Relevant bibliographies by topics / Dysplastic neoplasia

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Dysplastic neoplasia'

Author: Grafiati
Published: 7 June 2025
Last updated: 2 August 2025

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Journal articles on the topic "Dysplastic neoplasia"

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Neves, André, Massimiliano Di Pietro, Maria O’Donovan, et al. "Detection of early neoplasia in Barrett’s esophagus using lectin-based near-infrared imaging: an ex vivo study on human tissue." Endoscopy 50, no. 06 (2018): 618–25. http://dx.doi.org/10.1055/s-0043-124080.

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Abstract Background and study aims Endoscopic surveillance for Barrett’s esophagus (BE) is limited by long procedure times and sampling error. Near-infrared (NIR) fluorescence imaging minimizes tissue autofluorescence and optical scattering. We assessed the feasibility of a topically applied NIR dye-labeled lectin for the detection of early neoplasia in BE in an ex vivo setting. Methods Consecutive patients undergoing endoscopic mucosal resection (EMR) for BE-related early neoplasia were recruited. Freshly collected EMR specimens were sprayed at the bedside with fluorescent lectin and then ima
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Rickelt, Steffen, Azfar Neyaz, Charlene Condon, et al. "Agrin Loss in Barrett's Esophagus-Related Neoplasia and Its Utility as a Diagnostic and Predictive Biomarker." Clinical Cancer Research 28, no. 6 (2022): 1167–79. http://dx.doi.org/10.1158/1078-0432.ccr-21-2822.

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Abstract Purpose: There is an unmet need for identifying novel biomarkers in Barrett's esophagus that could stratify patients with regards to neoplastic progression. We investigate the expression patterns of extracellular matrix (ECM) molecules in Barrett's esophagus and Barrett's esophagus–related neoplasia, and assess their value as biomarkers for the diagnosis of Barrett's esophagus–related neoplasia and to predict neoplastic progression. Experimental Design: Gene-expression analyses of ECM matrisome gene sets were performed using publicly available data on human Barrett's esophagus, Barret
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Carballal, Sabela, Sandra Maisterra, Antonio López-Serrano, et al. "Real-life chromoendoscopy for neoplasia detection and characterisation in long-standing IBD." Gut 67, no. 1 (2016): 70–78. http://dx.doi.org/10.1136/gutjnl-2016-312332.

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ObjectiveOutside clinical trials, the effectiveness of chromoendoscopy (CE) for long-standing IBD surveillance is controversial. We aimed to assess the effectiveness of CE for neoplasia detection and characterisation, in real-life.DesignFrom June 2012 to 2014, patients with IBD were prospectively included in a multicentre cohort study. Each colonic segment was evaluated with white light followed by 0.4% indigo carmine CE. Specific lesions' features were recorded. Optical diagnosis was assessed. Dysplasia detection rate between expert and non-expert endoscopists and learning curve were ascertai
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KAPETANIOS, V., A. C. LAZARIS, P. BOGRIS, et al. "Extracellular regulated kinase-2 immunoreactivity increases in parallel with cervical intraepithelial neoplasia grade in cervical neoplasia." International Journal of Gynecologic Cancer 18, no. 3 (2008): 540–45. http://dx.doi.org/10.1111/j.1525-1438.2007.01057.x.

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The cell cycle control system includes cyclins, cyclin-dependent kinases (CDK), and their inhibitors (CDK1). Extracellular regulated kinase (ERK1/2) (p44 and p42 mitogen-activated protein kinases [MAPKs]) is a component of the MAPK pathway, which is associated with cyclin D1 and CDK. It is a critical signaling system for the induction of cell proliferation, differentiation, and cell survival. The aim of this study was to investigate the usefulness of ERK2 expression as a marker of biological aggressiveness complementary to cervical intraepithelial neoplasia (CIN) grade as well as to compare it
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Beggs, Andrew D., Jonathan James, Germaine Caldwell, et al. "Discovery and Validation of Methylation Biomarkers for Ulcerative Colitis Associated Neoplasia." Inflammatory Bowel Diseases 24, no. 7 (2018): 1503–9. http://dx.doi.org/10.1093/ibd/izy119.

