Relevant bibliographies by topics / E. coli 0157:H7

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Academic literature on the topic 'E. coli 0157:H7'

Author: Grafiati
Published: 4 June 2021
Last updated: 17 February 2022

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Journal articles on the topic "E. coli 0157:H7"

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Neill, Marguerite A. "Escherichia coli 0157:H7." Infectious Diseases Newsletter 10, no. 3 (March 1991): 19–24. http://dx.doi.org/10.1016/0278-2316(91)90046-3.

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OKREND, ANITA J. G., BONNIE E. ROSE, and CHARLES P. LATTUADA. "Isolation of Escherichia coli 0157:H7 Using 0157 Specific Antibody Coated Magnetic Beads." Journal of Food Protection 55, no. 3 (March 1, 1992): 214–17. http://dx.doi.org/10.4315/0362-028x-55.3.214.

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Escherichia coli 0157 specific antibody, coated on magnetic beads, was used to concentrate and remove the E. coli 0157:H7 from mixed cultures and meat samples. The problem of nontarget organism carryover was addressed by adding Protamine to the culture-bead sample, washing the beads three times in saline, and changing the test tubes with each wash. These modifications reduced the nontarget colony counts obtained from uninoculated meat samples. This procedure enabled consistent recovery of E. coli 0157:H7 from inoculated meat samples. The percentage of E. coli 0157:H7 cells captured, compared to the total number of cells captured, ranged from 48 to 100%. Two strains of E. coli 0157, H7 and :non-H7, appeared to compete with one another and thus reduce or prevent isolation.
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Roberts, James R. "Escherichia coli 0157:H7 Infections." Emergency Medicine News 26, no. 13 (January 2004): 20–22. http://dx.doi.org/10.1097/00132981-200401000-00020.

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Rowe, PaulM. "Eradicating E coli 0157:H7." Lancet 345, no. 8942 (January 1995): 117. http://dx.doi.org/10.1016/s0140-6736(95)90075-6.

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Hammack, Thomas S., Peter Feng, R. Miguel Amaguaña, Geraldine June, Patricia S. Sherrod, and Wallace H. Andrews. "Comparison of Sorbitol MacConkey and Hemorrhagic Coli Agars for Recovery of Escherichia coli 0157:H7 from Brie, Ice Cream, and Whole Milk." Journal of AOAC INTERNATIONAL 80, no. 2 (March 1, 1997): 335–40. http://dx.doi.org/10.1093/jaoac/80.2.335.

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Abstract The relative efficacies of hemorrhagic coli (HC) agar and several formulations of sorbitol Mac-Conkey (SorMac) agar, with and without 0.1 % (w/v) 4-methyllumbelliferyl-ß-D-glucuronide (MUG), in recovering unstressed and heat-stressed Escherichia coli 0157:H7 from Brie cheese, ice cream, and whole milk were determined. Recovery of unstressed E. coli 0157:H7 was determined quantitatively by spread-plating diluted samples onto different agars and performing plate counts. Recovery of stressed E. coli 0157:H7 was determined qualitatively by enriching samples in modified trypticase soy broth, streaking the incubated enrichments, and isolating E. coli 0157:H7 colonies from the agars. HC agar and the SorMac agar formulations did not differ significantly in their ability to recover unstressed E. coli 0157:H7 from ice cream and whole milk; however, HC agar recovered significantly more unstressed E. coli 0157:H7 from Brie cheese than did the SorMac agar formulations. Bacteriological Analytical Manual and Oxoid SorMac agar formulations made from individual ingredients, did not differ significantly in recovering unstressed E. coli 0157:H7 from Brie cheese. The efficiency of the commercially available Oxoid SorMac agar could not be determined because of overgrowth by indigenous microflora. HC and SorMac agars did not differ significantly in recovering stressed E. coli 0157:H7 from Brie cheese, ice cream, and whole milk. MUG had no apparent effect on recovery of either stressed or unstressed E. coli 0157:H7 from the dairy foods examined.
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Mcgowan, KarinL, Ellen Wickersham, and NancyA Strockbine. "ESCHERICHIA COLI 0157:H7 FROM WATER." Lancet 333, no. 8644 (April 1989): 967–68. http://dx.doi.org/10.1016/s0140-6736(89)92559-2.

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Yannelli, Barbara, Philip Domenico, Burke A. Cunha, and Sayed M. H. Quadri. "Nosocomial Escherichia coli 0157:H7 diarrhea." American Journal of Infection Control 18, no. 5 (October 1990): 341–42. http://dx.doi.org/10.1016/0196-6553(90)90236-l.

