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Journal articles on the topic 'Escape mechanisms'

Author: Grafiati

Published: 4 June 2021

Last updated: 6 February 2022

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1

&NA;. "Mechanisms of Escape." Journal of Immunotherapy 26, no. 6 (2003): S43—S45. http://dx.doi.org/10.1097/00002371-200311000-00015.

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2

Wildes, Tyler, Kyle Dyson, Connor Francis, et al. "IMMU-13. MECHANISMS OF IMMUNOLOGICAL ESCAPE DURING ADOPTIVE CELLULAR THERAPY IN HIGH GRADE GLIOMA." Neuro-Oncology 21, Supplement_6 (2019): vi121—vi122. http://dx.doi.org/10.1093/neuonc/noz175.507.

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Abstract INTRODUCTION Immunotherapy is remarkably effective, yet tumor escape is common. Herein, we investigated tumor escape after adoptive cellular therapy (ACT) in intractable glioma models. These studies revealed multiple mechanisms of escape including a shift in immunogenic tumor antigens, downregulation of MHC-I, and upregulation of checkpoint molecules. Despite these changes, we HYPOTHESIZED that a new population of escape variant-specific polyclonal T cells could be generated to target immune-escaped tumors through using tumor escape variant RNA. METHODS We studied KR158B-luc glioma-be

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3

Oudejans, J. J. "Immune escape mechanisms in ALCL." Journal of Clinical Pathology 56, >6 (2003): 423–25. http://dx.doi.org/10.1136/jcp.56.6.423.

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4

Moore, T. E., and G. V. Khazanov. "Mechanisms of ionospheric mass escape." Journal of Geophysical Research: Space Physics 115, A12 (2010): n/a. http://dx.doi.org/10.1029/2009ja014905.

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5

Chan, Wing C. "Immune escape mechanisms for TCRLBCL." Blood 137, no. 10 (2021): 1274–76. http://dx.doi.org/10.1182/blood.2020008766.

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6

Hara, Hiroyuki. "Mechanisms of Immune Escape in Cancer." Journal of Nihon University Medical Association 75, no. 4 (2016): 152–55. http://dx.doi.org/10.4264/numa.75.4_152.

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Capone, Emily, Pramudita R. Prasetyanti, and Gianluca Sala. "HER-3: hub for escape mechanisms." Aging 7, no. 11 (2015): 899–900. http://dx.doi.org/10.18632/aging.100850.

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Niederkorn, Jerry Y. "Immune escape mechanisms of intraocular tumors." Progress in Retinal and Eye Research 28, no. 5 (2009): 329–47. http://dx.doi.org/10.1016/j.preteyeres.2009.06.002.

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9

Bröcker, E. B., W. Dummer, and J. C. Becker. "Immune escape mechanisms in human melanoma." Melanoma Research 4, no. 2 (1994): 25. http://dx.doi.org/10.1097/00008390-199409001-00039.

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10

Poppema, S., M. Potters, L. Visser, and A. M. van den Berg. "Immune escape mechanisms in Hodgkin’s disease." Annals of Oncology 9 (1998): s21—s24. http://dx.doi.org/10.1093/annonc/9.suppl_5.s21.

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UNGEFROREN, HENDRIK, MARTINA VOSS, WOLFRAM V. BERNSTORFF, ANDREAS SCHMID, BERND KREMER, and HOLGER KALTHOFF. "Immunological Escape Mechanisms in Pancreatic Carcinoma." Annals of the New York Academy of Sciences 880, no. 1 CELL AND MOLE (1999): 243–51. http://dx.doi.org/10.1111/j.1749-6632.1999.tb09529.x.

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12

Miller, Ashley M., and Pavel Pisa. "Tumor escape mechanisms in prostate cancer." Cancer Immunology, Immunotherapy 56, no. 1 (2005): 81–87. http://dx.doi.org/10.1007/s00262-005-0110-x.

