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Journal articles on the topic 'Epidermidis'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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1

Coton, Emmanuel, Niels Mulder, Monika Coton, Sylvie Pochet, Hein Trip, and Juke S. Lolkema. "Origin of the Putrescine-Producing Ability of the Coagulase-Negative Bacterium Staphylococcus epidermidis 2015B." Applied and Environmental Microbiology 76, no. 16 (2010): 5570–76. http://dx.doi.org/10.1128/aem.00441-10.

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ABSTRACT A multiplex PCR method, aimed at the detection of genes associated with biogenic amine production, identified the odc gene encoding ornithine decarboxylase in 1 of 15 strains of Staphylococcus epidermidis. The ability of the positive strain, S. epidermidis 2015B, to produce putrescine in vitro was demonstrated by high-performance liquid chromatography (HPLC). In this strain, the odc gene was detected on plasmid DNA, suggesting that the ability to form putrescine is carried by a mobile element, which explains the fact that the trait is strain dependent within the S. epidermidis species
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2

Yu, Xianshui, Ting Chen, Ning Huang, Yanxia Jin, and Ling Yang. "Skin Commensal Bacteria Modulates the Immune Balance of Mice to Alleviate Atopic Dermatitis-Induced Damage." Evidence-Based Complementary and Alternative Medicine 2022 (September 17, 2022): 1–8. http://dx.doi.org/10.1155/2022/4731675.

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Objective. Although studies indicate that Staphylococcus epidermidis (S. epidermidis) can regulate inflammation and anti-inflammatory cytokines, there is limited evidence supporting their effects on atopic dermatitis (AD). Here, we aimed to investigate the effects and potential mechanism of skin commensal bacteria on the immunity of mice with AD. Methods. Twenty-four female BALB/C mice were selected and divided randomly into 4 groups: normal group, atopic dermatitis model group (AD), atopic dermatitis/substrate group (AD/substrates), and atopic dermatitis/substrates/epidermidis group (AD/S. ep
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3

Kiang, Michelle. "Staphylococcus epidermidis." Pediatrics in Review 24, no. 12 (2003): 430–31. http://dx.doi.org/10.1542/pir.24-12-430.

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4

Meskin, I. "Staphylococcus epidermidis." Pediatrics in Review 19, no. 3 (1998): 105–6. http://dx.doi.org/10.1542/pir.19-3-105.

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5

Meskin, I. "Staphylococcus epidermidis." Pediatrics In Review 19, no. 3 (1998): 105–6. http://dx.doi.org/10.1542/pir.19.3.105.

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6

Li, Xue, Cuiping Guan, Yulong He, Yujiong Wang, Xiaoming Liu, and Xuezhang Zhou. "Effects of Total Alkaloids ofSophora alopecuroideson Biofilm Formation inStaphylococcus epidermidis." BioMed Research International 2016 (2016): 1–10. http://dx.doi.org/10.1155/2016/4020715.

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Staphylococcus epidermidis(S. epidermidis) is an opportunistic pathogen with low pathogenicity and a cause of the repeated outbreak of bovine mastitis in veterinary clinical settings. In this report, a biofilm model ofS. epidermidiswas generated and the minimal inhibitory concentration (MIC) and sub-MIC (SMIC) on bacterial cultures were assessed for the following agents: total alkaloids ofSophora alopecuroides(TASA), ciprofloxacin (CIP), and erythromycin (ERY). The formation and characteristic parameters of biofilm were analyzed in terms of XTT assay, silver staining, and confocal laser scanni
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7

Kikuchi, Ken. "2. Staphylococcus Epidermidis." Nihon Naika Gakkai Zasshi 97, no. 11 (2008): 2673–77. http://dx.doi.org/10.2169/naika.97.2673.

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8

Fierer, Joshua. "Staphylococcus epidermidis Bacteremia." Annals of Internal Medicine 111, no. 1 (1989): 93. http://dx.doi.org/10.7326/0003-4819-111-1-93.

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9

Lampl, E., CH Richard, and PH Auzepy. "STAPHYLOCOCCUS EPIDERMIDIS SEPTICAEMIA." Lancet 331, no. 8585 (1988): 594–95. http://dx.doi.org/10.1016/s0140-6736(88)91394-3.

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10

Vuong, Cuong, and Michael Otto. "Staphylococcus epidermidis infections." Microbes and Infection 4, no. 4 (2002): 481–89. http://dx.doi.org/10.1016/s1286-4579(02)01563-0.

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11

Pugliese, Gina, and Martin S. Favero. "Staphylococcus epidermidis Biofilms." Infection Control & Hospital Epidemiology 22, no. 9 (2001): 598. http://dx.doi.org/10.1017/s0195941700066881.

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12

Dourado, Gabriela Sousa, Julio César Amaral Cardoso, Igor Feijão Cardoso, et al. "Avaliação da Capacidade Antimicrobiana de Nanopartículas de Prata Sintetizadas com Mel de Abelha." Ensaios e Ciência C Biológicas Agrárias e da Saúde 24, no. 3 (2020): 305–9. http://dx.doi.org/10.17921/1415-6938.2020v24n3p305-309.

