Relevant bibliographies by topics / Effect of drusg on

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Effect of drusg on'

Author: Grafiati
Published: 4 June 2021
Last updated: 12 February 2022

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Journal articles on the topic "Effect of drusg on"

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Bocharova, Inna Anatolevna, Vadim Agadzhanov, and Vadim Sagalaev. "Drug addiction. Drugs and their effects on man." Vestnik Volgogradskogo Gosudarstvennogo Universiteta. Serija 11. Estestvennye nauki, no. 2 (December 1, 2013): 22–27. http://dx.doi.org/10.15688/jvolsu11.2013.2.3.

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Slørdal, Lars, and Jarle Aarbakke. "Effect of anticancer drugs on drug metabolism." Pharmacology & Therapeutics 35, no. 1-2 (January 1987): 217–26. http://dx.doi.org/10.1016/0163-7258(87)90107-0.

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Tulegenova, Symbat, Raikhan Beisenova, and Almagul Auelbekova. "The sensitivity of algae to the effect of antifungal drugs." Bulletin of the Karaganda University. “Biology, medicine, geography Series” 97, no. 1 (March 30, 2020): 90–95. http://dx.doi.org/10.31489/2020bmg1/90-95.

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Sharifovna, Xidoyatova Zulfiya, Azimova Nozima Akramovna, and Azimova Munira Takhirovna. "Analysis Of The Assortment Of Drugs With A Sedative Effect." American Journal of Medical Sciences and Pharmaceutical Research 03, no. 01 (January 22, 2021): 81–86. http://dx.doi.org/10.37547/tajmspr/volume03issue01-12.

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A content analysis of the pharmaceutical market range of drugs with sedative effect was conducted. In the analysis of drugs with sedative effect, "West trade", "Floromed", "Grand farm", "Tabletka", "Dava", "Shafran farm", "Glucose", "Tetra", "Kobiljon Obidjon", "Pharmacy diabetes", "Navbahor", "777 pharmacy", "999 pharmacy", "pharmacy Malikabonu" pharmacies, the Department of neurology of the II clinic of the Tashkent Medical Academy, the State Department of medicines and medical products the basis of the register was. Determination of the share of products of manufacturing enterprises in the pharmaceutical market was the main objective of the study carried out.
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Hu, Baofang, Hong Wang, and Zhenmei Yu. "Drug Side-Effect Prediction Via Random Walk on the Signed Heterogeneous Drug Network." Molecules 24, no. 20 (October 11, 2019): 3668. http://dx.doi.org/10.3390/molecules24203668.

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Drug side-effects have become a major public health concern as they are the underlying cause of over a million serious injuries and deaths each year. Therefore, it is of critical importance to detect side-effects as early as possible. Existing computational methods mainly utilize the drug chemical profile and the drug biological profile to predict the side-effects of a drug. In the utilized drug biological profile information, they only focus on drug–target interactions and neglect the modes of action of drugs on target proteins. In this paper, we develop a new method for predicting potential side-effects of drugs based on more comprehensive drug information in which the modes of action of drugs on target proteins are integrated. Drug information of multiple types is modeled as a signed heterogeneous information network. We propose a signed heterogeneous information network embedding framework for learning drug embeddings and predicting side-effects of drugs. We use two bias random walk procedures to obtain drug sequences and train a Skip-gram model to learn drug embeddings. We experimentally demonstrate the performance of the proposed method by comparison with state-of-the-art methods. Furthermore, the results of a case study support our hypothesis that modes of action of drugs on target proteins are meaningful in side-effect prediction.
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Mohd Ali, Yousoff Effendy, Kiam Heong Kwa, and Kurunathan Ratnavelu. "Predicting new drug indications from network analysis." International Journal of Modern Physics C 28, no. 09 (September 2017): 1750118. http://dx.doi.org/10.1142/s0129183117501182.