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Griffin, Patricia M., Linda C. Olmstead, and Robert E. Petras. "Escherichia coli 0157:H7-associated colitis." Gastroenterology 99, no. 1 (July 1990): 142–49. http://dx.doi.org/10.1016/0016-5085(90)91241-w.

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RONNER, AMY B., and DEAN O. CLIVER. "Isolation and Characterization of a Coliphage Specific for Escherichia coli 0157:H7." Journal of Food Protection 53, no. 11 (November 1, 1990): 944–47. http://dx.doi.org/10.4315/0362-028x-53.11.944.

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Enterohemorrhagic Escherichia coli serogroup 0157:H7 is harbored by cattle and causes bloody diarrhea and hemolytic uremic syndrome in persons who consume raw milk and under-cooked beef. Samples of manure from Wisconsin dairy farms were tested for the presence of E. coli 0157:H7 as well as for bacteriophages (coliphages) specific for this microorganism. No E. coli 0157:H7 bacteria were isolated from any of the 21 manure samples taken from 12 farms. Nineteen of 20 samples yielded “nonspecific” coliphages that produced plaques both on 0157:H7 and on other E. coli. Only one sample yielded a coliphage that plaqued on 14 strains of 0157:H7 but not on other E. coli. This coliphage, designated “AR1,” is tailed and ca. 187 nm long; it produces distinct plaques ca. 0.5 mm in diameter; single-step growth experiments showed a latent period of 20 to 25 min and a burst size of 34 progeny plaque-forming units (PFU). AR1 was also tested against other enterobacteria, including: Escherichia hermanii, four species of Salmonella, four types of Yersinia enterocolitica, and a strain of Shigella dysenteriae which produces an enteric toxin similar to that produced by E. coli 0157:H7. Of these enteric bacteria, only S. dysenteriae yielded plaques, which suggests that there is a relationship between production of this toxin and susceptibility to coliphage AR1. Coliphage AR1 may be useful in detecting or identifying E. coli 0157:H7 and possibly other bacteria producing the same toxin, from human stool, animal manure, and food samples.
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OKREND, ANITA J. G., BONNIE E. ROSE, and CHARLES P. LATTUADA. "Use of 5-Bromo-4-Chloro-3-lndoxyl-β-D-Glucuronide in MacConkey Sorbitol Agar to Aid in the Isolation of Escherichia coli 0157:H7 from Ground Beef." Journal of Food Protection 53, no. 11 (November 1, 1990): 941–43. http://dx.doi.org/10.4315/0362-028x-53.11.941.

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The addition of 5-bromo-4-chloro-3-indoxyl-β-D-glucuronide (BCIG) at the 0.1 g/L level, to MacConkey sorbitol agar (MSA) plates aided in the isolation of Escherichia coli 0157:H7 from raw ground beef samples by differentiating β-glucuronidase positive from β-glucuronidase negative colonies. E. coli 0157:H7 colonies, being sorbitol negative, β-glucuronidase negative, remained white, while sorbitol negative, β-glucuronidase positive colonies turned green to blue. Addition of BCIG to the MSA agar reduced the number of false suspect colonies picked from the primary plating medium by 36% when compared to MSA. E. coli 0157:H7 was isolated from 11 out of 12 inoculated meat samples (0.7 E. coli 0157:H7/g) using MSA-BCIG as compared to 8 out of 12 samples using MSA without BCIG.
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Dissertations / Theses on the topic "E. coli 0157:H7"

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Silva, Neusely da. "Escherichia coli 0157:H7 em alimentos." [s.n.], 2004. http://repositorio.unicamp.br/jspui/handle/REPOSIP/256596.

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Orientador: Fumio Yokoya
Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Engenharia de Alimentos
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Zin, Noraziah Mohamad. "Studies on escherichia coli 0157:H7 and its toxins." Thesis, University of Strathclyde, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.366820.

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Garven, Sarah Jane. "Bovine dendritic cells & their interaction with E. coli 0157:H7." Thesis, University of Edinburgh, 2011. http://hdl.handle.net/1842/8741.

