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Books on the topic 'Edema pulmonar'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 April 2022

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1

Kenneth, Weir E., and Reeves John T, eds. Pulmonary edema. Armonk, NY: Futura Pub. Co., 1998.

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2

A, Matthay Michael, and Ingbar David H, eds. Pulmonary edema. New York: Marcel Dekker, 1998.

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3

A, Matthay Michael, ed. Symposium on pulmonary edema. Philadelphia: W.B. Saunders, 1985.

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4

Vásquez Bajaña, Viviana Beatriz, Madeleine Juliana Sarmiento Cabrera, Génesis Carolina Romoleroux Uquillas, Maite Guisella Santillan Arias, Pamela Elena Salas Espín, Yosselin Yolanda Gualancañay Zurita, Cirlei Elizabeth Pita Aveiga, et al. Introducción a la Medicina Interna: Conceptos fundamentales. Mawil Publicaciones de Ecuador, 2022, 2022. http://dx.doi.org/10.26820/978-9942-602-44-2.

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En este libro «Introducción a la medicina interna» se ofrece al lector información actualizada sobre diversos temas referentes a patologías que cursan como causa principal de alta morbimortalidad en el mundo contemporáneo. Todos de gran interés y vital importancia para la preservación de la salud. En el capítulo I, se enfoca en la discusión de los «conceptos elementales» relacionados con la medicina interna, las competencias profesionales del médico internista y la bioética en la práctica médica. En el capítulo II, se aborda el tema de las «enfermedades del sistema cardiovascular». Entre estas: La epidemiología de la enfermedad cardiovascular, los factores de riesgo de las enfermedades cardiovasculares, la hipertensión arterial, el edema agudo del pulmón, el paro cardiaco y la reanimación. En el capítulo III, se exponen algunas de las principales «enfermedades del sistema respiratorio». En primer lugar, se describe el aparato respiratorio humano, para seguidamente hacer referencia a la historia clínica en las patologías respiratorias y exponer con detalle la enfermedad pulmonar obstructiva crónica, el derrame plural y la disnea.

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5

Blanchard-Loeb. Heart Failure and Pulmonary Edema: Pathophysiology for Nurses Video Series. Delmar Learning, 2000.

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6

Stroman, Patrick William. The dynamic evaluation of alveolar fluid clearance using proton and deuteron nuclear magnetic resonance imaging. 1993.

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7

Honeyfield, Dale Cloyd. Enzymatic assay, induction and toxicity associated with the conversion of indoleacetic acid to 3-methylindole in a ruminal Lactobacillus. 1988, 1988.

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8

Shaefi, Shahzad, and Aaron Mittel. Disruption of Diffusion. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0019.

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Acute respiratory distress syndrome (ARDS), transfusion-related acute lung injury (TRALI), and transfusion-associated circulatory overload (TACO) are common conditions in critically ill patients that lead to pulmonary edema and hypoxemia. The nonhydrostatic edema characteristic of ARDS and TRALI is caused by an intense inflammatory response leading to increased microvascular permeability and alveolar injury. TACO is an acute hydrostatic edema temporally associated with events that precipitate lung injury. Lung-protective ventilation is the mainstay of therapy for ARDS and TRALI; optimizing gas exchange is the goal for all three. Prompt recognition is an important skill for perioperative practitioners.

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9

Blanchard-Loeb. Heart Failure and Pulmonary Edema: Expert Drug Therapy Series (Expert Drug Therapy Video Series). Delmar Learning, 2000.

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10

Gandhi, Shephali G. The effect of pulmonary edema fluid on ion transport by adult alveolar type II epithelial cells. 2007.

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11

Khan, Nayema, and John Pawlowski. Disruption of Diffusion. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0020.

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Adequate gas exchange in the lungs requires a balance between three key processes: ventilation (V), the flow of gas from the environment to the alveoli; perfusion (Q), the circulation to the pulmonary capillary beds; and diffusion of the gas from the alveolar space into the alveolar capillaries. This chapter discusses the management of diseases of the air space, which include secretions, pneumonia, pulmonary edema, and hemoptysis. Collectively these conditions result in the build-up of fluid in the alveolar space and thickening of the alveolar membrane, leading to a mismatch in ventilation and perfusion (V/Q mismatch). Both anesthesia and disease states can adversely affect gas exchange and the chapter discusses strategies to maximize a patient’s pulmonary status in order to minimize perioperative pulmonary complications.

