Relevant bibliographies by topics / Dystrophic neurites

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters

Academic literature on the topic 'Dystrophic neurites'

Author: Grafiati
Published: 4 June 2021
Last updated: 10 February 2022

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Journal articles on the topic "Dystrophic neurites"

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Mokhtar, Sara H., Maha M. Bakhuraysah, David S. Cram, and Steven Petratos. "The Beta-Amyloid Protein of Alzheimer’s Disease: Communication Breakdown by Modifying the Neuronal Cytoskeleton." International Journal of Alzheimer's Disease 2013 (2013): 1–15. http://dx.doi.org/10.1155/2013/910502.

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Alzheimer’s disease (AD) is one of the most prevalent severe neurological disorders afflicting our aged population. Cognitive decline, a major symptom exhibited by AD patients, is associated with neuritic dystrophy, a degenerative growth state of neurites. The molecular mechanisms governing neuritic dystrophy remain unclear. Mounting evidence indicates that the AD-causative agent,β-amyloid protein (Aβ), induces neuritic dystrophy. Indeed, neuritic dystrophy is commonly found decorating Aβ-rich amyloid plaques (APs) in the AD brain. Furthermore, disruption and degeneration of the neuronal micro
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Liberski, Paweł P., Agata Gajos, and Beata Sikorska. "Dystrophic neurites accumulating autophagic vacuoles show early stages of neuritic destruction." Folia Neuropathologica 56, no. 3 (2018): 175–78. http://dx.doi.org/10.5114/fn.2018.78696.

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MATTIACE, L. A., Y. KRESS, P. DAVIES, H. KSIEZAK-REDING, S. H. YEN та D. W. DICKSON. "Ubiquitin-immunoreactive Dystrophic Neurites in Downʼs Syndrome Brains". Journal of Neuropathology and Experimental Neurology 50, № 5 (1991): 547–59. http://dx.doi.org/10.1097/00005072-199109000-00003.

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Satoh, Jun-ichi, Hiroko Tabunoki, Tsuyoshi Ishida, Yuko Saito, and Kunimasa Arima. "Dystrophic neurites express C9orf72 in Alzheimer's disease brains." Alzheimer's Research & Therapy 4, no. 4 (2012): 33. http://dx.doi.org/10.1186/alzrt136.

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Chen, Tong, Wei-Ping Gai, and Catherine A. Abbott. "Dipeptidyl Peptidase 10 (DPP10789): A Voltage Gated Potassium Channel Associated Protein Is Abnormally Expressed in Alzheimer’s and Other Neurodegenerative Diseases." BioMed Research International 2014 (2014): 1–15. http://dx.doi.org/10.1155/2014/209398.

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The neuropathological features associated with Alzheimer’s disease (AD) include the presence of extracellular amyloid-βpeptide-containing plaques and intracellular tau positive neurofibrillary tangles and the loss of synapses and neurons in defined regions of the brain. Dipeptidyl peptidase 10 (DPP10) is a protein that facilitates Kv4 channel surface expression and neuronal excitability. This study aims to explore DPP10789protein distribution in human brains and its contribution to the neurofibrillary pathology of AD and other tauopathies. Immunohistochemical analysis revealed predominant neur
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Weghe, Jennifer Vande, Patrick Cras, Mitsuru Kawai, et al. "Dystrophic neurites infiltrate extracellular neurofibrillary tangles in Alzheimer disease." Brain Research 560, no. 1-2 (1991): 303–5. http://dx.doi.org/10.1016/0006-8993(91)91247-x.

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Paula, Vanessa de Jesus R. de, Fabiana Meira Guimarães, Breno Satler Diniz, and Orestes Vicente Forlenza. "Neurobiological pathways to Alzheimer's disease: Amyloid-beta, TAU protein or both?" Dementia & Neuropsychologia 3, no. 3 (2009): 188–94. http://dx.doi.org/10.1590/s1980-57642009dn30300003.

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Abstract Alzheimer's disease (AD) is a neurodegenerative disease characterized by progressive cognitive decline, including memory loss, behavioral and psychological symptoms and personality changes. The neuropathological hallmarks of AD are the presence of neuritic (senile) plaques (NP) and neurofibrillary tangles (NFT), along with neuronal loss, dystrophic neurites, and gliosis. Neuritic plaques are extracellular lesions and their main constituent is the amyloid-b42 peptide (Ab42). Neurofibrillary tangles are intracellular lesions that are mainly composed of hyperphosphorylated TAU protein. I
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de la Monte, S. M., and J. R. Wands. "Diagnostic utility of quantitating neurofilament-immunoreactive Alzheimer's disease lesions." Journal of Histochemistry & Cytochemistry 42, no. 12 (1994): 1625–34. http://dx.doi.org/10.1177/42.12.7983363.

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The diagnosis of Alzheimer's disease (AD) neurodegeneration is based on histopathological detection of paired helical filament-associated lesions. Silver stains are routinely used but the results are fraught with intra- and interinstitutional variability. This study employed monoclonal antibodies to middle and high molecular weight neurofilament subunits in an immunohistochemical assay to assess the extent of paired helical filament-associated lesions in brains with AD, Down's syndrome plus AD lesions (AD+DN), Parkinson's disease dementia (PD), AD+PD, and normal aging changes. The densities of
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Sharoar, Md Golam, Xiangyou Hu, Xin-Ming Ma, Xiongwei Zhu, and Riqiang Yan. "Dystrophic neurites labelled by pre-autophagosomal proteins in Alzheimer’s brains." Molecular Psychiatry 24, no. 9 (2019): 1247. http://dx.doi.org/10.1038/s41380-019-0476-3.

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Su, Joseph H., Brian J. Cummings, and Carl W. Cotman. "Identification and distribution of axonal dystrophic neurites in Alzheimer's disease." Brain Research 625, no. 2 (1993): 228–37. http://dx.doi.org/10.1016/0006-8993(93)91063-x.

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Dissertations / Theses on the topic "Dystrophic neurites"

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Wente, Sarah Luise. "Untersuchungen zu synaptischen Proteinen in einem Tiermodell für die Alzheimer-Krankheit." Doctoral thesis, 2011. http://hdl.handle.net/11858/00-1735-0000-0006-B20B-2.

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Book chapters on the topic "Dystrophic neurites"

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Ikeda, S., N. Yanagisawa, D. Allsop, and G. G. Glenner. "A variant of Gerstmann-Sträussler-Scheinker disease with ß -protein epitopes and dystrophic neurites in the peripheral regions of PrP -immunoreactive amyloid plaques." In Amyloid and Amyloidosis 1990. Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3284-8_180.

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Kosik, K. S. "The Neuritic Dystrophy of Alzheimer’s Disease: Degeneration or Regeneration?" In Growth Factors and Alzheimer’s Disease. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-46722-6_20.

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Journal articles
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