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E. coli O157:H7 is the most important serotype of enterohaemorrhagic E. coli (EHEC) which is of concern to public health worldwide. As a common cause of haemorrhagic colitis, EHEC infection can progress to life threatening sequelae including haemolytic uraemic syndrome (HUS) in humans. Human infection rates are higher in Scotland than found in the rest of the UK. Cattle are asymptomatic carriers of EHEC and are an important reservoir from which disease outbreaks can spread. The terminal rectum has been indicated as a site of E. coli O157:H7 colonisation in the bovine intestinal tract. This is the location of numerous lymphoid follicles which contain dendritic cells (DCs) which are professional antigen presenting cells and important directors of immune responses. DCs are likely to come into contact with EHEC and therefore could be key in this location for enabling EHEC to colonise the bovine host. The first aim of this project was to characterise dendritic cells within the bovine intestinal tract at various anatomical locations, including the terminal rectum, using immunohistochemistry techniques. Following this, work to extract and further phenotype dendritic cells from terminal rectal tissues was undertaken. Finally, a widely-used bovine dendritic cell model was employed to generate dendritic cells from circulating blood monocytes. This model was utilised to investigate the interactions of dendritic cells with EHEC strains compared with responses to bovine enterotoxigenic (ETEC) and bovine commensal E. coli strains. Early work identified that there are potentially numerous DCs within the bovine intestinal tissues and these cells were found in greater numbers at the terminal rectum. Protocols to extract and further characterise these cells were developed but proved inconsistent, with large variation between animals. Using the monocyte derived dendritic cells (moDCs), differences were observed between immunological responses to challenge with E. coli O157:H7 strains and bovine pathogenic or commensal E. coli strains. Cytokine production, cell surface molecule expression, cell phenotype and viability as well as intracellular bacterial counts were compared. The data presented here shows that the bovine moDCs respond differently to EHEC strains when compared with commensal or pathogenic E. coli in several key areas. This has important implications for the responses of the bovine host to various E. coli strains. This work also indicates that dendritic cells could be central to these responses and if studied further still, may hold the key to reducing the colonization and persistence of E. coli O157:H7 in cattle, and subsequent human disease outbreaks.
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Dlamini, Bhekisisa Chushuta. "Acid adaptation of Escherichia coli 0157:H7 in fermented goat milk." Pretoria : [s.n.], 2008. http://upetd.up.ac.za/thesis/available/etd-02102009-102022.

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Paton, Neil. "Bovine epithelial cell responses to colonisation by Escherichia coli 0157:H7." Thesis, University of Edinburgh, 2009. http://hdl.handle.net/1842/29314.

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E. coli 0157:H7 was the first identified pathogen of a group that have come to be referred to as Enterohaemorrhagic E. coli (EHEC) and was identified in 1983 as being associated with Haemolytic ureamic syndrome (HUS). This bacterium carries the potent cytotoxin Shigalike toxin (Stx), also known as verotoxin, which through the inhibition of protein synthesis causes cell necrosis in the endothelium of the renal vasculature and this leads to the triad of symptoms - renal failure, thrombocytopenia and microangiopathic haemolytic anaemia. EHEC is also associated with Haemorrhagic colitis in humans as endothelium of both the colonic vasculature and the renal system express Gb3 receptors. These receptors bind the toxin and internalisation of the toxin allows the inhibition of protein synthesis. EHEC is a food borne zoonosis and its reservoir host is the bovine and faecal contamination of the environment, the food chain and direct contact with cattle are the most recognised routes for human infection to occur. The bovine host is asymptomatically colonised in the field and detection and removal is problematic, any future intervention strategy to remove this pathogen from the national herd is likely to be expensive and labour intensive. EHEC has a number of virulence factors that are involved in colonisation and proinflammatory responses. These were examined in a bovine model system. The flagellum was the only virulence factor that produced a proinflammatory response when measured by quantitative and traditional RTPCR. Commensal bacteria were unable to produce a response although one motile strain was included in the panel and was presumed to express flagella which were shown to be pro-inflammatory when associated with EHEC. In the host animal there is limited evidence for a pathological effect and to elucidate the mechanisms that explain this lack of response to pathogenic effects microarray was utilised to tease out these mechanisms. The data produced identified a limited set of genes that were differentially regulated including CyclinC Angiopoetin-1 like protein, Jumonji domain containing protein 2B, Zinc finger protein 161 and Est-lplike protein, all of which were thought to be involved in cell cycle regulation. Quantitative RTPCR was unable to confirm the data from the array; further work is therefore required to determine whether colonisation does in fact alter the expression of these genes. EHEC was therefore hypothesized to alter the proliferation rate within the epithelium and an immunohistochemical approach was used to assess this. Proliferating cell nuclear antigen (PCNA) was used as a marker to identify replicating cells and counts of cells in the epithelium demonstrated a reduction in proliferating cells in colonised epithelium. Further analysis suggested that retinoblastoma protein was a central protein that was involved in pathways influenced by the proteins already outlined. IHC was used to study this protein and differences in the number of cells expressing this protein and localisation within the cells. Retinoblastoma appears to be retained in the cytoplasm in colonised cattle which limits its ability to induce proliferation through release of E2F. It is suggested that E.coli 0157:H7 can manipulate the epithelial cell proliferation rate in the bovine host and increases the time that the bacterium is retained in the host. It was hoped that microarray data and QtRT-PCR would identify proteins involved in this phenotype but the lack of support from the real time data for targets identified by the array makes it impossible to conclude that these proteins are defiantly involved. This increase in time allows for a greater chance of spread to other host animals within the herd. Further work to clarify the details of this pathway will allow interventions which limit colonisation the National herd to be designed.
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Byrne, Caitriona Martina. "Survival and persistence of Escherichia coli 0157:H7 within meat production processes." Thesis, University of Ulster, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.365916.

