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1

Kaper, James B. "Enterohemorrhagic Escherichia coli." Current Opinion in Microbiology 1, no. 1 (1998): 103–8. http://dx.doi.org/10.1016/s1369-5274(98)80149-5.

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2

Kilic, Abdullah. "Enterohemorrhagic Escherichia coli (EHEC)." TAF Preventive Medicine Bulletin 10, no. 4 (2011): 387. http://dx.doi.org/10.5455/pmb.20110823054010.

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3

Fan, K. T., G. J. Whitman, and F. S. Chew. "Enterohemorrhagic Escherichia coli colitis." American Journal of Roentgenology 166, no. 4 (1996): 788. http://dx.doi.org/10.2214/ajr.166.4.8610550.

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4

Welinder-Olsson, Christina, and Bertil Kaijser. "Enterohemorrhagic Escherichia coli (EHEC)." Scandinavian Journal of Infectious Diseases 37, no. 6-7 (2005): 405–16. http://dx.doi.org/10.1080/00365540510038523.

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5

Schnabel, Uta. "Inactivation of Escherichia coliK-12 and enterohemorrhagic Escherichia coli (EHEC) by atmospheric pressure plasma." Journal of Agricultural Science and Applications 03, no. 03 (2014): 81–91. http://dx.doi.org/10.14511/jasa.2014.030305.

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6

Oishi, Kazunori, Yuichiro Yahata, and Yukihiro Akeda. "8. Enterohemorrhagic Escherichia Coli Infection." Nihon Naika Gakkai Zasshi 102, no. 11 (2013): 2854–59. http://dx.doi.org/10.2169/naika.102.2854.

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7

Petras, Robert E. "Enterohemorrhagic Escherichia coli-Associated Colitis." Pathology Case Reviews 2, no. 2 (1997): 66–70. http://dx.doi.org/10.1097/00132583-199702020-00002.

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8

DESMARCHELIER, PATRICIA M. "Enterohemorrhagic Escherichia coli—The Australian Perspective†." Journal of Food Protection 60, no. 11 (1997): 1447–50. http://dx.doi.org/10.4315/0362-028x-60.11.1447.

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Food borne transmission of hemolytic uremic syndrome (HUS) was first reported in Australia in 1995 when an outbreak of HUS due to Escherichia coli O111 occurred following the consumption of locally produced mettwurst. Federal and state health and food authorities responded rapidly to bring the outbreak under control. Longer-term responses include the introduction by regulatory authorities of a code of practice for uncooked fermented comminuted meat products, the provision of government and industry funds to support the implementation of this code, and research into the ecology and epidemiology
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9

Orth, D., and R. Wurzner. "What Makes an Enterohemorrhagic Escherichia coli?" Clinical Infectious Diseases 43, no. 9 (2006): 1168–69. http://dx.doi.org/10.1086/508207.

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10

Benjamin, M. M., and A. R. Datta. "Acid tolerance of enterohemorrhagic Escherichia coli." Applied and environmental microbiology 61, no. 4 (1995): 1669–72. http://dx.doi.org/10.1128/aem.61.4.1669-1672.1995.

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11

SHINODA, Sumio, Shigeo YAMAMOTO, Kenichi TOMOCHIKA, and Shinichi MIYOSHI. "Enterohemorrhagic Escherichia coli O157: H7 Infection." Eisei kagaku 43, no. 1 (1997): 1–14. http://dx.doi.org/10.1248/jhs1956.43.1.

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12

IYODA, Sunao, Jun TERAJIMA, Akihito WADA, Hidemasa IZUMIYA, Kazumichi TAMURA, and Haruo WATANABE. "Molecular Epidemiology of enterohemorrhagic Escherichia coli." Nippon Saikingaku Zasshi 55, no. 1 (2000): 29–36. http://dx.doi.org/10.3412/jsb.55.29.

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13

Takeda, Yoshifumi. "Enterohemorrhagic Escherichia coli infection in Japan." Pediatrics International 41, no. 2 (1999): 198–201. http://dx.doi.org/10.1046/j.1442-200x.1999.4121050.x.