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Nos últimos anos, as nanopartículas de prata (AgNPs) têm atraído muita atenção devido às suas largas aplicações em diferentes campos como biotecnologia, microeletrônica, armazenamento de informação óptica, medicina e conversão de energia. O presente trabalho teve como objetivo sintetizar nanopartículas de prata a partir de amostras de mel de abelhas Apis mellifera produzidos na cidade de Santarém-Pará, Brasil. Além disso, avaliar a atividade antimicrobiana dessas amostras, obtidas em dois períodos distintos (seco e chuvoso), frente a patógenos de interesse clínico: Staphylococcus aureus, Staph
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13

Yao, Yufeng, Daniel E. Sturdevant, Amer Villaruz, Lin Xu, Qian Gao, and Michael Otto. "Factors Characterizing Staphylococcus epidermidis Invasiveness Determined by Comparative Genomics." Infection and Immunity 73, no. 3 (2005): 1856–60. http://dx.doi.org/10.1128/iai.73.3.1856-1860.2005.

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ABSTRACT Virulence mechanisms of the leading nosocomial pathogen Staphylococcus epidermidis are poorly understood. We used microarray-based genome-wide comparison of clinical and commensal S. epidermidis strains to identify putative virulence determinants. Our study revealed high genetic variability of the S. epidermidis genome, new markers for invasiveness of S. epidermidis, and potential targets for drug development against S. epidermidis infections.
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14

Cabrera-Contreras, Roberto, Rosa I. Santamaría, Patricia Bustos, et al. "Genomic diversity of prevalent Staphylococcus epidermidis multidrug-resistant strains isolated from a Children’s Hospital in México City in an eight-years survey." PeerJ 7 (November 20, 2019): e8068. http://dx.doi.org/10.7717/peerj.8068.

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Staphylococcus epidermidis is a human commensal and pathogen worldwide distributed. In this work, we surveyed for multi-resistant S. epidermidis strains in eight years at a children’s health-care unit in México City. Multidrug-resistant S. epidermidis were present in all years of the study, including resistance to methicillin, beta-lactams, fluoroquinolones, and macrolides. To understand the genetic basis of antibiotic resistance and its association with virulence and gene exchange, we sequenced the genomes of 17 S. epidermidis isolates. Whole-genome nucleotide identities between all the pairs
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15

Qin, Zhiqiang, Liang Yang, Di Qu, Soeren Molin, and Tim Tolker-Nielsen. "Pseudomonas aeruginosa extracellular products inhibit staphylococcal growth, and disrupt established biofilms produced by Staphylococcus epidermidis." Microbiology 155, no. 7 (2009): 2148–56. http://dx.doi.org/10.1099/mic.0.028001-0.

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Multiple bacterial species often coexist as communities, and compete for environmental resources. Here, we describe how an opportunistic pathogen, Pseudomonas aeruginosa, uses extracellular products to interact with the nosocomial pathogen Staphylococcus epidermidis. S. epidermidis biofilms and planktonic cultures were challenged with P. aeruginosa supernatant cultures overnight. Results indicated that quorum-sensing-controlled factors from P. aeruginosa supernatant inhibited S. epidermidis growth in planktonic cultures. We also found that P. aeruginosa extracellular products, mainly polysacch
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16

Otto, Michael, Hartmut Echner, Wolfgang Voelter, and Friedrich Götz. "Pheromone Cross-Inhibition betweenStaphylococcus aureus and Staphylococcus epidermidis." Infection and Immunity 69, no. 3 (2001): 1957–60. http://dx.doi.org/10.1128/iai.69.3.1957-1960.2001.

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ABSTRACT Cross-inhibition by quorum-sensing pheromones betweenStaphylococcus aureus and Staphylococcus epidermidis was investigated using all known S. aureus agr pheromone subgroups. All S. aureus subgroups were sensitive towards the S. epidermidis pheromone, with the exception of the recently identified subgroup 4. The subgroup 4 pheromone was also the only S. aureus pheromone able to inhibit the S. epidermidis agr response. The close relation of subgroup 4 to subgroup 1 suggests that subgroup 4 might have evolved from subgroup 1 by mutation under the selective pressure of competition with S.
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17

Martani, Natalia Sri, Oktaviani Naulita Turnip, Hanasia Hanasia, and Nawan Nawan. "Studi In Vitro Aktivitas Antibakteri Ekstrak Etanol Penawar Sampai (Tinospora crispa L.) terhadap Staphylococcus epidermidis." Journal of Experimental and Clinical Pharmacy (JECP) 4, no. 2 (2024): 95. http://dx.doi.org/10.52365/jecp.v4i2.1074.

