Relevant bibliographies by topics / EIF3j

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers
  5. Reports

Academic literature on the topic 'EIF3j'

Author: Grafiati
Published: 9 March 2023

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Journal articles on the topic "EIF3j"

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Herrmannová, Anna, Terezie Prilepskaja, Susan Wagner, et al. "Adapted formaldehyde gradient cross-linking protocol implicates human eIF3d and eIF3c, k and l subunits in the 43S and 48S pre-initiation complex assembly, respectively." Nucleic Acids Research 48, no. 4 (2019): 1969–84. http://dx.doi.org/10.1093/nar/gkz1185.

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Abstract One of the key roles of the 12-subunit eukaryotic translation initiation factor 3 (eIF3) is to promote the formation of the 43S and 48S pre-initiation complexes (PICs). However, particular contributions of its individual subunits to these two critical initiation reactions remained obscure. Here, we adapted formaldehyde gradient cross-linking protocol to translation studies and investigated the efficiency of the 43S and 48S PIC assembly in knockdowns of individual subunits of human eIF3 known to produce various partial subcomplexes. We revealed that eIF3d constitutes an important inter
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Sokabe, Masaaki, and Christopher S. Fraser. "A helicase-independent activity of eIF4A in promoting mRNA recruitment to the human ribosome." Proceedings of the National Academy of Sciences 114, no. 24 (2017): 6304–9. http://dx.doi.org/10.1073/pnas.1620426114.

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In the scanning model of translation initiation, the decoding site and latch of the 40S subunit must open to allow the recruitment and migration of messenger RNA (mRNA); however, the precise molecular details for how initiation factors regulate mRNA accommodation into the decoding site have not yet been elucidated. Eukaryotic initiation factor (eIF) 3j is a subunit of eIF3 that binds to the mRNA entry channel and A-site of the 40S subunit. Previous studies have shown that a reduced affinity of eIF3j for the 43S preinitiation complex (PIC) occurs on eIF4F-dependent mRNA recruitment. Because eIF
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Nielsen, Klaus H., Leos Valášek, Caroah Sykes, Antonina Jivotovskaya, and Alan G. Hinnebusch. "Interaction of the RNP1 Motif in PRT1 with HCR1 Promotes 40S Binding of Eukaryotic Initiation Factor 3 in Yeast." Molecular and Cellular Biology 26, no. 8 (2006): 2984–98. http://dx.doi.org/10.1128/mcb.26.8.2984-2998.2006.

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ABSTRACT We found that mutating the RNP1 motif in the predicted RRM domain in yeast eukaryotic initiation factor 3 (eIF3) subunit b/PRT1 (prt1-rnp1) impairs its direct interactions in vitro with both eIF3a/TIF32 and eIF3j/HCR1. The rnp1 mutation in PRT1 confers temperature-sensitive translation initiation in vivo and reduces 40S-binding of eIF3 to native preinitiation complexes. Several findings indicate that the rnp1 lesion decreases recruitment of eIF3 to the 40S subunit by HCR1: (i) rnp1 strongly impairs the association of HCR1 with PRT1 without substantially disrupting the eIF3 complex; (i
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Wei, Li, Lijun Liu, Lin Chen, et al. "Interfering Eukaryotic Translation Initiation Factor 3 Subunit J Antisense RNA1 Inhibits the Proliferation, Migration, and Invasion of Lung Cancer A549 Cells by Regulating microRNA-330-5p." Nanoscience and Nanotechnology Letters 12, no. 9 (2020): 1099–105. http://dx.doi.org/10.1166/nnl.2020.3208.

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This study investigated whether long noncoding RNA interfering EIF3J antisense RNA1 (EIF3JAS1) could affect lung cancer A549 cells as well as the role of microRNA-330-5p (miR-330-5p) during this process. To this end, quantitative real-time polymerase chain reaction was used to measure EIF3J-AS1 and miR-330-5p expression in 39 lung cancer cases. Small interfering RNA targeting EIF3J-AS1 (si-EIF3J-AS1), as well as the miR-330-5p inhibitor, was transfected into lung cancer A549 cells. The outcomes of cell proliferation, clone formation, migration, invasion, and E- and N-cadherin expression were a
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Luo, Yuhao, Rui Zhou, Na Huang, Li Sun, and Wangjun Liao. "Effect of long non-coding RNA EIF3J-AS1 on multi-drug resistance and autophagy in gastric cancer." Journal of Clinical Oncology 35, no. 15_suppl (2017): e15581-e15581. http://dx.doi.org/10.1200/jco.2017.35.15_suppl.e15581.

