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Books on the topic 'Epileptic Seizure'

Author: Grafiati
Published: 4 June 2021
Last updated: 6 September 2023

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1

J, Rowan A., and Gates John R, eds. Non-epileptic seizures. Boston: Butterworth-Heinemann, 1993.

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2

R, Gates John, and Rowan A. J, eds. Non-epileptic seizures. 2nd ed. Boston: Butterworth-Heinemann, 2000.

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3

Lennart, Gram, ed. Pseudo-epileptic seizures. Petersfield, UK: Wrightson Biomedical Pub., 1993.

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4

Panayiotopoulos, C. P. Imitators of epileptic seizures. London: Springer London, 2012. http://dx.doi.org/10.1007/978-1-4471-4023-8.

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5

Caplan, Rochelle, Julia Doss, Sigita Plioplys, and Jana E. Jones. Pediatric Psychogenic Non-Epileptic Seizures. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-55122-7.

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6

Hosten, Willard, and Aleph Burtsev. Seizures and anti-epileptic drugs. New York: Nova Science Publishers, 2012.

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7

Panayiotopoulos, C. P. Reflex seizures and related epileptic syndromes. London: Springer London, 2012. http://dx.doi.org/10.1007/978-1-4471-4042-9.

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8

Hans, Lüders, and Noachtar Soheyl, eds. Epileptic seizures: Pathophysiology and clinical semiology. New York: Churchill Livingstone, 2000.

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9

Lüders, Hans. Atlas of epileptic seizures and syndromes. Philadelphia: Saunders, 2001.

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10

Seizures and epilepsy. Philadelphia: F.A. Davis Co., 1989.

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11

1950-, Shinnar S., Amir N, and Branski D, eds. Childhood seizures. Basel: Karger, 1995.

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12

Recent advances in predicting and preventing epileptic seizures. Singapore: World Scientific Publishing Co. Pte. Ltd., 2013.

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13

Panayiotopoulos, C. P. Benign Childhood Focal Seizures and Related Epileptic Syndromes. London: Springer London, 2011. http://dx.doi.org/10.1007/978-1-84996-477-7.

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14

My seizure dog. [United States]: Moss Family Publishing, 2011.

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15

1959-, Mareels Iven, and Cook Mark 1960-, eds. Epileptic seizures and the EEG: Measurement, models, detection, and prediction. Boca Raton: Taylor & Francis, 2010.

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16

Nancy, Santilli, ed. Managing seizure disorders: A handbook for health care professionals. Philadelphia, PA: Lippincott=Raven Publishers, 1996.

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17

Seizure-alert dogs. New York, N.Y: Bearport Pub., 2010.

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18

C, Schachter Steven, ed. Epilepsy in our words: Personal accounts of living with seizures. New York: Oxford University Press, 2008.

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19

Ann, Jacoby, and Baker Gus A, eds. Quality of life in epilepsy: Beyond seizure counts in assessment and treatment. Amsterdam: Harwood Academic, 2000.

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20

Frederick, Andermann, Beaumanoir A, and Fondazione Pierfranco e Luisa Mariani., eds. Falls in epileptic and non-epileptic seizures during childhood: International colloquium of the Pierfranco e Luisa Mariani Foundation, Milan State University, 12-14 October 1995. London: J. Libbey, 1997.

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21

C, Schachter Steven, and Andermann Lisa Francesca, eds. Epilepsy in our world: Stories of living with seizures from around the world. New York: Oxford University Press, 2007.

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22

Epileptic Seizure. Bearport Publishing Company, Incorporated, 2022.

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23

Pointer, Charis Mather; Jasmine. Epileptic Seizure. BookLife Publishing Ltd., 2022.

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24

Epileptic Seizure. Bearport Publishing Company, Incorporated, 2022.

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25

Walter, Mark T. CBD Oil for Epileptic Seizure: The Therapeutic Effect of CBD Oil on Epileptic Seizure. Independently Published, 2019.

