Relevant bibliographies by topics / Epidermoid Lewis lung carcinoma

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Epidermoid Lewis lung carcinoma'

Author: Grafiati
Published: 5 June 2025
Last updated: 1 August 2025

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Journal articles on the topic "Epidermoid Lewis lung carcinoma"

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Kiseleva, M. P., V. S. Pokrovsky, L. M. Borisova, I. S. Golubeva, and L. V. Ektova. "N-glycosidesindolo[2,3,-a]pyrrolo[3,4,-c]carbazole derivatives chemical structure influence on antitumor activity." Russian Journal of Biotherapy 18, no. 2 (2019): 32–39. http://dx.doi.org/10.17650/1726-9784-2019-18-2-32-39.

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Introduction . The report considers the prospect of rational approach to the new anticancer agents creation based on indolocarbazole derivatives.Objective . To conduct a comparative study of 12 domestic N-glycosides, indolo[2,3-a]pirrolo[2,3-a]carbazole derivatives in the course of “structure – activity” bond analysis.Materials and methods . The investigation of influence of 12 carbohydrate – containing indolocarbazoles, synthesized in N. N. Blokhin Russian Cancer Research Center of the Ministry of Health of Russia, performed on models of solid transplantable mouse tumors: lung Lewis epidermoi
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G.G., Kudryashov,, Tochilnikov, G.V., Zmitrichenko, Yu.G., et al. "PECULIARITIES OF THE COURSE OF LEWIS LUNG EPIDERMOID CARCINOMA IN MICE INFECTED WITH MYCOBACTER TUBERCULOSIS." CARDIOMETRY, no. 24 (November 30, 2022): 23–24. http://dx.doi.org/10.18137/cardiometry.2022.24.conf.10.

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The potential relationship between pulmonary tuberculosis and lung cancer remains still poorly understood. The aim of our work was to study the features of the course of the tumor process and pulmonary tuberculosis in mice infected with Mycobacterium tuberculosis with transplanted Lewis epidermoid lung carcinoma.
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Dodokhova, Margarita A., Andrei V. Safronenko, Inga M. Kotieva, Margarita S. Alkhuseyn-Kulyaginova, Dmitry B. Shpakovsky, and Elena R. Milaeva. "Impact of organotin compounds on the growth of epidermoid Lewis carcinoma." Research Results in Pharmacology 7, no. (4) (2021): 81–88. https://doi.org/10.3897/rrpharmacology.7.71455.

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Introduction: Search for new compounds with a broad antitumor and antimetastatic potency due to multiple targeting remains important in medicinal chemistry, pharmacology and oncology. We report the efficacy of hybrid organotin agents bis-(3,5-di-tert-butyl-4-hydroxyphenylthiolate) dimethyltin (Ме3) and (3,5-di-tert-butyl-4-hydroxyphenylthiolate) triphenyltin (Ме5). Materials and methods: The compounds were administered to mice bearing the spontaneously metastatic epidermoid Lewis lung carcinoma (LLC). The efficacy of the treatment was evaluated by mean life span, percentage of tumor growth inh
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Dodokhova, M. A., M. A. Akimenko, O. V. Voronova, et al. "Comparative analysis of the use of B16 melanoma and epidermoid Lewis lung carcinoma models for preclinical studies of compounds with a putative antitumor effect." Ural Medical Journal 22, no. 5 (2023): 66–76. http://dx.doi.org/10.52420/2071-5943-2023-22-5-66-76.

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Introduction The search for new compounds with putative antitumor effects and the development of domestic anticancer and antimetastatic drugs based on them is a priority task for specialists in the field of medical chemistry, experimental pathophysiology and pharmacology. Melanoma B16 and Lewis lung epidermoid carcinoma are universal models for evaluating the effect of compounds with putative antitumor action on the primary focus of tumor growth and the process of tumor cells dissemination.The aim of the work is to determine the feasibility of using models of solid tumor growth and metastasis
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Kiseleva, M. P., I. S. Golubeva, V. P. Deryagina, et al. "Influence of Polysaccharide from Helianthus tuberosus L. on Antiproliferative Activity of N-Glycoside Indolo[2,3-a]carbazole Derivative LCS-1269." Biofizika 69, no. 5 (2024): 1109–17. http://dx.doi.org/10.31857/s0006302924050164.