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Ding, Yajun Mustapha Azlin. "Transfer of Listera monocytogenes and Escherichia coli 0157:H7 during food processing." Diss., Columbia, Mo. : University of Missouri-Columbia, 2008. http://hdl.handle.net/10355/6071.

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The entire thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file; a non-technical public abstract appears in the public.pdf file. Title from PDF of title page (University of Missouri--Columbia, viewed on Oct. 7, 2009). Thesis advisor: Dr. Azlin Mustapha. Includes bibliographical references.
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Bromberg, Renata. "Fatores que afetam a recuperação de celulas de Escherichia coli 0157:H7 termicamente injuriadas." [s.n.], 1997. http://repositorio.unicamp.br/jspui/handle/REPOSIP/256612.

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Orientadores: Fumio Yokoya, Michael William Peck
Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Engenharia de Alimentos
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Resumo: Um método popular de se prolongar a vida-de-prateleira de alimentos minimamente processados é através da utilização de tratamentos térmicos brandos e embalagem com atmosfera modificada (com baixas concentrações de oxigênio). Tradicionalmente, a eficácia dos tratamentos térmicos aplicados a estes alimentos é medida através da contagem em aerobiose das células injuriadas pelo calor. Contudo, no caso da bactéria Escheríchia coli 0157:H7, obtém-se uma melhor recuperação das células subletalmente injuriadas quando se utiliza técnicas estritamente anaeróbias. Estudou-se o efeito do tratamento térmico brando em células de E. Coli 0157H7 e as condições de recuperação da sua sensibilidade ao oxigênio em meio de cultura e em meio a base de alimentos. Células de E. Coli 0157:H7 em fase estacionária de crescimento, cultivadas em meio triptona de soja acrescido de 0,3% de extrato de levedura e 1% de glicose (TSYGB) a 30°C durante 24 horas, e submetidas a tratamento térmico a 59°C durante 5 minutos sob condições anaeróbias, recuperaram sua habilidade de crescer em presença de oxigênio em 6 horas. Similarmente, quando as células foram submetidas a tratamento térmico e mantidas em presença de 20% de oxigênio, estas apresentaram maior sensibilidade ao oxigênio, necessitando de um período maior de tempo para se recuperar do que as células mantidas em condições de anaerobiose (9 horas comparado a 6 horas). Células de E. coli 0157:H7 foram submetidas a tratamentos térmicos de letalidade equivalente a 55, 59 e 61 °C. Após tratamento térmico a 55°C por 100 minutos, as células necessitaram de mais de 20 horas para recuperar sua tolerância ao oxigênio, comparativamente a 6 e 2 horas para as células tratadas termicamente a 59°C por 5 minutos e a 61 °C por 1 minuto, respectivamente. Isto indica, que as células deste microrganismo recuperam sua sensibilidade ao oxigênio mais rapidamente após tratamentos térmicos mais curtos com temperaturas mais altas. Células de E. coli 0157:H7 submetidas a tratamento térmico a 59°C durante 5 minutos e incubadas a 10, 20 e 30°C, recuperaram sua tolerância ao oxigênio após aproximadamente 130, 50 e 6 horas, respectivamente. Contudo, quando mantidas a 5°C, estas células não recuperaram sua tolerância, sobrevivendo no mesmo nível durante o período de observação de 34 dias. A inclusão de 50% de dióxido de carbono ao meio de contagem, não apresentou efeito na recuperação das células sensíveis ao oxigênio, porém o aumento da concentração de oxigênio afetou a recuperação das células injuriadas. O choque térmico a 42°C durante 5 minutos, seguido de tratamento térmico a 59°C durante 5 minutos não influenciou a resistência térmica ou o reparo celular de E. coli 0157:H7. Entretanto, células deste microrganismo submetidas a choque térmico a 45°C durante 5 minutos, apresentaram um aumento em sua resistência térmica de cerca de 10 vezes, tanto