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This work adapts centrality measures commonly used in social network analysis to identify drugs with better positions in drug-side effect network and drug-indication network for the purpose of drug repositioning. Our basic hypothesis is that drugs having similar phenotypic profiles such as side effects may also share similar therapeutic properties based on related mechanism of action and vice versa. The networks were constructed from Side Effect Resource (SIDER) 4.1 which contains 1430 unique drugs with side effects and 1437 unique drugs with indications. Within the giant components of these networks, drugs were ranked based on their centrality scores whereby 18 prominent drugs from the drug-side effect network and 15 prominent drugs from the drug-indication network were identified. Indications and side effects of prominent drugs were deduced from the profiles of their neighbors in the networks and compared to existing clinical studies while an optimum threshold of similarity among drugs was sought for. The threshold can then be utilized for predicting indications and side effects of all drugs. Similarities of drugs were measured by the extent to which they share phenotypic profiles and neighbors. To improve the likelihood of accurate predictions, only profiles such as side effects of common or very common frequencies were considered. In summary, our work is an attempt to offer an alternative approach to drug repositioning using centrality measures commonly used for analyzing social networks.
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Krishnarao, Joshi Prema, and Kashinath K. Jadhav. "The Effect of Anticholinergic Drug on Thermoregulation in Paediatric Patients." International Physiology 6, no. 2 (2018): 121–25. http://dx.doi.org/10.21088/ip.2347.1506.6218.16.

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Seo, Sukyung, Taekeon Lee, Mi-hyun Kim, and Youngmi Yoon. "Prediction of Side Effects Using Comprehensive Similarity Measures." BioMed Research International 2020 (February 28, 2020): 1–10. http://dx.doi.org/10.1155/2020/1357630.

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Identifying the potential side effects of drugs is crucial in clinical trials in the pharmaceutical industry. The existing side effect prediction methods mainly focus on the chemical and biological properties of drugs. This study proposes a method that uses diverse information such as drug-drug interactions from DrugBank, drug-drug interactions from network, single nucleotide polymorphisms, and side effect anatomical hierarchy as well as chemical structures, indications, and targets. The proposed method is based on the assumption that properties used in drug repositioning studies could be utilized to predict side effects because the phenotypic expression of a side effect is similar to that of the disease. The prediction results using the proposed method showed a 3.5% improvement in the area under the curve (AUC) over that obtained when only chemical, indication, and target features were used. The random forest model delivered outstanding results for all combinations of feature types. Finally, after identifying candidate side effects of drugs using the proposed method, the following four popular drugs were discussed: (1) dasatinib, (2) sitagliptin, (3) vorinostat, and (4) clonidine.
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Song, Min, Seung Han Baek, Go Eun Heo, and Jeong-Hoon Lee. "Inferring Drug-Protein–Side Effect Relationships from Biomedical Text." Genes 10, no. 2 (February 19, 2019): 159. http://dx.doi.org/10.3390/genes10020159.

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Background: Although there are many studies of drugs and their side effects, the underlying mechanisms of these side effects are not well understood. It is also difficult to understand the specific pathways between drugs and side effects. Objective: The present study seeks to construct putative paths between drugs and their side effects by applying text-mining techniques to free text of biomedical studies, and to develop ranking metrics that could identify the most-likely paths. Materials and Methods: We extracted three types of relationships—drug-protein, protein-protein, and protein–side effect—from biomedical texts by using text mining and predefined relation-extraction rules. Based on the extracted relationships, we constructed whole drug-protein–side effect paths. For each path, we calculated its ranking score by a new ranking function that combines corpus- and ontology-based semantic similarity as well as co-occurrence frequency. Results: We extracted 13 plausible biomedical paths connecting drugs and their side effects from cancer-related abstracts in the PubMed database. The top 20 paths were examined, and the proposed ranking function outperformed the other methods tested, including co-occurrence, COALS, and UMLS by P@5-P@20. In addition, we confirmed that the paths are novel hypotheses that are worth investigating further. Discussion: The risk of side effects has been an important issue for the US Food and Drug Administration (FDA). However, the causes and mechanisms of such side effects have not been fully elucidated. This study extends previous research on understanding drug side effects by using various techniques such as Named Entity Recognition (NER), Relation Extraction (RE), and semantic similarity. Conclusion: It is not easy to reveal the biomedical mechanisms of side effects due to a huge number of possible paths. However, we automatically generated predictable paths using the proposed approach, which could provide meaningful information to biomedical researchers to generate plausible hypotheses for the understanding of such mechanisms.
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Inasu, Shiny, Biju Thomas, Satheesh Rao, Amitha Ramesh, and Smitha Shetty. "Antidepressant Drug Effect on Periodontal Status in Chronic Periodontitis Patients." Journal of Health and Allied Sciences NU 06, no. 02 (June 2016): 044–50. http://dx.doi.org/10.1055/s-0040-1708640.