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The Lewis model of epidermoid carcinoma that developed in the lungs of F1(C57Bl/6 × DBA/2) hybrid mice has been used while investigating antitumor activity of the N-glycoside derivative indolo[2,3-a]carbazole (LCS-1269) with a polysaccharide from Helianthus tuberosus L. as an adjuvant agent. The antitumor effect of LCS-1269 together with the polysaccharide was evaluated by the inhibition of tumor growth in treated ani mals in comparison to the control group. As a result, it was found that combination of LCS-1269 with the polysaccharide from Helianthus tuberosus L. provided more pronounced ther
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Dodokhova, Margarita A., Andrei V. Safronenko, Inga M. Kotieva, Margarita S. Alkhuseyn-Kulyaginova, Dmitry B. Shpakovsky, and Elena R. Milaeva. "Impact of organotin compounds on the growth of epidermoid Lewis carcinoma." Research Results in Pharmacology 7, no. 4 (2021): 81–88. http://dx.doi.org/10.3897/rrpharmacology.7.71455.

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Introduction: Search for new compounds with a broad antitumor and antimetastatic potency due to multiple targeting remains important in medicinal chemistry, pharmacology and oncology. We report the efficacy of hybrid organotin agents bis-(3,5-di-tert-butyl-4-hydroxyphenylthiolate) dimethyltin (Ме3) and (3,5-di-tert-butyl-4-hydroxyphenylthiolate) triphenyltin (Ме5). Materials and methods: The compounds were administered to mice bearing the spontaneously metastatic epidermoid Lewis lung carcinoma (LLC). The efficacy of the treatment was evaluated by mean life span, percentage of tumor growth inh
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Kudriashov, G. G., A. O. Nefedov, G. V. Tochilnikov, et al. "THE EFFECT OF TUBERCULOSIS ON THE COURSE OF LUNG CARCINOMA IN THE EXPERIMENT." Molekulyarnaya Meditsina (Molecular medicine) 21, no. 1 (2023): 43–49. http://dx.doi.org/10.29296/24999490-2023-01-06.

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Introduction. One of the problems of modern medicine is the low efficiency of treatment of patients with a coexistence of lung cancer and active tuberculosis. Optimal treatment regimens for coexistence pathology have not been developed at present. In recent years, fundamental research aimed to studying the unexplained mechanisms of the occurrence and course of coexistence pathology. The aim of the study was to investigate the peculiarities of the effect of tuberculosis with different drug resistance on the course of lung carcinoma in an experiment. Methods. The study was performed on 72 mice o
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Saprykina, N. S., L. M. Borisova, M. P. Kiseleva, et al. "Antitumor activity of ormustine against transplantable solid murine tumors. Part I." Russian Journal of Biotherapy 16, no. 4 (2017): 55–60. http://dx.doi.org/10.17650/1726-9784-2017-16-4-55-60.

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Background. Drugs of nitrosourea class are actively and successfully administered in oncology practice. However, at present, the search for new antitumor agents of this class is very urgent. Objective: to examine in vivo antitumor activity of ormustine, a new Russian drug from alkylnitrosourea class, against solid transplantable murine tumors: melanoma B16 and epidermoid Lewis lung carcinoma. Materials and methods. The assessment of antitumor activity of ormustine and a reference drug mustophoran (Les Labolatories Servier, France) was carried out in B6D2F1 mice. Ormustine was administered intr
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Kladnytska, Larysa, Viktor Tomchuk, Vladyslav Velychko, Volodymyr Salata, and Jakov Šengaut. "Influence of allogeneic mesenchymal stem cells of red bone marrow culture on the development of Lewis lung epidermoid carcinoma in vivo." Ukrainian journal of veterinary sciences 15, no. 2 (2024): 102–20. http://dx.doi.org/10.31548/veterinary2.2024.102.