em condições aeróbias como anaeróbias, não afetando porém, sua sensibilidade ao oxigênio. E. coli 0157:H7 foi inoculada em tubos contendo meios a base de alimentos, preparados em condições aeróbias e anaeróbias. Estes foram submetidos a tratamentos térmicos e incubados a 30°C. Foi verificada uma grande diferença na resistência térmica das células nos diferentes alimentos testados. Os valores Dsg-c nos tubos preparados em anaerobiose foram, para carne bovina 5,3 minutos, cogumelo 4 minutos, frango 3,7 minutos e de apenas 1,9 minutos para leite. Os valores D nos meios preparados em aerobiose foram similares aos obtidos no meio anaeróbio. Isto ocorreu porque os meios preparados em condições aeróbias apresentam potenciais de óxido-redução reduzidos, similares aos meios preparados em anaerobiose. Não se detectou crescimento de E. coli 0157:H7 em meios preparados a base de batata, brócolis e cenoura.
Abstract: A popular method of extending the shelf life of minimally processed foods is to subject the food to a mild heat treatment and package under a modified atmosphere (low level of oxygen). The effectiveness of the heat treatment is measured by aerobic enumeration of heat damaged cells. However, for Escherichia coli 0157:H7, the greatest recovery of sublethally damaged cells was obtained using strictly anaerobic techniques The effect of a mild heat treatment and the recovery conditions on oxygen sensitivity of E coli 0157:H7 cells was studied in media and foods. Stationary phase cells grown in tryptone soy agar with 0.3% yeast extract and 1 % glucose (TSYGB) at 30°C for 24 hours and heat treated at 59°C for 5 minutes recovered their ability to grow in oxygen in 6 hours when held in anaerobic conditions. Similarly when cells were heat treated and held in the presence of 20% oxygen, the cells were more sensitive to oxygen and took longer to recover than cells held in anaerobic conditions (9 hours compared with 6 hours). Cells of E coli 0157.H7 were subjected to heat treatments of equivalent lethality at 55, 59 and 61 °C. Following a heat treatment of 100 minutes at 55°C, cells took 20 hours to recover their tolerance to oxygen compared to 6 and 2 hours for cells heat treated for 5 minutes at 59°C and 1 minute at 61 °C, respectively. This shows that injured cells recover their oxygen tolerance faster after a short heat treatment at a higher temperature. Cells heat treated at 59°C for 5 minutes and held at 10, 20 and 30°C, recovered their oxygen tolerance after 130, 50 and 6 hours respectively. However, when held at 5°C, the cells did not recover their oxygen tolerance in 34 days. The inclusion of 50% carbon dioxide in the enumeration medium had no effect on the recovery of oxygen sensitive cells but oxygen concentration affected the recovery of injured cells A heat shock of 5 minutes at 42°C had no effect on heat resistance or recovery of damaged cells. However, 5 minutes at 45°C before a heat treatment of 5 minutes at 59°C increased the heat resistance of E. coli 0157:H7 by 10 fold but had no effect on its oxygen sensitivity. E. coli 0157:H7 was inoculated into tubes of food media prepared aerobically and anaerobicallly and given a heat treatment before incubating at 30°C. There was a large difference in heat resistance of cells in the different foods tested. Dsg-c in anaerobically prepared beef was 5.3 minutes, in mushroom, 4.0 minutes, in chicken, 3.7 minutes and in milk only 1.9 minutes. D values in aerobically prepared media were similar to those in anaerobic media. This was because aerobically prepared media had very low redox potentials, similar to those of the anaerobically prepared media. Growth of E. coli 0157:H7 was not detected in broccoli, carrot or potato media.
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Naylor, Stuart Warren. "Colonisation and persistence of Escherichia coli 0157:H7 in the bovine gastro-intestinal tract." Thesis, University of Edinburgh, 2003. http://hdl.handle.net/1842/27109.

