Academic literature on the topic 'EGF-TM7 receptor, GC-rich region, Transcription factor'

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'EGF-TM7 receptor, GC-rich region, Transcription factor.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "EGF-TM7 receptor, GC-rich region, Transcription factor"

1

Johnson, A. C., Y. Jinno, and G. T. Merlino. "Modulation of epidermal growth factor receptor proto-oncogene transcription by a promoter site sensitive to S1 nuclease." Molecular and Cellular Biology 8, no. 10 (1988): 4174–84. http://dx.doi.org/10.1128/mcb.8.10.4174.

Full text
Abstract:
The epidermal growth factor (EGF) receptor is the functional target of the mitogen EGF and the cellular homolog of the avian erythroblastosis virus erbB oncogene product. Regulation of expression of the proto-oncogene encoding the EGF receptor can be elucidated by studying the structure and function of the gene promoter outside the confines of the cell. Previously, we reported the isolation of the human EGF receptor gene promoter. The promoter is highly GC rich, contains no TATA or CAAT box, and has multiple transcription start sites. An S1 nuclease-sensitive site has now been found 80 to 110
APA, Harvard, Vancouver, ISO, and other styles
2

Johnson, A. C., Y. Jinno, and G. T. Merlino. "Modulation of epidermal growth factor receptor proto-oncogene transcription by a promoter site sensitive to S1 nuclease." Molecular and Cellular Biology 8, no. 10 (1988): 4174–84. http://dx.doi.org/10.1128/mcb.8.10.4174-4184.1988.

Full text
Abstract:
The epidermal growth factor (EGF) receptor is the functional target of the mitogen EGF and the cellular homolog of the avian erythroblastosis virus erbB oncogene product. Regulation of expression of the proto-oncogene encoding the EGF receptor can be elucidated by studying the structure and function of the gene promoter outside the confines of the cell. Previously, we reported the isolation of the human EGF receptor gene promoter. The promoter is highly GC rich, contains no TATA or CAAT box, and has multiple transcription start sites. An S1 nuclease-sensitive site has now been found 80 to 110
APA, Harvard, Vancouver, ISO, and other styles
3

Encarnacao, Priscilla C., Vincent P. Ramirez, Carmen Zhang та Brian J. Aneskievich. "Sp sites contribute to basal and inducible expression of the human TNIP1 (TNFα-inducible protein 3-interacting protein 1) promoter". Biochemical Journal 452, № 3 (2013): 519–29. http://dx.doi.org/10.1042/bj20121666.

Full text
Abstract:
TNIP1 [TNFα (tumour necrosis factor α)-induced protein 3-interacting protein 1] is a co-repressor of RAR (retinoic acid receptor) and PPAR (peroxisome-proliferator-activated receptor). Additionally, it can reduce signalling stemming from cell membrane receptors such as those for TNFα and EGF (epidermal growth factor). Consequently, it influences a variety of receptor-mediated events as diverse as transcription, programmed cell death and cell cycling. Thus changes in TNIP1 expression levels are likely to affect multiple important biological end points. TNIP1 expression level changes have been l
APA, Harvard, Vancouver, ISO, and other styles
4

Zhang, Zhiping, and Christina T. Teng. "Methoxychlor stimulates the mouse lactoferrin gene promoter through a GC-rich element." Biochemistry and Cell Biology 80, no. 1 (2002): 23–26. http://dx.doi.org/10.1139/o01-177.

Full text
Abstract:
The lactoferrin gene in the mouse uterus is a target gene for natural estrogens and xenoestrogens. One of the xenoestrogens is methyoxychlor, an insecticide that displays both estrogenic and antiandrogenic activities. Recently, methyoxychlor was found to stimulate lactoferrin gene expression in the uterus of an estrogen receptor null mouse. The present study is designed to uncover the methoxychlor response region in the mouse lactoferrin gene promoter. A series of different lengths of the mouse lactoferrin gene 5' flanking region were linked to a chloramphenicol acetyltransferase (CAT) reporte
APA, Harvard, Vancouver, ISO, and other styles
5

Salvatori, Luisa, Linda Ravenna, Maria Pia Felli, et al. "Identification of an Estrogen-Mediated Deoxyribonucleic Acid-Binding Independent Transactivation Pathway on the Epidermal Growth Factor Receptor Gene Promoter*." Endocrinology 141, no. 6 (2000): 2266–74. http://dx.doi.org/10.1210/endo.141.6.7521.

Full text
Abstract:
Abstract To investigate the estrogenic effects on the transcriptional regulation of the epidermal growth factor (EGF) receptor (EGFR) gene, we assayed its promoter ability to direct transcription of the luciferase reporter gene after transfection into HeLa cells. Our studies demonstrated a dose-dependent activation of the EGFR gene transcription by ligand-bound estrogen receptor α (ERα). This action was retained by the 36-bp core promoter fragment and did not require the receptor DNA binding domain, as demonstrated by analyzing the role of ERα deletion mutants on EGFR gene promoter-derived con
APA, Harvard, Vancouver, ISO, and other styles

Dissertations / Theses on the topic "EGF-TM7 receptor, GC-rich region, Transcription factor"

1

Wobus, Manja, Elke Wandel, Sonja Prohaska, Sven Findeiß, Katrin Tschöp, and Gabriela Aust. "Transcriptional regulation of the human CD97 promoter by Sp1/Sp3 in smooth muscle cells." 2008. https://ul.qucosa.de/id/qucosa%3A31891.

Full text
Abstract:
The EGF-TM7 receptor CD97 shows different features of expression and function in muscle cells compared to hematopoetic and tumor cells. Since the molecular function and regulation of CD97 are poorly understood, this study aimed at defining its basal transcriptional regulation in smooth muscle cells (SMCs). The computational analysis of the CD97 5′-flanking region revealed that the TATA box-lacking promoter possesses several GC-rich regions as putative Sp1/Sp3 binding sites. Transfection studies with serially deleted promoter constructs demonstrated that the minimal promoter fragment resided i
APA, Harvard, Vancouver, ISO, and other styles
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!