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Relevant bibliographies by topics / Estrogeen

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Academic literature on the topic 'Estrogeen'

Author: Grafiati

Published: 4 June 2021

Last updated: 14 February 2022

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Journal articles on the topic "Estrogeen"

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Wang, Po-Hsiang, Yi-Lung Chen, Sean Ting-Shyang Wei, et al. "Retroconversion of estrogens into androgens by bacteria via a cobalamin-mediated methylation." Proceedings of the National Academy of Sciences 117, no. 3 (2019): 1395–403. http://dx.doi.org/10.1073/pnas.1914380117.

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Steroid estrogens modulate physiology and development of vertebrates. Conversion of C19 androgens into C18 estrogens is thought to be an irreversible reaction. Here, we report a denitrifying Denitratisoma sp. strain DHT3 capable of catabolizing estrogens or androgens anaerobically. Strain DHT3 genome contains a polycistronic gene cluster, emtABCD, differentially transcribed under estrogen-fed conditions and predicted to encode a cobalamin-dependent methyltransferase system conserved among estrogen-utilizing anaerobes; an emtA-disrupted DHT3 derivative could catabolize androgens but not estroge

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Lee, Heehyoung, and Wenlong Bai. "Regulation of Estrogen Receptor Nuclear Export by Ligand-Induced and p38-Mediated Receptor Phosphorylation." Molecular and Cellular Biology 22, no. 16 (2002): 5835–45. http://dx.doi.org/10.1128/mcb.22.16.5835-5845.2002.

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ABSTRACT Estrogen receptors are phosphoproteins which can be activated by ligands, kinase activators, or phosphatase inhibitors. Our previous study showed that p38 mitogen-activated protein kinase was involved in estrogen receptor activation by estrogens and MEKK1. Here, we report estrogen receptor-dependent p38 activation by estrogens in endometrial adenocarcinoma cells and in vitro and in vivo phosphorylation of the estrogen receptor α mediated through p38. The phosphorylation site was identified as threonine-311 (Thr311), located in helix 1 of the hormone-binding domain. The mutation of thr

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Kumar, Anita, Antara Banerjee, Dipty Singh, et al. "Estradiol: A Steroid with Multiple Facets." Hormone and Metabolic Research 50, no. 05 (2018): 359–74. http://dx.doi.org/10.1055/s-0044-100920.

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AbstractSeventy-five glorious years have passed since estradiol was discovered by Edward Doisy. From discovery in the ovaries to delineation of diverse physiological effects, research on estrogens has covered a lot of ground. Estrogen receptors that mediate estrogenic effects, have been detected not only in reproductive organs, but also in other body organs. Estrogen receptors function either as conventional transcription factors or as rapid signal transducers. These different modes of action are opted by estrogens to elicit an array of reproductive and non-reproductive functions. It is well e

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Nwankudu, O. N. "Endocrine, Reproductive, Neurophysiologic and Extraneous Activities of Estrogen in Vertebrates." Nigerian Veterinary Journal 41, no. 2 (2021): 85–107. http://dx.doi.org/10.4314/nvj.v41i2.2.

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Estrogens are reproductive hormones synthesized in the gonads of both male and female vertebrates. This review is geared towards uncovering some endocrine, reproductive, neurophysiologic and extraneous activities of estrogen in vertebrates. The three most common naturally occurring estrogens are: Estrone (E1), estradiol (E2), and estriol (E3). In primates, estradiol is the most potent and predominant estrogen during reproductive years. Estrogens are synthesized primarily in the female ovaries and in small quantities in the male testes and the adrenal glands, brain, and fat of both sexes. Estro

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Birzniece, Vita, and Ken K. Y. Ho. "MECHANISMS IN ENDOCRINOLOGY: Paracrine and endocrine control of the growth hormone axis by estrogen." European Journal of Endocrinology 184, no. 6 (2021): R269—R278. http://dx.doi.org/10.1530/eje-21-0155.

