Relevant bibliographies by topics / Ebola

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Ebola'

Author: Grafiati
Published: 4 June 2021
Last updated: 30 July 2024

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Journal articles on the topic "Ebola"

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Scully, C., L. Samaranayake, S. Petti, and R. G. Nair. "Infection control: Ebola aware; Ebola beware; Ebola healthcare." British Dental Journal 217, no. 12 (December 2014): 661. http://dx.doi.org/10.1038/sj.bdj.2014.1108.

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Ahmad, Nadeem, Rubeena Bano, and Priyanka Singh. "Ebola Virus Disease." Indian Journal of Medical & Health Sciences 3, no. 2 (2016): 131–34. http://dx.doi.org/10.21088/ijmhs.2347.9981.3216.10.

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BOZKURT, İlkay, and Hakan LEBLEBİCİOĞLU. "Ebola Virus Infection." Mediterranean Journal of Infection Microbes and Antimicrobials 3, no. 1 (June 10, 2014): 1–7. http://dx.doi.org/10.5578/mjima.8945.

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Polz-Dacewicz, Małgorzata. "Ebola virus disease." Forum Zakażeń 5, no. 6 (January 30, 2015): 335–40. http://dx.doi.org/10.15374/fz2014058.

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Sizikova, T. E., V. N. Lebedev, N. V. Karulina, O. V. Chukhralya, S. I. Syromyatnikova, and S. V. Borisevich. "A SOME ECOLOGICAL CHARACTERISTICS OF EBOLA VIRUS IN NATURAL FOCIES." Journal of microbiology epidemiology immunobiology, no. 2 (April 28, 2018): 119–26. http://dx.doi.org/10.36233/0372-9311-2018-2-119-126.

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Ebola virus that composed Ebolavirus genus of Filoviridae Family causes severe hemorrhagic fever in humans with high case-fatality rates (up to 90%). The Ebolavirus genus includes Ebola-Zaire, Ebola-Sudan, Ebola-Reston, Ebola-Tai Forest and Ebola-Bundibugyo viruses. The date about epidemic outbreaks of disease, reservoirs of infection, accidental hosts of Ebola virus are presented in this review. The date about natural reservoirs of infection are accessed only for Ebola-Zaire and Ebola-Reston viruses. For Ebola-Sudan, Ebola-Tai Forest and Ebola-Bundibugyo viruses such information is absence. The bats are natural reservoirs for Ebola-Zaire and Ebola-Reston viruses. The formation of natural reservoirs of filoviruses assumes possibilities of existence of several hosts. The interrelation of Ebola virus and their hosts, dynamics of infection are the classical «susceptible-infected-immune» (recovered) cycle. The likely schemes of rises of epidemic outbreaks, caused by Ebola-Zaire virus are suggested.
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Aftab, Rabia. "Ebola." InnovAiT: Education and inspiration for general practice 10, no. 4 (February 7, 2017): 228–32. http://dx.doi.org/10.1177/1755738016689109.

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Ebola virus disease is responsible for a very high case fatality rate of around 50–90%; it presents as a severe, rapidly developing illness. Several outbreaks of Ebola virus disease have occurred in Central and recently West Africa. Infection is transmitted to humans from animals and spreads within the human population through direct contact with infected blood or bodily fluids. No curative treatment is yet available, but early supportive care with rehydration and symptomatic management improves the chance of survival. Community engagement is needed to control outbreaks. Control of outbreaks requires a package of interventions including case management, surveillance and contact tracing with a good laboratory service, safe burials and social mobilisation. An Ebola vaccination may become available in the near future. The very high case fatality rate and recent major outbreaks require GPs to be aware of the presentation and management of suspected Ebola virus disease.
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Feldmann, Heinz, Armand Sprecher, and Thomas W. Geisbert. "Ebola." New England Journal of Medicine 382, no. 19 (May 7, 2020): 1832–42. http://dx.doi.org/10.1056/nejmra1901594.

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Kiser, Erin. "Ebola." Journal of Christian Nursing 33, no. 3 (2016): 162–66. http://dx.doi.org/10.1097/cnj.0000000000000285.

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Thomas, James C., and Reid Miller. "Ebola." Journal of Public Health Management and Practice 21, no. 5 (2015): 507–8. http://dx.doi.org/10.1097/phh.0000000000000227.

