Relevant bibliographies by topics / Endogenous ROS

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Endogenous ROS'

Author: Grafiati
Published: 6 September 2023
Last updated: 31 July 2025

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Journal articles on the topic "Endogenous ROS"

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Supruniuk, Elżbieta, Jan Górski, and Adrian Chabowski. "Endogenous and Exogenous Antioxidants in Skeletal Muscle Fatigue Development during Exercise." Antioxidants 12, no. 2 (2023): 501. http://dx.doi.org/10.3390/antiox12020501.

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Muscle fatigue is defined as a decrease in maximal force or power generated in response to contractile activity, and it is a risk factor for the development of musculoskeletal injuries. One of the many stressors imposed on skeletal muscle through exercise is the increased production of reactive oxygen species (ROS) and reactive nitrogen species (RNS), which intensifies as a function of exercise intensity and duration. Exposure to ROS/RNS can affect Na+/K+-ATPase activity, intramyofibrillar calcium turnover and sensitivity, and actin–myosin kinetics to reduce muscle force production. On the oth
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Lu, Qing-Bin. "Reaction Cycles of Halogen Species in the Immune Defense: Implications for Human Health and Diseases and the Pathology and Treatment of COVID-19." Cells 9, no. 6 (2020): 1461. http://dx.doi.org/10.3390/cells9061461.

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There is no vaccine or specific antiviral treatment for COVID-19, which is causing a global pandemic. One current focus is drug repurposing research, but those drugs have limited therapeutic efficacies and known adverse effects. The pathology of COVID-19 is essentially unknown. Without this understanding, it is challenging to discover a successful treatment to be approved for clinical use. This paper addresses several key biological processes of reactive oxygen, halogen and nitrogen species (ROS, RHS and RNS) that play crucial physiological roles in organisms from plants to humans. These inclu
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Sharma, Ajay Kumar, Harshit Singh, and Harinath Chakrapani. "Photocontrolled endogenous reactive oxygen species (ROS) generation." Chemical Communications 55, no. 36 (2019): 5259–62. http://dx.doi.org/10.1039/c9cc01747j.

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Pan, Zhixiang, Jun Zhang, Kaili Ji, Vayou Chittavong, Xingyue Ji, and Binghe Wang. "Organic CO Prodrugs Activated by Endogenous ROS." Organic Letters 20, no. 1 (2017): 8–11. http://dx.doi.org/10.1021/acs.orglett.7b02775.

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Hole, Paul S., Lorna Pearn, Amanda J. Tonks, et al. "Ras-induced reactive oxygen species promote growth factor–independent proliferation in human CD34+ hematopoietic progenitor cells." Blood 115, no. 6 (2010): 1238–46. http://dx.doi.org/10.1182/blood-2009-06-222869.

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Abstract Excessive production of reactive oxygen species (ROS) is a feature of human malignancy and is often triggered by activation of oncogenes such as activated Ras. ROS act as second messengers and can influence a variety of cellular process including growth factor responses and cell survival. We have examined the contribution of ROS production to the effects of N-RasG12D and H-RasG12V on normal human CD34+ progenitor cells. Activated Ras strongly up-regulated the production of both superoxide and hydrogen peroxide through the stimulation of NADPH oxidase (NOX) activity, without affecting
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Federico, Cacciapuoti. "Oxidative Stress as "Mother" of Many Human Diseases at Strong Clinical Impact." Journal of Cardiovascular Medicine and Cardiology 3, no. 1 (2016): 001–6. https://doi.org/10.17352/2455-2976.000020.

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Likewise the 1991 Gulf War, known as a “mother of all battles”, oxidative stress (OS) can be considered as a “mother” of many human diseases life threatening. OS is a condition in which oxidation exceeds the anti-oxidant reactions, causing an imbalance between oxidative and anti-oxidant systems, with prevalence of reactive oxygen species ROS [1-5]. These include: peroxide, superoxide, hydroxyl radical, singlet oxygen and others. Under normal conditions ROS are maintained at physiological levels by several endogenous antioxidant systems, as superoxide dismutase, catalase
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HALVEY, Patrick J., Walter H. WATSON, Jason M. HANSEN, Young-Mi GO, Afshin SAMALI, and Dean P. JONES. "Compartmental oxidation of thiol–disulphide redox couples during epidermal growth factor signalling." Biochemical Journal 386, no. 2 (2005): 215–19. http://dx.doi.org/10.1042/bj20041829.