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There is a strong biological link between the growth hormone (GH) and gonadal systems in growth, development and metabolism; however, regulatory interactions are poorly understood. Advances in estrogen biology and endocrine physiology have provided insights into mechanistic links between the two systems. Estrogens are synthesized from androgens by aromatase which is widely distributed in extragonadal tissues. Local generation of estrogens raise the possibility of paracrine control as an additional level to classical endocrine regulation of the GH system. To explore the mechanistic links, we re

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Henriksson, Peter, and Reinhard Stege. "Cost Comparison of Parenteral Estrogen and Conventional Hormonal Treatment in Patients With Prostatic Cancer." International Journal of Technology Assessment in Health Care 7, no. 2 (1991): 220–26. http://dx.doi.org/10.1017/s0266462300005110.

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AbstractThe present study compares the cost of antitumor therapy and adverse cardiovascular effects during the first year of treatment with oral estrogens, nonoral estrogens, or surgical castration in patients with prostatic cancer. We found a much higher cost for patients treated with orchidectomy and oral estrogens than for patients treated with nonoral estrogens. Twenty-five percent of the patients treated with oral estrogen suffered cardiovascular complications, compared to none of the patients treated by orchidectomy or nonoral estrogens. The initial cost of orchidectomy as compared to no

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Imai, Yuuki, Shino Kondoh, Alexander Kouzmenko та Shigeaki Kato. "Minireview: Osteoprotective Action of Estrogens Is Mediated by Osteoclastic Estrogen Receptor-α". Molecular Endocrinology 24, № 5 (2010): 877–85. http://dx.doi.org/10.1210/me.2009-0238.

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Abstract The osteoprotective action of estrogen in women has drawn considerable attention because estrogen deficiency-induced osteoporosis became one of the most widely spread diseases in developed countries. In men, the significance of estrogen action for bone health maintenance is also apparent from the osteoporotic phenotype seen in male patients with genetically impaired estrogen signaling. Severe bone loss and high bone turnover, including typical osteofeatures seen in postmenopausal women, can also be recapitulated in rodents after ovariectomy. However, the expected osteoporotic phenotyp

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Kassi, E., and P. Moutsatsou. "Estrogen Receptor Signaling and Its Relationship to Cytokines in Systemic Lupus Erythematosus." Journal of Biomedicine and Biotechnology 2010 (2010): 1–14. http://dx.doi.org/10.1155/2010/317452.

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Dysregulation of cytokines is among the main abnormalities in Systemic Lupus Erythematosus (SLE). However, although, estrogens, which are known to be involved in lupus disease, influence cytokine production, the underlying molecular mechanisms remain poorly defined. Recent evidence demonstrates the presence of estrogen receptor in various cell types of the immune system, while divergent effects of estrogens on the cytokine regulation are thought to be implicated. In this paper, we provide an overview of the current knowledge as to how estrogen-induced modulation of cytokine production in SLE i

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Lizcano, Fernando, and Guillermo Guzmán. "Estrogen Deficiency and the Origin of Obesity during Menopause." BioMed Research International 2014 (2014): 1–11. http://dx.doi.org/10.1155/2014/757461.

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Sex hormones strongly influence body fat distribution and adipocyte differentiation. Estrogens and testosterone differentially affect adipocyte physiology, but the importance of estrogens in the development of metabolic diseases during menopause is disputed. Estrogens and estrogen receptors regulate various aspects of glucose and lipid metabolism. Disturbances of this metabolic signal lead to the development of metabolic syndrome and a higher cardiovascular risk in women. The absence of estrogens is a clue factor in the onset of cardiovascular disease during the menopausal period, which is cha

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Liu, Jia Wei, Elisabeth Jeannin, and Didier Picard. "The Anti-Estrogen Hydroxytamoxifen Is a Potent Antagonist in a Novel Yeast System." Biological Chemistry 380, no. 11 (1999): 1341–45. http://dx.doi.org/10.1515/bc.1999.172.