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Kulit merupakan jaringan terluar di tubuh manusia yang menjadi pertahanan pertama dari masuknya patogen. Kulit dihuni oleh berbagai flora normal, salah satunya adalah Staphylococcus epidermidis. S. epidermidis adalah patogen oportunistik yang menjadi penyebab infeksi nosokomial yang menetap pada perangkat medis. Kasus infeksi nosokomial banyak terjadi baik di negara maju maupun negara berkembang. Infeksi nosokomial karena S. epidermidis tergolong sulit diatasi karena adanya kemampuan spesies ini dalam membentuk biofilm dan resistensi antibiotik yang menyebabkan kasus nosokomial berujung kemati
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18

Yang, Albert-Jackson, Shinta Marito, John-Jackson Yang, et al. "A Microtube Array Membrane (MTAM) Encapsulated Live Fermenting Staphylococcus epidermidis as a Skin Probiotic Patch against Cutibacterium acnes." International Journal of Molecular Sciences 20, no. 1 (2018): 14. http://dx.doi.org/10.3390/ijms20010014.

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Antibiotics without selectivity for acne treatment may destroy the beneficial microbes in the human microbiome that helps to fight Cutibacterium acnes (C. acnes), a bacterium associated with inflammatory acne vulgaris. Probiotic treatment by direct application of live Staphylococcus epidermidis (S. epidermidis) onto the open acne lesions may run the risk of bloodstream infections. Here, we fabricated the polysulfone microtube array membranes (PSF MTAM) to encapsulate probiotic S. epidermidis. We demonstrate that the application of the encapsulation of S. epidermidis in PSF MTAM enhanced the gl
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19

Шипицына, Ирина В., та Елена В. Осипова. "Резистентость <i>S. epidermidis</i>, выделенных из ран пациентов с гнойной инфекцией костей и суставов, к антибактериальным препаратам". Экспериментальная и клиническая фармакология 86, № 9 (2023): 43–48. http://dx.doi.org/10.30906/0869-2092-2023-86-9-43-48.

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Проведено ретроспективное исследование чувствительности к антибактериальным препаратам штаммов S. epidermidis, выделенных в монокультуре и в составе ассоциаций из очагов инфекции у пациентов с гнойно-воспалительными заболеваниями костей и суставов. Показана резистентность исследуемых клинических изолятов S. epidermidis к амоксициллину (70,5 ± 5,3 %), цефтриаксону (70,2 ± 6,7 %), ампициллину/сулбактаму (69,3 ± 6,5 %). Обнаружено, что 43,5 % штаммов S. epidermidis обладают резистентностью к 6 и более протестированным антибактериальным препаратам. Доля MRSE среди всех штаммов — 51,4 %. Значительн
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20

Banaszkiewicz, Sylwia, Jessica K. Calland, Evangelos Mourkas, Samuel K. Sheppard, Ben Pascoe, and Jacek Bania. "Genetic Diversity of Composite Enterotoxigenic Staphylococcus epidermidis Pathogenicity Islands." Genome Biology and Evolution 11, no. 12 (2019): 3498–509. http://dx.doi.org/10.1093/gbe/evz259.

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Abstract The only known elements encoding enterotoxins in coagulase-negative staphylococci are composite Staphylococcus epidermidis pathogenicity islands (SePIs), including SePI and S. epidermidis composite insertion (SeCI) regions. We investigated 1545 Staphylococcus spp. genomes using whole-genome MLST, and queried them for genes of staphylococcal enterotoxin family and for 29 ORFs identified in prototype SePI from S. epidermidis FRI909. Enterotoxin-encoding genes were identified in 97% of Staphylococcus aureus genomes, in one Staphylococcus argenteus genome and in nine S. epidermidis genome
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21

Vadillo-Rodríguez, Virginia, Irene Fernández-Babiano, Ciro Pérez-Giraldo, and María Coronada Fernández-Calderón. "Anti-Biofilm Perspectives of Propolis against Staphylococcus epidermidis Infections." Biomolecules 14, no. 7 (2024): 779. http://dx.doi.org/10.3390/biom14070779.

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Staphylococcus epidermis has emerged as the main causative agent of medical device-related infections. Their major pathogenicity factor lies in its ability to adhere to surfaces and proliferate into biofilms, which increase their resistance to antibiotics. The main objective of this study was to evaluate the use and the mechanism of action of an ethanolic extract of Spanish propolis (EESP) as a potential alternative for preventing biofilm-related infections caused by S. epidermidis. The chemical composition of propolis is reported and its antibacterial activity against several strains of S. ep
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22

Herdiansyah, Al Fadil, La Ode Bariun, and Citra Dewi. "Uji Aktivitas Antibakteri Ekstrak Etanol Daun Suruhan (Peperomia Pellucida L.Kunth) Terhadap Bakteri Staphylococcus Aureus dan Staphylococcus Epidermidis." Jurnal Pharmacia Mandala Waluya 2, no. 2 (2023): 106–16. http://dx.doi.org/10.54883/28296850.v2i2.67.