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AbstractPatients with problems related to central nervous system dysfunctions are often treated with psychotropic drugs. These include antipsychotics, antidepressants, mood stabilizers, anticonvulsants, and drugs blocking specific receptors in the brain such as anticholinergics or beta-blockers. However, these medications have serious side effects affecting the oral health. The purpose of this study is to explore antidepressant drug effect in chronic periodontitis patients. Aim: To explore the effect of antidepressant drug in chronic periodontitis patients. Material and Methods: The study comprised of 100 subjects, 50 periodontally healthy subjects, 50 chronic periodontitis subjects.Clinical examination was done and the following parameters were assessed: Gingival index, Clinical Attachment Loss.
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Dissertations / Theses on the topic "Effect of drusg on"

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Chittchang, Montakarn Johnston Thomas P. "Effect of secondary structure on paracellular transport of polypeptides." Diss., UMK access, 2004.

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Thesis (Ph. D.)--School of Pharmacy and Dept. of Chemistry. University of Missouri--Kansas City, 2004.
"A dissertation in pharmaceutical sciences and chemistry." Advisor: Thomas P. Johnston. Typescript. Vita. Description based on contents viewed Feb. 23, 2006; title from "catalog record" of the print edition. Includes bibliographical references (leaves 202-223). Online version of the print edition.
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Dalal, Suntanu. "Amphetamine drugs potentiate morphine analgesia in the formalin test." Thesis, McGill University, 1994. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=55488.

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There has been a great deal of research investigating drug combinations which can increase analgesia. A number of studies have been conducted with one particular combination--opioids combined with the amphetamine drugs. Despite the existing literature, this combination is rarely used in clinical practice. One purpose of this thesis is to review the literature pertaining to the opioid-amphetamine combination. Another purpose of this thesis is to investigate whether dextroamphetamine sulfate ($ circler$Dexedrine) can potentiate morphine sulfate analgesia in rats in the formalin test (Experiment 1). To investigate whether these results can be generalized to another psychostimulant, methylphenidate hydrochloride ($ circler$Ritalin) is used in Experiment 2. Methylphenidate has been chosen instead of another amphetamine drug because it is currently being used in clinical studies without supporting evidence from animal studies. The results of the two experiments indicate that low doses of d-amphetamine and methylphenidate can potentiate the analgesic effects of morphine.
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Church, Donna Lea. "Depot medroxyprogesterone acetate discontinuation after weight gain in 17-19 year old adolescent girls." CSUSB ScholarWorks, 2002. https://scholarworks.lib.csusb.edu/etd-project/2047.

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Depot medroxyprogesterone acetate (DMPA) is a long acting progesterone only contraceptive agent. Side effects such as irregular bleeding patterns and weight gain are attributed to discontinuation. The purpose of this study was to describe depot medroxyprogesterone acetate discontinuation after weight gain in 17 to 19 year-old adolescent girls.
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Heiberg, Ludvig. "An electrophoretic study of fetal mouse brain proteins after in vivo exposure to phenytoin and disulfiram." Master's thesis, University of Cape Town, 1990. http://hdl.handle.net/11427/27187.

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Although there have been two-dimensional electrophoretic studies on fetal brain tissue (for instance, Yoshida and Takahashi, 1980), the emphasis in most of this work has been on developmental changes in protein expression, and not on the effects that drugs have on fetal brain protein complement. Klose and co-workers (1977) did an early study using two-dimensional gel electrophoresis to determine the effects of various teratogens on whole embryos. No protein changes were found and that line of research was not continued. In this study two-dimensional gel electrophoresis is extensively used, in the belief that the usefulness of this technique to experimental teratology has not been fully evaluated. It is reasonable to suppose that a central nervous system teratogen administered during critical periods of susceptibility will led to perturbations of orderly brain development, and that these perturbations will be reflected as changes to the protein complement. The total brain protein complement of mice that have been exposed to drugs in utero will therefore be analysed, in the hope that any inductions or deletions of proteins as a result of drug exposure may provide a clue to the molecular events underlying drug injury to the fetus.
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Goble, David. "The impact of low to moderate alcohol consumption on different types of human performance." Thesis, Rhodes University, 2013. http://hdl.handle.net/10962/d1006042.

