Relevant bibliographies by topics / Epiphyseal cartilage

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  2. Dissertations / Theses
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Academic literature on the topic 'Epiphyseal cartilage'

Author: Grafiati
Published: 28 May 2022
Last updated: 31 July 2025

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Journal articles on the topic "Epiphyseal cartilage"

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Klinger, P., S. Lukassen, F. Ferrazzi, et al. "PEDF Is Associated with the Termination of Chondrocyte Phenotype and Catabolism of Cartilage Tissue." BioMed Research International 2017 (2017): 1–13. http://dx.doi.org/10.1155/2017/7183516.

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Objective.To investigate the expression and target genes of pigment epithelium-derived factor (PEDF) in cartilage and chondrocytes, respectively.Methods.We analyzed the expression pattern of PEDF in different human cartilaginous tissues including articular cartilage, osteophytic cartilage, and fetal epiphyseal and growth plate cartilage, by immunohistochemistry and quantitative real-time (qRT) PCR. Transcriptome analysis after stimulation of human articular chondrocytes with rhPEDF was performed by RNA sequencing (RNA-Seq) and confirmed by qRT-PCR.Results.Immunohistochemically, PEDF could be d
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Carlson, C. S., H. D. Hilley, and D. J. Meuten. "Degeneration of Cartilage Canal Vessels Associated With Lesions of Osteochondrosis in Swine." Veterinary Pathology 26, no. 1 (1989): 47–54. http://dx.doi.org/10.1177/030098588902600108.

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Articular-epiphyseal cartilage complexes from the distal femur and humerus of five 3-month-old pigs were collected and preserved using either a conventional or a ruthenium hexammine trichloride (RHT)-supplemented fixation technique. Lesions were similar regardless of the fixation technique. Areas of necrosis were in epiphyseal but not articular cartilage from both sites of all pigs. Cartilage canals were confined to epiphyseal cartilage and contained vessels which had endothelial cells in varying stages of degeneration and necrosis. Areas of necrotic cartilage often were adjacent to or surroun
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Amizuka, N., H. Warshawsky, J. E. Henderson, D. Goltzman, and A. C. Karaplis. "Parathyroid hormone-related peptide-depleted mice show abnormal epiphyseal cartilage development and altered endochondral bone formation." Journal of Cell Biology 126, no. 6 (1994): 1611–23. http://dx.doi.org/10.1083/jcb.126.6.1611.

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To elucidate the role of PTHrP in skeletal development, we examined the proximal tibial epiphysis and metaphysis of wild-type (PTHrP-normal) 18-19-d-old fetal mice and of chondrodystrophic litter mates homozygous for a disrupted PTHrP allele generated via homologous recombination in embryonic stem cells (PTHrP-depleted). In the PTHrP-normal epiphysis, immunocytochemistry showed PTHrP to be localized in chondrocytes within the resting zone and at the junction between proliferative and hypertrophic zones. In PTHrP-depleted epiphyses, a diminished [3H]thymidine-labeling index was observed in the
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Kincaid, S. A., R. V. Allhands, and G. J. Pijanowski. "Chondrolysis associated with cartilage canals of the epiphyseal cartilage of the distal humerus of growing pigs." American Journal of Veterinary Research 46, no. 3 (1985): 726–32. https://doi.org/10.2460/ajvr.1985.46.03.726.

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SUMMARY The articular-epiphyseal (a-e) cartilage of the distal humeri of 7 pigs weighing 13.1 to 18.2 kg and of 3 pigs weighing 36.4 to 40.9 kg was studied. Frozen samples of a-e cartilage were stained for the presence of reduced nicotinamide adenine dinucleotide dehydrogenase, lactate dehydrogenase, isocitrate dehydrogenase, and uridine diphosphate galactose-4-epimerase. Additional frozen sections and paraffin-processed sections were stained using the Alcian blue-critical electrolyte concentration method, safranin 0-fast green, and hematoxylin and eosin. An area of grossly visible, opaque a-e
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Setiawan, Arum, Mammed Sagi, Widya Asmara, and Istriyati Istriyati. "Analisis Pertumbuhan Kartilago Epifisialis Os Tibia Fetus Mencit (Mus musculus L.) Swiss Webster Setelah Induksi Ochratoxin A Selama Periode Organogenesis." JURNAL BIOLOGI PAPUA 4, no. 1 (2018): 25–31. http://dx.doi.org/10.31957/jbp.532.