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The relevance of this study is conditioned by the widespread use of stem cells in veterinary medicine, a wide range of studies and ambiguous data on the oncoprotective properties of stem cells of different origins. In this regard, the purpose of this study was to investigate the course of the tumour process in Lewis lung carcinoma and the specific features of the effect of allogeneic mesenchymal stem cells of red bone marrow culture on it. The leading approach to investigating this problem was the method of modelling Lewis lung carcinoma in C57BL6 mice and the use of stem cells. The use of all
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Kotieva, Inga Movlievna, Margarita Avdeevna Dodokhova, Andrey Vladimirovich Safronenko, et al. "Antitumor effectiveness of combination therapy with platinum and hybrid organotin compound on the Lewis epidermoid carcinoma model with metronomic administration." Journal of Clinical Oncology 40, no. 16_suppl (2022): e15080-e15080. http://dx.doi.org/10.1200/jco.2022.40.16_suppl.e15080.

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e15080 Background: The prevalence of lung malignancies in Russia in 2019 was 100.5 per 100,000 population, which is 2.5% more than in the previous year. Drug therapy remains one of the main methods of treatment of malignant lung neoplasms. An urgent task for specialists in experimental pharmacology and oncology is searching for new antitumor compounds. Hybrid organotin compound (3,5-di-tert-butyl-4-hydroxyphenylthiolate)triphenylol) is a promising candidate for antitumor drugs [Milaeva E.R. et al. Novel selective anticancer agents based on Sn and Au complexes. Pure & Applied Chemistry. 202
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Dissertations / Theses on the topic "Epidermoid Lewis lung carcinoma"

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Yan, Dejun. "THE EFFECT OF CURCUMIN ON LEWIS LUNG CARCINOMA." Bowling Green State University / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1308588440.

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Nahreini, Piruz. "Regulation of Lewis lung carcinoma cell growth by prostaglandins." Virtual Press, 1985. http://liblink.bsu.edu/uhtbin/catkey/424559.

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A cloned metastatic variant of a murine tumor cell line, Lewis lung carcinoma (LLC(C3), was synchronized by double exposure to thymidine (5 x 10-4 M) at G1/S boundary. The effects of Prostaglandin (PG)E1, PGE2, PG synthetase inhibitors, and an analog of cAMP (dbut-cANP) on the life cycle of synchronized LLC(C3) cells were examined. When added to cells in G1 phase, PGE1 and dbut-cAMP enhanced 3H-thymidine uptake during S phase. Both of these agents suppressed S phase when they were added to cells entering S phase. The addition of PGE2 to LLC(C3) cells in G1 phase had no effect on S phase of the
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Wheeler, Elizabeth H. "Natural killer cell activity in mice bearing Lewis lung carcinoma." Virtual Press, 1985. http://liblink.bsu.edu/uhtbin/catkey/416656.

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Natural killer (NK) cells are important in limiting tumor dissemination. The NK activity in C57B1/6 mice bearing Lewis lung carcinoma (LLC) was monitored during tumor development. During the initial period of tumor growth, NK activity was enhanced. As tumor growth progressed, NK activity became suppressed. Depletion of macrophages from the spleen cells of tumor-bearing mice restored the NK cytotoxic response. Plasma prostaglandin E2 (PGE2) concentrations were measured by a radioimmunoassay and found to become elevated during the course of tumor growth. To determine whether the suppressed NK ac
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Endicott, Roger A. "Immunoregulation of T-lymphocyte proliferative activity by alveolar macrophages from mice bearing Lewis lung carcinoma tumors." Virtual Press, 1986. http://liblink.bsu.edu/uhtbin/catkey/458971.

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The immune regulatory abilities of alveolar macrophages from C57B1/6 mice bearing a metastatic variant of Lewis lung carcinoma were determined. During early stages of tumor development, or before tumors metastasized to the lungs, alveolar macrophages did not affect or slightly enhanced T-lymphocyte proliferation; as tumor growth progressed, or following tumor metastasis, alveolar macrophages suppressed the T-cell response. Macrophage suppressor activity was probably not mediated by their production of PGE, since macrophages of tumor-bearing mice secreted less 2 PGE than did macrophages of norm
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Shestowsky, William S. "Production and characterization of a monoclonal antibody to a highly metastatic and organ-selective variant of the Lewis lung carcinoma." Thesis, McGill University, 1988. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=61747.

