Journal articles: 'Embolie pulmonaire Thrombose Veines'

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Pernod, G. "Risque de récidive après thrombose veineuse profonde et embolie pulmonaire." Journal des Maladies Vasculaires 40, no. 2 (March 2015): 81. http://dx.doi.org/10.1016/j.jmv.2014.12.087.

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Cordeanu, Elena-Mihaela, J. Di Cesare, C. Mirea, A. M. Faller, A. Delatte, W. Younes, H. Lambach, et al. "Embolie pulmonaire et thrombose veineuse profonde concomitante : une association délétère ?" JMV-Journal de Médecine Vasculaire 44, no. 2 (March 2019): 156. http://dx.doi.org/10.1016/j.jdmv.2018.12.149.

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Soya, Esaï, F. Koffi, J. J. N’djessan, A. Ekou, A. Ahoua, E. Monney, S. Kouamé, and C. Konin. "Thrombose veineuse profonde et embolie pulmonaire sont-elles si identiques ?" JMV-Journal de Médecine Vasculaire 44, no. 2 (March 2019): 159. http://dx.doi.org/10.1016/j.jdmv.2018.12.156.

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Bressollette, L., and G. Le Gal. "Une embolie pulmonaire est plus grave qu’une thrombose veineuse profonde proximale." Journal des Maladies Vasculaires 37, no. 2 (March 2012): 57. http://dx.doi.org/10.1016/j.jmv.2011.12.110.

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Zuily, S. "Embolie pulmonaire et thrombose veineuse profonde : même maladie oui, mais même malades ?" Journal des Maladies Vasculaires 38, no. 2 (March 2013): 91. http://dx.doi.org/10.1016/j.jmv.2012.12.147.

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Vignat, N. "Prise en charge d’une thrombose veineuse profonde et/ou une embolie pulmonaire." Archives des Maladies du Coeur et des Vaisseaux - Pratique 2014, no. 231 (October 2014): 9–20. http://dx.doi.org/10.1016/s1261-694x(14)70673-x.

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Meneveau, N., and J. P. Bassand. "Quand suspecter une embolie pulmonaire chez un malade ayant une thrombose veineuse profonde ?" Annales de Cardiologie et d'Angéiologie 51, no. 3 (2002): 139–45. http://dx.doi.org/10.1016/s0003-3928(02)00086-0.

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Ly, K. H., E. Liozon, G. Gondran, H. Bezanahary, S. Palat, F. X. Lapébie, A. Cypierre, S. Nadalon, and A. Fauchais. "Thrombose veineuse profonde et embolie pulmonaire au cours de l’artérite à cellules géantes." La Revue de Médecine Interne 35 (December 2014): A46—A47. http://dx.doi.org/10.1016/j.revmed.2014.10.070.

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Fiori, S., S. Accassat, J. Catella-Chatron, L. Bertoletti, V. Chambefort, C. Duvillard, and B. Seffert. "Impact pronostique d’une thrombose veineuse profonde asymptomatique chez les patients pris en charge pour embolie pulmonaire." JMV-Journal de Médecine Vasculaire 43, no. 2 (March 2018): 118. http://dx.doi.org/10.1016/j.jdmv.2017.12.096.

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Minvielle, F. "Durée du traitement anticoagulant après une embolie pulmonaire ou une thrombose veineuse proximale des membres inférieurs." Archives des Maladies du Coeur et des Vaisseaux - Pratique 2021, no. 300 (September 2021): 35–38. http://dx.doi.org/10.1016/j.amcp.2021.06.011.

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Roux, M., P. Miranda, MC Trzeciak, L. Pinède, and J. Ninet. "Détermination des facteurs de risque biologiques de récidive de thrombose veineuse chez 90 patients présentant une thrombose veineuse profonde et/ou une embolie pulmonaire." La Revue de Médecine Interne 19 (January 1998): 414. http://dx.doi.org/10.1016/s0248-8663(98)90094-7.

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Baili, L., M. Smiti Khanfir, Z. Ayadi, H. Naffeti, T. Larbi, I. Ben Ghorbel, and M. H. Houman. "Profil épidémiologique, clinique, étiologique et évolutif des patients ayant une thrombose veineuse profonde compliquée d'une embolie pulmonaire." La Revue de Médecine Interne 27 (December 2006): S406. http://dx.doi.org/10.1016/j.revmed.2006.10.289.

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Hadj Ali, Z., F. Daoued, Z. Aydi, L. Baili, B. Ben Dhaou, and F. Boussema. "Caractéristiques épidémiologiques, cliniques, étiologiques et évolutives des patients ayant une embolie pulmonaire compliquant une thrombose veineuse profonde." Journal des Maladies Vasculaires 40, no. 5 (September 2015): 323–24. http://dx.doi.org/10.1016/j.jmv.2015.07.067.

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Ben Cheikh, J., J.-P. Cambou, H. Boccalon, and A. Bura-Rivière. "La maladie veineuse thromboembolique chez l’homme et chez la femme : thrombose veineuse profonde avec ou sans embolie pulmonaire (registre RIETE)." Journal des Maladies Vasculaires 33 (September 2008): S73. http://dx.doi.org/10.1016/j.jmv.2008.07.040.

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Hariri, A., G. Nourissat, C. Dumontier, and L. Doursounian. "Thrombose de la veine humérale et embolie pulmonaire après chirurgie arthroscopique de l’épaule du sujet jeune, à propos d’un cas." Revue de Chirurgie Orthopédique et Traumatologique 95, no. 5 (September 2009): 462–64. http://dx.doi.org/10.1016/j.rcot.2009.05.006.

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Auvray, M., C. Le Hello, A. Armand, V. Le Pennec, M. Ramakers, G. Zalcman, G. Grollier, et al. "P06 Un formulaire unique et simplifié de demande d’examens pour une meilleure prise en charge de la maladie thrombo-embolique veineuse (thrombose veineuse profonde et/ou embolie pulmonaire)." Journal des Maladies Vasculaires 31, no. 4 (September 2006): 33–34. http://dx.doi.org/10.1016/s0398-0499(06)76606-9.

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Willems, Ariane, Michèle David, and Marisa Tucci. "Prophylaxie des thromboembolies veineuses en réanimation pédiatrique : une mise au point." Médecine Intensive Réanimation 29, no. 2 (July 24, 2020): 116–27. http://dx.doi.org/10.37051/mir-00016.

