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Journal articles on the topic 'Escherichia coli. Pathology, Molecular'

Author: Grafiati

Published: 4 June 2021

Last updated: 11 February 2022

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1

Fleece, Molly E., Suporn Pholwat, Amy J. Mathers, and Eric R. Houpt. "Molecular diagnosis of antimicrobial resistance in Escherichia coli." Expert Review of Molecular Diagnostics 18, no. 3 (2018): 207–17. http://dx.doi.org/10.1080/14737159.2018.1439381.

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2

Vaishnavi, Chetana, Sukhminderjit Kaur, Lothar Beutin, and Ulrike Krueger. "Phenotypic and molecular characterization of clinically isolated Escherichia coli." Indian Journal of Pathology and Microbiology 53, no. 3 (2010): 509. http://dx.doi.org/10.4103/0377-4929.68298.

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3

Zahraei Salehi, Taghi, Mahdi Askari Badouei, and Iman Mehdizadeh Gohari. "Molecular detection and antibacterial susceptibility of enteropathogenic Escherichia coli (EPEC) and shigatoxigenic Escherichia coli (STEC) strains isolated from healthy and diarrhoeic dogs." Comparative Clinical Pathology 20, no. 6 (2010): 585–89. http://dx.doi.org/10.1007/s00580-010-1037-y.

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4

Rickert, C. H., C. August, M. Brandt, V. Wagner, and W. Paulus. "Cerebral malakoplakia associated with Escherichia coli infection." Acta Neuropathologica 99, no. 5 (2000): 595–98. http://dx.doi.org/10.1007/s004010051167.

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5

Wiles, Travis J., Richard R. Kulesus, and Matthew A. Mulvey. "Origins and virulence mechanisms of uropathogenic Escherichia coli." Experimental and Molecular Pathology 85, no. 1 (2008): 11–19. http://dx.doi.org/10.1016/j.yexmp.2008.03.007.

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6

Mundy, Rosanna, Francis Girard, Anthony J. FitzGerald, and Gad Frankel. "Comparison of colonization dynamics and pathology of mice infected with enteropathogenic Escherichia coli, enterohaemorrhagic E. coli and Citrobacter rodentium." FEMS Microbiology Letters 265, no. 1 (2006): 126–32. http://dx.doi.org/10.1111/j.1574-6968.2006.00481.x.

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7

Alexandre, Marcela, and Valeria Prado. "Detection of Shiga toxin-producing Escherichia coli in food." Expert Review of Molecular Diagnostics 3, no. 1 (2003): 105–15. http://dx.doi.org/10.1586/14737159.3.1.105.

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8

Saei, Habib Dastmalchi, Elham Ahmadi, Ali Kazemnia, and Mehrzad Ahmadinia. "Molecular identification and antibiotic susceptibility patterns of Escherichia coli isolates from sheep faeces samples." Comparative Clinical Pathology 21, no. 4 (2010): 467–73. http://dx.doi.org/10.1007/s00580-010-1134-y.

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9

Sasaki, Maiko, Shanti V. Sitaraman, Brian A. Babbin, et al. "Invasive Escherichia coli are a feature of Crohn's disease." Laboratory Investigation 87, no. 10 (2007): 1042–54. http://dx.doi.org/10.1038/labinvest.3700661.

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10

Makarova, Mariia A., Zoya N. Matveeva, E. V. Smirnova, et al. "LABORATORY ERRORS FOR IDENTIFICATION OF THE ESCHERICHIA COLI STRAINS OF THE SEROLOGICAL GROUPS O6 AND O25 AS ACUTE INTESTINAL INFECTIONS." Russian Clinical Laboratory Diagnostics 65, no. 6 (2020): 368–74. http://dx.doi.org/10.18821/0869-2084-2020-65-6-368-374.