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26

Ajmone-Marsan, Cosimo. Epileptic Seizure Its Functional Morphology and Diagnosis. Charles C Thomas Pub Ltd, 2000.

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27

JOHNSON, Thomas. Cbd Oil for Epileptic Seizure: Everything You Need to Know about Using CBD Oil to Cure Epileptic Seizure. Independently Published, 2019.

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28

EEG Brain Signal Classification for Epileptic Seizure Disorder Detection. Elsevier, 2019. http://dx.doi.org/10.1016/c2018-0-01888-5.

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29

Dehuri, Satchidananda, Alok Kumar Jagadev, Shruti Mishra, and Sandeep Kumar Satapathy. EEG Brain Signal Classification for Epileptic Seizure Disorder Detection. Elsevier Science & Technology Books, 2019.

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30

Dehuri, Satchidananda, Alok Kumar Jagadev, Shruti Mishra, and Sandeep Kumar Satapathy. EEG Brain Signal Classification for Epileptic Seizure Disorder Detection. Elsevier Science & Technology, 2019.

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31

Schachter, Steven C. Non-Epileptic Seizures in Our Experience. Edited by Markus Reuber and Gregg H. Rawlings. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780190927752.001.0001.

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To an outside observer, Psychogenic Non-Epileptic Seizures (PNES) look like epileptic seizures. The manifestations of PNES include collapses, impaired consciousness, and seizure-related injuries. However, unlike epileptic seizures, which are the result of abnormal electrical discharges in the brain, most PNES are an automatic psychological response to a trigger perceived as threatening. Not least because the changes in the brain that underpin PNES cannot be visualized easily with clinical tests (such as the EEG), there are many uncertainties and controversies surrounding the condition. Patients often provoke a mixture of emotions in healthcare professionals. In the authors’ previous book, In Our Words: Personal Accounts of Living with Non-Epileptic Seizures, over 100 individuals with PNES and their families wrote about their experiences with the condition. While some had positive care experiences, most were left feeling confused, angry, and abandoned by the clinicians they had encountered. Non-Epileptic Seizures in Our Experience: Accounts of Healthcare Professionals complements the previous book by presenting the perspectives of over ninety healthcare professionals from around the world. The anonymous publication format enabled many not only to share success stories but also to be open about difficulties and failures. There will be something to learn from this book for highly experienced professionals as well as for relative novices and those experiencing PNES. The hope is that this book will challenge negative attitudes surrounding the condition, improve understanding between healthcare professionals and patients, and, ultimately, advance the quality of care provided for those with PNES.
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32

Chadwick, David. Seizures, epilepsy, and other episodic disorders in adults. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0709.

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Epilepsy, or more correctly a seizure, is most easily defined in physiological terms, being ‘the name for occasional sudden, excessive, rapid, and local discharges of grey matter’ (Jackson 1873). It is more difficult to offer a comprehensive clinical definition of epileptic seizures and epilepsy because of the varied clinical manifestations produced by cerebral neuronal discharge. However, an epileptic seizure can be defined as an intermittent and stereotyped disturbance of consciousness, behaviour, emotion, motor function, or sensation that on clinical grounds is believed to result from cortical neuronal discharge. Epilepsy can then be defined as a condition in which seizures recur, usually spontaneously. The differential diagnosis of epilepsy is large because of the enormous range of symptoms that can occur during seizures. Inevitably, the differential diagnosis for tonic-clonic seizures is very different from that for simple partial seizures with autonomic symptoms. The most common clinical problem is the differential diagnosis from other causes of transient loss of consciousness associated with collapse, the commonest cause of which is syncope.
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33

Beal, Jules C., Monika Eisermann, Sunita Misra, Phillip L. Pearl, Perrine Plouin, Eli M. Mizrahi, and Solomon L. Moshe. Seizures and Epilepsy in Preterm and Term Neonates, Infants, Children, and Adolescents. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0018.