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Brown, Julian M., and Reston N. Smith. "Ebola." Journal of the Intensive Care Society 16, no. 1 (February 2015): 88. http://dx.doi.org/10.1177/1751143714564510.

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Dissertations / Theses on the topic "Ebola"

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DAINO, GIANLUCA. "Characterization of Ebola virus VP35-dsRNA binding for drug development against Ebola virus disease." Doctoral thesis, Università degli Studi di Cagliari, 2017. http://hdl.handle.net/11584/248693.

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The Ebola virus (EBOV) VP35 protein plays an important role in the inhibition of the initial innate immune responses to EBOV infection leading to Ebola virus disease development. In fact, VP35 interaction with the RIG-I like receptors (RLR) cascade components inhibits the interferon (IFN) production, impeding proper host immune response. Hence, it has been shown that VP35 is a validate drug target. Full-length His-tagged recombinant VP35 (rVP35) has been previously expressed in prokaryotic system and used to validate a biochemical pull-down assay for the screening of small molecules targeted to the VP35-double strand (ds)-RNA interactions. However, low rVP35 amount of purified protein and the use of radioactive substrate for binding evaluation, strongly limited the screening system. In the present study, a new method for high-yield rVP35 expression and purification, based on denaturation and subsequent protein refolding was established. Subsequently, a novel assay based on the use of Nickel-coated plates using a fluorescent labeled 30mer dsRNA was validated, showing a VP35 Kd value for dsRNA binding around 4 nM, comparable to the one previously reported, and a Z'-factor equal to 0.69, that indicate a good assay. The use of this biochemical assay to screen the ability of plant extracts and derived compounds to inhibit the rVP35 binding to dsRNA allowed to identified a few small molecules able to inhibit the VP35-dsRNA binding with IC50 values in the low micromolar range. Active plant extracts and derived compounds were also tested in a cellular assay to evaluate their ability to counteract the inhibitory activity of VP35 on the IFN response. We identified a number of compounds able to inhibit the VP35 function in biochemical assay but ineffective in the cellular system. Conversely, some other compound, unable to inhibit rVP35 binding to dsRNA in biochemical assay, subverted VP35 inhibition of IFN production in cellular assay. These results suggested that the VP35 binding to dsRNA may not be the driving force of the VP35 inhibition of the IFN cascade and that an alternative mechanisms of action could be hypothesized for those compounds not able to inhibit VP35-dsRNA binding but effective against the VP35 IFN inhibitory effect. An interesting compound showed activity in both biochemical and cellular assays, suggesting the possibility that some small molecules interact with VP35 in such way to disrupt its interaction with multiple targets. In order to understand the relative role of VP35-dsRNA binding in inhibiting the RLR cascade, we studied the effects of the lack of VP35 dimerization, considered essential for VP35 binding to dsRNA. For this purpose, three single point mutations, proposed to be essential for VP35 dimerization, were introduced into the coiled-coil VP35 domain. We confirmed by in silico studies that introduction of these three mutations disrupted coiled-coil dimerization. The importance of VP35 dimerization for VP35-dsRNA binding was confirmed by biochemical assay studies, where the mutant rVP35 showed a reduce ability to bind dsRNA. However, the mutant VP35 expressed in a cellular system showed a limited reduction of the ability of IFN production inhibition compared to the wild-type VP35. These results seem to confirm the hypothesis that VP35 binding to dsRNA is not the main interaction needed for VP35 inhibition of the IFN production and suggested that the VP35 interactions with cellular components do not require VP35 dimerization. In conclusion, we demonstrated that small molecules interacting with VP35 can subvert its inhibition of the IFN production, possibly inhibiting its interactions with cellular components of RLR pathway and suggested that VP35 binding to dsRNA is not the driving for VP35 inhibition RLR cascade activation.
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Dia, Emilia. ""Ebola har nu nått Europa" : En kritisk diskursanalys av nyhetsrapporteringen om ebola i svenska medier." Thesis, Stockholms universitet, JMK, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-120325.

