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1

Kay, Miller Ellen. Escherichia coli O157. National Agricultural Library, 1993.

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2

Kay, Miller Ellen. Escherichia coli O157. National Agricultural Library, 1992.

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3

United States. Animal and Plant Health Inspection Service. Veterinary Services. Centers for Epidemiology and Animal Health., ed. Escherichia coli O157:H7: Issues and ramifications. USDA:APHIS:VS Centers for Epidemiology and Animal Health, 1994.

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4

Kay, Miller Ellen. Escherichia coli O157: January 1994 - July 1995. National Agricultural Library, 1995.

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5

United States. Animal and Plant Health Inspection Service. and National Animal Health Monitoring System (U.S.), eds. Escherichia coli O157 in United States feedlots. U.S. Dept. of Agriculture, Animal and Plant Health Inspection Service, 2001.

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6

Miller, Ellen Kay. Escherichia coli O157: January 1993 - December 1993. National Agricultural Library, 1994.

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7

United States. Animal and Plant Health Inspection Service. Veterinary Services. Centers for Epidemiology and Animal Health. Escherichia coli O157:H7: Issues and ramifications : executive summary. USDA:APHIS:VS, Centers for Epidemiology and Animal Health, 1994.

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8

R, Palmer Stephen, ed. E.coli: Environmental health issues of VTEC O157. Spon Press, 2002.

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9

Urabi, Iftikhar. Virulence factors of verotoxin-producing Escherichia coli O157:H7. typescript, 1993.

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10

Heersink, Mary. E.coli O157: The true story of a mother's battle with a killer microbe. New Horizon Press, 1996.

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11

United States. Animal and Plant Health Inspection Service. Veterinary Services. Centers for Epidemiology and Animal Health., ed. An update, Escherichia coli O157:H7 in humans and cattle. USDA:APHIS:VS Centers for Epidemiology and Animal Health, 1997.

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12

Bukhari, Zia. Improved detection methods for E. coli 0157: H7. Awwa Research Foundation, 2005.

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13

Kerr, Marie. The survival of Escherichia coli O157:H7 in natural mineral water. The Author], 2001.

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14

Byrne, Caitriona Martina. Survival and persistence of Escherichia coli O157:H7 within meat production processes. The Author], 2001.

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15

Workshop on Methods to Isolate Escherichia Coli O157:H7 and Other Verotoxigenic E. Coli from Foods (1991 Ottawa, Ont.). Escherichia coli O157:H7 and other verotoxigenic E. coli in foods: Proceedings of a Workshop on Methods to Isolate Escherichia Coli O157:H7 and Other Verotoxigenic E. Coli from Foods, held on March 18-19, 1991 in Ottawa, Canada. Edited by Todd, E. C. D. 1939-, MacKenzie J. M, Canada Food Directorate, and Canadian Meat Council. Polyscience Publications, 1993.

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16

Assessment, Institute of Medicine (U S. ). Committee on the Review of the USDA E. coli O157:H7 Farm-to-Table Process Risk. Escherichia coli O157:H7 in ground beef: Review of a draft risk assessment. National Academies Press, 2002.

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17

Riordan, Denise Catherine Rose. A study on the survival of Escherichia coli O157:H7 in fermented meat. The Author], 1998.

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18

Atelier sur les méthodes permettant d'isoler Escherichia Coli O157 (H7 et d'autres E. Coli vérotoxigènes dans les aliments (1991 Ottawa, Ont.). Escherichia coli O157:H7 et d'autres E. coli vérotoxigènes dans les aliments : les actes de l'Atelier sur les méthodes permettant d'isoler Escherichia coli O157:H7 et d'autres E. coli vérotoxigènes dans les aliments, le 18-19 mars 1991 à Ottawa, Canada. Publications Polyscience, 1993.

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19

United States. Food Safety and Inspection Service. Draft risk assessment of the public health impact of Escherichia coli O157:H7 in ground beef. FSIS, 2001.

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20

Institute of Medicine (U.S.). Committee on the Review of the USDA E coli O157:H7 Farm-to-Table Process Risk Assessment. and Institute of Medicine (U.S.). Board on Health Promotion and Disease Prevention., eds. Review of the USDA draft risk assessment of the public health impact of escherichia coli o157:H7 in ground beef. National Academies Press, 2003.

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21

National Research Council (U.S.). Board on Assessment of NIST Programs., ed. An assessment of the National Institute of Standards and Technology measurement and standards laboratories: Fiscal years 2004-2005. National Academies Press, 2005.