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The aims of this study were determined the effects of Ochratoxin A (OTA) on growth of fetus tibia epiphyseal cartilage during organogenesis period. Twenty four pregnant mice were divided randomly into 4 groups of 6. Ochratoxin A was dissolved in sodium bicarbonateand administered orally on seventh to fourteenth days of gestation at dosage of 0.5, 1.0, 1.5 mg/kg bw, respectively. The remaining were used as control. The fetal tibia was taken after the 18 th day of pregnancy. The growth of tibia epiphyseal cartilages were observed histologically using Erlich’s Haematoxylin-Eosin Stain. The result
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Carlson, C. S., H. D. Hilley, and C. K. Henrikson. "Ultrastructure of normal epiphyseal cartilage of the articular-epiphyseal cartilage complex in growing swine." American Journal of Veterinary Research 46, no. 2 (1985): 306–13. https://doi.org/10.2460/ajvr.1985.46.02.306.

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SUMMARY Normal epiphyseal cartilage from the articular-epiphyseal cartilage complex (a-e complex) of the distal parts of the femur and humerus of growing commercial crossbred boars was collected, embedded in plastic, and studied by light and electron microscopies. The morphology of this cartilage was determined to provide a basis for comparison with cartilage affected with osteochondrosis, an important clinical disease in swine. Normal epiphyseal cartilage from the a-e complex in growing swine was divided into 4 major regions of cells; resting, proliferating, hypertrophic, and calcifying regio
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Ohtaka, Maiko, Izumi Hiramoto, Hiroshi Minagawa, Masashi Matsuzaki, and Hideya Kodama. "Screening of the Maturity Status of the Tibial Tuberosity by Ultrasonography in Higher Elementary School Grade Schoolchildren." International Journal of Environmental Research and Public Health 16, no. 12 (2019): 2138. http://dx.doi.org/10.3390/ijerph16122138.

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This study aimed to obtain screening data on the maturity status of the tibial tuberosity in schoolchildren of higher elementary school grades for risk management of Osgood–Schlatter disease (OSD). The maturity stages and cartilage thicknesses at the tibial tuberosity were determined by ultrasonography on the occasion of a school-based musculoskeletal examination for 124 grade 5–6 elementary schoolchildren, and their associations with the students’ demographic characteristics and OSD were examined. The time-dependent changes of the maturity status of the tibial tuberosity were also examined in
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Kambe, K., A. Yamamoto, T. Yoshimori, K. Hirayoshi, R. Ogawa, and Y. Tashiro. "Preferential localization of heat shock protein 47 in dilated endoplasmic reticulum of chicken chondrocytes." Journal of Histochemistry & Cytochemistry 42, no. 7 (1994): 833–41. http://dx.doi.org/10.1177/42.7.8014466.

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We investigated the distribution of heat shock protein 47 (hsp47) in cultured chicken embryonic chondrocytes and epiphyseal chondrocytes of tibial bones from 1-day-old to 6-week-old chickens. Northern blot and immunoblot analyses revealed that hsp47 exists in epiphyseal cartilage and cultured chondrocytes. Confocal laser immunofluorescence microscopy showed that hsp47 was localized mainly in the many granular structures found in the cytoplasm that contain Type II collagen. Epiphyseal cartilage and cultured chondrocytes were embedded in LR White resin and hsp47 was detected by protein A-immunog
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Sato, Shinsuke, Song Ho Chang, Taro Kasai, et al. "Juvenile Dysplasia Epiphysealis Hemimelica with Multiple Ankle Free Body." Case Reports in Orthopedics 2021 (April 9, 2021): 1–8. http://dx.doi.org/10.1155/2021/5579684.

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Dysplasia epiphysealis hemimelica (DEH), also known as Trevor’s disease, is a rare overgrowth of cartilage that commonly arises in the epiphyseal bone of children. We report a rare case of DEH originating from a talus accompanied by multiple intra-articular free bodies in a 7-year-old patient with ankle instability. After the primary surgery for free body removal and microfracture technique for the cartilage defects in the ankle joint, the free body recurred. Secondary surgery of arthroscopic free body removal with lateral ankle ligament repair succeeded in treating the patient, without furthe
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Moskalewski, S., and J. Malejczyk. "Bone formation following intrarenal transplantation of isolated murine chondrocytes: chondrocyte-bone cell transdifferentiation?" Development 107, no. 3 (1989): 473–80. http://dx.doi.org/10.1242/dev.107.3.473.