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Were studied the genes encoding the virulence factors of 221 strains: E. coli O6:H1 (194) and E. coli O25:H4 (27), isolated in 2014-2018 from stool samples of children and adults examined according to epidemic indications. Molecular methods included PCR with hybridization-fluorescence and electrophoresis detection of amplified products. The strains did not have virulence genes for diarrheagenic E. coli (DEC) pathogroups EPEC, ETEC, EIEC, EHEC, EAggEC, and belonged to the phylogenetic group B2. They contained from four to eight genes encoding virulence factors of ExPEC: E. coli O6:H1 - pap (68,

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11

Baines, Danica, Byron Lee, and Tim McAllister. "Heterogeneity in enterohemorrhagicEscherichia coliO157:H7 fecal shedding in cattle is related toEscherichia coliO157:H7 colonization of the small and large intestine." Canadian Journal of Microbiology 54, no. 12 (2008): 984–95. http://dx.doi.org/10.1139/w08-090.

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In the last decade, Escherichia coli O157:H7 have emerged as important pathogens of the gastrointestinal tract of humans. Healthy cattle have been identified as the primary reservoir, however, the factors affecting heterogeneous E. coli O157:H7 fecal shedding are not fully understood. The aim of this study was to investigate the contribution of E. coli O157:H7 colonization of small and large intestinal sites to the heterogeneity of fecal shedding in cattle. There was a dose-dependant E. coli O157:H7 E318N colonization of duodenum, jejunum, ileum, cecum, ascending colon, spiral colon, descendin

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12

Sepehriseresht, Saeed, Taghi Zahraei Salehi, Morteza Sattari, Hasan Tadjbakhsh, and Mohammad Mehdi Aslani. "Detection of shigatoxigenic Escherichia coli from fecal samples of calves and cattle by molecular and serological methods." Comparative Clinical Pathology 18, no. 1 (2008): 53–57. http://dx.doi.org/10.1007/s00580-008-0755-x.

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13

Chui, Linda, Marc R. Couturier, Theodore Chiu, et al. "Comparison of Shiga Toxin-Producing Escherichia coli Detection Methods Using Clinical Stool Samples." Journal of Molecular Diagnostics 12, no. 4 (2010): 469–75. http://dx.doi.org/10.2353/jmoldx.2010.090221.

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14

Łoś, Marcin, Joanna M. Łoś, and Grzegorz Węgrzyn. "Rapid Identification of Shiga Toxin-producing Escherichia coli (STEC) Using Electric Biochips." Diagnostic Molecular Pathology 17, no. 3 (2008): 179–84. http://dx.doi.org/10.1097/pdm.0b013e31815a5127.

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15

McAuliffe, Gary N., Fifi Tse, Helen Qiao, et al. "Isolate independent molecular typing improves the yield of O typing of infections due to Shiga toxin producing Escherichia coli." Pathology 52, no. 4 (2020): 460–65. http://dx.doi.org/10.1016/j.pathol.2020.02.008.

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16

Ceri, H., S. Schmidt, M. E. Olson, J. C. Nickel, and H. Benediktsson. "Specific mucosal immunity in the pathophysiology of bacterial prostatitis in a rat model." Canadian Journal of Microbiology 45, no. 10 (1999): 849–55. http://dx.doi.org/10.1139/w99-088.

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Mucosal immunity was established in the rat prostate by stimulating the common mucosal system through serosal exposure of formalin-killed Escherichia coli. Immunized but not sham-immunized rats developed bacterial specific IgG and IgA in prostatic fluid, and IgA in urine. Immunized (n = 21) and sham-immunized control rats (n = 30) were challenged by transurethral injection of E. coli into the prostate ducts. Mortality, gross and microscopic pathology, tissue bacterial counts, bacterial associated immunoglobulins, and antibody titers in serum and urine were assessed at 7 days following the chal

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17

Kaur, C., H. F. Too, and E. A. Ling. "Phagocytosis of Escherichia coli by amoeboid microglial cells in the developing brain." Acta Neuropathologica 107, no. 3 (2004): 204–8. http://dx.doi.org/10.1007/s00401-003-0798-7.

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18

OLOOMI, MANA, and SAEID BOUZARI. "Molecular profile and genetic diversity of cytolethal distending toxin (CDT)-producing Escherichia coli isolates from diarrheal patients." APMIS 116, no. 2 (2008): 125–32. http://dx.doi.org/10.1111/j.1600-0463.2008.00910.x.