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Jerawat sering terjadi dikarenakan bakteri Staphylococcus aureus dan Staphylococcus epidermidis. Tujuan penelitian ini adalah untuk mengetahui aktivitas ekstrak daun suruhan (Peperomia pellucida L.Kunth) terhadap pertumbuhan bakteri Staphylococcus aureus dan Staphylococcus epidermis. Penelitian ini merupakan penelitian analitik dengan menggunakan sampel daun suruhan (Peperomia pellucida L.Kunth) yang diekstraksi dengan metode maserasi menggunakan pelarut etanol 96% dipekatkan dalam rotary evaporator hingga mendapatkan ekstrak yang kental. Selanjutnya pengujian aktivitas antibakteri ekstrak eta
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23

Rademacher, Franziska, Maren Simanski, Bettina Hesse, et al. "Staphylococcus epidermidis Activates Aryl Hydrocarbon Receptor Signaling in Human Keratinocytes: Implications for Cutaneous Defense." Journal of Innate Immunity 11, no. 2 (2018): 125–35. http://dx.doi.org/10.1159/000492162.

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Bacterial challenge of keratinocytes with the abundant skin commensal Staphylococcus epidermidis induces distinct innate immune responses, but the underlying molecular mechanisms are still emerging. We report that the aryl hydrocarbon receptor (AhR) was activated in human primary keratinocytes infected with S. epidermidis, leading to induction of the AhR-responsive gene cytochrome P450 1A1 (CYP1A1). In addition, functional AhR was required for S. epidermidis-mediated induction of IL-1β expression in keratinocytes. AhR-dependent gene induction of IL-1β and CYP1A1 was mediated by factor(s) &lt;
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24

Benthien, Hannah, Beate Fresenborg, Linda Pätzold, et al. "The Transcription Factor SpoVG Is of Major Importance for Biofilm Formation of Staphylococcus epidermidis under In Vitro Conditions, but Dispensable for In Vivo Biofilm Formation." International Journal of Molecular Sciences 23, no. 6 (2022): 3255. http://dx.doi.org/10.3390/ijms23063255.

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Staphylococcus epidermidis is a common cause of device related infections on which pathogens form biofilms (i.e., multilayered cell populations embedded in an extracellular matrix). Here, we report that the transcription factor SpoVG is essential for the capacity of S. epidermidis to form such biofilms on artificial surfaces under in vitro conditions. Inactivation of spoVG in the polysaccharide intercellular adhesin (PIA) producing S. epidermidis strain 1457 yielded a mutant that, unlike its parental strain, failed to produce a clear biofilm in a microtiter plate-based static biofilm assay. A
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25

Jannah, Munaziatul, Haris Munandar Nasution, M. Pandapotan Nasution, and Yayuk Putri Rahayu. "Skrining fitokimia dan uji aktivitas antibakteri ekstrak etanol daun kayu Jawa ((Lannea coromandelica) (Houtt) Merr terhadap bakteri Propionibacterium acnes dan Staphylococcus epidermidis." Journal of Pharmaceutical and Sciences 6, no. 4 (2023): 1685–92. http://dx.doi.org/10.36490/journal-jps.com.v6i4.241.

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Kayu Jawa (Lannea coromandelica) (Houtt.) Merr. merupakan tanaman tropis yang berpotensi sebagai tanaman obat. Hampir seluruh bagian tanaman ini dapat dimanfaatkan sebagai bahan obat tradisional. Tumbuhan liar ini dapat ditemukan dengan mudah di berbagai lokasi seperti halaman rumah, tepi jalan, dan kebun masyarakat. Penelitian ini bertujuan untuk mengevaluasi potensi aktivitas antibakteri ekstrak etanol dari daun Kayu Jawa (Lannea coromandelia) (Houtt.) Merr. terhadap pertumbuhan bakteri Propionibacterium acnes dan Staphylococcus epidermidis. Penelitian ini menggunakan metode eksperimental ya
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26

Kaplan, Jeffrey B., Chandran Ragunath, Kabilan Velliyagounder, Daniel H. Fine, and Narayanan Ramasubbu. "Enzymatic Detachment of Staphylococcus epidermidis Biofilms." Antimicrobial Agents and Chemotherapy 48, no. 7 (2004): 2633–36. http://dx.doi.org/10.1128/aac.48.7.2633-2636.2004.

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ABSTRACT The gram-positive bacterium Staphylococcus epidermidis is the most common cause of infections associated with catheters and other indwelling medical devices. S. epidermidis produces an extracellular slime that enables it to form adherent biofilms on plastic surfaces. We found that a biofilm-releasing enzyme produced by the gram-negative periodontal pathogen Actinobacillus actinomycetemcomitans rapidly and efficiently removed S. epidermidis biofilms from plastic surfaces. The enzyme worked by releasing extracellular slime from S. epidermidis cells. Precoating surfaces with the enzyme p
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27

Nair, T. Bhaskaran, Radhika K. Rajendran, K. S. Deepak, and D. Deepa. "Comparative Study on Antibiogram of Staphylococcus epidermidis Isolated from Clinical Samples and from Healthy Skin of the same Patient." International Journal of Current Microbiology and Applied Sciences 12, no. 11 (2023): 168–76. http://dx.doi.org/10.20546/ijcmas.2023.1211.014.