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e15581 Background: Gastric cancer (GC) is a leading cause of cancer mortality worldwide, oxaliplatin and epirubicin based chemotherapy are one of the most important treatment options for GC patients. However, drug resistance, especially multi-drug resistance remains a major obstacle for successful chemotherapy. Recently, long non-coding RNAs (lncRNAs) have been widely identified to play emerging roles in diverse physiological and pathophysiological processes including drug resistance. Our previous bioinformatics analysis showed long non-coding RNA EIF3J-AS1 was a potential multi-drug resistanc
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ElAntak, Latifa, Andreas G. Tzakos, Nicolas Locker, and Peter J. Lukavsky. "Structure of eIF3b RNA Recognition Motif and Its Interaction with eIF3j." Journal of Biological Chemistry 282, no. 11 (2006): 8165–74. http://dx.doi.org/10.1074/jbc.m610860200.

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Aylett, Christopher H. S., Daniel Boehringer, Jan P. Erzberger, Tanja Schaefer, and Nenad Ban. "Structure of a Yeast 40S–eIF1–eIF1A–eIF3–eIF3j initiation complex." Nature Structural & Molecular Biology 22, no. 3 (2015): 269–71. http://dx.doi.org/10.1038/nsmb.2963.

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Chen, Xin, Zuyuan Yang, Chao Yang, Kan Xie, Weijun Sun, and Shengli Xie. "Sparse Gene Coexpression Network Analysis Reveals EIF3J-AS1 as a Prognostic Marker for Breast Cancer." Complexity 2018 (June 12, 2018): 1–12. http://dx.doi.org/10.1155/2018/1656273.

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Predictive and prognostic biomarkers facilitate the selection of treatment strategies that can improve the survival of patients. Accumulating evidence indicates that long noncoding RNAs (lncRNAs) play important roles in cancer progression, with diagnostic and prognostic potential. However, few prognostic lncRNAs are reported for breast cancer, and little is known about their functions that contribute to cancer pathogenesis. In this paper, we used weighted correlation network analysis (WGCNA) to construct networks containing noncoding and protein-coding genes based on their expression in 1097 b
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Chiu, Wen-Ling, Susan Wagner, Anna Herrmannová, et al. "The C-Terminal Region of Eukaryotic Translation Initiation Factor 3a (eIF3a) Promotes mRNA Recruitment, Scanning, and, Together with eIF3j and the eIF3b RNA Recognition Motif, Selection of AUG Start Codons." Molecular and Cellular Biology 30, no. 18 (2010): 4415–34. http://dx.doi.org/10.1128/mcb.00280-10.

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ABSTRACT The C-terminal domain (CTD) of the a/Tif32 subunit of budding yeast eukaryotic translation initiation factor 3 (eIF3) interacts with eIF3 subunits j/Hcr1 and b/Prt1 and can bind helices 16 to 18 of 18S rRNA, suggesting proximity to the mRNA entry channel of the 40S subunit. We have identified substitutions in the conserved Lys-Glu-Arg-Arg (KERR) motif and in residues of the nearby box6 element of the a/Tif32 CTD that impair mRNA recruitment by 43S preinitiation complexes (PICs) and confer phenotypes indicating defects in scanning and start codon recognition. The normally dispensable C
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Borgo, Christian, Cinzia Franchin, Valentina Salizzato, et al. "Protein kinase CK2 potentiates translation efficiency by phosphorylating eIF3j at Ser127." Biochimica et Biophysica Acta (BBA) - Molecular Cell Research 1853, no. 7 (2015): 1693–701. http://dx.doi.org/10.1016/j.bbamcr.2015.04.004.

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Dissertations / Theses on the topic "EIF3j"

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Borgo, Christian. "Coinvolgimento della proteinchinasi CK2 nella resistenza all'imatinib in cellule di leucemia mieloide cronica." Doctoral thesis, Università degli studi di Padova, 2010. http://hdl.handle.net/11577/3421631.

