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Relevant bibliographies by topics / Dysthyroidie

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Academic literature on the topic 'Dysthyroidie'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Dysthyroidie"

1

Khouri, Charles, Elodie Jean Bart, Sophie Logerot, Amandine Decker-Bellaton, Hervé Bontemps, and Michel Mallaret. "Dysthyroidie sous anti-VEGF, effet indésirable de classe ? À propos d’un cas." Therapies 69, no. 6 (November 2014): 521–24. http://dx.doi.org/10.2515/therapie/2014063.

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Grecescu, Mihai Leonard C. "COMPUTED TOMOGRAPHY FOR DIAGNOSING GRAVES OPHTALMOPATHY." Romanian Medical Journal 68, no. 2 (June 30, 2021): 268–72. http://dx.doi.org/10.37897/rmj.2021.2.21.

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Graves ophtalmopathy (GO), thyroid eye disease (TED) and thyroid - associated orbitopathy (TAO) are terms referring to an autoimmune inflammatory process that affects the adipogenesis of orbital fat and periorbital tissue, associated with dysthyroidism. On imaging, such as computed tomography (CT) thyroid -associated orbitopathy (or thyroid -associated ophtalmopathy) is characterised by symmetrical and bilateral proptosis, secondary to extra-ocular muscle (EOM) enlargement and expansion of orbital fat. Imaging studies ca be useful in identifying patients risk to develop dysthyroid optic neuropathy and initiating early treatment of the condition, avoiding permanent visual loss. The purpose of this study was to reveal the prevalence of thyroid ophtalmopathy imaging considerations in Graves disease patients. A total of 19 patients were evaluated on computed tomography scanning the diameters and density of extra-ocular muscles, muscular index, proptosis, width of the optic nerve, width of superior ophthalmic vein and lacrimal gland position.

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Kintu-Luwaga, Ronald, Timothy Makumbi, Cathy Kilyewala, and Jane O. Fualal. "Pattern of sub-clinical dysthyroidism in a postthyroidectomy cohort: Implications for supplementary treatment." East and Central African Journal of Surgery 23, no. 3 (December 31, 2018): 95–99. http://dx.doi.org/10.4314/ecajs.v23i3.2.

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Background: Defective thyroid functioning is referred to as dysthyroidism. Despite incomplete thyroidectomy or thyroxine supplementation, post-thyroidectomy patients may still experience dysthyroidism. Many times, this may be sub - clinical. This study aimed to assess the prevalence and pattern of sub-clinical dysthyroidism following thyroid surgery. Methods: In this prospective cohort study, 40 patients were consecutively recruited following conventional thyroidectomy and followed up to 12months. All patients were euthyroid at surgery. At 12 months serum TSH, T4 and T3 levels were measured and the patients clinically assessed. The prevalence and pattern of dysthyroidism was analysed statistically against the patient demographics, clinical and peri-operative variables for significance, using stata version 13. The confidence interval was at 95% and the statistical significance at a p-value of <0.05. Results: The mean age was 44.3 years (M:F= 1: 12.3). 20% of the patients had medical comorbidities. The types of surgery performed were sub-total thyroidectomy (55%), near total thyroidectomy (25%) and total thyroidectomy (20%). The prevalence of postoperative dysthyroidism was 52.5%. 22.7% of patients who underwent sub-total thyroidectomy had dysthyroidism. Most patients (90%) who were on thyroxine supplement (following total or near total thyroidectomy) still developed dysthyroidism (P= 0.017). The type of resection done had the greatest significance (P= 0.000). Other factors associated with dysthyroidism albeit non-significantly were history of pre-operative hyperthyroidism, middle age (40 - 60 years), and female gender. Conclusions: The prevalence of dysthyroidism in this cohort was high which may reflect the broader picture among post - thyroidectomy patients in this setting. Regular biochemical testing in post-thyroidectomy patients is important to identify and correct dysthyroidism early. This requires frequent follow-up and accurate dose adjustment, based on objective assessments like weight or body mass index. Keywords: post-thyroidectomy; dysthyroidism; hypothyroidism; hyperthyroidism; sub-clinical dysthyroidism; prospective; cohort

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Giordanetti, Stefano. "Dysthyroidism and heart failure: from clinical case to pathophysiology." Clinical Management Issues 4, no. 3S (October 13, 2015): 37–42. http://dx.doi.org/10.7175/cmi.v4i3s.1152.

