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Relevant bibliographies by topics / EBGP underlay

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Journal articles

Academic literature on the topic 'EBGP underlay'

Author: Grafiati

Published: 7 June 2025

Last updated: 2 August 2025

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Journal articles on the topic "EBGP underlay"

1

Dr.A.Shaji, George. "The Evolution of Data Center Networks: Strategies for Modern Infrastructure Design." Partners Universal Multidisciplinary Research Journal (PUMRJ) 02, no. 03 (2025): 141–59. https://doi.org/10.5281/zenodo.15450624.

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Data center networks have undergone dramatic transformation in response to shifting technological landscapes and business requirements. This article examines how traditional hierarchical network designs have given way to more efficient, scalable architectures as organizations adapt to cloud computing paradigms, AI workloads, and distributed systems. The analysis covers the foundational elements of modern data center networks, from physical topology selection to advanced fabric designs, security segmentation methodologies, and automation frameworks. By closely looking at spine-leaf architecture

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2

Barnsteiner, Jane, Scott Beebe, Rachel Dicker, et al. "Facilitating a Culture of Evidence-Based Practice and Quality Improvement Excellence." Journal for Nurses in Professional Development 39, no. 4 (2023): 207–13. http://dx.doi.org/10.1097/nnd.0000000000000995.

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This article defines evidence-based practice (EBP) and quality improvement (QI) and examines the positive impact on patient care when they are in place, as well as the barriers to their implementation. An effective tool, Ovid Synthesis, was created to enable clinicians and administrators to streamline the processes for EBP and QI, provide oversight on the initiatives underway, and enable clinical educators to help nursing staff develop the necessary competencies and successfully execute their EBP and/or QI projects.

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3

Mallidou, Anastasia A., Elizabeth Borycki, Noreen Frisch, and Lynne Young. "Research Competencies Assessment Instrument for Nurses: Preliminary Psychometric Properties." Journal of Nursing Measurement 26, no. 3 (2018): E159—E182. http://dx.doi.org/10.1891/1061-3749.26.3.e159.

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Background and Purpose: Clinician research competencies influence research use for evidence-based practice (EBP). We aimed to develop, refine, and psychometrically assess the Research Competencies Assessment Instrument for Nurses (RCAIN) to measure registered nurse research competencies (i.e., knowledge, skills, attitudes) focused on EBP-related domains: research process, knowledge synthesis, and knowledge translation activities. Methods: The preliminary psychometrics (face, content, construct/criterion validity) were evaluated based on 63 completed surveys. Results: The Cronbach’s α coefficie

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4

Salma, N., H. Xiao, and A. N. Imbalzano. "Temporal recruitment of CCAAT/enhancer-binding proteins to early and late adipogenic promoters in vivo." Journal of Molecular Endocrinology 36, no. 1 (2006): 139–51. http://dx.doi.org/10.1677/jme.1.01918.

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The CCAAT/enhancer-binding protein (C/EBP) family of transcriptional regulators is critically important for the activation of adipogenic genes during differentiation. The C/EBPβ and δ isoforms are rapidly induced upon adipocyte differentiation and are responsible for activating the adipogenic regulators C/EBPα and peroxisome proliferator activated receptor (PPAR)γ2, which together activate the majority of genes expressed in differentiating adipocytes. However, mitosis is required following the induction of adipogenesis, and the activation of C/EBPα and PPARγ2 gene expression is delayed until c

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Fernandes, Adelaide, Cláudia Caldeira, Carolina Cunha, Elisabete Ferreiro, Ana Rita Vaz, and Dora Brites. "Differences in Immune-Related Genes Underlie Temporal and Regional Pathological Progression in 3xTg-AD Mice." Cells 11, no. 1 (2022): 137. http://dx.doi.org/10.3390/cells11010137.

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The prevalence of Alzheimer’s disease (AD), the most common cause of age-associated dementia, is estimated to increase over the next decades. Evidence suggests neuro-immune signaling deregulation and risk genes beyond the amyloid-β (Aβ) deposition in AD pathology. We examined the temporal profile of inflammatory mediators and microglia deactivation/activation in the brain cortex and hippocampus of 3xTg-AD mice at 3- and 9-month-old. We found upregulated APP processing, decreased expression of CD11b, CX3CR1, MFG-E8, TNF-α, IL-1β, MHC-II and C/EBP-α and increased miR-146a in both brain regions i

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6

Zhang, Na, Mark McNicholas, Bob Anderhalt, Evan Slow, and Neil Colvin. "Microstructure of Pvd Al Deposition on CVD Tin." Microscopy and Microanalysis 3, S2 (1997): 493–94. http://dx.doi.org/10.1017/s1431927600009351.

