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Journal articles on the topic 'Endocrine abnormalities'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Osborn, Irene. "Endocrine Abnormalities." Anesthesiology Clinics of North America 5, no. 3 (1987): 521–29. http://dx.doi.org/10.1016/s0889-8537(21)00331-x.

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van Durme, Caroline MPG, Jérôme MH Kisters, Pieter van Paassen, Ronald W. van Etten, and Jan Willem Cohen Tervaert. "Multiple endocrine abnormalities." Lancet 378, no. 9790 (2011): 540. http://dx.doi.org/10.1016/s0140-6736(11)60831-3.

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3

VULLO, C., V. SANCTIS, M. KATZ, et al. "Endocrine Abnormalities in Thalassemia." Annals of the New York Academy of Sciences 612, no. 1 Sixth Cooley' (1990): 293–310. http://dx.doi.org/10.1111/j.1749-6632.1990.tb24317.x.

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4

Williams, D. J. "Malakoplakia and endocrine abnormalities." Postgraduate Medical Journal 63, no. 745 (1987): 1007. http://dx.doi.org/10.1136/pgmj.63.745.1007.

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5

Björntorp, Per. "Endocrine abnormalities of obesity." Metabolism 44 (September 1995): 21–23. http://dx.doi.org/10.1016/0026-0495(95)90315-1.

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6

Marinova, Elena, and Radina Dimitrova. "Endocrine abnormalities in thalassemia major." Varna Medical Forum 8, no. 1 (2019): 29. http://dx.doi.org/10.14748/vmf.v8i1.5795.

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7

Peiris, Alan, and Ahmed Kissebah. "Endocrine Abnormalities in Morbid Obesity." Gastroenterology Clinics of North America 16, no. 3 (1987): 389–98. http://dx.doi.org/10.1016/s0889-8553(21)00303-4.

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8

Shanks, Gabriella, Vinita Mishra, and Stanka Nikolova. "Endocrine abnormalities in lithium toxicity." Clinical Medicine 17, no. 5 (2017): 434–36. http://dx.doi.org/10.7861/clinmedicine.17-5-434.

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9

Freudenthal, Bernard. "Endocrine abnormalities in lithium toxicity." Clinical Medicine 18, no. 1 (2018): 110.1–110. http://dx.doi.org/10.7861/clinmedicine.18-1-110.

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10

Jain, Ankit, S. Ramakrishanan, and Rajesh Khadgawat. "Endocrine abnormalities in dilated cardiomyopathy." Journal of the Practice of Cardiovascular Sciences 1, no. 3 (2015): 247. http://dx.doi.org/10.4103/2395-5414.177235.

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11

Lawson, Elizabeth A., and Anne Klibanski. "Endocrine abnormalities in anorexia nervosa." Nature Clinical Practice Endocrinology & Metabolism 4, no. 7 (2008): 407–14. http://dx.doi.org/10.1038/ncpendmet0872.

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12

DONCKIER, J. E. "ECG abnormalities of endocrine origin." Heart 85, no. 6 (2001): 679. http://dx.doi.org/10.1136/heart.85.6.679.

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13

Douglas, R. H., and Norman Umphenour. "Endocrine Abnormalities and Hormonal Therapy." Veterinary Clinics of North America: Equine Practice 8, no. 1 (1992): 237–49. http://dx.doi.org/10.1016/s0749-0739(17)30478-9.

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14

Sklar, Charles A. "Craniopharyngioma: Endocrine Abnormalities at Presentation." Pediatric Neurosurgery 21, no. 1 (1994): 18–20. http://dx.doi.org/10.1159/000120856.

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15

Burra, Patrizia. "Liver abnormalities and endocrine diseases." Best Practice & Research Clinical Gastroenterology 27, no. 4 (2013): 553–63. http://dx.doi.org/10.1016/j.bpg.2013.06.014.

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16

Pelletier, F., A. Mirchi, FK Cayami, et al. "P.004 Endocrine and growth abnormalities in 4H leukodystrophy patients with a molecular diagnosis." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 43, S2 (2016): S22. http://dx.doi.org/10.1017/cjn.2016.110.

