Books: 'EEG wave'

1

Sanei, Saeid. EEG signal processing. Chichester: John Wiley & Sons, 2007.

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2

Driessen, Martin Michael. Een ware held. Amsterdam: Wereldbibliotheek, 2014.

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3

Arie Pieter van den Berg. A hybrid method for the solution of seismic wave propagation problems =: Een hybride oplossingsmethode voor seismische golfvoortplantingsproblemen. [Utrecht]: Instituut voor Aardwetenschappen der Rijksuniversiteit te Utrecht, 1987.

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4

Het ware leven, een roman. Amsterdam: De Arbeiderspers, 2006.

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5

Weisbrod, Harry. Wage-hour and EEO compliance and litigation prevention. New York, NY: American Institute of Certified Public Accountants, 1991.

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6

Aalbers, Bert. De ware Judas: Nieuw licht op een duister figuur. Kampen: Kok, 2006.

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7

Eynikel, Hilde. Over "Gastons oorlog": Het ware verhaal van een omstreden verzetsman. Leuven: Davidsfonds, 1997.

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8

Raad, Sociaal-Economische. Advies bijzondere verhoging minimumloon: Advies inzake de wenselijkheid van een bijzondere verhoging van het wettelijke minimumloon. ʼs-Gravenhage: De Raad, 1990.

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9

Kraft, Thomas. De verwarde geest: Ware verhalen uit de praktijk van een psychiater. Amsterdam: Veen, 1998.

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10

Houdijk, Cootje. Naar de ware Jacob: Dagboek van een voettocht naar Santiago de Compostela. Schoorl: Conserve, 1990.

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11

Raad, Sociaal-Economische. Advies bijzondere verhoging minimumloon en-vakantiebijslag: Advies inzake de wenselijkheid van een bijzondere verhoging van het wettelijke minimumloon en inzake de wenselijkheid van een aanpassing van de minimumvakantiebijslag. Den Haag: Sociaal-Economische Raad, 1986.

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12

Wae uri nŭn chŏngbu ege paesin tanghalkka: Minjujuŭi rŭl wihan kyŏngjehak. Sŏul T'ŭkpyŏlsi: Panbi, 2015.

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13

Het punt des tijds: De ware wereld achter 'Knielen op een bed violen'. Amsterdam: De Bezige Bij, 2008.

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14

Noens, Ludo. Subliem licht op de lijkwade van Turijn: Ware herkomst van een omstreden middeleeuwse relikwie. Soesterberg, Nederland: Uitgeverij Aspekt, 2015.

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15

An Ch'ŏl-su sindŭrom: Wae Han'gugin ŭn An Ch'ŏl-su ege yŏlgwang hanŭn'ga? Sŏul-si: P'yŏngdan, 2012.

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16

Het ware verhaal van de Kever: Hoe Hitler het ontwerp van een joods genie confisqueerde. Diemen: Veen Magazines, 2009.

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17

Dost, Bernard. The NARS array: A seismic experiment in Western Europe = Het NARS array : een seismisch experiment in West-Europa. [Utrecht: Instituut voor Aardwetenschappen der Rijksuniversiteit te Utrecht, 1987.

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18

Proveniers, Adri. New wave in building: A flexible way of design, construction and real estate management = een flexibele methode voor ontwerpen, uitvoeren en vastgoedbeheer. Assen: Van Gorcum, 1990.

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19

Sin, Hye-jŏng. Wae amu to na ege marhae chuji ananna: Sin Hye-jŏng Siin ŭi Taehan Min'guk wŏnjaryŏk palchŏnso kihaeng. Sŏul-si: Homi, 2015.

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20

Krol, Edzard (Edzard Z.), ed. Liefde in een verscheurd land: Joanna uit Koerdistan: het ware verhaal van haar ontsnapping aan de wraak van de Irakezen. Utrecht: Bruna, 2007.

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21

Hucbald, of Saint Amand, -930, ed. Terug naar de ware Lebuïnus: Hoe de evangelisator van Saksenland vanaf de IXe eeuw werd beschreven, vereerd en miskend : met een vertaling van de anonieme Vita Antiqua en de Vita Lebuini van Hucbaldus. Deventer: Deventer Universitaire Pers, 2010.

