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Books on the topic 'Epilepsy/pathology'

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1

V, Delgado-Escueta Antonio, National Institute of Neurological and Communicative Disorders and Stroke. Epilepsy Advisory Committee. Subcommittee on Basic Research., and International Symposium on Basic Mechanisms of the Epilepsies (1983 : San Diego, Calif.), eds. Basic mechanisms of the epilepsies: Molecular and cellular approaches. Raven Press, 1986.

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2

Stephenson, John B. P. Fits and faints / John B.P. Stephenson. MacKeith, 1990.

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3

S, Fisher Robert, and Coyle Joseph T, eds. Neurotransmitters and epilepsy. Wiley-Liss, 1991.

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4

Lebrun, Yvan. Language and epilepsy. Whurr, 2002.

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5

Stephenson, John B. P. Fits and faints. MacKeith Press, 1990.

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6

P, Ottersen O., Langmoen Iver Arne, and Gjerstad L, eds. The glutamate synapse as a therapeutical target: Molecular organization and pathology of the glutamate synapse. Elsevier, 1998.

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7

Henry, Sieveking Edward. On Epilepsy and Epileptiform Seizures: Their Causes, Pathology, and Treatment. Adamant Media Corporation, 2005.

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8

The neuropathology of temporal lobe epilepsy. Oxford University Press, 1988.

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9

Bertram, Edward H. Temporal Lobe Epilepsy. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0038.

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Temporal lobe epilepsy, as discussed in this chapter, is a focal epilepsy that involves primarily the limbic structures of the medial temporal lobe (amygdala, hippocampus, and entorhinal cortex). In recent years animal models have been developed that mirror the pathology and pathophysiology of this disease. This chapter reviews the human condition, the structural and physiological changes that support the development of seizures. The neural circuitry of seizure initiation will be reviewed with a goal of creating a framework for developing more effective treatments for this disease.
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10

Kuzniecky, Ruben, and Graeme D. Jackson. Magnetic Resonance in Epilepsy, Second Edition: Neuroimaging Techniques, Second Edition. 2nd ed. Academic Press, 2004.

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11

So, Elson L., and Philippe Ryvlin. MRI-Negative Epilepsy: Evaluation and Surgical Management. Cambridge University Press, 2015.

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12

So, Elson L., and Philippe Ryvlin. MRI-Negative Epilepsy: Evaluation and Surgical Management. Cambridge University Press, 2015.

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13

So, Elson L., and Philippe Ryvlin. MRI-Negative Epilepsy: Evaluation and Surgical Management. Cambridge University Press, 2015.

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14

Lebrun, Yvan, and Franco Fabbro. Language and Epilepsy. Wiley & Sons, Incorporated, John, 2008.

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15

Lebrun, Yvan, and Franco Fabbro. Language and Epilepsy. Wiley & Sons, Limited, John, 2002.

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16

Fits and faints. Mac Keith Press, 1990.

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17

Chen, David K., and W. Curt LaFrance. Nonepileptic Events. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0024.

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Nonepileptic events (NEE) represent important differential diagnoses in patients with neurobehavioral paroxysms, especially those with apparent drug-resistant epilepsy. Errant recognition of NEE may not only subject the patient to potential complications of unnecessary epilepsy treatment, but delay the delivery of treatment that properly addresses the underlying pathology. For many patients with NEE, such as those with the conversion disorder psychogenic nonepileptic seizures (PNES) or with physiologic NEE (e.g., cardiac-induced syncope), delays in the provision of proper treatment have been s
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18

Haig, Alexander. Uric Acid As a Factor in the Causation of Disease: A Contribution to the Pathology of High Blood Pressure, Headache, Epilepsy, Nervousness, Mental ... Bright's Disease, Diabetes, Gout, Rheumatis. Arkose Press, 2015.

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19

Royal College of Physicians of Edinbu and 1862-1931 Hare H a (Hobart Amory). Epilepsy : Its Pathology and Treatment: Being an Essay to Which Was Awarded a Prize of Four Thousand Francs by the Academie Royale de Médécine de Belgique, December 31 1889. Creative Media Partners, LLC, 2021.