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Despite extensive research into the effects of alcohol consumption, there is no clear understanding into the mechanisms underlying human information processing impairment. The acute consumption of alcohol was investigated to determine the implications for human information processing capabilities, and to identify the extent to which these implications were stage-specific. Further aims included the investigation and quantification of caffeine-induced antagonism of alcohol impairment. Moreover, the aforementioned relationships were investigated in morning versus evening conditions. A test battery of six resource-specific tasks was utilised to measure visual perceptual, cognitive and sensory-motor performance, fashioned to return both simple and complex measures of each task. The tasks implemented were: visual perceptual performance (accommodation, visual detection, visual pattern recognition); cognition (memory recall- digit span); and motor output (modified Fitts‟ and a driving simulated line-tracking). Performance measures were recorded by the respective computer based tasks. Physiological variables measured included heart rate frequency, heart rate variability (RMSSD, High and Low Frequency Power) and body temperature. Saccade speed, saccade amplitude, pupil size and fixation duration were the oculomotor parameters measured. Three groups of participants (alcohol, caffeine+alcohol and control) n=36 were studied, split evenly between sexes in a mixed repeated/non-repeated measures design. The control group performed all test batteries under no influence. The alcohol group performed test batteries one and two sober, and three and four under the influence of a 0.4 g/kg dose of alcohol. Group caffeine+alcohol conducted test battery one sober, two under the effect of caffeine only (4 mg/kg), and three and four under the influence of both caffeine and alcohol (0.4 g/kg). The third test battery demonstrated the effects of alcohol during the inclining phase of the blood alcohol curve, and the fourth represented the declining phase. Morning experimentation occurred between 10:00 - 12: 45 and 10:30 -13:15 with evening experimentation between 19:00 - 21:30 and 19:30 - 22:00. Acute alcohol consumption at a dose of approximately 0.4 g/kg body weight effected an average peak breath alcohol concentration of 0.062 % and 0.059 % for the alcohol and caffeine+alcohol groups respectively. Task-related visual perceptual performance demonstrated significant decrements for simple reaction time, choice reaction time and error rate. Cognitive performance demonstrated no significant performance decrements, while motor performance indicated significant decrements in target accuracy only. Physiological parameters in response to alcohol consumption showed significantly decreased heart rate variability (RMSSD) in the modified Fitts‟ task only. A significant decrease in saccade amplitude in the memory task was the only change in oculomotor parameters. Prior caffeine consumption demonstrated limited antagonism to task-related alcohol impairment, significantly improving performance only in reduced error rate while reading. Caffeine consumption showed stimulating effects on physiological parameters, significantly increasing heart rate and heart rate variability when compared to alcohol alone. The design of the tasks allows for comparison between complex and simple task performance, indicating resource utilisation and depletion. Complex tasks demonstrated higher resource utilisation, however with no statistical performance differences to simple tasks. Physiological parameters showed greater change in response to alcohol consumption, than did the performance measures. Alcohol consumption imposed significant changes in physiological and oculomotor parameters for cognitive tasks only, significantly increasing heart rate frequency and decreasing heart rate variability, skin temperature and saccade amplitude. Caffeine consumption showed no antagonism of alcohol-induced performance measures. Physiological measures showed that caffeine consumption imposed stimulating effects in only the neural reflex and memory tasks, significantly increasing heart rate frequency and heart rate variability. Prior caffeine consumption significantly decreased fixation duration in the memory task only. The time of day at which alcohol was consumed demonstrated significant performance and physiological implications. Results indicated that morning consumption of alcohol imposes greater decrements in performance and larger fluctuations in physiological parameters than the decrements in evening experimental sessions. It can be concluded that alcohol consumption at a dose of 0.4 g/kg affects all stages in the information processing chain. Task performance indicates that alcohol has a greater severity on the early stages of information processing. Conversely, under the influence of alcohol an increased task complexity induces greater effects on central stage information processing. In addition, caffeine consumption at a dose of 4 mg/kg prior to alcohol does not antagonise the alcohol-induced performance decrements.
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Bhatnagar, Barkha. "An examination of the effects of ivermectin on Brugia malayi adult worms /." Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=100768.