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Abstract Background and aims Ulcerative colitis (UC) is associated with a higher background risk of dysplasia and/or neoplasia due to chronic inflammation. There exist few biomarkers for identification of patients with dysplasia, and targeted biopsies in this group of patients are inaccurate in reliably identifying dysplasia. We aimed to examine the epigenome of UC dysplasia and to identify and validate potential biomarkers Methods Colonic samples from patients with UC-associated dysplasia or neoplasia underwent epigenome-wide analysis on the Illumina 450K methylation array. Markers were valid
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Tariq, Raseen, Sarah Enslin, Maham Hayat, and Vivek Kaul. "Efficacy of Cryotherapy as a Primary Endoscopic Ablation Modality for Dysplastic Barrett’s Esophagus and Early Esophageal Neoplasia: A Systematic Review and Meta-Analysis." Cancer Control 27, no. 1 (2020): 107327482097666. http://dx.doi.org/10.1177/1073274820976668.

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Introduction: Cryotherapy is a cold-based ablative therapy used primarily as second line therapy in patients with Barrett’s esophagus (BE) who have persistent dysplasia after undergoing endoscopic treatment with radiofrequency ablation (RFA). Few studies have described the use of cryotherapy as a primary treatment modality for dysplastic or neoplastic BE. Aim: To evaluate the efficacy of cryotherapy as primary treatment of dysplastic and/or neoplastic BE by conducting a systemic review and meta-analysis. Methods: A systematic search of Medline, Embase, and Web of Science was performed from Jan
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Claessen, Marian M. H., Marguerite E. I. Schipper, Bas Oldenburg, Peter D. Siersema, G. Johan A. Offerhaus, and Frank P. Vleggaar. "WNT-Pathway Activation in IBD-Associated Colorectal Carcinogenesis: Potential Biomarkers for Colonic Surveillance." Analytical Cellular Pathology 32, no. 4 (2010): 303–10. http://dx.doi.org/10.1155/2010/957698.

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Objectives: The Wnt-pathway dominates the sporadic carcinogenesis whereas p53 plays a pivotal role in the colitis-associated counterpart. The expression of Wnt-signaling proteins and p53 during colitis-associated carcinogenesis was determined.Methods: A tissue microarray was constructed with colonic samples from 5 groups of patients: controls (C, n=10), IBD without neoplasia (IBD, n=12), non-dysplastic IBD with neoplasia elsewhere in the colon (IBD-NE, n=12), dysplastic lesion in IBD (IBD-DYS, n=12), and IBD-associated colorectal cancer (IBD-CRC, n=10). Immunohistochemistry was performed for β
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Saiz Chumillas, R. M., L. Alba, Y. Gonzalez-Lama, et al. "P122 Low Risk of new dysplastic lesions during inflammatory bowel disease surveillance with dye-cromoendoscopy: a multicenter population-based retrospective study." Journal of Crohn's and Colitis 16, Supplement_1 (2022): i211—i213. http://dx.doi.org/10.1093/ecco-jcc/jjab232.250.

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Abstract Background The 21st century has witnessed advances in endoscopic surveillance technology such as high-definition imaging and virtual or dye-based chromoendoscopy, which have led to increased detection of dysplasia. It is unknown the rates of new dysplastic lesions or cancer progression with these techniques though it seems lower than previously described. Methods We developed a multicenter, population-based and retrospective cohort from 7 Spanish hospitals from the Group of Inflammatory bowel disease of Castilla Y Leon (GEICYL) including sequentially all patients with inflammatory bow
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Huang, Lee-Wen, Hun-Shan Pan, Yu-Hung Lin, Kok-Min Seow, Heng-Ju Chen, and Jiann-Loung Hwang. "P16 Methylation Is an Early Event in Cervical Carcinogenesis." International Journal of Gynecologic Cancer 21, no. 3 (2011): 452–56. http://dx.doi.org/10.1097/igc.0b013e31821091ea.