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AbstractThe budding yeastSaccharomyces cerevisiaehas been used extensively as a biological ‘test tube’ to study the regulation of the human estrogen receptor (ER) α. However, anti-estrogens, which are of great importance as therapeutic agents and research tools, fail to antagonize the activation by estrogen in yeast. Here, we have surveyed the antagonistic potential of five different anti-estrogens of diverse chemical nature. While they all act as agonists for wild-type ERα we have established a novel yeast assay system for anti-estrogens, in which at least the commonly used anti-estrogen hydr

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Dissertations / Theses on the topic "Estrogeen"

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Järvenpää, Paula. "Intestinal metabolism of estrogens including some studies on medroxiprogesterone acetate and megestrol acetate." Helsinki : Finnish Society of Sciences and Letters, 1991. http://books.google.com/books?id=ee5qAAAAMAAJ.

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Lee, Isaish Chi Kin. "Measuring the binding kinetics of estrogen receptor alpha and dietary estrogens." HKBU Institutional Repository, 2014. https://repository.hkbu.edu.hk/etd_oa/28.

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Anti-estrogen drugs such as Tamoxifen and Raloxifene are widely prescribed for breast cancer patients. While they are effective, they also have serious side effects. Alternative drugs are therefore being developed. In the drug discovery process, the in vitro binding of estrogen receptors and lead compounds were studied. The binding strength was conventionally quantified in terms of equilibrium dissociation constants (K0 ). However, the binding kinetic rates and especially off-rates (k0 ff) were recently shown to be better indicators of drug potency. In this thesis, we identified a few dietary

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Wade, Christian Bernard. "Mechanisms of estrogen rapid signaling /." Thesis, Connect to this title online; UW restricted, 2002. http://hdl.handle.net/1773/6272.

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Scherr, Frank. "Sorption, degradation and transport of estrogens and estrogen sulphates in agricultural soils." Diss., Lincoln University, 2009. http://hdl.handle.net/10182/1017.

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The fate and behaviour of estrogens in the environment are of concern due to the compounds’ endocrine disruption potential. Estrogens, namely 17β-estradiol (E2), estrone (E1), and estrogen sulphates, i.e. 17β-estradiol-3-sulphate (E2-3S) and estrone-3-sulphate (E1-3S) excreted by livestock constitute a potential source for estrogen contamination in the environment. A method was developed to separate and quantify the hormones by high-performance-liquid-chromatography (HPLC) and ultraviolet detection (UV). A combination of dichloromethane (DCM) and dicyclohexylamine hydrochloride (DCH·HCl) gave

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Graham, Lisa Anne. "Environmental Estrogens: Assessing Human Gestational Exposure and Interactions with the Estrogen Receptor." Thesis, University of Canterbury. Chemistry, 2012. http://hdl.handle.net/10092/7173.

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Environmental xenoestrogens (EEs) are chemicals that when they enter the body, the body responds to them as it would to endogenous estrogens. Humans are exposed to these chemicals on a daily basis via natural components, additives and contaminants in food and water, through the use of pharmaceuticals and personal care products such as sunscreens, lotions and toothpaste. Exposure to EEs is thought to result in adverse effects on humans such as decreased fertility, increased susceptibility to hormone-sensitive cancers, deformities of the male genitalia and precocious puberty in females. Th

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Lambert, K. Chad. "The effects of estrogen signaling in innate and adaptive immune cells /." Free to MU Campus, others may purchase, 2005. http://wwwlib.umi.com/cr/mo/fullcit?p3189934.

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Nilsson, Ola. "The role of estrogen in growth plate chondrogenesis /." Stockholm, 2002. http://diss.kib.ki.se/2002/91-7349-410-0/.

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Van, Wyk Susan. "The effects of growth stimulants used at cattle feedlots, on reproductive health and thyroid function of Sprague-Dawley rats." Diss., University of Pretoria, 2011. http://hdl.handle.net/2263/24885.