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19

Jiang, Chenggang, Tongqing An, Shujie Wang, et al. "Role of theehxAgene fromEscherichia coliserotype O82 in hemolysis, biofilm formation, and in vivo virulence." Canadian Journal of Microbiology 61, no. 5 (2015): 335–41. http://dx.doi.org/10.1139/cjm-2014-0824.

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Enterohemorrhagic Escherichia coli (EHEC) strains cause serious gastrointestinal disease, which can lead to potentially life-threatening systemic complications such as hemolytic uremic syndrome. Although the ehx gene is established as a major virulence factor of EHEC, the role of this gene in colonization and biofilm formation remains to be elucidated. We constructed recombinant isogenic mutants of the ehxA locus of E. coli HLJ1122 (serotype O82) using the λ Red homologous recombination system. Significantly higher levels of adherence to human epithelial cells (HEp-2) cells were observed for s

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20

Luperchio, Steven A., Joseph V. Newman, Charles A. Dangler, et al. "Citrobacter rodentium, the Causative Agent of Transmissible Murine Colonic Hyperplasia, Exhibits Clonality: Synonymy of C. rodentium and Mouse-PathogenicEscherichia coli." Journal of Clinical Microbiology 38, no. 12 (2000): 4343–50. http://dx.doi.org/10.1128/jcm.38.12.4343-4350.2000.

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Citrobacter rodentium (formerly Citrobacter freundii biotype 4280 and Citrobacter genomospecies 9) was described on the basis of biochemical characterization and DNA-DNA hybridization data and is the only Citrobacterspecies known to possess virulence factors homologous to those of the human pathogens enteropathogenic Escherichia coli and enterohemorrhagic E. coli. These virulence factors are encoded on the locus of enterocyte effacement (LEE), a pathogenicity island required for the characteristic attaching and effacing (AE) pathology seen in infection with these three pathogens. C. rodentium,

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21

Wu, Hsueh-Hsia, Yi-Yuan Yang, Wen-Shyang Hsieh, Chi-Hsin Lee, Sy-Jye C. Leu, and Mei-Ru Chen. "OmpA Is the Critical Component for Escherichia coli Invasion-Induced Astrocyte Activation." Journal of Neuropathology and Experimental Neurology 68, no. 6 (2009): 677–90. http://dx.doi.org/10.1097/nen.0b013e3181a77d1e.

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22

Bleich, Andre, John P. Sundberg, Anna Smoczek, et al. "Sensitivity to Escherichia coli Nissle 1917 in mice is dependent on environment and genetic background." International Journal of Experimental Pathology 89, no. 1 (2007): 45–54. http://dx.doi.org/10.1111/j.1365-2613.2007.00560.x.

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23

McPhee, Joseph B., Cherrie L. Small, Sarah A. Reid-Yu, John R. Brannon, Hervé Le Moual, and Brian K. Coombes. "Host Defense Peptide Resistance Contributes to Colonization and Maximal Intestinal Pathology by Crohn's Disease-Associated Adherent-Invasive Escherichia coli." Infection and Immunity 82, no. 8 (2014): 3383–93. http://dx.doi.org/10.1128/iai.01888-14.

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ABSTRACTHost defense peptides secreted by colonocytes and Paneth cells play a key role in innate host defenses in the gut. In Crohn's disease, the burden of tissue-associatedEscherichia colicommonly increases at epithelial surfaces where host defense peptides concentrate, suggesting that this bacterial population might actively resist this mechanism of bacterial killing. Adherent-invasiveE. coli(AIEC) is associated with Crohn's disease; however, the colonization determinants of AIEC in the inflamed gut are undefined. Here, we establish that host defense peptide resistance contributes to host c

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24

Matussek, Andreas, Joerg Lauber, Anna Bergau, et al. "Molecular and functional analysis of Shiga toxin–induced response patterns in human vascular endothelial cells." Blood 102, no. 4 (2003): 1323–32. http://dx.doi.org/10.1182/blood-2002-10-3301.