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Objectif : Le but de ce manuscrit est de revoir la littérature actuelle sur la prévention des thromboses veineuses chezl’enfant admis en réanimation.Matériel et méthode : Revue systématique basée sur les articles trouvés dans PubMed et Embase entre janvier 1975 etmars 2018. Les mots clés suivants ont été utilisés : paediatric, children, venous thromboembolism, venous thrombosis,pulmonary embolism, thromboprophylaxis.Résultats : La thrombose veineuse est fréquente en réanimation pédiatrique et souvent sous-estimée malgré sonassociation avec une morbi-mortalité importante. La cause principale est la présence d’un cathéter veineux central.Comparativement à l’adulte, il n’existe chez l’enfant aucune évidence d’un bénéfi ce d’une prophylaxie mis à part peut-être chez l’adolescent traumatisé.Conclusions : La maladie thrombo-embolique veineuse est une préoccupation chez l’enfant gravement malade.En absence de preuves, des études sont nécessaires afi n de cibler les patients à risque et qui bénéfi cieraientd’une prophylaxie et ceux à risque de saignement chez qui l’administration d’anticoagulation pourrait êtredélétère.

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Elhammady, Gina, Andrew T. Schubeck, Vicky El-Najjar, and Morton J. Robinson. "Postmortem Demonstration of the Source of Pulmonary Thromboembolism: The Importance of the Autopsy." Case Reports in Vascular Medicine 2011 (2011): 1–3. http://dx.doi.org/10.1155/2011/108215.

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Periprostatic or paravaginal venous thromboses are rarely considered clinically as sites of clot origin in patients with pulmonary thromboembolism. The majority of emboli have been demonstrated to originate in the veins of the legs. This report raises awareness of pelvic vein thrombosis as a potential source of pulmonary embolism that is rarely considered or detected clinically, and which usually requires postmortem examination for recognition. It also reviews the possible routes emboli may take to reach the lungs.

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Emmerich, J., G. Meyer, D. Haley, E. Arnaud, V. Nicaud, M. Alhenc-Gelas, M. Aiach, A. M. Fischer, J. N. Fiessinger, and H. Sors. "Prévalence du facteur V Leiden et de la mutation 20210A du facteur II dans une large cohorte de patients non sélectionnés avec thrombose veineuse profonde ou embolie pulmonaire." La Revue de Médecine Interne 20 (January 1999): 600s. http://dx.doi.org/10.1016/s0248-8663(00)87656-0.

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O’Brien, Danielle M., and Sundus H. Mohammad. "Acute Thrombotic Complication Post-Endovenous Ablation With Mobile Thrombus: A Case Study." Journal of Diagnostic Medical Sonography 36, no. 2 (December 21, 2019): 163–69. http://dx.doi.org/10.1177/8756479319892850.

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Endovenous ablation is a common procedure performed on the superficial veins of the lower extremities to treat symptoms of venous insufficiency and varicose veins. While endovenous ablation is a minimally invasive procedure, patients may develop thrombotic complications including endovenous heat-induced thrombosis. Endovenous heat-induced thrombosis is a rare complication of thrombus extension from the superficial vein into or near the deep system postprocedure. In this case report, a female with a history of recent endovenous ablation presented to the emergency department with symptoms of a pulmonary embolism. Sonographic evaluation of the right lower extremity revealed extension of thrombus from the ablated superficial vein into the common femoral vein. Thrombus was also noted in the left lower extremity extending from the ablated superficial vein to approximately the saphenofemoral junction. While the left lower extremity was being interrogated, the thrombus was documented, via a cine clip, liberating from the occluded great saphenous vein. Further assessment utilizing computed tomography revealed bilateral pulmonary emboli. This unique case emphasizes the importance of sonographic evaluation post-endovenous ablation.

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El Mouknia, M., A. Harti, L. Barrou, K. Mjahed, S. Fedouach, and M. Benaguida. "Thrombose de l'oreillette droite révélée par une embolie pulmonaire grave." Annales Françaises d'Anesthésie et de Réanimation 13, no. 2 (January 1994): 255–57. http://dx.doi.org/10.1016/s0750-7658(05)80560-x.

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Bounzira, T., D. Hakem, S. Médaoud, D. Zemmour, N. Ouadahi, L. Baghli, S. Harriti, and A. Berrah. "RPCA familiale d’expressions sévères chez trois frères (thrombose porte, thrombose cérébrale et embolie pulmonaire)." Journal des Maladies Vasculaires 36, no. 2 (March 2011): 127. http://dx.doi.org/10.1016/j.jmv.2010.12.091.

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Zaghba, N., A. Ech-cherrate, H. Benjelloun, N. Yassine, A. Bakhatar, and A. Bahlaoui. "Thrombose cardiaque, anévrisme de l’artère pulmonaire et embolie pulmonaire révélant une maladie de Behçet." Revue de Pneumologie Clinique 68, no. 5 (October 2012): 307–10. http://dx.doi.org/10.1016/j.pneumo.2012.04.004.

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Kamalov, I. A., I. R. Agliullin, and M. G. Tukhbatullin. "Optimization of proper diagnosis of thromboses associated with high risk of embolism in patients with neoplasms." Kazan medical journal 94, no. 2 (April 15, 2013): 202–7. http://dx.doi.org/10.17816/kmj1589.

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Aim. To optimize a proper diagnosis of thromboses associated with high risk of embolism and pulmonary embolism prevention in patients with neoplasms. Methods. Ultrasonography of veins of lower extremities, iliac veins and the distal part of inferior vena cava was performed in patients with and without neoplasms in a prospective study to detect thromboses associated with high risk of embolism and thrombophlebitis. Ultrasonography was performed once in control group subjects, and before and during specialized antineoplastic treatment (surgical, chemotherapy, radiotherapy) in patients of the main group, the results were compared. A detection of a new thrombus in previously intact venous segment of inferior vena cava system was assessed as a high risk for pulmonary embolism. Results. Thromboses associated with high risk of embolism and thrombophlebitis were found in 6 patients of control group, in 5 patients of the main group before and in 27 patients of the main group while at specialized antineoplastic treatment. Specific measures for pulmonary embolism prevention were taken immediately in all of the cases according to ultrasonography results after the detection of thromboses associated with high risk of embolism. No fatal cases of pulmonary embolism were registered both in main (before and while at treatment) and control groups. Conclusion. Ultrasonography of veins of lower extremities, iliac veins and the distal part of inferior vena cava in patients with neoplasms before the start of specialized antineoplastic treatment allows to optimize the choice of prevention measures for pulmonary embolism and thus significantly decreases mortality from pulmonary embolism.

