Relevant bibliographies by topics / Eozinofilai

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Academic literature on the topic 'Eozinofilai'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Eozinofilai"

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Constantin, Marilena, Gabriel Zamfirescu, Cristina Voinea, and Sorin Constantinescu. "EOZINOFILIA – FACTOR TRIGGER PENTRU TROMBOZA VENOASĂ PROFUNDĂ ŞI EMBOLIA PULMONARĂ LA UN PACIENT CU TROMBOFILIE." Romanian Journal of Infectious Diseases 20, no. 4 (December 31, 2017): 212–15. http://dx.doi.org/10.37897/rjid.2017.4.8.

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Introducere. Infiltratele pulmonare şi eozinofilia reprezintă un grup heterogen de afecţiuni determinate de factori intrinseci si extrinseci. Factorii extrinseci reprezentaţi de medicaţie sau agenţi infecţioşi declanşează un răspuns eozinofilic imun. Raportăm cazul unui pacient de 53 ani, bărbat, cu infiltrat pulmonar si eozinofilie, secundare infecţiei cu Toxocara canis, care a fost diagnosticat cu tromboză venoasă profundă şi embolie pulmonară la o lună de la diagnosticul infecţiei cu Toxocara canis. Investigaţiile ulterioare au arătat prezenţa statusului procoagulant. Prezentarea cazului. În martie 2015, pacientul, în vârstă de 53 ani, s-a prezentat la consultaţie pentru durere intensă la nivelul toracelui posterior accentuată de inspir profund. Examenul clinic a fost în limite normale, dar tomografia computerizată fără substanţă de contrast efectuată în urgenţă a arătat infiltrat pulmonar la baza plămânului stâng cu reacţie pleurală. Analizele de sânge au indicat leucocite la limita superioară, eozinofilie (21,5%, 2.050/mm3) şi sindrom inflamator. Investigaţiile pentru eozinofilie au arătat reacţie pozitivăpentru Toxocara canis. Se începe tratamentul cu Albendazol 800 mg/zi cu răspuns favorabil. La interval de o lună, pacientul se prezintă pentru durere la nivelul gambei drepte. Testul pentru D-Dimer a fost intens pozitiv, ecografia Doppler venos confirmă diagnosticul de tromboză venoasă profundă, iar tomografia computerizată cu substanţă de contrast a descris embolie pulmonară. Pacientul a început tratamentul anticoagulant. A efectuat analizele de trombofilie care au fost pozitive pentru MTHFR A 1298C homozigot şi gena PAI1 675. Discuţii. Infecţiile cu helminţi se asociază cu eozinofilie. Helminţii care migrează spre viscere, cum este şi cazul Toxocarei canis, produc răspuns eozinofilic înalt. Întrebarea la care am încercat să răspundem a fost dacă eozinofilia a fost responsabilă pentru tromboze sau a reprezentat doar un factor de risc. Întrucât douăteste genetice pentru trombofilie au fost pozitive (MTHFR A 1298C homozigot şi gena PAI1 675), s-a considerat apariţia trombozei venoase şi a emboliei pulmonare în contextul statusului procoagulant al pacientului. Concluzii. Acest caz clinic subliniază implicarea eozinofiliei ca factor trigger pentru embolia pulmonară şi tromboza venoasă profundă.
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Stănciulescu, Cristiana, Mirela Chiru, Alina Oprea, Daniela Păcurar, Carmen Zăpucioiu, Elena Petrişor, and Dumitru Orăşeanu. "FENO ŞI ATOPIA ÎN ASTMUL BRONŞIC LA COPII." Romanian Journal of Pediatrics 64, no. 2 (June 30, 2015): 199–202. http://dx.doi.org/10.37897/rjp.2015.2.20.