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Staphylococcus epidermidis is the most common normal flora of human skin. There is a delicate balance between skin barrier and innate immunity to maintain healthy skin and disturbance of the balance can predispose the host to a number of cutaneous and systemic infections. Aim-The present study is intended to find out the frequency of Staphylococcus epidermidis (S. epidermidis) of the patient normal flora causing skin infections by comparing the antibiotic sensitivity pattern of the strains isolated from clinically proven cases and strains from the healthy skin sites of the same patient. Method
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Srilistiany, Annisa Tri, Erlin Syahril, Zulfahmidah Zulfahmidah, Nurelly Noro Waspodo, and Dahlia Dahlia. "Uji Aktivitas Antibakteri Air Perasan Jeruk Nipis Terhadap Pertumbuhan Bakteri Staphylococcus epidermidis." Indonesian Journal of Health 1, no. 03 (2022): 141–49. http://dx.doi.org/10.33368/inajoh.v1i03.62.

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Penyakit infeksi merupakan salah satu permasalahan kesehatan di masyarakat yang sulit diatasi secara tuntas. Bakteri yang dapat menimbulkan penyakit infeksi adalah bakteri Staphylococcus epidermidis. Penyakit yang disebabkan oleh infeksi bakteri umumnya diobati dengan pemberian antibakteri. Tumbuhan yang memiliki potensi untuk dikembangkan sebagi antibakteri adalah jeruk nipis (Citrus aurantifolia swingle). Jeruk nipis memiliki kandungan flavonoid, fenol, dan minyak atsiri yang mampu menghambat pertumbuhan bakteri Staphylococcus epidermidis. Untuk mengetahui konsentrasi hambat minimum dan maks
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Dohare, Suhaga, Devendra Singh, Deepmala Sharma, and Vishnu Agarwal. "EFFECT OF Staphylococcus epidermidis ON Pseudomonas aeruginosa BIOFILM IN MIXED-SPECIES CULTURE." Journal of Experimental Biology and Agricultural Sciences 9, no. 3 (2021): 325–34. http://dx.doi.org/10.18006/2021.9(3).325.334.

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Staphylococcus epidermidis and Pseudomonas aeruginosa, are clinically relevant pathogens that often produce biofilms. To investigate the co-survivability of S. epidermidis and P. aeruginosa in mixed cultures biofilm and planktonic form, it is important to understand more about the interspecies interaction of both species. The interspecies interaction was analyzed using streak and drop agar plate assay, cell viability assay (CFU), spectrophotometry-based method, and microscopic analysis. The findings suggest that both cells and supernatant of P. aeruginosa inhibit the planktonic growth of S. ep
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30

Ohkubo, Tomotaka, Yasuhiko Matsumoto, Otomi Cho, Yuki Ogasawara, and Takashi Sugita. "Delftia acidovorans secretes substances that inhibit the growth of Staphylococcus epidermidis through TCA cycle-triggered ROS production." PLOS ONE 16, no. 7 (2021): e0253618. http://dx.doi.org/10.1371/journal.pone.0253618.

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The proportion of Staphylococcus aureus in the skin microbiome is associated with the severity of inflammation in the skin disease atopic dermatitis. Staphylococcus epidermidis, a commensal skin bacterium, inhibits the growth of S. aureus in the skin. Therefore, the balance between S. epidermidis and S. aureus in the skin microbiome is important for maintaining healthy skin. In the present study, we demonstrated that the heat-treated culture supernatant of Delftia acidovorans, a member of the skin microbiome, inhibits the growth of S. epidermidis, but not that of S. aureus. Comprehensive gene
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31

Sun, Daqian, M. A. Accavitti, and J. D. Bryers. "Inhibition of Biofilm Formation by Monoclonal Antibodies against Staphylococcus epidermidis RP62A Accumulation-Associated Protein." Clinical Diagnostic Laboratory Immunology 12, no. 1 (2005): 93–100. http://dx.doi.org/10.1128/cdli.12.1.93-100.2005.

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ABSTRACT Staphylococcus epidermidis expresses a 140-kDa cell wall-bound protein accumulation-associated protein (AAP) to adhere to and accumulate as a biofilm on a surface. Potentially blocking AAP with a monoclonal antibody (MAb) could reduce or eliminate S. epidermidis bacterial colonization of biomedical devices. Here, we report on our efforts to (i) isolate AAP, (ii) generate MAbs against AAP, and (iii) determine the efficacy of MAbs to inhibit S. epidermidis biofilm formation. An M7 S. epidermidis mutant, reportedly deficient in AAP expression, was used as a negative control. Postinoculat
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32

Izano, Era A., Matthew A. Amarante, William B. Kher, and Jeffrey B. Kaplan. "Differential Roles of Poly-N-Acetylglucosamine Surface Polysaccharide and Extracellular DNA in Staphylococcus aureus and Staphylococcus epidermidis Biofilms." Applied and Environmental Microbiology 74, no. 2 (2007): 470–76. http://dx.doi.org/10.1128/aem.02073-07.