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Brugia malayi is one of the causative agents of the disabling and disfiguring disease known as Lymphatic Filariasis (LF). This infection is a well-established ailment in tropical and subtropical countries and recently the drug ivermectin has been introduced for the LF control programs. Ivermectin (IVM) is an excellent microfilaricide, but is not markedly macrofilaricidal. However, it causes a long-lasting reduction in the production of new larvae by female worms, suggesting that adult stages are affected. However, the mechanism by which IVM produces such effect in the adult worm is not well understood. One major reason is our incomplete understanding about the biological effect of IVM on adult stages. The present study was carried out to examine the in vitro effects of IVM on B. malayi adult worms using Brugia-gerbil animal model. And also to have some leads in understanding the drug-uptake and location of probable targets in the worm body by using fluorescent labeled IVM and confocal microscopy.
The antifilarial effects of IVM were examined using three parameters: mf release by female worms, and motility, and viability in both male and female worms. The results reported in this study demonstrate that although IVM did not kill the adult worm, but showed significant antifilarial effects on B. malayi adult stages when examined in an in vitro system. Confocal microscopy images of the worms incubated in bodipy FITC-IVM showed strong specific localization signal in the anterior cephalic region of both male and female worms. These observations suggest the early/initial interactions of the drug with its probable receptors that could be located specifically in the head region.
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Kim, Chang Kwon. "The role of the drug-effect contingency in the development of cross tolerence to anticonvulsant drug effects." Thesis, University of British Columbia, 1989. http://hdl.handle.net/2429/28249.

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It was recently demonstrated that tolerance develops to the anticonvulsant effect of ethanol on kindled convulsions elicited in rats by electrical stimulation of the amygdala, following each of a series of ethanol injections delivered on a bidaily schedule (once every 48 hr). The tolerance developed only when the convulsive stimulation was administered during the periods of ethanol exposure: subjects that received ethanol 1.5 hr before each convulsive stimulation demonstrated tolerance after just five ethanol injections; whereas, no tolerance was evident in subjects that received ethanol 1.5 hr after each stimulation. Such tolerance, which is not the inevitable product of drug exposure but is contingent upon the expression of the drug effect--the anticonvulsant effect in this case--has been termed contingent tolerance. In Experiment 1A, tolerance developed to the anticonvulsant effects of bidaily IP injections of phenobarbital (30 mg/kg), trimethadione (270 mg/kg) and clonazepam (0.40 or 0.35 mg/kg) delivered 1 hr before each convulsive stimulation. In Experiment IB, the rats tolerant to the anticonvulsant effects of phenobarbital, trimethadione, or clonazepam received bidaily injections of carbamazepine (35 mg/kg, IP), administered 1 hr before each stimulation. There was a statistically significant transfer of tolerance from phenobarbital to carbamazepine, but not from either trimethadione or clonazepam to carbamazepine. Thus, cross tolerance appears to be greatest between anticonvulsant drugs that are effective against a similar profile of clinical and experimental seizures and that have similar mechanisms of action. In Experiment 2A, tolerance developed to the anticonvulsant effect of bidaily pentobarbital (15 mg/kg, IP) injections only in those rats that received the drug 1 hr before the convulsive stimulation, but not in those rats that had received the drug 1 hr after each stimulation. Furthermore, those rats that had received the convulsive stimulations while under the influence of pentobarbital subsequently displayed a greater degree of cross tolerance to the anticonvulsant effect of ethanol (1.5 g/kg, IP) than those that had received the drug after each stimulation (contingent cross tolerance). Experiment 2B was the converse of Experiment 2A: contingent tolerance was demonstrated to ethanol and contingent cross tolerance to pentobarbital. This study provided the first unambiguous and bidirectional demonstration that the drug-effect contingency plays an important role in the development of cross tolerance. In Experiment 3, tolerance to the anticonvulsant effect of ethanol dissipated when bidaily pentobarbital (15 mg/kg, IP) injections were delivered 1 hr after each convulsive stimulation (contingent cross-dissipation of tolerance), but did not dissipate when it was delivered 1 hr before each stimulation. Thus, the drug-effect contingency was shown to be important in the dissipation of tolerance to one drug following the administration of another drug. In Experiment 4, different groups of rats received different doses of pentobarbital (10-50 mg/kg, IP) on a bidaily schedule 1 hr before the convulsive stimulation. Greater tolerance was found to the anticonvulsant effect of pentobarbital in rats that had received successively larger doses of the drug, none of which were large enough to suppress the convulsions, than those rats that were maintained on a high dose of the drug that completely suppressed the convulsions. The greater tolerance in the group that received successively greater doses was attributed to the fact that the convulsions were experienced in the drugged state. This study challenged the generally accepted view that tolerance develops more rapidly and to a greater extent with larger drug doses . This thesis provides the first unambiguous and systematic evidence of the role of the drug-effect contingency in the transfer of tolerance from one drug to another, and in the dissipation of tolerance to one drug following the administration of another. On the basis of the present experiments, several elaborations were proposed to the drug-effect theory of drug tolerance, which claims that tolerance develops to drug effects and not to drug exposure per se.
Arts, Faculty of
Psychology, Department of
Graduate
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Chawla, Monica Kapoor 1950. "THE ROLE OF SEVERAL DRUGS AND COSOLVENTS ON INFUSION RELATED PHLEBITIS (THERMOGRAPHY)." Thesis, The University of Arizona, 1986. http://hdl.handle.net/10150/276915.

