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Journal articles on the topic 'Eozinofilai'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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1

Constantin, Marilena, Gabriel Zamfirescu, Cristina Voinea, and Sorin Constantinescu. "EOZINOFILIA – FACTOR TRIGGER PENTRU TROMBOZA VENOASĂ PROFUNDĂ ŞI EMBOLIA PULMONARĂ LA UN PACIENT CU TROMBOFILIE." Romanian Journal of Infectious Diseases 20, no. 4 (2017): 212–15. http://dx.doi.org/10.37897/rjid.2017.4.8.

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Introducere. Infiltratele pulmonare şi eozinofilia reprezintă un grup heterogen de afecţiuni determinate de factori intrinseci si extrinseci. Factorii extrinseci reprezentaţi de medicaţie sau agenţi infecţioşi declanşează un răspuns eozinofilic imun. Raportăm cazul unui pacient de 53 ani, bărbat, cu infiltrat pulmonar si eozinofilie, secundare infecţiei cu Toxocara canis, care a fost diagnosticat cu tromboză venoasă profundă şi embolie pulmonară la o lună de la diagnosticul infecţiei cu Toxocara canis. Investigaţiile ulterioare au arătat prezenţa statusului procoagulant. Prezentarea cazului. Î
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2

Stănciulescu, Cristiana, Mirela Chiru, Alina Oprea, et al. "FENO ŞI ATOPIA ÎN ASTMUL BRONŞIC LA COPII." Romanian Journal of Pediatrics 64, no. 2 (2015): 199–202. http://dx.doi.org/10.37897/rjp.2015.2.20.

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Obiective. Stabilirea relaţiei între valoarea concentraţiei oxidului nitric expirat (FENO) şi atopia în astmul bronşic la copii. Metode. Au fost examinaţi 92 de pacienţi cu diagnosticul de astm bronşic stabilit, cu vârste cuprinse între 5 şi 18 ani, care s-au prezentat în Clinica de Pediatrie a Spitalului Clinic de Urgenţe pentru Copii „Grigore Alexandrescu“. Terenul atopic a fost evaluat prin date anamnestice, clinice şi investigaţii de laborator (eozinofile serice şi IgE totale). Tuturor pacienţilor li s-a determinat concentraţia oxidului nitric expirat. Rezultate. S-au obţinut valori crescu
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3

Ünler, Gülhan Kanat, Gülsüm Teke Özgür, Özgür Hilal Erinanç, and Hüseyin Savaş Göktürk. "Eozinofilik Gastroenteritin Nadir Bir Formu: Eozinofilik Asit." Cukurova Medical Journal 40, no. 3 (2015): 597. http://dx.doi.org/10.17826/cutf.27001.

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4

Gülcan, E. Mahir, and Ayça Vitrinel. "Eozinofilik özofajit." Türk Pediatri Arşivi 45, no. 3 (2010): 232–37. http://dx.doi.org/10.4274/tpa.45.232.

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5

Cvenkel, Klara, and Mateja Starbek Zorko. "EOZINOFILNI PUSTULOZNI FOLIKULITIS OTROŠKE DOBE." Slovenska pediatrija, revija pediatrov Slovenije in specialistov šolske ter visokošolske medicine Slovenije 28, no. 2 (2021): 94–97. http://dx.doi.org/10.38031/slovpediatr-2021-2-04.

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6

Çengel, Gülden, Esra Gürlek Olgun, Semih Alper Kureş, Cafer Uysal, Mehmet Ali Çakmak, and İsmail Özgür Can. "Eozinofilik Arterite Bağlı Spontan Koroner Arter Diseksiyonu Bir Olgu Sunumu." Bulletin of Legal Medicine 20, no. 1 (2015): 53–55. http://dx.doi.org/10.17986/blm.2015110924.

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Koroner arterin spontan diseksiyonu ani ölüm ve myokard enfarktüsünün nadir nedenlerinden biri olarak kabul edilmektedir. Spontan travmatik olmayan koroner arter diseksiyonu seyrek olarak meydana gelir. Koroner arterin eozinofilden zengin periarteriti (eozinofilik periarterit) koroner arter diseksiyonuna yol açabilmektedir. Koroner eozinofilik periarteritin intimai bozulmada ve diseksiyonda predispozan bir faktör olduğu ileri sürülmektedir. Literatürdeki eozinofilik periarteritli kadın olguların 1/3’ü gebe ya da péripartum dönemdedir. Olgumuzda, 35 yaşında, evinde fenalaşan, postpartum dönemde
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7

YILDIZ, Levent, Mehmet KEFELİ, and Seda GÜN. "Behçet Hastalığında Angiolenfoid Hiperplazi ve Eozinofili." Journal of Experimental and Clinical Medicine 26, no. 4 (2009): 186–89. http://dx.doi.org/10.5835/jecm.omu.26.04.008.