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BackgroundAberrant gene promoter methylation is a critical event in tumorigenesis. The aim of this study was to explore the promoter hypermethylation of p16 and DAPK1 during the progression of cervical precancerous lesions.MethodsA series of 98 cervical neoplasms (72 cervical intraepithelial neoplasia and 26 cervical carcinomas) were evaluated. The promoter methylation status of p16 and DAPK1 was assessed from cervical scrapings by methylation-specific polymerase chain reaction.ResultsFor p16, the frequency of promoter hypermethylation showed an increasing trend from normal to dysplastic to in
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Farhat, Nada, Nora Laver, and José Caro. "9. Cellular immune response to anal dysplasia in HIV-positive men on highly active antiretroviral therapy." Sexual Health 10, no. 6 (2013): 574. http://dx.doi.org/10.1071/shv10n6ab9.

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Background The role of the cellular immune response to anal dysplasia progression is poorly understood. Extrapolated from the cervical model, CD4+ (killed by HIV) and CD8+ cells may have crucial anti-tumour activity. We report pilot data on key immune responses in HIV+ patients on HAART with anal dysplasia. Methods: High-resolution anoscopy and biopsies were performed. Ten tissue biopsies with high-grade anal intraepithelial neoplasia (HGAIN) were stained with immunohistochemistry for CD4+, CD8+, CXCL12+ and FOXP3+, and compared with 10 samples with low-grade anal intraepithelial neoplasia (LG
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Dissertations / Theses on the topic "Dysplastic neoplasia"

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Alnasser, Saleh. "Predictors of dysplastic and neoplastic progression for Barrett's esophagus." Thesis, McGill University, 2012. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=110608.

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Abstract (English)Introduction: Barrett's esophagus (BE) is a premalignant condition that may progress through a stepwise process to esophageal adenocarcinoma (EAC). It is unknown why some BE patients progress to EAC rapidly while others do so more slowly, or not at all. The aim of this study is to identify the demographic and endoscopic factors that can be used as predictors of dysplastic and neoplastic progression in BE patients, and thereby be used to stratify BE patients into different surveillance protocols according to the risk of neoplastic progression. Method: All BE patients at Mc
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Gattoc, Leda, Paula M. Frew, Shontell N. Thomas, et al. "Phase I dose-escalation trial of intravaginal curcumin in women for cervical dysplasia." DOVE MEDICAL PRESS LTD, 2016. http://hdl.handle.net/10150/622729.

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Background: This is a Phase I trial demonstrating safety and tolerability of intravaginal curcumin for future use in women with cervical neoplasia. Objective: The objective of this study was to assess the safety, tolerability, and pharmacokinetics of intravaginal curcumin in healthy women. Study design: We conducted a 3+3 dose-escalation Phase I trial in a group of women aged 18-45 years. Thirteen subjects were given one of four doses of curcumin powder (500 mg, 1,000 mg, 1,500 mg, and 2,000 mg) packed in gelatin capsules, which was administered intra-vaginally daily for 14 days. The primary e
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Wilkins, Bridget Sally. "An immunohistochemical study of the spatial organisation and differentiation of haemopoietic cells within bone marrow in reactive, dysplastic and neoplastic myeloproliferative states." Thesis, University of Southampton, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.316483.

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Majumdar, Debabrata. "Expression and post-translational modification of intermediate filament proteins in colonic mucosa of patients with ulcerative colitis : role in pathogenesis of colitis associated colorectal neoplasia and dysplasia." Thesis, University of Sheffield, 2016. http://etheses.whiterose.ac.uk/16564/.

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Background: Ulcerative colitis(UC) is a chronic inflammatory disease of the colon associated with increased cancer risk of colitis associated cancer(CAC). Keratins(K) are intermediate filament(IF) proteins, and are key component of the cellular cytoskeleton. K8 null mice develop colitis; a subset of IBD patients have mutation in K8 gene. Keratins play a role in cell-death signalling pathways; epithelial cells lacking K8 and K18 are more sensitive to TNF-mediated apoptosis. Methods: Colonic biopsies obtained from UC patients (and controls) were grouped into eight categories based on risk of can
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Bernardes, Carlos Alberto Nascimento. "A expressão de Angiopoetina 1 e Trombospondina 1 em desordens potencialmente malignas bucais e no carcinoma espinocelular." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2015. http://hdl.handle.net/10183/130347.