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Weise-Kelly, Lorraine Ann. "Drug-induced ataxia : effect of the self-administration contingency /." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape10/PQDD_0030/NQ66245.pdf.

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Gabriel, Kara Irene. "Effects of prenatal ethanol exposure and postnatal handling on cognition/behavior and hypothalamic-pituitary-adrenal stress responsiveness in Sprague-Dawley rats." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ56547.pdf.

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Books on the topic "Effect of drusg on"

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Fraser, Suzanne, and David Moore, eds. The Drug Effect. Cambridge: Cambridge University Press, 2011. http://dx.doi.org/10.1017/cbo9781139162142.

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Prebble, Lucy. The effect. London: Methuen Drama, 2012.

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Drug actions and interactions. New York: McGraw-Hill Medical, 2011.

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Aronson, J. K. Side effects of drugs annual 28: A worldwide yearly survey of new data and trends in adverse drug reactions and interactions. Amsterdam, Netherlands: Elsevier, 2005.

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Side effects of drugs annual: A worldwide yearly survey of new data in adverse drug reactions. Amsterdam: Elsevier, 2011.

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Levinthal, Charles F. Drugs, behavior, and modern society. 7th ed. Boston: Allyn & Bacon, 2012.

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National Institutes of Health (U.S.). Clinical Center, ed. Drugs and the brain. Bethesda, Md. (9000 Rockville Pike, Bethesda 20892): National Institutes of Health, The Clinical Center, 1993.

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Friedman, David P. Drugs and the brain. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1991.

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J, Collard Keith, ed. Endocrine and metabolic effects of lithium. New York: Plenum Medical Book Co., 1986.

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1963-, Moore David, ed. The drug effect: Health, crime and society. Cambridge: Cambridge University Press, 2011.

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Book chapters on the topic "Effect of drusg on"

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Schäfer, H. "Chemical Constitution and Pharmacological Effect." In Antiepileptic Drugs, 199–243. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-69518-6_9.

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Mattison Faye, Amy C. M., and Donald R. Mattison. "Drug Disposition and Effect." In Handbook of Clinical Gender Medicine, 473–79. Basel: KARGER, 2012. http://dx.doi.org/10.1159/000336450.

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Klebe, Gerhard. "Optical Activity and Biological Effect." In Drug Design, 89–110. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-17907-5_5.

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Holford, N. H. G., and T. M. Ludden. "Time Course of Drug Effect." In Pharmacokinetics of Drugs, 333–52. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78680-8_11.

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Martin, X. "Diabetogenic effect of immunosuppressive drugs." In Immunosuppression under Trial, 101–3. Dordrecht: Springer Netherlands, 1999. http://dx.doi.org/10.1007/978-94-011-4643-2_12.

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Gralinski, Michael, Liomar A. A. Neves, and Olga Tiniakova. "Effect of Different Peptides." In Drug Discovery and Evaluation: Pharmacological Assays, 1–45. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27728-3_147-1.

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Gralinski, Michael, Liomar A. A. Neves, and Olga Tiniakova. "Effect of Different Peptides." In Drug Discovery and Evaluation: Pharmacological Assays, 513–53. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-05392-9_147.

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Simola, Nicola, Micaela Morelli, Tooru Mizuno, Suzanne H. Mitchell, Harriet de Wit, H. Valerie Curran, Celia J. A. Morgan, et al. "Drug (Dose)-Effect Function (Curve)." In Encyclopedia of Psychopharmacology, 430. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_1086.

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Deth, Richard C. "The Effect of Drugs on Attention." In Molecular Origins of Human Attention, 111–24. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0335-4_10.

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Simpson, N. B. "The Effect of Drugs on Hair." In Pharmacology of the Skin II, 495–508. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74054-1_37.

