Relevant bibliographies by topics / Estrogen receptor. eng

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Academic literature on the topic 'Estrogen receptor. eng'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Estrogen receptor. eng"

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Albiñana, Virginia, Maria Bernabeu-Herrero, Roberto Zarrabeitia, Carmelo Bernabeu, and Luisa Botella. "Estrogen therapy for hereditary haemorrhagic telangiectasia (HHT): Effects of raloxifene, on Endoglin and ALK1 expression in endothelial cells." Thrombosis and Haemostasis 103, no. 03 (2010): 441–51. http://dx.doi.org/10.1160/th09-07-0425.

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SummaryHereditary haemorrhagic telangiectasia (HHT), or Rendu-Osler-Weber syndrome, is an autosomal dominant vascular disease. The clinical manifestations are epistaxis, mucocutaneous and gastrointestinal telangiectases, and arteriovenous malformations. There are two predominant types of HHT caused by mutations in Endoglin (ENG) and activin receptor-like kinase 1 (ALK1) (ACVRL1) genes, HHT1 and HHT2, respectively. No cure for HHT has been found and there is a current need to find new effective drug treatments for the disease. Some patients show severe epistaxis which interferes with their qual
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Albiñana, Virginia, Angel M. Cuesta, Isabel de Rojas-P, et al. "Review of Pharmacological Strategies with Repurposed Drugs for Hereditary Hemorrhagic Telangiectasia Related Bleeding." Journal of Clinical Medicine 9, no. 6 (2020): 1766. http://dx.doi.org/10.3390/jcm9061766.

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The diagnosis of hereditary hemorrhagic telangiectasia (HHT) is based on the Curaçao criteria: epistaxis, telangiectases, arteriovenous malformations in internal organs, and family history. Genetically speaking, more than 90% of HHT patients show mutations in ENG or ACVRL1/ALK1 genes, both belonging to the TGF-β/BMP9 signaling pathway. Despite clear knowledge of the symptoms and genes of the disease, we still lack a definite cure for HHT, having just palliative measures and pharmacological trials. Among the former, two strategies are: intervention at “ground zero” to minimize by iron and blood
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Abramenko, Nikita, Fréderic Vellieux, Petra Tesařová, et al. "Estrogen Receptor Modulators in Viral Infections Such as SARS−CoV−2: Therapeutic Consequences." International Journal of Molecular Sciences 22, no. 12 (2021): 6551. http://dx.doi.org/10.3390/ijms22126551.

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COVID-19 is a pandemic respiratory disease caused by the SARS−CoV−2 coronavirus. The worldwide epidemiologic data showed higher mortality in males compared to females, suggesting a hypothesis about the protective effect of estrogens against severe disease progression with the ultimate end being patient’s death. This article summarizes the current knowledge regarding the potential effect of estrogens and other modulators of estrogen receptors on COVID-19. While estrogen receptor activation shows complex effects on the patient’s organism, such as an influence on the cardiovascular/pulmonary/immu
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Zarrabeitia, Roberto, Luisa Ojeda-Fernandez, Lucia Recio, et al. "Bazedoxifene, a new orphan drug for the treatment of bleeding in hereditary haemorrhagic telangiectasia." Thrombosis and Haemostasis 115, no. 06 (2016): 1167–77. http://dx.doi.org/10.1160/th15-03-0239.

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SummaryHereditary haemorrhagic telangiectasia (HHT), or Rendu-Osler-Weber syndrome, is a dominant genetic vascular disorder. In HHT, blood vessels are weak and prone to bleeding, leading to epistaxis and anaemia, severely affecting patients’ quality of life. Development of vascular malformations in HHT patients is originated mainly by mutations in ACVRL1/ALK1 (activin receptor-like kinase type I) or Endoglin (ENG) genes. These genes encode proteins of the TGF-β signalling pathway in endothelial cells, controlling angiogenesis. Haploinsufficiency of these proteins is the basis of HHT pathogenic
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Pentikäinen, Virve, Krista Erkkilä, Laura Suomalainen, Martti Parvinen, and Leo Dunkel. "Estradiol Acts as a Germ Cell Survival Factor in the Human Testis in Vitro*." Journal of Clinical Endocrinology & Metabolism 85, no. 5 (2000): 2057–67. http://dx.doi.org/10.1210/jcem.85.5.6600.