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Chronic myeloid leukemia (CML) is a clonal myeloproliferative disorder of hematopoietic stem cells, characterized by clonal expansion of a primitive pluripotent stem cell with the consequence of the increasing number of granulocytes in the blood. A hallmark of CML is the Philadelphia chromosome (Ph+), originated from a reciprocal chromosomal translocation t(9;22)(q34;q11), that leads to the formation of the BCR/ABL1 fusion gene, which encodes for the constitutively active Bcr/Abl tyrosine kinase oncoprotein. Imatinib (STI-571) selectively targets the Bcr/Abl oncoprotein and represents now th
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TRIA, VALERIA. "CHARACTERIZATION OF EIF3E TRANSCRIPT: ROLE IN MAMMARY DEVELOPMENT AND CARCINOGENESIS." Doctoral thesis, Università degli Studi di Milano, 2013. http://hdl.handle.net/2434/217466.

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Il gene EIF3E (eukaryotic translation initiation factor 3 subunit e) è stato identificato inizialmente in topo come sito di integrazione del virus MMTV (mouse mammary tumor virus). Nel genoma umano EIF3E è un gene di 45 Kb localizzato nella porzione 22-23 del braccio lungo del cromosoma 8 e codifica per un RNA messaggero di 1516 nucleotidi e una proteina di 445 amminoacidi (52 KDa). L’integrazione del virus MMTV negli introni (5, 9, 12) del gene EIF3E causa la trascrizione di RNA tronchi. L’espressione di tali trascritti tronchi è in grado di indurre un fenotipo tumorale sia in vitro che in v
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Benyelles, Maname. "Le rôle de l'oncoprotéine INT6 dans la maintenance des télomères." Thesis, Lyon, École normale supérieure, 2015. http://www.theses.fr/2015ENSL0978/document.

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La protéine INT6/EIF3E codée par le gène mammalien correspondant au site d’intégration du rétrovirus Mouse Mammary tumor virus (MMTV) n°6 (int-6), a été impliquée dans le cancer du sein chez la souris et l’homme. Malgré qu’INT6 soit une sous-unité du facteur d’initiation de la traduction eIF3, elle n’est pas essentielle pour la traduction générale mais pour l’expression d’ARNm spécifiques tel qu’il a été montré pour la traduction d’ARNm histones. Elle a aussi été impliquée dans la réplication d’ADN en stabilisant le facteur de licence de la réplication MCM7, dans la réponse aux dommages à l’AD
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Raibon, Audrey. "Le facteur d'initiation de la traduction eIF3f dans le muscle squelettique : étude in vitro et obtention de modèles animaux." Thesis, Montpellier 1, 2013. http://www.theses.fr/2013MON1T023/document.

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Le facteur d'initiation de la traduction eIF3f est une des sous-unités constituant le facteur d'initiation de la traduction eIF3. Au niveau musculaire la surexpression de eIF3f dans les myotubes induit une hypertrophie associée à une augmentation de la synthèse protéique. A l'inverse, l'inhibition de l'expression de eIF3f entraîne une atrophie associée à une diminution de la synthèse protéique. Ce travail de thèse a permis (i) in vitro de mettre en évidence les fonctions inhibitrices du facteur eIF3f au cours de la prolifération des myoblastes C2C12 et par une étude transcriptomique sur les fr
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Guillon, Laurent. "Etude des facteurs du démarrage de la traduction eIF5B et eIF3." Phd thesis, Ecole Polytechnique X, 2008. http://pastel.archives-ouvertes.fr/pastel-00004650.

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Le démarrage de la traduction est un processus central dans toute cellule. L'étude des protéines assistant le ribosome pour réaliser cette étape, les facteurs de démarrage (Initiation Factors Ifs), permet d'obtenir des informations sur les mécanismes moléculaires complexes assurant la fidélité et l'efficacité du démarrage. La comparaison des jeux de facteurs protéiques dans les trois règnes du monde vivant a permis de mettre en évidence la présence de trois facteurs universellement conservés. Parmi ceux-ci, le facteur eucaryotique/archéen e/aIF5B, homologue au facteur bactérien IF2, stimule l'
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Wang, Xiaoshan. "Regulation of eIF3-Paip1 interaction by extracellular stimuli and phosphorylation status." Thesis, McGill University, 2010. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=86958.

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The tertiary interaction of poly(A)-binding protein (PABP), with eukaryotic translation initiation factor 4G (eIF4G) and 3'poly(A) tail of mRNA acts to stimulate translation initiation. Subsequently, the interaction of PABP-interacting protein 1 (Paip1) with PABP and eukaryotic translation initiation factor 3 (eIF3) (via the eIF3g subunit) further stimulates translation. Here, we demonstrate that the interaction of eIF3 and Paip1 is regulated by the presence of amino acids through an mTORC1 dependent signaling pathway. This interaction is inhibited by addition of mTORC1 signaling pathway inhib
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Morais, Ana Theresa Silveira de [UNESP]. "Caracterização da interação entre a proteínas NS5 do vírus da febre amarela e EIF3L." Universidade Estadual Paulista (UNESP), 2012. http://hdl.handle.net/11449/103873.

