the Cleveland Museum of Natural History, the Hamann Todd Human Osteological Collection. With over 4,000 complete human skeletons, the Hamann Todd osteological collection at the Cleveland Museum of Natural History is the largest collection of osteological specimens with documented demographic data in the Western Hemisphere. We evaluated femora from over 1,000 individuals aged 16-40 years. Each specimen was evaluated for the presence of trochlear dysplasia and supratrochlear spurs. We excluded skeletally immature specimens as well as specimens with visible signs of osteoarthritis or abnormal, bony deformity. The presence or absence of supratrochlear spurs was documented. Abnormal femoral trochleae were photographed in A/P, axial, and lateral views. The width and length of supratrochlear spurs were measured using digital calipers. Results: Of the 1,148 specimens studied, 135 femora from 93 individuals were found to be dysplastic. Amongst these, 9 femora with trochlear dysplasia had supratrochlear spurs. The average age of individuals with dysplastic trochleae in this study was 32.86. Of the 93 specimens identified, 24 were female (25.8%). Additionally, 52% of individuals with dysplastic trochleae were white while 48% were black. Conclusions: There may be an association of supratrochlear spurs with trochlear dysplasia. Though 25.8% of individuals with dysplastic trochleae were female, it is important to note that the Hamann Todd Collection as a whole is 19% female.

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23

Gyawali, Bigyan Raj, and Rabindra Bhakta Pradhananga. "Monostotic fibrous dysplasia of temporal bone: a case series and literature review." International Journal of Scientific Reports 6, no. 4 (March 24, 2020): 167. http://dx.doi.org/10.18203/issn.2454-2156.intjscirep20201273.

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<p>Fibrous dysplasia is a benign, progressive fibro-osseus disease. Typically, there are three variants monostotic, polyostotic and McCune Albright syndrome. Monostotic variant being the commonest of all, affects mainly the craniofacial bones and ribs. Fibrous dysplasia affecting the temporal bone may be challenging at times. Here, we present our case series of three temporal bone fibrous dysplasias along with the literature review.</p>

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24

Natrajan, Srivalli, Usha Asnani, Jyotsna S. Galinde, and Mahesh Sonar. "Florid Cemento-Osseous Dysplasia." Journal of Contemporary Dentistry 2, no. 1 (2012): 53–56. http://dx.doi.org/10.5005/jcd-2-1-53.

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Abstract Florid cemento-osseous dysplasia (FCOD) is an uncommon benign multifocal dysplastic lesion affecting the jaws that consists of cellular fibrous connective tissue with bone and cementum like tissue. It is most commonly seen in middle aged black woman although it can occur in Caucasians and Asians. A case of FCOD in a 63 year old Indian woman with unique radiographic and clinical presentation is reported.

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25

Akbulut, Sevtap, Mehmet Gökhan Demir, Kayhan Basak, and Mustafa Paksoy. "Maxillectomy for Cementifying Osseous Dysplasia of the Maxilla: A Case Report." Acta Medica (Hradec Kralove, Czech Republic) 58, no. 1 (2015): 32–34. http://dx.doi.org/10.14712/18059694.2015.89.

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Objectives: Osseous dysplasias are the most common subtype of fibro-osseous lesions of the maxillofacial bones. They are benign and often present as incidental asymptomatic lesions. Diagnosis can be made with clinical and radiographic examination. Case report: This article reports the case of a 47-year-old man with a huge cementifying osseous dysplasia of the maxilla that presented with secondary infection after teeth extraction and repair of oroantral fistula. A subtotal maxillectomy had to be done after conservative treatment failed to resolve the infection. Discussion/Conclusion: Biopsy, tooth extraction or surgical trauma to the affected bones of osseous dysplasia could easily lead to severe secondary infection, the treatment of which may be very difficult, and thus should be avoided if possible.