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14

TAKEDA, Yoshifumi. "Vibrio parahaemolyticus, enterotoxigenic Escherichia coli, enterohemorrhagic Escherichia coli and Vibrio cholerae." Proceedings of the Japan Academy, Series B 87, no. 1 (2011): 1–12. http://dx.doi.org/10.2183/pjab.87.1.

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15

SCHAMBERGER, GERRY P., and FRANCISCO DIEZ-GONZALEZ. "Characterization of Colicinogenic Escherichia coli Strains Inhibitory to Enterohemorrhagic Escherichia coli." Journal of Food Protection 67, no. 3 (2004): 486–92. http://dx.doi.org/10.4315/0362-028x-67.3.486.

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A previously identified set of anti– Escherichia coli O157:H7 colicinogenic E. coli were characterized to assess the suitability of these isolates as a preharvest food safety intervention in cattle. This collection of 23 E. coli strains were screened for virulence factors, antibiotic resistance, type of colicin(s) present, and their ability to inhibit other pathogenic E. coli. With the use of PCR, pathogen genes were detected in six of the 23 colicinogenic E. coli. When the nonpathogenic strains were assessed for antibiotic resistance, four strains showed resistance to at least one antibiotic.
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16

TERAJIMA, Jun, Sunao IYODA, Hidemasa IZUMIYA, et al. "Current Status of Enterohemorrhagic Escherichia coli Infections." Japanese Journal of Food Microbiology 29, no. 2 (2012): 88–93. http://dx.doi.org/10.5803/jsfm.29.88.

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17

Yi, C. r., and M. B. Goldberg. "Enterohemorrhagic Escherichia coli raises the I-BAR." Proceedings of the National Academy of Sciences 106, no. 16 (2009): 6431–32. http://dx.doi.org/10.1073/pnas.0902773106.

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18

Mellmann, Alexander, Martina Bielaszewska, and Helge Karch. "Intrahost Genome Alterations in Enterohemorrhagic Escherichia coli." Gastroenterology 136, no. 6 (2009): 1925–38. http://dx.doi.org/10.1053/j.gastro.2008.12.072.

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19

PICKERING, LARRY K., TOM G. OBRIG, and BRUDER F. STAPLETON. "Hemolytic-uremic syndrome and enterohemorrhagic Escherichia coli." Pediatric Infectious Disease 13, no. 6 (1994): 459–75. http://dx.doi.org/10.1097/00006454-199406000-00001.

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20

Verweyen, Hege M., Helge Karch, Matthias Brandis, and Lothar B. Zimmerhackl. "Enterohemorrhagic Escherichia coli infections: following transmission routes." Pediatric Nephrology 14, no. 1 (2000): 73–83. http://dx.doi.org/10.1007/s004670050018.

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21

Song, J. Y., R. H. Yoo, S. Y. Jang, et al. "Genome Sequence of Enterohemorrhagic Escherichia coli NCCP15658." Journal of Bacteriology 194, no. 14 (2012): 3749–50. http://dx.doi.org/10.1128/jb.00653-12.

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22

Békássy, Zivile D., Carla Calderon Toledo, Gustav Leoj, et al. "Intestinal damage in enterohemorrhagic Escherichia coli infection." Pediatric Nephrology 26, no. 11 (2011): 2059–71. http://dx.doi.org/10.1007/s00467-010-1616-9.

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23

Masiga, Fredrick, Edgar Kigozi, Christine Florence Najjuka, Henry Kajumbula, and David Patrick Kateete. "Diarrhoeagenic Escherichia coli isolated from children with acute diarrhoea at Rakai hospital, Southern Uganda." African Health Sciences 22, no. 1 (2022): 581–8. http://dx.doi.org/10.4314/ahs.v22i1.67.

