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Relevant bibliographies by topics / Endomysial antibody

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Academic literature on the topic 'Endomysial antibody'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Endomysial antibody"

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Chan, K. N., A. D. Phillips, R. Mirakian, and J. A. Walker‐Smith. "Endomysial Antibody Screening in Children." Journal of Pediatric Gastroenterology and Nutrition 18, no. 3 (1994): 316–20. http://dx.doi.org/10.1002/j.1536-4801.1994.tb11181.x.

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It has been suggested that endomysial antibodies are specific markers for coeliac disease. In a 13‐month study, we examined the usefulness of screening for these antibodies in the diagnosis of coeliac disease in children. Twenty‐one of 223 (9.4%) serum samples [or 17 of 192 (9%) children undergoing investigation for GI disorders] were found to be positive for serum IgA class endomysial antibodies. These included eight strong positives, eight positives, and five weak positives. One‐hundred‐thirty‐four children had small bowel biopsies performed. Endomysial antibodies were found in all children

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Chan, K. N., A. D. Phillips, R. Mirakian, and J. A. Walker-Smith. "Endomysial Antibody Screening in Children." Journal of Pediatric Gastroenterology and Nutrition 18, no. 3 (1994): 316–20. http://dx.doi.org/10.1097/00005176-199404000-00011.

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Murray, Joseph A., Judith Herlein, Frank Mitros, and James A. Goeken. "Serologic Testing for Celiac Disease in the United States: Results of a Multilaboratory Comparison Study." Clinical Diagnostic Laboratory Immunology 7, no. 4 (2000): 584–87. http://dx.doi.org/10.1128/cdli.7.4.584-587.2000.

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ABSTRACT The aim of this study was to compare the efficiencies of six reference laboratories for serologic testing for celiac disease. Serum from 20 patients with untreated celiac disease and from 20 controls was thawed, divided, and distributed to each participating laboratory, which performed endomysial antibody tests. Five laboratories also performed antigliadin antibody tests. Sensitivity for endomysial antibody immunoglobulin A (IgA) varied from 57 to 90%. In all laboratories, the specificity for celiac disease was 100%. The sensitivity and specificity for both IgA and IgG antigliadin ant

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4

Karabey, Mehmet, Havva Kaya, Alperen Ceylan, Kadir Kaba, Mehmet Özdemir, and Bahadır Feyzioğlu. "The effect of the pandemic on autoantibody rates in the general population." Archives of Rheumatology 39, no. 4 (2024): 541–48. https://doi.org/10.46497/archrheumatol.2024.10330.

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Objectives: The study aimed to investigate the possible effects of coronavirus disease 2019 (COVID-19) on autoantibodies. Patients and methods: Samples of 89,108 individuals (29,033 males, 60,075 females; median: 36 years; range, 0 to 96 years) who underwent autoimmune testing between January 2017 and May 2022 were retrospectively analyzed. The prepandemic period was defined as May 1, 2017, to March 20, 2020, while the pandemic period was defined as March 20, 2020, to May 31, 2022. Results: Of the participants, 0.55% were of foreign nationality. The positivity rate was 18.12%. Autoantibody pos

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Picarelli, Antonio, Luigi Sabbatella, and Marco Di Tola. "Endomysial antibody production after in vitro gliadin challenge." European Journal of Gastroenterology & Hepatology 13, no. 2 (2001): 213. http://dx.doi.org/10.1097/00042737-200102000-00022.

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Ciclitira, P. J., F. Biagi, H. J. Ellis, and N. D. G. Parnell. "Endomysial antibody production after in vitro gliadin challenge." European Journal of Gastroenterology & Hepatology 13, no. 2 (2001): 214. http://dx.doi.org/10.1097/00042737-200102000-00023.

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Peleg, Roni, Z. Itzhak Ben-Zion, Aya Peleg, et al. "“Bread madness” revisited: screening for specific celiac antibodies among schizophrenia patients." European Psychiatry 19, no. 5 (2004): 311–14. http://dx.doi.org/10.1016/j.eurpsy.2004.06.003.

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AbstractPurposeA possible association between gluten consumption and schizophrenia has been reported. The objective was to compare patients with chronic schizophrenia and matched controls for sociodemographic variables, prevalence of celiac-specific anti-endomysial antibodies and disease-related variables.Subjects and methodsThe study group was comprised of 50 consecutive patients diagnosed with schizophrenia, 18 years of age and older attending the out-patient clinic of the Mental Health Center in Beer-Sheva, Israel. The control group was comprised of mentally normal volunteers who came to pr

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Biagi, F., P. I. Bianchi, J. Campanella, et al. "The prevalence and the causes of minimal intestinal lesions in patients complaining of symptoms suggestive of enteropathy: a follow-up study: Table 1." Journal of Clinical Pathology 61, no. 10 (2008): 1116–18. http://dx.doi.org/10.1136/jcp.2008.060145.