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22

Sanei, Saeid, and Jonathon A. Chambers. EEG Signal Processing. Wiley & Sons, Incorporated, John, 2008.

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23

Sanei, Saeid, and Jonathon A. Chambers. EEG Signal Processing. Wiley & Sons, Incorporated, John, 2013.

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24

EEG Signal Processing. Wiley-Interscience, 2007.

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25

Steiger, Axel. Sleep in endocrine disorders. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0044.

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Related to bidirectional interaction between electrophysiological and endocrine activity during sleep, which are assessed by sleep electroencephalography (EEG) and hormone profiles, respectively, sleep changes occur frequently in endocrine disorders. In most of these disorders, sleep is impaired. Only in patients with prolactinoma is slow-wave sleep elevated. This chapter summarizes the current knowledge on sleep in disorders of the hypothalamic–pituitary–adrenocortical (HPA) and hypothalamic–pituitary–somatotropic (HPS) systems, in hypo- and hyperthyroidism, in diabetes mellitus, in prolactinoma, in disorders related to gonadal hormones, and with regard to disturbed endocrine rhythms related to environmental influences.

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26

Youngblood, Mark W., and Hal Blumenfeld. Biological Basis of Primary Generalized Epilepsies—Pathophysiology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0037.

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The primary generalized epilepsies include a heterogeneous group of seizures including absence, myoclonic, and generalized tonic-clonic seizures that are not strictly localized on EEG and not secondary to another disorder. The seizures are often associated with a loss of consciousness and may present with motor manifestations, including convulsions and arrest of respiration. Generalized spike-and-wave discharges on electroencephalogram are a uniting feature, and this pattern of activity is a direct manifestation of the underlying mechanism of these disorders. A review of important underlying circuitry will set the stage to discuss the pathological and genetic basis of these disorders, and the chapter will conclude with a review of current and potential therapeutics.

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27

Sperling, George, and Zhong-Lin Lu. Objectless Motion. Oxford University Press, 2017. http://dx.doi.org/10.1093/acprof:oso/9780199794607.003.0079.

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The sum of two sine waves of the same frequency is yet another sine wave. When a moving sinewave grating (e.g., continuously translating from left to right) is added to (superimposed on) a stationary sinewave grating (the pedestal) with twice the amplitude, the sum is a sine-wave grating that wobbles back and forth. Remarkably, the left–right direction of the moving grating can be perceived just as accurately in pedestalled motion as in normal motion. At temporal frequencies of 10 Hz and greater, the wobble is too quick to be perceived. The moving pedestalled sine-wave grating is perceived as an invisible left-to-right horizontal wind above the summed sine-wave grating that wobbles back and forth at low temporal frequencies of motion but appears to be absolutely stationary at high temporal frequencies.

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28

MD, Edward B. Bromfield, and Wendi M. Nugent REEGT RPSGT. Atlas of Adult EEG: Rhythms in Sleep and Wake. Butterworth-Heinemann, 2000.

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29

Nussbaumer-Ochsner, Yvonne, and Konrad E. Bloch. Sleep at high altitude and during space travel. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0054.

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This chapter summarizes data on sleep–wake disturbances in humans at high altitude and in space. High altitude exposure is associated with periodic breathing and a trend toward reduced slow-wave sleep and sleep efficiency in healthy individuals. Some subjects are affected by altitude-related illness (eg, acute and chronic mountain sickness, high-altitude cerebral and pulmonary edema). Several drugs are available to prevent and treat these conditions. Data about the effects of microgravity on sleep are limited and do not allow the drawing of firm conclusions. Microgravity and physical and psychological factors are responsible for sleep–wake disturbances during space travel. Space missions are associated with sleep restriction and disruption and circadian rhythm disturbances encouraging use of sleep medication. An unexplained and unexpected finding is the improvement in upper airway obstructive breathing events and snoring during space flight.