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Duffie, Gordon Patrick. "Tumoricidal activity of pulmonary alveolar macrophages isolated from C57BL/6 mice bearing either a cloned metastatic or nonmetastatic variant of Lewis lung carcinoma." Virtual Press, 1988. http://liblink.bsu.edu/uhtbin/catkey/558376.

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The spontaneous tumoricidal ability of pulmonary alveolar macrophages (PAM) isolated from C57B1/6 mice bearing either a metastatic or a nonmetastatic cloned variant of Lewis lung carcinoma (LLC) was examined in vitro. During the early weeks of tumor development the cytotoxicity mediated by macrophages was enhanced in the tumor-bearing mice, especially in the metastatic tumor bearers. Later in tumor progress (week 4) the spontaneous cytotoxicity of both groups typically declined to levels less than those of normal macrophages. Experiments were performed to determine if macrophages could be acti
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Moraes, Adriel dos Santos 1976. "Administração de nanotubos de carbono "multiwalled" in vivo ativa citotoxicidade tumor-específica de linfócitos T CD8+." [s.n.], 2012. http://repositorio.unicamp.br/jspui/handle/REPOSIP/259942.

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Orientador: Vitor Baranauskas<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Engenharia Elétrica e de Computação<br>Made available in DSpace on 2018-08-20T09:27:39Z (GMT). No. of bitstreams: 1 Moraes_AdrieldosSantos_M.pdf: 1300108 bytes, checksum: bcd1099ef486cc4a2842627fe2b4df57 (MD5) Previous issue date: 2012<br>Resumo: A administração "in vivo" de nanotubos de carbono "multi-walled" (MWCNT's), mesmo sem serem funcionalizados, estimula o aparecimento de uma reposta imune em camundongos normais. Nesse trabalho nós fornecemos evidências de que essas nanopartícula
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Gallot, Yann. "Invalidation du gène de la myostatine dans un modèle murin de cachexie associée au cancer : implication dans la régulation de la masse musculaire." Phd thesis, Université Jean Monnet - Saint-Etienne, 2013. http://tel.archives-ouvertes.fr/tel-01058905.

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La cachexie est un syndrome clinique et métabolique caractérisé par une perte de tissu adipeux et de tissu musculaire, fréquemment observé chez les patients atteints de cancer. La myostatine (Mstn) régule négativement la masse musculaire. Bien que la régulation des mécanismes moléculaires impliqués dans le contrôle de la masse musculaire joue un rôle central dans la cachexie associée au cancer, les relations existant entre la Mstn et les mécanismes physiopathologiques restent largement inconnues. Suite à l'inoculation de cellules Lewis lung carcinoma (LLC) à des souris, nous avons montré que l
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Puka, Juliana. "Perfil biomolecular do derrame pleural maligno experimentalmente induzido: frequência de mutações e impacto de terapias-alvo." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/5/5150/tde-06022017-154634/.

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INTRODUÇÃO: O câncer de pulmão é a principal causa de morte por câncer em todo o mundo e muitos pacientes apresentam derrame pleural em um estágio avançado da doença, com alta morbidade e mortalidade. Entretanto, a patogênese do derrame maligno é ainda pouco compreendida e as opções terapêuticas são limitadas. OBJETIVO: 1) Estudar a fisiopatologia do derrame pleural maligno em modelo animal com células de Lewis em diferentes concentrações; 2) Avaliar os efeitos da terapia intrapleural com anti-VEGF e anti-EGFR e a frequência de mutações de EGFR e KRAS neste modelo. MÉTODOS: Foi utilizado model
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Akramienė, Dalia. "Assessment of the modulation of photodynamic effect by β-glucan and characteristics of anti-CD7 monoclonal antibody during tumor process". Doctoral thesis, Lithuanian Academic Libraries Network (LABT), 2011. http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2011~D_20110309_111259-65000.