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Background: 4H or POLR3-related leukodystrophy is an autosomal recessive disorder characterized by hypomyelination, hypodontia and hypogonadotropic hypogonadism caused by mutations in POLR3A, POLR3B and POLR1C. The endocrine abnormalities have never been systematically studied. Methods: A cross sectional international multicenter study was performed and the following variables were assessed: weight, height, head circumference, pubertal history, hormone levels and neurological and non-neurological features. Data was analyzed to determine whether there was a correlation between the presence of e

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17

Morshed, Md Shahed. "Endocrine and metabolic aspects of dengue syndrome." Journal of Association of Clinical Endocrinologist and Diabetologist of Bangladesh 2, no. 2 (2025): 66–70. https://doi.org/10.3329/jacedb.v2i2.78446.

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Dengue virus infection is increasing in frequency as well as its complications. Profound/prolonged dengue shock syndrome causes metabolic acidosis leading to different metabolic and electrolyte abnormalities. Endocrine glands may be involved by shock, bleeding, and inflammation and may present with transient abnormalities in hormone levels. This unusual expanded dengue syndrome may require close follow up for proper management. Besides, diabetes mellitus and obesity are risk factors for severe dengue and mortality. This review article summarizes the endocrine and metabolic aspects of dengue in

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18

Verrotti, Alberto, Mariapaola Ciampani, Mariapina Pomilio, Giuseppe Latini, and Francesco Chiarelli. "Endocrine Abnormalities Induced by Antiepileptic Drugs." Current Pediatric Reviews 2, no. 1 (2006): 85–92. http://dx.doi.org/10.2174/157339606775518313.

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19

Kelner, Michael J., and Nicholas M. Alexander. "Endocrine Hormone Abnormalities in Amanita Poisoning." Journal of Toxicology: Clinical Toxicology 25, no. 1-2 (1987): 21–37. http://dx.doi.org/10.3109/15563658708992611.

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20

Mehr, A. J., B. M. Paulus, and K. T. Weber. "A VARIANT OF MULTIPLE ENDOCRINE ABNORMALITIES." Journal of Investigative Medicine 55, no. 1 (2007): S257. http://dx.doi.org/10.1097/00042871-200701010-00577.

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21

Leavey, Sean F., and William F. Weitzel. "Endocrine abnormalities in chronic renal failure." Endocrinology and Metabolism Clinics of North America 31, no. 1 (2002): 107–19. http://dx.doi.org/10.1016/s0889-8529(01)00006-8.

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22

El-Reshaid, Kamel A. M., Abass A. Hakim, Hana A. Hourani, and Mandalam S. Seshadri. "Endocrine Abnormalities in Patients with Amyloidosis." Renal Failure 16, no. 6 (1994): 725–30. http://dx.doi.org/10.3109/08860229409044902.

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23

Lawrence, Cara, Irene Hong-McAtee, Bryan Hall, et al. "Endocrine abnormalities in townes-brocks syndrome." American Journal of Medical Genetics Part A 161, no. 9 (2013): 2266–73. http://dx.doi.org/10.1002/ajmg.a.36104.

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24

Kuczera, Piotr, Marcin Adamczak, and Andrzej Wiecek. "Endocrine Abnormalities in Patients with Chronic Kidney Disease." PRILOZI 36, no. 2 (2015): 109–18. http://dx.doi.org/10.1515/prilozi-2015-0059.

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Abstract In patients with chronic kidney disease the alterations of the endocrine system may arise from several causes. The kidney is the site of degradation as well as synthesis of many different hormones. Moreover, a number of concomitant pathological conditions such as inflammation, metabolic acidosis and malnutrition may participate in the pathogenesis of endocrine abnormalities in this group of patients. The most pronounced endocrine abnormalities in patients with chronic kidney disease are the deficiencies of: calcitriol, testosterone, insulin-like growth factor and, erythropoietin (EPO)

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25

Rosenfeld, Cheryl R., and S. Sethu K. Reddy. "Endocrine and Metabolic Manifestations of HIV Infection and AIDS." Journal of Intensive Care Medicine 16, no. 4 (2001): 161–68. http://dx.doi.org/10.1177/088506660101600401.

