Relevant bibliographies by topics / ED attentional set shifting

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'ED attentional set shifting'

Author: Grafiati
Published: 10 December 2022
Last updated: 31 July 2025

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Journal articles on the topic "ED attentional set shifting"

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Rogers, R. D., T. C. Andrews, P. M. Grasby, D. J. Brooks, and T. W. Robbins. "Contrasting Cortical and Subcortical Activations Produced by Attentional-Set Shifting and Reversal Learning in Humans." Journal of Cognitive Neuroscience 12, no. 1 (2000): 142–62. http://dx.doi.org/10.1162/089892900561931.

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Much evidence suggests that lesions of the prefrontal cortex (PFC) produce marked impairments in the ability of subjects to shift cognitive set, as exemplified by performance of the Wisconsin Card Sorting Test (WCST). However, studies with humans and experimental primates have suggested that damage to different regions of PFC induce dissociable impairments in two forms of shift learning implicit in the WCST (that is, extradimensional (ED) shift learning and reversal shift learning), with similar deficits also being apparent after damage to basal ganglia structures, especially the caudate nucle
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Patel, D. D., K. R. Laws, A. Padhi, et al. "The neuropsychology of the schizo-obsessive subtype of schizophrenia: a new analysis." Psychological Medicine 40, no. 6 (2009): 921–33. http://dx.doi.org/10.1017/s0033291709991255.

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BackgroundInterest in the neuro-cognitive profile of patients with schizophrenia and co-morbid obsessive compulsive disorder (schizo-OCD) is rising in response to reports of high co-morbidity rates. Whereas schizophrenia has been associated with global impairment in a wide range of neuro-cognitive domains, OCD is associated with specific deficits featuring impaired performance on tasks of motor and cognitive inhibition involving frontostriatal neuro-circuitry.MethodWe compared cognitive function using the CANTAB battery in patients with schizo-OCD (n=12) and a schizophrenia group without OCD s
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Tournikioti, Kalliopi, Maria Alevizaki, Ioannis Michopoulos, et al. "Cortisol to Dehydroepiandrosterone Sulphate Ratio and Executive Function in Bipolar Disorder." Neuropsychobiology 80, no. 4 (2021): 342–51. http://dx.doi.org/10.1159/000513512.

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<b><i>Introduction:</i></b> Bipolar disorder (BD) is associated with impairment in cognitive domains such as verbal memory and executive functions. Very few studies have assessed dehydroepiandrosterone sulphate (DHEA-S) in BD and its relation to cognitive functioning despite evidence showing its regulatory effects on glucocorticoid action. The aim of our study was to explore the association of cortisol, DHEA-S, and cortisol to DHEA-S ratio with visuospatial memory and executive functioning in BD. <b><i>Methods:</i></b> Cognitive performance of 60
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Livingston, Nicholas R., Peter CT Hawkins, James Gilleen, et al. "Preliminary evidence for the phosphodiesterase type-4 inhibitor, roflumilast, in ameliorating cognitive flexibility deficits in patients with schizophrenia." Journal of Psychopharmacology 35, no. 9 (2021): 1099–110. http://dx.doi.org/10.1177/02698811211000778.

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Background: Cognitive flexibility deficits are present in patients with schizophrenia and are strong predictors of functional outcome but, as yet, have no pharmacological treatments. Aims: The purpose of this study was to investigate whether the phosphodiesterase type-4 inhibitor, roflumilast, can improve cognitive flexibility performance and functional brain activity in patients with schizophrenia. Methods: This was a within-subject, randomised, double-blind, placebo-controlled, three-period crossover study using a version of the Intradimensional/Extradimensional (ID/ED) task, optimised for f
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Pedersen, Christian Spang, Pascal Goetghebeur, and Rebecca Dias. "Chronic infusion of PCP via osmotic mini-pumps: A new rodent model of cognitive deficit in schizophrenia characterized by impaired attentional set-shifting (ID/ED) performance." Journal of Neuroscience Methods 185, no. 1 (2009): 66–69. http://dx.doi.org/10.1016/j.jneumeth.2009.09.014.