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Dysthyroidism affects cardiovascular function in many ways and can cause heart failure. The physiopathological mechanisms underlying the development of heart failure involve both direct intranuclear transcriptional effects of thyroid hormones and specific haemodynamic consequences of vascular modifications induced by dysthyroidism. Phospholamban regulatory action on diastolic ventricular function appears to play a pivotal role in mediating both direct T3 action and adrenal effects on myocardial contractility, possibly explaining the way dysthyroidism mimics sympathoadrenergic alterations on cardiovascular function. Therapeutical approach to cardiovascular disorders in dysthyroidism should focus on both thyroid hormones dysregulation and sympathoadrenergic activity, to attempt a reversal of the associated derangements.

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Jacobson, Daniel M. "Dysthyroid Orbitopathy." Seminars in Neurology Volume 20, Number 1 (2000): 0043–54. http://dx.doi.org/10.1055/s-2000-6832.

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Dresner, S. C., and J. S. Kennerdell. "Dysthyroid orbitopathy." Neurology 35, no. 11 (November 1, 1985): 1628. http://dx.doi.org/10.1212/wnl.35.11.1628.

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Luz, Inês Romão, João Rio Martins, Mónica Jerónimo, Joana Serra Caetano, Rita Cardoso, Isabel Dinis, and Alice Mirante. "Neonates Born to Mothers with Graves’ Disease: 15 Year Experience of a Pediatric Endocrinology Department." Acta Médica Portuguesa 33, no. 7-8 (July 1, 2020): 483. http://dx.doi.org/10.20344/amp.12279.

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Introduction: Graves disease is characterized by the existence of autoantibodies directed to the thyrotropin receptor, which can have a stimulatory/inhibitory action, in women with the condition, their fetus or neonate. Our aim was to review the case series of these neonates in order to establish neonatal thyroid function predictors.Material and Methods: Retrospective cohort study of the database of the Department of Pediatric Endocrinology, including patients born to mothers with Graves’ disease, between 2002 and 2017. Clinical and biochemical data were collected from mothers and offspring.Results: Fifty newborns, from 46 women with a median of 3.5 years after diagnosis, were included. During all trimesters of pregnancy, more than half of women had positive autoantibodies directed to the thyrotropin receptor. Not every woman had a complete thyroid function evaluation every trimester. In 32 newborns, cord blood screening was done. During the neonatal period, there were three cases of hypothyroidism and two of hyperthyroidism. The mothers of these five newborns had higher levels of free thyroid hormones during the second trimester (p = 0.03). The level of antibodies directed to the thyrotropin receptor was significantly higher in the cord blood (p = 0.03) and in the first neonatal test (p = 0.03) of these dysthyroid newborns.Discussion: Our results reinforce the need for every pregnant woman with Graves’ disease to be subject to thyroid function and autoantibodies evaluation during every trimester, as well as the importance of evaluating these antibodies in cord blood.Conclusion: High levels of free thyroid hormones during the second trimester of pregnancy and antibodies directed to the thyrotropin receptor value in cord blood are predictors of dysthyroidism in neonates born from women with Grave’s disease.

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Signorelli, Diego, Maria Silvia Cona, Milena Vitali, Giuseppe Lo Russo, Claudia Proto, Martina Imbimbo, Nicoletta Zilembo, et al. "Correlation between dysthyroidism and efficacy in patients treated with anti PD-1 or anti PDL-1 agents." Journal of Clinical Oncology 35, no. 7_suppl (March 1, 2017): 67. http://dx.doi.org/10.1200/jco.2017.35.7_suppl.67.