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Since the first descriptions of acquired immune deficiency syndrome (AIDS) and human immunodeficiency virus (HIV), a great deal of knowledge has been accumulated on how these entities interact with the endocrine system. Recently new information has been received on autoimmune dysregulation causing thyroid abnormalities after highly active antiretroviral therapy (HAART), as well as metabolic dysregulation occurring in the setting of protease inhibitors. We review the pathophysiologic abnormalities of the endocrine systems due to HIV infection as well as the endocrine effects of drugs commonly u

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26

Newman, Connie B., Michael J. Blaha, Jeffrey B. Boord, et al. "Lipid Management in Patients with Endocrine Disorders: An Endocrine Society Clinical Practice Guideline." Journal of Clinical Endocrinology & Metabolism 105, no. 12 (2020): 3613–82. http://dx.doi.org/10.1210/clinem/dgaa674.

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Abstract Objective This guideline will provide the practicing endocrinologist with an approach to the assessment and treatment of dyslipidemia in patients with endocrine diseases, with the objective of preventing cardiovascular (CV) events and triglyceride-induced pancreatitis. The guideline reviews data on dyslipidemia and atherosclerotic cardiovascular disease (ASCVD) risk in patients with endocrine disorders and discusses the evidence for the correction of dyslipidemia by treatment of the endocrine disease. The guideline also addresses whether treatment of the endocrine disease reduces ASCV

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27

Stosel, Helen, та Glenn D. Braunstein. "Endocrine Abnormalities Associated with Langerhansʼ Cell Histiocytosis". Endocrinologist 1, № 6 (1991): 393–98. http://dx.doi.org/10.1097/00019616-199112000-00007.

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28

Schoeters, Greet, Elly Den Hond, Willem Dhooge, Nik Van Larebeke, and Marike Leijs. "Endocrine Disruptors and Abnormalities of Pubertal Development." Basic & Clinical Pharmacology & Toxicology 102, no. 2 (2008): 168–75. http://dx.doi.org/10.1111/j.1742-7843.2007.00180.x.

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29

SAITO, Shiro. "Pathophysiology and Gene Abnormalities of Endocrine Tumors." Folia Endocrinologica Japonica 68, no. 12 (1992): 1225–39. http://dx.doi.org/10.1507/endocrine1927.68.12_1225.

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30

Franchini, Massimo, Giuseppe Lippi, Franco Manzato, Pier Paolo Vescovi, and Giovanni Targher. "Hemostatic abnormalities in endocrine and metabolic disorders." European Journal of Endocrinology 162, no. 3 (2010): 439–51. http://dx.doi.org/10.1530/eje-09-0958.

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The hemostatic balance is a complex system where the delicate equilibrium is regulated by several factors, including hormones. This review summarizes current knowledge of the effects of most frequent endocrine and metabolic diseases (such as hypothyroidism, hyperthyroidism, Cushing's syndrome, GH-related pituitary dysfunctions, pituitary prolactin-producing adenomas, polycystic ovary syndrome, primary hyperparathyroidism, and metabolic syndrome) on coagulation and fibrinolysis. Overt hypothyroidism appears to be associated with a bleeding tendency, whereas all other endocrine diseases appear t

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31

Giri, Neelam, Dalia L. Batista, Blanche P. Alter, and Constantine A. Stratakis. "Endocrine Abnormalities in Patients with Fanconi Anemia." Journal of Clinical Endocrinology & Metabolism 92, no. 7 (2007): 2624–31. http://dx.doi.org/10.1210/jc.2007-0135.

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Abstract Background: Fanconi anemia (FA) is an inherited disorder with chromosomal instability, bone marrow failure, developmental defects, and a predisposition to cancer. Systematic and comprehensive endocrine function data in FA are limited. Objective: We studied a cohort of FA patients enrolled in the National Cancer Institute’s Inherited Bone Marrow Failure Syndrome study. Study Design and Patients: Retrospective review of the medical records of 45 FA patients (ages 2–49 yr), 23 of whom were intensively evaluated at the National Institutes of Health. Anthropometric measurements, GH, IGF-I,

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32

Franceschini, R., G. L. Tenconi, F. Zoppoli, and T. Barreca. "Endocrine abnormalities and outcome of ischaemic stroke." Biomedicine & Pharmacotherapy 55, no. 8 (2001): 458–65. http://dx.doi.org/10.1016/s0753-3322(01)00086-5.