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Exogenously added ROS (reactive oxygen species) cause generalized oxidation of cellular components, whereas endogenously generated ROS induced by physiological stimuli activate discrete signal transduction pathways. Compartmentation is an important aspect of such pathways, but little is known about its role in redox signalling. We measured the redox states of cytosolic and nuclear Trx1 (thioredoxin-1) and mitochondrial Trx2 (thioredoxin-2) using redox Western blot methodologies during endogenous ROS production induced by EGF (epidermal growth factor) signalling. The glutathione redox state was
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Koval, M., S. T. Geist, E. M. Westphale, et al. "Transfected connexin45 alters gap junction permeability in cells expressing endogenous connexin43." Journal of Cell Biology 130, no. 4 (1995): 987–95. http://dx.doi.org/10.1083/jcb.130.4.987.

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Many cells express multiple connexins, the gap junction proteins that interconnect the cytosol of adjacent cells. Connexin43 (Cx43) channels allow intercellular transfer of Lucifer Yellow (LY, MW = 443 D), while connexin45 (Cx45) channels do not. We transfected full-length or truncated chicken Cx45 into a rat osteosarcoma cell line ROS-17/2.8, which expresses endogenous Cx43. Both forms of Cx45 were expressed at high levels and colocalized with Cx43 at plasma membrane junctions. Cells transfected with full-length Cx45 (ROS/Cx45) and cells transfected with Cx45 missing the 37 carboxyl-terminal
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Sarniak, Agata, Joanna Lipińska, Karol Tytman, and Stanisława Lipińska. "Endogenous mechanisms of reactive oxygen species (ROS) generation." Postępy Higieny i Medycyny Doświadczalnej 70 (November 14, 2016): 1150–65. http://dx.doi.org/10.5604/17322693.1224259.

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Wu, Jiaye, Yue Zhang, Ruizhi Hao, Yuan Cao, Xiaoyi Shan, and Yanping Jing. "Nitric Oxide Enhances Cytotoxicity of Lead by Modulating the Generation of Reactive Oxygen Species and Is Involved in the Regulation of Pb2+ and Ca2+ Fluxes in Tobacco BY-2 Cells." Plants 8, no. 10 (2019): 403. http://dx.doi.org/10.3390/plants8100403.

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Lead is a heavy metal known to be toxic to both animals and plants. Nitric oxide (NO) was reported to participate in plant responses to different heavy metal stresses. In this study, we analyzed the function of exogenous and endogenous NO in Pb-induced toxicity in tobacco BY-2 cells, focusing on the role of NO in the generation of reactive oxygen species (ROS) as well as Pb2+ and Ca2+ fluxes using non-invasive micro-test technology (NMT). Pb treatment induced BY-2 cell death and rapid NO and ROS generation, while NO burst occurred earlier than ROS accumulation. The elimination of NO by 2-4-car
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Dissertations / Theses on the topic "Endogenous ROS"

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Singhapol, Chatchawan. "Mitochondrial localisation of hTERT protects against nuclear DNA damage and mitochondrial ROS production after endogenous and exogenous stress." Thesis, University of Newcastle upon Tyne, 2013. http://hdl.handle.net/10443/2216.

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Under oxidative stress condition, telomerase catalytic subunit can shuttle from the nucleus and localises within mitochondria. hTERT can improve mitochondrial functions and contribute to a decreased oxidative stress suggesting an entirely new function of telomerase in protecting mitochondria and cells under stress. However, there are still many questions about the mechanism and what factors influence the protective function of telomerase. In this study we investigated the kinetic exclusion of hTERT, the catalytic subunit of telomerase, in various cell lines under different oxidative stress con
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Mutneja, Manpreet S. "Endogenous RGS proteins modulate acute desensitization of GIRK currents /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2003. http://wwwlib.umi.com/cr/ucsd/fullcit?p3099926.