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Genom kritiska diskursanalyser ämnar studien att undersöka nyhetsrapporteringen av ebola i de svenska tidningarna Dagens Nyheter och Aftonbladet. Delvis för att få förståelse för hur mediepanik skapas, men även hur relationen mellan nyhetsläsaren och den lidande konstrueras. Undersökningsperioden för studien är oktober 2014. Den tidigare forskningen som belyses i denna undersökning är studier om nyhetsrapporteringar kring svininfluensan, SARS-smittan och salmonellabakterien. Dessa visar på att de intensiva perioderna i nyhetsrapporteringen ofta innefattar användningen av visuella och lingvistiska medel som bidrar till att skapa en mediepanik. De teoretiska ramarna som denna uppsats utgår ifrån är Simon Cottles teorier om hur medier konstruerar mediepaniker i rapporterandet om globala kriser, Roger Fowlers teorier om nyheternas språk vid rapporteringar om kriser och Lilie Chouliarakis teorier om lidandets spektakel. Resultatet visar således att nyhetsmedierna talar om ebolan som hot mot Europa och USA, om regional spridning, katastrofhjälp, geopolitik och sjukvård. De röster som främst får en talan i dessa artiklar är Världshälsoorganisationen, myndigheter, politiker och experter som uttalar sig om olika situationer. Patienter är de som inte får komma till tals även om de nämns i artiklarna. Resultatet av de ingående analyserna i denna studie visar att avståndet till nyhetshändelserna påverkar hur nyheter om ebola skildras i medierapporteringen. I artiklar som behandlade nyhetshändelser om ebola i Afrika, betonades det att ebolasmittan fanns på en avlägsen plats. Samtidigt upptäcktes det att innehållet såsom språket blev intensivare när medierapporteringen skildrade nyheter om ebola i Europa. Därmed skapades även en känsla av kris och risk.
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Miller, Michele. "Development and Validation of Virus and Ebola Misconceptions Assessment (VirEMiA): Ebola Virus Misconceptions in College Students." Wright State University / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=wright1459333659.

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LaTouche, Alina S. "East Harlem seniors' knowledge about ebola." Thesis, Icahn School of Medicine at Mount Sinai, 2016. http://pqdtopen.proquest.com/#viewpdf?dispub=10107190.

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Older adults’ knowledge and perceptions about Ebola have not been previously explored in the literature. A survey, administered during the Ebola outbreak in 2014, was performed to capture East Harlem senior residents’ knowledge about Ebola and identify their major sources of information, their self-reported levels of trust in city officials, and whether or not they would follow officials’ directions. Results using frequency testing showed that senior respondents were not knowledgeable about disease transmission. The major trusted sources of information identified were television and newspapers. Overall, respondents trusted city officials and were very likely to follow city instructions. This study is important because it elucidates the need for better information to be more efficiently disseminated through media during a health emergency.

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Seiler, Nina. "Inkubationszeit und Übertragungsparameter der Ebola-Viruskrankheit." [S.l. : s.n.], 2008.

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Lennemann, Nicholas Joseph. "Biochemical characterization of Ebola virus GP." Diss., University of Iowa, 2014. https://ir.uiowa.edu/etd/3127.