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Abstract Enterohemorrhagic Escherichia coli (EHEC) is the major cause of hemolyticuremic syndrome (HUS) characterized by microangiopathic hemolytic anemia, thrombocytopenia, and acute renal failure. EHEC produces one or more Shiga toxins (Stx1 and Stx2), and it was assumed that Stx's only relevant biologic activity was cell destruction through inhibition of protein synthesis. However, recent data indicate that in vivo the cytokine milieu may determine whether endothelial cells survive or undergo apoptosis/necrosis when exposed to Stxs. In this study, we analyzed the genome-wide expression patt

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25

Zink, Albert, Udo Reischl, Hans Wolf, and Andreas G. Nerlich. "Molecular Evidence of Bacteremia by Gastrointestinal Pathogenic Bacteria in an Infant Mummy From Ancient Egypt." Archives of Pathology & Laboratory Medicine 124, no. 11 (2000): 1614–18. http://dx.doi.org/10.5858/2000-124-1614-meobbg.

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Abstract In this study, we describe an infant mummy from ancient Egypt that showed macromorphologic signs of chronic anemia and vitamin C deficiency. From this infant, we have obtained a sterile sample from a metatarsal bone to extract ancient bacterial DNA. Following polymerase chain reaction amplification and subcloning of the amplicons, the sequence of the 16S ribosomal DNA was determined in several resulting clones. The presence of pathogenic and apathogenic bacteria, such as Escherichia coli, are indicated by our result, providing evidence of bacteremia, which probably contributed to deat

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26

Ballares, Lea D., Frederick R. Masangkay, Joseph Dionisio, Oliver Villaflores, Maria Ruth Pineda-Cortel, and Giovanni D. Milanez. "Molecular detection of Acanthamoeba spp. in Seven Crater Lakes of Laguna, Philippines." Journal of Water and Health 18, no. 5 (2020): 776–84. http://dx.doi.org/10.2166/wh.2020.146.

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Acanthamoeba spp. are ubiquitous free-living amoeba with genotypes that cause severe pathology of the eyes, central nervous systems, and rare reports of cutaneous infections. The Seven Crater Lakes are freshwater water resources in Laguna, Philippines primarily used for aquaculture and tourism. A total of 16 surface water samples were collected from different sampling areas per Crater Lake and placed in sterile plastic containers. Samples were filtered using 1.2 μm pore size, glass microfiber filter. Filtered sediments were placed on non-nutrient agar lawned with Escherichia coli and incubated

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27

Galeone, Massimiliano, Roberta Colucci, Angelo Massimiliano D'Erme, Silvia Moretti, and Torello Lotti. "Potential Infectious Etiology of Behçet's Disease." Pathology Research International 2012 (December 29, 2012): 1–4. http://dx.doi.org/10.1155/2012/595380.

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Behçet's disease is a multisystem inflammatory disorder characterized by recurrent oral aphthous ulcers, genital ulcers, uveitis, and skin lesions. The cause of Behçet's disease remains unknown, but epidemiologic findings suggest that an autoimmune process is triggered by an environmental agent in a genetically predisposed individual. An infectious agent could operate through molecular mimicry, and subsequently the disease could be perpetuated by an abnormal immune response to an autoantigen in the absence of ongoing infection. Potentia bacterial are Saccharomyces cerevisiae, mycobacteria, Bor

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28

Mohawk, Krystle L., and Alison D. O'Brien. "Mouse Models ofEscherichia coliO157:H7 Infection and Shiga Toxin Injection." Journal of Biomedicine and Biotechnology 2011 (2011): 1–17. http://dx.doi.org/10.1155/2011/258185.

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Escherichia coliO157:H7 has been responsible for multiple food- and waterborne outbreaks of diarrhea and/or hemorrhagic colitis (HC) worldwide. More importantly, a portion ofE. coliO157:H7-infected individuals, particularly young children, develop a life-threatening sequela of infection called hemolytic uremic syndrome (HUS). Shiga toxin (Stx), a potent cytotoxin, is the major virulence factor linked to the presentation of both HC and HUS. Currently, treatment ofE. coliO157:H7 and other Stx-producingE. coli(STEC) infections is limited to supportive care. To facilitate development of therapeuti

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29

Ranford, Julia C., Anthony R. M. Coates, and Brian Henderson. "Chaperonins are cell-signalling proteins: the unfolding biology of molecular chaperones." Expert Reviews in Molecular Medicine 2, no. 8 (2000): 1–17. http://dx.doi.org/10.1017/s1462399400002015.