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Kumar, Priyatham. "Deep Vein Thrombosis and Pulmonary Embolism in Sickle Cell Disease." Biomedical Research and Clinical Reviews 1, no. 5 (December 4, 2020): 01–04. http://dx.doi.org/10.31579/2692-9406/024.

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Sickle Cell Disease (SCD) is considered a group of genetic red blood cell (RBC) disorders. Healthy red blood cells (RBC) are round in shape and migrates throughout the body to carry oxygen in the small blood vessels. In SCD, the RBC turns into hard and sticky, and the shape is similar to a C-Shaped tool called "SICKLE." Because of the early death of the sickle cells, a constant shortage of red blood cells arises. Because of the typical shape of the sickle cells, their movement in the blood vessel is not as smooth as normal RBC and get stuck and clog the blood flow leading to anemia. The changes in shape make the cells more easily destroyed, causing anemia. Defective hemoglobin is the primary cause of SCD.

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Thomas, M. Lea, and G. Solis. "The Phlebographic Distribution of Deep Venous Thrombosis in the Calf and its Relevance to Duplex Ultrasound." Phlebology: The Journal of Venous Disease 7, no. 2 (June 1992): 64–66. http://dx.doi.org/10.1177/026835559200700204.

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Objective: To assess the distribution of deep vein thrombosis in the calf by phlebography. Setting: Department of Vascular Radiology, St. Thomas' Hospital, London, England. Patients: Seventy patients with suspected deep vein thrombosis or pulmonary embolism were examined. Interventions: Bilateral ascending contrast phlebography was performed in all patients. Main Outcome Measures: The sites of any thrombus in the stem or muscle veins of the calf below the popliteal vein were recorded. Results: One hundred legs contained thrombus. In fifty-three legs thrombus was present solely in the calf veins below the popliteal vein. Isolated thrombus in either one or more of the three paired stem veins or the muscle veins was present in twenty-two calves. Conclusions: Because of the difficulty in visualising some calf veins by duplex ultrasound it is suggested that a detailed knowledge of the distribution of thrombus may assist ultrasonographers.

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Gabrielli, Roberto, Maria Sofia Rosati, Andrea Siani, and Luigi Irace. "Management of Symptomatic Venous Aneurysm." Scientific World Journal 2012 (2012): 1–6. http://dx.doi.org/10.1100/2012/386478.

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Venous aneurysms (VAs) have been described in quite of all the major veins. They represent uncommon events but often life-threatening because of pulmonary or paradoxical embolism. We describe our twelve patients’ series with acute pulmonary emboli due to venous aneurysm thrombosis. Our experience underlines the importance of a multilevel case-by-case approach and the immediate venous lower limbs duplex scan evaluation in pulmonary embolism events. Our data confirm that anticoagulant alone is not effective in preventing pulmonary embolism. We believe that all the VAs of the deep venous system of the extremities should be treated with surgery as well as symptomatic superficial venous aneurysm. A simple excision can significantly improve symptoms and prevent pulmonary embolism.

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Kamalov, I. A., I. R. Agliullin, M. G. Tukhbatullin, and I. R. Safin. "Required frequency of ultrasonography for detection of thrombosis with high risk for embolism in patients with malignancies." Kazan medical journal 94, no. 3 (June 15, 2013): 335–39. http://dx.doi.org/10.17816/kmj2180.

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Aim. To determine the optimal terms for detection of thrombosis with high risk for embolism in patients with malignancies receiving specialized treatment. Methods. 117 patients (50 males, 67 females - the main group) with malignancies were randomly picked out (using the random numbers tables) underwent daily ultrasonography of inferior vena cava tributaries for detection of thrombosis with high risk for embolism. Ultrasonography of inferior vena cava distal part, both common iliac veins and veins of lower extremities was done in patients while on surgical treatment, chemotherapy or radiotherapy. The control group consisted of 130 patients (58 males, 72 females) with malignancies in whom ultrasonography was performed only if clinical signs of venous thrombosis were present. Results. Ultrasonic features of venous blood flow decrease (spontaneous contrast phenomenon) in veins of lower extremities were found in 27 out of 117 main group patients on the second day. On the third day features of saphenous veins thrombosis were found in 13 patients. On the fourth day, 4 patients were diagnosed with iliofemoral thrombosis. 5 more patients developed ultrasonic features of venous blood flow decrease (spontaneous contrast phenomenon) on the fourth and fifth day. Signs of thrombosis progression and floating thrombus were found in 6 patients on the 6th and 7th day. No fatal cases of pulmonary embolism were registered in the main group. 10 patients of the control group had clinical signs of inferior vena cava tributaries and underwent distal part of inferior vena cava, both common iliac veins and veins of lower extremities ultrasonography while on specialized treatment. 5 cases of pulmonary embolism were reported in the rest of the control group patients (120 patients). Conclusion. Inferior vena cava tributaries thrombosis with high risk for embolism in patients with malignancies can be reliably detected by repeating ultrasonography every 3-4 days; Reliable diagnosis of thrombosis with high risk for embolism by means of ultrasonography during the post-surgical period allows to prevent pulmonary embolism in a timely and targeted manner.

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Mahé, V., T. Bonnal, J. Y. Riou, F. Bernage, and C. Ecoffey. "Embolie pulmonaire massive due à une thrombose sur cathéter central chez un enfant." Annales Françaises d'Anesthésie et de Réanimation 12, no. 5 (January 1993): 505–7. http://dx.doi.org/10.1016/s0750-7658(05)81000-7.

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Stefano, Valerio De, and Ida Martinelli. "Rare thromboses of cerebral, splanchnic and upper-extremity veins." Thrombosis and Haemostasis 103, no. 06 (2010): 1136–44. http://dx.doi.org/10.1160/th09-12-0873.