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Abstract The necessity of estrogens for male fertility was recently discovered in studies on both estrogen receptor α knockout and aromatase (cyp 19 gene) knockout mice. However, direct testicular effects of estrogens in male reproduction have remained unclear. Here we studied the protein expression of ERα and the recently described estrogen receptor β in the human seminiferous epithelium and evaluated the role of 17β-estradiol, the main physiological estrogen, in male germ cell survival. Interestingly, both estrogen receptors α and β were found in early meiotic spermatocytes and elongating sp
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Dama, Aida, Chiara Baggio, Carlotta Boscaro, Mattia Albiero, and Andrea Cignarella. "Estrogen Receptor Functions and Pathways at the Vascular Immune Interface." International Journal of Molecular Sciences 22, no. 8 (2021): 4254. http://dx.doi.org/10.3390/ijms22084254.

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Estrogen receptor (ER) activity mediates multiple physiological processes in the cardiovascular system. ERα and ERβ are ligand-activated transcription factors of the nuclear hormone receptor superfamily, while the G protein-coupled estrogen receptor (GPER) mediates estrogenic signals by modulating non-nuclear second messengers, including activation of the MAP kinase signaling cascade. Membrane localizations of ERs are generally associated with rapid, non-genomic effects while nuclear localizations are associated with nuclear activities/transcriptional modulation of target genes. Gender depende
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Jordan, V. Craig. "The new biology of estrogen-induced apoptosis applied to treat and prevent breast cancer." Endocrine-Related Cancer 22, no. 1 (2014): R1—R31. http://dx.doi.org/10.1530/erc-14-0448.

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The successful use of high-dose synthetic estrogens to treat postmenopausal metastatic breast cancer is the first effective ‘chemical therapy’ proven in clinical trial to treat any cancer. This review documents the clinical use of estrogen for breast cancer treatment or estrogen replacement therapy (ERT) in postmenopausal hysterectomized women, which can either result in breast cancer cell growth or breast cancer regression. This has remained a paradox since the 1950s until the discovery of the new biology of estrogen-induced apoptosis at the end of the 20th century. The key to triggering apop
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Sonnenschein, C., A. M. Soto, M. F. Fernandez, N. Olea, M. F. Olea-Serrano, and M. D. Ruiz-Lopez. "Development of a marker of estrogenic exposure in human serum." Clinical Chemistry 41, no. 12 (1995): 1888–95. http://dx.doi.org/10.1093/clinchem/41.12.1888.

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Abstract The deleterious, disruptive effects of estrogen mimics on the endocrine system were discovered after the compounds were released into the environment. Their chemical structure does not obviously resemble that of steroid hormones; hence, their estrogenic effects were totally unexpected. In addition to occupational exposures, environmental estrogens may have played a role in decreasing the quantity and quality of human semen during the last 50 years and in increasing the incidences of testicular cancer and cryptorchidism in men and breast cancer in women and men in industrialized countr
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Martin, Olwenn, Tassos Shialis, John Lester, Mark Scrimshaw, Alan Boobis, and Nikolaos Voulvoulis. "Testicular dysgenesis syndrome and the estrogen hypothesis: a quantitative meta-analysis." Ciência & Saúde Coletiva 13, no. 5 (2008): 1601–18. http://dx.doi.org/10.1590/s1413-81232008000500024.