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Garzon, D. "HIGH RESOLUTION MASS SPECTROMETRIC STRATEGIES FOR DETECTION OF PROTEINS AND PEPTIDES COVALENTLY MODIFIED BY ELECTROPHILIC XENOBIOTICS AND ENDOGENOUS INTERMEDIATES." Doctoral thesis, Università degli Studi di Milano, 2015. http://hdl.handle.net/2434/250677.

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Non enzymatic protein covalent modifications are involved in the toxic effects induced by electrophilic xenobiotics as well as by endogenous cytotoxic oxidation by-products. Aim of my Ph.D work was to set-up MS methods for the identification, characterization and quantification of non-enzymatic covalently modified proteins and peptides in biological matrices. To reach this goal both tandem MS and high resolution approaches were employed due to the wealth of structural and molecular information that these techniques can provide. As a first step the MS methods were applied for understanding in
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Mourão, Carlos Manuel Pombo Martins. "Enzimas antioxidantes e patologias neurodegenerativas: estado da arte." Master's thesis, 2013. http://hdl.handle.net/10284/7043.

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A literatura apresenta uma evidência crescente de que o stress oxidativo promovido pelas espécies oxigénio-reativas (ROS) pode ter um papel vital na etiologia das doenças neurodegenerativas como a doença de Alzheimer, a doença de Parkinson ou a esquizofrenia, entre outras. As ROS possuem um papel duplo, tanto benéfico quanto prejudicial, no organismo humano. Estas são geralmente reguladas por enzimas antioxidantes endógenas para que o balanço oxidativo seja mantido mas, por factores endógenos e exógenos, pode ocorrer uma alteração com produção excessiva que resulta em stress oxidativo. Este st
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Shee, Somnath. "Manipulating Bacterial and Host Reactive Oxygen Species (ROS)- based mechanisms to potentiate killing of Mycobacterium tuberculosis (Mtb)." Thesis, 2021. https://etd.iisc.ac.in/handle/2005/5680.

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Mycobacterium tuberculosis (Mtb) is evolutionarily equipped to resist exogenous reactive oxygen species but shows vulnerability to an increase in endogenous ROS (eROS). Since eROS is an unavoidable consequence of aerobic metabolism, understanding how eROS levels are controlled is essential yet remains uncharacterized. By combining the Mrx1-roGFP2 redox biosensor with transposon mutagenesis, we identified 368 genes (redoxosome) responsible for maintaining non-toxic levels of eROS in Mtb. Integrating redoxosome with a global network of protein-protein interactions and transcriptional regulators
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Lin, Xu Hong, and 林旭宏. "Effects of endogenous carotenoids and hemoglobin on lipid oxidation and browning of dried mullet roe." Thesis, 1996. http://ndltd.ncl.edu.tw/handle/52511642557435067505.

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Books on the topic "Endogenous ROS"

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Abidde, Sabella Ogbobode, ed. Africa-China-Taiwan Relations, 1949–2020. Rowman & Littlefield, 2022. https://doi.org/10.5040/9781666982961.

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The ongoing tension and hostility between China and Taiwan in Africa are a continuation of the Chinese Civil War (1927-1949) between the forces of the Chinese Communist Party (CCP) which remained in mainland China, and the Kuomintang (KMT) of the Republic of China (ROC) which fled to the island of Taiwan. In the intervening years, China has claimed Taiwan as part of its territory and through persistent and aggressive political and economic efforts convinced much of the world to accept her as the sole and legitimate seat of the Chinese people and government. Africa-China-Taiwan Relations, 1949-
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Book chapters on the topic "Endogenous ROS"

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Elliott, Michael H., and Abboud J. Ghalayini. "Phosphorylation of Caveolin-1 in Bovine Rod Outer Segments in vitro by an Endogenous Tyrosine Kinase." In Advances in Experimental Medicine and Biology. Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-74904-4_39.