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Children are often affected by seizure types and epilepsy syndromes that are specific to their age group and distinct from those seen in adults. At the same time, certain epilepsy syndromes affecting the adult population, such as Lennox–Gastaut syndrome and juvenile myoclonic epilepsy, often begin during childhood, as do seizures related to genetic abnormalities. The use of electroencephalography (EEG) and prolonged EEG monitoring has allowed for further insight and greater specificity in identifying and understanding seizures and epilepsy syndromes in children. This chapter reviews the role of EEG in pediatric seizures and the pediatric epilepsies, including electrographic findings in the ictal state and in the interictal period, as well as the correlation with clinical seizure semiology as it contributes to the diagnosis of epileptic phenomena. The chapter discusses EEG patterns, seizure types, and epilepsy syndromes specific to neonates, infants, children, and adolescents.
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34

Drislane, Frank W., Susan T. Herman, and Peter W. Kaplan. Convulsive Status Epilepticus. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0020.

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Generalized convulsive status epilepticus (GCSE) is a serious neurologic illness causing unresponsiveness, major physiologic disturbances, risk of injury and, if prolonged enough, neuronal damage. Causes are many, and the outcome often depends as much on the etiology as on the epileptic seizure itself. Several anti-seizure medications are used in treatment of GCSE, but some cases continue electrographically when clinical convulsions cease (nonconvulsive SE), and EEG is essential in their diagnosis. About 20% of cases become refractory to initial treatment, and the EEG becomes even more crucial in diagnosis and management. This chapter also covers other forms of SE with significant motor manifestations including: focal motor status (including epilepsia partialis continua); myoclonic status, which includes some relatively benign forms as well as some with a very poor prognosis; and clonic and tonic status. It reviews the many different EEG findings in those forms of status, and the use of EEG in their treatment and management, especially in prolonged cases.
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35

Lynch, Tara A., and J. Christopher Glantz. Seizure Medications Effects on Fetus, Neonate, and Lactation. Edited by Emma Ciafaloni, Cheryl Bushnell, and Loralei L. Thornburg. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190667351.003.0021.

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Medication use in pregnancy requires a careful balance between the risks of fetal teratogenicity and the maternal benefits of disease treatment. For women with epilepsy, there are many antiepileptic medications available for use in pregnancy. Each varies in their safety profile, risk for fetal anomalies, and effectiveness of seizure control. In most scenarios, the benefits of maternal treatment outweigh the risk of fetal effects, especially in cases of refractory epilepsy or severe disease. Many of the newer anti-epileptic drugs appear to have less teratogenic risk than the older medications. The ideal AED is one that is effective from the woman, is least teratogenic, and used at the lowest possible dose. Overall, a detailed understanding of antiepileptic efficacy, the pharmacologic differences in pregnancy, and the potential adverse fetal effects are required for optimal treatment of pregnant patients with epilepsy.
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36

Kalitzin, Stiliyan, and Fernando Lopes da Silva. EEG-Based Anticipation and Control of Seizures. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0023.

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Early seizure-prediction paradigms were based on detecting electroencephalographic (EEG) features, but recent approaches are based on dynamic systems theory. Methods that attempted to detect predictive features during the preictal period proved difficult to validate in practice. Brain systems can display bistability (both normal and epileptic states can coexist), and the transitions between states may be initiated by external or internal dynamic factors. In the former case prediction is impossible, but in the latter case prediction is conceivable, leading to the hypothesis that as seizure onset approaches, the excitability of the underlying neuronal networks tends to increase. This assumption is being explored using not only the ongoing EEG but also active probes, applying appropriate stimuli to brain areas to estimate the excitability of the neuronal populations. Experimental results support this assumption, suggesting that it may be possible to develop paradigms to estimate the risk of an impending transition to an epileptic state.
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37

Aggarwala, Neha, and Pirjo H. Manninen. Seizures and Status Epilepticus. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0065.