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13

Frey, S. E., M. K. Gingras, and S. E. Dashtgard. "Experimental Studies of Gas-Escape and Water-Escape Structures: Mechanisms and Morphologies." Journal of Sedimentary Research 79, no. 11 (2009): 808–16. http://dx.doi.org/10.2110/jsr.2009.087.

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14

Wu, Haichao, Benjamin Greydanus, and Daniel K. Schwartz. "Mechanisms of transport enhancement for self-propelled nanoswimmers in a porous matrix." Proceedings of the National Academy of Sciences 118, no. 27 (2021): e2101807118. http://dx.doi.org/10.1073/pnas.2101807118.

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Micro/nanoswimmers convert diverse energy sources into directional movement, demonstrating significant promise for biomedical and environmental applications, many of which involve complex, tortuous, or crowded environments. Here, we investigated the transport behavior of self-propelled catalytic Janus particles in a complex interconnected porous void space, where the rate-determining step involves the escape from a cavity and translocation through holes to adjacent cavities. Surprisingly, self-propelled nanoswimmers escaped from cavities more than 20× faster than passive (Brownian) particles,

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Zhai, Weijie, Fengjuan Wu, Yiyuan Zhang, Yurong Fu, and Zhijun Liu. "The Immune Escape Mechanisms of Mycobacterium Tuberculosis." International Journal of Molecular Sciences 20, no. 2 (2019): 340. http://dx.doi.org/10.3390/ijms20020340.

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Epidemiological data from the Center of Disease Control (CDC) and the World Health Organization (WHO) statistics in 2017 show that 10.0 million people around the world became sick with tuberculosis. Mycobacterium tuberculosis (MTB) is an intracellular parasite that mainly attacks macrophages and inhibits their apoptosis. It can become a long-term infection in humans, causing a series of pathological changes and clinical manifestations. In this review, we summarize innate immunity including the inhibition of antioxidants, the maturation and acidification of phagolysosomes and especially the apo

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16

Muller, Ludmila, Rolf Kiessling, Robert C. Rees, and Graham Pawelec. "Escape Mechanisms in Tumor Immunity: An Update." Journal of Environmental Pathology, Toxicology and Oncology 21, no. 4 (2002): 54. http://dx.doi.org/10.1615/jenvironpatholtoxicoloncol.v21.i4.10.

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17

Santer, Frédéric R., Holger H. H. Erb, and Rhiannon V. McNeill. "Therapy escape mechanisms in the malignant prostate." Seminars in Cancer Biology 35 (December 2015): 133–44. http://dx.doi.org/10.1016/j.semcancer.2015.08.005.

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18

Roberts, B. L. "Neural mechanisms underlying escape behaviour in fishes." Reviews in Fish Biology and Fisheries 2, no. 3 (1992): 243–66. http://dx.doi.org/10.1007/bf00045039.

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19

Lucas, Michaela, URS Karrer, Andrew Lucas, and Paul Klenerman. "Viral escape mechanisms - escapology taught by viruses." International Journal of Experimental Pathology 82, no. 5 (2008): 269–86. http://dx.doi.org/10.1046/j.1365-2613.2001.00204.x.

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20

ROSENBERG, W. "Mechanisms of immune escape in viral hepatitis." Gut 44, no. 5 (1999): 759–64. http://dx.doi.org/10.1136/gut.44.5.759.

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21

Smith, Leanne M., and Robin C. May. "Mechanisms of microbial escape from phagocyte killing." Biochemical Society Transactions 41, no. 2 (2013): 475–90. http://dx.doi.org/10.1042/bst20130014.

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Phagocytosis and phagosome maturation are crucial processes in biology. Phagocytosis and the subsequent digestion of phagocytosed particles occur across a huge diversity of eukaryotes and can be achieved by many different cells within one organism. In parallel, diverse groups of pathogens have evolved mechanisms to avoid killing by phagocytic cells. The present review discusses a key innate immune cell, the macrophage, and highlights the myriad mechanisms microbes have established to escape phagocytic killing.