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26

Bagheri, Fatemeh, Jamileh Bigom Taheri, Somayyeh Azimi, and Zahra Elmi. "Florid Cemento-osseous Dysplasia: Report of Three Unusual Cases." International Journal of Experimental Dental Science 3, no. 2 (2014): 103–5. http://dx.doi.org/10.5005/jp-journals-10029-1081.

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ABSTRACT Florid cemento-osseous dysplasia (FLCOD) is a reactive process that involves tooth-bearing areas of the jaws. Reactive or dysplastic changes in periodontal ligament have been suggested as an etiologic factor. It usually affects females and is less frequently seen in Caucasians and Asians groups. Multiple sclerotic masses involving more than one quadrant are it is radiographic appearance. Because of its benign nature periodic recall is all things to do but in symptomatic cases surgical intervention may be required. How to cite this article Bagheri F, Taheri JB, Azimi S, Elmi Z. Florid Cemento-osseous Dysplasia: Report of Three Unusual Cases. Int J Experiment Dent Sci 2014;3(2):103-105.

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27

BENBELLAL, Amina, Hanène BELABBASSI, Sarrah AIT ZIANE, Redha ALLOUTI, and Houria KACED. "Clinical and etiological aspects of vertebral deviations secondary to osteochondrdysplasias." Batna Journal of Medical Sciences (BJMS) 5, no. 1 (December 25, 2018): 68–73. http://dx.doi.org/10.48087/bjmsoa.2018.5116.

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Les dysplasies osseuses ou maladies osseuses constitutionnelles (MOC) sont une des causes des déformations vertébrales secondaires. Ce sont des affections génétiques congénitales rares touchant le squelette, liées à des anomalies du cartilage et/ou de l’os. Elles sont responsables de retard de la croissance et des déformations osseuses d’importances variables. Elles se divisent en ostéochondrodysplasies et en dysostoses multiples. Notre objectif est de décrire l’aspect étiologique des déformations vertébrales secondaires aux ostéochondrodysplasies et leurs tableaux cliniques correspondants, à travers trois observations de patients âgés entre 3 et 7 ans. Tous nos patients présentent un retard statural avec des signes cliniques squelettiques, à savoir des déformations thoraciques et vertébrales sous la forme soit d’une cyphose pathologique, une scoliose ou une cyphoscoliose, des troubles orthopédiques des membres supérieurs et inférieurs, associés à des signes extra squelettiques. L’approche clinique d’un patient atteint d’une déformation vertébrale secondaire à une ostéochondro-dysplasie se heurte à de multiples difficultés en raison de la grande diversité étiologique, et chacune d’entre elles demande une prise en charge adaptée et multidisciplinaire.

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28

Minic, Arsa. "Fibro-osseous lesions of the jaws." Serbian Dental Journal 50, no. 4 (2003): 171–78. http://dx.doi.org/10.2298/sgs0304171m.

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Fibro-osseous lesions of the jaws usually present a diagnostic dilemma for the clinician as well as the pathologist. This group includes fibrous dysplasia, ossifying fibroma, focal cemento-osseous dysplasia, periapical cemento-osseous dysplasia, and florid cemento-osseous dysplasia. As in other sites, appropriate clinicopathologic correlations are needed to reach a definitive diagnosis. The clinical and pathologic features of fibro-osseous lesions of the jaws are considered, including adequate therapeutic strategies.

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29

Akram, Arifa, Tajinder Lidhar, Katy Marie Valentine, and Roger Webb. "Diagnosis and management of chronic osteomyelitis and cemento-osseous dysplasia of the mandible." Dental Update 46, no. 11 (December 2, 2019): 1062–66. http://dx.doi.org/10.12968/denu.2019.46.11.1062.

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Chronic osteomyelitis is a severe and persistent infection of the bone including the bone marrow. Cemento-osseous dysplasias are a group of conditions arising from the periodontal ligament tissue and are categorized into peri-apical, focal and florid. A case of a patient with buccal and submandibular swelling associated with a discharging sinus between LR45 was presented. History, examination and investigations confirmed a diagnosis of chronic osteomyelitis and an incidental finding of florid cemento-osseous dysplasia of the mandible. The patient's treatment is discussed within the context of important clinical considerations required in managing these patients. CPD/Clinical Relevance: It is pertinent for a general practitioner to be aware of signs and symptoms associated with chronic osteomyelitis and understand the management options.