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8

Uyar, Belkız, and Serap Karaarslan. "Yaygın Eozinofilik Anjiolenfoid Hiperplazi." Turkish Journal of Dermatology / Türk Dermatoloji Dergisi 7, no. 3 (2013): 161–63. http://dx.doi.org/10.4274/tdd.1362.

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9

Knežević, Snežana, and Branimir Dugalić. "Eosinophilic esophagitis: A potential complication of sublingual immunotherapy." Opsta medicina 27, no. 1-2 (2021): 32–39. http://dx.doi.org/10.5937/opmed2102032k.

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Introduction. Eosinophilic esophagitis (EoE) represents chronic, a local immune-mediated disease with symptoms of esophageal dysfunction and histologically eosinophil-predominant inflammation and requires immediate endoscopy. Male gender is a strong risk factor. Case report. We presented a case of a 25-year-old young man with a history of allergic rhino-conjunctivitis, asthma, and intermittent severe feeding disturbance. The patient had begun sublingual immunotherapy therapy, containing specific soluble allergens for ambrosia. Six weeks after starting the ambrosia sublingual immunotherapy he d
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10

ENSARİ(), Arzu, and Yasemin YUYUCU KARABULUT(). "Çocukluk çağı eozinofilik özofajitlerinde histopatolojik bulgular." Endoskopi Gastrointestinal 20, no. 2 (2015): 40. http://dx.doi.org/10.17940/endoskopi.74829.

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11

Biazar, Tahmineh, Parisa Sabbagh, Soheil Ebrahimpour, and Masomeh Bayani. "Eosinophilic gastroenteritis with acute abdomen: A case report." Acta Facultatis Medicae Naissensis 38, no. 1 (2021): 96–100. http://dx.doi.org/10.5937/afmnai2101096b.

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Eosinophilic gastroenteritis (EGE) as a rare inflammatory gastrointestinal (GI) disorder is primarily characterized by long-term or recurrent abdominal pain. Peripheral eosinophilia is usually caused by eosinophilic infiltrations into the multiple layers of the GI tract. In this report, a case of EGE with an acute abdomen is presented. A 14-year-old female experienced severe abdominal pain and hypereosinophilia. She underwent appendectomy due to severe abdominal pain approximately one week before readmission. Because of the persistence of pain, the upper GI endoscopy and biopsy were performed
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12

MERTOĞLU, Aydan, Yasemin YURT, Şevket DERELİ, Zühre TAYMAZ, and Günseli BALCI. "Eosinophilic Pneumonia Caused by Mesalazine: Case Report." Turkiye Klinikleri Journal of Case Reports 23, no. 4 (2015): 401–5. http://dx.doi.org/10.5336/caserep.2014-40142.

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13

CANPOLAT, Filiz, Hatice AKPINAR, Bengü ÇEVİRGEN CEMİL, Fatma ESKİOĞLU, and Demet YILMAZER. "Report of Two Cases with Eosinophilic Cellulitis." Turkiye Klinikleri Journal of Medical Sciences 31, no. 4 (2011): 1034–37. http://dx.doi.org/10.5336/medsci.2009-13460.

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14

DEMİR, Melike, Fatih METEROĞLU, Halide KAYA, et al. "PET/BT’de pozitif tutulum saptanan eozinofilik granüloma olgusu." Tuberkuloz ve Toraks 63, no. 3 (2015): 207–9. http://dx.doi.org/10.5578/tt.9794.

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15

Emine;YÜCESOY, ÖZTÜRK. "Memede Tanımlanan Eozinofilik Anjiolenfoid Hiperplazi Olgusunun Ultrasonografi Bulguları." Ankara Üniversitesi Tıp Fakültesi Mecmuası 63, no. 3 (2010): 89–91. http://dx.doi.org/10.1501/tipfak_0000000768.