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Filoviruses cause sporadic outbreaks of highly lethal hemorrhagic fever throughout central Africa. Virus entry is mediated by the sole viral glycoprotein, GP. Furthermore, GP is the main target for neutralizing antibodies. Thus, a better understanding of GP and its functions is critical for the development of antivirals and vaccines. GP contains a high number of N- and O-linked glycans, which shield the majority of the protein. These glycans are required for cell surface interactions with C-type lectins that mediate internalization of the virus. We found that GP1, but not GP2, N-linked glycans were required for efficient entry into cells expressing the C-type lectins: L-SIGN, DC-SIGN, and LSECtin expressing cells, but O-linked glycans were sufficient for ASGPRI- and hMGL-dependent entry. However, filoviruses also utilize phosphatidylserine (PS) receptors, which bind PS in the viral membrane, to mediate entry into host cells. We found that all N-linked glycosylation sites in GP1 could be mutated without significant impact on expression. Furthermore, removal of all N-linked glycans increased entry into a PS receptor-dependent cell line and primary murine macrophages. These results correlated with an increase in sensitivity to proteolysis, which is required within the late endosome/lysosome to expose the receptor-binding domain. Surprisingly, removal of N-linked glycans that directly shield the receptor-binding domain did not allow for binding to the intracellular receptor, NPC1. Thus, proteolytic removal of heavily glycosylated domains within the late endosome/lysosome exposes critical receptor-binding residues that are masked by polypeptides and not N-linked glycans. Furthermore, removal of the conserved N-linked glycan on the heptad repeat 1 region in GP2 led to an increase in entry. Conversely, removal of the conserved N-linked glycan on the heptad repeat 2 region decreased entry. Removal of either glycan resulted in a decrease in entry mediated by protease-treated GP. Together, these results suggest N-linked glycans on GP2 are involved in controlling fusion. Interestingly, removal of N-linked glycans masking conserved regions of GP led to a significant increase in convalescent antibody-mediated neutralization. Overall, these results indicate that there is an evolutionary trade-off that results in a decrease in entry efficiency in order to protect virus from the immune system. Analysis of entry mediated by multiple species of ebolavirus indicated that the residue occupying position 95 is a critical determinant of entry. For Ebola virus (EBOV) GP, Sudan virus (SUDV) GP, and Bundibugyo (BDBV) GP, a lysine at position 95 imparts dependence on the cysteine protease cathepsin B. However, a glutamine at this position alleviates this dependence and is found in some early isolates of SUDV. Furthermore, cathepsin B dependence inversely correlated with an increase in sensitivity to protease-mediated degradation of GP. Mutation of K95 to a glutamine in EBOV GP and BDBV GP led to decreased sensitivity to NPC1 and voltage-operated calcium channel inhibitors. Conversely, mutation of the Q95 to a lysine in SUDV GP decreased sensitivity to NPC1 inhibitors and had no impact on voltage-operated calcium channel inhibitors. However, all proteins regardless of the residue at position 95 required NPC1 for entry. Together these results indicate that a single amino acid polymorphism in GP of ebolaviruses has dramatic impacts on entry factor dependence, suggesting potential differences in entry pathways.
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Dapiaggi, F. "MOLECULAR MODELING OF EBOLA VIRUS INHIBITORS." Doctoral thesis, Università degli Studi di Milano, 2018. http://hdl.handle.net/2434/545872.

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In this PhD thesis computational methods have been employed in order to study different biologically relevant systems. In the first part of the thesis two Ebola virus proteins were studied, namely Viral Protein 24 (VP24) and Viral Protein 35 (VP35), responsible for the inhibition of the immune response . After a brief theoretical introduction to the main computational methods employed in the thesis, a study of VP35 in complex with small organic molecules is presented. These compounds are able to inhibit the interaction between VP35 and viral nucleoprotein. This study confirms the experimental findings highlighting new important key interactions between the protein the inhibitors. Moreover, an Essential Dynamics analysis points out an interesting collective motion of the apo-form that is hindered by the presence of the ligands. Afterwards, the protein-protein interaction VP24-Karyopherin (KPNA) is studied. An atomistic analysis of the interactions at the interface leads to the design of a nonapeptide with VP24 binding capability. The peptide is derived from a KPNA subsequence and could potentially inhibit the VP24-KPNA interaction. Subsequently an analysis on the pockets present on VP24 surface in different solvents is performed. Once the most promising pocket has been located, a virtual screening on a subset of ZINC database is carried out, leading to the identification of few classes of molecules potentially able to bind VP24. Finally the effect of the osmolytes on VP24 protein structure is studied, pointing out how osmoprotectants and urea have opposite effects on the protein, the former stabilizing the folded state and the latter shifting the equilibrium to the denatured state. In the second part of the manuscript the study of the interaction of an antimicrobial peptide with a lipid membrane is presented. This work was carried out in the University of Groningen under the supervision of Prof. Siewert Jan Marrink in order to deepen the Coarse Grain method.
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Elmberg, Emma, and Mathilda Nordahl. "”Världen förlorar kampen mot ebola” : En kvantitativ innehållsanalys om hur ebola gestaltades i svensk storstadspress hösten 2014." Thesis, Linnéuniversitetet, Institutionen för medier och journalistik (MJ), 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-45042.

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Mbala-Kingebeni, Placide. "Virus Ebola à l’interface homme – faune sauvage et réservoir animal des virus Ebola en République Démocratique du Congo." Thesis, Montpellier, 2019. http://www.theses.fr/2019MONTT035.