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The principal aim during this study was to develop appropriate in vitro and in vivo systems to examine colonisation mechanisms of E. coli O157:H7 in the bovine GIT. An adherence assay on cultured tissue explants was developed to compare the ability of different aspects of E. coli O157:H7 adherence. In two separate experiments the contribution of intimate attachment, thought to be essential for virulence in humans, was assessed. The ability to intimately attach did not affect the level of E. coli O157:H7 adherence to bovine intestinal epithelium in vitro. A strain lacking the genes required for intimate attachment however exhibited enhanced adherence to bovine Peyer’s patch. The other strains did not exhibit tropism for any of the tissue type examined. The most relevant system to assess the behaviour of E. coli O157:H7 is within its natural host. Persistent colonisation of weaned calves was achieved for a number of isolates marked by nalidixic acid resistance, including a Stx negative strain that colonised at a similar level and duration to the equivalent Stx positive strain. At the conclusion of each calf colonisation experiment, those individuals still shedding the organism were examined under necropsy to determine its distribution. The first attempts failed to recover the organism in significant numbers at any site examined despite it being present in ante-mortem faeces. One explanation was that the organism was multiplying primarily in the distal rectum. Further necropsies revealed that the organism was colonising the mucosal surface of the distal 3 cm of the rectum via intimate attachment and confirmed that this phenomenon was indeed typical of persistently colonised calves. This small region contained a high density of lymphoid tissue.
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Emmerson, James R. "Tagging of the type three secretion system basal apparatus of enterohaemorrhagic Escherichia coli 0157:H7." Thesis, University of Edinburgh, 2008. http://hdl.handle.net/1842/30180.

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The aim of this project was to label inner membrane basal apparatus proteins with fluorescent markers or immunogenic tags in order to investigate their function, regulation and localization. Cloning strategies were designed to insert tags at the 3’ end of eight genes encoding putative inner membrane proteins and to use these fusions to replace the wild type sequences by allelic exchange. From the eight strategies, escR and escU were taken forward to produce five EHEC O157:H7 strains. One strain contained EscR labelled at the C-terminus with an HA (haemagglutinin) epitope tag and the other four strains contained different labelled versions of EscU. Work with these strains demonstrated that the T3SS could not be visualized using fluorescence microscopy. However, Western blot analysis did show that the EscU protein was cleaved into 30kDa and 10kDa peptides, both of which localized to the membrane fraction of the bacterial cell. This cleavage was most likely occurring at the conserved cleavage site NPTH. Interestingly all the mutant strains constructed did not secrete a detectable level of EspD, apart from one fusion that had previously been shown to cleave the tag from EscU. This indicated that, despite the small size of the HA tag, all the tags interfered with the function of EscR or EscU. Fusions to the 10kDa fragment of EscU, along with a deletion of this domain, were used to elucidate its function in the T3SS. All the EscU mutants did not form EspA filaments, secrete EspD at wild type levels or secrete detectable levels of Tir, whilst the expression from the LEE1-5 promoters remained unaffected. These phenotypes could not be restored upon supplying the 10kDa peptide in trans. It is proposed that the uncleaved EscU protein is needed to secrete EscF. After a defined period EscU is cleaved (possibly auto-catalytically) and this allows the secretion of EspA, B and D. The lack of wild type EscU in the mutant strains may not allow EscF to be secreted or assembled correctly and this in turn leads to the inhibition of EspA, B and D translocation. It was evident that even minimal changes were not tolerated and inhibited secretion by the system. Future research will have to proceed with either alternative target proteins or the generation of high affinity antibodies coupled with sensitive imaging technology.
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Books on the topic "E. coli 0157:H7"

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Bukhari, Zia. Improved detection methods for E. coli 0157: H7. Denver, CO: Awwa Research Foundation, 2005.

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Middleton, Karen Elizabeth. Investigation of the effect of acids and detergents on the biocide susceptibility of Escherichia Coli 0157:H7. Wolverhampton: University of Wolverhampton, 2003.

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Makwana, Bhanumati. The effect of low pH on the survival of Escherichia coli 0157:H7 (EHEC) in ground beef. Wolverhampton: University of Wolverhampton, 1995.

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Ching, Cheung Yee Joyce. Mechanisms of escherichia coli 0157:H7-derived shiga-like toxins induction of programmed cell death in epithelial cells. Ottawa: National Library of Canada, 2002.

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Service, United States Food Safety and Inspection. E. coli 0157:H7: What you need to know if there is an outbreak in your community : background information, prevention guidelines, protecting your children, USDA-E. coli control efforts. Washington, D.C.]: U.S. Dept. of Agriculture, Food Safety and Inspection Service, 1995.