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ABSTRACT Staphylococcus aureus and Staphylococcus epidermidis are major human pathogens of increasing importance due to the dissemination of antibiotic-resistant strains. Evidence suggests that the ability to form matrix-encased biofilms contributes to the pathogenesis of S. aureus and S. epidermidis. In this study, we investigated the functions of two staphylococcal biofilm matrix polymers: poly-N-acetylglucosamine surface polysaccharide (PNAG) and extracellular DNA (ecDNA). We measured the ability of a PNAG-degrading enzyme (dispersin B) and DNase I to inhibit biofilm formation, detach prefo
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33

Oliveira, Fernando, Vânia Gaio, Susana Brás, Sofia Oliveira, and Angela França. "Host Soluble Factors Cause Changes in Staphylococcus epidermidis Antibiotic Susceptibility and Biofilm Formation Ability." Pathogens 12, no. 8 (2023): 1064. http://dx.doi.org/10.3390/pathogens12081064.

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Staphylococcus epidermidis is a major nosocomial pathogen with a remarkable ability to adhere to the surfaces of indwelling medical devices and form biofilms. Unlike other nosocomial pathogens, the interaction of S. epidermidis with host factors has not been the focus of substantial research. This study aimed to assess the alterations in the antibiotic susceptibility and biofilm formation ability of S. epidermidis in the presence of host serum factors. S. epidermidis strain RP62A was cultured in a laboratory culture medium with or without human serum/plasma, and changes in antibiotic susceptib
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34

Hadi Rahman, Ilman, Asep Dermawan, Iis Kurniati, and Mamat Rahmat. "EFEKTIVITAS EKSTRAK DAUN TEH PUTIH SEBAGAI ANTIBAKTERI DALAM MENGHAMBAT DAN MEMBUNUH Staphylococcus epidermidis." Jurnal Kesehatan Siliwangi 4, no. 1 (2023): 241–50. http://dx.doi.org/10.34011/jks.v4i1.1454.

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Jerawat disebabkan karena kondisi pori-pori kulit yang tersumbat dan mengakibatkan kantong berisi nanah yang meradang. Jerawat dapat disebabkan oleh beberapa bakteri yang berflora normal dikulit, salah satunya Staphylococcus epidermidis. Bakteri ini berkontribusi mengubah bagian cair sebum menjadi massa padat dan menyumbat saluran kelenjar sebasea. Penggunaan methicillin dapat menyebabkan resistensi terhadap antibiotik lain. Oleh karena itu, diperlukan bahan alam yang berpotensi sebagai antibakteri seperti daun teh putih (Camellia Sinensis L. Kuntze). Tujuan dari penelitian ini adalah untuk me
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35

Kiang, Michelle. "Staphylococcus epidermidis." Pediatrics In Review 24, no. 12 (2003): 430–31. http://dx.doi.org/10.1542/pir.24.12.430.

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36

HENRICKSON, KELLY J., and JERRY L. SHENEP. "Fulminating Staphylococcus epidermidis Bacteremia." Southern Medical Journal 83, no. 2 (1990): 231–34. http://dx.doi.org/10.1097/00007611-199002000-00025.

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Scheifele, David, and Gordean Bjornson. "Staphylococcus epidermidis-associated enterocolitis." Journal of Pediatrics 110, no. 2 (1987): 330. http://dx.doi.org/10.1016/s0022-3476(87)80185-3.

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38

Gruskay, Jeffrey A., Soraya Abbasi, Endia Anday, Stephen Baumgart, and Jeffrey Gerdes. "Staphylococcus epidermidis-associated enterocolitis." Journal of Pediatrics 109, no. 3 (1986): 520–24. http://dx.doi.org/10.1016/s0022-3476(86)80135-4.

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39

Ikeda, Y., Y. Ohara-Nemoto, S. Kimura, K. Ishibashi, and K. Kikuchi. "PCR-based identification ofStaphylococcus epidermidistargetinggseAencoding the glutamic-acid-specific protease." Canadian Journal of Microbiology 50, no. 7 (2004): 493–98. http://dx.doi.org/10.1139/w04-055.

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The frequency of the gseA gene encoding a glutamic acid-specific serine protease, GluSE, of Staphylococcus epidermidis was investigated. DNA hybridization analysis demonstrated that gseA existed exclusively in S. epidermidis but not in other bacteria examined. A single step PCR assay with a set of designed primers yielded amplification of gseA from all 69 clinical isolates of S. epidermidis taken from patients and healthy adults, whereas production of GluSE was observed in 74% (51/69) of the isolates. Furthermore, none of the 46 clinical isolates of other species of coagulase-negative staphylo
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Broekhuizen, Corine A. N., Leonie de Boer, Kim Schipper, et al. "Peri-Implant Tissue Is an Important Niche for Staphylococcus epidermidis in Experimental Biomaterial-Associated Infection in Mice." Infection and Immunity 75, no. 3 (2006): 1129–36. http://dx.doi.org/10.1128/iai.01262-06.