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Chaque épidémie de la maladie à virus Ebola résulte très probablement d'événements zoonotiques indépendants. Plus de quatre décennies après la première identification du virus Ebola, son réservoir demeure encore inconnu. Nous avons montré dans ce travail que la prévalence du virus Ebola dans la faune sauvage était très faible avec une présence d’anticorps anti-virus Ebola (Zaïre et/ou Sudan) chez moins de 1% des chauves-souris testées de la RDC, Guinée et Cameroun ; et de 0% chez les primates non humains de la RDC, Cote d’Ivoire et Cameroun, en période inter-épidémique. Aucun anticorps n’a été détecté dans les échantillons prélevés en période épidémique en RDC. La recherche de l’ARN du virus Ebola, au cours de ces études, était négative. Néanmoins, nous avons confirmé et caractérisé chez l’homme, les nouveaux variants du virus Ebola responsables des récentes épidémies de 2018 en RDC. Le séquençage génomique précoce et continu a permis d'orienter les interventions en matière de santé publique.Ainsi, malgré la présence d'anticorps du virus Ebola, le rôle des chauves-souris en tant qu'espèce réservoir reste flou, car la détection de l'ARN viral est encore rare. Les anticorps anti-virus Ebola sont très rares chez les primates non humains, ce qui confirme que les PNH ne sont pas des espèces réservoirs. Les efforts pour retrouver le réservoir de ce virus doivent continuer car c’est le seul moyen qui nous permettra de prévenir efficacement les prochaines épidémies
Every Ebola outbreak is most likely the result of independent zoonotic events. More than four decades after the first identification of the Ebola virus, its reservoir remains unknown. We have shown in this work that the prevalence of Ebola virus in wildlife was very low with antibodies against Ebola virus (Zaire and / or Sudan) detected in less than 1% of bats tested in the DRC, Guinea and Cameroon; and 0% in non-human primates from the DRC, Cote d'Ivoire and Cameroon, during inter-epidemic period. No antibodies were detected in samples collected during the epidemic period in the DRC, and the search for Ebola RNA in these studies was negative. Nevertheless, we have confirmed and characterized in humans, new variants of the Ebola virus which caused the recent outbreaks of 2018 in the DRC. Early and ongoing genomic sequencing has been used to guide public health interventions.Thus, despite the presence of antibodies to the Ebola virus, the role of bats as a reservoir species remains unclear, as the detection of viral RNA is still rare. Ebola virus antibodies are very rare in non-human primates, confirming that PNH are not reservoir species. Efforts to recover the reservoir of this virus must continue because it is the only way that will allow us to effectively prevent future outbreaks
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Victoriano, Analisa Machado. "Ébola: uma revisão bibliográfica." Bachelor's thesis, [s.n.], 2020. http://hdl.handle.net/10284/9282.

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Projeto de Graduação apresentado à Universidade Fernando Pessoa para obtenção do grau de Licenciada em Enfermagem
O vírus Ébola é um agente patogénico causador de uma doença sistémica, grave e com alta taxa de mortalidade em humanos e primatas não-humanos. Foi descoberto pela primeira vez em 1976 na República Democrática do Congo. Existem cinco espécies conhecidas do vírus, das quais quatro foram identificadas na África Equatorial. Uma vez que entra na população humana a transmissão ocorre principalmente pelo contato com fluídos corporais contaminados. O vírus causa uma supressão do sistema imunitário, bem como uma resposta inflamatória acentuada e grande perda de fluídos e eletrólitos. Embora existam vacinas e tratamentos em desenvolvimento, a gestão dos casos baseia-se na sintomatologia e manutenção e reposição de fluídos e eletrólitos, para prevenção e controlo da falência de órgãos. Em 2014 a Organização Mundial da Saúde classificou a epidemia causada pelo vírus Ébola como um problema da saúde pública em África e que preocupa o mundo. O Centro de Controlo e Prevenção de Doenças dos Estados Unidos listou o vírus Ébola como um agente de bioterrorismo de categoria A.
The Ebola virus is a pathogen causing a systemic, severe disease with a high mortality rate in humans and non-human primates. It was first discovered in 1976 in the Democratic Republic of congo. There are five known species of the virus, four of which have been identified in Equatorial Africa. Once it enters the human population transmission occurs mainly by contact with infected body fluids. The virus causes a suppression of the immune system, as well as a marked inflammatory response and large loss of fluids and electrolytes. Although vaccines and treatments are in development, case management is based on symptomatology and maintenance and replacement of fluids and electrolytes, for prevention and control of organ failure. In 2014, the World Health Organization classified the Ebola virus epidemic as a public health problem in Africa that worries the world. The U.S. Centers for Disease Control and Prevention has listed the Ebola virus as a Category A bioterrorism agent.
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Books on the topic "Ebola"

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Ladouceur, Jeff. Ebola. Montreal: L'Oie de Cravan, 2002.