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Miller, Ellen Kay. Escherichia coli 0157: January 1994 - July 1995. Beltsville, Md: National Agricultural Library, 1995.

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Miller, Ellen Kay. Escherichia coli 0157: January 1994 - July 1995. Beltsville, Md: National Agricultural Library, 1995.

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Kay, Miller Ellen. Escherichia coli 0157: January 1994 - July 1995. Beltsville, Md: National Agricultural Library, 1995.

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United States. Animal and Plant Health Inspection Service. Veterinary Services. Centers for Epidemiology and Animal Health. Escherichia coli O157:H7: Issues and ramifications : executive summary. Fort Collins, Colo: USDA:APHIS:VS, Centers for Epidemiology and Animal Health, 1994.

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Miller, Ellen Kay. Escherichia coli O157: January 1993 - December 1993. Beltsville, Md: National Agricultural Library, 1994.

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Book chapters on the topic "E. coli 0157:H7"

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Griffin, Patricia M., and Thomas G. Boyce. "Escherichia coli O157:H7." In Emerging Infections 1, 137–45. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816940.ch9.

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Smith, David R. "Vaccination of Cattle against Escherichia coli O157:H7." In Enterohemorrhagic Escherichia coli and Other Shiga Toxin-Producing E. coli, 487–501. Washington, DC, USA: ASM Press, 2015. http://dx.doi.org/10.1128/9781555818791.ch25.

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Besser, Thomas E., Margaret A. Davis, and Seth T. Walk. "Escherichia coli O157:H7 in Reservoir Hosts." In Population Genetics of Bacteria, 303–24. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555817114.ch18.

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Bailey, C. W., and C. A. Carson. "Variation in Manifestation of E. coli H7 Antigen." In Advances in Experimental Medicine and Biology, 83–85. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1828-4_11.

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Lacher, David W. "The Evolutionary Model of Escherichia coli O157:H7." In Population Genetics of Bacteria, 225–39. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555817114.ch13.

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Laury, Angela, Alejandro Echeverry, and Mindy Brashears. "Fate of Escherichia coli O157:H7 in Meat." In Safety of Meat and Processed Meat, 31–53. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-0-387-89026-5_2.

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Kendall, Melissa M. "Interkingdom Chemical Signaling in Enterohemorrhagic Escherichia coli O157:H7." In Microbial Endocrinology: Interkingdom Signaling in Infectious Disease and Health, 201–13. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-20215-0_9.

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Dean-Nystrom, Evelyn A., Brad T. Bosworth, and Harley W. Moon. "Pathogenesis of Escherichia Coli O157:H7 in Weaned Calves." In Mechanisms in the Pathogenesis of Enteric Diseases 2, 173–77. Boston, MA: Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4143-1_16.

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Szu, Shousun Chen, and Amina Ahmed. "Clinical Studies of Escherichia coli O157:H7 Conjugate Vaccines in Adults and Young Children." In Enterohemorrhagic Escherichia coli and Other Shiga Toxin-Producing E. coli, 477–85. Washington, DC, USA: ASM Press, 2015. http://dx.doi.org/10.1128/9781555818791.ch24.

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Dean-Nystrom, Evelyn A., Brad T. Bosworth, and Harley W. Moon. "Pathogenesis of O157:H7 Escherichia Coli Infection in Neonatal Calves." In Advances in Experimental Medicine and Biology, 47–51. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1828-4_5.

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Conference papers on the topic "E. coli 0157:H7"

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Tu, Shu-I., Joseph Uknalis, Deidre Patterson, and Andrew G. Gehring. "Detection of immunomagnetically captured 4',6-diamidino-2-phenyl-indole (DAPI)-labeled Escherichia coli 0157:H7 by fluorescent microscopic imaging." In Photonics East (ISAM, VVDC, IEMB), edited by Yud-Ren Chen. SPIE, 1999. http://dx.doi.org/10.1117/12.335780.

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Tu, Shu-I., and Andrew Gehring. "Detection of Escherichia coli O157:H7 using immuno beads." In Optics East 2005, edited by Yud-Ren Chen, George E. Meyer, and Shu-I. Tu. SPIE, 2005. http://dx.doi.org/10.1117/12.629949.