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Made available in DSpace on 2014-06-11T19:32:53Z (GMT). No. of bitstreams: 0 Previous issue date: 2012-08-10Bitstream added on 2014-06-13T19:22:41Z : No. of bitstreams: 1 morais_ats_dr_sjrp.pdf: 1276143 bytes, checksum: 62a89d8b555ac92b5faf4baa19e4db2f (MD5)<br>O vírus da Febre Amarela (YFV) pertence ao gênero Flavivirus e causa uma importante doença. Nos últimos anos, uma alarmante ressurgência da circulação viral e expansão do vírus em áreas endêmicas têm sido detectadas na África e América do Sul. NS5 é uma proteína viral não estrutural com duas atividades essenciais para a replicação vir
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Morais, Ana Theresa Silveira de. "Caracterização da interação entre a proteínas NS5 do vírus da febre amarela e EIF3L /." São José do Rio Preto : [s.n.], 2012. http://hdl.handle.net/11449/103873.

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Orientador: Maurício Lacerda Nogueira<br>Banca: Fátima Pereira de Souza<br>Banca: Cleslei Fernando Zanelli<br>Banca: Eurico de Arruda Neto<br>Banca: Luciana Barros de Arruda<br>Resumo: O vírus da Febre Amarela (YFV) pertence ao gênero Flavivirus e causa uma importante doença. Nos últimos anos, uma alarmante ressurgência da circulação viral e expansão do vírus em áreas endêmicas têm sido detectadas na África e América do Sul. NS5 é uma proteína viral não estrutural com duas atividades essenciais para a replicação viral, uma de metiltransferase e outra de RNA Polimerase dependente de RNA (RdRp).
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Bertorello, Juliette. "Reprogrammation traductionnelle par eIF3 liée à la résistance aux traitements des glioblastomes." Thesis, Toulouse 3, 2020. http://www.theses.fr/2020TOU30096.

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La résistance intrinsèque aux thérapies actuelles, conduisant à une rechute quasi systématique des patients, est une caractéristique des glioblastomes multiformes (GBM), la tumeur cérébrale la plus courante et la plus agressive. Comprendre les mécanismes sous-jacents d'une telle tumeur maligne est donc un besoin médical urgent. Il a été démontré par de nombreux travaux que la dérégulation de la machinerie de traduction des ARNm, notamment pendant l'étape d'initiation, contribue à la transformation maligne et à la progression des cancers en partie via une traduction sélective des transcrits spé
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Lagirand-Cantaloube, Julie. "MyoD et eIF3f : cibles majeures du complexe ubiquitine-ligase SCFMAFbx au cours de l'atrophie musculaire." Paris 11, 2008. http://www.theses.fr/2008PA11T020.

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Book chapters on the topic "EIF3j"

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Hershey, John W. B. "eIF3." In Translation and Its Regulation in Cancer Biology and Medicine. Springer Netherlands, 2014. http://dx.doi.org/10.1007/978-94-017-9078-9_8.

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Choudhuri, Avik, Anirban Ray, Arunima Biswas, and Umadas Maitra. "eIF3." In Encyclopedia of Signaling Molecules. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4614-6438-9_101984-1.

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Yin, Ji-Ye, Zizheng Dong, and Jian-Ting Zhang. "eIF3 Regulation of Protein Synthesis, Tumorigenesis, and Therapeutic Response." In Methods in Molecular Biology. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-6518-2_9.

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Conference papers on the topic "EIF3j"

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Tumia, Rima A. "Abstract 3029: Role of eIF3a expression in cellular sensitivity to radiation treatment." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-3029.

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Cui, Jia-Jia, Lei-Yun Wang, and Ji-Ye Yin. "Abstract 1407: Translational regulation of RPA2 via IRES by UNR and eIF3a." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-1407.

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Xu, Pengfei, and Dake Li. "Abstract 6000: eIF3A regulate the Warburg effect via ENO1 in cervical cancer cells." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-6000.

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Yin, Jiye, Zizheng Dong, Zhaoqian Liu, and Jianting Zhang. "Abstract 1749: Role of eIF3a in DNA repair and lung cancer chemo sensitivity." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-1749.