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ABSTRACT Biomaterial-associated infections (BAI), which are predominantly caused by Staphylococcus epidermidis, are a significant problem in modern medicine. Biofilm formation is considered the pivotal element in the pathogenesis, but in previous mouse studies we retrieved S. epidermidis from peri-implant tissue. To assess the kinetics and generality of tissue colonization, we investigated BAI using two S. epidermidis strains, two biomaterials, and two mouse strains. With small inocula all implants were culture negative, whereas surrounding tissues were positive. When higher doses were used, t
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Suharyanisa, Suharyanisa, Syarifah Roslianizar, and Makarios Harefa. "UJI AKTIVITAS ANTIBAKTERI EKSTRAK ETANOL BENGKUANG (Pachyrhizus erosus) TERHADAP Staphylococcusepidermidis dan Staphylococcus aureus." JURNAL FARMANESIA 10, no. 2 (2023): 31–37. http://dx.doi.org/10.51544/jf.v10i2.4594.

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Infeksi kulit merupakan salah satu penyakit yang masih menjadi masalah kesehatan yang disebabkan oleh mikroorganisme yang dapat menginfeksi luka pada permukaan kulit. Umbi bengkuang mengandung serat, inulin, vitamin C, flavonoid, dan daidzein yang bermanfaat untuk prebiotik, antidiabetes mellitus, immunomodulator, fitoestrogen, dan kecantikan. Bakteri Staphylococcus epidermidis dan Staphylococcus aureus menyebabkan infeksi yang disertai nanah karena terjadi kerusakan jaringan. Berdasarkan uraian pada di atas maka, pada penelitian ini akan dilakukan uji aktivitas antibakteri ekstrak etanol beng
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Kumar, Manish, Binderiya Myagmardoloonjin, Sunita Keshari, Indira Putri Negari, and Chun-Ming Huang. "5-methyl Furfural Reduces the Production of Malodors by Inhibiting Sodium l-lactate Fermentation of Staphylococcus epidermidis: Implication for Deodorants Targeting the Fermenting Skin Microbiome." Microorganisms 7, no. 8 (2019): 239. http://dx.doi.org/10.3390/microorganisms7080239.

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Staphylococcus epidermidis (S. epidermidis) is a common bacterial colonizer on the surface of human skin. Lactate is a natural constituent of skin. Here, we reveal that S. epidermidis used sodium l-lactate as a carbon source to undergo fermentation and yield malodors detected by gas colorimetric tubes. Several furan compounds such as furfural originating from the fermentation metabolites play a role in the negative feedback regulation of the fermentation process. The 5-methyl furfural (5MF), a furfural analog, was selected as an inhibitor of sodium l-lactate fermentation of S. epidermidis via
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43

Ortega-Peña, Silvestre, Sandra Rodríguez-Martínez, Mario E. Cancino-Diaz, and Juan C. Cancino-Diaz. "Staphylococcus epidermidis Controls Opportunistic Pathogens in the Nose, Could It Help to Regulate SARS-CoV-2 (COVID-19) Infection?" Life 12, no. 3 (2022): 341. http://dx.doi.org/10.3390/life12030341.

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Staphylococcus epidermidis is more abundant in the anterior nares than internal parts of the nose, but its relative abundance changes along with age; it is more abundant in adolescents than in children and adults. Various studies have shown that S. epidermidis is the guardian of the nasal cavity because it prevents the colonization and infection of respiratory pathogens (bacteria and viruses) through the secretion of antimicrobial molecules and inhibitors of biofilm formation, occupying the space of the membrane mucosa and through the stimulation of the host’s innate and adaptive immunity. The
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44

Juárez-Verdayes, Marco A., Pilar M. González-Uribe, Humberto Peralta, et al. "Detection of hssS, hssR, hrtA, and hrtB genes and their expression by hemin in Staphylococcus epidermidis." Canadian Journal of Microbiology 58, no. 9 (2012): 1063–72. http://dx.doi.org/10.1139/w2012-086.

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Staphylococcus aureus employs a heme sensing system (HssR–HssS) and a heme-regulated transporter efflux pump (HrtA–HrtB) to avoid the accumulation of heme, which is toxic at high concentrations. The detoxification system to heme has not been studied in Staphylococcus epidermidis . In this work, the hssR, hssS, hrtA, and hrtB genes were detected, and their expression when stimulated by hemin in S. epidermidis was explored. In silico genomic analyses exhibited that the genetic organization of the hssRS and hrtAB genes was identical in 11 Staphylococcus species analyzed, including S. epidermidis.
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Masuda-Kuroki, Kana, Shahrzad Alimohammadi, and Anna Di Nardo. "S. epidermidis Rescues Allergic Contact Dermatitis in Sphingosine 1-Phosphate Receptor 2-Deficient Skin." International Journal of Molecular Sciences 24, no. 17 (2023): 13190. http://dx.doi.org/10.3390/ijms241713190.