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SummaryVenous thrombosis typically involves the lower extremity circulation. Rarely, it can occur in the cerebral or splanchnic veins and these are the most frightening manifestations because of their high mortality rate. A third site of rare venous thrombosis is the deep system of the upper extremities that, as for the lower extremity, can be complicated by pulmonary embolism and post-thrombotic syndrome. The authors conducted a narrative review focused on clinical manifestations, risk factors, and treatment of rare venous thromboses. Local risk factors such as infections or cancer are frequent in thrombosis of cerebral or portal veins. Upper extremity deep-vein thrombosis is mostly due to local risk factors (catheter- or effort-related). Common systemic risk factors for rare venous thromboses are inherited thrombophilia and oral contraceptive use; chronic myeloproliferative neoplasms are closely associated with splanchnic vein thrombosis. In the acute phase rare venous thromboses should be treated conventionally with low-molecular-weight heparin. Use of local or systemic fibrinolysis should be considered in the case of clinical deterioration in spite of adequate anticoagulation. Anticoagulation with vitamin K-antagonists is recommended for 3–6 months after a first episode of rare venous thrombosis. Indefinite anticoagulation is recommended for Budd-Chiari syndrome, recurrent thrombosis or unprovoked thrombosis and permanent risk factors. In conclusion, the progresses made in the last couple of decades in diagnostic imaging and the broadened knowledge of thrombophilic abnormalities improved the recognition of rare venous thromboses and the understanding of pathogenic mechanisms. However, the recommendations for treatment mainly derive from observational studies.

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Papysheva, O. V., A. E. Voynovskiy, A. I. Davydov, and M. B. Tairova. "Postpartum ovarian vein thrombosis." Voprosy ginekologii, akušerstva i perinatologii 19, no. 5 (2020): 147–49. http://dx.doi.org/10.20953/1726-1678-2020-5-147-149.

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A clinical case of thrombosis of the right ovarian vein in the postpartum period - an extremely rare pathology in which acute appendicitis is often suspected due to the identity of the symptoms of the diseases. Performed laparoscopy, during which a large fragment of a thrombosed vein was excised. Conclusion. Extensive right ovarian vein thrombosis with a normal hemostasiogram could not only not be resolved by anticoagulants, but also potentially spread to the renal veins and inferior vena cava, increasing the risk of pulmonary embolism. Key words: vein dissection, laparoscopy, postpartum period, ovarian vein thrombosis

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Heidrich, Konau, and Hesse. "Asymptomatic venous thrombosis in cancer patients – a problem often overlooked. Results of a retrospective and prospective study." Vasa 38, no. 2 (May 1, 2009): 160–66. http://dx.doi.org/10.1024/0301-1526.38.2.160.

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Background: Venous thrombosis with and without pulmonary embolism is a frequent complication of malignancies and second among the causes of death in tumour patients. Its incidence is reported to be 10 to 15%. Since for methodological reasons, this rate can be assumed to be too low and to disregard asymptomatic venous thrombosis, a combined retrospective and prospective study was performed to examine the actual frequency of venous thrombosis in tumour patients. Patients and methods: The histories of 409 patients (175 women, 234 men, mean age 69 years [19 to 96 years]) with different tumours, consecutively enrolled in the order of their altogether 426 inpatient treatments, were checked in retrospect for the frequency of venous thrombosis and pulmonary embolism. Subsequently, 97 tumour inpatients (36 women, 61 men, mean age 70 years [42 to 90 years]) were systematically screened, by means of duplex sonography and/or venography, for venous thromboses in the veins of the pelvis and both legs. Results: In the retrospective analysis, where no systematic screening for thromboses was performed and only symptomatic thrombosis was recorded, venous thrombosis was found in 6.6% of all tumour patients, whereas in the prospective examination with systematic duplex sonography and / or venography of all patients, the percentage was 33%. In the prospective study, 31.3% of venous thromboses were symptomatic and 68.7% asymptomatic. In 39.3% of the cases in the retrospective analysis and 25% in the prospective analysis, venous thrombosis occurred during chemotherapy, surgery or radiation therapy. Venous thrombosis was most often seen in metastasizing tumours and in colorectal carcinoma (40%), haematological system diseases (28.6%), gastric cancer (30%), bronchial, pancreas and ovarian carcinoma (28.6%), and carcinoma of the prostate (16.7%). Conclusion: Regular screening for thrombosis is indicated even in asymptomatic tumour patients because asymptomatic venous thrombosis is frequent, can lead to pulmonary embolism and has to be treated like symptomatic venous thrombosis. This is particularly true for metastasization during chemotherapy, surgical interventions, or radiation.

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Anfinogenova, Nina D., Oksana Y. Vasiltseva, Alexander V. Vrublevsky, Irina N. Vorozhtsova, Sergey V. Popov, Andrey V. Smorgon, Olga V. Mochula, and Wladimir Y. Ussov. "Right Atrial Thrombosis and Pulmonary Embolism: A Narrative Review." Seminars in Thrombosis and Hemostasis 46, no. 08 (November 2020): 895–907. http://dx.doi.org/10.1055/s-0040-1718399.

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AbstractPrompt diagnosis of pulmonary embolism (PE) remains challenging, which often results in a delayed or inappropriate treatment of this life-threatening condition. Mobile thrombus in the right cardiac chambers is a neglected cause of PE. It poses an immediate risk to life and is associated with an unfavorable outcome and high mortality. Thrombus residing in the right atrial appendage (RAA) is an underestimated cause of PE, especially in patients with atrial fibrillation. This article reviews achievements and challenges of detection and management of the right atrial thrombus with emphasis on RAA thrombus. The capabilities of transthoracic and transesophageal echocardiography and advantages of three-dimensional and two-dimensional echocardiography are reviewed. Strengths of cardiac magnetic resonance imaging (CMR), computed tomography, and cardiac ventriculography are summarized. We suggest that a targeted search for RAA thrombus is necessary in high-risk patients with PE and atrial fibrillation using transesophageal echocardiography and/or CMR when available independently on the duration of the disease. High-risk patients may also benefit from transthoracic echocardiography with right parasternal approach. The examination of high-risk patients should involve compression ultrasonography of lower extremity veins along with the above-mentioned technologies. Algorithms for RAA thrombus risk assessment and protocols aimed at identification of patients with RAA thrombosis, who will potentially benefit from treatment, are warranted. The development of treatment protocols specific for the diverse populations of patients with right cardiac thrombosis is important.