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30

Santosh Hunasgi. "FLORID OSSEOUS DYSPLASIA." Annals and essences of dentistry 2, no. 4 (September 14, 2010): 214–17. http://dx.doi.org/10.5368/aedj.2010.2.4.214-217.pdf.

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31

Bansal, Shivani, Deepika Bablani, Vinay Kumar, Subraj Shetty, Sandip Kulkarni, and Rajiv Desai. "Florid osseous dysplasia." Journal of Oral and Maxillofacial Pathology 15, no. 2 (2011): 197. http://dx.doi.org/10.4103/0973-029x.84497.

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32

Mohan, R. P. S., S. Verma, U. Singh, and N. Agarwal. "Florid osseous dysplasia." Case Reports 2013, jun27 1 (June 27, 2013): bcr2013010431. http://dx.doi.org/10.1136/bcr-2013-010431.

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33

Potochny, Evelyn M., and Aaron R. Huber. "Focal Osseous Dysplasia." Head and Neck Pathology 5, no. 3 (July 9, 2011): 265–67. http://dx.doi.org/10.1007/s12105-011-0280-0.

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34

OLIVEIRA, Maiolino Thomaz Fonseca, Sérgio Vitorino CARDOSO, Cláudia Jordão SILVA, Darceny ZANETTA-BARBOSA, and Adriano Mota LOYOLA. "Failure of dental implants in cemento-osseous dysplasia: a critical analysis of a case." Revista de Odontologia da UNESP 43, no. 3 (June 2014): 223–27. http://dx.doi.org/10.1590/rou.2014.035.

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Introduction: Osseointegrated dental implants have been routinely used in oral rehabilitation. Patients with dysplastic bone diseases represent a particular challenge for oral rehabilitation with dental implants. Nevertheless, the installation of implants in patients with cemento-osseous dysplasia (COD) has been reported with apparent success. Objective: In this paper, we present a case of a patient with COD in which a dental implant had been installed and lost six months later. In this regard, we analyzed pertinent aspects of the indications for dental implants in patients with COD considering the pathogenesis, dysplastic nature, and behavior of the disease. Conclusion: The present case report illustrates that COD can be considered a limiting factor in the recommendation for a dental implant. Because of this, it is imperative to inform the patient about the risks of surgically manipulating the diseased bone.

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35

Srivastava, Adit, Rahul Agarwal, Romesh Soni, Avesh Sachan, G. C. Shivakumar, and T. P. Chaturvedi. "Familial Florid Cemento-Osseous Dysplasia: A Rare Manifestation in an Indian Family." Case Reports in Dentistry 2012 (2012): 1–5. http://dx.doi.org/10.1155/2012/574125.

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Florid cemento-osseous dysplasia (FCOD) is one of the uncommon dysplasias affecting the maxillofacial region. The age group may vary from 19 to 76 years and typically presents in the 4th and 5th decades. In most cases patients do not have hereditary basis of disease, and only a few familial cases have been documented. As far as we know this is the 1st reported case of familial FCOD in an Indian family. The mother and son exhibited multiple sclerotic masses in both jaws. The mode of transmission appeared to be autosomal dominant with variable phenotypic expression.

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36

Agha Hosseini, Farzaneh, and Elham Moslemi. "Central ossifying fibroma, periapical cemento-osseous dysplasia and complex odontoma occurring in the same jaw." Clinics and Practice 1, no. 2 (May 17, 2011): 36. http://dx.doi.org/10.4081/cp.2011.e36.