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Male reproductive tract abnormalities such as hypospadias and cryptorchidism, and testicular cancer have been proposed to comprise a common syndrome together with impaired spermatogenesis with a common etiology resulting from the disruption of gonadal development during fetal life, the testicular dysgenesis syndrome (TDS). The only quantitative summary estimate of the link between prenatal exposure to estrogenic agents and testicular cancer was published over 10 years ago; other reviews of the link between estrogenic compounds, other than the potent pharmaceutical estrogen diethylstilbestrol (
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Bernard, Daniel J., George E. Bentley, Jacques Balthazart, Fred W. Turek та Gregory F. Ball. "Androgen Receptor, Estrogen Receptor α, and Estrogen Receptorβ Show Distinct Patterns of Expression in Forebrain Song Control Nuclei of European Starlings1". Endocrinology 140, № 10 (1999): 4633–43. http://dx.doi.org/10.1210/endo.140.10.7024.

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Abstract In songbirds, singing behavior is controlled by a discrete network of interconnected brain nuclei known collectively as the song control system. Both the development of this system and the expression of singing behavior in adulthood are strongly influenced by sex steroid hormones. Although both androgenic and estrogenic steroids have effects, androgen receptors (AR) are more abundantly and widely expressed in song nuclei than are estrogen receptors (ERα). The recent cloning of a second form of the estrogen receptor in mammals, ERβ, raises the possibility that a second receptor subtype
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Dissertations / Theses on the topic "Estrogen receptor. eng"

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Dune, Ana Cláudia. "Expressão de recptor de estrógeno, vimentina, TGF"beta", e marcador de macrófagos em tumor ósseo de células gigantes em gatos domésticos /." Jaboticabal : [s.n.], 2008. http://hdl.handle.net/11449/95976.

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Orientador: Mirela Tinucci Costa<br>Banca: Gisele Fabrino Machado<br>Banca: Gervásio Henrique Bechara<br>Resumo: O tumor ósseo de células gigantes apresenta 3 diferentes tipos celulares, sendo duas estromais: fibroblastos neoplásicos e células mononucleares; e o terceiro tipo, células gigantes. Propôs-se que este tumor é de linhagem monócito-macrofágica e acredita-se que as células gigantes se formam por fusão de células mononucleares. Aparentemente os fibroblastos neoplásicos que expressam o fator transformador de crescimento TGF"beta"1 estão envolvidos no recrutamento das células gigantes pa
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Vladimir, Vidović. "Značaj određivanja androgenih receptora u odgovoru na hormonsku terapiju kod estrogen receptor pozitivnih pacijenata sa karcinomom dojke." Phd thesis, Univerzitet u Novom Sadu, Medicinski fakultet u Novom Sadu, 2020. https://www.cris.uns.ac.rs/record.jsf?recordId=114127&source=NDLTD&language=en.

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Glavni problem u lečenju karcinoma dojke je kako na osnovu kliničke klasifikacije i morfolo&scaron;kih osobina tumora predvideti njegovo dalje pona&scaron;anje. Vrlo često ni kombinacija standardnih prognostičkih faktora ne daje odgovor o potrebi davanja adjuvantne hemioterapije. U cilju sprovođenja adekvatne dalje terapije karcinoma dojke i otkrivanja agresivnih tipova tumora, a nakon hirur&scaron;kog lečenja, postoji stalna potreba za pronalaženjem novih pokazatelja pomoću kojih bi se identifikovale bolesnice koje imaju povećan rizik od razvoja relapsa bolesti. Ciljevi ove studije su bili da
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Aleksandra, Fejsa Levakov. "Ekspresija estrogenog receptora β u prekanceroznim lezijama i adenokarcinomu prostate". Phd thesis, Univerzitet u Novom Sadu, Medicinski fakultet u Novom Sadu, 2016. http://www.cris.uns.ac.rs/record.jsf?recordId=99993&source=NDLTD&language=en.

