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Books on the topic 'Epilepsy and sleep'

Author: Grafiati

Published: 5 June 2025

Last updated: 1 August 2025

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1

R, Degen, and Rodin Ernst A. 1925-, eds. Epilepsy, sleep, and sleep deprivation. 2nd ed. Elsevier, 1991.

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2

Elaine, Wyllie, Gupta Ajay 1965-, and Lachhwani Deepak K, eds. The treatment of epilepsy: Principles and practice. 4th ed. Lippincott Williams & Wilkins, 2006.

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3

Teresa, Paiva, and Penzel Thomas, eds. European Neurological Network: ENN. IOS Press, 2000.

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4

Epilepsy and Sleep. Elsevier, 2001. http://dx.doi.org/10.1016/b978-0-122-16770-6.x5020-3.

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5

Nobili, Lino, Paola Proserpio, Steve Gibbs, and Giuseppe Plazzi. Sleep and epilepsy. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0028.

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This chapter on sleep and epilepsy examines the activating and deactivating properties of NREM and REM sleep states on interictal epileptic activity and seizures. It reviews specific epileptic syndromes in which seizures manifest a tendency to present exclusively or predominantly during sleep or upon wakening. Particular attention is paid to the description of the different forms of nocturnal frontal lobe epilepsy: autosomal dominant and lesional. There is also a discussion of the negative bidirectional relationship between epilepsy and sleep disorders (sleep apneas and parasomnias) and the ef

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6

(Editor), Carl W. Bazil, Beth A. Malow (Editor), and Michele R. Sammaritano (Editor), eds. Sleep and Epilepsy: The Clinical Spectrum. Elsevier Science Publishing Company, 2002.

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7

Lozsadi, Dora A. Sleep and epilepsy—chicken or egg? Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198778240.003.0011.

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Epilepsy is the commonest serious chronic neurological condition, affecting 0.5% of the population in the UK. Subjective sleep disturbance and excessive daytime sleepiness are reported to be 50% more frequent in those with epilepsy than in controls. Causes are multiple. Both poor seizure control and nocturnal attacks are known to contribute to such sleep disorders. Epilepsy also increases the risk of associated sleep disorders, and additional neurological conditions, such as dementia, learning disability, and depression. These all affect sleep hygiene. Prescribed anti-epileptic drugs will furt

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8

Sleep and epilepsy: The clinical spectrum. Elsevier, 2002.

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9

Epilepsy and Sleep: Physiological and Clinical Relationships. Academic Press, 2001.

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10

Steriade, Mircea. Neuronal Substrates of Sleep and Epilepsy. Cambridge University Press, 2009.

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11

Steriade, Mircea. Neuronal Substrates of Sleep and Epilepsy. Cambridge University Press, 2005.

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12

Steriade, Mircea. Neuronal Substrates of Sleep and Epilepsy. Cambridge University Press, 2003.

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13

Steriade, Mircea. Neuronal Substrates of Sleep and Epilepsy. Cambridge University Press, 2003.

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14

Steriade, Mircea. Neuronal Substrates of Sleep and Epilepsy. Cambridge University Press, 2003.

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15

Luders, Hans O., and Dudley S. Dinner. Epilepsy and Sleep: Physiological and Clinical Relationships. Elsevier Science & Technology Books, 2001.

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16

(Editor), Dudley S. Dinner, and Hans O. Luders (Editor), eds. Epilepsy and Sleep: Physiological and Clinical Relationships. Academic Press, 2001.

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17

Halasz, Peter, and Anna Szucs. Sleep, Epilepsies, and Cognitive Impairment. Elsevier Science & Technology Books, 2018.

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18

Halasz, Peter, and Anna Szucs. Sleep, Epilepsies, and Cognitive Impairment. Elsevier Science & Technology Books, 2017.

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19

Tassinari, Carlo Alberto, and Guido Rubboli. Encephalopathy Related to Status Epilepticus During Slow Sleep: Linking Epilepsy, Sleep Disruption and Cognitive Impairment. Editions John Libbey Eurotext, 2019.

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20

Reading, Paul. Sleep disorders. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0736.

