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Journal articles on the topic 'EBGP underlay'

Author: Grafiati

Published: 7 June 2025

Last updated: 2 August 2025

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1

Dr.A.Shaji, George. "The Evolution of Data Center Networks: Strategies for Modern Infrastructure Design." Partners Universal Multidisciplinary Research Journal (PUMRJ) 02, no. 03 (2025): 141–59. https://doi.org/10.5281/zenodo.15450624.

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Data center networks have undergone dramatic transformation in response to shifting technological landscapes and business requirements. This article examines how traditional hierarchical network designs have given way to more efficient, scalable architectures as organizations adapt to cloud computing paradigms, AI workloads, and distributed systems. The analysis covers the foundational elements of modern data center networks, from physical topology selection to advanced fabric designs, security segmentation methodologies, and automation frameworks. By closely looking at spine-leaf architecture

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2

Barnsteiner, Jane, Scott Beebe, Rachel Dicker, et al. "Facilitating a Culture of Evidence-Based Practice and Quality Improvement Excellence." Journal for Nurses in Professional Development 39, no. 4 (2023): 207–13. http://dx.doi.org/10.1097/nnd.0000000000000995.

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This article defines evidence-based practice (EBP) and quality improvement (QI) and examines the positive impact on patient care when they are in place, as well as the barriers to their implementation. An effective tool, Ovid Synthesis, was created to enable clinicians and administrators to streamline the processes for EBP and QI, provide oversight on the initiatives underway, and enable clinical educators to help nursing staff develop the necessary competencies and successfully execute their EBP and/or QI projects.

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3

Mallidou, Anastasia A., Elizabeth Borycki, Noreen Frisch, and Lynne Young. "Research Competencies Assessment Instrument for Nurses: Preliminary Psychometric Properties." Journal of Nursing Measurement 26, no. 3 (2018): E159—E182. http://dx.doi.org/10.1891/1061-3749.26.3.e159.

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Background and Purpose: Clinician research competencies influence research use for evidence-based practice (EBP). We aimed to develop, refine, and psychometrically assess the Research Competencies Assessment Instrument for Nurses (RCAIN) to measure registered nurse research competencies (i.e., knowledge, skills, attitudes) focused on EBP-related domains: research process, knowledge synthesis, and knowledge translation activities. Methods: The preliminary psychometrics (face, content, construct/criterion validity) were evaluated based on 63 completed surveys. Results: The Cronbach’s α coefficie

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4

Salma, N., H. Xiao, and A. N. Imbalzano. "Temporal recruitment of CCAAT/enhancer-binding proteins to early and late adipogenic promoters in vivo." Journal of Molecular Endocrinology 36, no. 1 (2006): 139–51. http://dx.doi.org/10.1677/jme.1.01918.

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The CCAAT/enhancer-binding protein (C/EBP) family of transcriptional regulators is critically important for the activation of adipogenic genes during differentiation. The C/EBPβ and δ isoforms are rapidly induced upon adipocyte differentiation and are responsible for activating the adipogenic regulators C/EBPα and peroxisome proliferator activated receptor (PPAR)γ2, which together activate the majority of genes expressed in differentiating adipocytes. However, mitosis is required following the induction of adipogenesis, and the activation of C/EBPα and PPARγ2 gene expression is delayed until c

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Fernandes, Adelaide, Cláudia Caldeira, Carolina Cunha, Elisabete Ferreiro, Ana Rita Vaz, and Dora Brites. "Differences in Immune-Related Genes Underlie Temporal and Regional Pathological Progression in 3xTg-AD Mice." Cells 11, no. 1 (2022): 137. http://dx.doi.org/10.3390/cells11010137.