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Central ossifying fibroma is a rare, benign fibro-osseous lesion that arises from the periodontal ligament. Periapical cemento-osseous dysplasia is another variant of fibro-osseous lesion which occurs in the anterior region of the mandible of females. Odontoma is a benign odontogenic tumor that contains enamel, dentine cement and pulp tissue. A 46-year-old woman was referred to the Department of Oral Medicine, School of Dentistry, Tehran University of Medical Sciences, with two nonpainful swellings on both sides of the mandible, which had been slowly growing over a period of one year. Our differential diagnosis was florid cemento-osseous dysplasia, focal cemento-osseous dysplasia for the right side, complex odontoma for the left side and periapical cement-osseous dysplasia for the anterior side. The historical feature revealed ossifying fibroma, complex odontoma and periapical cemento-osseous dysplasia. The occurrence of these three lesions in the same jaw has been rarely reported in the literature. The relationship between the occurrence of these three lesions is not obvious it could be coincidental. It seems that more case reports are needed to establish the relationship between them.

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37

Bulut, Emel Uzun, Aydan Acikgoz, Bora Ozan, Ayse Zeynep Zengin, and Omer Gunhan. "Expansive Focal Cemento-Osseous Dysplasia." Journal of Contemporary Dental Practice 13, no. 1 (2012): 115–18. http://dx.doi.org/10.5005/jp-journals-10024-1105.

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ABSTRACT Aim To present a case of expansive focal cemento-osseous dysplasia and emphasize the importance of differential diagnosis. Background Cemento-osseous dysplasia is categorized into three subtypes on the basis of the clinical and radiographic features: Periapical, focal and florid. The focal type exhibits a single site of involvement in any tooth-bearing or edentulous area of the jaws. These lesions are usually asymptomatic; therefore, they are frequently diagnosed incidentally during routine radiographic examinations. Lesions are usually benign, show limited growth, and do not require further surgical intervention, but periodic follow-up is recommended because occasionally, this type of dysplasia progresses into florid osseous dysplasia and simple bone cysts are formed. Case report A 24-year-old female patient was referred to our clinic for swelling in the left edentulous mandibular premolarmolar region and felt discomfort when she wore her prosthetics. She had no pain, tenderness or paresthesia. Clinical examination showed that the swelling in the posterior mandible that was firm, nonfluctuant and covered by normal mucosa. On panoramic radiography and computed tomography, a welldefined lesion of approximately 1.5 cm in diameter of mixed density was observed. The swelling increased slightly in size over 2 years making it difficult to use prosthetics and, therefore, the lesion was totally excised under local anesthesia, and surgical specimens were submitted for histopathological examination. The histopathological diagnosis was focal cemento-osseous dysplasia. Conclusion In the present case, because of the increasing size of the swelling making it difficult to use prosthetics, young age of the patient and localization of the lesion, in the initial examination, cemento-ossifying fibroma was suspected, and the lesion was excised surgically; the histopathological diagnosis confirmed it as focal cemento-osseous dysplasia. Clinical significance We present a case of expansive focal cemento-osseous dysplasia. Differential diagnosis is essential because ossifying fibroma is a real neoplastic entity. How to cite this article Uzun Bulut E, Acikgoz A, Ozan B, Zengin AZ, Gunhan O. Expansive Focal Cemento-Osseous Dysplasia. J Contemp Dent Pract 2012;13(1):115-118.

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Kalifa, Gabriel, and Solè;ne Ferey. "Dysplasies osseuses identifiables à; la naissance." EMC - Radiologie et imagerie médicale - Musculosquelettique - Neurologique - Maxillofaciale 1, no. 1 (January 2006): 1–14. http://dx.doi.org/10.1016/s1879-8551(06)74019-2.

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Gupta, Aprna, Suryakanti Nayak, BijayKumar Das, and SuryaNarayan Das. "Florid cemento-osseous dysplasia." Journal of Oral and Maxillofacial Pathology 17, no. 1 (2013): 150. http://dx.doi.org/10.4103/0973-029x.110735.