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30

Koelemeijer, Judith. HET ZWIJGEN VAN MARIA ZACHEA: EEN WARE FAMILIEGESCHIEDENIS. Plataan, 2001.

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31

Daniel, Meijers, ed. Ware fictie: Een experiment in antropologie en literatuur. Leuven: Garant, 1996.

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32

Sutter, Raoul, Trudy Pang, and Peter W. Kaplan. EEG in Metabolic Disorders, Intoxications, and Epileptic Encephalopathies. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0017.

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This chapter provides a systematic overview of the diagnostic and prognostic value of electroencephalography (EEG) in adult patients with different types of encephalopathies in association with metabolic, toxic, and epileptic disorders. Most encephalopathies present with a fluctuating course characterized by typical but not pathognomonic symptoms such as cognitive impairment, altered mental status or confusion, lethargy, decreased or rarely increased motor activity, and disturbed sleep/wake cycles. EEG enables rapid, bedside electrophysiological monitoring, providing dynamic real-time information on the integrity of neocortical brain activity. Hence, EEG complements clinical and neuroimaging assessments of encephalopathic patients. Progressive slowing of EEG background activity with increasing cerebral dysfunction, emergence of intermittent transients, electrographic seizures, and impaired background reactivity to external stimuli all provide important diagnostic and prognostic information to guide medical management.

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33

Barold, S. Serge. Atrioventricular conduction abnormalities and atrioventricular blocks: ECG patterns and diagnosis. Edited by Giuseppe Boriani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0453.

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The diagnosis of first-degree and third-degree atrioventricular (AV) block is straightforward but that of second-degree AV block is more involved. Type I block and type II second-degree AV block are electrocardiographic patterns that refer to the behaviour of the PR intervals (in sinus rhythm) in sequences (with at least two consecutive conducted PR intervals) where a single P wave fails to conduct to the ventricles. Type I second-degree AV block describes visible, differing, and generally decremental AV conduction. Type II second-degree AV block describes what appears to be an all-or-none conduction without visible changes in the AV conduction time before and after the blocked impulse. The diagnosis of type II block requires a stable sinus rate, an important criterion because a vagal surge (generally benign) can cause simultaneous sinus slowing and AV nodal block, which can resemble type II block. The diagnosis of type II block cannot be established if the first post-block P wave is followed by a shortened PR interval or by an undiscernible P wave. A narrow QRS type I block is almost always AV nodal, whereas a type I block with bundle branch block barring acute myocardial infarction is infranodal in 60–70% of cases. All correctly defined type II blocks are infranodal. A 2:1 AV block cannot be classified in terms of type I or type II block, but it can be AV nodal or infranodal. Concealed His bundle or ventricular extrasystoles may mimic both type I or type II block (pseudo-AV block), or both

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34

Inkomens van jongeren: Naar een geïntegreerd beleid. Rijswijk: Ministerie van Welzijn, Volksgezondheid en Cultuur, 1989.

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35

Venner, Anne, and Patrick M. Fuller. An overview of sleep–wake circuitry. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198778240.003.0005.

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How and when we wake and sleep are under the control of incredibly complex neural circuitry, consisting of neuronal populations (or nodes), neurotransmitters, and pathways that form orchestrated wake- or sleep-promoting networks. When any aspect of this neural circuitry is impaired (e.g. disease) or altered by external factors (e.g. stress), sleep and wake can be disrupted, sometimes quite profoundly. As one example, selective loss of orexin neurons in the lateral hypothalamus results in the sleep disorder narcolepsy. While our understanding of how discrete circuit elements in the brain work together to regulate wake and sleep remains incomplete, the relatively recent development of genetically driven tools and techniques has enabled a far more detailed understanding of the functional and structural basis of this circuitry. In this chapter, we review the current state of our understanding of the brain circuitry regulating sleep and wake, including how disruption of discrete circuit elements underlies a myriad of sleep- and wake-disorders.

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36

Archer, Nick, and Nicky Manning. Fetal cardiac rhythm. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199230709.003.0013.