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The chaperonins are a subgroup of oligomeric molecular chaperones; the best-studied examples are chaperonin 60 (GroEL) and chaperonin 10 (GroES), both from the bacterium Escherichia coli. At the end of the 20th century, the paradigm of chaperonins as protein folders had emerged, but it is likely that during the 21st century these proteins will come to be viewed as intercellular signals. Indeed, it is possible that the chaperonins were among the first intercellular signalling proteins to evolve. During the past few years, it has emerged that chaperonin 10 and chaperonin 60 can be found on the s

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30

Vallance, Bruce A., Wanyin Deng, Leigh A. Knodler, and B. Brett Finlay. "Mice Lacking T and B Lymphocytes Develop Transient Colitis and Crypt Hyperplasia yet Suffer Impaired Bacterial Clearance during Citrobacter rodentium Infection." Infection and Immunity 70, no. 4 (2002): 2070–81. http://dx.doi.org/10.1128/iai.70.4.2070-2081.2002.

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ABSTRACT The bacterial pathogen Citrobacter rodentium belongs to a family of gastrointestinal pathogens that includes enteropathogenic and enterohemorrhagic Escherichia coli and is the causative agent of transmissible colonic hyperplasia in mice. The molecular mechanisms used by these pathogens to colonize host epithelial surfaces and form attaching and effacing (A/E) lesions have undergone intense study. In contrast, little is known about the host's immune response to these infections and its importance in tissue pathology and bacterial clearance. To address these issues, wild-type mice and m

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ÖNNBERG, ANNA, PAULA MÖLLING, JOHANNA ZIMMERMANN та BO SÖDERQUIST. "Molecular and phenotypic characterization of Escherichia coli and Klebsiella pneumoniae producing extended-spectrum β-lactamases with focus on CTX-M in a low-endemic area in Sweden". APMIS 119, № 4-5 (2011): 287–95. http://dx.doi.org/10.1111/j.1600-0463.2011.02730.x.

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32

NASEER, UMAER, BJØRG HALDORSEN, STÅLE TOFTELAND, et al. "Molecular characterization of CTX-M-15-producing clinical isolates of Escherichia coli reveals the spread of multidrug-resistant ST131 (O25:H4) and ST964 (O102:H6) strains in Norway." APMIS 117, no. 7 (2009): 526–36. http://dx.doi.org/10.1111/j.1600-0463.2009.02465.x.

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33

Holst-Hansen, Claus, Ross Wentworth Stephens, Bente Egholm Johannessen, Peter Buhl Jensen та Nils Brünner. "Plasma Escherichia Coli β-Galactosidase as a Marker of Tumor Burden and Response to Experimental Anti-Neoplastic Therapy in Nude Mice Xenografted with lacZ Transduced Human Tumor Cells". Laboratory Investigation 80, № 5 (2000): 719–24. http://dx.doi.org/10.1038/labinvest.3780075.

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34

Flieger, Antje, Kerstin Rydzewski, Sangeeta Banerji, Markus Broich, and Klaus Heuner. "Cloning and Characterization of the Gene Encoding the Major Cell-Associated Phospholipase A of Legionella pneumophila, plaB, Exhibiting Hemolytic Activity." Infection and Immunity 72, no. 5 (2004): 2648–58. http://dx.doi.org/10.1128/iai.72.5.2648-2658.2004.

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ABSTRACT Legionella pneumophila, the causative agent of Legionnaires' disease, is an intracellular pathogen of amoebae, macrophages, and epithelial cells. The pathology of Legionella infections involves alveolar cell destruction, and several proteins of L. pneumophila are known to contribute to this ability. By screening a genomic library of L. pneumophila, we found an additional L. pneumophila gene, plaB, which coded for a hemolytic activity and contained a lipase consensus motif in its deduced protein sequence. Moreover, Escherichia coli harboring the L. pneumophila plaB gene showed increase

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35

Hayashi, Tetsutaro, Naohide Oue, Naoya Sakamoto, et al. "Identification of Transmembrane Protein in Prostate Cancer by the Escherichia coli Ampicillin Secretion Trap: Expression of CDON Is Involved in Tumor Cell Growth and Invasion." Pathobiology 78, no. 5 (2011): 277–84. http://dx.doi.org/10.1159/000329588.

