Relevant bibliographies by topics / Early steps of retroviral replication cycle

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  2. Dissertations / Theses
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Academic literature on the topic 'Early steps of retroviral replication cycle'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Early steps of retroviral replication cycle"

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Masson, Christel, Stéphanie Bury-Moné, Elvire Guiot, et al. "Ku80 Participates in the Targeting of Retroviral Transgenes to the Chromatin of CHO Cells." Journal of Virology 81, no. 15 (2007): 7924–32. http://dx.doi.org/10.1128/jvi.02015-06.

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ABSTRACT The heterodimer Ku70/80 Ku is the DNA-binding component of the DNA-PK complex required for the nonhomologous end-joining pathway. It participates in numerous nuclear processes, including telomere and chromatin structure maintenance, replication, and transcription. Ku interacts with retroviral preintegration complexes and is thought to interfere with the retroviral replication cycle, in particular the formation of 2-long terminal repeat (LTR) viral DNA circles, viral DNA integration, and transcription. We describe here the effect of Ku80 on both provirus integration and the resulting t
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Morcock, David R., James A. Thomas, Tracy D. Gagliardi, et al. "Elimination of Retroviral Infectivity by N-Ethylmaleimide with Preservation of Functional Envelope Glycoproteins." Journal of Virology 79, no. 3 (2005): 1533–42. http://dx.doi.org/10.1128/jvi.79.3.1533-1542.2005.

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ABSTRACT The zinc finger motifs in retroviral nucleocapsid (NC) proteins are essential for viral replication. Disruption of these Cys-X2-Cys-X4-His-X4-Cys zinc-binding structures eliminates infectivity. To determine if N-ethylmaleimide (NEM) can inactivate human immunodeficiency virus type 1 (HIV-1) or simian immunodeficiency virus (SIV) preparations by alkylating cysteines of NC zinc fingers, we treated infectious virus with NEM and evaluated inactivation of infectivity in cell-based assays. Inactivation was rapid and proportional to the NEM concentration. NEM treatment of HIV-1 or SIV result
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Kim, Steve S., Xue Juan You, Mary-Elizabeth Harmon, Julie Overbaugh, and Hung Fan. "Use of Helper-Free Replication-Defective Simian Immunodeficiency Virus-Based Vectors To Study Macrophage and T Tropism: Evidence for Distinct Levels of Restriction in Primary Macrophages and a T-Cell Line." Journal of Virology 75, no. 5 (2001): 2288–300. http://dx.doi.org/10.1128/jvi.75.5.2288-2300.2001.

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ABSTRACT Cell tropism of human and simian immunodeficiency viruses (HIV and SIV, respectively) is governed in part by interactions between the viral envelope protein and the cellular receptors. However, there is evidence that envelope-host cell interactions also affect postentry steps in viral replication. We used a helper-free replication-defective SIV macaque (SIVmac)-based retroviral vector carrying the enhanced jellyfish green fluorescent protein inserted into thenef region (V1EGFP) to examine SIV tropism in a single cycle of infection. Vector stocks containing envelope proteins from three
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Zhu, Kai, Charles Dobard, and Samson A. Chow. "Requirement for Integrase during Reverse Transcription of Human Immunodeficiency Virus Type 1 and the Effect of Cysteine Mutations of Integrase on Its Interactions with Reverse Transcriptase." Journal of Virology 78, no. 10 (2004): 5045–55. http://dx.doi.org/10.1128/jvi.78.10.5045-5055.2004.

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ABSTRACT Retroviral integrase catalyzes the essential step of integrating a double-stranded DNA copy of the viral genome into a host cell chromosome. Mutational studies have revealed that integrase is involved in additional steps of viral replication, but the mechanism for the pleiotropic effect is not well characterized. Since Cys residues generally play crucial roles in protein structure and function, we introduced Cys-to-Ser substitutions at positions 56, 65, and 130 of human immunodeficiency virus type 1 (HIV-1) integrase to determine their effects on integration activity and viral replica
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Griffith, Jacqulyn L., Laura E. Coleman, Adam S. Raymond, et al. "Functional Genomics Reveals Relationships Between the Retrovirus-Like Ty1 Element and Its Host Saccharomyces cerevisiae." Genetics 164, no. 3 (2003): 867–79. http://dx.doi.org/10.1093/genetics/164.3.867.

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Abstract Retroviruses and their relatives, the long terminal repeat (LTR) retrotransposons, carry out complex life cycles within the cells of their hosts. We have exploited a collection of gene deletion mutants developed by the Saccharomyces Genome Deletion Project to perform a functional genomics screen for host factors that influence the retrovirus-like Ty1 element in yeast. A total of 101 genes that presumably influence many different aspects of the Ty1 retrotransposition cycle were identified from our analysis of 4483 homozygous diploid deletion strains. Of the 101 identified mutants, 46 h
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Heusinger, Elena, Silvia F. Kluge, Frank Kirchhoff, and Daniel Sauter. "Early Vertebrate Evolution of the Host Restriction Factor Tetherin." Journal of Virology 89, no. 23 (2015): 12154–65. http://dx.doi.org/10.1128/jvi.02149-15.