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Introduction 166Identification of cardiac rhythm 168Normal rhythms 172Fast abnormal rhythms 184Slow abnormal rhythms 194Irregular rhythms 200Normal cardiac rhythm originates in the sinus node, a RA structure. Atrial electrical depolarization is manifest on the electrocardiogram (ECG) by a P wave and is followed by atrial contraction....

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37

Books, Arie Coloring. Easter Coloring Ideas Activity and Coloring Book: 50 Funny for Boys Easter Egg, Rabbit, Calendar, Easter Egg, Egg Hunt, Willow, Wave, Dove Image Quiz Words and Coloring Book. Independently Published, 2020.

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38

Wohlbier, Thomas. Metallic Glasses and Their Composites. Materials Research Forum LLC, 2021. http://dx.doi.org/10.21741/9781644901014.

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Metallic glasses and their crystal/glass composites find ever more applications in such fields as mini transformers, microelectromechanical devices, pressure sensors, precision surgical instruments, biological implants and sportive goods (springs, diaphragms, membranes, knife blades, electromagnetic wave shields, optical mirrors, power inductors, Coriolis flow meters, etc.). The book reviews recent research and suggests future developments, e.g. in the area of dual-phase composite/hybrid materials.

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39

Katirji, Bashar. Specialized Electrodiagnostic Studies. Edited by Bashar Katirji. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603434.003.0003.

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In addition to nerve conduction nerve studies and needle EMG, the clinical EMG study include more specialized examinations Some of the tests, such as the F waves and H reflexes are now often used as part of the routine nerve conduction studies. Others are utilized for specific indications: Repetitive nerve stimulation and single fiber EMG are used predominantly in patients with suspected neuromuscular junction disorders such as myasthenia gravis, Lambert-Eaton myasthenic syndrome or botulism; the blink reflexes are used in patients with disorders of the facial and trigeminal nerves as well as brainstem conditions. This chapter covers the late responses, including the F waves, H reflexes and blink reflexes and their applications in the diagnosis of peripheral nerve disorders. This is a followed by a discussion of the basic concepts of slow and rapid repetitive nerve stimulation and single fiber EMG, as well as their applications in the diagnosis of neuromuscular junction disorders.

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40

Shirodaria, Cheerag, and Sam Dawkins. Acute coronary syndromes. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0090.

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The term ‘acute coronary syndrome’ includes unstable angina, ST-elevation myocardial infarction (STEMI), and non-ST-elevation myocardial infarction (NSTEMI). The difference between these three syndromes is as follows. In STEMI and NSTEMI, there is evidence of myocardial necrosis, as evidenced by raised cardiac enzymes, specifically, the very sensitive cardiac biomarker troponin. STEMI is diagnosed when the ECG shows persisting ST elevation in an appropriate territory consistent with STEMI whereas, in NSTEMI, there can be any or no ECG changes, or very transient, self-limiting ST elevation. In unstable angina, there is no myocardial necrosis, and troponins are normal. The ECG is as for NSTEMI and often shows no change, ST depression, or T-wave inversion. The prognoses in STEMI and NSTEMI are identical; unstable angina has a better prognosis than either STEMI or NSTEMI.

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41

Beninger, Richard J. Mechanisms of dopamine-mediated incentive learning. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198824091.003.0012.

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Mechanisms of dopamine-mediated incentive learning explains how sensory events, resulting from an animal’s movement and the environment, activate cortical glutamatergic projections to dendritic spines of striatal medium spiny neurons to initiate a wave of phosphorylation. If no rewarding stimulus is encountered, a subsequent wave of phosphatase activity undoes the phosphorylation. If a rewarding stimulus is encountered, dopamine initiates a cascade of events in D1 receptor-expressing medium spiny neurons that may prevent the phosphatase effects and work synergistically with signaling events produced by glutamate. As a result, corticostriatal synapses have a greater impact on response systems; this may be part of the mechanism of incentive learning. Dopamine acting on dendritic spines of D2 receptor-expressing medium spiny neurons may prevent synaptic strengthening by inhibiting adenosine signaling; these synapses may be weakened through mechanisms involving endocannabinoids. When dopamine concentrations drop, e.g. during negative prediction errors, the opposite may occur, producing inverse incentive learning.