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van Langevelde, Kirsten, Alexandr Srámek, Patrice W. J. Vincken, Jan-Kees van Rooden, Frits R. Rosendaal, and Suzanne C. Cannegieter. "Is Deep-Vein Thrombosis Always the Origin of Pulmonary Embolism? Imaging of Veins with a Total-Body Direct Thrombus MRI Technique." Blood 118, no. 21 (November 18, 2011): 2297. http://dx.doi.org/10.1182/blood.v118.21.2297.2297.

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Abstract Abstract 2297 Introduction Pulmonary embolism (PE) is thought to originate from embolisation of a deep-vein thrombosis (DVT), resulting in two clinical manifestations of one disease: venous thrombosis. However, in up to 50% of patients with PE, no DVT is found with ultrasound. A new technique in diagnosing DVT is ‘MR Direct Thrombus Imaging’ (MRDTI), using methaemoglobin in the thrombus as endogenous contrast. We performed total body MRIs to visualize thrombi from calf veins to subclavian veins, to assess the origin of PE. Methods Consecutive patients with a first PE diagnosed by Multi Detector CT scan are included. All patients undergo a 1.5 T MRDTI scan. In addition, blood is sampled for determination of genetic risk factors: the Factor V Leiden (FVL) and prothrombin G20210A (FII) mutations. Results 99 patients have been enrolled in the study. In 44/99 (44%) a thrombus was identified on MR images. Most thrombi originated from the left side of the body (27/44). The upper leg was involved most often (22/44), with thrombi extending into the calf in 16 of these patients. Other thrombi were present in the pelvic veins (4/44), and deep calf veins alone (12/44). 6 patients had merely thrombophlebitis. The FVL mutation was present in 14% (95% CI 6–27) of patients with a thrombus on MRI, and in 7% (95% CI 3–18) of patients without a thrombus. For FII, the prevalence in the group with a DVT on MRI was 2% (95% CI 0.4–12) and without a DVT 4% (95% CI 1–13). Conclusion In less than half of PE patients a thrombus can be found with total body MRI. When thrombi were present, they were mainly found in the upper leg, extending into the calf veins. Interestingly, 27% of PEs with a thrombus on MRI was related to isolated calf vein thrombosis and 14% to thrombophlebitis. In PE patients with no thrombus on MRI, a local origin of PE, a thrombus from cardiac origin or complete embolisation needs to be considered. Supported by Netherlands Heart Foundation grant number 2009B050 Disclosures: No relevant conflicts of interest to declare.

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Ramazanov, G. R., A. E. Talypov, A. A. Kanibolotskiy, Kh V. Korigova, V. N. Stepanov, L. Kh Akhmatkhanovа, M. V. Sinkin, and S. S. Petrikov. "Superior sagittal sinus thrombosis as a focal point of pulmonary embolism." Russian neurological journal 26, no. 1 (March 26, 2021): 39–44. http://dx.doi.org/10.30629/2658-7947-2021-26-1-39-44.

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This article represents the discussion of a clinical case of superior sagittal sinus thrombosis as a focal point of fatal pulmonary embolism. Pulmonary embolism is a life-threatening condition, with a mortality rate of up to 40%. The direct source of pulmonary embolism is deep vein thrombosis of the lower extremities and pelvis in 80–90% of all cases. The veins of the upper extremities and venous heart cause it less often. Pulmonary embolism in patients with cerebral venous thrombosis is observed in 1.4% of patients.Cerebral venous thrombosis is a cerebrovascular disease manifested by venous outflow disorders due to acute occlusion of the sinuses and veins of the brain. It requires immediate treatment in order to prevent the development of intracranial hemorrhage, venous infarction, disability and death. Cerebral venous thrombosis accounts for approximately 0.5% of all cases of cerebrovascular disease worldwide. In contrast to ischemic stroke, cerebral venous thrombosis is more common in younger patients. Currently, the diagnosis of cerebral venous thrombosis is based on neuroimaging data, and timely treatment leads to a decrease in disability and mortality.

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Marty and Depairon. "Prise en charge de la phlegmasia cerulea dolens." Praxis 95, no. 21 (May 1, 2006): 845–48. http://dx.doi.org/10.1024/0369-8394.95.21.845.

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La phlegmasia cerulea dolens (PCD) résulte d'une thrombose aiguë et massive du réseau veineux profond du membre inférieur envahissant souvent les veines iliaques. Elle se caractérise par une extrémité oedématiée fortement cyanosée intense et s'accompagnant de fortes douleurs. Le but thérapeutique est de sauver l'extrémité, d'éviter l'embolie pulmonaire et de prévenir le syndrome post-thrombotique. En cas d'échec thérapeutique conservateur, le traitement de choix se compose d'une thrombectomie iliaque à l'aide d'un ballon et d'une fibrinolyse périphérique simultanée avec expulsion des thrombi fémoro-poplités. La thrombectomie n'est possible qu'au niveau de l'axe iliaque avalvulé. La thrombolyse reste le moyen idéal pour une ablation efficace des thromboses des systèmes veineux superficiel et profond puisque ces derniers sont valvulés sur la plus grande partie de leur trajet. Cet article a essentiellement pour objet le traitement de la phlegmasia. Toutefois, la plupart des études citées concerne le traitement d'une thrombose veineuse profonde importante (TVP) touchant l'axe iliaque et souligne de ce fait que les thromboses ilio-fémorales massives peuvent également êtres considérées comme des indications au traitement invasif.

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Aşker, Selvi, Müntecep Aşker, Özgür Gürsu, Rıdvan Mercan, and Özgür Bülent Timuçin. "A Behcet’s Disease Patient with Right Ventricular Thrombus, Pulmonary Artery Aneurysms, and Deep Vein Thrombosis Complicating Recurrent Pulmonary Thromboembolism." Case Reports in Pulmonology 2013 (2013): 1–4. http://dx.doi.org/10.1155/2013/492321.

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Intracardiac thrombus, pulmonary artery aneurysms, deep vein thrombosis, and pulmonary thromboembolism are rarely seen symptoms of Behcet’s disease. A 20-year-old female patient was admitted for complaints of cough, fever, palpitations, and chest pain. On the dynamic thorax computed tomograms (CT) obtained because of significantly enlarged hilar structures seen on chest radiograms, aneurysmal dilatation of the pulmonary artery segments bilaterally, chronic thrombus with collapse, and consolidation substances compatible with pulmonary embolism involving both lower lobes have been observed. It is learned that, four years ago, the patient had been diagnosed with Behcet’s disease and received colchicine treatment but not regularly. The patient was hospitalized. On the transthoracic echocardiogram, a thrombosis with a dimension of 4.2 × 1.6 cm was recognized in the right ventricle. On abdomen CT, aneurysmal iliac veins and deep vein thrombus on Doppler ultrasonograms were diagnosed. At the controls after three months of immunosuppressive and anticoagulant therapies, some clinical and radiological improvements were recognized. The patient suspended the treatment for a month and the thrombus recurred. We present our case in order to show the effectiveness of immunosuppressive and anticoagulant therapies and rarely seen pulmonary thromboembolism in recurrent Behcet’s disease.