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Despite major advances in our understanding of its neurobiology, sleep remains an enigma. Its true function and even the amount needed for optimum brain performance remain uncertain (Frank 2006). However, the need to sleep is imperative, reflecting the fact that sleepiness, like hunger and thirst, is a true drive state. Sleepiness can only be satiated by sleep itself. Moreover, severely disordered sleep can profoundly affect cognition, mental health, and physical well-being.Although sleep medicine has a traditionally low profile in neurology teaching and practice, sleep-related phenomena are f

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21

Gupta, Ajay, Elaine Wyllie, and Deepak K. Lachhwani. The Treatment of Epilepsy: Principles and Practice. 4th ed. Lippincott Williams & Wilkins, 2005.

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22

Cappuccio, Francesco P., Michelle A. Miller, Steven W. Lockley, and Shantha M. W. Rajaratnam, eds. Sleep, Health, and Society. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198778240.001.0001.

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Sleep disturbances are common in modern society. Since the beginning of the century, populations have shown a decline in sleep duration, owing to changes in environmental and social conditions. Industry was the first to appreciate the detrimental effects of sleep disturbances on health and wellbeing. It has taken, however, many decades to understand the implications for individuals and populations of sustained sleep deprivation. This second edition follows in the footsteps of the first edition published in 2010. It maintains the focus on sleep disturbances as a universal behaviour that affects

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23

Gowers, W. R. 1845-1915. Border-Land of Epilepsy: Faints, Vagal Attacks, Vertigo, Migraine, Sleep Symptoms and Their Treatment. Creative Media Partners, LLC, 2023.

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24

Gowers, W. R. 1845-1915. Border-Land of Epilepsy: Faints, Vagal Attacks, Vertigo, Migraine, Sleep Symptoms and Their Treatment. Creative Media Partners, LLC, 2023.

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25

Gowers, W. R. Border-Land of Epilepsy: Faints, Vagal Attacks, Vertigo, Migraine, Sleep Symptoms, and Their Treatment. Creative Media Partners, LLC, 2018.

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26

(Editor), Teresa Paiva, and Thomas Penzel (Editor), eds. European Neurological Network (Studies in Health Technology and Informatics, V. 78). Ios Pr Inc, 2000.

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27

Reading, Paul J. Neurological diseases and their effects on the sleep–wake cycle. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0035.

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This chapter addresses current neurobiological knowledge of how wake- and sleep-promoting systems interact to produce the daily circadian rhythm of wake and sleep and how this may be adversely affected by a variety of neurological diseases. The crucial importance of sleep quality for optimal brain function is stressed and the potential hazards of prolonged wakefulness highlighted. Insomnia relating to either sleep onset or maintenance is common and increases with normal aging. Many neurodegenerative diseases such as Alzheimer disease appear to enhance the effects of aging on the sleep–wake cyc

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28

Bowker, Lesley K., James D. Price, Ku Shah, and Sarah C. Smith. Neurology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198738381.003.0007.

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This chapter provides information on the ageing brain and nervous system, tremor, neuropathic pain/neuralgia, presentation of Parkinson’s disease, management of Parkinson’s disease, diseases masquerading as Parkinson’s disease, epilepsy and its drug treatment, neuroleptic malignant syndrome, motor neuron disease, peripheral neuropathies, subdural haematoma, sleep and insomnia, and other sleep disorders.

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29

Johnson, Chris, Jon Dallimore, Alistair R. M. Cobb, et al. Treatment: head and neck. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199688418.003.0010.

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Anatomy - Facial injuries - Minor injuries to head and neck - Fractured facial bones - Head injury - Blackouts, syncope, and epilepsy - Migraine - Sleep disturbances - The eye - Ear problems - Nasal problems - Upper respiratory tract

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30

Johnson, Chris, Jon Dallimore, Alistair R. M. Cobb, et al. Treatment: head and neck. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199688418.003.0010_update_001.

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Anatomy - Facial injuries - Minor injuries to head and neck - Fractured facial bones - Head injury - Blackouts, syncope, and epilepsy - Migraine - Sleep disturbances - The eye - Ear problems - Nasal problems - Upper respiratory tract

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31

Koutroumanidis, Michalis, Dimitrios Sakellariou, and Vasiliki Tsirka. Electroencephalography. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0011.

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This chapter concentrates on essential technical aspects of the electroencephalogram (EEG) and its role in the clinical and aetiological diagnosis of people with epilepsy. The technical subsection explores important stages of the largely ‘mystifying’ process from the generation of the abnormal signals in the brain to their final visualization on the screen, including digitalization of the signal and sampling rate, montages, and derivations, focusing on their clinical relevance. The second part reviews the behavioural attributes of the interictal and ictal discharges in the different epilepsy t

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32

Mills, Kerry R., ed. Oxford Textbook of Clinical Neurophysiology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.001.0001.