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Kumar, Shwetha, and Rahul Kadam. "Focal Cemento-osseous Dysplasia." Journal of Contemporary Dentistry 3, no. 2 (2013): 112–15. http://dx.doi.org/10.5005/jp-journals-10031-1048.

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ABSTRACT Focal cemento-osseous dysplasia (FCOD) is a benign fibroosseous condition that can be seen in dentulous and edentulous patients. It is an asymptomatic lesion and needs no treatment; however, follow-up is essential due to the possibility that it can progress to a condition called florid cemento-osseous dysplasia. Clinically, the lesion resembles periapical pathosis of odontogenic origin. FCOD is an asymptomatic lesion and occurs in the periapical area of teeth with vital pulps or in regions of extractions. The lesion is detected only on radiographic examination varying from completely radiolucent to densely radiopaque. The histopathologic appearance consists of trabeculae of bone and cementum like material present within a vascular fibrous stroma. Presented here is a case of FCOD in the mandible that occurred in the periapical region of a vital tooth. How to cite this article Kadam R, Patel S, Pathak J, Swain N, Kumar S. Focal Cemento-osseous Dysplasia. J Contemp Dent 2013;3(2):112-115.

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Miyake, Minoru, and Shunichiro Nagahata. "Florid cemento-osseous dysplasia." International Journal of Oral and Maxillofacial Surgery 28, no. 1 (February 1999): 56–57. http://dx.doi.org/10.1016/s0901-5027(99)80679-1.

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Gilchrist, Dawna M., and Frances L. Harley. "Schimke immuno-osseous dysplasia." Journal of Pediatrics 120, no. 3 (March 1992): 497. http://dx.doi.org/10.1016/s0022-3476(05)80933-3.

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Beylouni, I., P. Farge, J. F. Mazoyer, and J. L. Coudert. "Florid cemento-osseous dysplasia." Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology, and Endodontology 85, no. 6 (June 1998): 707–11. http://dx.doi.org/10.1016/s1079-2104(98)90039-7.

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Oğuz, Yener, Ülkem Aydın, Burcu Şengüven, and Taner Köroğlu. "Focal Cemento-Osseous Dysplasia." Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology 119, no. 3 (March 2015): e151. http://dx.doi.org/10.1016/j.oooo.2014.07.211.

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Gumusok, Mustafa, MeryemToraman Alkurt, Kaan Hamurcu, DervisKaan Kilavuz, Emre Baris, and Baris Simsek. "Focal cemento-osseous dysplasia." Journal of Oral and Maxillofacial Radiology 2, no. 3 (2014): 92. http://dx.doi.org/10.4103/2321-3841.144683.

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Ravikumar, SShamala, G. Vasupradha, TR Menaka, and SPradeep Sankar. "Focal cemento-osseous dysplasia." Journal of Oral and Maxillofacial Pathology 24, no. 4 (2020): 19. http://dx.doi.org/10.4103/jomfp.jomfp_209_19.

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Ariji, Yoshiko, Eiichiro Ariji, Yoshinori Higuchi, Shuro Kubo, Eiji Nakayama, and Sigenobu Kanda. "Florid cemento-osseous dysplasia." Oral Surgery, Oral Medicine, Oral Pathology 78, no. 3 (September 1994): 391–96. http://dx.doi.org/10.1016/0030-4220(94)90074-4.

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Parham, David M., Julia A. Bridge, Janet L. Lukacs, Yiling Ding, A. Francine Tryka, and Jeffrey R. Sawyer. "Cytogenetic Distinction among Benign Fibro-osseous Lesions of Bone in Children and Adolescents: Value of Karyotypic Findings in Differential Diagnosis." Pediatric and Developmental Pathology 7, no. 2 (March 2004): 148–58. http://dx.doi.org/10.1007/s10024-003-6065-z.