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Christiansen, F., T. Kellerth, T. Andersson, A. Ragnarsson, and F. Hjortevang. "Ultrasound at Scintigraphic “Intermediate Probability of Pulmonary Embolism”." Acta Radiologica 37, no. 1P1 (January 1996): 14–17. http://dx.doi.org/10.1177/02841851960371p104.

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For diagnosis of pulmonary embolism (PE), ventilation/perfusion lung scintigraphy is routinely used; approximately one-third of the patients will have the diagnosis “intermediate probability of PE” (inconclusive). In this group only about 33% are found to have pulmonary emboli if examined with pulmonary angiography. To evaluate the diagnostic, therapeutic, and economic consequences of ultrasound of the legs as a complementary diagnostic investigation to “intermediate probability”, 72 consecutive patients were investigated with bilateral ultrasound of the proximal deep veins of the legs and pulmonary angiography in a prospective study. Ten patients had PE, of whom 7 had deep venous thrombosis, and 62 had no PE, of whom 2 had deep venous thrombosis. The negative predictive value of ultrasound was 0.95. In view of the importance of adequate treatment and rational use of public health care expenditure, complementary diagnostics should be performed, and ultrasound is an adequate complementary investigation.

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Zou, Junjie, Hongyu Yang, Hao Ma, Shui Wang, and Xiwei Zhang. "Embolie pulmonaire provoquée par un anévrysme veineux iliaque externe thrombosé." Annales de Chirurgie Vasculaire 25, no. 7 (October 2011): 1046.e19–1046.e22. http://dx.doi.org/10.1016/j.acvfr.2012.10.013.

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Alias, Sherin, Bassam Redwan, Adelheid Panzenböck, Max P. Winter, Uwe Schubert, Robert Voswinckel, Maria K. Frey, et al. "Defective Angiogenesis Delays Thrombus Resolution." Arteriosclerosis, Thrombosis, and Vascular Biology 34, no. 4 (April 2014): 810–19. http://dx.doi.org/10.1161/atvbaha.113.302991.

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Objective— Restoration of patency is a natural target of vascular remodeling after venous thrombosis that involves vascular endothelial cells and smooth muscle cells, as well as leukocytes. Acute pulmonary emboli usually resolve <6 months. However, in some instances, thrombi transform into fibrous vascular obstructions, resulting in occlusion of the deep veins, or in chronic thromboembolic pulmonary hypertension (CTEPH). We proposed that dysregulated thrombus angiogenesis may contribute to thrombus persistence. Approach and Results— Mice with an endothelial cell–specific conditional deletion of vascular endothelial growth factor receptor 2/kinase insert domain protein receptor were used in a model of stagnant flow venous thrombosis closely resembling human deep vein thrombosis. Biochemical and functional analyses were performed on pulmonary endarterectomy specimens from patients with CTEPH, a human model of nonresolving venous thromboembolism. Endothelial cell–specific deletion of kinase insert domain protein receptor and subsequent ablation of thrombus vascularization delayed thrombus resolution. In accordance with these findings, organized human CTEPH thrombi were largely devoid of vascular structures. Several vessel-specific genes, such as kinase insert domain protein receptor, vascular endothelial cadherin, and podoplanin, were expressed at lower levels in white CTEPH thrombi than in organizing deep vein thrombi and organizing thrombi from aortic aneurysms. In addition, red CTEPH thrombi attenuated the angiogenic response induced by vascular endothelial growth factor. Conclusions— In the present work, we propose a mechanism of thrombus nonresolution demonstrating that endothelial cell–specific deletion of kinase insert domain protein receptor abates thrombus vessel formation, misguiding thrombus resolution. Medical conditions associated with the development of CTEPH may be compromising early thrombus angiogenesis.

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Baili, L., F. Daoud, Z. Aydi, B. Ben Dhaou, F. Boussema, and L. Rokbani. "Caractéristiques épidémiologiques, cliniques, étiologiques et évolutives des patients ayant une embolie pulmonaire compliquant une thrombose veineuses profonde." La Revue de Médecine Interne 33 (June 2012): S108. http://dx.doi.org/10.1016/j.revmed.2012.03.156.

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Malhotra, Rahul. "Audit on venous thromboembolism risk assessment in mental health inpatient wards." BJPsych Open 7, S1 (June 2021): S331—S332. http://dx.doi.org/10.1192/bjo.2021.870.

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AimsFrom May 2015 NHS organisations in Wales are expected to report the number of VTE cases associated with hospital admissions which are possible hospital acquired thrombosis (HAT) per calendar month. NICE Quality Standards (QS3) recommend that All patients, on admission, receive an assessment of VTE and bleeding risk using the clinical risk assessment criteria described in the national tool.BackgroundVTE is a condition in which a blood clot (thrombus) forms in a vein, most commonly in the deep veins of the legs, known as a deep vein thrombosis (DVT). The thrombus can dislodge from its original site and travel in the blood (embolism). If it becomes lodged in the lungs, a condition known as a pulmonary embolism (PE) arises and can cause sudden death. Hospital acquired thrombosis is avoidable and unfortunately kills patients under our care.MethodCollected data using a standardised form for 131 patients from 3 inpatient mental health units on documentation of a VTE risk assessment in the inpatient notes. For those patients who had a documented risk assessment, further data were collected on documentation of contraindications, eisk factors, sign and date of prescription and the appropriateness of prescribing.Conclusion8% of patients from one mental health unit (n = 48) had a documented risk assessment in the notes. The subsections of documented risk assessment including contraindications, risk factors, sign and date of prescriptions and appropriateness of prescribing were complete at 100%. No patients from the other 2 mental health units (n = 39,44) had a risk assessment documented in the notes.Recommendations: All adult inpatients in Mental Health units must receive a venous thrombo-embolism risk assessment. This must be documented on the Inpatient Medication Chart. Consider adding a risk assessment checklist tool mapped from the Department of Health guidelines into the Mental Health Inpatient Clerking in pro-forma.