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The prevalence of Alzheimer’s disease (AD), the most common cause of age-associated dementia, is estimated to increase over the next decades. Evidence suggests neuro-immune signaling deregulation and risk genes beyond the amyloid-β (Aβ) deposition in AD pathology. We examined the temporal profile of inflammatory mediators and microglia deactivation/activation in the brain cortex and hippocampus of 3xTg-AD mice at 3- and 9-month-old. We found upregulated APP processing, decreased expression of CD11b, CX3CR1, MFG-E8, TNF-α, IL-1β, MHC-II and C/EBP-α and increased miR-146a in both brain regions i

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6

Zhang, Na, Mark McNicholas, Bob Anderhalt, Evan Slow, and Neil Colvin. "Microstructure of Pvd Al Deposition on CVD Tin." Microscopy and Microanalysis 3, S2 (1997): 493–94. http://dx.doi.org/10.1017/s1431927600009351.

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CVD TiN films offer promise as a barrier to Al deposition as a result of the improved conformai step coverage of this film in 0.25 μm contact holes. As an underlayer, the TiN performs a secondary role by improving electromigration (EM) resistance. This is a result of the crystal orientation of the TiN film and its influence on the orientation of the subsequent Al layer. A <111>Al orientation shows improved EM resistance; however, CVD TiN has a preferred <200> orientation as opposed to a <111>PVD TiN orientation.In this study, two parts were investigated: 1) obtain a qualified

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7

Grimes, H. Leighton, Chinavenmeni S. Velu, Shane Horman, et al. "Gfi1 as a Regulator of Hematopoietic Differentiation." Blood 112, no. 11 (2008): sci—16—sci—16. http://dx.doi.org/10.1182/blood.v112.11.sci-16.sci-16.

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Abstract Growth Factor Independent-1 (Gfi1) is a transcriptional repressor originally identified as a target of Moloney leukemia virus insertion mutagenesis. Gfi1 potently collaborates with Myc and Pim oncogenes to mediate T cell transformation. Retroviral insertions which activate Gfi1 are among the most frequent events in MoMLV-induced T cell, but not myeloid, leukemias. Orthologous proteins in Drosophila (Senseless) and C.elegans (Pag-3) are key regulators of developmental decisions. Gfi1−/− mice display lymphopenia, neutropenia, and abnormally proliferative HSC and progenitors which eventu

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8

Kuang, Ping-Ping, and Ronald H. Goldstein. "Regulation of elastin gene transcription by proteasome dysfunction." American Journal of Physiology-Cell Physiology 289, no. 3 (2005): C766—C773. http://dx.doi.org/10.1152/ajpcell.00525.2004.

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Elastin, a major extracellular matrix protein and the core component of elastic fiber, is essential to maintain lung structural integrity and normal physiological function. We previously found that the downregulation of elastin gene transcription by IL-1β is mediated via activation of NF-κB and CCAAT/enhancer binding protein (C/EBP)β, both targets of the ubiquitin-proteasome pathway. To further investigate the molecular mechanisms that underlie the control of elastin gene expression, we disrupted the ubiquitin-proteasome pathway with specific proteasome inhibitors. We found that specific prote

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9

Hiroki, Tomoko, Stephen A. Liebhaber, and Nancy E. Cooke. "An Intronic Locus Control Region Plays an Essential Role in the Establishment of an Autonomous Hepatic Chromatin Domain for the Human Vitamin D-Binding Protein Gene." Molecular and Cellular Biology 27, no. 21 (2007): 7365–80. http://dx.doi.org/10.1128/mcb.00331-07.

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ABSTRACT The human vitamin D-binding protein (hDBP) gene exists in a cluster of four liver-expressed genes. A minimal hDBP transgene, containing a defined set of liver-specific DNase I hypersensitive sites (HSs), is robustly expressed in mouse liver in a copy-number-dependent manner. Here we evaluate these HSs for function. Deletion of HSI, located 5′ to the promoter (kb −2.1) had no significant effect on hDBP expression. In contrast, deletion of HSIV and HSV from intron 1 repressed hDBP expression and eliminated copy number dependency without a loss of liver specificity. Chromatin immunopreci

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10

Diakogiannaki, Eleftheria, Hannah J. Welters та Noel G. Morgan. "Differential regulation of the endoplasmic reticulum stress response in pancreatic β-cells exposed to long-chain saturated and monounsaturated fatty acids". Journal of Endocrinology 197, № 3 (2008): 553–63. http://dx.doi.org/10.1677/joe-08-0041.