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Background: Diarrhoeagenic Escherichia coli (DEC) is a leading cause of childhood diarrhoea. This study estimated the prevalence of DEC and DEC pathotypes among children with acute diarrhoea in Southern Uganda.
 Methods: A cross-sectional study was conducted on 267 children less than 5 years with acute diarrhoea, admitted to Rakai General Hospital in Southern Uganda. Faecal samples were collected from the children and processed for isolation of E. coli. The presence of DEC and the distribution of DEC pathotypes were determined by polymerase chain reaction.
 Results: A total of 102 (3
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24

WANG, GUODONG, and MICHAEL P. DOYLE. "Survival of Enterohemorrhagic Escherichia coli O157:H7 in Water." Journal of Food Protection 61, no. 6 (1998): 662–67. http://dx.doi.org/10.4315/0362-028x-61.6.662.

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Several recent Escherichia coli O157:H7 outbreaks associated with both drinking and recreational water raise concerns about waterborne illness caused by this pathogen. The survival characteristics of a mixture of five nalidixic acid-resistant E. coli O157:H7 strains (103 CFU/ml) in filtered and autoclaved municipal water, in reservoir water, and in water from two recreational lakes were determined for a period of 91 days at 8, 15, or 25°C. Greatest survival was in filtered autoclaved municipal water and least in lake water. Regardless of the water source, survival was greatest at 8°C and least
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25

Nataro, James P., and James B. Kaper. "Diarrheagenic Escherichia coli." Clinical Microbiology Reviews 11, no. 1 (1998): 142–201. http://dx.doi.org/10.1128/cmr.11.1.142.

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SUMMARY Escherichia coli is the predominant nonpathogenic facultative flora of the human intestine. Some E. coli strains, however, have developed the ability to cause disease of the gastrointestinal, urinary, or central nervous system in even the most robust human hosts. Diarrheagenic strains of E. coli can be divided into at least six different categories with corresponding distinct pathogenic schemes. Taken together, these organisms probably represent the most common cause of pediatric diarrhea worldwide. Several distinct clinical syndromes accompany infection with diarrheagenic E. coli cate
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26

Shan, Xueling, Takuya Yamauchi, Yojiro Yamamoto, et al. "Spontaneous and specific binding of enterohemorrhagic Escherichia coli to overoxidized polypyrrole-coated microspheres." Chemical Communications 53, no. 27 (2017): 3890–93. http://dx.doi.org/10.1039/c7cc00244k.

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27

Foster, Geoffrey, Judith Evans, Hazel I. Knight, et al. "Analysis of Feces Samples Collected from a Wild-Bird Garden Feeding Station in Scotland for the Presence of Verocytotoxin-Producing Escherichia coli O157." Applied and Environmental Microbiology 72, no. 3 (2006): 2265–67. http://dx.doi.org/10.1128/aem.72.3.2265-2267.2006.

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ABSTRACT Composite wild bird feces collected at regular intervals from a garden feeding station in southwest Scotland over a 3-year period were examined for verocytotoxin-producing Escherichia coli O157. One sample was positive for Escherichia coli O157. The isolate belonged to phage type 21/28 and possessed vtx 2, eaeA, and enterohemorrhagic E. coli hlyA genes.
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28

Ferrer, M. D., Y. Pallardó, and E. Nogués. "Enterohemorrhagic colitis caused by Escherichia coli: CT findings." American Journal of Roentgenology 169, no. 3 (1997): 912. http://dx.doi.org/10.2214/ajr.169.3.9275927.

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29

MAKINO, Sou-ichi, Hiroshi ASAKURA, Toshikazu SHIRAHATA, et al. "Enterohemorrhagic Escherichia coli O157 Outbreak in Obihiro-city." Journal of the Japanese Association for Infectious Diseases 71, no. 11 (1997): 1131–36. http://dx.doi.org/10.11150/kansenshogakuzasshi1970.71.1131.

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30

MAKINO, Sou-ichi, Hiroshi ASAKURA, Toshikazu SHIRAHATA, et al. "Enterohemorrhagic Escherichia coli O157 Outbreak in Obihiro-City." Journal of the Japanese Association for Infectious Diseases 72, no. 2 (1998): 89–96. http://dx.doi.org/10.11150/kansenshogakuzasshi1970.72.89.