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Recent studies have identified a subtype of the S1P-receptor family called sphingosine-1-phosphate receptor 2 (S1PR2), which plays a crucial role in maintaining the skin barrier. It has been observed that S1PR2 and Staphylococcus epidermidis (S. epidermidis) work together to regulate the skin barrier. However, the interaction between these two factors is still unclear. To investigate this, a study was conducted on healthy skin and allergic contact dermatitis (ACD) using 3,4-Dibutoxy-3-cyclobutene-1,2-dione (SADBE) on the ears of S1pr2fl/fl and S1pr2fl/flK14-Cre mice and using 1 × 106 CFU of S.
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Sangitaa, Km, Anita Pandeyb, Geeta Guptaa, Peetam Singhb, and Taibat Zahoor b. "Molecular characterization of linezolid resistant Staphylococcus epidermidis from a tertiary care hospital." Biomedicine 45, no. 1 (2024): 75–81. https://doi.org/10.51248/v45i1.135.

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Introduction and Aim: Staphylococcus species are important bacterial pathogens implicated in invasive infections. Staphylococcus aureus and coagulase negative staphylococci including Staphylococcus epidermidis have been considered as multi drug resistant and emergence of linezolid resistance in staphylococci has been reported. The molecular characterization of linezolid resistance in Staphylococcus epidermidis isolated from clinical specimens of blood culture was planned in this study by targeting cfr and optrA genes associated with linezolid resistance. Materials and Methods: A total of 120 c
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Rennermalm, Anna, Martin Nilsson, and Jan-Ingmar Flock. "The Fibrinogen Binding Protein of Staphylococcus epidermidis Is a Target for Opsonic Antibodies." Infection and Immunity 72, no. 5 (2004): 3081–83. http://dx.doi.org/10.1128/iai.72.5.3081-3083.2004.

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ABSTRACT Antibodies against the fibrinogen binding protein (Fbe) of Staphylococcus epidermidis significantly increased macrophage phagocytosis. Antibodies against autolysin E were opsonic but to a lesser extent. Antibodies against a novel, putatively surface-located antigen were unable to enhance phagocytosis. The severity of systemic infection of mice with S. epidermidis was reduced if the bacteria were preopsonized with anti-Fbe prior to administration. Fbe is thus a strong candidate for protein vaccination against S. epidermidis infection, and antibodies against Fbe can be used to prevent o
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Qomar, Moh Syaifuddin, Moch Agus Krisno Budiyanto, Sukarsono Sukarsono, Sri Wahyuni, and Husamah Husamah. "EFEKTIVITAS BERBAGAI KONSENTRASI EKSTRAK DAUN KAYU MANIS (Cinnamomum burmannii [Ness.] BI) TERHADAP DIAMETER ZONA HAMBAT PERTUMBUHAN BAKTERI Staphylococcus epidermidis." Jurnal Biota 4, no. 1 (2018): 12–18. http://dx.doi.org/10.19109/biota.v4i1.1454.

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Staphylococcus epidermidis is one of the bacteria that can cause infectious diseases. In general Staphylococcus epidermidis can cause diseases of swelling (abscess) such as skin infections or acne. Cinnamon (Cinnamomum burmannii [Ness.] BI) leaf extract has a substance which gives antimicrobial effects that can be used as an inhibitor of the Staphylococcus epidermidis bacteria’s growth because it contains substances such as essential oils, flavonoids, saponins, tannins, and alkaloids. The aim of this research is to analyse the effect of the concentration of cinnamon leaf extract and to determi
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Fluckiger, Ursula, Christiane Wolz, and Ambrose L. Cheung. "Characterization of a sar Homolog ofStaphylococcus epidermidis." Infection and Immunity 66, no. 6 (1998): 2871–78. http://dx.doi.org/10.1128/iai.66.6.2871-2878.1998.

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ABSTRACT Coagulase-negative staphylococci are common nosocomial pathogens. A regulatory element, designated sar, partially controls exoprotein synthesis in coagulase-positive Staphylococcus aureus by modulating the expression of another regulatory locus, called agr. We report here the cloning of a sarhomolog in S. epidermidis. The major open reading frame within sar in S. epidermidis is highly homologous (84%) to the S. aureus SarA protein. Primer extension studies revealed three sar transcripts (0.64, 0.76, and 0.85 kb) initiated from three distinct promoters. The interpromoter region in S. e
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Pribadhi, Adhitya Naufal, Sri Mastuti, and Ellyka Purwaningrum. "Aktivitas Antibakteri Dari Bakteri Probiotik Dalam Melawan Propionibacterium acnes Dan Staphylococcus epidermidis." Indobiosains 5, no. 1 (2023): 1–7. http://dx.doi.org/10.31851/indobiosains.v5i1.9659.

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Introduction; Acne vulgaris or commonly called acne, is a disease that is generally suffered by teenagers. Acne is caused by the formation of colonies of pathogenic bacteria such as Propionibacterium acnes and S. epidermidis. Excessive use of antibiotics can lead to bacterial resistance. Therefore, there is a need for other alternatives to the use of antibiotics. Probiotics can be an alternative as an antimicrobial against P. acnes and S. epidermidis bacteria. The aim of this study was to determine the antibacterial activity of probiotic bacteria, namely W. confusa against P. acnes and S. epid
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