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Adenokarcinom prostate (PCa) je najče&scaron;ći karcinom u mu&scaron;karaca. Intraepitelne prostatične neoplazme visokog gradusa (HGPIN) su lezije koje prethode nastanku invazivnog karcinoma i podrazumevaju kompletno odsustvo bazalnih ćelija i invaziju strome malignim acinusima. Estrogeni receptor &beta; (ER&beta;) se nalazi u jedrima bazalnih i sekretornih ćelija acinusa i delimično u stromalnim ćelijama. Cilj istraživanja je da prikaže i lokalizuje ER&beta; u različitim morfolo&scaron;kim lezijama prostate: hiperplaziji (BHP), PINu i u PCa sa različitim Gleason scorom. Pretpostavlja se da pr
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Smith, Matthew John. "Exploring the effects of estrogen receptor beta polymorphisms on wound repair." Thesis, University of Manchester, 2017. https://www.research.manchester.ac.uk/portal/en/theses/exploring-the-effects-of-estrogen-receptor-beta-polymorphisms-on-wound-repair(af2ae557-16ef-43a2-b5a7-db541b7c5d65).html.

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Estrogen is an important regulator and promoter of epithelial wound healing. This is facilitated by increased keratinocyte and fibroblast migration and proliferation, as well as promotion of angiogenesis, matrix deposition and inflammatory response dampening. The potential to target this pathway for therapeutics is highlighted by observations that post-menopausal women on hormone replacement therapy have a significantly lower incidence of venous ulcers. Previous work from this laboratory identified four SNPs (single nucleotide polymorphisms) in the 5’UTR of estrogen receptor beta (ERβ) gene th
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Alexander, Amy. "GPR30 : a role for a novel estrogen-sensing receptor in hippocampal function?" Thesis, University of Dundee, 2014. https://discovery.dundee.ac.uk/en/studentTheses/a51c1614-e36e-40b6-aad3-7efacdce0f48.

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17ß-estradiol (E2), the most potent form of the steroid hormone class, the estrogens, is a known regulator of hippocampal function and is capable of being synthesised within hippocampal neurons. E2 can modulate glutamatergic synaptic transmission and hippocampal synaptic plasticity, as well as enhance hippocampal dependent learning in rodent models. However, not all effects of estradiol are mediated by the two canonical estrogen receptors, ERa and ERß. Recently, a novel estrogen-sensing G protein coupled receptor (GPR30) was identified, and hippocampal expression of GPR30 indicates a potential
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Tanja, Lakić. "Klinička vrednost određivanja Ki-67 proliferativnog indeksa u karcinomima dojke sa pozitivnim hormonskim receptorima." Phd thesis, Univerzitet u Novom Sadu, Medicinski fakultet u Novom Sadu, 2018. https://www.cris.uns.ac.rs/record.jsf?recordId=107631&source=NDLTD&language=en.

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<b>Uvod: </b>Karcinom dojke je heterogena bolest koju karakteri&scaron;u različita morfologija, imunohisto-hemijski profil, klinički tok i terapijski odgovor. Ki-67 proliferativni indeks je jedan od markera sa prognostičkim i prediktivnim značajem, čije metodolo&scaron;ko određivanje i analiza jo&scaron; uvek nisu standardizovani. <b>Cilj: </b>Utvrditi graničnu (&ldquo;cut-off&rdquo;) prognostičku vrednost Ki-67 indeksa, kao i povezanost vrednosti Ki-67 u ranom luminalnom karcinomu dojke sa prognostičkim i prediktivnim parametrima karcinoma dojke, kao &scaron;to su životna dob bolesnica, velič
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Dzida, Tomasz. "Predicting context specific enhancer-promoter interactions from ChIP-Seq time course data." Thesis, University of Manchester, 2017. https://www.research.manchester.ac.uk/portal/en/theses/predicting-context-specific-enhancerpromoter-interactions-from-chipseq-time-course-data(281a8cee-87f9-49e7-8106-a9d76603a806).html.

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We develop machine learning approaches to predict context specific enhancer-promoter interactions using evidence from changes in genomic protein occupancy over time. Occupancy of estrogen receptor alpha (ER-alpha), RNA polymerase (Pol II) and histone marks H2AZ and H3K4me3 were measured over time using ChIP-Seq experiments in MCF7 cells stimulated with estrogen. Two Bayesian classifiers were developed, unsupervised and supervised. The supervised approach uses the correlation of temporal binding patterns at enhancers and promoters and genomic proximity as features and predicts interactions. The
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"A functional study of the orphan nuclear receptor estrogen-related receptor alpha in advanced growth of prostate cancer: 孤兒受體ERRα在前列腺癌中惡性增殖的功能研究". 2014. http://repository.lib.cuhk.edu.hk/en/item/cuhk-1291462.