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Zimmermann, Oliver, Jan Torzewski, Ekkehard Reichenbach-Klinke, and Christine Zenk. "Large diverticulum of the urinary bladder: A rare cause of deep vein thrombosis with consecutive pulmonary embolism." Canadian Urological Association Journal 9, no. 5-6 (May 13, 2015): 321. http://dx.doi.org/10.5489/cuaj.2533.

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A 73-year-old man was admitted with progressive dyspnea; he also had benign prostatic hyperplasia (BPH). An angio computed tomography scan showed pulmonary embolism with thrombi in both main pulmonary arteries. By duplex ultrasonography, we detected a thrombus in the right vena femoralis superficialis and vena femoralis communis. Simultaneously, we also noticed a large diverticulum on the right side of the urinary bladder and urinary stasis II of the left kidney. We consider the BPH as the trigger for a secondary diverticulum of the urinary bladder. As a result of its large dimensions, mechanical compression of the deep right pelvic veins resulted in thrombosis which finally caused the pulmonary embolism. With respect to the urinary stasis II, surgical excavation of the diverticulum with infravesical desobstruction was planned. The potentially lethal course of large diverticula may require surgery.

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Ushijima, Akiko, Taichi Komai, Atsuko Masukawa, Keiko Oikawa, Norishige Morita, Satomi Asai, Saki Mukai, Nobuo Okumura, Yoshinori Kobayashi, and Hayato Miyachi. "Hypodysfibrinogenemia with a Heterozygous Mutation of γCys326Ser by the Novel Transversion of TGT to TCT in a Patient with Pulmonary Thromboembolism and Right Ventricular Thrombus." Cardiology 137, no. 3 (2017): 167–72. http://dx.doi.org/10.1159/000457899.

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We encountered a 45-year-old Japanese man who suffered from pulmonary thromboembolism and huge right ventricular thrombus after inferior vena cava (IVC) filter implantation without apparent thrombus in either the deep veins or inside the IVC filter. The biochemical data showed a discrepancy in the level of fibrinogen between the immunological and thrombin time methods, suggesting hypodysfibrinogenemia. The sequencing of the fibrinogen γ-chain gene (FGG) revealed a novel heterozygous missense mutation in exon 8 - a TGT to TCT transversion in codon 326 - resulting in an amino acid substitution of serine for cysteine (γCys326Ser). The characterization of the protein did not show known mechanisms for thrombosis in dysfibrinogenemia, such as dimer or albumin-binding complex formation. In summary, the current case with a life-threatening thrombotic event was found to have a novel heterozygous missense mutation resulting in γCys326Ser, which was suggested as a predisposing factor of the thrombosis. Known mechanisms responsible for thrombosis in the current case were not demonstrated, suggesting other mechanisms including superimposing inherited and/or acquired risk factors. When a patient presents with unusual thrombosis such as breakthrough pulmonary embolism and huge thrombus in the right ventricle, as in the current case, the laboratory process for heritable thrombophilia should be considered.

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Barbaryan, Aram, Stefania Bailuc, Travis Abicht, Sergey Barsamyan, Yonatan Gizaw, and Aibek E. Mirrakhimov. "Saddle Pulmonary Embolus Caught in Transit across a Patent Foramen Ovale." Case Reports in Pulmonology 2019 (May 2, 2019): 1–6. http://dx.doi.org/10.1155/2019/5747598.

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Impending paradoxical embolism (IPE) also described in the literature as thrombus straddling a patent foramen ovale (PFO) or paradoxical embolus in transit is a rare condition when thrombus (originating mostly in deep veins of lower extremities) embolized to the heart gets caught in PFO or in atrial septal defect without systemic embolization. We present a case of a 39-year-old female on oral contraceptive pills who presented to the emergency department with chief complaint of dyspnea and chest pain. She was found to have saddle pulmonary embolus (PE) extending through PFO to left atrium and into the left ventricle. Patient underwent emergent open pulmonary embolectomy, removal of right and left atrial thrombi, and closure of patent foramen ovale. She tolerated the surgery well and was discharged home on chronic anticoagulation therapy.

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Tejpal, Ambika, Peter Economopoulos, Roseann Andreou, and James Stevenson. "Vaccine-Induced Immune Thrombotic Thrombocytopenia after Receiving the ChAdOx1 nCoV-19 Vaccine." Canadian Journal of General Internal Medicine 16, no. 2 (June 21, 2021): 34–37. http://dx.doi.org/10.22374/cjgim.v16i2.559.

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Vaccine-induced prothrombotic immune thrombocytopenia (VIPIT) is a rare complication noted after immunization against coronavirus disease with the ChAdOx1 nCoV-19 (AstraZeneca) vaccine. Here we report such a case in a 61-year-old gentleman who presented twenty days after receiving the vaccine and was diagnosed with massive bilateral pulmonary emboli with a saddle embolus. This is the third such case in Canada. This case serves to highlight that venous thromboembolism in an older, male patient presenting in a typical manner can still be VIPIT, despite reports of this occurring predominantly in young females with intracranial and splanchnic thromboses. It is important to make the diagnosis accurately and in a timely fashion because the management strategy differs from that of classic venous thromboembolism. Heparin anticoagulants should be avoided, and intravenous immunoglobulin is critical to consider as part of the treatment. RésuméLa thrombocytopénie thrombotique immunitaire induite par le vaccin (TTIV) est une complication rare, signalée après l’immunisation contre la maladie à coronavirus par le vaccin ChAdOx1 nCoV-19 (AstraZeneca). Cet article expose le cas d’un homme de 61 ans qui s’est présenté à l’hôpital 20 jours après avoir reçu le vaccin et a reçu un diagnostic d’embolie pulmonaire bilatérale massive avec une embolie en selle. Il s’agit du troisième cas du genre au Canada. Ce cas vient démontrer qu’un homme plus âgé qui présente les symptômes typiques d’une thromboembolie veineuse peut être atteint d’une TTIV, même si l’on rapporte qu’elle survient essentiellement chez les jeunes femmes atteintes de thromboses intracrâniennes et splanchniques. Il est important de poser le diagnostic rapidement et avec précision, car la stratégie de prise en charge de la TTIV diffère de celle de la thromboembolie veineuse classique. Les anticoagulants à base d’héparine doivent être évités, et il est essentiel de considérer l’administration d’immunoglobulines par voie intraveineuse dans le cadre du traitement.