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Masci, Joseph R., and Elizabeth Bass. Ebola. Boca Raton : Taylor & Francis, 2018.: CRC Press, 2017. http://dx.doi.org/10.1201/9781315119854.

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Stimola, Aubrey. Ebola. New York: Rosen Pub., 2011.

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Wallace, Robert G., and Rodrick Wallace, eds. Neoliberal Ebola. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40940-5.

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Lado, Marta, ed. Ebola Virus Disease. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-94854-6.

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Jespersen, Sanne. Ebola-lægens dagbog. Aarhus: Mediart, 2014.

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Ramazzotti, Sergio. Ground zero ebola. Milano: Piemme, 2015.

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Klenk, Hans-Dieter. Marburg and Ebola Viruses. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-59949-1.

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Open Society Initiative for West Africa. Ebola through the lens. Dakar, Senegal: Open Society Initiative for West Africa, 2015.

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Shkarin, Anatoliĭ. Ebola: Shalenyĭ kvest : roman. Kyïv: Knyhonosha, 2018.

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Book chapters on the topic "Ebola"

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Masci, Joseph R., and Elizabeth Bass. "The 2014–2016 Epidemic and Earlier Outbreaks." In Ebola, 3–34. Boca Raton : Taylor & Francis, 2018.: CRC Press, 2017. http://dx.doi.org/10.1201/9781315119854-1.

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Masci, Joseph R., and Elizabeth Bass. "Potential Bioterrorism Concerns." In Ebola, 147–54. Boca Raton : Taylor & Francis, 2018.: CRC Press, 2017. http://dx.doi.org/10.1201/9781315119854-10.

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Masci, Joseph R., and Elizabeth Bass. "Frequently Asked Questions." In Ebola, 155–79. Boca Raton : Taylor & Francis, 2018.: CRC Press, 2017. http://dx.doi.org/10.1201/9781315119854-11.

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Masci, Joseph R., and Elizabeth Bass. "Tabletop Exercises for Preparedness." In Ebola, 181–203. Boca Raton : Taylor & Francis, 2018.: CRC Press, 2017. http://dx.doi.org/10.1201/9781315119854-12.

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Masci, Joseph R., and Elizabeth Bass. "Cases outside Africa." In Ebola, 35–46. Boca Raton : Taylor & Francis, 2018.: CRC Press, 2017. http://dx.doi.org/10.1201/9781315119854-2.

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Masci, Joseph R., and Elizabeth Bass. "Prevention and Containment." In Ebola, 47–56. Boca Raton : Taylor & Francis, 2018.: CRC Press, 2017. http://dx.doi.org/10.1201/9781315119854-3.

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Masci, Joseph R., and Elizabeth Bass. "Global Response to the Epidemic." In Ebola, 57–69. Boca Raton : Taylor & Francis, 2018.: CRC Press, 2017. http://dx.doi.org/10.1201/9781315119854-4.

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Masci, Joseph R., and Elizabeth Bass. "Challenges in the Aftermath of Ebola in West Africa." In Ebola, 71–81. Boca Raton : Taylor & Francis, 2018.: CRC Press, 2017. http://dx.doi.org/10.1201/9781315119854-5.

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Masci, Joseph R., and Elizabeth Bass. "Virus." In Ebola, 85–95. Boca Raton : Taylor & Francis, 2018.: CRC Press, 2017. http://dx.doi.org/10.1201/9781315119854-6.

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Masci, Joseph R., and Elizabeth Bass. "Pathophysiology and Clinical Features of Ebola Virus Infection." In Ebola, 97–118. Boca Raton : Taylor & Francis, 2018.: CRC Press, 2017. http://dx.doi.org/10.1201/9781315119854-7.

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Conference papers on the topic "Ebola"

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Tithi, Saima Sultana, and Mohammad Shabbir Hasan. "Modeling ebola outbreak." In BCB '15: ACM International Conference on Bioinformatics, Computational Biology and Biomedicine. New York, NY, USA: ACM, 2015. http://dx.doi.org/10.1145/2808719.2811458.