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36

Okada, Y. "Historical overview of research on the tobacco mosaic virus genome: genome organization, infectivity and gene manipulation." Philosophical Transactions of the Royal Society of London. Series B: Biological Sciences 354, no. 1383 (1999): 569–82. http://dx.doi.org/10.1098/rstb.1999.0408.

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Early in the development of molecular biology, TMV RNA was widely used as a mitochondrial RNA that could be purified easily, and it contributed much to research on protein synthesis. Also, in the early stages of elucidation of the genetic code, artificially produced TMV mutants were widely used and provided the first proof that the genetic code was non–overlapping. In 1982, Goelet et al. determined the complete TMV RNA base sequence of 6395 nucleotides. The four genes (130K, 180K, 30K and coat protein) could then be mapped at precise locations in the TMV genome. Furthermore it had become clear

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37

Bergstrom, Kirk S. B., Julian A. Guttman, Mohammad Rumi, et al. "Modulation of Intestinal Goblet Cell Function during Infection by an Attaching and Effacing Bacterial Pathogen." Infection and Immunity 76, no. 2 (2007): 796–811. http://dx.doi.org/10.1128/iai.00093-07.

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ABSTRACT The attaching and effacing (A/E) bacterial pathogens enteropathogenic Escherichia coli and enterohemorrhagic E. coli and the related mouse pathogen Citrobacter rodentium colonize their hosts' intestines by infecting the apical surfaces of enterocytes, subverting their function, and they ultimately cause diarrhea. Surprisingly, little is known about the interactions of these organisms with goblet cells, which are specialized epithelial cells that secrete the protective molecules Muc2 and trefoil factor 3 (Tff3) into the intestinal lumen. C. rodentium infection leads to dramatic goblet

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38

Mayer, Alejandro M., Mary L. Hall, Peer B. Jacobson та Jan L. Powell. "Differential effect of Vibrio vulnificus and Escherichia coli LPS on rat brain neonatal microglia release of superoxide anion, thromboxane B2 and tumor necrosis factor-α". Journal of Neuropathology and Experimental Neurology 66, № 5 (2007): 455. http://dx.doi.org/10.1097/01.jnen.0000268964.93966.78.

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Song, Baifen, Xiaoting Li, Jinzhu Ma та ін. "Prokaryotic Expression and Anti-IBDV Activity of Chicken Interleukin-18 and Interferon-γ". Cytogenetic and Genome Research 153, № 1 (2017): 36–45. http://dx.doi.org/10.1159/000481522.

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Interferon-γ (IFN-γ), a cytokine produced by activated natural killer cells and T lymphocytes, is an important regulator of innate and adaptive immunity. Interleukin (IL)-18, also known as IFN-γ-inducing factor, is a cytokine that induces T and natural killer cells to produce IFN-γ. In this study, the chicken IL-18 (ChIL-18) and chicken IFN-γ (ChIFN-γ) genes were inserted into the pET28a prokaryotic expression vector, resulting in pET28a-IL-18 and pET28a-IFN-γ, respectively. These plasmids were transformed into Escherichia coli strain BL21, and the ChIL-18 and ChIFN-γ proteins were expressed a

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40

Pryce, J. W., A. R. Bamber, M. T. Ashworth, N. J. Klein, and N. J. Sebire. "Immunohistochemical expression of inflammatory markers in sudden infant death; ancillary tests for identification of infection." Journal of Clinical Pathology 67, no. 12 (2014): 1044–51. http://dx.doi.org/10.1136/jclinpath-2014-202489.