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22

Lisiecka, Urszula, and Krzysztof Kostro. "Mechanisms of tumour escape from immune surveillance." Journal of Veterinary Research 60, no. 4 (2016): 453–60. http://dx.doi.org/10.1515/jvetres-2016-0068.

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Abstract The progressive growth and spread of tumour cells in the form of metastases requires an interaction of healthy host cells, such as endothelial cells, fibroblasts, and other cells of mesenchymal origin with immune cells taking part in innate and adaptive responses within the tumour lesion and entire body. The host cells interact with tumour cells to create a dynamic tumour microenvironment, in which healthy cells can both positively and negatively influence the growth and spread of the tumour. The balance of cellular homeostasis and the effect of substances they secrete on the tumour m

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23

Seliger, Barbara. "Immune Escape Mechanisms of Renal Cell Carcinoma." European Urology Supplements 6, no. 10 (2007): 616–22. http://dx.doi.org/10.1016/j.eursup.2007.03.009.

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24

Joshua, D. E., J. Gibson, and R. D. Brown. "Mechanisms of the escape phase of myeloma." Blood Reviews 8, no. 1 (1994): 13–20. http://dx.doi.org/10.1016/0268-960x(94)90003-5.

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25

Winger, Paul D., and Philip J. Walsh. "The feasibility of escape mechanisms in conical snow crab traps." ICES Journal of Marine Science 64, no. 8 (2007): 1587–91. http://dx.doi.org/10.1093/icesjms/fsm125.

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Abstract Winger, P. D., and Walsh, P. J. 2007. The feasibility of escape mechanisms in conical snow crab traps. – ICES Journal of Marine Science, 64: 1587–1591. Laboratory observations and morphometric measurements of snow crab (Chionoecetes opilio) were conducted to examine the feasibility of incorporating rigid escape mechanisms into conical snow crab traps to improve trap selectivity. Under laboratory conditions, undersized adolescent male snow crab (≤94 mm carapace width) were capable of detecting, approaching, and interacting with escape mechanisms, and the location of the mechanisms was

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26

Qian, Jin. "Immune escape mechanism of B-cell malignancies on Anti-CD19 Chimeric Antigen Receptor T-cell treatment and solution." E3S Web of Conferences 271 (2021): 03038. http://dx.doi.org/10.1051/e3sconf/202127103038.

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Relapse or refractory B-cell malignancies have been reported in multiple clinical trials after treatment of Anti-CD19 Chimeric Antigen Receptor (CAR) T-cells. Many clinical studies have demonstrated the potential immune escape mechanism for B-cell malignancies like genetic mutation, transcriptional deregulation, lineage switch, loss of CAR T-cells, and trogocytosis. The study of these mechanisms can provide us insights in designs of future immunotherapies regarding both B-cell malignancies and even other solid tumors. The potential solution for the immune escape mechanisms regarding CAR T-cell

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27

Páez, Rocío I., and Christos Efthymiopoulos. "Modeling Trojan dynamics: diffusion mechanisms through resonances." Proceedings of the International Astronomical Union 9, S310 (2014): 96–97. http://dx.doi.org/10.1017/s1743921314007959.

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AbstractIn the framework of the ERTBP, we study an example of the influence of secondary resonances over the long term stability of Trojan motions. By the integration of ensembles of orbits, we find various types of chaotic diffusion, slow and fast. We show that the distribution of escape times is bi-modular, corresponding to two populations of short and long escape times. The objects with long escape times produce a power-law tail in the distribution.

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28

O. Croci, D., and M. Salatino. "Tumor Immune Escape Mechanisms that Operate During Metastasis." Current Pharmaceutical Biotechnology 12, no. 11 (2011): 1923–36. http://dx.doi.org/10.2174/138920111798376987.