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16

EYÜPOĞLU, Damla, Ebru ORTAÇ ERSOY, Kazım ROLLAS, and Arzu TOPELİ. "Acute eosinophilic pneumonia secondary to heroin inhalation." Tuberkuloz ve Toraks 65, no. 2 (2017): 154–56. http://dx.doi.org/10.5578/tt.10438.

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17

YALÇIN, Mihriban, Celali KURT, and Hilayda KARAKÖK GÜNGÖR. "Fatal DRESS Syndrome Without Eosinophilia After Coronary Artery Bypass Grafting." Turkiye Klinikleri Journal of Dermatology 30, no. 2 (2020): 66–68. http://dx.doi.org/10.5336/dermato.2019-65810.

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18

KANZA GÜL, Derya, and Ayça ŞOLT. "Kan eozinofil-bazofil oranı, eozinofil-lenfosit oranı ve bazofil-lenfosit oranı değerleri gebelikteki intrahepatik kolestazın şiddetini tahmin edebilir mi?" Cukurova Medical Journal 45, no. 4 (2020): 1742–49. http://dx.doi.org/10.17826/cumj.834063.

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19

KANZA GÜL, Derya, and Ayça ŞOLT. "Kan eozinofil-bazofil oranı, eozinofil-lenfosit oranı ve bazofil-lenfosit oranı değerleri gebelikteki intrahepatik kolestazın şiddetini tahmin edebilir mi?" Cukurova Medical Journal 45, no. 4 (2020): 1742–49. http://dx.doi.org/10.17826/cumj.834063.

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20

Erkan;OKUTAN, KAPTANOĞLU. "Lomber Disk Herniasyonunu Taklit Eden Eozinofilik Granulom: Olgusu Sunumu." Ankara Üniversitesi Tıp Fakültesi Mecmuası 55, no. 2 (2002): 1. http://dx.doi.org/10.1501/tipfak_0000000020.

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21

Özgen, Hasan, Fatma Merve Tepetam, İsmet Bulut, and Cihan Örçen. "The significance of eosinophil and eosinophil lymphocyte ratio (ELR) in predicting response to omalizumab treatment in patients with severe allergic asthma." Tuberk Toraks 69, no. 1 (2021): 39–48. http://dx.doi.org/10.5578/tt.20219905.

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22

ALTINTAŞ KAKŞİ, Sümeyye, Ali BALEVİ, Mavişe YÜKSEL, Mustafa ÖZDEMİR, and Aslı ÇAKIR. "Angiolenfoid Hyperplasia with Eosinophilia Treated with Cryotherapy: Case Report." Turkiye Klinikleri Journal of Dermatology 26, no. 1 (2016): 50–53. http://dx.doi.org/10.5336/dermato.2015-45868.

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23

YILMAZ, İnsu. "Eosinophilic asthma with chronic rhinosinusitis/nasal polyps and biologic agents." Tuberk Toraks 67, no. 4 (2019): 292–99. http://dx.doi.org/10.5578/tt.68953.

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24

Mikoviny Kajzrlíková, Ivana. "Jorveza® – the expected preparation for the treatment of eosinophilic esophagitis." Gastroenterologie a hepatologie 74, no. 4 (2020): 357–59. http://dx.doi.org/10.14735/amgh2020357.

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25

Menclová, Alena, Michal Hojný, Martina Dulavová, et al. "New options for treating eosinophilic esophagitis by using compounded medication." Praktické lékárenství 12, no. 1 (2016): 26–28. http://dx.doi.org/10.36290/lek.2016.006.

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26

UGIŞ, Ayfer, İbrahim DOĞAN, Özgür EKİNCİ, Mustafa CENGİZ, and Selahattin ÜNAL. "Prevalence of Eosinophilic Esophagitis in Patients with Dysphagia in the Turkish Population." Turkiye Klinikleri Journal of Medical Sciences 33, no. 3 (2013): 888–93. http://dx.doi.org/10.5336/medsci.2012-33171.

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27

Pecl, Jakub, Eva Karásková, Lumír Kunovský, et al. "Eosinophilic esophagitis – 10 years of experience in five Czech pediatric endoscopy centers." Gastroenterologie a hepatologie 74, no. 6 (2020): 469–80. http://dx.doi.org/10.48095/ccgh2020469.