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AimsSudden unexpected death in infancy (SUDI) investigation requires extensive ancillary investigations, the results of which, such as postmortem microbiology, can be difficult to interpret. Markers of an inflammatory response, including interleukin 6 (IL-6), c-reactive protein (CRP) and cellular adhesion molecules are elevated in infections, yet little attention has been paid to their assessment after death. This study investigates the role of inflammatory markers in SUDI autopsies for determining cause of death.MethodsCases of SUDI over a 14 year period were identified from an autopsy databa

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41

Brom, René van den, Inge Santman-Berends, Remco Dijkman, Piet Vellema, Reinie Dijkman, and Erik van Engelen. "An Accessible Diagnostic Toolbox to Detect Bacterial Causes of Ovine and Caprine Abortion." Pathogens 10, no. 9 (2021): 1147. http://dx.doi.org/10.3390/pathogens10091147.

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Results of laboratory investigations of ovine and caprine cases of abortion in the lambing season 2015–2016 were analyzed, using pathology records of submissions to Royal GD (Deventer, the Netherlands) from January until and including April 2016, in comparison with the results of two accessible alternative techniques for sampling aborted lambs and kids, swabbing the fetal oropharynx and puncture of the fetal lung. Chlamydia abortus was the main cause of abortion in sheep as well as in goats. Other causes of abortion were Campylobacter spp., Listeria spp., Escherichia coli, and Yersinia enteroc

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42

Samokovlija, Ana, and Bosiljka Djuricic. "Escherichia coli: Old problem, new pathology." Veterinarski glasnik 63, no. 1-2 (2009): 113–23. http://dx.doi.org/10.2298/vetgl0902113s.

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Even though the presence of Escherichia coli in the organism of humans and animals as a commensal has been known for a long time, attention has been paid to it only recently as the cause of diseases in humans and animals. The disease that is caused by this bacteria is linked to new variants-strains (eg. Verotoxic 0157:H7) which have, due to the toxic effects, a pathogensis and clinical picture that differs from those which have so far been linked to the conventional E.coli. Namely, it has been proved that there is a connection between the presence of certain strains of this bacteria in animals

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43

Goodsell, David S. "Escherichia coli." Biochemistry and Molecular Biology Education 37, no. 6 (2009): 325–32. http://dx.doi.org/10.1002/bmb.20345.

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44

Lorenz, E., M. D. Plamann, and G. V. Stauffer. "Escherichia coli." MGG Molecular & General Genetics 250, no. 1 (1996): 81. http://dx.doi.org/10.1007/s004380050053.

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45

Tuychiev, L. N., M. D. Ahkmedova, I. A. Imamova, N. U. Ibragimova, and S. D. Igamberdieva. "Predisposing factors contributing to the development of renal dysfunction in acute infectious diarrhea associated with hemocolitis (AIDH)." Journal Infectology 12, no. 5 (2021): 123–29. http://dx.doi.org/10.22625/2072-6732-2020-12-5-123-129.

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Outcomes of severe variants of acute infectious diarrhea in children with acute renal injury/failure (ARI/ARF) remain unsatisfactory, and mortality reaches 70% and more. ARI/ ARF therapy requires high material costs, which represent significant burden on health financing systems. It makes us consider AFR as one of the most important medical and social problems.The aim of our study was to investigate predisposing factors contributing to the development of renal dysfunction in acute infectious diarrhea associated with hemocolitis (AIDH), as well as to study the relationship of the developed rena

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Kelly, J., A. Oryshak, M. Wenetsek, J. Grabiec, and S. Handy. "The Colonic Pathology of Escherichia coli 0157." American Journal of Surgical Pathology 14, no. 1 (1990): 87–92. http://dx.doi.org/10.1097/00000478-199001000-00010.

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Petras, Robert E. "Enterohemorrhagic Escherichia coli-Associated Colitis." Pathology Case Reviews 2, no. 2 (1997): 66–70. http://dx.doi.org/10.1097/00132583-199702020-00002.

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Robins-Browne, Roy. "Pathogenic escherichia coli – an update." Pathology 45 (2013): S50. http://dx.doi.org/10.1097/01.pat.0000426839.87188.0a.

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Weiss, D. S. "Escherichia coli Shapeshifters." Journal of Bacteriology 195, no. 11 (2013): 2449–51. http://dx.doi.org/10.1128/jb.00306-13.

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O'Neill, M. C. "Escherichia coli Promoters." Journal of Biological Chemistry 264, no. 10 (1989): 5522–30. http://dx.doi.org/10.1016/s0021-9258(18)83576-1.

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