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Reports of endocrine disrupting potential of common environmental chemicals and the effects on reproductive health are well documented in literature. It has been suggested that deteriorating male reproductive health could be due to in utero exposures to these chemicals. The effects mediated through endocrine disrupting chemicals (EDCs) are on the fetus and may therefore be trans-generational. Ultimately, these chemicals land up in aquatic systems and affect wildlife and humans. Humans are exposed to these chemicals through multiple routes including atmosphere, water, occupational, domestic and

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Zhang, Qiu-Xia. "Estrogen receptor gene alterations in human breast cancer." Lund : Jubileumsinstitutionen, Dept. of Oncology,Lund University, 1997. http://catalog.hathitrust.org/api/volumes/oclc/39738537.html.

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Linford, Nancy J. "Effects of estrogenic compounds on neuronal apoptotic pathways /." Thesis, Connect to this title online; UW restricted, 2001. http://hdl.handle.net/1773/6289.

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Books on the topic "Estrogeen"

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Angerer, Erwin von. The estrogen receptor as a target for rational drug design. Springer, 1995.

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Angerer, Erwin von. The estrogen receptor as a target for rational drug design. Springer, 1995.

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D, Lindsay Robert Ph, Dempster David W, and Jordan V. Craig, eds. Estrogens and antiestrogens: Basic and clinical aspects. Lippincott-Raven, 1997.

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Rattner, Heilman Joan, ed. Estrogen. HarperPerennial, 1991.

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Nachtigall, Lila. Estrogen. 3rd ed. Harper Resource, 2000.

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Nachtigall, Lila. Estrogen. 2nd ed. HarperPerennial, 1995.

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The estrogen decision: A self-help program. Westchester Pub. Co., 1994.

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Eyster, Kathleen M., ed. Estrogen Receptors. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3127-9.

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International Congress on the Menopause (8th 1996 Sydney, N.S.W.). Progress in the management of the menopause: The proceedings of the 8th International Congress on the Menopause, Sydney, Australia, November 1996. Parthenon Pub. Group, 1997.

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Estrogen versus cancer. Nova Science Publishers, 2009.

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Book chapters on the topic "Estrogeen"

1

Lindsay, Robert. "Estrogens and Estrogen Agonists/Antagonists." In Osteoporosis. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-459-9_15.

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Watts, Nelson B. "Estrogens, Estrogen Agonists/Antagonists, and Calcitonin." In Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism. John Wiley & Sons, Inc., 2013. http://dx.doi.org/10.1002/9781118453926.ch48.

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Bidlingmaier, M. "Estrogene." In Springer Reference Medizin. Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_1051.

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Bidlingmaier, M. "Estrogene." In Lexikon der Medizinischen Laboratoriumsdiagnostik. Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-662-49054-9_1051-1.

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Leeth, Chris. "Estrogen." In Encyclopedia of Child Behavior and Development. Springer US, 2011. http://dx.doi.org/10.1007/978-0-387-79061-9_1032.

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Metzler, Marnie. "Estrogen." In Encyclopedia of Animal Cognition and Behavior. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-47829-6_481-1.

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Cooley, Laura A., Daniel G. Bausch, Marija Stojkovic, et al. "Estrogen." In Encyclopedia of Intensive Care Medicine. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_1566.

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Proske, Uwe, David L. Morgan, Tamara Hew-Butler, et al. "Estrogen." In Encyclopedia of Exercise Medicine in Health and Disease. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2366.

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LaCaille, Lara, Anna Maria Patino-Fernandez, Jane Monaco, et al. "Estrogen." In Encyclopedia of Behavioral Medicine. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_249.

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Oladipo, A., and J. C. Stevenson. "Estrogen." In Management of Fractures in Severely Osteoporotic Bone. Springer London, 2000. http://dx.doi.org/10.1007/978-1-4471-3825-9_29.

