Relevant bibliographies by topics / Epigenetic reprogramming

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  2. Dissertations / Theses
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Academic literature on the topic 'Epigenetic reprogramming'

Author: Grafiati
Published: 4 June 2021
Last updated: 26 July 2025

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Journal articles on the topic "Epigenetic reprogramming"

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Stoker, A. "Epigenetic reprogramming." Trends in Genetics 14, no. 2 (1998): 53. http://dx.doi.org/10.1016/s0168-9525(98)01410-3.

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de Lima Camillo, Lucas Paulo, and Robert B. A. Quinlan. "A ride through the epigenetic landscape: aging reversal by reprogramming." GeroScience 43, no. 2 (2021): 463–85. http://dx.doi.org/10.1007/s11357-021-00358-6.

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AbstractAging has become one of the fastest-growing research topics in biology. However, exactly how the aging process occurs remains unknown. Epigenetics plays a significant role, and several epigenetic interventions can modulate lifespan. This review will explore the interplay between epigenetics and aging, and how epigenetic reprogramming can be harnessed for age reversal. In vivo partial reprogramming holds great promise as a possible therapy, but several limitations remain. Rejuvenation by reprogramming is a young but rapidly expanding subfield in the biology of aging.
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Tada, Takashi, Hironobu Kimura, and Masako Tada. ""Nuclear Reprogramming" and "Epigenetic Reprogramming"." Journal of Mammalian Ova Research 21, no. 3 (2004): 97–104. http://dx.doi.org/10.1274/jmor.21.97.

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Watanabe, Akira, Yasuhiro Yamada, and Shinya Yamanaka. "Epigenetic regulation in pluripotent stem cells: a key to breaking the epigenetic barrier." Philosophical Transactions of the Royal Society B: Biological Sciences 368, no. 1609 (2013): 20120292. http://dx.doi.org/10.1098/rstb.2012.0292.

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The differentiation and reprogramming of cells are accompanied by drastic changes in the epigenetic profiles of cells. Waddington's classical model clearly describes how differentiating cells acquire their cell identity as the developmental potential of an individual cell population declines towards the terminally differentiated state. The recent discovery of induced pluripotent stem cells as well as of somatic cell nuclear transfer provided evidence that the process of differentiation can be reversed. The identity of somatic cells is strictly protected by an epigenetic barrier, and these cell
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Barneda-Zahonero, Bruna, Lidia Roman-Gonzalez, Olga Collazo, Tokameh Mahmoudi, and Maribel Parra. "Epigenetic Regulation of B Lymphocyte Differentiation, Transdifferentiation, and Reprogramming." Comparative and Functional Genomics 2012 (2012): 1–10. http://dx.doi.org/10.1155/2012/564381.

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B cell development is a multistep process that is tightly regulated at the transcriptional level. In recent years, investigators have shed light on the transcription factor networks involved in all the differentiation steps comprising B lymphopoiesis. The interplay between transcription factors and the epigenetic machinery involved in establishing the correct genomic landscape characteristic of each cellular state is beginning to be dissected. The participation of “epigenetic regulator-transcription factor” complexes is also crucial for directing cells during reprogramming into pluripotency or
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Li, Anqi, Rui Wang, Yuqiang Zhao, Peiran Zhao, and Jing Yang. "Crosstalk between Epigenetics and Metabolic Reprogramming in Metabolic Dysfunction-Associated Steatotic Liver Disease-Induced Hepatocellular Carcinoma: A New Sight." Metabolites 14, no. 6 (2024): 325. http://dx.doi.org/10.3390/metabo14060325.

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Epigenetic and metabolic reprogramming alterations are two important features of tumors, and their reversible, spatial, and temporal regulation is a distinctive hallmark of carcinogenesis. Epigenetics, which focuses on gene regulatory mechanisms beyond the DNA sequence, is a new entry point for tumor therapy. Moreover, metabolic reprogramming drives hepatocellular carcinoma (HCC) initiation and progression, highlighting the significance of metabolism in this disease. Exploring the inter-regulatory relationship between tumor metabolic reprogramming and epigenetic modification has become one of
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Giuliani, Stefano, Celeste Accetta, Simona di Martino, et al. "Metabolic Reprogramming in Melanoma: An Epigenetic Point of View." Pharmaceuticals 18, no. 6 (2025): 853. https://doi.org/10.3390/ph18060853.