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Sempéré, Antoine J., Robert Mauget, and André Lacroix. "Seasonal Regulation of the Sexual Cycle and Antler Growth in Roe Deer: Evidence for an Endogenous Rhythm." In The Biology of Deer. Springer New York, 1992. http://dx.doi.org/10.1007/978-1-4612-2782-3_119.

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Di Girolamo, M., T. Cacciamani, M. Pallas, and D. Corda. "Activities Involved in the Modulation of the G Protein Endogenous ADP-Ribosylation in Normal and K-ras-Transformed Thyroid Cells." In Molecular Oncology and Clinical Applications. Birkhäuser Basel, 1993. http://dx.doi.org/10.1007/978-3-0348-5663-8_20.

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Kaczmarczyk, Jan A., Gordon R. Whiteley, and Josip Blonder. "Detection and Quantitation of Endogenous Membrane-Bound RAS Proteins and KRAS Mutants in Cancer Cell Lines Using 1D-SDS-PAGE LC–MS2." In Methods in Molecular Biology. Springer US, 2024. http://dx.doi.org/10.1007/978-1-0716-3922-1_17.

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Ahsan, Haseeb, Mohammad Yusuf Hasan, and Rizwan Ahmad. "Reactive Oxygen Species (ROS) in the Pathophysiology of Rheumatoid Arthritis (RA)." In Reactive Oxygen Species [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.101333.

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Free radicals are highly reactive molecules that are unstable and have extremely short-short half-life. They are derived from either oxygen (reactive oxygen species, ROS) or nitrogen (reactive nitrogen species, RNS) in mitochondria, plasma membrane and endoplasmic reticulum due to oxidative stress and damage. ROS/RNS are physiologically useful at low concentrations and are responsible for the activation of redox-sensitive signaling pathways, phagocytosis of infected cells and removal of abnormal and aging cells. The endogenous sources of ROS are the electron transport chain, the respiratory bu
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Gupta, Dr Madhu. "Endogenous Antioxidants-I." In Edited Book of Dietary Supplements and Nutraceuticals [According to latest syllabus of B. Pharm-VIII Semester of Pharmacy Council of India]. Iterative International Publishers, Selfypage Developers Pvt Ltd, 2024. http://dx.doi.org/10.58532/nbennurdsch12.

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Endogenous antioxidants are critical components of the body's defense system against oxidative stress caused by free radicals. These antioxidants, both enzymatic and nonenzymatic, work together to neutralize reactive oxygen species (ROS) and prevent cellular damage. The enzymatic antioxidant defense includes key enzymes such as superoxide dismutase (SOD) and catalase, which play pivotal roles in detoxifying harmful free radicals. Superoxide dismutase (SOD) is one of the primary enzymatic antioxidants, responsible for catalyzing the dismutation of the superoxide radical into oxygen and hydrogen
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Kageyama, Hakuto. "Biological Activities of MAAs and their Applications 2: Antioxidative Properties." In An Introduction to Mycosporine-Like Amino Acids. BENTHAM SCIENCE PUBLISHERS, 2023. http://dx.doi.org/10.2174/9789815136081123010008.

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It is known that the generation of reactive oxygen species (ROS) caused by UV irradiation and oxidative reactions accelerate skin aging. Substances that suppress or eliminate the generation of ROS are called antioxidants. So far, various mycosporine-like amino acids (MAAs) have been reported to have antioxidative activities. To prevent damage to the skin caused by ROS and maintain the homeostasis of the epidermis, skin cells have an endogenous antioxidant system consisting of enzymatic reactions. Although many points are unclear about the regulatory mechanisms, it has been suggested that MAAs
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Gallo, Giuseppe. "FREE RADICALS AND ANTIOXIDANTS: WHO THEY ARE, WHO GENERATES THEM AND WHAT IS THE MECHANISM OF ACTION." In Research in Biological Sciences - Challenges and Opportunities. Iterative International Publishers, Selfypage Developers Pvt Ltd, 2024. http://dx.doi.org/10.58532/nbennurrbch9.