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Isolated syngeneic epiphyseal chondrocytes transplanted into a muscle formed cartilage in which matrix resorption and endochondral ossification began at the end of the second week after transplantation. After 56 days cartilage was converted into an ossicle. In 7-day-old intrarenal transplants, epiphyseal chondrocytes formed nodules of cartilage. In 10-day-old transplants, islands of bone appeared. Slight resorption of cartilage was first noted in 14-day-old transplants of chondrocytes. After eight weeks, transplants contained mainly bone. Intramuscularly transplanted rib chondrocytes formed ca
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Dissertations / Theses on the topic "Epiphyseal cartilage"

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Johnson, Colleen Joy. "Transport of interleukin 1[beta] in calf epiphyseal cartilage." Thesis, Massachusetts Institute of Technology, 1995. http://hdl.handle.net/1721.1/39371.

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Thesis (M.S.)--Massachusetts Institute of Technology, Dept. of Electrical Engineering and Computer Science, 1995.<br>On t.p. "[beta]" appears as the lower-case Greek letter.<br>Includes bibliographical references (leaves 140-143).<br>by Colleen Joy Johnson.<br>M.S.
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Chong, Ka-Wing. "Cellular responses of murine epiphyseal cartilage to mechanical injury." Thesis, Imperial College London, 2012. http://hdl.handle.net/10044/1/9487.

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Our laboratory has defined a rapid cellular response in porcine articular cartilage to injury. To enable study of the injury response in genetically modified animals I have characterized a novel model of cartilage injury in the mouse, involving avulsion of femoral head epiphyseal cartilage from 4-6 week old animals, and maintaining the explanted tissue in culture. Avulsion injury (explantation) of murine cartilage rapidly activated mitogen activated protein kinases (MAPKs) and nuclear factor κB (NFκB), thus establishing a close similarity to porcine tissue. Gene expression (mRNA) was analysed
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Романюк, Анатолій Миколайович, Анатолий Николаевич Романюк, Anatolii Mykolaiovych Romaniuk, K. Romaniuk, and A. Nwaogy. "Influence of salts of heavy metals on the structure of epiphyseal cartilage." Thesis, Вид-во СумДУ, 2007. http://essuir.sumdu.edu.ua/handle/123456789/5526.

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Among the known unfavorable influences on the skeletal bone, there is reaction on epiphyseal cartilage of bones under the influence of salts of heavy metals in the organism. To study the morphological changes of epiphyseal cartilage of skeletal bones using salts of heavy metals (chrome, zinc and lead). When you are citing the document, use the following link http://essuir.sumdu.edu.ua/handle/123456789/5526
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Gray, Martha Lane. "Physical regulation of epiphyseal cartilage biosynthesis : responses to electrical, mechanical, and chemical signals." Thesis, Massachusetts Institute of Technology, 1986. http://hdl.handle.net/1721.1/17205.

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Thesis (Ph. D.)--Massachusetts Institute of Technology, Harvard-MIT Division of Health Sciences and Technology Program in Medical Engineering and Medical Physics, 1986.<br>MICROFICHE COPY AVAILABLE IN ARCHIVES AND SCIENCE.<br>Bibliography: leaves 194-205.<br>by Martha Lane Gray.<br>Ph.D.
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Jakkula, E. (Eveliina). "Defects in the genes coding for cartilage extracellular matrix proteins as a cause of osteoarthritis and multiple epiphyseal dysplasia." Doctoral thesis, University of Oulu, 2005. http://urn.fi/urn:isbn:9514277333.

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Abstract The role of sequence variations in genes encoding cartilage extracellular matrix (ECM) proteins were studied in osteoarthritis (OA) and multiple epiphyseal dysplasia (MED). The cartilage collagen genes COL2A1, COL9A1, COL9A2, COL9A3, COL11A1, and COL11A2 were screened for sequence variations in 72 Finnish probands and one US family with primary early-onset hip and/or knee OA. Altogether 239 sequence variations were found, of which 16 were not present in the controls. Seven of the unique variations — four in COL11A1, two in COL11A2, and one in COL2A1 — were studied further, because the
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Ekman, Stina. "The normal and osteochondrotic porcine articular-epiphyseal cartilage complex : studies on cellular and matrix components /." Uppsala : Sveriges lantbruksuniv, 1990. http://epsilon.slu.se/avh/1990/91-576-4316-4.gif.