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The Oxford Textbook of Clinical Neurophysiology provides a comprehensive account from world experts of the modern practice of the specialty. It deals with the full range of techniques giving the underpinning basic science and clinical use. The importance of clinical skills, as well as technical expertise are emphasized. Section I reviews the physiology of nerve, muscle, and cortex, and the digital techniques used to study them. Section II discusses the techniques for nerve conduction, electromyography (EMG), electroencephalography (EEG), magnetoencephalography, evoked potentials, and transcran

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33

Non-epileptic paroxysmal childhood disorders. Oxford University Press, 2009.

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34

Forsyth, Rob, and Richard Newton. Signs and symptoms. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198784449.003.0003.

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This chapter addresses the diagnostic approach to the patterns of symptoms and signs commonly seen in the paediatric neurology clinic. It encourages pattern recognition. The presentations considered are: altered mental state (agitation/confusion); motor disorders (exercise limitation and muscle pain; eye or facial movement abnormalities; the floppy infant; a funny gait; weakness; unsteadiness or falls; toe-walking; disordered sensation, numbness, pain, dysaesthesia; deafness, loss or disturbance of hearing or vision; paroxysmal disorders (funny turns, loss of awareness, epilepsy, headache, mov

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35

Iversen, Leslie. Medical Uses of Marijuana—Fact or Fantasy? Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190846848.003.0005.

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Marijuana has a long history of medical use. This chapter discusses the modern revival of interest in marijuana in the United States, Europe, and elsewhere. The fully licensed cannabinoid medicines dronabinol (Marinol), nabilone (Cesamet), and nabiximols (Sativex) and their uses are discusses. Medical indications include multiple sclerosis, pain, nausea and vomiting, epilepsy, appetite stimulation, mood, sleep, and migraine. Apart from the treatment of spasticity and pain in multiple sclerosis, there is little or no scientific evidence of efficacy because of the lack of controlled clinical tri

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36

Jolly, Elaine, Andrew Fry, and Afzal Chaudhry, eds. Neurology and neurosurgery. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199230457.003.0014.

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Chapter 14 covers the basic science and clinical topics relating to neurology and neurosurgery which trainees are required to learn as part of their basic training and demonstrate in the MRCP. It covers the approach to the neurological Patient, neurological examination, neurological investigations, coma, acquired brain injury, encephalopathies, alcohol and the nervous system, brainstem disorders, common cranial nerve disorders, migraine, other primary headaches, secondary headache, neuro-ophthalmology, vertigo and hearing loss, seizures and epilepsy, intracranial pressure, stroke, central nerv

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37

Stafstrom, Carl E. Dietary Therapy for Neurological Disorders. Edited by Jong M. Rho. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0018.

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Dietary and metabolic therapies such as the high-fat, low-carbohydrate ketogenic diet (KD) are best known for the treatment of intractable epilepsy. Yet, dietary and metabolic approaches have also found some efficacy in a wide variety of other neurological diseases, including autism spectrum disorder, brain trauma, Alzheimer’s disease, sleep disorders, brain tumors, pain, and multiple sclerosis, as discussed in other chapters of this volume. This chapter provides an overview of clinical and experimental studies using the KD in an array of other neurologic disorders: amyotrophic lateral scleros

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38

Forsyth, Rob, and Richard Newton. Specific conditions. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198784449.003.0004.

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This chapter adopts a systematic approach to common diagnoses in paediatric neurology, aetiologies, management to include investigation and treatment, and outcome. For each condition current knowledge on cause and underlying biology is summarized. A rational approach to investigation and treatment is summarized for each topic. These include: acquired brain injury; autoimmune and autoinflammatory disease of the CNS; cerebral palsy and neurodisability which covers feeding, communication, special senses, and respiratory disease; demyelinating disease; epilepsy including its impact on daily life;

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39

Peñagarikano, Olga, and Daniel H. Geschwind. CNTNAP2 and Autism Spectrum Disorders. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199744312.003.0016.