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Metabolic reprogramming and epigenetic alterations are fundamental hallmarks of cancer cells, contributing to adaptation, progression, and resistance. In melanoma, high metabolic-epigenetic plasticity enables the rapid modulation of cell states in response to environmental and therapeutic pressures. Recent studies have highlighted a bidirectional crosstalk between cellular metabolism and epigenetic regulation. Epigenetic modifications influence the transcriptional control of metabolic genes, thereby shaping metabolic phenotypes. Conversely, specific metabolites are essential cofactors or subst
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Bunsick, David A., Jenna Matsukubo, and Myron R. Szewczuk. "Cannabinoids Transmogrify Cancer Metabolic Phenotype via Epigenetic Reprogramming and a Novel CBD Biased G Protein-Coupled Receptor Signaling Platform." Cancers 15, no. 4 (2023): 1030. http://dx.doi.org/10.3390/cancers15041030.

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The concept of epigenetic reprogramming predicts long-term functional health effects. This reprogramming can be activated by exogenous or endogenous insults, leading to altered healthy and different disease states. The exogenous or endogenous changes that involve developing a roadmap of epigenetic networking, such as drug components on epigenetic imprinting and restoring epigenome patterns laid down during embryonic development, are paramount to establishing youthful cell type and health. This epigenetic landscape is considered one of the hallmarks of cancer. The initiation and progression of
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Meiliana, Anna, and Andi Wijaya. "Epigenetic Reprogramming Induced Pluripotency." Indonesian Biomedical Journal 3, no. 2 (2011): 93. http://dx.doi.org/10.18585/inabj.v3i2.139.

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BACKGROUND: The ability to reprogram mature cells to an embryonic-like state by nuclear transfer or by inducing the expression of key transcription factors has provided us with critical opportunities to linearly map the epigenetic parameters that are essential for attaining pluripotency.CONTENT: Epigenetic reprogramming describes a switch in gene expression of one kind of cell to that of another unrelated cell type. Early studies in frog cloning provided some of the first experimental evidence for reprogramming. Subsequent procedures included mammalian somatic cell nuclear transfer, cell fusio
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Hörmanseder, Eva. "Epigenetic memory in reprogramming." Current Opinion in Genetics & Development 70 (October 2021): 24–31. http://dx.doi.org/10.1016/j.gde.2021.04.007.

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Dissertations / Theses on the topic "Epigenetic reprogramming"

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Bagci, Hakan. "Epigenetic reprogramming and DNA demethylation." Thesis, Imperial College London, 2014. http://hdl.handle.net/10044/1/45352.

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Embryonic development begins with fertilization of the egg, a progressive process that gives rise to the zygote and subsequently to the formation of somatic tissues. Normally once cells acquire a fate, it is stably maintained. Conversion back to a multipotent state occurs rarely in-vivo, but can be achieved experimentally by inducing ‘reprogramming’. In this study I am looking at the epigenetic mechanisms that underlie reprogramming and, in particular, DNA methylation and demethylation. To address this I am taking advantage of the cellular fusion system. Fusion of pluripotent cells with differ
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Hajkova, Petra. "Epigenetic reprogramming in mouse germ cells." [S.l. : s.n.], 2002. http://deposit.ddb.de/cgi-bin/dokserv?idn=970526938.

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Rao, Venkata Lakshmi Prakruthi. "Epigenetic Reprogramming at the Th2 Locus." University of Cincinnati / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1543838686940608.

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Ribeiro, Lemos Pereira Carlos Filipe. "Epigenetic events underlying somatic cell reprogramming." Thesis, Imperial College London, 2008. http://hdl.handle.net/10044/1/4439.

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Although differentiated cells normally retain cell-type-specific gene expressionpatterns throughout their lifetime, cell identity can sometimes be modified or reversedin vivo by transdifferentiation, or experimentally through cell fusion or by nucleartransfer. Several studies have illustrated the importance of chromatin remodelling, DNAdemethylation and dominant transcriptional factor expression for changes in lineageidentity. Here the epigenetic mechanisms required to ?reset? genome function wereinvestigated using experimental heterokaryons.To examine the epigenetic changes that are required
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Hajkova, Petra. "Epigenetic reprogramming in mouse germ cells." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2004. http://dx.doi.org/10.18452/15020.

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Bei Säugerkeimzellen, Zygoten und Embryos in frühen Stadien kommt der epigenetischen Neuprogammierung eine außergewöhnlich wichtige Rolle in der Regulation der Genomfunktionen in entscheidenden Entwicklungsstadien zu. Die epigenetische Neuprogrammierung in Keimzellen löscht zuerst die Imprinting-Markierungen und Epi-Mutationen und stellt dann geschlechtsspezifische Markierungen (genomische Prägung) wieder her. Die vorliegende Arbeit bezieht sich auf das Löschen epigenetischer Modifikationen in primordialen Mauskeimzellen (primordial germ cells (PGCs)) zwischen dem 10.5 bis 13.5 Tag nach der
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Oksuz, Samet. "Targeting IL-4 locus for epigenetic reprogramming." University of Cincinnati / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1423581203.