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Tian, Jamie. "The Interaction of Ebola Virus with the Immune System Ebola and Immunity." In ICBEA '21: 2020 4th International Conference on Biometric Engineering and Applications. New York, NY, USA: ACM, 2021. http://dx.doi.org/10.1145/3476779.3476794.

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Tian, Jamie. "The Interaction of Ebola Virus with the Immune System Ebola and Immunity." In ICBEA '21: 2020 4th International Conference on Biometric Engineering and Applications. New York, NY, USA: ACM, 2021. http://dx.doi.org/10.1145/3476779.3476794.

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Ganesan, Adarsh Venkataraman. "A Novel MEMS Based Immunosensor for Ebola Virus Detection." In ASME 2013 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/imece2013-66025.

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According to WHO, “The Ebola virus can cause several viral haemorrhagic fever (VHF) outbreaks in humans with a case fatality rate of up to 90%.” On top of these physical health issues, the Ebola virus infected patients are also subjected to many psychological problems. Recently, the ebola virus had its outbreak in Uganda on May 2011. In such places, the resources are scarce and the access to hospitals is limited. Hence, there is a need for point-of-care device for the diagnosis of Ebola virus. Currently, an expensive Immunofluorescence method is used to address this need. In this paper, the authors present the design of a robust, reliable and inexpensive microfluidic patch that is capable of detecting Ebola Virus from a fingerprick sample of whole blood. The fluidic model of the device is analyzed in COMSOL Multiphysics. The analyses show that for a contact time of 1s, approximately 5.4 μL of blood can be drawn. Further, the designed micro-cantilever was analyzed and found to effectively produce 0.23557 μV for every 10 ng of the Ebola virus present.
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Xueying Bai, Wenlong Song, and Jiahe Chen. "Ebola prediction with epidemic model." In 2016 2nd IEEE International Conference on Computer and Communications (ICCC). IEEE, 2016. http://dx.doi.org/10.1109/compcomm.2016.7924822.

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Tanade, Cyrus, Nathanael Pate, Elianna Paljug, Ryan A. Hoffman, and May Dongmei Wang. "Hybrid Modeling of Ebola Propagation." In 2019 IEEE 19th International Conference on Bioinformatics and Bioengineering (BIBE). IEEE, 2019. http://dx.doi.org/10.1109/bibe.2019.00044.

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ALDILA, DIPO, EDY SOEWONO, and TRI JULIANSYAH. "Epidemic Model For Ebola Disease." In Second International Conference on Advances in Applied Science and Environmental Technology - ASET 2015. Institute of Research Engineers and Doctors, 2015. http://dx.doi.org/10.15224/978-1-63248-075-0-41.

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Yom-Tov, Elad. "Ebola data from the Internet." In DH '15: Digital Health 2015 Conference. New York, NY, USA: ACM, 2015. http://dx.doi.org/10.1145/2750511.2750512.

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Guo, He. "Inhibit the Spread of Ebola." In 3d International Conference on Applied Social Science Research (ICASSR 2015). Paris, France: Atlantis Press, 2016. http://dx.doi.org/10.2991/icassr-15.2016.30.

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Qu, Weilin, Nan Geng, and Yidi Xu. "The Strategy of Eradicating Ebola." In 2015 Seventh International Conference on Measuring Technology and Mechatronics Automation (ICMTMA). IEEE, 2015. http://dx.doi.org/10.1109/icmtma.2015.315.

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Reports on the topic "Ebola"

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Geisbert, Thomas W., and Peter B. Jahrling. Towards a Vaccine Against Ebola Virus. Fort Belvoir, VA: Defense Technical Information Center, January 2003. http://dx.doi.org/10.21236/ada428607.

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Bray, Mike, and Thomas W. Geisbert. Ebola Virus: The Role of Macrophages and Dendritic Cells in the Pathogenesis of Ebola Hemorrhagic Fever. Fort Belvoir, VA: Defense Technical Information Center, February 2005. http://dx.doi.org/10.21236/ada434710.

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Daughton, Ashlynn Rae, and Alina Deshpande. SWAP Applications to the 2014 Ebola Outbreak. Office of Scientific and Technical Information (OSTI), February 2015. http://dx.doi.org/10.2172/1170691.

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Leffel, Elizabeth K., and Douglas S. Reed. Marburg and Ebola Viruses as Aerosol Threats. Fort Belvoir, VA: Defense Technical Information Center, January 2004. http://dx.doi.org/10.21236/ada428619.