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Exposure of pancreatic β-cells to long-chain fatty acids leads to the activation of some components of the endoplasmic reticulum (ER) stress pathway and this mechanism may underlie the ability of certain fatty acids to promote β-cell death. We have studied ER stress in BRIN-BD11 β-cells exposed to either the saturated fatty acid palmitate (C16:0) or the monounsaturated palmitoleate (C16:1). Palmitate (0.025–0.25 mM) induced the expression of various markers of the RNA-dependent protein kinase-like ER eukaryotic initiation factor 2α (eIF2α) kinase (PERK)-dependent pathway of ER stress (phospho-

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11

Bannigan, Katrina, Nicole Jade Larkan, Emmanuelle Renee Rogelia Meurgue, and Jason Chun Hin Sze. "Characteristics of Occupational Therapy Interventions to Promote Healthy Aging: Protocol for a Scoping Review." JMIR Research Protocols 13 (March 18, 2024): e55198. http://dx.doi.org/10.2196/55198.

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Background Healthy aging is a pressing public health priority. Focusing on what people do every day may be a meaningful approach to lifestyle change, suggesting a need for occupation therapy interventions to promote healthy aging. A preliminary database search was conducted, and no current or underway systematic or scoping reviews on the topic were identified. Developing an overview of studies of occupational therapy interventions to promote healthy aging is a necessary first step to understanding the existing knowledge and increasing the impact of future interventions. This scoping review wil

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12

Amengual, Jennifer E., Paul M. Johannet, Kelly Zullo, Maximilian Lombardo, and Owen A. O'Connor. "Development and Characterization of Two Lymphoma Cell Lines Resistant to the Selective HDAC6 Inhibitor Ricolinostat (ACY-1215) As a Tool to Better Understand the Biology of HDACs on Lymphoma." Blood 124, no. 21 (2014): 1777. http://dx.doi.org/10.1182/blood.v124.21.1777.1777.

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Abstract Histone deacetylase (HDAC) inhibitors possess marked activity in lymphoma and induce pleiotropic effects. HDAC6 is cytoplasmic and binds polyubiquitinated misfolded proteins to facilitate their transport to the aggresome for proteasome-independent degradation. The aggresome is a key outlet for the unfolded protein response (UPR), a quality control mechanism that promotes survival during endoplasmic reticulum stress and signals CHOP (C/EBP-homologous protein) mediated apoptosis when homeostasis cannot be reestablished. To better understand the discrete function of HDAC6 and its role in

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13

Amengual, Jennifer E., Maximilian Lombardo, Kelly M. Zullo, et al. "Mechanisms of Acquired Drug Resistance to the Isoform Selective HDAC6 Inhibitor Ricolinostat Reveals Markedly Upregulated Elements of the BTK Pathway Revealing Rational Drug : Drug Combinations." Blood 126, no. 23 (2015): 3708. http://dx.doi.org/10.1182/blood.v126.23.3708.3708.

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Abstract HDAC6 is cytoplasmic with non-histone substrates and binds poly-ubiquinated misfolded proteins facilitating transport to the aggresome for proteasome-independent degradation. The aggresome is a key outlet for the unfolded protein response (UPR), a quality control mechanism that promotes survival during endoplasmic reticulum stress and signals CHOP (C/EBP-homologous protein) mediated apoptosis when protein homeostasis cannot be reestablished. To better understand the discrete function of HDAC6 and its role in lymphoma, we developed lymphoma cell lines resistant to the selective HDAC6 i

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14

Amengual, Jennifer E., Paul M. Johannet, Xavier Jirau-Serrano, et al. "Dual Targeting With The Selective Histone Deacetylase (HDAC) 6 Inhibitor, ACY-1215, and Bortezomib (BOR) Leads To Marked Disruption Of Protein Degradation Pathways and Apoptosis In Preclinical Models Of Lymphoma." Blood 122, no. 21 (2013): 648. http://dx.doi.org/10.1182/blood.v122.21.648.648.