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42

Tussen droom en werkelijkheid: Een keuze uit de literaire pagina van De ware tijd. Paramaribo: Uitgeverij Okopipi, 2001.

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43

Jones, Michael, Norman Qureshi, and Kim Rajappan. Atrial flutter. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0117.

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Atrial flutter is the term given to one of the four types of supraventricular tachycardia; in it, atrial activation occurs as a consequence of a continuous ‘short circuit’: a defined and fixed anatomical route, resulting in a fairly uniform atrial rate, and uniform atrial flutter waves on the ECG. The ventricles are not a part of this arrhythmia circuit, and ventricular activation is variable, dependent on atrioventricular (AV) nodal conduction. Given that the atrial rate is essentially uniform (e.g. 300 min−1), ventricular activation tends to be regular (i.e. 150 min−1, 100 min−1, 75 min−1, etc., if the atrial rate is 300 mins−1), or regularly irregular if changes are occurring in the fraction of conducted impulses to the ventricles. When AV nodal conduction permits only 4:1 conduction or less, atrial flutter is usually obvious, but when ventricular rates are higher (150 min−1 or more) the flutter waves can be obscured by the QRS complexes, making diagnosis more difficult. Atrial flutter is of two types, typical and atypical. Typical atrial flutter is a right atrial tachycardia, with electrical activation proceeding around the tricuspid valve annulus. This arrhythmia is dependent on a zone of slow electrical conduction through the cavotricuspid isthmus (the tissue lying between the origin of the inferior vena cava and the posterior tricuspid valve). The resulting circuit can be either anticlockwise (activation proceeds up the inter-atrial septum, across the atrial roof, down the free wall, and then through the cavotricuspid isthmus to the basal septum) or clockwise (down the inter-atrial septum and around the circuit in the opposite direction). Anticlockwise typical atrial flutter is more common. Atypical atrial flutter refers to all other atrial flutters, and this includes other right atrial flutters (e.g. pericristal flutter), left atrial flutters, post-ablation or post-surgical flutters, and pulmonary vein flutters. The feature common to all types of flutter and which differentiates flutter from other types of supraventricular tachycardia is the presence of a macro-re-entrant anatomical circuit around which the electrical impulse travels continuously and repeatedly, thereby generating the flutter. Even though typical atrial flutter has a fairly obvious and specific appearance on the ECG, atypical flutters do not, and often it is only possible to differentiate atypical flutter from atrial tachycardias by invasive electrophysiology studies, as the ECG alone may be insufficient.

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44

Sŏng-wi, Kang, ed. Wae sanŭngarŭl munnŭn chŏrmŭnidŭl ege: 50-in ŭi chisŏngdŭl i ponaenŭn myŏngsang esei. Sŏul Tʻŭkpyŏlsi: Munhak Sasangsa, 1993.

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45

Tibaldi, Stefano, and Franco Molteni. Atmospheric Blocking in Observation and Models. Oxford University Press, 2018. http://dx.doi.org/10.1093/acrefore/9780190228620.013.611.