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Dastider, S. Ghosh, S. Barizuddin, N. Yuksek, M. Dweik, and M. Almasri. "Impedance biosensor for rapid detection of low concentration of E.coli 0157:H7." In 2016 IEEE 29th International Conference on Micro Electro Mechanical Systems (MEMS). IEEE, 2016. http://dx.doi.org/10.1109/memsys.2016.7421620.

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Stephen Radke and Evangelyn Alocilja. "Impedimetric Biosensor for the Rapid Detection of Escherichia coli O157:H7." In 2003, Las Vegas, NV July 27-30, 2003. St. Joseph, MI: American Society of Agricultural and Biological Engineers, 2003. http://dx.doi.org/10.13031/2013.14181.

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Dujuan Li, Jianping Wang, Yibin YIng, and Yanbin Li. "Rapid Detection of Escherichia coli O157:H7 Using Electrochemical Impedance Immunosensor." In 2007 Minneapolis, Minnesota, June 17-20, 2007. St. Joseph, MI: American Society of Agricultural and Biological Engineers, 2007. http://dx.doi.org/10.13031/2013.23116.

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Tu, Shu-I., Joseph Uknalis, and Andrew Gehring. "Optical methods for detecting Escherichia coli O157:H7 spiked on cantaloupes." In Optics East, edited by Yud-Ren Chen and Shu-I. Tu. SPIE, 2004. http://dx.doi.org/10.1117/12.569269.

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C.A. Meeusen, E.C. Alocilja, and W. Osburn. "Detection of E. coli O157:H7 Using a Surface Plasmon Resonance Biosensor." In 2001 Sacramento, CA July 29-August 1,2001. St. Joseph, MI: American Society of Agricultural and Biological Engineers, 2001. http://dx.doi.org/10.13031/2013.5549.

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Dujuan (or initial) Li, Jianping (or initial) Wang, Yibin (or initial) Ying, Zunzhong (or initial) Ye, and Yanbin (or initial) Li. "Development of a Capacitive Immunosensor for Detection of Escherichia coli O157:H7." In 2008 Providence, Rhode Island, June 29 - July 2, 2008. St. Joseph, MI: American Society of Agricultural and Biological Engineers, 2008. http://dx.doi.org/10.13031/2013.24936.

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Danyelle D. Small, Chandra S. Theegala, and Todd W. Monroe. "Detection of Escherichia coli O157:H7 in Water using an Amperometric Biosensor." In 2006 Portland, Oregon, July 9-12, 2006. St. Joseph, MI: American Society of Agricultural and Biological Engineers, 2006. http://dx.doi.org/10.13031/2013.21018.

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Ratna R. Sharma and Ali Demirci. "Treatment of E. coli O157:H7 Contaminated Alfalfa Seeds with Ozonated Water." In 2001 Sacramento, CA July 29-August 1,2001. St. Joseph, MI: American Society of Agricultural and Biological Engineers, 2001. http://dx.doi.org/10.13031/2013.7513.

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Reports on the topic "E. coli 0157:H7"

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Ko, Kyung Yuk, Aubrey F. Mendonca, and Dong U. Ahn. EDTA and Lysozyme Improves Antimicrobial Activities of Ovotransferrin against Escherichia coli O157:H7. Ames (Iowa): Iowa State University, January 2010. http://dx.doi.org/10.31274/ans_air-180814-1042.

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Clothier, Kris. An Investigation in to the Fecal Shedding of E. Coli O157:H7 from Steers on Rations Containing Corn Co-Products. Ames (Iowa): Iowa State University, January 2009. http://dx.doi.org/10.31274/ans_air-180814-876.

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Kudra, Li, Joseph G. Sebranek, James S. Dickson, Aubrey F. Mendonca, Armitra Jackson-Davis, Qijing Zhang, Kenneth J. Prusa, and Zheng Lu. Controlling Listeria monocytogenes, Campylobactor jejuni, Salmonella enterica Typhimurium and Escherichia coli O157:H7 in Meat Products by Irradiation Combined with Modified Atmosphere Packaging. Ames (Iowa): Iowa State University, January 2013. http://dx.doi.org/10.31274/ans_air-180814-13.

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Ko, Kyung Yuk, Aubrey F. Mendonca, and Dong U. Ahn. Influence of Zn2 + , Sodium Bicarbonate, and Citric Acid on the Antibacterial Activity of Ovotransferrin against E. coli O157:H7 and L. monocytogenes in Model Systems and Ham. Ames (Iowa): Iowa State University, January 2010. http://dx.doi.org/10.31274/ans_air-180814-1020.

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