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Abstract The development of isoform selective HDAC inhibitors has opened the opportunity to better define and target pathways germane to lymphoma. Class IIb HDAC6 binds polyubiquinated, misfolded proteins facilitating their transport to the aggresome for proteosome-independent degradation. The aggresome is a key outlet for the unfolded protein response (UPR), a quality control mechanism that promotes survival during endoplasmic reticulum stress and signals CHOP (C/EBP-homologous protein) mediated apoptosis when homeostasis cannot be reestablished. We investigated the impact of the selective HD

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15

Borck, Patricia, Isabella Boyle, Kristina Jankovic, et al. "Abstract SY14-03: Exploiting dysregulated ribosomal homeostasis in chromosome 9p21.3 deleted cancers and microsatellite unstable cancers." Cancer Research 85, no. 8_Supplement_2 (2025): SY14–03—SY14–03. https://doi.org/10.1158/1538-7445.am2025-sy14-03.

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Abstract Synthetic lethality, a phenomenon in which alterations in two or more genes together induce cell death while changes in either gene alone do not, provides a promising approach for cancer treatment. Indeed, various inhibitors targeting synthetic lethal interactions are approved by the Food and Drug Administration or in clinical trials. Seeking to nominate additional synthetic lethal targets, we analyzed large-scale CRISPR knockout screening data and identified Pelota mRNA surveillance and ribosome rescue factor (PELO) as a promising therapeutic target for two independent and frequently

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16

Abbate, Santina, and Carol Della Ratta. "Transforming Undergraduate Evidence-Based Practice Education." Nurse Educator, June 20, 2025. https://doi.org/10.1097/nne.0000000000001917.

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Background: Despite evidence-based practice (EBP) concepts having a presence in undergraduate curricula, many practicing nurses do not meet EBP competency. Problem: Schools of nursing are addressing this gap as major curricular revisions with a competency focus are underway to promote “practice ready” graduates. Approach: This article describes a semester-long EBP activity that involves team-based active learning within a large classroom environment and leverages academic-practice partner engagement to enhance undergraduate student learning and EBP competency attainment. Outcomes: This multifa

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17

Connor, Megan C., and Jeffrey R. Raker. "Factors associated with chemistry faculty members’ cooperative adoption of evidence-based instructional practices: results from a national survey." Chemistry Education Research and Practice, 2024. http://dx.doi.org/10.1039/d3rp00194f.

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Despite institutional reform efforts to increase use of evidence-based instructional practices (EBIPs) in undergraduate chemistry and STEM courses, didactic lecture remains the predominant mode of instruction. Research to inform these initiatives routinely focuses on drivers and barriers to EBIP adoption, with recent work investigating factors associated with faculty members’ cooperative adoption of EBIPs from five STEM disciplines including chemistry. To understand the role of these specific factors within undergraduate chemistry education across a broad set of institutions, we conducted a na

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18

Zhang, Huan, Zhen Chen, and Qiujie Zhang. "Predictors of Social Workers Attitudes Towards Evidence-based Practice: Self-determination and the Moderating Role of Organisational Learning." British Journal of Social Work, February 2, 2022. http://dx.doi.org/10.1093/bjsw/bcac014.

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Abstract Social workers’ attitudes towards evidence-based practice (EBPA) are critical to evidence-based practice (EBP) implementation. However, current studies on EBPA have not yet established a theoretical framework based on the intrinsic motivations that underlie social workers’ attitudes. By applying self-determination theory, this study constructed an explanatory framework that focussed on intrinsic motivation and used organisational learning capability (OLC) as a moderating role. Furthermore, this study verified the constructed framework using data from the Chinese Social Workers Survey

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