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Obiective. Stabilirea relaţiei între valoarea concentraţiei oxidului nitric expirat (FENO) şi atopia în astmul bronşic la copii. Metode. Au fost examinaţi 92 de pacienţi cu diagnosticul de astm bronşic stabilit, cu vârste cuprinse între 5 şi 18 ani, care s-au prezentat în Clinica de Pediatrie a Spitalului Clinic de Urgenţe pentru Copii „Grigore Alexandrescu“. Terenul atopic a fost evaluat prin date anamnestice, clinice şi investigaţii de laborator (eozinofile serice şi IgE totale). Tuturor pacienţilor li s-a determinat concentraţia oxidului nitric expirat. Rezultate. S-au obţinut valori crescute ale FENO la copiii cu astm bronşic alergic faţă de cei cu astm bronşic nonalergic, valori care se corelează cu eozinofilia şi creşterea imunoglobulinelor E totale. Concluzii. Măsurarea FENO este utilă pentru evaluarea inflamaţiei mucoasei bronşice la copiii cu astm bronşic atopic.
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Ünler, Gülhan Kanat, Gülsüm Teke Özgür, Özgür Hilal Erinanç, and Hüseyin Savaş Göktürk. "Eozinofilik Gastroenteritin Nadir Bir Formu: Eozinofilik Asit." Cukurova Medical Journal 40, no. 3 (November 3, 2015): 597. http://dx.doi.org/10.17826/cutf.27001.

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Gülcan, E. Mahir, and Ayça Vitrinel. "Eozinofilik özofajit." Türk Pediatri Arşivi 45, no. 3 (September 15, 2010): 232–37. http://dx.doi.org/10.4274/tpa.45.232.

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Cvenkel, Klara, and Mateja Starbek Zorko. "EOZINOFILNI PUSTULOZNI FOLIKULITIS OTROŠKE DOBE." Slovenska pediatrija, revija pediatrov Slovenije in specialistov šolske ter visokošolske medicine Slovenije 28, no. 2 (2021): 94–97. http://dx.doi.org/10.38031/slovpediatr-2021-2-04.

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Çengel, Gülden, Esra Gürlek Olgun, Semih Alper Kureş, Cafer Uysal, Mehmet Ali Çakmak, and İsmail Özgür Can. "Eozinofilik Arterite Bağlı Spontan Koroner Arter Diseksiyonu Bir Olgu Sunumu." Bulletin of Legal Medicine 20, no. 1 (May 19, 2015): 53–55. http://dx.doi.org/10.17986/blm.2015110924.

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Koroner arterin spontan diseksiyonu ani ölüm ve myokard enfarktüsünün nadir nedenlerinden biri olarak kabul edilmektedir. Spontan travmatik olmayan koroner arter diseksiyonu seyrek olarak meydana gelir. Koroner arterin eozinofilden zengin periarteriti (eozinofilik periarterit) koroner arter diseksiyonuna yol açabilmektedir. Koroner eozinofilik periarteritin intimai bozulmada ve diseksiyonda predispozan bir faktör olduğu ileri sürülmektedir. Literatürdeki eozinofilik periarteritli kadın olguların 1/3’ü gebe ya da péripartum dönemdedir. Olgumuzda, 35 yaşında, evinde fenalaşan, postpartum dönemdeki kadın olgu hastaneye ölü olarak getirilmiştir. Adli Tıp Kurumu’nda yapılan otopsisinde alman kalbe ait doku örneklerinin histopatolojik incelenmesinde koroner arterin sol ön inen dalında eozinofilik periarterite bağlı diseksiyon; tiroide ait doku örneklerinde ise tiroidit saptanmıştır. Literatürde otoimmun tiroidit ile birlikte eozinofilik periarterite bağlı koroner arter diseksiyonu sonucu ölüm olgusu sayısı oldukça azdır. Genç postpartum dönemdeki kadınların ani ölümlerinde akla getirilmesi gereken bir patoloji olması nedeniyle önemli bulunmuştur.Anahtar kelimeler: Postpartum, Arterit, Eozinofilik, Koroner, Diseksiyon.
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YILDIZ, Levent, Mehmet KEFELİ, and Seda GÜN. "Behçet Hastalığında Angiolenfoid Hiperplazi ve Eozinofili." Journal of Experimental and Clinical Medicine 26, no. 4 (December 30, 2009): 186–89. http://dx.doi.org/10.5835/jecm.omu.26.04.008.