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Yong, Qian Yu. "A screen for modifiers of epigenetic reprogramming." Thesis, Queensland University of Technology, 2011. https://eprints.qut.edu.au/50955/1/Qian_Yu_Yong_Thesis.pdf.

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Epigenetic modifiers are the proteins involved in establishing and maintaining the epigenome of an organism. They are particularly important for development. Changes in epigenetic modifiers have been shown be lethal, or cause diseases. Our laboratory has developed an ENU mutagenesis screen to produce mouse mutants displaying altered epigenetic gene silencing. The screen relies on a GFP transgene that is expressed in red blood cells in a variegated manner. In the orginal transgenic FVB mice expression occurs in approximately 55% of red blood cells. During the course of my Masters, I ch
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Aguilar, Sanchez Cristina. "Epigenetic transitions in cardiovascular development and cell reprogramming." Thesis, University of Edinburgh, 2017. http://hdl.handle.net/1842/28787.

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Epigenetic modifications are alterations in the cell nucleus that affect gene expression and can occur in chromatin at the level of DNA methylation or histone modifications. Such ‘epigenetic marks’ can be heritable through cell division but leave the DNA sequence unchanged. Post-­translational modifications can be found on the histone proteins associated with DNA; the majority of histone modifications are found on the lysine-­rich N-‐terminal amino acid “tails”. Histone acetylation and methylation influence the chromatin structure by loosening or tightening the packaging of DNA, respectively,
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Wanichnopparat, Wachiraporn [Verfasser]. "Epigenetic reprogramming of hepatocyte-like cells / Wachiraporn Wanichnopparat." Saarbrücken : Saarländische Universitäts- und Landesbibliothek, 2020. http://d-nb.info/1239645333/34.

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Eggan, Kevin C. (Kevin Carl) 1974. "Cloning, stem cells and epigenetic reprogramming after nuclear transfer." Thesis, Massachusetts Institute of Technology, 2003. http://hdl.handle.net/1721.1/29931.

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Thesis (Ph. D.)--Massachusetts Institute of Technology, Dept. of Biology, 2003.<br>Includes bibliographical references (leaves 128-146).<br>The process by which a single totipotent cell becomes a complex organism is a unidirectional program, with each mitotic division generating new cells that gradually differentiate towards more specified fates and specialized functions. Nuclear transfer (NT) experiments have demonstrated the epigenetic nature of development and showed, that although differentiated cells have a very limited developmental potential, the nuclei of these cells retain the potency
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Books on the topic "Epigenetic reprogramming"

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Meissner, Alexander, and Jörn Walter, eds. Epigenetic Mechanisms in Cellular Reprogramming. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-31974-7.

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Ancelin, Katia, and Maud Borensztein, eds. Epigenetic Reprogramming During Mouse Embryogenesis. Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-0958-3.

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Pei, Gang, ed. Epigenetic Mechanisms of Cell Programming and Reprogramming. Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-19-7419-9.

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Meissner, Alexander, and Jörn Walter. Epigenetic Mechanisms in Cellular Reprogramming. Springer, 2015.

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Meissner, Alexander, and Jö Walter. Epigenetic Mechanisms in Cellular Reprogramming. Springer London, Limited, 2014.

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Epigenetic Mechanisms In Cellular Reprogramming. Springer-Verlag Berlin and Heidelberg GmbH &, 2014.

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Meissner, Alexander, and Jörn Walter. Epigenetic Mechanisms in Cellular Reprogramming. Springer, 2016.

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Pei, Gang. Epigenetic Mechanisms of Cell Programming and Reprogramming. Springer, 2022.

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Ancelin, Katia, and Maud Borensztein. Epigenetic Reprogramming During Mouse Embryogenesis: Methods and Protocols. Springer, 2021.

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Ancelin, Katia, and Maud Borensztein. Epigenetic Reprogramming During Mouse Embryogenesis: Methods and Protocols. Springer, 2020.

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Book chapters on the topic "Epigenetic reprogramming"

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Mravec, Boris. "Nonmutational Epigenetic Reprogramming." In Neurobiology of Cancer. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-68590-3_19.

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Lindroth, Anders M., Yoon Jung Park, and Christoph Plass. "Epigenetic Reprogramming in Cancer." In Epigenetic Mechanisms in Cellular Reprogramming. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-31974-7_9.