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Conference papers on the topic "Effect of drusg on"

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Balachandran, Ram K., and Victor H. Barocas. "Effect of Active Transport and Loss to Choroidal Blood Flow on Transscleral Drug Delivery to the Posterior Eye." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-175366.

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Delivering drugs to the posterior eye has been a challenge for many years. Systemic delivery of drugs is not a viable option because the eye does not receive enough blood supply, because of its small size, for the drug delivery process to be effective. Topical delivery in the form of eye drops is also ineffective in generating therapeutic concentrations in the posterior eye, because of the resistance offered by the corneal epithelium to the transport of drugs, and rapid elimination due to aqueous humor flow and tear dilution. Intravitreal delivery of drugs through implants and injections has been associated with serious side effects like endophthalmitis, hemorrhage, and retinal detachment. In recent years, transcleral delivery of drugs has received attention due to the relatively high permeability of the sclera.
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Hwang, Sohee, Jungrim Kim, and Sanghyun Park. "Recommend alternative drugs to minimize side-effect using generic name of drug." In EDB: 2016 International Conference on Emerging Databases. New York, NY, USA: ACM, 2016. http://dx.doi.org/10.1145/3007818.3007846.

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Nikkhah, Mehdi, Jeannine S. Strobl, and Masoud Agah. "Study the Effect of Anticancer Drugs on Human Breast Cancer Cells Using Three Dimensional Silicon Microstructures." In ASME 2008 International Mechanical Engineering Congress and Exposition. ASMEDC, 2008. http://dx.doi.org/10.1115/imece2008-66680.

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In this paper we report development of three dimensional silicon microenvironments in order to test the morphological changes and adhesion properties of human breast cancer cells after treatment with different anticancer drugs such as Trichostatin A (TSA), suberoylanilide hydroxamic acid (SAHA) and Scriptaid. Our results indicate that the cancer cells reorganize their cytoskeleton structure after treatment with TSA and Scriptaid. However, SAHA does not change the behavior of the cells inside the three dimensional microstructures while TSA and Scriptaid evoked striking changes in the cells morphology. TSA and Scriptaid drugs cause the cells to stretch inside the isotropic microchambers to avoid contact with curved sidewalls in contrast to their originally rounded shape. The proposed microstructures can be used to evaluate mechanical properties and the pathological grade of various cancer cell lines after different conditions i.e. drug exposure.
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Madhav, Bhumika, Aparna Iyer, and T. K. Jayalakshmi. "Side effect profile of 2ndline drugs in multi drug resistant (MDR) and extensively drug resistant (XDR) tuberculosis." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa2708.

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Kazantsev, S. O., and M. S. Korovin. "Nanomaterials potentiating standard chemotherapy drugs’ effect." In PHYSICS OF CANCER: INTERDISCIPLINARY PROBLEMS AND CLINICAL APPLICATIONS: Proceedings of the International Conference on Physics of Cancer: Interdisciplinary Problems and Clinical Applications (PC IPCA’17). Author(s), 2017. http://dx.doi.org/10.1063/1.5001610.

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Poliakov, V. V., D. S. Mokshin, A. E. Kalenova, Z. A. Koshchugulova, N. Sadyrbekuly, and O. N. Chikunova. "Radioprotective effect of the drug "Topolin"." In Наука России: Цели и задачи. НИЦ «Л-Журнал», 2018. http://dx.doi.org/10.18411/sr-10-12-2018-26.

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Sun, Chengcheng, Yi Zheng, Yan Jia, and Liang Gan. "Drug Side-effect Prediction based on Comprehensive Drug Similarity." In 2016 International Forum on Mechanical, Control and Automation (IFMCA 2016). Paris, France: Atlantis Press, 2017. http://dx.doi.org/10.2991/ifmca-16.2017.28.

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Raugale, A., P. Gaidelis, and A. Januskevicius. "New researches on teratogenic effect of drugs." In SPIE Proceedings, edited by Leonardo Longo, Alfons G. Hofstetter, Mihail-Lucian Pascu, and Wilhelm R. A. Waidelich. SPIE, 2004. http://dx.doi.org/10.1117/12.584335.