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Uyar, Belkız, and Serap Karaarslan. "Yaygın Eozinofilik Anjiolenfoid Hiperplazi." Turkish Journal of Dermatology / Türk Dermatoloji Dergisi 7, no. 3 (September 5, 2013): 161–63. http://dx.doi.org/10.4274/tdd.1362.

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Knežević, Snežana, and Branimir Dugalić. "Eosinophilic esophagitis: A potential complication of sublingual immunotherapy." Opsta medicina 27, no. 1-2 (2021): 32–39. http://dx.doi.org/10.5937/opmed2102032k.

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Introduction. Eosinophilic esophagitis (EoE) represents chronic, a local immune-mediated disease with symptoms of esophageal dysfunction and histologically eosinophil-predominant inflammation and requires immediate endoscopy. Male gender is a strong risk factor. Case report. We presented a case of a 25-year-old young man with a history of allergic rhino-conjunctivitis, asthma, and intermittent severe feeding disturbance. The patient had begun sublingual immunotherapy therapy, containing specific soluble allergens for ambrosia. Six weeks after starting the ambrosia sublingual immunotherapy he developed burning epigastric pain, dysphagia, and odynophagia. Six days later, he was admitted to an emergency department due to choking on a solid of food. Esophageal histopathological findings were in favor of EoE. Sublingual immunotherapy was discontinued. He feels well now. Conclusion. The majority of cases of Eosinophilic esophagitis are diagnosed in spring or fall, 1-2 months following the peak of pollen season. Dysphagia, chest pain, food sticking, and bolus obstruction are the most common symptoms. Endoscopic findings are Schatzki ring, edema, exudates, furrows, and strictures. Six biopsies should be taken from areas with endoscopic mucosal abnormalities, and infiltration of eosinophils (more than 15 eosinophils/HRI) (HRI - high resolution imaging) is necessary for the diagnosis confirmation. Treatment options are proton pump inhibitors - oral dispersible tablets of budesonide or fluticasone propionate, an elimination diet. Sublingual immunotherapy should be discontinued. Family physicians should be aware of this complication in evaluating patients with dysphagia.
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ENSARİ(), Arzu, and Yasemin YUYUCU KARABULUT(). "Çocukluk çağı eozinofilik özofajitlerinde histopatolojik bulgular." Endoskopi Gastrointestinal 20, no. 2 (April 3, 2015): 40. http://dx.doi.org/10.17940/endoskopi.74829.

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Dissertations / Theses on the topic "Eozinofilai"

1

Lavinskienė, Simona. "Peripheral blood neutrophil and eosinophil activity during allergen-induced late-phase airway inflammation in asthma." Doctoral thesis, Lithuanian Academic Libraries Network (LABT), 2015. http://vddb.library.lt/obj/LT-eLABa-0001:E.02~2014~D_20150106_083713-90371.