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Lin, Jer-Young, and Tzung-Fu Hsieh. "Epigenetic Reprogramming During Plant Reproduction." In Plant Epigenetics. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-55520-1_20.

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Nakamura, Toshinobu, and Toru Nakano. "Stella and Zygotic Reprogramming." In Epigenetic Mechanisms in Cellular Reprogramming. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-31974-7_2.

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Alberio, Ramiro, and Andrew D. Johnson. "Epigenetic Reprogramming with Oocyte Molecules." In Nuclear Reprogramming and Stem Cells. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-225-0_5.

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Kádár, András, and Tibor A. Rauch. "Epigenetic Reprogramming in Lung Carcinomas." In Patho-Epigenetics of Disease. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-3345-3_7.

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Schwarzer, Caroline, and Michele Boiani. "The Oocyte Determinants of Early Reprogramming." In Epigenetic Mechanisms in Cellular Reprogramming. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-31974-7_1.

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Bošković, Ana, and Maria-Elena Torres-Padilla. "Histone Variants and Reprogramming in Early Development." In Epigenetic Mechanisms in Cellular Reprogramming. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-31974-7_3.

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Lepikhov, Konstantin, Julia Arand, Sarah Fuchs, Jie Lan, Mark Wossidlo, and Jörn Walter. "DNA Methylation Reprogramming in Preimplantation Development." In Epigenetic Mechanisms in Cellular Reprogramming. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-31974-7_4.

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Liao, Jing, and Alexander Meissner. "Establishing and Maintaining Pluripotency: An Epigenetic Perspective." In Epigenetic Mechanisms in Cellular Reprogramming. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-31974-7_5.

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Conference papers on the topic "Epigenetic reprogramming"

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Parfenova, P. S., N. A. Mikhailova, M. G. Khotin, and N. A. Kraskovskaya. "DIRECT REPROGRAMMING OF PATIENT FIBROBLAST INTO NEURON-LIKE CELLS AS A PROMISING MODEL OF HUNTINGTON’S DISEASE." In X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-111.

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The existing research of molecular mechanisms of Huntington’s disease (HD) progression, is expanded here by providing a modified method of direct reprogramming fibroblasts into neuron-like cells that keeps epigenetic changes important for late-onset HD progression. The HD cause seen as an expansion of CAG repeats within the Huntingtin gene. We anticipate our assay to be a starting point for preclinical trials on human cells rather than on mouse models.
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Shiao, Yih-Horng, W. G. Alvord, Xin Ge, et al. "Abstract 184: Stress-induced father-mediated45S rRNAgenetic and epigenetic reprogramming." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-184.

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Benavente, Claudia A., and Michael A. Dyer. "Abstract A163: A role for epigenetic reprogramming in retinoblastoma tumorigenesis." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Nov 12-16, 2011; San Francisco, CA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1535-7163.targ-11-a163.

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Teng, Shuaishuai, Yang Li, Ming Yang, et al. "Abstract 4334: Epigenetic reprogramming of tissue-specific transcription promotes metastasis." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-4334.

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Teng, Shuaishuai, Yang Li, Ming Yang, et al. "Abstract 4334: Epigenetic reprogramming of tissue-specific transcription promotes metastasis." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-4334.

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Puca, Loredana, Dong Gao, Myriam Kossai, et al. "Abstract B41: Targeting androgen-independent prostate cancer through epigenetic reprogramming." In Abstracts: AACR Special Conference: Chromatin and Epigenetics in Cancer; September 24-27, 2015; Atlanta, GA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.chromepi15-b41.

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Estecio, Marcos R. H., Mhair Dekmezian, Juan Gallegos, Shoudan Liang, and Jean-Pierre Issa. "Abstract 2791: SINE retrotransposons cause epigenetic reprogramming of adjacent gene promoters." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-2791.

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Raggi, Chiara, Jens U. Marquardt, Daekwan Seo, Jesper B. Andersen, Valentina M. Factor, and Snorri S. Thorgeirsson. "Abstract 4261: Epigenetic reprogramming affects malignant properties of human liver cancer cells." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-4261.

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Denstaedt, S. J., R. L. Zemans, M. W. Newstead, T. J. Standiford, and B. H. Singer. "Epigenetic Immune Reprogramming in Murine Sepsis Survival Enhances Lipopolysaccharide-Induced Lung Injury." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a7652.

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Cunningham, Isabel, Diane Hamele-Bena, Audrey Papp, et al. "Abstract 1748: Organ-specific epigenetic reprogramming of leukemia cells: Clues to chemoresistance." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-1748.

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