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Books on the topic 'Endocrine resistance'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Cohen, Margo Panush, and Piero P. Foà, eds. Hormone Resistance and Other Endocrine Paradoxes. Springer New York, 1987. http://dx.doi.org/10.1007/978-1-4612-4758-6.

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2

P, Cohen Margo, and Foà Piero P. 1911-, eds. Hormone resistance and other endocrine paradoxes. Springer-Verlag, 1987.

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3

Galland, Leo. The fat resistance diet: Reprogram your body to stay thin forever. Rodale, 2008.

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4

Evanthia, Diamanti-Kandarkis, ed. Insulin resistance and polycystic ovarian syndrome: Pathogenesis, evaluation, and treatment. Humana Press, 2007.

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5

Berns, P. M. J. J., Romijn J. C, and Schröder F. H, eds. Mechanisms of progression to hormone-independent growth of breast and prostatic cancer. Parthenon Pub. Group, 1991.

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6

Banyameen, Hene. Coping with pollution: Alteration in metabolic gene expression among sensitive, resistant, and hybrid Fundulus heterolitus embryos during organogenesis. National University, 2015.

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7

Cohen, Margo P., and Piero P. Foa. Hormone Resistance and Other Endocrine Paradoxes. Springer, 2012.

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8

Cohen, Margo P., and Piero P. Foa. Hormone Resistance and Other Endocrine Paradoxes. Springer, 2011.

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9

Hormone Resistance and Other Endocrine Paradoxes. Springer-Verlag Berlin and Heidelberg GmbH & Co. K, 1987.

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10

Cohen, Margo P., and Piero P. Foa. Hormone Resistance and Other Endocrine Paradoxes. Island Press, 1987.

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11

Lifshitz, Fima. Pediatric Endocrinology: Obesity, Diabetes Mellitus, Insulin Resistance, and Hypoglycemia. Taylor & Francis Group, 2006.

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12

Lifshitz, Fima. Pediatric Endocrinology: Obesity, Diabetes Mellitus, Insulin Resistance, and Hypoglycemia. Taylor & Francis Group, 2006.

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13

Beck-Peccoz, Paolo. Syndromes of Hormone Resistance on the Hypothalamic-Pituitary-Thyroid Axis (Endocrine Updates). Springer, 2004.

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14

Wiffen, Philip, Marc Mitchell, Melanie Snelling, and Nicola Stoner. Therapy-related issues: endocrine system. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199603640.003.0021.

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Diabetes mellitus 444Monitoring and control 449Thyroid disorders 450Diabetes mellitus (DM) affects approximately 4% of the UK population. In 2009, Diabetes UK reported that 2.6 million people in the UK have diabetes.Type 2 diabetes accounts for 90% of all diabetes and is a result of insulin resistance and pancreatic β-cell dysfunction. Type 1 diabetes results from an absolute insulin deficiency secondary to autoimmune dysfunction....

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15

Cabot, Sandra. Can't Lose Weight?: Unlock the Secrets That Keep You Fat! Ten Speed Press, 2002.

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16

Panidis, Dimitrios, Evanthia Diamanti-Kandarakis, John E. Nestler, and Renato Pasquali. Insulin Resistance and Polycystic Ovarian Syndrome: Pathogenesis, Evaluation, and Treatment. Humana Press, 2010.

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17

Goldie, James H., and Nicholas Bruchovsky. Drug and Hormone Resistance in Neoplasia: Volume 2 Clinical Concepts. Taylor & Francis Group, 2019.

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18

Goldie, James H., and Nicholas Bruchovsky. Drug and Hormone Resistance in Neoplasia: Volume 2 Clinical Concepts. Taylor & Francis Group, 2019.

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19

Goldie, James H., and Nicholas Bruchovsky. Drug and Hormone Resistance in Neoplasia: Volume 2 Clinical Concepts. Taylor & Francis Group, 2019.

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20

Goldie, James H., and Nicholas Bruchovsky. Drug and Hormone Resistance in Neoplasia: Volume 2 Clinical Concepts. Taylor & Francis Group, 2019.

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21

Drug and Hormone Resistance in Neoplasia: Volume 2 Clinical Concepts. Taylor & Francis Group, 2019.

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22

(Editor), John E. Nestler, Evanthia Diamanti-Kandarakis (Editor), Renato Pasquali (Editor), and Dimitrios Panidis (Editor), eds. Insulin Resistance and Polycystic Ovarian Syndrome: Pathogenesis, Evaluation, and Treatment (Contemporary Endocrinology). Humana Press, 2007.

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23

Jolly, Elaine, Andrew Fry, and Afzal Chaudhry, eds. Diabetes. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199230457.003.0008.

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Chapter 8 covers the basic science and clinical topics relating to the endocrine system which trainees are required to learn as part of their basic training and demonstrate in the MRCP. It covers diabetes mellitus, diabetic emergencies, diabetes-long-term management, diabetic retinopathy, diabetic neuropathy, diabetic nephropathy, the diabetic foot, diabetic skin, the diabetic pregnancy, and metabolic syndrome and insulin resistance.

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24

Lancellotti, Patrizio, and Bernard Cosyns. Systemic Disease and Other Conditions. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713623.003.0017.

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This chapter describes the effect of various activities on the heart and associated disorders. It details the echocardiographic findings of athlete’s heart and differential diagnosis. It considers pregnancy which induces several haemodynamic changes: increase in heart rate, stroke volume, cardiac output, and decrease in systemic vascular resistance. Several echocardiographic changes may also present in normal pregnancy and these must be recognized. Echocardiography should be performed in each pregnant woman with cardiac signs or symptoms to search for new cardiac disease occurring during pregn

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25

C Diaz, Eva, Celeste C Finnerty, and David N. Herndon. Severe Burn Injuries and Their Long-Term Implications. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0016.

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Burn injury is notable for the degree and duration of pathophysiological alterations that it induces. Burn triggers profound changes in metabolism, immune function, and endocrine function, leading to a host of negative effects, including catabolism of muscle and bone and insulin resistance. These changes may persist or evolve for years after the injury has occurred, delaying recovery. This chapter discusses all of these consequences of burn injury, along with other adverse outcomes, specifically growth delay in children and hypertrophic scarring. Particular attention is placed on what is known

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26

Dussaule, Jean-Claude, Martin Flamant, and Christos Chatziantoniou. Function of the normal glomerulus. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0044_update_001.

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Glomerular filtration, the first step leading to the formation of primitive urine, is a passive phenomenon. The composition of this primitive urine is the consequence of the ultrafiltration of plasma depending on renal blood flow, on hydrostatic pressure of glomerular capillary, and on glomerular coefficient of ultrafiltration. Glomerular filtration rate (GFR) can be precisely measured by the calculation of the clearance of freely filtrated exogenous substances that are neither metabolized nor reabsorbed nor secreted by tubules: its mean value is 125 mL/min/1.73 m² in men and 110 mL/min/1.73 m

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