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Background: Eosinophilic esophagitis (EoE) is a chronic, progressive inflammatory disease of the esophagus characterized by local eosinophilic infiltration accompanied by symptoms of esophageal dysfunction. The aim of this study was to characterize features of EoE dia gnosed regionally and to describe local strategies for its treatment. Methods: The observational survey retrospectively analyzed a data set of child patients with histologically proven EoE from five pediatric endoscopy centers. This analysis focused on describing their general situation (age, gender, symptoms) and also aimed to i
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28

Hloušková, Eliška, Kateřina Bajerová, Jakub Pecl, Petr Jabandžiev, Marta Ježová, and Jiří Tůma. "Eosinophilic enteritis – case report of a rare manifestation and review of updates." Gastroenterologie a hepatologie 74, no. 6 (2020): 492–96. http://dx.doi.org/10.48095/ccgh2020492.

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Eosinophilic enteritis is one of the eosinophilic gastrointestinal disorders characterised by various clinical symptoms. Histopathologic proof of dense eosinophilic infiltration is the cornerstone for the diagnosis. There is no consensus on how dense the infiltration should be, and cut-off values of the eosinophilic count in HPF have not been defined yet. Therapy for eosinophilic enteritis is based on an elimination diet and corticosteroid treatment. Herein the authors report a case of 14-year-old boy who suffered from acute abdominal pain. Computer tomography of the abdomen revealed a thicken
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29

ÖZDEL ÖZTÜRK, Betül, and Sevim BAVBEK. "Anti-IL-5 treatments in severe eosinophilic asthma: real life datas." Tuberk Toraks 68, no. 2 (2020): 148–59. http://dx.doi.org/10.5578/tt.69416.

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30

Appak, Yeliz Çağan, Semin Ayhan, and Erhun Kasırga. "Çölyak hastalığı ve eozinofilik özefajit birlikteliği: Bu iki hastalık için bir tesadüf mü?" Ege Tıp Dergisi 54, no. 4 (2015): 193–95. http://dx.doi.org/10.19161/etd.344161.

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31

Kömür, Süheyla, Aslıhan Ulu, Behice Kurtaran, et al. "Daptomisin alan hastalarda gelişen ani solunum yetmezliği ve eozinofilik pnömoni: beş olgunun sunumu." Cukurova Medical Journal 41, no. 2 (2016): 396. http://dx.doi.org/10.17826/cutf.208261.

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32

Aydoğdu, İmran. "Nazal polipozis hastalarında nötrofil/lenfosit oranı ve eozinofil/lenfosit oranının prognostik değeri." Praxis of Otorhinolaryngology 6, no. 3 (2018): 92–95. http://dx.doi.org/10.5606/kbbu.2018.83723.

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33

Benlidayı, İlke Coşkun, Neslihan Gökçen, Aylin Gökşen, and Tunay Sarpel. "Romatoid artritli hastalarda kan eozinofil sayısının hastalık aktivitesi ve inflamatuvar belirteçlerle ilişkisi." Cukurova Medical Journal (Çukurova Üniversitesi Tıp Fakültesi Dergisi) 42, no. 1 (2017): 73. http://dx.doi.org/10.17826/cutf.280122.

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34

KORKMAZ, Deniz, and İsmail Şah HAREM. "Deve (Camelus dromedarius) İnce Barsak Mukozasında Plazma Hücresi ve Eozinofil Granulosit Dağılımının Belirlenmesi." Harran Üniversitesi Veteriner Fakültesi Dergisi 7, no. 1 (2018): 108–13. http://dx.doi.org/10.31196/huvfd.470980.

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35

Šimovičová, V., M. Danková, M. Luknár, et al. "Eozinofil – „dobrý sluha, zlý pán“ / Eosinophil – “a good servant but a bad master”." Cardiology letters 30, no. 01 (2021): 57–61. http://dx.doi.org/10.4149/cardiol_2021_1_10.

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36

ÇOBAN AĞCA, Meltem, Emine Aksoy, Dildar Duman, et al. "Does eosinophilia and neutrophil to lymphocyte ratio affect hospital re-admission in cases of copd exacerbation?" Tuberkuloz ve Toraks 65, no. 4 (2017): 282–90. http://dx.doi.org/10.5578/tt.57278.