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ABSTRACTTetherin is an interferon-inducible restriction factor targeting a broad range of enveloped viruses. Its antiviral activity depends on an unusual topology comprising an N-terminal transmembrane domain (TMD) followed by an extracellular coiled-coil region and a C-terminal glycosylphosphatidylinositol (GPI) anchor. One of the two membrane anchors is inserted into assembling virions, while the other remains in the plasma membrane of the infected cell. Thus, tetherin entraps budding viruses by physically bridging viral and cellular membranes. Although tetherin restricts the release of a la
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Wainberg, Mark A., Andre Dascal, and Jack Mendelson. "Anti-Retroviral Strategies for AIDS and Related Diseases." Canadian Journal of Infectious Diseases 2, no. 3 (1991): 121–28. http://dx.doi.org/10.1155/1991/487657.

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The replication cycle of human immunodeficiency virus type 1 (HIV-1) and other retroviruses consists of four stages: attachment of the virus to specific receptors on the cell surface; uncoating of the viral nucleic acid and conversion to DNA; production of viral RNA and proteins; and assembly and liberation of progeny virus from the cell. Each of these steps represents a potential target for antiviral chemotherapy. Combinations of drugs which act against different steps in the viral replication cycle might be expected to have synergistic potential. Zidovudine (AZT) is the most widely used drug
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Katz, Richard A., James G. Greger, and Anna Marie Skalka. "Effects of cell cycle status on early events in retroviral replication." Journal of Cellular Biochemistry 94, no. 5 (2005): 880–89. http://dx.doi.org/10.1002/jcb.20358.

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Sabahi, Ali. "Hepatitis C Virus entry: the early steps in the viral replication cycle." Virology Journal 6, no. 1 (2009): 117. http://dx.doi.org/10.1186/1743-422x-6-117.

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Lehmann-Che, Jacqueline, Marie-Lou Giron, Olivier Delelis, et al. "Protease-Dependent Uncoating of a Complex Retrovirus." Journal of Virology 79, no. 14 (2005): 9244–53. http://dx.doi.org/10.1128/jvi.79.14.9244-9253.2005.

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ABSTRACT Although retrovirus egress and budding have been partly unraveled, little is known about early stages of the replication cycle. In particular, retroviral uncoating, a process during which incoming retroviral cores are altered to allow the integration of the viral genome into host chromosomes, is poorly understood. To get insights into these early events of the retroviral cycle, we have used foamy complex retroviruses as a model. In this report, we show that a protease-defective foamy retrovirus is noninfectious, although it is still able to bud and enter target cells efficiently. Simi
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Dissertations / Theses on the topic "Early steps of retroviral replication cycle"

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Inacio, Mamede Joao Filipe. "Interactions de la capside de lentivirus de primates avec les facteurs cellulaires de l’hôte." Thesis, Montpellier 1, 2012. http://www.theses.fr/2012MON13524/document.

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Depuis la découverte du virus de l'Immunodéficience humaine, un lentivirus, comme agent pathogène responsable de l'épidémie du SIDA en 1983, beaucoup de progrès sur le sujet ont été réalisés. Il existe deux types de virus différents pouvant infecter l'Homme, le HIV-1 et le HIV-2. Ces deux virus se regroupent en différents groupes et sous-types qui témoignent d'une grande diversité inter et intra individus (notions de quasi-espèces). La découverte de lentivirus infectant naturellement au moins quarante-cinq espèces de primates en Afrique sub-saharienne, a permis un enrichissement des connaissan
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Books on the topic "Early steps of retroviral replication cycle"

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Young, John A. T., ed. Cellular Factors Involved in Early Steps of Retroviral Replication. Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-19012-4.

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T, Young J. A., ed. Cellular factors involved in early steps of retroviral replication. Springer, 2003.

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Young, John A. T. Cellular Factors Involved in Early Steps of Retroviral Replication. Current Topics in Microbiology and Immunology, No. 281. Springer, 2003.

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Book chapters on the topic "Early steps of retroviral replication cycle"

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"Genes." In Examining the Causal Relationship Between Genes, Epigenetics, and Human Health. IGI Global, 2019. http://dx.doi.org/10.4018/978-1-5225-8066-9.ch007.

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Two types of nucleic acids, DNA and RNA, carry genetic information of organisms across generations. Many researchers are credited with the early work that laid the foundation of the discovery of the structure of DNA. During cell division, the cell replicates its DNA and organelles during the synthesis (S) phase of the cell cycle. Four main steps are involved in the processes of replication. DNA replication errors and cells have evolved a complex system of fixing most (but not all) of those replication errors proofreading and mismatch repair. With repeated cell division, the DNA molecule shortens with the loss of critical genes, leading to cell death. In gonads, a special enzyme called telomerase lengthens telomeres from its own RNA sequence which serves as a template to synthesize new telomeres. Although most DNA is packaged within the nucleus, mitochondria have a small amount of their own DNA called mitochondrial DNA. This chapter explores this aspect of genes.
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Journal articles
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