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Coghill, D. R., D. Hayward, S. M. Rhodes, C. Grimmer, and K. Matthews. "A longitudinal examination of neuropsychological and clinical functioning in boys with attention deficit hyperactivity disorder (ADHD): improvements in executive functioning do not explain clinical improvement." Psychological Medicine 44, no. 5 (2013): 1087–99. http://dx.doi.org/10.1017/s0033291713001761.

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BackgroundAttention deficit hyperactivity disorder (ADHD) often, but not always, persists into adulthood. Investigations of the associations between clinical and biological markers of persistence can shed light on causal pathways. It has been proposed that compensatory improvements in executive neuropsychological functioning are associated with clinical improvements. This is the first study to test this hypothesis prospectively.MethodThe clinical and neuropsychological functioning of 17 boys with ADHD (mean age 10.45 years at time 1; 14.65 years at time 2) and 17 typically developing (TYP) boy
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Gargaro, B. A., Tamara May, B. J. Tonge, D. M. Sheppard, J. L. Bradshaw, and N. J. Rinehart. "Attentional Mechanisms in Autism, ADHD, and Autism-ADHD Using a Local–Global Paradigm." Journal of Attention Disorders 22, no. 14 (2015): 1320–32. http://dx.doi.org/10.1177/1087054715603197.

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Objective: Cognitive flexibility or attentional set-shifting capacity has long been considered a core area of executive dysfunction for individuals with autism. Whether these difficulties are due to higher-level attentional difficulties associated with comorbid ADHD remains unclear. Method: The current study compared the performance of 48 participants with autism, ADHD, autism-ADHD, and a comparison group ( N = 12 per group) on a set-shifting task, which included a local–global paradigm. Results: Results of this study revealed that participants with attentional difficulties (autism + ADHD and
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Gauntlett-Gilbert, Jeremy, Richard C. Roberts, and Verity J. Brown. "Mechanisms underlying attentional set-shifting inParkinsons disease." Neuropsychologia 37, no. 5 (1999): 605–16. http://dx.doi.org/10.1016/s0028-3932(98)00049-9.

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Van der Molen, M. J. W., M. W. Van der Molen, K. R. Ridderinkhof, B. C. J. Hamel, L. M. G. Curfs, and G. J. A. Ramakers. "Attentional set-shifting in fragile X syndrome." Brain and Cognition 78, no. 3 (2012): 206–17. http://dx.doi.org/10.1016/j.bandc.2011.12.008.

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Bergvall, Å. H., T. Nilsson, and S. Hansen. "Exploring the link between character, personality disorder, and neuropsychological function." European Psychiatry 18, no. 7 (2003): 334–44. http://dx.doi.org/10.1016/j.eurpsy.2003.03.008.

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AbstractPersonality deviations and deficits in cognitive executive function are common among forensic populations. The present study on incarcerated offenders explored whether there are links between the two domains. Personality was assessed using the Temperament and Character Inventory (TCI). Neuropsychological performance, including visual working memory, attentional set-shifting and planning, were tested with the Cambridge Neuropsychological Test Automated Battery (CANTAB). Subjects with personality disorders scored high on harm avoidance, and low on self-directedness and cooperativeness. P
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Dissertations / Theses on the topic "ED attentional set shifting"

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Wang, Jiachao. "Bayesian analysis for quantification of individual rat and human behavioural patterns during attentional set-shifting tasks." Thesis, University of St Andrews, 2018. http://hdl.handle.net/10023/14843.