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Background and aims of the study. Prostate cancer (PCa) is one of the most common hormone-dependent cancers in men in Western and also Asian countries. The standard treatment options for localized PCa include surgery and androgen-deprivation therapy (ADT). However, most patients upon ADT therapy invariably relapse and progress to a more aggressive and metastatic stage termed as castration-resistant PCa (CRPC). Accumulating studies indicate that androgen receptor (AR) transcriptional activity is dysregulated during the advanced progression of CRPC. One important mechanism responsible for the gr
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"Expression of G protein-coupled estrogen receptor (GPER) and its effects on P2Y receptor-mediated Ca²⁺ signalling and cytokine secretion in human bronchial epithelia." 2014. http://repository.lib.cuhk.edu.hk/en/item/cuhk-1290692.

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The airway epithelium plays a central role in respiratory physiology through its transport and immunological functions. Our previous study suggested that P2Y receptors are expressed in airway epithelia and play a significant role in regulating transepithelial ion transport. P2Y receptors belong to the family of purinergic receptors, which can be stimulated by nucleotides such as UTP and UDP. P2Y receptors are G protein-coupled receptors and classically signal through G[subscript q], resulting in an increase in intracellular Ca²⁺ concentration ([Ca²⁺]ᵢ) and thereby in the activation of Ca²⁺-dep
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"The role of estrogen receptors in mechanical signal transduction during osteoporotic fracture healing augmented by mechanical stimulation." 2014. http://repository.lib.cuhk.edu.hk/en/item/cuhk-1292088.

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Chow, Kwoon Ho Simon.<br>Thesis Ph.D. Chinese University of Hong Kong 2014.<br>Includes bibliographical references (leaves 108-115).<br>Abstracts also in Chinese.<br>Title from PDF title page (viewed on 21, December, 2016).
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Books on the topic "Estrogen receptor. eng"

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Test No. 457: BG1Luc Estrogen Receptor Transactivation Test Method for Identifying Estrogen Receptor Agonists and Antagonists. OECD, 2012. http://dx.doi.org/10.1787/9789264185395-en.

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Test No. 455: The Stably Transfected Human Estrogen Receptor-alpha Transcriptional Activation Assay for Detection of Estrogenic Agonist-Activity of Chemicals. OECD, 2009. http://dx.doi.org/10.1787/9789264076372-en.

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Test No. 455: Performance-Based Test Guideline for Stably Transfected Transactivation In Vitro Assays to Detect Estrogen Receptor Agonists. OECD, 2012. http://dx.doi.org/10.1787/9789264185388-en.

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Test No. 455: Performance-Based Test Guideline for Stably Transfected Transactivation In Vitro Assays to Detect Estrogen Receptor Agonists and Antagonists. OECD, 2016. http://dx.doi.org/10.1787/9789264265295-en.

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Test No. 455: Performance-Based Test Guideline for Stably Transfected Transactivation In Vitro Assays to Detect Estrogen Receptor Agonists and Antagonists. OECD, 2015. http://dx.doi.org/10.1787/9789264243040-en.

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Test No. 250: EASZY assay - Detection of Endocrine Active Substances, acting through estrogen receptors, using transgenic tg(cyp19a1b:GFP) Zebrafish embrYos. OECD, 2021. http://dx.doi.org/10.1787/0a39b48b-en.

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Test No. 493: Performance-Based Test Guideline for Human Recombinant Estrogen Receptor (hrER) In Vitro Assays to Detect Chemicals with ER Binding Affinity. OECD, 2015. http://dx.doi.org/10.1787/9789264242623-en.

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Conference papers on the topic "Estrogen receptor. eng"

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Davies, Julia, Deyarina Gonzalez, and R. Steven Conlan. "Abstract 5504: Regulation of the receptor for advanced glycation end products by estrogen receptor ligands in endometrial cancer." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-5504.

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