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12

Chidambaran, Vidya, and Senthilkumar Sadhasivam. Foreign Body in the Airway. Edited by Erin S. Williams, Olutoyin A. Olutoye, Catherine P. Seipel, and Titilopemi A. O. Aina. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190678333.003.0012.

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Anesthetic management of suspected foreign body aspiration in the airway can be challenging. It is critical to develop a coordinated plan with the surgeon. Removal of a foreign body may necessitate laryngoscopy, bronchoscopy (commonly used), thoracoscopy, thoracotomy, or even a tracheotomy. Anesthesia could be induced using inhalation or intravenous anesthetics, while maintaining spontaneous ventilation. However, there is no consensus as to whether controlled or spontaneous ventilation is more advantageous. Maintaining deep planes of anesthesia, with minimal airway reflexes, during bronchoscopy is essential. In the event that total airway obstruction due to a tracheal foreign body occurs, a potentially life-saving technique is to push the object deeper into one of the main bronchi, for temporary relief. Postoperatively, steroids, racemic epinephrine, and intubation/ventilation may be necessary for airway edema. A chest x-ray may be indicated to rule out postobstructive pulmonary edema, pneumothorax, and pneumonia.

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13

Nussbaumer-Ochsner, Yvonne, and Konrad E. Bloch. Sleep at high altitude and during space travel. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0054.

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This chapter summarizes data on sleep–wake disturbances in humans at high altitude and in space. High altitude exposure is associated with periodic breathing and a trend toward reduced slow-wave sleep and sleep efficiency in healthy individuals. Some subjects are affected by altitude-related illness (eg, acute and chronic mountain sickness, high-altitude cerebral and pulmonary edema). Several drugs are available to prevent and treat these conditions. Data about the effects of microgravity on sleep are limited and do not allow the drawing of firm conclusions. Microgravity and physical and psychological factors are responsible for sleep–wake disturbances during space travel. Space missions are associated with sleep restriction and disruption and circadian rhythm disturbances encouraging use of sleep medication. An unexplained and unexpected finding is the improvement in upper airway obstructive breathing events and snoring during space flight.

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14

Karon, Barry L., and Naveen L. Pereira. Heart Failure and Cardiomyopathies. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0046.

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Heart failure is a clinical syndrome characterized by the inability of the heart to maintain adequate cardiac output to meet the metabolic demands of the body while still maintaining normal or near-normal ventricular filling pressures. Heart failure may be present at rest, but often it is present only during exertion as a result of the dynamic nature of cardiac demands. For correct treatment of heart failure, the mechanism, underlying cause, and any reversible precipitating factors must be identified. Typical manifestations of heart failure are dyspnea and fatigue that limit activity tolerance and fluid retention leading to pulmonary or peripheral edema. The most recent proposed categorization divided the cardiomyopathies into primary and secondary cardiomyopathies, and the primary disorders are further subdivided as genetic, acquired, or mixed. Although this proposal takes into account our progressive understanding of this heterogeneous group of disorders, the previous phenotypic classification of dilated, hypertrophic, and restrictive diseases still provides utility in day-to-day understanding and management of these disorders.

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15

1946-, Scharf Steven M., ed. Cardiopulmonary physiology in critical care. New York: Dekker, 1992.

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16

AlJaroudi, Wael. Risk Assessment Before Noncardiac Surgery. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199392094.003.0014.

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Perioperative risk assessment is essential in screening patients before noncardiac surgery. Cardiovascular complications such as fatal and non-fatal myocardial infarction (MI), ventricular arrhythmia, pulmonary edema, and stroke are important in-hospital causes of morbidity and mortality intra and post-operatively. The optimal approach is to identify patients at increased risk so that appropriate testing and therapeutic interventions are undertaken a priori to minimize such risk. The initial preoperative evaluation includes identification of surgery-specific risk, patient exercise functional capacity and clinical risk profile. Patients with major predictors of events such as acute coronary syndromes, recent MI, unstable arrhythmia, and severe valvular disease warrant further management and optimization that often lead to delaying surgery. Those with three or more predictors (history of ischemic heart disease, compensated heart failure, diabetes, renal insufficiency, or history of cerebrovascular disease) undergoing high- risk surgery often require stress testing. Although data from randomized prospective trials are lacking, numerous studies have demonstrated the utility of myocardial perfusion imaging (MPI) for determination of perioperative cardiac risk. The goal of this chapter is to review the use of MPI for preoperative risk assessment and the recommendations from the current guidelines. The focus will be on short-term and long-term prognosis including special groups such as after coronary stenting and before vascular surgery, liver and renal transplantation.

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