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31

Garcia, A., J. G. Fox, and T. E. Besser. "Zoonotic Enterohemorrhagic Escherichia coli: A One Health Perspective." ILAR Journal 51, no. 3 (2010): 221–32. http://dx.doi.org/10.1093/ilar.51.3.221.

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32

Tosa, K., and T. Hirata. "Photoreactivation of enterohemorrhagic Escherichia coli following UV disinfection." Water Research 33, no. 2 (1999): 361–66. http://dx.doi.org/10.1016/s0043-1354(98)00226-7.

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33

Lin, J., M. P. Smith, K. C. Chapin, H. S. Baik, G. N. Bennett, and J. W. Foster. "Mechanisms of acid resistance in enterohemorrhagic Escherichia coli." Applied and environmental microbiology 62, no. 9 (1996): 3094–100. http://dx.doi.org/10.1128/aem.62.9.3094-3100.1996.

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34

Mellies, Jay L., Alex M. S. Barron, and Anna M. Carmona. "Enteropathogenic and Enterohemorrhagic Escherichia coli Virulence Gene Regulation." Infection and Immunity 75, no. 9 (2007): 4199–210. http://dx.doi.org/10.1128/iai.01927-06.

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35

Lukyanenko, Valeriy, Irina Malyukova, Ann Hubbard, et al. "EnterohemorrhagicEscherichia coliinfection stimulates Shiga toxin 1 macropinocytosis and transcytosis across intestinal epithelial cells." American Journal of Physiology-Cell Physiology 301, no. 5 (2011): C1140—C1149. http://dx.doi.org/10.1152/ajpcell.00036.2011.

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Gastrointestinal infection with Shiga toxins producing enterohemorrhagic Escherichia coli causes the spectrum of gastrointestinal and systemic complications, including hemorrhagic colitis and hemolytic uremic syndrome, which is fatal in ∼10% of patients. However, the molecular mechanisms of Stx endocytosis by enterocytes and the toxins cross the intestinal epithelium are largely uncharacterized. We have studied Shiga toxin 1 entry into enterohemorrhagic E. coli-infected intestinal epithelial cells and found that bacteria stimulate Shiga toxin 1 macropinocytosis through actin remodeling. This e
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36

Stanford, Kim. "Introduction to the Special Issue “Molecular Basis and the Pathogenesis of Enterohemorrhagic Escherichia coli Infections”." Toxins 12, no. 12 (2020): 763. http://dx.doi.org/10.3390/toxins12120763.

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37

STOPFORTH, J. D., M. LOPES, J. E. SHULTZ, R. R. MIKSCH, and M. SAMADPOUR. "Location of Bung Bagging during Beef Slaughter Influences the Potential for Spreading Pathogen Contamination on Beef Carcasses." Journal of Food Protection 69, no. 6 (2006): 1452–55. http://dx.doi.org/10.4315/0362-028x-69.6.1452.

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Preevisceration carcass washing prior to bung bagging during beef slaughter may allow pooling of wash water in the rectal area and consequent spread of potential pathogens. The objective of this study was to compare protocols for bung bagging after preevisceration washing with an alternative method for bung bagging before preevisceration washing for the potential to spread enterohemorrhagic Escherichia coli, E. coli O157:H7, and Salmonella on carcass surfaces. The study evaluated incidence rates of pathogens in preevisceration wash water (10 ml) samples (n = 120) and on surface (100 cm2) spong
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38

Allerberger, F., L. Beutin, T. Cheasty, G. Gonedera, and H. Karch. "Sporadic infection caused by enterohemorrhagic d’Escherichia coli O157." Eurosurveillance 4, no. 10 (1999): 107–8. http://dx.doi.org/10.2807/esm.04.10.00053-en.