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Wang, Hong, Xuejiao Wang, Yuanbin Ru, Mark R. Conaway, Jeffery S. Kieft, and Dan Theodorescu. "Abstract 4200: Translation initiation factor eIF3b expression and its role in cancer cell growth and metastases." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-4200.

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Shi, Jiaqi, Brittany Silverman, and Lili Zhao. "Abstract B26: Altered eIF3f subcellular localization and expression in pancreatic ductal adenocarcinomas and its precursor lesions." In Abstracts: AACR Special Conference on Pancreatic Cancer: Advances in Science and Clinical Care; May 12-15, 2016; Orlando, FL. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.panca16-b26.

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Wu, Xiling, Yen-Lin Chu, and Chengtao Her. "Abstract 640: Human MutS homologue 4 (hMSH4) interacts with eIF3f and inhibits NHEJ-mediated DNA repair." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-640.

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Paluncic, Jasmina, Darius J. R. Lane, Federica Saletta, Yohan S. Rahmanto, Zaklina Kovacevic, and Des R. Richardson. "Abstract B212: N-myc downstream regulated 1 (NDRG1) is regulated by eukaryotic initiation factor 3a (eIF3a) during cellular stress caused by iron depletion." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics; October 26-30, 2017; Philadelphia, PA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1535-7163.targ-17-b212.

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Cuesta, Rafael, and Marina K. Holz. "Abstract B28: Estrogen receptor alpha (ERa) promotes protein synthesis by fine-tuning the expression of the eukaryotic translation initiation factor 3 subunit f (eIF3f)." In Abstracts: AACR Special Conference on Targeting PI3K/mTOR Signaling; November 30-December 8, 2018; Boston, MA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1557-3125.pi3k-mtor18-b28.

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Reports on the topic "EIF3j"

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Chamovitz, Daniel, and Albrecht Von Arnim. Translational regulation and light signal transduction in plants: the link between eIF3 and the COP9 signalosome. United States Department of Agriculture, 2006. http://dx.doi.org/10.32747/2006.7696515.bard.

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The COP9 signalosome (CSN) is an eight-subunit protein complex that is highly conserved among eukaryotes. Genetic analysis of the signalosome in the plant model species Arabidopsis thaliana has shown that the signalosome is a repressor of light dependent seedling development as mutant Arabidopsis seedlings that lack this complex develop in complete darkness as if exposed to light. These mutant plants die following the seedling stage, even when exposed to light, indicating that the COP9 signalosome also has a central role in the regulation of normal photomorphogenic development. The biochemical
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Chamovitz, Daniel A., and Albrecht G. Von Arnim. eIF3 Complexes and the eIF3e Subunit in Arabidopsis Development and Translation Initiation. United States Department of Agriculture, 2009. http://dx.doi.org/10.32747/2009.7696545.bard.

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The original working hypothesis of our proposal was that The “e” subunit of eIF3 has multiple functions from both within the nucleus and in the cytoplasm. Within this model, we further hypothesized that the “e” subunit of eIF3 functions in translation as a repressor. We proposed to test these hypotheses along the following specific aims: 1) Determine the subcellular localization of the interaction between eIF3e and other eIF3 subunits, or the COP9 signalosome. 2) Elucidate the biological significance of the varied subcellular localizations of eIF3e through generating Arabidopsis eIF3e alleles
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von Arnim, Albrecht G. Eukaryotic initiation factor 3 (eIF3) and 5’ mRNA leader sequences as agents of translational regulation in Arabidopsis. Final report. Office of Scientific and Technical Information (OSTI), 2015. http://dx.doi.org/10.2172/1169186.

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Chamovitz, Daniel, and Xing-Wang Deng. Morphogenesis and Light Signal Transduction in Plants: The p27 Subunit of the COP9-Complex. United States Department of Agriculture, 1997. http://dx.doi.org/10.32747/1997.7580666.bard.

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Plants monitor environmental signals and modulate their growth and development in a manner optimal for the prevailing light conditions. The mechanisms by which plants transduce light signals and integrate them with other environmental and developmental signals to regulate plant pattern development are beginning to be unraveled. A large body of knowledge has accumulated regarding the roles of specific photoreceptors in perceiving light signals, and about the downstream developmental responses responding to light (Batschauer, 1999; Chamovitz and Deng, 1996; Deng and Quail, 1999). Still, little i
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