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Seizures are common with 5%–10% of the general population experiencing a seizure in their lifetime and 1%–3% having a seizure disorder. Patients undergoing neurosurgical procedures are at an increased risk of perioperative seizures, including seizures occurring during surgery. To prevent and minimize perioperative seizures, the preoperative optimization of patients with seizures is crucial, including administration of regular antiepileptic agents. Status epilepticus is a true neurological emergency, with early and aggressive treatment being imperative for successful management. This chapter reviews the diagnosis, risk factors for seizures, and the initial treatment of an acute perioperative seizure as well as the management of status epilepticus.
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38

Snars, Jeff, and Neil Buchanan. Pseudo-epileptic Seizures. MacLennan & Petty Pty.Ltd ,Australia, 1995.

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39

Luders Epileptic Seizures. WB Saunders, 2000.

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40

Chakravarty, Ambar. MIMICS of Epileptic Seizures. Jaypee Brothers Medical Publishers, 2020.

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41

Panayiotopoulos, C. P. Imitators of epileptic seizures. Springer, 2012.

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42

Panayiotopoulos, C. P. Imitators of epileptic seizures. Springer, 2012.

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43

Varsavsky, Andrea, Iven Mareels, and Mark Cook. Epileptic Seizures and the EEG. CRC Press, 2016. http://dx.doi.org/10.1201/b10459.

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44

Cook, Mark, Iven Mareels, and Andrea Varsavsky. Epileptic Seizures and the Eeg. Taylor & Francis Group, 2010.

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45

Avanzini, G., A. Beaumanoir, and A. Beaumanoir. Falls in Epileptic and Non-epileptic Seizures During Childhood. John Libbey Eurotext Limited, 1997.

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46

Nita, Dragos A., Miguel A. Cortez, Jose Luis Perez Velazquez, and O. Carter Snead. Biological Bases of Symptomatic Generalized Epilepsies in Children. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0040.

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Symptomatic generalized epilepsies represent a group of challenging epilepsy syndromes, most often seen in children, which share the hallmark of a triad encompassing multiple seizure types, electroencephalographical (EEG) evidence of diffuse brain involvement, and dysfunction in the intellectual domain (global developmental delay or mental retardation). SGEs include the early myoclonic encephalopathy, early infantile epileptic encephalopathy (Ohtahara syndrome), West syndrome, epilepsy with myoclonic-astatic seizures, epilepsy with myoclonic absence, Lennox-Gastaut syndrome, and the progressive myoclonic epilepsies. SGEs may arise from various genetic, developmental, or acquired brain pathologies and also can be associated with other cerebral or systemic defects and thus being part of a broader epilepsy syndrome phenotype. SGEs are associated with significant mortality and morbidity and most patients with SGE grow up to have intractable epilepsy, mental retardation, and depend on parents and institutions for the activities of the daily living. The mechanisms of SGE are numerous and heterogeneous and the EEG findings usually reflect the age-related changes as the brain matures.
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47

Walker, Matthew C. Convulsive and non-convulsive status epilepticus. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0030.

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This chapter describes the definition, epidemiology, classification, diagnosis, and treatment of status epilepticus, concentrating on the roles that electroencephalography (EEG) plays. The term status epilepticus now encompasses a range of conditions from continuous convulsive seizures to clinically subtle non-convulsive seizures, which may manifest as changes in behaviour or personality. EEG is critical for the diagnosis of non-convulsive status epilepticus. Furthermore, the progression of convulsive status epilepticus is to an electromechanical dissociation in which continuous electrical seizure activity may have no or minimal clinical manifestations. In the later stages of status epilepticus, EEG is necessary to monitor treatment, but is confounded by the interpretation of periodic EEG patterns, which represent a continuum from interictal through to ictal activity. Post-status epilepticus EEG patterns have prognostic value: periodic epileptiform discharges, burst suppression patterns (off anaesthesia) and repetitive seizure activity are indicative of a poor long-term prognosis.
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48

Seizure Free. Leanne Chilton, 1999.

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49

Atlas of Epileptic Seizures and Syndromes. W.B. Saunders Company, 2001.

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50

Panayiotopoulos, C. P. Reflex seizures and related epileptic syndromes. Springer, 2012.

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