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O carcinoma espinocelular é responsável por 90% das lesões malignas na cavidade bucal, sendo o oitavo tipo de câncer em todo o mundo e apresenta uma taxa de sobrevida de apenas 50% em 5 anos. Os carcinomas podem se desenvolver diretamente de células epiteliais normais ou a partir das desordens potencialmente malignas, as quais envolvem as hiperplasias e as displasias. Para que estas lesões se desenvolvam é necessário que nutrientes e oxigênio sejam disponibilizados, o que ocorre a partir da formação de uma nova vasculatura. Este processo, denominado angiogênese, é complexo e envolve a regulaçã
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Strander, Björn. "Cervical cancer prevention : studies on possible improvements /." Göteborg : Dept. of Obstetrics and Gynecology, Institute of Clinical Sciences, Sahlgrenska Academy, Göteborg University, 2008. http://hdl.handle.net/2077/8513.

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Karlsson, Pia. "Cutaneous melanoma in children and adolescents and aspects of naevus phenotype in melanoma risk assessment." Doctoral thesis, Linköping : Univ, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-7703.

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Milana, Ivkov Simić. "Kliničke i dermoskopske karakteristike displastičnih nevusa." Phd thesis, Univerzitet u Novom Sadu, Medicinski fakultet u Novom Sadu, 2015. http://www.cris.uns.ac.rs/record.jsf?recordId=90291&source=NDLTD&language=en.

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Uvod: Od kada je prvi put opisan pre 30 godina, kod porodica obolelih od melanomakože, displastični nevus je predmet debate. Klinički najznačajnija kontroverza je da sedisplastični nevus klinički često smatra sumnjivim i da se teško razlikuje od ranogmelnoma kože. Ovakav klinički izgled displastičnog nevusa je razlog čestih nepotrebnihekscizija, jednog od najčešćih nevusa čoveka. Ciljevi: Ispitati kliničke i dermoskopske karakteristike patohistološki potvrđenih displastičnih nevusa, i utvrditi učestalost displastičnih nevusa među sumnjivim melanocitnim lezijama koje su oda
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Tomita, Luciana Yuki. "Consumo alimentar e concentrações séricas de micronutrientes: associação com lesões neoplásicas e câncer cervical." Universidade de São Paulo, 2007. http://www.teses.usp.br/teses/disponiveis/6/6133/tde-20122007-160208/.

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O câncer cervical é o segundo câncer mais comum entre as mulheres em todo o mundo. A infecção por Papilomavirus (HPV) do tipo oncogênico é causa necessária. Estudos internacionais sugerem importante papel de carotenóides e tocoferóis séricos e dietéticos na redução do risco para lesões precursoras, mas os resultados dos estudos prévios são inconsistentes. Indivíduos e métodos: O presente estudo de casos e controles de base hospitalar conduzido na cidade de São Paulo analisou a associação entre concentrações séricas de carotenóides (licopeno, β-caroteno), tocoferóis (α- e γ-), co
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Klann, Alexandra. "The effect of cervical intraepithelial neoplasia and treatment surgeries on fecundability." Thesis, 2018. https://hdl.handle.net/2144/32976.

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INTRODUCTION: Approximately 6 million couples in the United States experience infertility. Because few risk factors for infertility are known, identification of modifiable determinants is an important public health goal. Cervical intraepithelial neoplasia, CIN, occurs when the surface cells of the cervical tissue begin to change, and is caused by infection with a high-risk type of human Papillomavirus (HPV). CIN may affect the cervix’s immunological function, resulting in changes in mucus production, reduced protection against infections, and alterations in sperm transport through the cervic
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Books on the topic "Dysplastic neoplasia"

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S, Cibas Edmund, and Lee Kenneth R. 1941-, eds. Pathology of early cervical neoplasia. Churchill Livingstone, 1997.