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Attentional set-shifting tasks, consisting of multiple stages of discrimination learning, have been widely used in animals and humans to investigate behavioural flexibility. However, there are several learning criteria (e.g., 6-correct-choice-in-a-row, or 10-out- of-12-correct) by which a subject might be judged to have learned a discrimination. Furthermore, the currently frequentist approach does not provide a detailed analysis of individual performance. In this PhD study, a large set of archival data of rats performing a 7-stage intra-dimensional/extra-dimensional (ID/ED) attentional set- sh
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Pershing, Michelle. "Acute elevations in kynurenic acid result in cognitive inflexibility in an attentinal set-shfiting task via an alpha 7-mediated mechanism." The Ohio State University, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=osu1354032404.

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Stanislaus-Carter, Rudi. "Behavioural examination of the role of the thalamic reticular nucleus in attention." Thesis, University of St Andrews, 2017. http://hdl.handle.net/10023/16298.

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The ability to selectively attend to aspects of the environment which signal opportunity or danger, while marginalising irrelevant stimuli is critical to an animal's survival. With finite cognitive resources, the brain must dedicate resources to only those stimuli that are biologically significant. Incoming thalamic information must therefore be filtered. The thalamic reticular nucleus has long been considered critically involved in modulating thalamic sensory processing. Sharing connections with both the thalamus and cortex, it is ideally located to modulate the transfer of pertinent incoming
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Crofts, Harriet Sarah. "Methodology for the repeated assessment of cognitive and physiological parameters within a home cage environment in the common marmoset (Callithrix jacchus) : assessment of repeated attentional set shifting and the sleep electroencephalogram." Thesis, University of Bristol, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.299537.

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Foister, Nicola. "Behavioural phenotyping of mice with genetic alterations of the GABA[subscript A] receptor." Thesis, University of St Andrews, 2010. http://hdl.handle.net/10023/988.

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GABA is the main inhibitory neurotransmitter of the central nervous system. GABA[subscript A]Rs are multimeric transmembrane receptors, which are composed of 5 subunits. It is known that there are 19 subunits that can make up the GABA[subscript A]Rs, allowing for a vast array of receptor subtypes. In addition to the GABA binding site GABA[subscript A]Rs have distinct allosteric binding sites for benzodiazepines, barbiturates, ethanol, certain general anaesthetics and neuroactive steroids. The molecular heterogeneity of the GABA[subscript A]R is accompanied by distinct pharmacological profiles
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Phenis, David Anthony. "Performance of Adult Rats Exposed to Elevated Levels of Kynurenic Acid during Gestation in a Rodent Target Detection Task: A Translational Model for Studying the Effects of Cognitive Training." The Ohio State University, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=osu154211727456543.

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Miguel, Patrícia Maidana. "Hipóxia-isquemia neonatal e o desenvolvimento de características relacionadas ao transtorno de déficit de atenção/hiperatividade em ratos wistar machos : análises comportamentais e dano tecidual cerebral." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2014. http://hdl.handle.net/10183/134839.

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A hipóxia-isquemia (HI) encefálica neonatal pode gerar sequelas neurológicas permanentes nos indivíduos que sobrevivem a este evento precoce. Dentre estas sequelas, o diagnóstico de Transtorno de déficit de atenção e hiperatividade (TDAH) já foi relacionado em pesquisa clínica. Sabendo que não há consenso de um modelo adequado para o estudo do TDAH em pesquisa experimental, novas abordagens que contribuam para o desenvolvimento desse modelo são necessárias. Assim, o objetivo do presente estudo foi investigar se a HI neonatal contribui para o desenvolvimento das características comportamentais
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García, Aguirre Ana I. "An evaluation of cognitive deficits in a rat-model of Huntington's disease." Thesis, University of St Andrews, 2016. http://hdl.handle.net/10023/8827.