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Activation of the immune system during photodynamic therapy (PDT) and improvement of the effector functions of mAbs - these are the ways to use and enhance the potential of the immune system to fight cancer. Tumor cells lack β-glucan as a surface compo¬nent and can‘t trigger complement receptor 3-dependent cellular cytoto¬xicity and initiate tumor-killing activity during PDT. So, it gave rise to the hypothesis that β-glucan in combination with PDT will produce more effective killing of by iC3b fragment opsonized tumor cells. The human Fc portion is essential for the recruiting of human effecto
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Books on the topic "Epidermoid Lewis lung carcinoma"

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Tu, Chau-Khac. Interaction between C57BL-6 mouse bone marrow-derived maturing macrophages and Lewis lung carcinoma cell-recruited supernatant: An in-vitro study. 2000.

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Book chapters on the topic "Epidermoid Lewis lung carcinoma"

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Herbertsson, Helena, Tobias Kühme, and Sven Hammarström. "A 12(s)-Hete Receptor in Lewis Lung Carcinoma Cells." In Advances in Experimental Medicine and Biology. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4861-4_18.

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Orel, V. E., A. D. Shevchenko, A. V. Romanov, et al. "Magnetic Properties of Lewis Lung Carcinoma and Antitumor Magneto-Sensitive Complex." In IFMBE Proceedings. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-34197-7_22.

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Shen, Rong-Nian, Li Lu, Xiao-Qing Jia, Mo-Lam Wong, and Hans E. Kaiser. "The Therapeutic Effect of Naturin-2 on Lewis Lung Carcinoma and Murine-AIDS." In Advances in Experimental Medicine and Biology. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1813-0_82.

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Jones, C. L., and K. V. Honn. "Enhanced Membrane Expression Of Cytoadhesion αIIbβ3 in Lewis Lung Carcinoma Cells by Epoxyeicosatrienoic Acids." In Eicosanoids and Other Bioactive Lipids in Cancer, Inflammation and Radiation Injury. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-3520-1_131.

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Young, M. R., and M. Newby. "Differential Induction of Macrophage Prostaglandin E2 Secretion and Suppressor Activity by Lewis Lung Carcinoma Variants." In Proceedings in Life Sciences. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71904-2_27.

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Rimbach, S., D. Wallwiener, D. Pollmann, et al. "Experimental CO2 Laser Surgery on the Lewis Lung Carcinoma Tumor Model in C56BL/6 Mice." In Lasers in Gynecology. Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-45683-1_63.

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Ismail, M. S., C. Dressler, B. Röder, H. Weitzel, and H. P. Berlien. "Pharmacokinetics Comparison of 132-Hydroxy-Bacteriopheophorbid-A Methyl Ester and Octa-α-Butyloxy-Zinc Phthalocyanine in Mice Bearing Lewis Lung Carcinoma." In Laser in der Medizin Laser in Medicine. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-60306-8_47.

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Duniec, Z. M., L. J. Marnett, and K. V. Honn. "Transformation of Exogenous Arachidonic Acid into a New Metabolite, 12-Keto- 5,8,10,14-Eicosatetraenoic Acid by Lewis Lung Carcinoma Cells: Implications in Tumor Cell Metastasis." In Eicosanoids and Other Bioactive Lipids in Cancer, Inflammation and Radiation Injury. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-3520-1_130.

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Lassie, M. W., B. R. Zetter, S. E. Brightman, and G. L. Blatch. "Mammalian Mtj." In Guidebook to Molecular Chaperones and Protein-Folding Catalysts. Oxford University PressOxford, 1997. http://dx.doi.org/10.1093/oso/9780198599494.003.0051.

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Abstract The cDNA encoding Mtj1 was isolated from an M27 Lewis lung carcinoma expression library (Brightman et al., 1995). Antibodies used for the screen were prepared against 50-60 kDa proteins that were retained on a hydrophobic peptide affinity column (YVGVAPG).
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Zhang, Boyang. "The Effect of Oxaliplatin on the Immunogenic Cell Death and Cell Apoptosis of Human Merkel Cell Cancerous Tumor." In Studies in Health Technology and Informatics. IOS Press, 2023. http://dx.doi.org/10.3233/shti230870.