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67 Background: Immune checkpoints inhibitors have clearly improved patients’outcomes in different tumors, but definitive predictive biomarkers are still missing. Since thyroiditis have been reported, we have evaluated whether clinical responses are more frequent in patients who underwent anti PD-1 or anti PDL-1 agents and experienced dysthyroidism. Methods: We retrospectively evaluated 135 metastatic solid tumor patients, treated at our Institute with anti PD-1/PDL-1 antibodies since 2013. Thyroid toxicity was defined according to CTCAE version 4.0 and disease control (DC: CR+PR+SD) was chosen as efficacy endpoint. Correlation between dysthyroidism and DC rate was assessed by Fisher’s exact test. Results: Of 135 patients, 76 (56.3%) were treated by anti PD-1 and 59 (43.7%) by anti PDL-1 agents. Population was heterogeneous, including patients from the 1st to the 9th line of therapy, affected by the following cancers: 57 (42.2%) NSCLC, 18 (13.3%) melanoma, 13 (9.6%) RCC, 7 (5.2%) bladder and urothelial, 6 (4.5%) mesothelioma, 5 (3.7%) SCLC, 5 (3.7%) sarcoma, 5 (3.7%) biliary tract, 5 (3.7%) head and neck, 3 (2.2%) gastric, 3 (2.2%) colon, 2 (1.5%) Merkel cells and one each for thyroid, HCC, ovary, cervix, anal carcinoma and germ cells tumor. Median follow up was 8.6 months. Best responses were: 5 CR, 30 PR, 51 SD and 49 PD; 86 (63.7%) patients achieved DC. Overall, 38 patients developed dysthyroidism (subclinical, G1, G2 hypo/hyperthyroidism): 18/76 (23.7%) in anti PD-1, 20/59 (33.9%) in anti PDL-1 group; 31/38 (81.6%) of them achieved DC. The median time of dysthyroidism appearance was the 6th cycle of therapy (range 1st-22th). Of 97 patients who did not develop thyroid toxicity, 55 (56.7%) achieved DC. Dysthyroidism significantly correlated with DC rate (p=0.009). Conclusions: We found a significant correlation between dysthyroidism and clinical responses in solid tumor metastatic patients treated by anti PD-1 or anti PDL-1 antibodies. Our observation suggests that evaluation of thyroid function should be regularly performed during therapy with these agents. These retrospective findings must be confirmed in a prospective trial powered to see whether thyroid dysfunction is a surrogate marker of response.

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Elouazzani, H., K. Sirajedine, M. Aladib, and H. Colomb. "Les dysthyroidies chez l’hémodialysé chronique." Néphrologie & Thérapeutique 7, no. 5 (September 2011): 309–10. http://dx.doi.org/10.1016/j.nephro.2011.07.104.

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Desailloud, R. "Dysthyroidies des traitements par cytokines." Annales d'Endocrinologie 74, no. 4 (September 2013): 244. http://dx.doi.org/10.1016/j.ando.2013.07.035.

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Dissertations / Theses on the topic "Dysthyroidie"

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André, Laurence. "Vitiligo et dysthyroi͏̈dies." Montpellier 1, 1995. http://www.theses.fr/1995MON11159.

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CARON, LAVIOLETTE XAVIER. "Etude de l'association dysthyroidies auto-immunes - maladies systemiques : analyse de cent cas (40 polyarthrites rhumatoides, 38 lupus erythemateux dissemines, 12 sclerodermies et 10 syndromes de gougerot-sjogren) et revue de la litterature." Reims, 1990. http://www.theses.fr/1990REIMM081.

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LEVY, CAROLE. "Dysthyroidies auto-immunes du post-partum." Aix-Marseille 2, 1988. http://www.theses.fr/1988AIX20355.

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Tison, Eric. "Dysthyroidies sous amiodarone : observations recueillies a partir de 250 dossiers." Lille 2, 1988. http://www.theses.fr/1988LIL2M307.

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GUILLERAND, MARIE-AUDE. "Dysthyroidies au cours de l'immunotherapie par interleukine 2 en oncologie." Aix-Marseille 2, 1993. http://www.theses.fr/1993AIX20805.

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DAUPHIN, MARC. "Aspects actuels des dysthyroidies a l'amiodarone dans la litterature internationale." Clermont-Ferrand 1, 1989. http://www.theses.fr/1989CLF13825.

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SCHMIDT, XAVIER. "Interet d'un depistage systematique des dysthyroidies secondaires a un traitement par l'amiodarone." Reims, 1988. http://www.theses.fr/1988REIMM085.

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Hocquelet, Joël. "Variations des lipoprotéines au cours des dysthyroidies : à propos de 30 cas." Bordeaux 2, 1988. http://www.theses.fr/1988BOR25432.

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9

Leclercq, Stéphanie Veysseyre Anne-Marie. "Les dysthyroïdies étude des pratiques des médecins généralistes en ambulatoire /." Créteil : Université de Paris-Val-de-Marne, 2008. http://doxa.scd.univ-paris12.fr:80/theses/th0487187.pdf.

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Guérin, Violaine. "Implication du m. A. L. T. (mucosal associated lymphoid tissue) dans la physiopathologie des dysthyroidies auto-immunes." Nancy 1, 1988. http://www.theses.fr/1988NAN11214.