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37

Kıykım, Ayça, Ezgi Gizem Yüce, Ezgi Barış, et al. "Serum IgE Düzeyi ve Eozinofil Sayısı ile Atopik Hastalıklar Hiper IgE Sendromu’ndan Ayırt Edilebilir mi?" Asthma Allergy Immunology 15, no. 2 (2017): 73–81. http://dx.doi.org/10.21911/aai.13.

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38

Yakar, Halil İbrahim, and Asiye Kanbay. "A new predictor to determine the exacerbation and treatment of chronic obstructive pulmonary disease: eosinophil/neutrophil ratio." Tuberk Toraks 68, no. 3 (2020): 260–67. http://dx.doi.org/10.5578/tt.69600.

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39

YILMAZ, Güldal, Berna SAVAŞ, Çetin ATASOY, Kaan KARAYALÇIN, and Esra ERDEN. "Autoimmune Pancreatitis Accompanied by Eosinophilic Cholecystitis Diagnosed After Surgery: Report of a Case with Histopathological, Radiological, and Clinical Approach." Turkiye Klinikleri Journal of Medical Sciences 32, no. 5 (2012): 1471–76. http://dx.doi.org/10.5336/medsci.2010-16865.

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40

Damian, Raluca, Cristina Oana Mărginean, Nicoleta Suciu, Maria Oana Mărginean, Simona Mocan, and Lorena Elena Meliţ. "METAPLAZIA PANCREATICĂ A MUCOASEI GASTRICE LA COPIL." Romanian Journal of Pediatrics 65, no. 3 (2016): 321–23. http://dx.doi.org/10.37897/rjp.2016.3.18.

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Metaplazia pancreatică este definită drept apariţia ţesutului pancreatic într-o locaţie anormală şi fară conectare directă la pancreas. Ţesutul pancreatic heterotopic este cel mai frecvent observat la nivelul mucoasei antrale gastrice, în duoden şi jejun. Prezentăm cazul unei paciente în vârstă de 13 ani care s-a internat în Clinica Pediatrie 1 Târgu-Mureş pentru dureri abdominale recurente şi pirozis. Investigaţiile de laborator efectuate au evidenţiat limfocitoză (41%) cu monocitoză (8,9%), eozinofilie (4,9%), un număr crescut de reticulocite (20%), un nivel seric scăzut al trigliceridelor ş
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41

Şovărel, Ana-Maria Moiceanu, Eugenia Buzoianu, Mariana Moiceanu, and Doina Anca Pleşca. "Nivelul seric al proteinei cationice eozinofilice în raport cu eozinofilia şi valoarea oxidul nitric exhalat la copiii cu astm bronşic atopic sensibilizaţi la aeroalergeni." Romanian Journal of Pediatrics 67, no. 1 (2018): 44–48. http://dx.doi.org/10.37897/rjp.2018.1.10.

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42

Meliţ, Lorena Elena, Cristina Oana Mărginean, Andreea Dincă, Raluca Damian, and Cristian Dan Mărginean. "ENTEROPATIA EXUDATIVĂ DUPĂ ENTEROCOLITĂ INFECŢIOASĂ LA COPIL – PREZENTARE DE CAZ ŞI DATE DIN LITERATURĂ." Romanian Journal of Infectious Diseases 20, no. 1 (2017): 53–56. http://dx.doi.org/10.37897/rjid.2017.1.9.

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Enteropatia exudativă (EE) reprezintă o pierdere proteică anormală la nivelul mucosei tractului digestiv, fiind în majoritatea cazurilor secundară altor patologii gastro-intestinale sau extraintestinale. Cu toate acestea, există, de asemenea, cauze primare, genetice asociate cu EE. Prezentăm cazul unui copil mic, de sex feminin, în vârstă de 1 an şi 1 lună, cu un episod de enterocolită infecţioasă cauzată de Escherichia coli enteropatogen în antecedente, care s-a internat în clinica noastră cu edeme palpebrale matinale de aproximativ 1 săptămână. Analizele de laborator au evidenţiat hipoalbumi
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43

Gerlinger, Imre, András Fittler, Anna Mayer, et al. "Postoperative application of amphotericin B nasal spray in chronic rhinosinusitis with nasal polyposis. Can recidive polyposis be prevented?" Orvosi Hetilap 149, no. 37 (2008): 1737–46. http://dx.doi.org/10.1556/oh.2008.28410.