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A, Jordan Joseph, Luesley David, and Richart Ralph M. 1933-, eds. Intraepithelial neoplasia of the lower genital tract. Churchill Livingstone, 1995.

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Baron, Mary Michele. Fractal characterisation of nuclear morphology in cervical intraepithelial neoplasia (dysplasia and carcinoma in situ) in humans. Laurentian University, 1994.

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1943-, Riddell Robert H., Crohn's & Colitis Foundation of America., National Institutes of Health (U.S.), and Rebecca Meyerhoff Philanthropic Fund, eds. Dysplasia and cancer in colitis. Elsevier, 1991.

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Ann, McPherson, Cancer Research Campaign (Great Britain), and Imperial Cancer Research Fund (Great Britain), eds. Cervical screening. 2nd ed. Oxford University Press, 1992.

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Husain, O. A. N. A colour atlas of gynaecological cytology. Wolfe Medical Pub., 1989.

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Sybert, Virginia P. Tumors/Hamartomas. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780195397666.003.0010.

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Basal Cell Nevus Syndrome – Bathing Trunk Nevus – Cowden Disease – Cylindromatosis – Dysplastic Nevus Syndrome – Epidermal Nevus – Gardner Syndrome – Hereditary Keratoacanthomas – Infantile Myofibromatosis – Multiple Endocrine Neoplasia Types 1, 2A, and 2B/3 – Multiple Leiomyomatosis – Pilomatricoma – Proteus Syndrome – Sebaceous Nevus Syndrome – Tumoral Calcinosis
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Sybert, Virginia P. Tumors/Hamartomas. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190276478.003.0010.

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Chapter 10 covers Basal Cell Nevus Syndrome, Cowden Syndrome, Cylindromatosis, Dysplastic Nevus Syndrome, Epidermal Nevus, Gardner Syndrome, Giant congenital nevocytic nevus, Hereditary Keratoacanthomas, Hereditary Leiomyomatosis and renal cell cancer, Infantile Myofibromatosis, Multiple Endocrine Neoplasia Types 1, 2A, and 2B/3, Pilomatricoma, Proteus Syndrome, Sebaceous Nevus Syndrome, and Tumoral Calcinosis. Each condition is discussed in detail, including dermatologic features, associated anomalies, histopathology, basic defect, treatment, mode of inheritance, prenatal diagnosis, and diffe
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Floch, Martin H. Colorectal Neoplasia and the Colorectal Microbiome: Dysplasia, Probiotics, and Fusobacteria. Elsevier Science & Technology Books, 2020.

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Colorectal Neoplasia and the Colorectal Microbiome: Dysplasia, Probiotics, and Fusobacteria. Elsevier Science & Technology, 2020.

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Book chapters on the topic "Dysplastic neoplasia"

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Robin, Marie, and Carmelo Gurnari. "Myelodysplastic Neoplasms/Syndromes (MDS)." In The EBMT Handbook. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-44080-9_74.

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Masterpol, Kasia Szyfelbein, Andrea Primiani, and Lyn McDivitt Duncan. "Vulvar Dysplasia and Neoplasia." In Atlas of Essential Dermatopathology. Springer London, 2012. http://dx.doi.org/10.1007/978-1-4471-4471-7_26.

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Nwaneshiudu, Adaobi I., Jon A. Reed, Victor G. Prieto, and Christopher R. Shea. "Dysplastic Nevi Versus Melanoma." In Pathology of Challenging Melanocytic Neoplasms. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1444-9_9.

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Rothschild, Bruce M., Dawid Surmik, and Filippo Bertozzo. "Bone Neoplasia and Skeletal Dysplasia." In Modern Paleopathology, The Study of Diagnostic Approach to Ancient Diseases, their Pathology and Epidemiology. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-28624-7_13.

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He, Yue, Hui Shan Ong, Zhen Tian, Lin Zhu, and Yu Han. "Fibrous Dysplasia." In Inflammatory and Neoplastic Diseases of Craniofacial Bones. Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-97-4155-7_17.