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29

Scarsbrook, J. R., G. A. McFarlane, and W. Shaw. "Effectiveness of Experimental Escape Mechanisms in Sablefish Traps." North American Journal of Fisheries Management 8, no. 2 (1988): 158–61. http://dx.doi.org/10.1577/1548-8675(1988)008<0158:eoeemi>2.3.co;2.

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30

Zimmermann-Klemd, Amy Marisa, Seema Devi, Carmen Steinborn, et al. "Does mistletoe interact with tumor immune-escape mechanisms?" Phytomedicine 61 (2019): 5. http://dx.doi.org/10.1016/j.phymed.2019.09.125.

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31

Barsoum, Ivraym B., Madhuri Koti, D. Robert Siemens, and Charles H. Graham. "Mechanisms of Hypoxia-Mediated Immune Escape in Cancer." Cancer Research 74, no. 24 (2014): 7185–90. http://dx.doi.org/10.1158/0008-5472.can-14-2598.

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32

Khaliq and Fallahi-Sichani. "Epigenetic Mechanisms of Escape from BRAF Oncogene Dependency." Cancers 11, no. 10 (2019): 1480. http://dx.doi.org/10.3390/cancers11101480.

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About eight percent of all human tumors (including 50% of melanomas) carry gain-of-function mutations in the BRAF oncogene. Mutated BRAF and subsequent hyperactivation of the MAPK signaling pathway has motivated the use of MAPK-targeted therapies for these tumors. Despite great promise, however, MAPK-targeted therapies in BRAF-mutant tumors are limited by the emergence of drug resistance. Mechanisms of resistance include genetic, non-genetic and epigenetic alterations. Epigenetic plasticity, often modulated by histone-modifying enzymes and gene regulation, can influence a tumor cell’s BRAF dep

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33

Sapoznik, Sivan, Ohad Hammer, Rona Ortenberg, et al. "Novel Anti-Melanoma Immunotherapies: Disarming Tumor Escape Mechanisms." Clinical and Developmental Immunology 2012 (2012): 1–9. http://dx.doi.org/10.1155/2012/818214.

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The immune system fights cancer and sometimes temporarily eliminates it or reaches an equilibrium stage of tumor growth. However, continuous immunological pressure also selects poorly immunogenic tumor variants that eventually escape the immune control system. Here, we focus on metastatic melanoma, a highly immunogenic tumor, and on anti-melanoma immunotherapies, which recently, especially following the FDA approval of Ipilimumab, gained interest from drug development companies. We describe new immunomodulatory approaches currently in the development pipeline, focus on the novel CEACAM1 immune

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34

Cohen, Tara N., Andrew C. Griggs, Falisha F. Kanji, et al. "Advancing team cohesion: Using an escape room as a novel approach." Journal of Patient Safety and Risk Management 26, no. 3 (2021): 126–34. http://dx.doi.org/10.1177/25160435211005934.

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Objective An escape room was used to study teamwork and its determinants, which have been found to relate to the quality and safety of patient care delivery. This pilot study aimed to explore the value of an escape room as a mechanism for improving cohesion among interdisciplinary healthcare teams. Methods This research was conducted at a nonprofit medical center in Southern California. All participants who work on a team were invited to participate. Authors employed an interrupted within-subjects design, with two pre- and post-escape room questionnaires related to two facets of group cohesion

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35

Procházka, V., M. Jarošová, Z. Prouzová, R. Nedomová, T. Papajík, and K. Indrák. "Immune Escape Mechanisms in Diffuse Large B-Cell Lymphoma." ISRN Immunology 2012 (December 23, 2012): 1–6. http://dx.doi.org/10.5402/2012/208903.