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The atmospheric circulation in the mid-latitudes of both hemispheres is usually dominated by westerly winds and by planetary-scale and shorter-scale synoptic waves, moving mostly from west to east. A remarkable and frequent exception to this “usual” behavior is atmospheric blocking. Blocking occurs when the usual zonal flow is hindered by the establishment of a large-amplitude, quasi-stationary, high-pressure meridional circulation structure which “blocks” the flow of the westerlies and the progression of the atmospheric waves and disturbances embedded in them. Such blocking structures can have lifetimes varying from a few days to several weeks in the most extreme cases. Their presence can strongly affect the weather of large portions of the mid-latitudes, leading to the establishment of anomalous meteorological conditions. These can take the form of strong precipitation episodes or persistent anticyclonic regimes, leading in turn to floods, extreme cold spells, heat waves, or short-lived droughts. Even air quality can be strongly influenced by the establishment of atmospheric blocking, with episodes of high concentrations of low-level ozone in summer and of particulate matter and other air pollutants in winter, particularly in highly populated urban areas.Atmospheric blocking has the tendency to occur more often in winter and in certain longitudinal quadrants, notably the Euro-Atlantic and the Pacific sectors of the Northern Hemisphere. In the Southern Hemisphere, blocking episodes are generally less frequent, and the longitudinal localization is less pronounced than in the Northern Hemisphere.Blocking has aroused the interest of atmospheric scientists since the middle of the last century, with the pioneering observational works of Berggren, Bolin, Rossby, and Rex, and has become the subject of innumerable observational and theoretical studies. The purpose of such studies was originally to find a commonly accepted structural and phenomenological definition of atmospheric blocking. The investigations went on to study blocking climatology in terms of the geographical distribution of its frequency of occurrence and the associated seasonal and inter-annual variability. Well into the second half of the 20th century, a large number of theoretical dynamic works on blocking formation and maintenance started appearing in the literature. Such theoretical studies explored a wide range of possible dynamic mechanisms, including large-amplitude planetary-scale wave dynamics, including Rossby wave breaking, multiple equilibria circulation regimes, large-scale forcing of anticyclones by synoptic-scale eddies, finite-amplitude non-linear instability theory, and influence of sea surface temperature anomalies, to name but a few. However, to date no unique theoretical model of atmospheric blocking has been formulated that can account for all of its observational characteristics.When numerical, global short- and medium-range weather predictions started being produced operationally, and with the establishment, in the late 1970s and early 1980s, of the European Centre for Medium-Range Weather Forecasts, it quickly became of relevance to assess the capability of numerical models to predict blocking with the correct space-time characteristics (e.g., location, time of onset, life span, and decay). Early studies showed that models had difficulties in correctly representing blocking as well as in connection with their large systematic (mean) errors.Despite enormous improvements in the ability of numerical models to represent atmospheric dynamics, blocking remains a challenge for global weather prediction and climate simulation models. Such modeling deficiencies have negative consequences not only for our ability to represent the observed climate but also for the possibility of producing high-quality seasonal-to-decadal predictions. For such predictions, representing the correct space-time statistics of blocking occurrence is, especially for certain geographical areas, extremely important.

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46

1956-, Pak Chun-sŏng, ed. Wae 80 i 20 ege chibae tanghanŭnʼga?: Chagŭn Chʻaek sŭtʻa ka para pon sesang. Sŏul-si: Chʻŏlsu wa Yŏnghŭi, 2007.

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47

1956-, Pak Chun-sŏng, ed. Wae 80 i 20 ege chibae tanghanŭnʼga?: Chagŭn Chʻaek sŭtʻa ka para pon sesang. Sŏul-si: Chʻŏlsu wa Yŏnghŭi, 2007.

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48

1956-, Pak Chun-sŏng, ed. Wae 80 i 20 ege chibae tanghanŭnʼga?: Chagŭn Chʻaek sŭtʻa ka para pon sesang. Sŏul-si: Chʻŏlsu wa Yŏnghŭi, 2007.

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49

Cabrelli, David. 8. Pay and Working Time. Oxford University Press, 2018. http://dx.doi.org/10.1093/he/9780198813149.003.0008.

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This chapter examines the statutory regulation of the wage–work bargain and the working conditions of ‘employees’ and ‘workers’, analysing their historical background and the justifications for their introduction. It covers the rights conferred on employees and workers under the National Minimum Wage Act 1998 and the Working Time Regulations 1998, including working time rights and the right to annual leave. Both laws have the capacity to over-ride the mutually agreed contractual arrangements struck by the parties. The chapter also addresses the provisions of the Employment Rights Act 1996 relating to wages (e.g. the statutory right not to suffer unauthorized deductions from wages, and the right to a guarantee payment).

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50

Fassbinder, Helga, and Adri Proveniers. New Wave in Building: A Flexible Way of Design, Construction and Real Estate Management / Een Flexibele Methode Voor Ontwerpen, Uitvoeren and Vastgoedbeheer. Van Gorcum and Comp BV, 1992.

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Books on the topic 'EEG wave'

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