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Books on the topic "Dysthyroidie"

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Kennard, Christopher. Ocular motor disorders. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0274.

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This chapter discusses motor disorders of the eye. The first part of the chapter describes the proper examination of eye movements to facilitate identification of ocular motor disorder pathology. The effects of nerve palsies on ocular motor function are then described.Eye movement disorders can also have their cause in the central nervous system; both the brainstem, and cerebellum have been implicated as causal factors in some eye movement disorders. Disorders of the pupil, which affect the pupillary light reflex, can be caused by lesions to central, afferent and efferent pupillary pathways as well as sympathetic pathways lesions.Finally, this chapter describes diseases of the eye orbits, including dysthyroid eye disease, idiopathic orbital inflammation, orbital tumours, vascular disorders and orbital infections.

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Book chapters on the topic "Dysthyroidie"

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Joseph, Shannon S., and Neil R. Miller. "Management of Dysthyroid Optic Neuropathy." In Emergencies of the Orbit and Adnexa, 235–57. New Delhi: Springer India, 2016. http://dx.doi.org/10.1007/978-81-322-1807-4_28.

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Lane, C. M., and A. Boschi. "Management of Very Severe Graves’ Orbitopathy (Dysthyroid Optic Neuropathy)." In Graves' Orbitopathy, 153–59. Basel: KARGER, 2007. http://dx.doi.org/10.1159/000107474.

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Rizzo, Caterina, Margherita Ilaria Gioia, Giuseppe Parisi, Vincenzo Triggiani, and Massimo Iacoviello. "Dysthyroidism and Chronic Heart Failure: Pathophysiological Mechanisms and Therapeutic Approaches." In Advances in Experimental Medicine and Biology, 239–53. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/5584_2017_132.

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Nakase, Yoshiko, Toshi Higashide, Keiji Yoshikawa, and Youichi Inoue. "Color Doppler Imaging of Orbital Blood Flow in Dysthyroid Ophthalmopathy." In Documenta Ophthalmologica Proceedings Series, 216–24. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0025-0_41.

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Elston, J. S., and P. N. Plowman. "The Effect of Radiotherapy on Abnormal Eye Movements in Dysthyroid Orbitopathy." In Strabismus and Ocular Motility Disorders, 517. London: Macmillan Education UK, 1990. http://dx.doi.org/10.1007/978-1-349-11188-6_75.

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Lane, C. M., and A. Boschi. "Management of Very Severe Graves’ Orbitopathy (Dysthyroid Optic Neuropathy and Corneal Breakdown)." In Graves' Orbitopathy, 159–66. Basel: KARGER, 2010. http://dx.doi.org/10.1159/000320436.

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"Dysthyroid Ophthalmopathy." In Encyclopedia of Ophthalmology, 671. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_100559.

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"Dysthyroid Eye Disease." In Garner and Klintworth's Pathobiology of Ocular Disease (Part B), 273–84. CRC Press, 2007. http://dx.doi.org/10.1201/9781420093919-23.

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"Dysthyroid Eye Disease." In Garner and Klintworth's Pathobiology of Ocular Disease, 1158–69. CRC Press, 2008. http://dx.doi.org/10.3109/9781420020977-55.

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"Dysthyroidal/Thyroid-Associated Orbitopathy." In Encyclopedia of Ophthalmology, 671. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_100560.

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Conference papers on the topic "Dysthyroidie"

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Signorelli, Diego, Michele Del Vecchio, Marina Garassino, Alice Intini, Maria Silvia Cona, Michele Magni, Ettore Seregni, Massimo Di Nicola, and Filippo De Braud. "Abstract A018: Onset of dysthyroidism during treatment with immune checkpoint inhibitors is increased in responder patients." In Abstracts: CRI-CIMT-EATI-AACR Inaugural International Cancer Immunotherapy Conference: Translating Science into Survival; September 16-19, 2015; New York, NY. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/2326-6074.cricimteatiaacr15-a018.

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Reports on the topic "Dysthyroidie"

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Xu, Mingna, Zhaoqi Pan, Yunhai Tu, and Wencan Wu. Orbital Decompression versus Intravenous High-dose Glucocorticoids in Treatment for Dysthyroid Optic Neuropathy. A Systematic Review of the Literature. International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2020. http://dx.doi.org/10.37766/inplasy2020.5.0005.

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