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Diffuse large B-cell lymphoma (DLBCL) is the most frequent subtype of non-Hodgkin lymphomas in Western countries. Implementation of immunotherapy using monoclonal antibodies to therapeutic protocols has led to dramatic improvements in overall survival. DLBCL became a model of a successful immunochemotherapy concept. Despite this fact, there is still a proportion of patients who do not respond to or relapse early after treatment. Growing evidence suggests that host antitumor immunity is suppressed by lymphoma cells in many ways. First, host cytotoxic T cells are directly suppressed by interacti

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Setiadi, A. Francesca, Muriel D. David, Robyn P. Seipp, Jennifer A. Hartikainen, Rayshad Gopaul, and Wilfred A. Jefferies. "Epigenetic Control of the Immune Escape Mechanisms in Malignant Carcinomas." Molecular and Cellular Biology 27, no. 22 (2007): 7886–94. http://dx.doi.org/10.1128/mcb.01547-07.

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ABSTRACT Downregulation of the transporter associated with antigen processing 1 (TAP-1) has been observed in many tumors and is closely associated with tumor immunoevasion mechanisms, growth, and metastatic ability. The molecular mechanisms underlying the relatively low level of transcription of the tap-1 gene in cancer cells are largely unexplained. In this study, we tested the hypothesis that epigenetic regulation plays a fundamental role in controlling tumor antigen processing and immune escape mechanisms. We found that the lack of TAP-1 transcription in TAP-deficient cells correlated with

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37

Zeiser, Robert, and Luca Vago. "Mechanisms of immune escape after allogeneic hematopoietic cell transplantation." Blood 133, no. 12 (2019): 1290–97. http://dx.doi.org/10.1182/blood-2018-10-846824.

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Abstract Relapse of the original disease is a major cause of death after allogeneic hematopoietic cell transplantation for acute leukemias. There is growing evidence that relapses may be explained not only by resistance to chemotherapy but also by the escape of tumor cells from the control of the allogeneic immune response. Mechanisms of immune evasion can involve abrogation of leukemia cell recognition due to loss of HLA genes, immunosuppression by immune-checkpoint ligand expression, production of anti-inflammatory factors, release of metabolically active enzymes, loss of proinflammatory cyt

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38

Weymouth, G. D., and M. S. Triantafyllou. "Ultra-fast escape of a deformable jet-propelled body." Journal of Fluid Mechanics 721 (March 13, 2013): 367–85. http://dx.doi.org/10.1017/jfm.2013.65.

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AbstractIn this work a cephalopod-like deformable body that fills an internal cavity with fluid and expels it to propel an escape manoeuvre, while undergoing a drastic external shape change through shrinking, is shown to employ viscous as well as mainly inviscid hydrodynamic mechanisms to power an impressively fast start. First, we show that recovery of added-mass energy enables a shrinking rocket in a dense inviscid flow to achieve greater escape speed than an identical rocket in a vacuum. Next, we extend the shrinking body results of Weymouth & Triantafyllou (J. Fluid Mech., vol. 702, 20

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39

Velázquez-Moctezuma, Rodrigo, Elias H. Augestad, Matteo Castelli, et al. "Mechanisms of Hepatitis C Virus Escape from Vaccine-Relevant Neutralizing Antibodies." Vaccines 9, no. 3 (2021): 291. http://dx.doi.org/10.3390/vaccines9030291.

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Hepatitis C virus (HCV) is a major causative agent of acute and chronic hepatitis. It is estimated that 400,000 people die every year from chronic HCV infection, mostly from severe liver-related diseases such as cirrhosis and liver cancer. Although HCV was discovered more than 30 years ago, an efficient prophylactic vaccine is still missing. The HCV glycoprotein complex, E1/E2, is the principal target of neutralizing antibodies (NAbs) and, thus, is an attractive antigen for B-cell vaccine design. However, the high genetic variability of the virus necessitates the identification of conserved ep

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40

Anichini, Andrea, Valentina E. Perotti, Francesco Sgambelluri, and Roberta Mortarini. "Immune Escape Mechanisms in Non Small Cell Lung Cancer." Cancers 12, no. 12 (2020): 3605. http://dx.doi.org/10.3390/cancers12123605.