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Paassilta, P. (Petteri). "The human COL9A3 gene:structure of the gene for the α3 chain of type IX collagen and its role in human cartilage and intervertebral disc diseases". Doctoral thesis, University of Oulu, 1999. http://urn.fi/urn:isbn:9514254562.

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Abstract The nucleotide sequence of the entire COL9A3 gene, coding for the human α3(IX) chain, was determined. The gene was approximately 23 kb in length and consisted of 32 exons. The polymerase chain reaction (PCR)-based procedure of conformation-sensitive gel electrophoresis (CSGE) was used to screen the gene for sequence variations and mutations in 83 unrelated patients with generalized primary osteoarthritis (OA), 31 with rheumatoid arthritis (RA), 171 with intervertebral disc disease (IDD), and 50 with various osteochondrodysplasias. The frequencies of certain sequence variations in heal
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Rutt, Julianne Eileen. "Molecular Analysis Of The Epiphyseal Growth Plate In Rachitic Broilers: Evidence For The Etilogy Of The Condition." Columbus, Ohio : Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1223319403.

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Moraes, Vivian Vargas de. "Efeito da desnutrição protéica precocemente corrigida na morfologia da epífise de crescimento de coelhos (Nova Zelândia)." Universidade de São Paulo, 2006. http://www.teses.usp.br/teses/disponiveis/10/10132/tde-06082007-080608/.

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Diversos autores comprovam a influência de um episódio de desnutrição crônica no crescimento pôndero-estatural. Entretanto, não são definidas as repercussões da desnutrição protéica precocemente corrigida sobre o potencial genético de crescimento, uma vez que é difícil distinguir o papel da desnutrição do ambiente onde habitualmente ocorre. Deste modo, esta pesquisa teve como objetivo analisar os efeitos da desnutrição protéica precocemente corrigida na morfologia da epífise de crescimento. Assim, como modelos experimentais foram utilizados 13 coelhos adultos da raça Nova Zelândia Branco, send
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Edwards, Sarah. "Investigating the role of a novel ER molecular chaperone : Creld2 in the physiology and pathophysiology of endochondral bone growth." Thesis, University of Manchester, 2015. https://www.research.manchester.ac.uk/portal/en/theses/investigating-the-role-of-a-novel-er-molecular-chaperone-creld2-in-the-physiology-and-pathophysiology-of-endochondral-bone-growth(6fd49909-beec-42d1-a546-8b2411616e59).html.

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Cysteine rich with EGF-like domains 2 (Creld2) is a novel endoplasmic reticulum (ER) resident molecular chaperone that has been recently implicated in the ER stress signalling response (ERSS) and the unfolded protein response (UPR). Global transcriptomic data derived from in vivo mouse models of rare chondrodysplasias; Multiple Epiphyseal Dysplasia (MED Matn3 p.V194D) and Metaphyseal chondrodysplasia type Schmid (MCDS Col10a1 p.N617K), identified a significant upregulation in Creld2 expression in mutant chondrocytes. These chondrodysplasias share a common disease signature consisting of aberra
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Books on the topic "Epiphyseal cartilage"

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Ekman, Stina. Normal and osteochondrotic porcine articular-epiphyseal cartilage complex: Studies on cellular and matrix components. Sveriges Lantbruksuniversitet, 1990.

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Parker, James N., and Philip M. Parker. Recessive multiple epiphyseal dysplasia: A bibliography and dictionary for physicians, patients, and genome researchers [to Internet references]. ICON Health Publications, 2007.

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Book chapters on the topic "Epiphyseal cartilage"

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Donzelli, Onofrio, Leonardo Marchesini Reggiani, Manuele Lampasi, and Stefano Stilli. "Physeal and Epiphyseal Cartilage Injuries." In Pediatric and Adolescent Sports Traumatology. Springer Milan, 2013. http://dx.doi.org/10.1007/978-88-470-5412-7_5.

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Briggs, Michael D., Peter Bell, and Katarzyna A. Piróg. "Pseudoachondroplasia and Multiple Epiphyseal Dysplasia: Molecular Genetics, Disease Mechanisms and Therapeutic Targets." In Cartilage. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-45803-8_7.

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Dearden, Lyle C. "Glucocorticoid effects on the ultrastructure of epiphyseal cartilage." In Ultrastructure of Skeletal Tissues. Springer US, 1990. http://dx.doi.org/10.1007/978-1-4613-1487-5_8.