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In 1997 the Austrian Federal Ministry of Health established a reference laboratory for enterohaemorrhagic Escherichia coli at the federal public health laboratory in Innsbruck. The reference laboratory investigates sources of infection causing food poison
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39

GRANT, MICHAEL A., CHERYL A. EKLUND, and SUE C. SHIELDS. "Monitoring Dairy Silage for Five Bacterial Groups with Potential for Human Pathogenesis." Journal of Food Protection 58, no. 8 (1995): 879–83. http://dx.doi.org/10.4315/0362-028x-58.8.879.

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Two types of silage routinely used at a dairy research facility were monitored over a 20 month period for five bacterial groups which have been associated with human pathogenesis. Those monitored were Listeria monocytogenes, Yersinia enterocolitica (motile), motile aeromonads, Campylobacter sp., and enterohemorrhagic Escherichia coli. Of 46 total silage samples, 8.7% were positive for L monocytogenes, 6.5% for Y. enterocolitica, and 10.9% for motile aeromonads. Campylobacter spp. and enterohemorrhagic E. coli were not isolated. The presence of pathogens was associated with elevated pH in silag
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40

TKALCIC, SUZANA, TONG ZHAO, BARRY G. HARMON, MICHAEL P. DOYLE, CATHY A. BROWN, and PING ZHAO. "Fecal Shedding of Enterohemorrhagic Escherichia coli in Weaned Calves following Treatment with Probiotic Escherichia coli." Journal of Food Protection 66, no. 7 (2003): 1184–89. http://dx.doi.org/10.4315/0362-028x-66.7.1184.

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The fecal shedding and pathogenicity of enterohemorrhagic E. coli (EHEC) O26:H11, EHEC O111:NM, and EHEC O157:H7 in weaned calves (8 to 10 weeks of age) were compared with and without treatment with a three-strain mixture of probiotic bacteria (competitive-exclusion E. coli). Three groups of 12 calves were each perorally given a five-strain mixture of one of the EHEC serotypes (1010 CFU of total bacteria per calf). Seventy-two hours later, six calves from each group were each administered 1010 CFU of probiotic bacteria. None of the EHEC serotypes caused significant clinical disease, although a
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41

ZHAO, TONG, and MICHAEL P. DOYLE. "Fate of Enterohemorrhagic Escherichia coli O157: H7 in Commercial Mayonnaise." Journal of Food Protection 57, no. 9 (1994): 780–83. http://dx.doi.org/10.4315/0362-028x-57.9.780.

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The fate of enterohemorrhagic Escherichia coli O157:H7 was determined in three different lots of commercial mayonnaise, including four different samples from a lot implicated in an outbreak of E. coli O157:H7 infection. The initial pH of the products ranged from 3.6 to 3.9. Products were inoculated with 6.5 × 103 E. coli O157:H7/g and incubated at 5 or 20°C. Escherichia coli O157:H7 did not grow at either temperature but survived for 34 to 55 days at 5°C and for 8 to 21 days at 20°C, depending on the lot. Survival was greatest in real mayonnaise purchased at retail among six mayonnaise samples
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42

Low, J. Christopher, Iain J. McKendrick, Caroline McKechnie, et al. "Rectal Carriage of Enterohemorrhagic Escherichia coli O157 in Slaughtered Cattle." Applied and Environmental Microbiology 71, no. 1 (2005): 93–97. http://dx.doi.org/10.1128/aem.71.1.93-97.2005.

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ABSTRACT Escherichia coli O157:H7 is an important cause of diarrhea, hemorrhagic colitis, and potentially fatal human illness. Cattle are considered a primary reservoir of infection, and recent experimental evidence has indicated that the terminal rectum is the principal site of bacterial carriage. To test this finding in naturally colonized animals, intact rectum samples from 267 cattle in 24 separate lots were obtained immediately after slaughter, and fecal material and mucosal surfaces were cultured for E. coli O157 by direct and enrichment methods. Two locations, 1 and 15 cm proximal to th
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43

Itoh, Yoshinori, Yoshiko Sugita-Konishi, Fumiko Kasuga, et al. "Enterohemorrhagic Escherichia coli O157:H7 Present in Radish Sprouts." Applied and Environmental Microbiology 64, no. 4 (1998): 1532–35. http://dx.doi.org/10.1128/aem.64.4.1532-1535.1998.