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20

Epilepsy : Its Pathology and Treatment: Being an Essay to Which Was Awarded a Prize of Four Thousand Francs by the Académie Royale de Médécine [Sic] de Belgique, December 31 1889. Creative Media Partners, LLC, 2023.

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21

Reading, Paul J. Neurological diseases and their effects on the sleep–wake cycle. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0035.

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This chapter addresses current neurobiological knowledge of how wake- and sleep-promoting systems interact to produce the daily circadian rhythm of wake and sleep and how this may be adversely affected by a variety of neurological diseases. The crucial importance of sleep quality for optimal brain function is stressed and the potential hazards of prolonged wakefulness highlighted. Insomnia relating to either sleep onset or maintenance is common and increases with normal aging. Many neurodegenerative diseases such as Alzheimer disease appear to enhance the effects of aging on the sleep–wake cyc
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22

Researches on Epilepsy: Its Artificial Production in Animals, and Its Etiology, Nature and Treatment in Man. First Part of a New Series of Experimental and Clinical Researches Applied to Physiology and Pathology. Creative Media Partners, LLC, 2023.

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23

Banerjee, Ashis, and Clara Oliver. Neurological emergencies. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198786870.003.0011.

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A number of neurological conditions can present to the emergency department in a variety of presentations. This chapter summarizes the common neurological problems that may be examined in the Intermediate FRCEM short-answer question (SAQ) paper. This chapter includes the pathophysiology and management of an unconscious patient which may commonly appear in the SAQ paper. In addition, it also includes sections of epilepsy, headaches and strokes, and their subclassification and diagnosis. Many individuals find the differentiation of the cause of motor weakness complicated. This chapter summarizes
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24

Quarrell, Oliver W. J. Juvenile Huntington’s Disease. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199929146.003.0004.

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In approximately 5% of cases of Huntington’s disease, onset is before 20 years of age, and these cases are defined as juvenile Huntington’s disease (JHD). The clinical and pathologic features of JHD overlap with those of typical adult-onset disease, so it is not a distinct entity. As a generalization, however, dystonia and bradykinesia are more likely to occur earlier in the disease process, epilepsy is more frequent, the average CAG repeat length is longer, and the pathology within the brain is more widespread. There are conflicting reports in the literature regarding both disease duration an
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25

Haig, Alexander. Uric Acid As a Factor in the Causation of Disease: A Contribution to the Pathology of High Arterial Tension, Headache, Epilepsy, Mental Depression, Paroxysmal Hæmoglobinuria and anæmia, Bright's Disease, Diabetes, Gout, Rheumatism, and Other Disorders. Creative Media Partners, LLC, 2018.

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26

Uric Acid As a Factor in the Causation of Disease: A Contribution to the Pathology of High Blood Pressure, Headache, Epilepsy, Nervousness, Mental Diseases, Asthma, Hay Fever, Paroxysmal Hæmoglobinuria, anæmia, Bright's Disease, Diabetes, Gout, Rheum. Creative Media Partners, LLC, 2022.

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27

Taylor, Eric. Developmental Neuropsychiatry. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198827801.001.0001.

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Neurodevelopmental disorders are a group of conditions involving alterations of behaviour, thinking, and emotions. They have onsets in early childhood, persistence into adult life, and high rates of altered cognitive and neurological function. They are frequent reasons for referral to psychiatry, paediatrics, and clinical psychology and often require team approaches to meet a variety of needs for service. This book includes accounts of the typical development and possible pathology of key functions whose alterations can underlie problems of mental development: motor function, attention, memory
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28

Gill, Steven J., and Michael H. Nathanson. Central nervous system pathologies and anaesthesia. Edited by Philip M. Hopkins. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0081.

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Anaesthesia induces changes in many organ systems within the body, though clearly none more so than the central nervous system. The physiology of the normal central nervous system is complex and the addition of chronic pathology and polypharmacy creates a significant challenge for the anaesthetist. This chapter demonstrates a common approach for the anaesthetist and specific considerations for a wide range of neurological conditions. Detailed preoperative assessment is essential to gain understanding of the current symptomatology and neurological deficit, including at times restrictions on mov
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