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Oxidative stress it is a form of "chemical stress" induced in our organism by an imbalance between the production of highly reactive chemical species (free radicals) and the level of physiological defense capabilities (antioxidants). Free Radicals (ROS) are molecules that, due to their instability, are constantly looking for other molecules to attach to, like molecular sharks. While the antioxidants are "scavengers", molecular, exogenous scavengers and endogenous. They swallow‖ excess radicals, preventing them from damaging the body. But sometimes they can be genetically altered (endogenous) o
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Reyed, Reyed M., and P. K. Prabhakar. "Impact of Dietary Antioxidants on Redox Homeostasis and Its Effects on Health and Disease." In Advances in Medical Diagnosis, Treatment, and Care. IGI Global, 2025. https://doi.org/10.4018/979-8-3693-7919-6.ch003.

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This chapter discusses the roles of reactive oxygen species (ROS) in cells, including their role in oxidative stress and redox balance. It explores how dietary antioxidants affect redox homeostasis, their sources, mechanisms of action, and antioxidant capacity. The chapter also discusses how antioxidants affect ROS-mediated signaling cascades and endogenous antioxidant defense mechanisms. It discusses the use of antioxidants in pharmacological approaches to treat chronic illnesses like cancer, neurological disorders, and cardiometabolic diseases. It also addresses the antioxidant paradox, wher
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Eddaikra, Atika, and Naouel Eddaikra. "Endogenous Enzymatic Antioxidant Defense and Pathologies." In Antioxidants - Benefits, Sources, Mechanisms of Action. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.95504.

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Oxidative stress is an important component of various diseases. It manifests as an imbalance caused by an excessive production of reactive oxygen species (ROS) which are associated with a deficit of antioxidant activity. This deficit can be the consequence of genetic factors, environmental ones, metabolic imbalance, toxicity or direct attacks by the accumulation of free radicals. These can induce metabolic dysfunction affecting biological macromolecules in their structures or activities. From a physiological perspective, the neutralization of free radicals is ensured by enzymatic, antioxidant
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Conference papers on the topic "Endogenous ROS"

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Filippova, Maria. "Abstract 3766: The response of cervical cancer cells to chemotherapeutic agents depends on endogenous ROS levels as well as the drug-induced ROS profile." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-3766.

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Rabbani, Naila, Paul John Thornalley, Maryam Al-Motawa, and Mingzhan Xue. "Vulnerabilities of the SARS-Cov-2 Virus to Proteotoxicity – Opportunity for Repurposed Chemotherapy of COVID-19 Infection." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0291.

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The global pandemic of COVID-19 disease caused by infection with the SARS-CoV-2 Coronavirus, has produced an urgent requirement and search for improved treatments whilst effective vaccines are developed. A strategy for improved drug therapy is to increase levels of endogenous reactive metabolites for selective toxicity to SARS-CoV-2 by preferential damage to the viral proteome. Key reactive metabolites producing major quantitative damage to the proteome in physiological systems are: Reactive oxygen species (ROS) and the reactive glycating agent methylglyoxal (MG); cysteine residues and arginin
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Yi Fang, Chin. "EXAMINING THE ENDOGENEITY OF ESG PRACTICE AND FIRM PERFORMANCE OF SPORT-RELATED ENTERPRISES: THE MODERATING EFFECT OF R&D INTENSITY." In 2025 SoRes Singapore – International Conference on Interdisciplinary Research in Social Sciences, 03-04 March. Global Research & Development Services Publishing, 2025. https://doi.org/10.20319/icssh.2025.275276.

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The number of sports-related enterprises has increased; however, there is little research on the factors influencing the performance of these enterprises and the sustainability of their companies. Enterprises have expanded their focus from pursuing financial returns to embracing social responsibility and sustainable development, marking a transition from corporate social responsibility to Environmental, Social, and Governance (ESG). Hence, the objectives of this paper are (1) establishing an efficiency index incorporating multiple inputs/outputs for sports-related companies; (2) examining the
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Hermán, F., P. Hadházy, and K. Magyar. "QUANTITATIVE DETERMINATION OF ENDOGENOUS PGI2 RELEASE IN ANAESTHETIZED BEAGLE DOGS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643184.