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Development of strong immune evasion has been traditionally associated with the late stages of solid tumor progression, since advanced cancers are more likely to have reached the third phase of the immunoediting process. However, by integrating a variety of approaches, evidence for active immune escape mechanisms has been found even in the pre-invasive lesions that later progress to the main NSCLC histotypes. Pre-invasive lesions of adenocarcinoma (LUAD) and of squamous cell carcinoma (LUSC) can show impaired antigen presentation, loss of heterozygosity at the Human Leukocyte Antigen (HLA) reg

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41

Sokolova, M. V., M. V. Konopleva, Т. A. Semenenko, V. G. Akimkin, A. V. Tutelyan, and A. P. Suslov. "THE MECHANISMS OF IMMUNE ESCAPE BY HEPATITIS B VIRUS." Annals of the Russian academy of medical sciences 72, no. 6 (2017): 408–19. http://dx.doi.org/10.15690/vramn866.

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The high prevalence of the hepatitis B virus (HBV) in population occurs mainly due to numerous mechanisms formed in the process of the virus evolution, contributing to its survival under immunological pressure. The review presents the most complete systematization and classification of various HBV protective mechanisms basing on their influence on different parts of congenital and adaptive immune response. The analysis of literature data allows for the conclusion that two basic principles underlie the mechanisms: the strategy of the «stealth virus» (virus’s escape from recognition by the immun

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42

Denham, Steven, and Jessica Brown. "Mechanisms of Pulmonary Escape and Dissemination by Cryptococcus neoformans." Journal of Fungi 4, no. 1 (2018): 25. http://dx.doi.org/10.3390/jof4010025.

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43

Wilczynski, Jacek R. "Cancer and Pregnancy Share Similar Mechanisms of Immunological Escape." Chemotherapy 52, no. 3 (2006): 107–10. http://dx.doi.org/10.1159/000092537.

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44

Beatty, Gregory L., and Whitney L. Gladney. "Immune Escape Mechanisms as a Guide for Cancer Immunotherapy." Clinical Cancer Research 21, no. 4 (2014): 687–92. http://dx.doi.org/10.1158/1078-0432.ccr-14-1860.

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45

Gow, Paul John, and David Mutimer. "Mechanisms of hepatitis B virus escape after immunoglobulin therapy." Current Opinion in Infectious Diseases 13, no. 6 (2000): 643–46. http://dx.doi.org/10.1097/00001432-200012000-00011.

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46

Restifo, Nicholas P., Yutaka Kawakami, Franco Marincola, et al. "Molecular Mechanisms Used by Tumors to Escape Immune Recognition." Journal of Immunotherapy 14, no. 3 (1993): 182–90. http://dx.doi.org/10.1097/00002371-199310000-00004.

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47

Friedl, Peter, and Katarina Wolf. "Tumour-cell invasion and migration: diversity and escape mechanisms." Nature Reviews Cancer 3, no. 5 (2003): 362–74. http://dx.doi.org/10.1038/nrc1075.

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48

Ridolfi, Laura, Massimiliano Petrini, Laura Fiammenghi, Angela Riccobon, and Ruggero Ridolfi. "Human embryo immune escape mechanisms rediscovered by the tumor." Immunobiology 214, no. 1 (2009): 61–76. http://dx.doi.org/10.1016/j.imbio.2008.03.003.

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49

Zeng, Xianmin. "Human Embryonic Stem Cells: Mechanisms to Escape Replicative Senescence?" Stem Cell Reviews 3, no. 4 (2007): 270–79. http://dx.doi.org/10.1007/s12015-007-9005-x.

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50

Tang, Sha, Qian Ning, Ling Yang, Zhongcheng Mo, and Shengsong Tang. "Mechanisms of immune escape in the cancer immune cycle." International Immunopharmacology 86 (September 2020): 106700. http://dx.doi.org/10.1016/j.intimp.2020.106700.

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