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22

United States. Congress. Senate. Committee on Appropriations. Subcommittee on Agriculture, Rural Development, and Related Agencies. Food safety recall procedures: Hearing before a subcommittee of the Committee on Appropriations, United States Senate, One Hundred Seventh Congress, second session, special hearing, December 11, 2002, Billings, Montana. U.S. G.P.O., 2003.

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23

Pennington, T. H. When food kills: BSE, E. coli, and disaster science. Oxford University Press, 2003.

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24

(Editor), C. S. Stewart, and H. J. Flint (Editor), eds. Escherichia Coli O157 in Farm Animals. CABI, 2000.

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25

Lamden, Ken, Sam Rowell, and Andrew Fox. Vero cytotoxigenic Escherichia coli (VTEC) O157. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198745471.003.0005.

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This chapter describes a cluster of diarrhoeal disease caused by E.coli O157 in a case scenario involving infection following a farm visit, and possible subsequent spread within a nursery. Background information on the epidemiology of E.coli O157 indicates the role of farm contact in infection, and the risk factors for transmission. In addition to providing clear case definitions, the risk groups of contacts are described in detail. ‘Top tips’ are given, to assist the health protection practitioner take timely and appropriate action, and ‘tools of the trade’ list the laboratory, epidemiological, and veterinary components of the investigation. The statutory safety legislation required at the source farm is explained, and the importance of communication with the affected families is emphasized. The final part of the chapter considers other possible scenarios, including the possibility of food from the farm café being the source.
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26

Escherichia coli O157:H7 in Ground Beef. National Academies Press, 2002. http://dx.doi.org/10.17226/10528.

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27

Kay, Miller Ellen. Escherichia Coli O157 January 93- December 93. Diane Books Publishing Company, 2004.

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28

Parry, Sharon, and S. Palmer. E. Coli: Environmental Health Issues of Vtec O157. Taylor & Francis Group, 2001.

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29

Duffy, Geraldine, Patricia Garvey, and David A. McDowell. Verocytotoxigenic E. Coli. Wiley & Sons, Incorporated, John, 2008.

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30

Escherichia coli O157:H7 and other shiga toxin-producing E. coli strains. ASM Press, 1998.

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31

Enterohemorrhagic Escherichia coli and Other Shiga Toxin-Producing E. coli. ASM Press, 2015.

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32

Hayhurst, Chris. E. Coli (Epidemics). Rosen Publishing Group, 2003.

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33

Cooper, Ian Richard. The role of freshwater biofilms as reservoirs of escherichia coli O157. 2004.

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34

Parry, Sharon, and Stephen Palmer. E. coli: Environmental Health Issues of VTEC 0157. CRC Press LLC, 2002.

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35

Parry, Sharon, and Stephen Palmer. E. coli: Environmental Health Issues of VTEC 0157. CRC Press LLC, 2002.

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36

Parry, Sharon, and Stephen Palmer. E. coli: Environmental Health Issues of VTEC 0157. CRC Press LLC, 2002.

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37

Parry, Sharon, and Stephen Palmer. E. coli: Environmental Health Issues of VTEC 0157. CRC Press LLC, 2002.

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38

Karmali, Mohamed A., and Jan M. Sargeant. Verocytotoxin-producing Escherichia coli (VTEC) infections. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0008.

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Verocytotoxin (VT)-producing Escherichia coli (VTEC), also known as Shiga toxin producing E. coli (STEC), are zoonotic agents, which cause a potentially fatal illness whose clinical spectrum includes diarrhoea, haemorrhagic colitis, and the haemolytic uraemic syndrome (HUS). VTEC are of serious public health concern because of their association with large outbreaks and with HUS, which is the leading cause of acute renal failure in children. Although over 200 different OH serotypes of VTEC have been associated with human illness, the vast majority of reported outbreaks and sporadic cases of VTEC-infection in humans have been associated with serotype O157:H7.VTs constitute a family of related protein subunit exotoxins, the major ones implicated in human disease being VT1, VT2, and VT2c. Following their translocation into the circulation, VTs bind to endothelial cells of the renal glomeruli, and of other organs and tissues via a specific receptor globotriosylceramide (Gb 3), are internalized by a process of receptor-mediated endocytosis, and cause subcellular damage that results in the characteristic microangiopathic disease observed in HUS.The incubation period of VTEC-associated illness is about 3–5 days. After ingestion VTEC (especially of serotype O157:H7) multiply in the bowel and colonize the mucosa of probably the large bowel with a characteristic attaching and effacing (AE) cytopathology. Colonization is followed by the translocation of VTs into the circulation and the subsequent manifestation of disease.The majority of patients with uncomplicated VTEC infection recover fully with general supportive measures. Historically, the case-fatality rate was high for HUS. However, improvement in the treatment of renal failure and the attendant biochemical disturbances has substantially improved the outlook, although long-term sequelae may develop.Ruminants, especially cattle, are the main reservoirs of VTEC. Infection is acquired through the ingestion of contaminated food, especially under-cooked hamburger, through direct contact with animals, via contaminated water or environments, or via personto-person transmission.The occurrence of large outbreaks of food-borne VTEC-associated illness has promoted close scrutiny of this zoonoses at all levels in the chain of transmission, including the farm, abattoir, food processing, packaging and distribution plants, the wholesaler, the retailer and the consumer. While eradication of VTEC O157 at the farm may not be an option, interventions to increase animal resistance or to decrease animal exposure are being developed and validated. Hazard Analysis and Critical Control Programmes are being implemented in the processing sector and appear to be associated with temporal decreases in VTEC serotype O157 illness in humans. Education programmes targeting food handling procedures and hygiene practices are being advocated at the retail and consumer level. Continued efforts at all stages from the farm to the consumer will be necessary to reduce the risk of VTEC-associated illness in humans.
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39

