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Books on the topic 'Enteric pathogen'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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1

Farrah, Samuel R. Inactivation of enteric pathogens during aerobic digestion of wastewater sludge. U.S. Environmental Protection Agency, Water Engineering Research Laboratory, 1986.

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2

Farrah, Samuel R. Inactivation of enteric pathogens during aerobic digestion of wastewater sludge. U.S. Environmental Protection Agency, Water Engineering Research Laboratory, 1986.

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3

Sterling, Charles R., and Rodney D. Adam, eds. The Pathogenic Enteric Protozoa: Giardia, Entamoeba, Cryptosporidium and Cyclospora. Springer US, 2004. http://dx.doi.org/10.1007/b113653.

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4

S, Paul Prem, Francis David H, and International Rushmore Conference on Mechanisms in the Pathogenesis of Enteric Diseases (2nd : 1998 : Rapid City, S.D.), eds. Mechanisms in the pathogenesis of enteric diseases 2. Kluwer Academic/Plenum Publishers, 1999.

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5

Heufelder, George R. Survival and transport of enteric bacteria and viruses in the nearshore marine environment: An annotated bibliography. Barnstable County Health and Environmental Department, 1988.

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6

Friedman, Herman, Mauro Bendinelli, and Lois J. Paradise. Enteric Infections and Immunity. Springer, 2014.

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7

Paradise, Lois J. Enteric Infections and Immunity. Springer, 2013.

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8

Friedman, Herman, Mauro Bendinelli, and Lois J. Paradise. Enteric Infections and Immunity. Springer, 2013.

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9

Sterling, Charles R., and Rodney D. Adam. Pathogenic Enteric Protozoa: Giardia, Entamoeba, Cryptosporidium and Cyclospora. Springer, 2014.

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10

Sterling, Charles R., and Rodney D. Adam. Pathogenic Enteric Protozoa : : Giardia, Entamoeba, Cryptosporidium and Cyclospora. Springer London, Limited, 2006.

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11

(Editor), Charles R. Sterling, and Rodney D. Adam (Editor), eds. The Pathogenic Enteric Protozoa:: Giardia, Entamoeba, Cryptosporidium and Cyclospora (World Class Parasites). Springer, 2004.

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12

Bentley, Tony. Bacterial infection. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0310.

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13

McAdam, Alexander J. Diagnostic Testing for Enteric Pathogens, an Issue of Clinics in Laboratory Medicine 35-2. Elsevier, 2015.

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14

Zürich, Eidgenössische Technische Hochschule, ed. Effects of mannanoligosaccharide on different cecal parameters and on cecal concentrations of enteric pathogens in poultry. 1996.

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15

Scoullos, Iosif Marios. Assessment of the Fate of Surrogates for Enteric Pathogens Resulting from the Surcharging of Combined Sewer Systems. Taylor & Francis Group, 2020.

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16

Assessment of the Fate of Surrogates for Enteric Pathogens Resulting from the Surcharging of Combined Sewer Systems. Taylor & Francis Group, 2020.

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17

Scoullos, Iosif Marios. Assessment of the Fate of Surrogates for Enteric Pathogens Resulting from the Surcharging of Combined Sewer Systems. Taylor & Francis Group, 2020.

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18

Scoullos, Iosif Marios. Assessment of the Fate of Surrogates for Enteric Pathogens Resulting from the Surcharging of Combined Sewer Systems. Taylor & Francis Group, 2020.

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19

Scoullos, Iosif Marios. Assessment of the Fate of Surrogates for Enteric Pathogens Resulting from the Surcharging of Combined Sewer Systems. Taylor & Francis Group, 2020.

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20

(Editor), Prem S. Paul, and David H. Francis (Editor), eds. Mechanisms in the Pathogenesis of Enteric Diseases 2 (ADVANCES IN EXPERIMENTAL MEDICINE AND BIOLOGY Volume 473). Springer, 2000.

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21

Livingston, Schuyler, Benjamin Young, Martin Markowitz, Poonam Mathur, and Bruce L. Gilliam. HIV Virology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0017.

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HIV is a member of the lentivirus subfamily of retroviruses. Two distinct groups of viruses are pathogenic in humans: HIV-1 and HIV-2. Both are transmitted sexually and known to cause immunodeficiency disease. HIV enters the cell through use of the CD4 receptor and chemokine co-receptors, primarily CCR5 and CXCR4. The viral genome is transcribed from RNA to DNA by reverse transcriptase and integrated into the host genome by integrase. The HIV genome encodes 15 proteins, comprising three categories: structural, regulatory, and accessory. After budding from the host cell, the virus matures into

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22

Rello, Jordi, and Bárbara Borgatta. Pathophysiology of pneumonia. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0115.

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Airway colonization, ventilator-associated tracheobronchitis (VAT), and hospital-acquired (HAP) and ventilator-associated pneumonia (VAP) are three manifestations having the presence of micro-organisms in airways in common. Newer definitions have to consider worsening of oxygenation, in addition to purulent respiratory secretions, chest-X rays opacities, and biomarkers of inflammation. Bacteria are the main causes of HAP/VAP. During hospitalization there’s a shift of airway’s colonizing flora from core organisms to enteric and non-fermentative ones. Macro- and micro-aspiration is the most impo

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23

Litell, John M., and Nathan I. Shapiro. Pathophysiology of septic shock. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0297.

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The pathophysiology of sepsis is the result of a dysregulated host response to infection. Interactions between conserved pathogenic signals and host recognition systems initiate a systemic reaction to local infection. Pro- and anti-inflammatory intermediates and associated coagulatory abnormalities lead to altered macrovascular, microvascular, and mitochondrial function. Uncorrected, these processes yield similar patterns of failure in multiple organ systems. Mortality increases with successive organ failures. Although commonly thought to be a manifestation of impaired renal circulation, septi

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