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Benign fibro-osseous lesions of bone (BFOL) comprise a group of clinically distinct entities with significant histologic overlap and often occur in children and adolescents. Because of prior studies indicating that these lesions possess distinct karyotypic abnormalities, we conducted a retrospective review of cytogenetic analyses performed in a series of 16 BFOL in children and adolescents diagnosed at two institutions. These comprised five cases with the diagnosis of ossifying fibroma, four with osteofibrous dysplasia, and seven with fibrous dysplasia arising in the skeleton of 16 children and adolescents. All cases were analyzed using standard G-banding techniques on fresh tumors explanted in tissue culture media. Spectral karyotyping (SKY) and fluorescence in situ hybridization (FISH) were used to analyze selected metaphases of a talar lesion with the histologic features of ossifying fibroma. All four confirmed ossifying fibromas, including the talar lesion, contained clonal aberrations fusing breakpoints on Xq26 and 2q33, and one case with dissimilar histology did not. Three of four osteofibrous dysplasias contained multiple copies of chromosomes 8, 12, and/or 21. All but two fibrous dysplasia cases exhibited either a completely normal karyotype or single cell aberrations. One fibrous dysplasia had subtle chromosomal abnormalities not seen in other cases in the series, and another had complex abnormalities involving multiple chromosomes. Our current and published results indicate that cytogenetics might be of ancillary use in the diagnosis of BFOL and that a characteristic chromosomal arrangement is associated with ossifying fibroma.

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Samaha, Mark, and Ralph Metson. "Image-Guided Resection of Fibro-Osseous Lesions of the Skull Base." American Journal of Rhinology 17, no. 2 (March 2003): 115–18. http://dx.doi.org/10.1177/194589240301700210.

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Background Endoscopic resection of sinonasal fibro-osseous lesions remains a technical challenge because of the loss of anatomic landmarks and the frequent need to resect bone along the skull base. The purpose of this study was to evaluate the usefulness of image-guidance systems for the resection of these lesions. Methods Endoscopic surgery was performed in 10 patients with fibro-osseous lesions of the sinuses and skull base (six fibrous dysplasias and four osteomas). Indications for surgical intervention were recurrent infection (7), facial pain (3), mucocele (1), and compressive optic neuropathy (1). All surgeries were performed with an image-guidance system, which included an integrated high-speed drill system. Results Anatomic localization was accurate to within 1 mm when monitoring the depth of bone removal along the skull base. Complete resection of the fibro-osseous lesion was possible in six patients (60%) and partial removal was possible in four patients (40%). In the latter group, a margin of dysplastic bone was preserved along the ethmoid roof to prevent dural exposure. There were no intraoperative complications. Surgery resulted in resolution of infection and facial pain in nine patients (90%). Additional surgery was required in one patient (10%) who developed recurrent optic neuropathy because of continued dysplastic bone growth. Mean follow-up was 34 months (range, 13–67 months). Conclusion Image-guidance technology appears to be ideally suited for the treatment of patients with fibro-osseous lesions of the sinonasal cavity. Real-time monitoring of the depth of bone removal relative to the skull base may enhance the safety and efficacy of such surgery.

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Piróg, Katarzyna A., and Michael D. Briggs. "Skeletal Dysplasias Associated with Mild Myopathy—A Clinical and Molecular Review." Journal of Biomedicine and Biotechnology 2010 (2010): 1–13. http://dx.doi.org/10.1155/2010/686457.

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Musculoskeletal system is a complex assembly of tissues which acts as scaffold for the body and enables locomotion. It is often overlooked that different components of this system may biomechanically interact and affect each other. Skeletal dysplasias are diseases predominantly affecting the development of the osseous skeleton. However, in some cases skeletal dysplasia patients are referred to neuromuscular clinics prior to the correct skeletal diagnosis. The muscular complications seen in these cases are usually mild and may stem directly from the muscle defect and/or from the altered interactions between the individual components of the musculoskeletal system. A correct early diagnosis may enable better management of the patients and a better quality of life. This paper attempts to summarise the different components of the musculoskeletal system which are affected in skeletal dysplasias and lists several interesting examples of such diseases in order to enable better understanding of the complexity of human musculoskeletal system.

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