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Grandi, L., R. A. Grandi, C. D. Tomasi, J. L. Da Rocha, V. Cardoso, and F. Dal-Pizzol. "Acute and chronic consequences of polidocanol foam injection in the lung in experimental animals." Phlebology: The Journal of Venous Disease 28, no. 8 (May 6, 2013): 441–44. http://dx.doi.org/10.1258/phleb.2012.012120.

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Objective To assess the presence of pulmonary embolism and inflammation after polidocanol foam injection into the peripheral veins of rabbits. Method The animals were treated with polidocanol foam (1 or 3 mg/kg) or vehicle. Early (15 minutes) and late (30 days) animals were evaluated by perfusional lung scintigraphy and histopathological examination. Results In the control group no alterations were found. After polidocanol foam injection it was observed that an important reduction of pulmonary perfusion in the early periods, was mainly in the left lung ( P < 0.001), with consequent embolism in the histological evaluation. In late periods it was observed that the presence of thrombus was with fibrin in small veins, compatible with chronic thrombus and the presence of chronic pulmonary inflammation. Conclusions The injection of polidocanol foam in experimental animals can induce venous embolism and chronic inflammatory infiltration.

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von Hodenberg, E., C. Zerweck, M. Knittel, T. Zeller, and T. Schwarz. "Endovenous laser ablation of varicose veins with the 1470 nm diode laser using a radial fiber – 1-year follow-up." Phlebology: The Journal of Venous Disease 30, no. 2 (November 29, 2013): 86–90. http://dx.doi.org/10.1177/0268355513512825.

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Background: Endovenous laser ablation is one of the most accepted treatment options for insufficient great and small saphenous veins. The aim of this study was to investigate the long-term efficacy and safety of the radial fiber (ELVeS-radial kit™) for the 1470 nm diode laser in a 1-year follow-up. Methods: A total of 308 lower limbs with primary insufficiency of great and small saphenous veins or insufficient tributaries were included in the prospective observational cohort study. The primary efficacy endpoint of the study was ultrasonographic proven elimination of venous reflux after at least 1 year. Secondary efficacy and further safety end points after 1 year were as follows: (1) sonographic exclusion of recanalization of the treated vein segments, (2) deep vein thrombosis, clinical pulmonary embolism or superficial vein thrombosis as defined by objective testing, (3) death from any cause, (4) persistent clinical complaints such as pain and paresthesia, (5) recurrent varicose veins. Patient satisfaction was assessed using a CIVIQ-2 questionnaire after 1 year. Results: Follow-up could be completed in 91.2% of the patients. Excellent efficacy numbers with 99.6% occlusion of the treated varicose veins as elimination of reflux could be demonstrated. After 1 year, 96% of the treated veins disappeared completely sonographically; one recanalization was observed. No deep vein thrombosis or pulmonary embolism occurred, three superficial vein thrombosis were diagnosed in follow-up examinations. Four patients died, not related to pulmonary embolism. No persistent pain or paresthesia occurred in the follow-up. Recurrent varicose veins were diagnosed in 10 patients (2.81%). Conclusion: One-year follow-up showed that endovenous laser treatment of varicose veins with 1470 nm diode laser using the radial fiber is highly effective, also regarding in a 1-year follow-up.

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Lorut, C., G. Meyer, C. Leroyer, and F. Parent. "La maladie thrombo-embolique veineuse." Revue des Maladies Respiratoires 22, no. 1 (February 2005): 169–73. http://dx.doi.org/10.1016/s0761-8425(05)85452-x.

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Hunt, N., R. K. Strachan, A. N. Nicolaides, and K. T. Delis. "Incidence, Natural History and Risk Factors of Deep Vein Thrombosis in Elective Knee Arthroscopy." Thrombosis and Haemostasis 86, no. 09 (2001): 817–21. http://dx.doi.org/10.1055/s-0037-1616137.

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Summary Aims: to determine the incidence, anatomical distribution and extent of deep vein thrombosis (DVT) in limbs undergoing elective unilateral knee arthroscopy without active prophylaxis, to evaluate its effect on venous function following early diagnosis, and to quantify the impact of risk factors on its incidence. Methods: 102 consecutive patients undergoing unilateral knee arthroscopy without prophylaxis were studied. A history was obtained with emphasis on the risk factors for thromboembolism, and physical examination and colour duplex were performed prior to and within a week after surgery. Patients who developed calf DVT were given aspirin (150 mg) and compression stockings; those with proximal DVT were admitted for anticoagulation (heparin followed by warfarin). Follow-up (mean 118 [range 84-168] days) entailed weekly physical and duplex examinations during the first month and monthly thereafter. Results: 8 patients developed calf DVT in the operated leg (incidence 7.84% [95% Cl: 2.7%-13.2%]); thrombosis was asymptomatic in 4 of those (50%), caused calf tenderness in 4 (50%) and a positive Homan’s sign in one (12.5%). DVT occurred in the following veins: peroneal 4 subjects (50%), soleal 4 (50%), gastrocnemial 2 (25%) and tibial 2 (25%). Propagation of a calf DVT to the popliteal vein was identified in 1 patient (12.5%). After a median period of 118 days, total clot lysis was found in 50% of DVTs, with partial thrombus resorption in the rest; reflux in the thrombosed veins was present in 75% of limbs with DVT. 43% of patients had 1 risk factor for DVT and 20% had ≥2. The incidence of DVT was higher amongst those with two or more risk factors for thromboembolism (p <.05) or those with previous thrombosis alone (p <.005). Symptoms or signs of pulmonary embolism were not documented. Conclusions: Elective unilateral knee arthroscopy performed without prophylaxis is complicated by ipsilateral calf DVT in 7.8% (95% CI: 2.7%-13.2%) of cases. The risk is higher in the presence of previous thrombosis (relative risk: 8.2) and two or more risk factors for DVT (relative risk: 2.94). Thrombosis may propagate to the proximal veins, despite early diagnosis. 50% of calf clots totally lyse in 4 months, yet reflux develops in at least 75% of limbs with DVT. Further studies to determine optimal prophylaxis are warranted.

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Journal articles on the topic 'Embolie pulmonaire Thrombose Veines'

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