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Oxaliplatin, as previously studied in the paper, is a derivative of Cisplatin that is effective in treating the Lewis Lung Carcinoma (LLC)4. As it can actively induce immunogenic cell death of the cancer cells, and result in apoptosis, which increases the therapeutic efficacy in the LLC cancer treatment.4 Merkel cell caner is a type of skin cancer that is rare but highly aggressive, with high metastasizing and reoccurring rate. In this study, we aim the determine the potential of Oxaliplatin to induce apoptosis and ICD in cancerous Merkel cell line MCC1, in associate with the PD-1 inhibitor Ni
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Conference papers on the topic "Epidermoid Lewis lung carcinoma"

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Dibra, Denada, Jeffry Cutrera, Xueqing Xia, and Shulin Li. "Abstract 3821: Expression of WSX1 receptor in Lewis Lung Carcinoma promotes tumor growth." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3821.

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Jamasbi, Roudabeh J., Dejun Yan, and Michael E. Geusz. "Abstract 2341: Curcumin targets a stem-like subpopulation of Lewis lung carcinoma cells." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-2341.

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Ge, Wei, Yongfa Zheng, Ling Zhang, et al. "Endostar enhances the radioresponse on lewis lung carcinoma by regulating HIF-1α." In 2011 4th International Conference on Biomedical Engineering and Informatics (BMEI). IEEE, 2011. http://dx.doi.org/10.1109/bmei.2011.6098592.

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Mantareva, Vanya. "Influence of photodynamic therapy on the delay of metastasis development in Lewis lung carcinoma." In Photodynamic Therapy of Cancer II. SPIE, 1995. http://dx.doi.org/10.1117/12.199167.

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Mathew, Biji, Frances E. Lennon, Jessica Siegler, et al. "Abstract C78: The mu opioid receptor regulates Lewis lung carcinoma tumor growth and metastasis." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Nov 15-19, 2009; Boston, MA. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/1535-7163.targ-09-c78.

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Nikolaienko, Tetiana, Liudmyla Garmanchuk, Olena Dzhus, and Larisa Kladnytska. "Influence mesenchymal stem cells on aneuploidy of the tumor cells of Lewis lung carcinoma." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa3655.

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Stepanov, Yurii. "INHIBITION OF LACTATE DEHYDROGENASE REDUCES THE SURVIVAL OF METASTATICALLY ACTIVE LEWIS LUNG CARCINOMA CELLS." In THEORETICAL AND EMPIRICAL SCIENTIFIC RESEARCH: CONCEPT AND TRENDS, chair Denis Kolesnik, Iryna Prokhorova, and Galina Solyanik. European Scientific Platform, 2022. http://dx.doi.org/10.36074/logos-14.10.2022.11.

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Maiak, M. A., D. Sh Dzhalilova, and O. V. Makarova. "MOLECULAR-BIOLOGICAL CHARACTERISTICS OF LEWIS LUNG CARCINOMA PROGRESSION IN MICE WITH DIFFERENT HYPOXIA TOLERANCE." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-257.

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In the present study the features of progression of Lewis lung carcinoma in animals with low and high hypoxia resistance were characterized using morphological and molecular-biological methods. A more pronounced pro-inflammatory phenotype was identified in low resistant to hypoxia animals, a higher mitotic index of primary tumors was found in high resistant to hypoxia animals. There were no differences in metastasis between two groups.
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Zhang, Yong, Nan Zhang, Robert M. Hoffman, and Ming Zhao. "Abstract 704: Comparison of Salmonella typhimurium A1-R and VNP20009 on the Lewis Lung carcinoma." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-704.

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Yan, Lin. "Abstract B77: Curcumin deteriorates trabecular and cortical bone in mice bearing metastatic Lewis lung carcinoma." In Abstracts: AACR Special Conference on Tumor Invasion and Metastasis - January 20-23, 2013; San Diego, CA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.tim2013-b77.

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Reports on the topic "Epidermoid Lewis lung carcinoma"

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Valeri, C. R., John Dittmer, Toshio Ichikura, Hong Qu, and Linda E. Pivacek. Effects of Syngeneic Fresh and Liquid Preserved Red Blood Cells on Primary and Metastatic Growth of the Lewis Lung Carcinoma in Mice. Defense Technical Information Center, 1993. http://dx.doi.org/10.21236/ada360153.

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