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The purpose of this thesis was to develop methodology by which treatments for the cognitive impairments in Huntington's disease (HD) could be tested. As such, the thesis focused mainly on evaluating rats with quinolinic acid (QA) lesions of the striatum, as this manipulation mimics some aspects of the neural damage in Huntington's disease, to try to identify cognitive deficits of HD resulting from cell loss in the striatum. In the first part (Chapters 3-5), the role of the striatum in implicit memory was investigated. Chapter 3 compared the performance of rats and humans on a reaction time tas
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Jeng, Ren-Tang, and 鄭任唐. "Attentional Set-shifting in Spontaneously Hypertensive Rats." Thesis, 2011. http://ndltd.ncl.edu.tw/handle/13284139201871439196.

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碩士<br>國立中正大學<br>心理學研究所<br>99<br>Attention-deficit/hyperactivity disorder (ADHD), a common psychiatric disorder occurring in childhood and adolescence, is characterized by inattention, hyperactivity, and impulsivity. The spontaneously hypertensive rat (SHR) is a widely used animal model for ADHD research. A number of studies suggest that the prefrontal cortex dysfunctions can impair the inhibition system of ADHD patients, resulting in their impulsive behaviors. In human studies, the attentional set-shifting task (ASST) is frequently used to study the inhibition system, and its influences on att
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Ou, Hui-Shan, and 歐惠姍. "Deficit in attentional set shifting of MPTP-induced Parkinson's disease rat model." Thesis, 2017. http://ndltd.ncl.edu.tw/handle/st9v8k.

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碩士<br>國立中正大學<br>心理學系研究所<br>105<br>Parkinson’s disease (PD) is a neurodegenerative disorder. In addition to the typical motor symptoms, some patients display cognitive deficits in the mid or later phases of the disease. To study the pathology and physiology of the disease, the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)- lesioned rat has been widely used as an animal model. However, behavioral flexibility of MPTP-lesioned rats has not been thoroughly studied yet. Furthermore, previous studies on PD have been focused mostly on the dopaminergic innervation on the classic cortex-basal gan
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Books on the topic "ED attentional set shifting"

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Lovic, Vedran. Artificially reared female rats show reduced prepulse inhibition and deficits in attentional set shifting task. National Library of Canada, 2002.

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Book chapters on the topic "ED attentional set shifting"

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Brown, Verity J., and David S. Tait. "Attentional Set-Shifting Across Species." In Translational Neuropsychopharmacology. Springer International Publishing, 2015. http://dx.doi.org/10.1007/7854_2015_5002.

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"Attentional Set-Shifting Test." In Encyclopedia of Animal Cognition and Behavior. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-319-55065-7_300197.

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Robbins, T. W. "Shifting and stopping: frontostriatal substrates, neurochemical modulation and clinical implications." In Mental Processes in the Human Brain. Oxford University PressOxford, 2008. http://dx.doi.org/10.1093/oso/9780199230617.003.0013.

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Abstract The neuropsychological basis of attentional set-shifting, task-set switching and stop-signal inhibition is reviewed through comparative studies of humans and experimental animals. Using human functional neuroimaging, plus neuropsychological investigation of patients with frontal damage quantified by structural magnetic resonance imaging, and through parallels with effects of specific lesions of the prefrontal cortex (PFC) and striatum in rats and marmosets, it is possible to define both distinct and overlapping loci for tasks such as extra-dimensional shifting and reversal learning, s
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Roberts, Angela C. "Dopaminergic and Serotonergic Modulation of Two Distinct Forms of Flexible Cognitive Control: Attentional Set‐Shifting and Reversal Learning." In Neuroscience of Rule-Guided Behavior. Oxford University Press, 2007. http://dx.doi.org/10.1093/acprof:oso/9780195314274.003.0016.

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Conference papers on the topic "ED attentional set shifting"

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Fernandes, L. S., D. S. Ferreira, P. R. Almeida, and N. S. Dias. "Aging and attentional set shifting on WCST: An event-related EEG study." In 2015 7th International IEEE/EMBS Conference on Neural Engineering (NER). IEEE, 2015. http://dx.doi.org/10.1109/ner.2015.7146817.

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