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33

Hörl, W. H., W. Riegel, C. Wanner, et al. "Endocrine and metabolic abnormalities following kidney transplantation." Klinische Wochenschrift 67, no. 17 (1989): 907–18. http://dx.doi.org/10.1007/bf01717348.

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34

Polgreen, Lynda, Melissa Plog, Paul Orchard, Bradley Miller, Anna Petryk, and Chester B. Whitley. "40 Endocrine abnormalities in patients with mucopolysaccharidoses." Molecular Genetics and Metabolism 92, no. 4 (2007): 20–21. http://dx.doi.org/10.1016/j.ymgme.2007.08.045.

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35

Rajiwade, Siddhesh R., Haritha Sagili, R. Soundravally, and L. Subitha. "Endocrine Abnormalities in Adolescents with Menstrual Disorders." Journal of Obstetrics and Gynecology of India 68, no. 1 (2017): 58–64. http://dx.doi.org/10.1007/s13224-017-1035-y.

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36

Wacharasindhu, S., U. Asawutmangkul, and S. Srivuthana. "Endocrine Abnormalities in Patients with Frontoethmoidal Encephalomeningocele." Hormone Research in Paediatrics 64, no. 2 (2005): 64–67. http://dx.doi.org/10.1159/000087691.

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37

Oerter, Karen E., Theodore C. Friedman, Hans C. Anderson, and Fernando G. Cassorla. "Familial syndrome of endocrine and neuroectodermal abnormalities." American Journal of Medical Genetics 44, no. 4 (1992): 487–91. http://dx.doi.org/10.1002/ajmg.1320440421.

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38

Bongsebandhu-phubhakdi, Chansuda, Therdpong Tempark, and Vichit Supornsilpchai. "Endocrine manifestations of PHACE syndrome." Journal of Pediatric Endocrinology and Metabolism 32, no. 8 (2019): 797–802. http://dx.doi.org/10.1515/jpem-2019-0126.

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Abstract PHACE syndrome is an uncommon disorder of posterior fossa anomalies, cervicofacial infantile hemangiomas, arterial anomalies, cardiac defects, eye anomalies, and midline/ventral defects. Endocrine abnormalities including hypopituitarism and ectopic thyroid were rarely described. In this article we review occurrence, onset, presenting symptoms, hormonal treatments and outcomes of all endocrine abnormalities in PHACE syndrome. Eleven of 20 (55%) had hypothalamic-pituitary dysfunction and 10 of 20 (50%) had thyroid dysgenesis. A thorough understanding of the endocrine manifestations is i

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39

Nonglait, Phibakordor L., Srivenkata Madhu, Nishant Raizada, Amitesh Aggarwal, Rafat Ahmed, and Mohammad Aslam. "Abnormalities of Endocrine Function Tests in COVID-19-Recovered Individuals." Indian Journal of Endocrinology and Metabolism 29, no. 3 (2025): 325–31. https://doi.org/10.4103/ijem.ijem_32_25.

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Abstract Introduction: Impact of COVID-19 on endocrine system due to the widespread distribution of Angiotensin Converting Enzyme-2 (ACE2) receptors in different endocrine organs of the body has been shown in several studies. However, most of these studies were in the setting of acute COVID-19. The present study was planned to comprehensively evaluate endocrine abnormalities in COVID-19-recovered individuals. Methods: Eighty-three COVID-19-recovered individuals were recruited 8-20 weeks following the recovery. They were further stratified according to disease severity as defined by the Indian

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40

Hill, J., C. Elliott, and I. Colquhoun. "Audiological, vestibular and radiological abnormalities in Kallman's syndrome." Journal of Laryngology & Otology 106, no. 6 (1992): 530–34. http://dx.doi.org/10.1017/s0022215100120067.