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A 30μm diameter pore size screen was inserted into an arteriovenous bypass system in anaesthetized, heparin-treated beagle dogs. The arterial blood was directed through the screen by a roller pump at a constant rate. As a result, the pressure proximal to the filter continuously increased (F.Hermdn et al.Thromb. Res.44 /1986/,575). The concentration of proximally infused PGI2 that stabilized the filtration pressure curve (pressure stabilizing concentration= PSC) was determined. If it was low enough (between 0.4 and 1.5 nmol/1) we administered the PGI2-releaser bradykinin (1/ug/kg), angiotensin
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Roth, Sascha O. M., Thomas J. Macartney, Agnieszka Konopacka, Markus Queisser, and Gopal P. Sapkota. "Abstract B34: Targeted destruction of endogenous K-RAS using an Affinity directed PROtein Missile (AdPROM)." In Abstracts: AACR Special Conference on Targeting RAS-Driven Cancers; December 9-12, 2018; San Diego, CA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1557-3125.ras18-b34.

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Chi, Xin-Zi, You-Soub Lee, and Suk-Chul Bae. "Abstract B35: Runx3 contributes for the defense against endogenous level of oncogenic K-Ras-induced tumorigenesis." In Abstracts: AACR International Conference: New Frontiers in Cancer Research; January 18-22, 2017; Cape Town, South Africa. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.newfront17-b35.

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Mohammed, Altaf, Naveena B. Janakiram, Misty Brewer, et al. "Abstract A111: Endogenous conversion of n-6 PUFAs to n-3 PUFAs prevents progression of K-Ras activated pancreatic lesion development to pancreatic ductal carcinoma in Fat-1 transgenic mice." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Oct 22-25, 2011; Boston, MA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1940-6207.prev-11-a111.

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Carvalho, Tamyres MIngorance, Tayana Schultz Jukoski, Guillermo Ortiz Brasil, Flavia Kuroda, and Enilze M. S. F. Ribeiro. "EXPRESSION OF miRNAS SUGGESTS A POTENTIAL ROLE IN BREAST CANCER." In Scientifc papers of XXIII Brazilian Breast Congress - 2021. Mastology, 2021. http://dx.doi.org/10.29289/259453942021v31s1050.

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Introduction: MicroRNAs (miRNAs) are regulators of gene expression in biological processes, mainly repressing translation or degrading messenger RNA (mRNA) from their target genes. Their deregulation is associated with a wide variety of diseases, including cancer. Breast cancer (BC) is the most common cancer among women worldwide. Understanding the mechanisms involved in this pathology is essential for the discovery of new diagnostic and prognostic markers. Objectives: Investigate the differential expression of selected miRNAs in luminal A (LA) and triple-negative breast cancer (TNBC). Methods
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Miguel Marques, Fernando. "“ALImentáRIO” - Holder for the Supplementary Feeding of Wild Birds." In 13th International Conference on Applied Human Factors and Ergonomics (AHFE 2022). AHFE International, 2022. http://dx.doi.org/10.54941/ahfe1001397.

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This is an animal design project (design thinking to meet the needs of animals) which aims to bring birds into contact with humans and - through this solution - to bring the sound and movement produced by them into our lives (whether in the countryside, whether in the urban landscape), which can be translated into joy and peace achieved by the harmony of natural wildlife in our domestic environment.Starting question Knowing that birds are elusive and fearful animals, but also can bring joy and movement to our daily lives, how can we attract them to our coexistence? Supplementary feeding can be
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Reports on the topic "Endogenous ROS"

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Friedman, Haya, Chris Watkins, Susan Lurie, and Susheng Gan. Dark-induced Reactive Oxygen Species Accumulation and Inhibition by Gibberellins: Towards Inhibition of Postharvest Senescence. United States Department of Agriculture, 2009. http://dx.doi.org/10.32747/2009.7613883.bard.