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Conference papers on the topic "Estrogeen"

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Islam, Nazmul. "Real-Time Detection of Estrogen by Microcantilever Sensor in Microfluidic Channel." In ASME 2008 International Mechanical Engineering Congress and Exposition. ASMEDC, 2008. http://dx.doi.org/10.1115/imece2008-68903.

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As the world becomes increasingly concerned with endocrine disruptor from estrogenic activity, and since the threat of estrogen can be substantially mitigated if detected early, the demand for real-time/on-site detection of estrogen is expanding quickly. However, current technology is expensive, technically complicated and not available for estrogen level determination at the contamination sites. The primary objective of this research is to develop and validate a robust, rapid lab-on-chip for detecting estrogen in environmental water samples. Anti-estrogen antibodies can be layered to the micr

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Thornton, Gail M., Soraya J. Bailey, Xinxin Shao, Douglas Morck, David A. Hart, and Yamini Achari. "Influence of Early Ovariohysterectomy on the Mechanical Properties of Rabbit Medial Collateral Ligament." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176260.

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Female athletes have significantly higher incidence of anterior cruciate ligament (ACL) injuries than males participating in similar sports [1]. To date, no clear explanation has emerged for this disparity. However, hormonal differences may provide an explanation because some ACL injuries have been linked to physiologic fluctuations in estrogen levels over the menstrual cycle [2]. Receptors for estrogen have been identified in rabbit and human ACLs and medial collateral ligaments (MCLs) [3]. Increased estrogen levels caused decreased fibroblast proliferation and collagen synthesis in cell cult

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Falk, Roni T., Gretchen L. Gierach, Xia Xu, Joanne F. Dorgan, Timothy D. Veenstra, and Louise A. Brinton. "Abstract A90: Relationship of serum estrogens and estrogen metabolites with postmenopausal breast cancer risk." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Oct 22-25, 2011; Boston, MA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1940-6207.prev-11-a90.

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Gu, Fangyi, Neil E. Caporaso, A. Heather Eliassen, Xia Xu, Susan E. Hankinson, and Regina G. Ziegler. "Abstract 3738: Associations between cigarette smoking and urinary estrogens/estrogen metabolites (EM) in premenopausal women." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-3738.

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Liu, Aiping, and Naomi Chesler. "Effects of Estrogen on Pulmonary Vascular Remodeling in Pulmonary Artery Hypertension." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14736.

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Pulmonary artery hypertension (PAH) is a female dominant, fatal disease characterized by progressive increase of pulmonary vascular resistance and loss of compliance. The role of estrogen in these pulmonary vascular changes with PAH progression remains unclear. Our objective was to study the effects of estrogen on pulmonary arterial (PA) remodeling in a mouse model of progressive PAH, created via a combination of a VEGF inhibitor Sugen and chronic hypoxia (SuHx). To quantify PA hemodynamics, we measured in vivo pressure and flow simultaneously in live mice in order to obtain pulmonary vascular

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Cowden, Courtney, Naveen Chandrashekar, Javad Hashemi, Vaughan H. Lee, Daniel M. Hardy, and James R. Slauterbeck. "Effect of Estrogen on the Mechanical Properties of Prepubertal Rabbit ACL." In ASME 2004 International Mechanical Engineering Congress and Exposition. ASMEDC, 2004. http://dx.doi.org/10.1115/imece2004-59986.

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Recent research suggests that estradiol affects the material properties of the Anterior Cruciate Ligament (ACL). Here we experimentally determine the cross-sectional area and tensile properties of ACLs from the groups of prepubertal rabbits: untreated control (C), estrogen treated (E) and estrogen and epidermal growth factor treated (E+EGF). Estradiol decreased the ultimate tensile strength and toughness of the ACL. Addition of EGF decreased the modulus of elasticity.