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AbstractKallman's syndrome is a multifaceted congenital disorder with predominantly endocrine abnormalities. We have characterized the associated mixed hearing loss and identified consistent radiological evidence of abnormal temporal bone anatomy. Abnormal labyrinthine morphology is accompanied by a complete absence of response to vestibular stimulation with caloric or rotational chair testing.The endocrine abnormalities are correctable and Kallman's syndrome is a diagnosis worthy of consideration when assessing children with congenital hearing loss.

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41

Rattan, Saniya, Changqing Zhou, Catheryne Chiang, Sharada Mahalingam, Emily Brehm, and Jodi A. Flaws. "Exposure to endocrine disruptors during adulthood: consequences for female fertility." Journal of Endocrinology 233, no. 3 (2017): R109—R129. http://dx.doi.org/10.1530/joe-17-0023.

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Endocrine disrupting chemicals are ubiquitous chemicals that exhibit endocrine disrupting properties in both humans and animals. Female reproduction is an important process, which is regulated by hormones and is susceptible to the effects of exposure to endocrine disrupting chemicals. Disruptions in female reproductive functions by endocrine disrupting chemicals may result in subfertility, infertility, improper hormone production, estrous and menstrual cycle abnormalities, anovulation, and early reproductive senescence. This review summarizes the effects of a variety of synthetic endocrine dis

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42

Iguchi, Taisen, and Tomomi Sato. "Endocrine Disruption and Developmental Abnormalities of Female Reproduction1." American Zoologist 40, no. 3 (2000): 402–11. http://dx.doi.org/10.1668/0003-1569(2000)040[0402:edadao]2.0.co;2.

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43

Diegel, Cassandra R., Steven Hann, Ugur M. Ayturk, et al. "An osteocalcin-deficient mouse strain without endocrine abnormalities." PLOS Genetics 16, no. 5 (2020): e1008361. http://dx.doi.org/10.1371/journal.pgen.1008361.

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44

Brinker, Mark R., Daniel P. O??Connor, Yomna T. Monla, and Thomas P. Earthman. "Metabolic and Endocrine Abnormalities in Patients With Nonunions." Journal of Orthopaedic Trauma 21, no. 8 (2007): 557–70. http://dx.doi.org/10.1097/bot.0b013e31814d4dc6.

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45

&NA;. "Undiagnosed Metabolic and Endocrine Abnormalities May Explain Nonunions." Lippincott's Bone and Joint Newsletter 14, no. 1 (2008): 8–9. http://dx.doi.org/10.1097/01.bonej.0000305503.91042.37.

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46

Iguchi, Taisen, and Tomomi Sato. "Endocrine Disruption and Developmental Abnormalities of Female Reproduction." American Zoologist 40, no. 3 (2000): 402–11. http://dx.doi.org/10.1093/icb/40.3.402.

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47

Brinker, Mark R., Daniel P. OʼConnor, Yomna T. Monla, and Thomas P. Earthman. "Metabolic and endocrine abnormalities in patients with nonunions." Current Orthopaedic Practice 19, no. 4 (2008): 430–42. http://dx.doi.org/10.1097/bco.0b013e32830b8711.

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48

Suzuki, T., H. Sasano, T. Sawai, et al. "Small Adrenocortical Tumors without Apparent Clinical Endocrine Abnormalities." Pathology - Research and Practice 188, no. 7 (1992): 883–89. http://dx.doi.org/10.1016/s0344-0338(11)80248-7.

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49

Elovic, Elie, Hima S. Doppalapudi, Michelle Miller, et al. "Endocrine Abnormalities and Fatigue After Traumatic Brain Injury." Journal of Head Trauma Rehabilitation 21, no. 5 (2006): 426–27. http://dx.doi.org/10.1097/00001199-200609000-00044.

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50

Haimovici, Robert, Shimon Rumelt, and James Melby. "Endocrine abnormalities in patients with central serous chorioretinopathy." Ophthalmology 110, no. 4 (2003): 698–703. http://dx.doi.org/10.1016/s0161-6420(02)01975-9.

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