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Relevant bibliographies by topics / EHC-93

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Academic literature on the topic 'EHC-93'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 February 2022

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Journal articles on the topic "EHC-93"

1

Boyd, Nathan, Hugh Brock, Albert Meier, Richard Miller, Gary Mlady, and Keikhosrow Firoozbakhsh. "Extensor Hallucis Capsularis: Frequency and Identification on MRI." Foot & Ankle International 27, no. 3 (2006): 181–84. http://dx.doi.org/10.1177/107110070602700305.

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Background: The extensor hallucis capsularis (EHC) is the most common name given to the accessory tendon sporadically seen medial to the extensor hallucis longus (EHL). We performed cadaver dissections and MRI evaluation to determine the frequency of its occurrence, the pattern of its origin and insertion, and its potential suitability as tendon graft. Methods: The EHC was examined by dissection in 81 cadaver feet. Physical parameters pertaining to EHC size and location were recorded. MRI was performed on six cadaver legs to determine if the EHC can be identified radiographically. MRI images w

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Kooter, I. M., J. L. A. Pennings, A. Opperhuizen, and F. R. Cassee. "Gene Expression Pattern in Spontaneously Hypertensive Rats Exposed to Urban Particulate Matter (EHC-93)." Inhalation Toxicology 17, no. 1 (2005): 53–65. http://dx.doi.org/10.1080/08958370590885717.

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3

Bissell, Richard A., and Jean Conover. "International Emergency Health Care Systems Survey." Prehospital and Disaster Medicine 6, no. 2 (1991): 149–58. http://dx.doi.org/10.1017/s1049023x00028272.

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AbstractMedical emergencies occur in every country regardless of its level of socio-economic development. Little comparative data are available which define the characteristics of the system by which some emergencies are managed. Without such comparisons, it is difficult for countries to establish appropriate priorities within their geographic, cultural, and economic constraints. In an effort to gather some of these needed data, a survey was distributed to the participants in an International Conference on Emergency Health Care (EHC) Development convened in Washington, D. C, in August 1989. Ea

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Mondaca, Sebastian, Pedram Razavi, Chongrui Xu, et al. "Genomic Characterization of ERBB2-Driven Biliary Cancer and a Case of Response to Ado-Trastuzumab Emtansine." JCO Precision Oncology, no. 3 (December 2019): 1–9. http://dx.doi.org/10.1200/po.19.00223.

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PURPOSE Biliary tract cancers (BTCs), which include intrahepatic cholangiocarcinoma (ICC), extrahepatic cholangiocarcinoma (EHC), and gallbladder cancer (GBC), have limited treatment options. We sought to comprehensively examine the clinical and molecular characteristics of BTCs with amplification or mutation of ERBB2. METHODS Demographic, outcome, and treatment response data were collected for patients with ERBB2-altered BTC identified by next-generation sequencing with Memorial Sloan Kettering-Integrated Mutation Profiling of Actionable Cancer Targets from 2014 to 2018. RESULTS A total of 51

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Sakamoto, Noriho, Shizu Hayashi, Hiroshi Mukae, Renaud Vincent, James C. Hogg, and Stephan F. van Eeden. "Effect of Atorvastatin on PM10-induced Cytokine Production by Human Alveolar Macrophages and Bronchial Epithelial Cells." International Journal of Toxicology 28, no. 1 (2009): 17–23. http://dx.doi.org/10.1177/1091581809333140.

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Exposure to ambient air pollution particles (PM10) has been associated with increased cardiovascular morbidity and mortality. Inhaled pollutants induce a pulmonary and systemic inflammatory response that is thought to exacerbate cardiovascular disease. The 3-hydroxy-3-methylglutaryl coenzyme A (HMG-CoA) reductase inhibitors (statins) have been shown to have anti-inflammatory effects that could contribute to their beneficial effect in cardiovascular disease. The aim of this study is to determine the effects of statins on PM10-induced cytokine production in human bronchial epithelial cells (HBEC

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Yatera, Kazuhiro, Joanne Hsieh, James C. Hogg, et al. "Particulate matter air pollution exposure promotes recruitment of monocytes into atherosclerotic plaques." American Journal of Physiology-Heart and Circulatory Physiology 294, no. 2 (2008): H944—H953. http://dx.doi.org/10.1152/ajpheart.00406.2007.

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Epidemiologic studies have shown an association between exposure to ambient particulate air pollution <10 μm in diameter (PM10) and increased cardiovascular morbidity and mortality. We previously showed that PM10 exposure causes progression of atherosclerosis in coronary arteries. We postulate that the recruitment of monocytes from the circulation into atherosclerotic lesions is a key step in this PM10-induced acceleration of atherosclerosis. The study objective was to quantify the recruitment of circulating monocytes into vessel walls and the progression of atherosclerotic plaques induced

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Kumarathasan, P., E. Blais, P. Goegan та ін. "90-Day Repeated Inhalation Exposure of Surfactant Protein-C/Tumor Necrosis Factor-α (SP-C/TNF-α) Transgenic Mice to Air Pollutants". International Journal of Toxicology 24, № 1 (2005): 59–67. http://dx.doi.org/10.1080/10915810590921379.

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Tumor necrosis factor (TNF)- α, a cytokine present in inflammed lungs, is known to mediate some of the adverse effects of ozone and inhaled particles. The authors evaluated transgenic mice with constitutive pulmonary expression of TNF- αunder transcriptional regulation of the surfactant protein-C promoter as an animal model of biological susceptibility to air pollutants. To simulate a repeated, episodic exposure to air pollutants, wild-type and TNF mice inhaled air or a mixture of ozone (0.4 ppm) and urban particles (EHC-93, 4.8 mg/m3) for 4 h, once per week, for 12 consecutive weeks and were

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Dissertations / Theses on the topic "EHC-93"

1

Vuong, Ngoc Quang. "Fingerprinting the effect of airborne particulate matter via in vitro toxicoproteomics." Thesis, Université d'Ottawa / University of Ottawa, 2017. http://hdl.handle.net/10393/36939.

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It is a challenge to assess the toxicity of environmental air particulate matter (PM) because PM composition is complex and variable, due to source contribution and atmospheric transformation. The goal of this study is to establish an in vitro model that can fingerprint the cytotoxic effects of airborne PM and their associated toxicity mechanisms. For this purpose, the cytotoxic effects of different reference and environmental particles on A549 human lung epithelial cells were characterized using multiple endpoint assays (cytokine release, LDH release, BrdU incorporation, cellular ATP and resa

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