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CVD TiN films offer promise as a barrier to Al deposition as a result of the improved conformai step coverage of this film in 0.25 μm contact holes. As an underlayer, the TiN performs a secondary role by improving electromigration (EM) resistance. This is a result of the crystal orientation of the TiN film and its influence on the orientation of the subsequent Al layer. A <111>Al orientation shows improved EM resistance; however, CVD TiN has a preferred <200> orientation as opposed to a <111>PVD TiN orientation.In this study, two parts were investigated: 1) obtain a qualified

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7

Grimes, H. Leighton, Chinavenmeni S. Velu, Shane Horman, et al. "Gfi1 as a Regulator of Hematopoietic Differentiation." Blood 112, no. 11 (2008): sci—16—sci—16. http://dx.doi.org/10.1182/blood.v112.11.sci-16.sci-16.

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Abstract Growth Factor Independent-1 (Gfi1) is a transcriptional repressor originally identified as a target of Moloney leukemia virus insertion mutagenesis. Gfi1 potently collaborates with Myc and Pim oncogenes to mediate T cell transformation. Retroviral insertions which activate Gfi1 are among the most frequent events in MoMLV-induced T cell, but not myeloid, leukemias. Orthologous proteins in Drosophila (Senseless) and C.elegans (Pag-3) are key regulators of developmental decisions. Gfi1−/− mice display lymphopenia, neutropenia, and abnormally proliferative HSC and progenitors which eventu

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8

Kuang, Ping-Ping, and Ronald H. Goldstein. "Regulation of elastin gene transcription by proteasome dysfunction." American Journal of Physiology-Cell Physiology 289, no. 3 (2005): C766—C773. http://dx.doi.org/10.1152/ajpcell.00525.2004.

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Elastin, a major extracellular matrix protein and the core component of elastic fiber, is essential to maintain lung structural integrity and normal physiological function. We previously found that the downregulation of elastin gene transcription by IL-1β is mediated via activation of NF-κB and CCAAT/enhancer binding protein (C/EBP)β, both targets of the ubiquitin-proteasome pathway. To further investigate the molecular mechanisms that underlie the control of elastin gene expression, we disrupted the ubiquitin-proteasome pathway with specific proteasome inhibitors. We found that specific prote

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Hiroki, Tomoko, Stephen A. Liebhaber, and Nancy E. Cooke. "An Intronic Locus Control Region Plays an Essential Role in the Establishment of an Autonomous Hepatic Chromatin Domain for the Human Vitamin D-Binding Protein Gene." Molecular and Cellular Biology 27, no. 21 (2007): 7365–80. http://dx.doi.org/10.1128/mcb.00331-07.

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ABSTRACT The human vitamin D-binding protein (hDBP) gene exists in a cluster of four liver-expressed genes. A minimal hDBP transgene, containing a defined set of liver-specific DNase I hypersensitive sites (HSs), is robustly expressed in mouse liver in a copy-number-dependent manner. Here we evaluate these HSs for function. Deletion of HSI, located 5′ to the promoter (kb −2.1) had no significant effect on hDBP expression. In contrast, deletion of HSIV and HSV from intron 1 repressed hDBP expression and eliminated copy number dependency without a loss of liver specificity. Chromatin immunopreci

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10

Diakogiannaki, Eleftheria, Hannah J. Welters та Noel G. Morgan. "Differential regulation of the endoplasmic reticulum stress response in pancreatic β-cells exposed to long-chain saturated and monounsaturated fatty acids". Journal of Endocrinology 197, № 3 (2008): 553–63. http://dx.doi.org/10.1677/joe-08-0041.

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Exposure of pancreatic β-cells to long-chain fatty acids leads to the activation of some components of the endoplasmic reticulum (ER) stress pathway and this mechanism may underlie the ability of certain fatty acids to promote β-cell death. We have studied ER stress in BRIN-BD11 β-cells exposed to either the saturated fatty acid palmitate (C16:0) or the monounsaturated palmitoleate (C16:1). Palmitate (0.025–0.25 mM) induced the expression of various markers of the RNA-dependent protein kinase-like ER eukaryotic initiation factor 2α (eIF2α) kinase (PERK)-dependent pathway of ER stress (phospho-

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