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ABSTRACT Using cultivation, immunofluorescence microscopy, and scanning electron microscopy, we demonstrated the presence of viable enterohemorrhagic Escherichia coli O157:H7 not only on the outer surfaces but also in the inner tissues and stomata of cotyledons of radish sprouts grown from seeds experimentally contaminated with the bacterium. HgCl2 treatment of the outer surface of the hypocotyl did not kill the contaminating bacteria, which emphasized the importance of either using seeds free from E. coli O157:H7 in the production of radish sprouts or heating the sprouts before they are eaten
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44

Schwach, T. S., and E. A. Zottola. "Adherence of enterohemorrhagic Escherichia coli in a beef system." Proceedings, annual meeting, Electron Microscopy Society of America 53 (August 13, 1995): 1022–23. http://dx.doi.org/10.1017/s0424820100141482.

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Microbiologists have traditionally studied pure cultures growing in laboratory media. Food microbiologists, interested in interactions between the food system, pathogen, and host have tried to extrapolate information in the same manner. However, clinical isolates readily lose virulence characteristics when repeatedly subcultured into laboratory media. Expression of many virulence factors, such as fimbriae, extracellular polymers and outer membrane adhesins are affected by growth environment; an environment which encompasses not only nutrient availability and growth temperature, but also physic
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45

MAYERHAUSER, CAROLYN M. "Survival of Enterohemorrhagic Escherichia coli O157:H7 in Retail Mustard." Journal of Food Protection 64, no. 6 (2001): 783–87. http://dx.doi.org/10.4315/0362-028x-64.6.783.

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Escherichia coli O157:H7 survival in acid foods such as unpasteurized apple cider and fermented sausage is well documented. Researchers have determined that E. coli O157:H7 can survive in refrigerated acid foods for weeks. The potential of acid foods to serve as a vector of E. coli O157:H7 foodborne illness prompted this study to determine the fate of this organism in retail mustard containing acetic acid when stored at room and refrigerated temperatures. Various retail brands of dijon, yellow, and deli style mustard, pH ranging from 3.17 to 3.63, were inoculated individually with three test s
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46

Mokhtari, Forough, Azam Mokhtari, Mohammadreza Mahzounieh, Somayeh Shahrokh Shahraki, and Somayeh Gheisarbegi. "Isolation of bacteriophages affecting Enterohemorrhagic Escherichia coli O157:H7." Iranian Journal of Medical Microbiology 15, no. 1 (2021): 130–39. http://dx.doi.org/10.30699/ijmm.15.1.130.

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47

Ceponis, Peter JM, Jason D. Riff, and Philip M. Sherman. "Epithelial cell signaling responses to enterohemorrhagic Escherichia coli infection." Memórias do Instituto Oswaldo Cruz 100, suppl 1 (2005): 199–203. http://dx.doi.org/10.1590/s0074-02762005000900034.

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48

Horne, Cathy, Bruce A. Vallance, Wanyin Deng, and B. Brett Finlay. "Current progress in enteropathogenic and enterohemorrhagic Escherichia coli vaccines." Expert Review of Vaccines 1, no. 4 (2002): 483–93. http://dx.doi.org/10.1586/14760584.1.4.483.

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49

BAKHSHI, Bita, Sakine NAJIBI, and Saeed SEPEHRI-SERESHT. "Molecular Characterization of Enterohemorrhagic Escherichia coli Isolates from Cattle." Journal of Veterinary Medical Science 76, no. 9 (2014): 1195–99. http://dx.doi.org/10.1292/jvms.13-0237.

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50

Izumiya, Hidemasa. "Molecular Epidemiological Analysis of Enterohemorrhagic Escherichia coli." Japanese Journal of Food Microbiology 36, no. 1 (2019): 10–12. http://dx.doi.org/10.5803/jsfm.36.10.

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