Food Poisoning E Coli And The Food Supply. Rosen Publishing Group, 2011.

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40

Escherichia coli 0157:H7 and other Vero cytotoxin producing strains: Report to the Minister for Health and the Minister for Agriculture, Food and Forestry. Stationery Office, 1994.

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41

Escherichia coli O157:H7 in Ground Beef: Review of a Draft Risk Assessment. National Academies Press, 2002.

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42

Board, Food and Nutrition, Institute of Medicine, Board on Health Promotion and Disease Prevention, Committee on the Review of the USDA E. coli O157:H7, and Farm-to-Table Process Risk Assessment. Escherichia Coli O157 : H7 in Ground Beef: Review of a Draft Risk Assessment. National Academies Press, 2002.

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43

Committee on the Review of the USDA E. coli O157:H7 Farm-to-Table Process Risk Assessment, Food and Nutrition Board, Institute of Medicine, and Board on Health Promotion and Disease Prevention. Escherichia Coli O157 : H7 in Ground Beef: Review of a Draft Risk Assessment. National Academies Press, 2002.

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44

Committee on the Review of the USDA E. coli O157:H7 Farm-to-Table Process Risk Assessment, Food and Nutrition Board, Institute of Medicine, and Board on Health Promotion and Disease Prevention. Escherichia Coli O157 : H7 in Ground Beef: Review of a Draft Risk Assessment. National Academies Press, 2002.

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45

Deisingh, Anil Kenneth. Detection of Escherichia coli O157: H7 and related species by acoustic wave sensor. 2002.

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46

Kru⁺ger, William. Molekulargenetische und pha⁺notypische Untersuchungen an Plasmiden enteroha⁺morrhagischer E. coli O157: H7. 1988.

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47

Oakley, Jeremy E., and Helen E. Clough. Sensitivity analysis in microbial risk assessment: Vero-cytotoxigenic E. coli O157 in farm-pasteurized milk. Edited by Anthony O'Hagan and Mike West. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780198703174.013.4.

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This article discusses the use of Bayesian methods for performing uncertainty analysis in complex computer models, focusing on a mechanistic model that has been applied in a risk assessment of contamination of farm-pasteurized milk with the bacterium Vero-cytotoxigenic E. coli (VTEC) O157. The VTEC model has uncertain input parameters, which makes outputs from the model used to inform the risk assessment also uncertain. The question that arises is how to reduce output uncertainty in the most efficient manner possible. The article first provides an overview of microbial risk assessment before analysing the frequency and consequences of food-borne outbreaks associated with VTEC O157. It then introduces the risk assessment model, along with model input distributions. Finally, it presents the results of a variance-based sensitivity analysis that was conducted to identify the most important uncertain model inputs.
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48

Sanderson, Michael William. Sensitivity of bacteriologic culture for detection of Escherichia coli O157:H7 in bovine feces, and effects on apparent prevalence in cattle. 1995.

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49

Characterization of the interaction of Escherichia coli O157:H7 intimin with a human colonic epithelial cell line (HCT-8). National Library of Canada, 1995.

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50

US GOVERNMENT. Food Safety Recall Procedures: Hearing Before a Subcommittee of the Committee on Appropriations, United States Senate, One Hundred Seventh Congress,. Government Printing Office, 2003.

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