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There is no doubt that eosinophils and neutrophils are important cells participating in asthma pathogenesis. The most prominent feature reflecting asthma pathogenesis is late-phase airway inflammation, which occurs a few hours after allergen inhalation. The worldwide published studies on asthma show that most attention is paid to individual, not complex, functions of neutrophils and eosinophils in the airways. Moreover, associations between peripheral blood neutrophil and eosinophil activity and infiltration of these cells in the airways during asthma have not been com¬pletely elucidated yet. There are no data about peripheral blood neutrophil and eosinophil activity during allergen-induced late-phase airway inflam¬mation in asthma patients. Therefore, the aim of this study was to evaluate peripheral blood neutrophil and eosinophil functional activity during allergen-induced late-phase airway inflammation in asthma. We found that an inhaled allergen activates peripheral blood neutrophil and eosinophil chemotaxis, phagocytosis, generation of reactive oxygen species and also reduces apoptosis during late-phase airway inflammation in asthma. Furthermore, altered peripheral blood neutrophil and eosinophil functional activity is related with airway neutrophilia and eosinophilia. Our findings provide new evidence about neutrophil and eosinophil functional activity during allergen-induced late-phase airway inflammation in asthma patients.
Mokslininkai neabejoja, jog eozinofilai ir neutrofilai yra vienos svarbiausių ląstelių, dalyvaujančių astmos patogenezėje, kurią labiausiai atspindi vėlyva kvėpavimo takų uždegimo fazė, išsivystanti praėjus kelioms valandoms po alergeno įkvėpimo. Pasaulinėje literatūroje publikuojami darbai, nagrinėja atskirus kvė¬pavimo takų neutrofilų ir eozinofilų aktyvumo pokyčius. Ypač mažai darbų apie periferinio kraujo neutrofilų ir eozinofilų funkcijas bei jų ryšį su šių ląstelių pagausėjimu kvėpavimo takuose, sergant astma. Taip pat nėra tyrimų, vertinančių periferinio kraujo uždegimo ląstelių (neutrofilų ir eozi¬nofilų) funkcijų alergeno sukeltos vėlyvos fazės kvėpavimo takų uždegimo metu. Todėl šio tyrimo tikslas buvo įvertinti periferinio kraujo neutrofilų ir eozinofilų funkcinį aktyvumą alergeno sukeltos vėlyvos fazės kvėpavimo takų uždegimo metu sergant astma. Tyrimo metu nustatėme, kad įkvėptas alergenas aktyvina periferinio kraujo neutrofilų ir eozinofilų funkcijas - chemotaksį, fagocitozę, reaktyvių deguonies formų susidarymą, degranuliaciją bei silpnina apoptozę vėlyvos fazės kvėpavimo takų uždegimo metu. O šių ląstelių aktyvumo pokyčiai yra susiję su kvėpavimo takų neutrofilija ir eozinofilija. Moksliniame darbe pateikiami rezultatai suteikia naujų duomenų apie sergančiųjų alergine astma periferinio kraujo neutrofilų ir eozinofilų funkcinių savybių ypatumus ir parodo jų pokyčius alergeno sukeltos vėlyvos fazes kvėpavimo takų uždegimo metu.
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Arıkan, Mehmet Salih Demirci Mustafa. "Toxocariasis hastalarında eozinofilik katyonik protein düzeylerinin araştırılması /." Isparta : SDÜ Tıp Fakültesi, 2007. http://tez.sdu.edu.tr/Tezler/TT00329.pdf.

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Ivana, Koledin. "Strukturne promene sluzokože debelog creva pacova pod uticajem akrilamida." Phd thesis, Univerzitet u Novom Sadu, Prirodno-matematički fakultet u Novom Sadu, 2016. http://www.cris.uns.ac.rs/record.jsf?recordId=100831&source=NDLTD&language=en.

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Akrilamid je supstanca koja se prirodno stvara pečenjem i prženjem hrane bogate skrobom.  Cilj rada je bio da se ispita subhronični i akutni uticaj akrilamida na debelo crevo prepubertalnih pacova. Dobijeni rezultati su pokazali da akrilamid ne narušava morfologiju zida creva, ali dovodi do promena u volumenskoj gustini njegovih tunika i lamina. Najizraženije promene  su bile na  vezivnom tkivu  debelog creva.  Analizom peharastih ćelija  i sadržaja mucina pokazano je da akrilamid utiče i na ugljenohidratnu i na proteinsku komponentu mucina. Subhronični tretman je  doveo do smanjenja broja limfocita i  eozinofila, a  kod akutnog tretmana  je primećeno nakupljanje limfocita i eozinofila u kolonu akrilamidom tretiranih jedinki. Broj mastocita je u sva tri eksperimenta bio smanjen kod tretiranih životinja. Duže izlaganje akrilamidu ima imunosupresivno dejstvo kod pacova.
Acrylamide is natural product of cooking (baking, roasting) starchy food.  Aim of the study was to evaluate the risk of subchronic and acute acrylamide treatment on juvenile  rat colon.  Changes  in colon wall morphology was detected by stereological methods since histological evaluation reveal normal colon architecture  after acrylamide intoxication.  The changes was  most prominent on  connective tissue of rat colon.  Acrylamide affected both protein  component of mucins and glycans linked to peptide backbone.  In subchronic  treatment acrylamide caused reduction of lymphocytes  and eosinophils  number, while acute experiment lead to lymphocytes  and  eosinophils  accumulation in colon tissue. Acrylamide intoxication decreased mast cell number in all experiments. Longer acrylamide exposure had immunosuppressive effect in rats.
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