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A krónikus rhinosinusitis (KRS) a felnőttpopuláció 1–4%-át érintő, jelentős életminőség-romlást okozó idült, multifaktoriális, gyakran orrpolipképződéssel járó, etiológiáját illetően azonban pontosan nem tisztázott megbetegedés. Az elmúlt évtizedben felmerült, hogy az orrüregi nyálkahártya jellegzetes eozinofil sejtes beszűrődését okozó megbetegedésnek az orrüregi nyákban található gombák elleni fokozott – nem IgE típusú klasszikus allergiás – immunválasz a kiváltó oka. Célkitűzés: Ha igaz ez a feltételezés, akkor kézenfekvőnek tűnik, hogy az endoszkópos orrpolypectomiákat követően megfelelő i
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44

Janulaitytė, Ieva, Andrius Januškevičius, Virginija Kalinauskaitė-Žukauskė, Rokas Stonkus, and Kęstutis Malakauskas. "Eozinofilai, aktyvindami WNT-5a signalinį kelią, skatina bronchų lygiųjų raumenų kontraktiliškumą sergant astma." Pulmonology and allergology 2, no. 1 (2018). http://dx.doi.org/10.37499/pia.194.

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Įvadas. Astma yra lėtinė uždegiminė plaučių liga, kuriai būdinga kvėpavimo takų eozinofilija. Uždegimas apima ir plaučių struktūrines ląsteles: bronchų lygiųjų raumenų ląsteles, fibroblastus, kurių aktyvumas valdomas per uždegiminių ląstelių išskiriamus mediatorius. Iki šiol neaišku, kaip eozinofilai veikia bronchų lygiųjų raumenų ląstelių kontraktiliškumą. Tyrimo tikslas. Įvertinti eozinofilų įtaką WNT-5a signalinio kelio aktyvumui ir bronchų lygiųjų raumenų ląstelių kontraktiliškumui sergant astma. Metodai. Į tyrimą įtraukti penki sergantieji alergine astma ir penki sveiki asmenys. Kraujo eo
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45

Kalinauskaitė, Virginija, and Kęstutis Malakauskas. "Nauji astmos patogenezės aspektai." Pulmonology and allergology 14, no. 1 (2014). http://dx.doi.org/10.37499/pia.344.

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Astma yra lėtinė apatinių kvėpavimo takų uždegimo liga, kuriai būdinga praeinanti bronchų obstrukcija, bronchų hiperreakty-vumas; vyrauja uždegimo ląstelės ir jų išskiriami mediatoriai, struktūriniai bronchų pokyčiai (remodeliavimasis). Bronchų lygiųjų raumenų ląstelės aktyviai dalyvauja bronchų remodeliavimosi procese išskirdamos prouždegimo citokinus, chemokinus ir tarpląstelinės medžiagos baltymus. Eozinofilai, vienos pagrindinių uždegimo ląstelių, yra svarbūs vykstant bronchų remodeliavimuisi. Eozinofilai, išskirdami cisteinil-leukotrienus, skatina bronchų lygiųjų raumenų proliferaciją. Ma
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46

Januškevičius, Andrius, Ieva Janulaitytė, and Kęstutis Malakauskas. "Sergant astma po sąveikos su bronchų lygiųjų raumenų ląstelėmis didėja eozinofilų gyvybingumas." Pulmonology and allergology 1, no. 2 (2017). http://dx.doi.org/10.37499/pia.215.

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Įvadas. Periferiniame kraujyje cirkuliuojančių eozinofilų gyvybingumas yra mažesnis lyginant su migravusių į kvėpavimo takus. Todėl galima daryti prielaidą, kad eozinofilų sąveika su plaučių struktūrinėmis ląstelėmis gali prailginti jų gyvybingumą ir aktyvumą, taip sąlygodama kvėpavimo takų remodeliacijos vystymąsi sergant astma. Tikslas. Įvertinti bronchų lygiųjų raumenų ląstelių poveikį eozinofilų gyvybingumui sergant astma. Metodai. Periferinio kraujo eozinofilai buvo izoliuoti iš penkių sergančiųjų astma ir keturių sveikų asmenų, pritaikant centrifugavimą aukšto tankio gradiente ir magneti
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47

Kalinauskaitė-Žukauskė, Virginija. "Biologinė terapija benralizumabu gydant sunkią eozinofilinę astmą. Privalumai ir kylantys klausimai." Pulmonology and allergology 4, no. 2 (2020). http://dx.doi.org/10.37499/pia.120.