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He, Yue, Hui Shan Ong, Zhen Tian, Lin Zhu, and Qing Long Wu. "Cemento-osseous Dysplasia." In Inflammatory and Neoplastic Diseases of Craniofacial Bones. Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-97-4155-7_18.

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Ozawa, Soji, Yoshifumi Ikeda, Kazuo Koyanagi, et al. "Molecular Alterations in Dysplasia and Superficial Cancer of the Esophagus." In Superficial Esophageal Neoplasm. Springer Japan, 2002. http://dx.doi.org/10.1007/978-4-431-67873-1_25.

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Onida, Francesco, and Yves Chalandon. "Myelodysplastic/Myeloproliferative Neoplasms." In The EBMT Handbook. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-44080-9_76.

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AbstractThe myelodysplastic syndrome-myeloproliferative neoplasms (MDS/MPNs) are a heterogeneous group of hematologic malignancies characterized by dysplastic and myeloproliferative clinical, laboratory, and morphological overlapping features, both in marrow and in blood. MDS/MPNs include chronic myelomonocytic leukemia (CMML), MDS/MPN with neutrophilia, MDS/MPN with SF3B1 mutation (in its absence with ringed sideroblasts) and thrombocytosis (MDS/MPN-SF3B1-T), and MDS/MPN not otherwise specified (MDS/MPN-NOS). Prognosis of MDS/MPN is highly variable, being dismal in the majority of patients wi
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Riddell, R. H. "The case for the reversibility of gastric dysplasia/neoplasia." In Helicobacter pylori. Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-011-3927-4_57.

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Mingazzini, P. L., L. Scucchi, D. Di Stefano, et al. "Interphasic Nucleolar Organizer Regions in Colorectal Dysplastic and Neoplastic Lesions." In Hereditary Colorectal Cancer. Springer Japan, 1990. http://dx.doi.org/10.1007/978-4-431-68337-7_57.

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Conference papers on the topic "Dysplastic neoplasia"

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Pu, Yang, Jaidip Jagtap, Asima Pradhan, and R. R. Alfano. "Dysplastic Cervical Intraepithelial Neoplasia (CIN) Tissues Detection using Spatial Frequency Analysis." In Frontiers in Optics. OSA, 2013. http://dx.doi.org/10.1364/fio.2013.jw3a.24.

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Santiago-García, Jose, Jacobo Ortiz Fernández-Sordo, Philip Kaye, Mirela Pana, and Krish Ragunath. "PTH-126 Neoplasia progression in barrett´s oesophagus with baseline low-grade dysplasia undergoing radiofrequency ablation." In British Society of Gastroenterology, Annual General Meeting, 4–7 June 2018, Abstracts. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2018. http://dx.doi.org/10.1136/gutjnl-2018-bsgabstracts.300.

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Cui, Changxu, Reika Takamatsu, Hiroshi Doguchi, Akiko Matsuzaki, Masanao Saio, and Naoki Yoshimi. "Abstract 4416: The pre-neoplastic lesion, mucin-depleted foci, reveals asde novohigh-grade dysplasia in rat colon carcinogenesis." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-4416.

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Glanzmann, Thomas M., Pascal Uehlinger, Jean-Pierre Ballini, et al. "Time-Resolved Autofluorescence Spectroscopy of the Bronchial Mucosa for the Detection of Early Cancer: Clinical Results." In European Conference on Biomedical Optics. Optica Publishing Group, 2001. http://dx.doi.org/10.1364/ecbo.2001.4432_199.

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Time-resolved measurements of endogenous tissue autofluorescence were carried out on the bronchial mucosa of 18 patients during endoscopy by the means of an optical fibre-based spectrometer. The objective was to assess the fluorescence lifetime as a new contrast parameter between normal and malignant tissue and to explain the origin of a previously observed contrast in fluorescence intensity. The intra- and interpatient variation of tissue autofluorescence intensity and decay on normal tissue was determined, with the outcome that a strong fluctuation in autofluorescence intensity but not in li
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