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Ciani, Cesare, Paula A. Ramirez Marin, Stephen B. Doty, and Susannah P. Fritton. "Estrogen Depletion Increases Osteocyte Canalicular Diameter in Cortical and Cancellous Bone of the Rat Proximal Tibia." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-205382.

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Estrogen depletion has been shown to cause bone loss in the proximal metaphysis of the rat tibia [1,2]. A decrease in bone volume fraction is frequently reported, yet there is little analysis in the literature related to changes in microporosities during osteoporosis. Our recent work quantifying microporosity changes due to estrogen depletion has shown an increase in the lacunar-canalicular porosity surrounding osteocytes in the proximal metaphysis of the rat tibia [3].

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Dallal, Cher M., Louise A. Brinton, Charles E. Matthews, et al. "Abstract 2519: Is accelerometer-measured physical activity associated with urinary estrogens and estrogen metabolites among postmenopausal women?." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-2519.

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Ferreri, Suzanne, and Yi-Xian Qin. "Alteration of Bone’s Nonlinear Elastic and Viscoelastic Nanomechanical Properties Is Triggered by Low Intensity Pulsed Ultrasound." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206711.

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Dynamic mechanotransduction, particularly under high frequency, low amplitude signals, has been proven effective in mediating bone loss and improving mechanical strength for tissues affected by estrogen deficient osteopenia. Ultrasound, which behaves as an alternating pressure wave in bone, may offer an effective, non-invasive technology for delivery of anabolic signals. In vitro, dynamic mechanical signals delivered using ultrasound have been shown to increase osteoblast proliferation [1], and in vivo studies have established ultrasound as an effective treatment for delayed and non-union frac

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Ragaz, J., K. Wilson, G. Muraca, J. Budlovsky, and J. Froehlich. "Abstract P6-09-09: Dual Estrogen Effects on Breast Cancer: Endogenous Estrogen Stimulates, Exogenous Estrogen Protects. Further Investigation of Estrogen Chemoprevention Is Warranted." In Abstracts: Thirty-Third Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 8‐12, 2010; San Antonio, TX. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/0008-5472.sabcs10-p6-09-09.

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Reports on the topic "Estrogeen"

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DeSombre, E. R., R. C. Mease, A. Hughes, P. V. Harper, O. T. DeJesus, and A. M. Friedman. Bromine-80m-labeled estrogens: Auger-electron emitting, estrogen receptor-directed ligands with potential for therapy of estrogen receptor positive cancers. Office of Scientific and Technical Information (OSTI), 1988. http://dx.doi.org/10.2172/6347502.

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Wuttke, Deborah S. Breast Cancer Therapeutics, Environmental Estrogens, and the Estrogen Receptor (ER); Characterization of the Diverse Ligand Binding Properties of the ER. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada408777.

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Wuttke, Deborah S. Breast Cancer Therapeutics, Environmental Estrogens, and the Estrogen Receptor (ER); Characterization of the Diverse Ligand Binding Properties of the ER. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada420485.

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Bhat, Abhijit S. Analogs of Estrogen Metabolites as Probes of Estrogen-Induced Tumorigenesis. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada371246.

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Smith, Carolyn L. Activation of Estrogen Receptor-Beta-Dependent Transcription by Estrogen-Independent Pathways. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada400004.

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Smith, Carolyn L. Activation of Estrogen Receptor-Beta-Dependent Transcription by Estrogen-Independent Pathways. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada383071.

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Smith, Carolyn L. Activation of Estrogen Receptor-Beta-Dependent Transcription by Estrogen-Independent Pathways. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada413466.

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Smith, Carolyn L. Activation of Estrogen Receptor-Beta-Dependent Transcription by Estrogen-Independent Pathways. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada393484.

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Berglund, J. Andrew. RNA Regulation of Estrogen. Defense Technical Information Center, 2010. http://dx.doi.org/10.21236/ada541794.

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Russo, Jose. Estrogen and Breast Cancer. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada428032.

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