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Bonucci, Ermanno, and Benedetto Nicoletti. "Achondroplastic Mice: Morphological Investigations of Epiphyseal Cartilage and Bone." In Human Achondroplasia. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-8712-1_12.

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Engfeldt, Bengt, and Finn P. Reinholt. "Structure and Calcification of Epiphyseal Growth Cartilage." In Calcification in Biological Systems. CRC Press, 2020. http://dx.doi.org/10.1201/9781003068396-11.

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Hellemans, Jan, and Geert R. Mortier. "IHH and Acrocapitofemoral Dysplasia and Brachydactyly A1." In Inborn Errors Of Development. Oxford University PressNew York, NY, 2008. http://dx.doi.org/10.1093/oso/9780195306910.003.0022.

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Abstract The 7rst steps in skeletal morphogenesis involve the induction of undifferentiated mesenchyme and its differentiation into condensed mesenchyme. In the human skeleton, only the membranous bones of the skull and part of the clavicle are formed by direct ossi7cation of condensed mesenchyme (intramembranous ossi7cation). For the large majority of bones, bone formation and growth proceed through an intermediate cartilaginous template or anlage in a process called endochondral ossi7cation. These cartilaginous models are formed by further differentiation of condensed mesenchymal cells into
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Havránek, Petr. "INJURY TO GROWTH CARTILAGE (physeal and epiphyseal injuries, classification)." In Selected chapters in orthopedics and traumatology for medical students. Karolinum Press, Charles University, 2023. http://dx.doi.org/10.2307/jj.9669278.35.

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Rock, Matthew J., and Daniel H. Cohn. "COMP and Pseudoachondroplasia." In Inborn Errors Of Development. Oxford University PressNew York, NY, 2008. http://dx.doi.org/10.1093/oso/9780195306910.003.0167.

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Abstract Pseudoachondroplasia (PSACH) is a dominantly inherited form of short-limb short stature. Both mild and typical forms are recognized, comprising a spectrum of clinical severity. A definitive diagnosis can be made on the basis of radiographs showing delayed epiphyseal ossification, epiphyseal and metaphyseal irregularities, and a characteristic anterior beaking of the vertebral bodies apparent on lateral projection. In addition to short stature, loose joints due to ligamentous laxity, joint pain, varus and/or valgus deformities at the knees and scoliosis characterize the phenotype. The
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Clunie, Gavin. "Osteonecrosis, osteochondrosis, and osteochondritis dissecans." In Oxford Textbook of Medicine, edited by Cyrus Cooper. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0470.

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This chapter examines the bone conditions of osteonecrosis, osteochondrosis, and osteochondritis dissecans. Osteonecrosis is ischaemia of bone caused by a range of conditions, including trauma, which cause intravascular or extravascular obstruction of blood flow to bone. Many cases are associated with pro-thrombotic conditions. Osteonecrosis can be asymptomatic, but if progressive often leads to secondary (adjacent) joint destruction. Diagnosis is made by magnetic resonance imaging. Aside from treatment of any underlying cause, treatment options include analgesics, vasodilators, and surgery. T
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"IMMUNOREACTIVE l,25(OH)2D3, RECEPTORS AND CALBINDINS D-9k AND D-28k IN THE RAT EPIPHYSEAL CARTILAGE." In Vitamin D. De Gruyter, 1991. http://dx.doi.org/10.1515/9783110850345-195.

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Reports on the topic "Epiphyseal cartilage"

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Leach, Roland M., Mark Pines, Carol V. Gay, and Shmuel Hurwitz. In vivo and in vitro Chondrocyte Metabolism in Relationship to the Developemnt of Tibial Dyschondroplasia in Broiler Chickens. United States Department of Agriculture, 1993. http://dx.doi.org/10.32747/1993.7568090.bard.

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Skeletal deformities are a significant financial and welfare problem for the world poultry industry. Tibial dyschondroplasia (TD) is the most prevalent skeletal abnormality found in young broilers, turkeys and ducks. Tibial dyschondroplasia results from a perturbation of the sequence of events in the epiphyseal growth plate, the tissue responsible for longitudinal bone growth. The purpose of this investigation was to test the hypothesis that TD was the result of a failure of growth plate chondrocytes to differentiate and express the chemotactic molecules required for cartilage vascularization.
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