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Biologinės terapijos sukūrimas yra proveržis eozinofilinės astmos gydyme. Slopinant interleukino-5 signalinį kelią, pvz., benralizumabu, veiksmingaimažinamas eozinofilinis uždegimas, retinami astmos paūmėjimai, pavyksta pasiekti geresnės astmos simptomų kontrolės, sumažinti geriamųjų gliukokortikoidų dozes arba juos visiškai nutraukti. Šis gydymas pasižymi ir geru saugumo profiliu. Tačiau kyla klausimų: ar sunaikinus eozinofilus ir pagerinus astmos kontrolę, nesumažinamas priešvirusinis eozinofilų poveikis? Ar neskatinama tumorogenezė? Atlikta nemažai tyrimų, kuriuose aktyviai diskutuojama šia
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48

Januškevičius, Andrius, Ieva Janulaitytė, Virginija Kalinauskaitė-Žukauskė, Aigirdas Rimkūnas, Beatričė Tamašauskaitė, and Kęstutis Malakauskas. "Eozinofilų adhezijos poveikis bronchų lygiųjų raumenų proliferacijai sergant astma." Pulmonology and allergology 3, no. 1 (2020). http://dx.doi.org/10.37499/pia.150.

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Įvadas. Eozinofilinis kvėpavimo takų uždegimas – svarbus astmos patogenezės komponentas. Eozinofilai yra gebančios adhezuoti ląstelės, nes turi pagrindinius adhezijos receptorius – integrinus, kurie ne tik atlieka prisitvirtinimo funkciją, bet gali elgtis kaip signalo perdavėjai abipus komunikuojančių ląstelių. Manoma, kad ne visi eozinofilai pasižymi vienodomis adhezinėmis savybėmis ir priklausomai nuo to gali skirtis savo atliekamomis funkcijomis. Taip pat dėl adhezijos per paviršiaus integrinus eozinofilai yra ne tik užlaikomi plaučių audiniuose greta struktūrinių ląstelių, bet gali pakisti
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49

Janulaitytė, Ieva, and Simona Lavinskienė. "Branduolio faktorius kappa B ir eozinofilai astmos patogenezėje." Pulmonology and allergology 16, no. 1 (2015). http://dx.doi.org/10.37499/pia.314.

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Astma yra kompleksinė uždegimo liga, kuriai būdingas kvėpavimo takų uždegimas bei remodeliavimasis ir bronchų reaktyvumas. Eozinofilai – tai kraujyje cirkuliuojantys granuliocitai, dalyvaujantys astmos patogenezėje. Eozinofilų gyvavimas sergant alergine astma užsitęsia, kas gali sąlygoti kvėpavimo takų gleivinės pažeidimą ir remodeliavimąsi. Branduolio faktoriaus kappa B kelias buvo laikomas prototipiniu uždegimo signaliniu keliu, kurio pagrindas – uždegimo citokinų, chemokinų ir adhezijos molekulių genų raiška, reguliuojama NF-κB transkripcijos faktoriaus. NF-κB ilgai buvo laikomas „šventuoju
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Jurkevičiūtė, Eglė, Andrius Januškevičius, and Kęstutis Malakauskas. "Nekoduojančiųjų RNR reikšmė astmos patogenezei." Pulmonology and allergology 4, no. 1 (2020). http://dx.doi.org/10.37499/pia.64.

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Astma serga daugiau nei 300 milijonų žmonių visame pasaulyje ir 250 000 kasmet jų miršta, o ligos patogenezė vis dar nėra pakankamai ištirta. Tai itin heterogeninė, lėtinė uždegiminė kvėpavimo takų liga, kuriai būdingi dusulio, švokštimo, kosulio epizodai, pasireiškiantys dėl kvėpavimo takų obstrukcijos ir hiperreaktyvumo. Astmos uždegiminis atsakas apima daugybę skirtingų specializuotų imuninių ląstelių, iš kurių svarbiausi yra eozinofilai. Šios ląstelės patenka į kvėpavimo takus per kraują bei išskiria katijoninius baltymus, citokinus, chemokinus ir lipidų mediatorius, esančius jų citoplazmi
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