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Zarandi, Marjan Molavi, Rosaire Mongrain, and Olivier F. Bertrand. "Modeling Drug Eluting Stents for Coronary Artery Bifurcation Considering Non-Newtonian Effects." In ASME 2010 3rd Joint US-European Fluids Engineering Summer Meeting collocated with 8th International Conference on Nanochannels, Microchannels, and Minichannels. ASMEDC, 2010. http://dx.doi.org/10.1115/fedsm-icnmm2010-31190.

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Drug Eluting Stents (DES) are commonly used for the treatment of stenotic arteries. Restenosis can be treated by delivering anti-thrombotic and anti-proliferative drugs to the arterial wall. The main mechanism of the drug eluting stent is to allow diffusion of the drug from the coating on the stent, into the arterial wall over a prolonged period of time. Investigation of blood flow hemodynamics and shear stress are of great importance in understanding the transport of drugs through the circulatory systems and predicting the performance of drug eluting stents. While drug eluting stent effectively reduces restenosis rate, the conventional drug eluting stent should be optimized to be used in the bifurcation stenting. Various flow patterns due to specific designs of drug eluting stent influence drug delivery. Numerical simulation techniques are appropriate approaches to study such phenomena which can be used to optimize the design of drug eluting stents for bifurcations. In this paper, the complexity of drug eluting stent function in the bifurcation is presented by employing computational fluid dynamics analysis for various stent strut designs. Drug transportation through the lumen and determination of local drug concentrations in arterial wall is carried out for both Newtonian and non-Newtonian flow conditions. It is, to the author’s best knowledge, the first investigation of drug dispersion in arterial bifurcation considering the effects of both the blood rheological properties and stent strut design.
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Kayastha, Tanay, Pranjal Gupta, and Pushpak Bhattacharyya. "BERT based Adverse Drug Effect Tweet Classification." In Proceedings of the Sixth Social Media Mining for Health (#SMM4H) Workshop and Shared Task. Stroudsburg, PA, USA: Association for Computational Linguistics, 2021. http://dx.doi.org/10.18653/v1/2021.smm4h-1.15.

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Reports on the topic "Effect of drusg on"

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Kamimori, Gary H. Effect of Time of Menstrual Cycle on Drug Pharmacokinetics and Pharmacodynamics. Fort Belvoir, VA: Defense Technical Information Center, May 2001. http://dx.doi.org/10.21236/ada409105.

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Lichtenberg, Frank. The Effect of New Drugs on Mortality from Rare Diseases and HIV. Cambridge, MA: National Bureau of Economic Research, December 2001. http://dx.doi.org/10.3386/w8677.

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Miron, Jeffrey. The Effect of Drug Prohibition on Drug Prices: Evidence from the Markets for Cocaine and Heroin. Cambridge, MA: National Bureau of Economic Research, May 2003. http://dx.doi.org/10.3386/w9689.

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Buchmueller, Thomas, and Colleen Carey. The Effect of Prescription Drug Monitoring Programs on Opioid Utilization in Medicare. Cambridge, MA: National Bureau of Economic Research, February 2017. http://dx.doi.org/10.3386/w23148.

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Kaestner, Robert. The Effect of Illicit Drug Use on the Wages of Young Adults. Cambridge, MA: National Bureau of Economic Research, December 1990. http://dx.doi.org/10.3386/w3535.

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Wen, Hefei, Jason Hockenberry, and Janet Cummings. The Effect of Medical Marijuana Laws on Marijuana, Alcohol, and Hard Drug Use. Cambridge, MA: National Bureau of Economic Research, May 2014. http://dx.doi.org/10.3386/w20085.

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Khan, Nasreen, Robert Kaestner, and Swu Jane Lin. Prescription Drug Insurance and Its Effect on Utilization and Health of the Elderly. Cambridge, MA: National Bureau of Economic Research, January 2007. http://dx.doi.org/10.3386/w12848.

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Kaestner, Robert. The Effect of Illicit Drug Use on the Labor Supply of Young Adults. Cambridge, MA: National Bureau of Economic Research, October 1992. http://dx.doi.org/10.3386/w4187.

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Lichtenberg, Frank. The Effect of Changes in Drug Utilization on Labor Supply and Per Capita Output. Cambridge, MA: National Bureau of Economic Research, September 2002. http://dx.doi.org/10.3386/w9139.

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Lichtenberg, Frank. The Effect of Drug Vintage on Survival: Micro Evidence from Puerto Rico's Medicaid Program. Cambridge, MA: National Bureau of Economic Research, November 2004. http://dx.doi.org/10.3386/w10884.

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