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Dark-induced senescence could pose a major problem in export of various crops including cuttings. The assumption of this work was that ROS which is increased at a specific organelle can serve as a signal for activation of cell senescence program. Hormones which reduce senescence in several crops like gibberellic acid (GA) and possibly cytokinin (CK) may reduce senescence by inhibiting this signal. In this study we worked on Pelargonium cuttings as well as Arabidopsis rosette. In Pelargonium the increase in ROS occurred concomitantly with increase in two SAGs, and the increase persisted in isol
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Sritularak, Boonchoo, Kittisak Likhitwitayawuid, Pithi Chanvorachote, and Varisa Pongrakhananon. Cytotoxic constituents from Dendrobium spp. : Final report. Chulalongkorn University, 2014. https://doi.org/10.58837/chula.res.2014.28.

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The MeOH extracts of Dendrobium pullchellum Roxb. Ex Lind1. And D. ellipsophyllum Tang & wang (Orchidaceae) showed significant cytotoxic activity against human lung cancer cells. Phytochemical study of the whole plant of D. pullchellum Roxb. Ex Lind1 led to the isolation of seven phenolic compounds, namely, chrysotobibenzyl, chrysotoxine, crepidatin, moscatilin, fimbriatone, (-) –shikimic acid and liriodendrin. The whole plant of D. ellipsophyllum Tang & wang yielded ten phenolic compounds indluding moscatilin, 5,7-dihydroxy-chromen-4-one, 4,5-dihydroxy-2,3-dimethoxy-9,10-dihydrophenan
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Estevadeordal, Antoni. Negotiating Preferential Market Access: The Case of NAFTA. Inter-American Development Bank, 1999. http://dx.doi.org/10.18235/0011086.

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There is a growing interest related to the theoretical analysis of Free Trade Agreements (FTAs). Nevertheless, there has been as yet very little empirical research on the topic, in particular, on the negotiating dynamics of these types of agreements. This paper attempts to make a contribution in this direction examining the relationship between the two most important market access instruments in the case of NAFTA negotiations: the preferential tariff phase-outs and the accompanying rules of origin (RoO). The traditional literature has viewed market access negotiations solely in terms of tariff
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Straume, Odd Rune, and Paulo Bastos. Globalization, Product Differentiation and Wage Inequality. Inter-American Development Bank, 2010. http://dx.doi.org/10.18235/0010990.

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This paper develops a two-country, general equilibrium model of oligopoly in which the degree of horizontal product differentiation is endogenously determined by rms strategic investments in product innovation. Consumers seek variety and product innovation is more skill intensive than production. Stronger import competition increases innovation incentives, and thereby the relative demand for skill. An intraindustry trade expansion following trade liberalization can therefore increase wage inequality between skilled and unskilled workers. In addition, since product differentiation is resource c
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Sands, Ronald, and Man-Keun Kim. Modeling the Competition for Land: Methods and Application to Climate Policy. GTAP Working Paper, 2008. http://dx.doi.org/10.21642/gtap.wp45.

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*Chapter 7 of the forthcoming book "Economic Analysis of Land Use in Global Climate Change Policy," edited by Thomas W. Hertel, Steven Rose, and Richard S.J. Tol The Agriculture and Land Use (AgLU) model was developed at Pacific Northwest National Laboratory to assess the impact of a changed climate or a climate policy on land use, carbon emissions from land use change, production of field crops, and production of biofuels. The level of analysis to date is relatively aggregate, at the global or national scale, but the model captures important interactions such as endogenous land use change in
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6

Calderón, César, Alberto E. Chong, and Ernesto H. Stein. Trade Intensity and Business Cycle Synchronization: Are Developing Countries any Different? Inter-American Development Bank, 2003. http://dx.doi.org/10.18235/0010806.

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Some key criteria in the optimal currency area literature are that countries should join a currency union if they have closer international trade links and more symmetric business cycles. However, both criteria are endogenous. Frankel and Rose (1998) find that trade intensity increases cycle correlation among industrial countries. We study whether the same result holds true for the case of developing countries, as their different patterns of international trade and specialization may lead to cyclical asymmetries among them and between industrial and developing countries. We gather annual infor
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