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Aims:Although they are non-specific, minimal intestinal lesions are at the end of the coeliac histological damage spectrum. To investigate whether minimal intestinal lesions in patients without endomysial antibodies are due to coeliac disease, their prevalence, causes and risk of evolving into frank coeliac disease were studied.Methods:From January 2000 to December 2005, 645 duodenal biopsies were performed. In 209 patients, duodenal biopsies were performed independently of endomysial antibody results. Clinical data and HLA-typing of all the patients negative to endomysial antibodies but with

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Anbardar, Mohammad Hossein, Neda Soleimani, Ehsan Torabi Dashtaki, Naser Honar, Mozhgan Zahmatkeshan, and Sahand Mohammadzadeh. "Do Serological Tests Eliminate the Need for Endoscopic Biopsy for the Diagnosis of Symptomatic Patients with Celiac Disease? A Retrospective Study with Review of Literature." Middle East Journal of Digestive Diseases 15, no. 4 (2023): 263–69. http://dx.doi.org/10.34172/mejdd.2023.356.

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Background: Celiac disease is one of the most common genetic allergies worldwide. The prevalence of celiac disease in Iran is similar to or even higher than the global prevalence. Celiac disease is a chronic inflammatory disease that affects the small intestine. Affected patients are allergic to gluten protein that exists in some grains, such as wheat and barley. Methods: Serological endomysial IgA antibody (EMA-AB) and tissue transglutaminase IgA antibody (TTG-IgA) tests were performed on 114 patients aged the ages of 0–18 years with histopathological findings of celiac disease. The results o

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Aktay, Atiye N., P. C. Lee, Vijay Kumar, Elaine Parton, David T. Wyatt, and Steven L. Werlin. "The Prevalence and Clinical Characteristics of Celiac Disease in Juvenile Diabetes in Wisconsin." Journal of Pediatric Gastroenterology and Nutrition 33, no. 4 (2001): 462–65. http://dx.doi.org/10.1002/j.1536-4801.2001.tb07502.x.

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ABSTRACTBackgroundThe relationship between celiac disease and juvenile diabetes has long been known. Only a single study in the United States, from Buffalo, New York, has reported the prevalence of celiac disease in a pediatric diabetic population. This study was conducted to determine the prevalence and clinical presentation of celiac disease in children and adolescents with juvenile diabetes in Wisconsin, U.S.A., using serum antiendomysial antibody as a screening test.MethodsTwo hundred eighteen patients with diabetes (113 males; age range, 4–21 years) and 117 age‐and gender‐matched control

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Book chapters on the topic "Endomysial antibody"

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Beutner, Ernst H., Olavi Hällström, Tadeusz P. Chorzelski, Vijay Kumar, Annemarie Bürgin-Wolff, and Jadwiga Sulej. "Standardization of Endomysial, Reticulin, and Gliadin Antibody Tests for the Diagnosis of Gluten-Sensitive Enteropathy." In Serologic Diagnosis of Celiac Disease. CRC Press, 2020. http://dx.doi.org/10.1201/9780429281839-7.

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Conference papers on the topic "Endomysial antibody"

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Lima, GLN, JR Campos, AB Fernandes, EDL Cabral, and LS Santos. "VERIFICAÇÃO DE DESEMPENHO DO REAGENTE PARA DETECÇÃO DE ANTICORPOS ANTIENDOMÍSIO IGA E IGG, UTILIZANDO SUBSTRATO CONTENDO FÍGADO DE MACACO." In Resumos do 54º Congresso Brasileiro de Patologia Clínica/Medicina Laboratorial. Zeppelini Editorial e Comunicação, 2022. http://dx.doi.org/10.5327/1516-3180.140s1.6649.

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Objetivo: Avaliar o desempenho do kit comercial para detecção de anticorpos antiendomísio IgA e IgG, utilizando substrato de fígado de macaco. Método: Foram incluídas 85 amostras previamente analisadas no substrato de esôfago, sendo caracterizadas como: 36 não reagentes e seis reagentes para endomísio IgA; 41 não reagentes e duas reagentes para endomísio IgG. As lâminas contendo substrato fígado de macaco foram preparadas no equipamento Sprinter XL, utilizando kit Euroimmun® pelo método de imunofluorescência indireta para posterior leitura em microscópio de ultravioleta. Para análise estatísti

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