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Bedford, Juliet. SSHAP Roundtable: 2021 Ebola Outbreak in Guinea. Institute of Development Studies (IDS), March 2021. http://dx.doi.org/10.19088/sshap.2021.019.

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SSHAP convened a virtual roundtable of expert advisors on Friday 12 March 2021 to discuss the outbreak of Ebola in Guinea declared on 14 February 2021. At the time of writing (19 March 2021), there have been 18 cases (14 confirmed, 4 probable), 9 deaths (including 5 in the community; CFR 50%) and 6 recoveries. Six of the 7 first cases identified were from the family of the first case, a 51-year-old nurse from Gouecke who died in N’Zérékoré on 28 January. Vaccination was launched on 23 February, and as of 17 March, 3,492 people had been vaccinated. The last new case was reported on 4 March 2021.
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Dr. Shawn Carbonell, Dr Shawn Carbonell. Developing a new treatment for Ebola Virus Disease. Experiment, April 2015. http://dx.doi.org/10.18258/4932.

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Marsden, Willard, Aurelia Fedenisn, Jennifer Greer, Jon Herstein, Maria Powers, Mark Sampson, Ward Sax, Kristen Vosburgh, Kevin Walsh, and Bradford Walters. Ebola as an Organizational Policy Challenge: A Brief Primer. RTI Press, December 2014. http://dx.doi.org/10.3768/rtipress.2014.pb.0006.1412.

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Dr. Shawn Carbonell, Dr Shawn Carbonell. Can We Defeat EBOLA with an Experimental CANCER Drug? Experiment, August 2014. http://dx.doi.org/10.18258/3094.

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Barry Pittendrigh, Barry Pittendrigh. Ebola video voice-over recording in 10 African local languages. Experiment, December 2014. http://dx.doi.org/10.18258/4155.

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Moro, Leben, and Alice Robinson. Key Considerations: Cross-Border Dynamics between Uganda and South Sudan in the Context of the Outbreak of Ebola, 2022. Institute of Development Studies, December 2022. http://dx.doi.org/10.19088/sshap.2022.045.

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This brief summarises key considerations concerning cross-border dynamics between South Sudan and Uganda in the context of the 2022 outbreak of Ebola in Uganda, and the risk of the spread of the virus into South Sudan. It is one of four briefs exploring cross-border dynamics in the context of the outbreak, alongside Kenya, Rwanda and Tanzania. The outbreak is of the Sudan strain of Ebola (Sudan Virus Disease, SVD). SVD is used in this paper to refer to the current outbreak in East Africa, whereas outbreaks of Zaire Ebolavirus disease or general references to Ebola are referred to as EVD. The outbreak of SVD began in Mubende, Uganda, on 19 September 2022. At the time of writing (25 November), there had been 141 confirmed cases and 55 deaths, including seven health workers. Infections had been confirmed in nine districts in Uganda, including in Kampala – a major transport hub. Vaccines used in previous Ebola outbreaks are effective against the Zaire strain of Ebola, and vaccines that could work against the Sudan strain remain under investigation. As of November 2022, there have been no confirmed cases of SVD imported into South Sudan, although several alerts have been investigated. However, the fear that travellers from Uganda might bring the disease into South Sudan has spurred preparations by government institutions and partner organisations, building on the experiences acquired during past outbreaks, particularly Ebola and COVID-19. An EVD High Level Taskforce has been formed, chaired by the Minister for Cabinet Affairs and co-chaired by the Minister of Health. The South Sudan Ministry of Health (MoH) has activated the Public Health Emergency Operation Centre (PHEOC) and Incident Management System (IMS). A national EVD Readiness Plan has been developed and endorsed by the government. A free hotline (number 6666) is in place, which can be used either to report suspected cases or for information on Ebola. Training of staff at border entry points has started. This brief is based on a rapid review of published and grey literature, and informal discussions with the South Sudan Red Cross, IOM, academics from University of Juba, and the PHEOC. It was requested by the Collective Service and was written by Leben Nelson Moro (University of Juba) and Alice Robinson (London School of Economics). It was reviewed by colleagues at the University of Bath, the PHEOC, Internews, Anthrologica, the Institute of Development Studies and the Collective Service. The brief is the responsibility of the Social Science in Humanitarian Action Platform (SSHAP).
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