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Although autism was described in the early 1940s as a disorder of affective contact (Kanner, 1943), it was not classified as a neurodevelopmental disorder with a biological basis until the early 1980s, when studies reported its high heritability (Folstein & Rutter, 1977; Ritvo et al., 1985) and co-occurrence with chromosomal abnormalities (Gillberg & Wahlstrom, 1985; Wahlström et al., 1986). Today, autism is considered a heterogeneous neurodevelopmental syndrome and therefore termed autism spectrum disorder (ASD), characterized by variable deficits in social behavior and language, rest

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40

Cheng, Ning, Susan A. Masino, and Jong M. Rho. Metabolic Therapy for Autism Spectrum Disorder and Comorbidities. Edited by Jong M. Rho. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0014.

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Autism spectrum disorder (ASD) is a heretogenous developmental disorder characterized by deficits in sociability and communication and by repetitive and/or restrictive behaviors. Currently, only comorbid manifestations can be alleviated (such as seizures and sleep disturbance) not core behavioral symptoms. Recent studies have increasingly implicated mitochondrial dysfunction as a cause of ASD. Mitochondria play an integral role in many cellular functions and are susceptible to many pathophysiological insults. Derangements in mitochondrial structure and function provide a scientific rationale f

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41

Hawkes, Christopher H., Kapil D. Sethi, and Thomas R. Swift. Instant Neurological Diagnosis. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190930868.001.0001.

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Experienced neurologists work fast. They ask few questions, maybe perform a brief examination, and they come up with the right answer. Sometimes they do neither and their conclusions are accurate—but how do they do it? This book holds the answers. The book is divided into 14 chapters which, for the most part, focus on a particular neurologic condition, namely: demyelination, headache, epilepsy and sleep, myopathy and motor neuron disorders, movement disorders, stroke, peripheral neuropathy, cerebellar ataxia, and dementia. The remaining chapters are concerned with the clinician’s initial impre

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42

Parker, Linda A., Erin M. Rock, and Raphael Mechoulam. CBD. The MIT Press, 2022. http://dx.doi.org/10.7551/mitpress/13686.001.0001.

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A comprehensive review of the scientific literature on the possible benefits of CBD, describing findings from both preclinical and human clinical studies. CBD (cannabidiol), a nonintoxicating compound derived from the cannabis plant, can be found in products ranging from lotion and smoothies to chewable gummies and pet treats. It's been promoted—but not always scientifically validated—as a treatment for medical conditions including psychosis, anxiety, pain, and even cancer. In this book, three leading cannabis researchers look at the science of CBD, offering a comprehensive review of the scien

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43

Rubin, Devon I., and Jasper R. Daube. Clinical Neurophysiology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190259631.001.0001.

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Clinical neurophysiologic testing is an important component of evaluating patients with complaints that may be attributed to diseases of the central or peripheral nervous system. This classic volume in the Contemporary Neurology Series covers the basic concepts underlying each of the testing techniques and provides comprehensive descriptions of the methods and wide range of electrophysiologic testing available for patients with epilepsy, neuromuscular diseases, movement disorders, demyelinating diseases, sleep disorders, autonomic disorders and those undergoing orthopedic and neurosurgical pro

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44

Joshi, Charuta N., and Renzo Guerrini, eds. Reflex Epilepsies for the Practicing Clinician. Oxford University PressNew York, 2025. https://doi.org/10.1093/med/9780197634547.001.0001.

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Abstract Most practicing neurologists will have seen patients with reflex epilepsies—in other words, patients with seizures induced by light stimuli or by sudden, unexpected noises. In some patients, seizures may be triggered by more complex precipitants such as reading, emotion or certain movements or activities. The neurophysiological bases and clinical manifestations associated with these conditions are variable and must be distinguished from those related to other commonly observed seizure precipitating factors such as sleep deprivation, alcohol intake or menstrual cycles whose relationshi

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45

Hawkes, Christopher H., Kapil D. Sethi, and Thomas R. Swift. Instant Neurological Diagnosis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199361953.001.0001.

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Experienced neurologists work fast. They ask few questions, maybe perform a brief examination, and come up with the right answer. Sometimes they do neither and their conclusions are accurate—but how do they do it? Traditionally, the diagnostic process includes pattern recognition and probabilistic, causal, or deterministic methods. The aeronautical expert uses so-called demons—which are essentially memorized diagnostic shortcuts that help to solve problems rapidly. In this text they are called Handles. Another complementary diagnostic process is the use of caveats, or Red Flags, which are nega

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