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Journal articles on the topic 'Estrogenization'

Author: Grafiati

Published: 4 June 2021

Last updated: 15 February 2022

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1

Santti, Risto, Sari Mäkelä, Liisa Pylkkänen, Retha R. Newbold, and John A. McLachlan. "Developmental estrogenization and prostatic neoplasia." Prostate 24, no. 2 (February 1994): 67–78. http://dx.doi.org/10.1002/pros.2990240204.

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2

LONGHURST, PENELOPE A. "In Vitro Rat Bladder Function After Neonatal Estrogenization." Journal of Urology 168, no. 6 (December 2002): 2695–99. http://dx.doi.org/10.1016/s0022-5347(05)64246-2.

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3

Zurawa-Janicka, Dorota, Jaroslaw Kobiela, Tomasz Stefaniak, Agnieszka Wozniak, Joanna Narkiewicz, Michał Wozniak, Janusz Limon, and Barbara Lipinska. "Changes in expression of serine proteases HtrA1 and HtrA2 during estrogen-induced oxidative stress and nephrocarcinogenesis in male Syrian hamster." Acta Biochimica Polonica 55, no. 1 (January 30, 2008): 9–20. http://dx.doi.org/10.18388/abp.2008_3123.

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Serine proteases HtrA1 and HtrA2 are involved in cellular stress response and development of several diseases, including cancer. Our aim was to examine the involvement of the HtrA proteins in acute oxidative stress response induced in hamster kidney by estrogen treatment, and in nephrocarcinogenesis caused by prolonged estrogenization of male Syrian hamster. We used semi-quantitative RT-PCR to estimate the HtrA1 and HtrA2 mRNA levels in kidney tissues, and Western blotting to monitor the amount of the HtrA proteins. Within the first five hours following estrogen administration both HtrA1 mRNA

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4

Saffarini, Camelia M., Elizabeth V. McDonnell-Clark, Ali Amin, and Kim Boekelheide. "A Human Fetal Prostate Xenograft Model of Developmental Estrogenization." International Journal of Toxicology 34, no. 2 (January 29, 2015): 119–28. http://dx.doi.org/10.1177/1091581815569364.

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Prostate cancer is a common disease in older men. Rodent models have demonstrated that an early and later-life exposure to estrogen can lead to cancerous lesions and implicated hormonal dysregulation as an avenue for developing future prostate neoplasia. This study utilizes a human fetal prostate xenograft model to study the role of estrogen in the progression of human disease. Histopathological lesions were assessed in 7-, 30-, 90-, 200-, and 400-day human prostate xenografts. Gene expression for cell cycle, tumor suppressors, and apoptosis-related genes (ie, CDKN1A, CASP9, ESR2, PTEN, and TP

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5

Kopylova, I. V., and M. A. Kareva. "Risk factors of impaired fertility in the patients presenting with congenital adrenal cortical hyperplasia." Problems of Endocrinology 59, no. 3 (June 15, 2013): 51–56. http://dx.doi.org/10.14341/probl201359351-56.

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The analysis of publications concerning the impairment of fertility in the patients presenting with congenital adrenal cortical hyperplasia was focused on the reduction of the degree of estrogenization. It included the data on the course of sexual development and the outcomes of feminizing plastic procedures in the girls suffering this disease depending on the degree of its compensation and the date of onset of the treatment.

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Lehtimaki, Jyrki, Sari Makela, Jaakko Viljamaa, Ahmed Yagi, Jorma Paranko, and Risto Santti. "Neonatal Estrogenization of the Male Mouse Results in Urethral Dysfunction." Journal of Urology 156, no. 6 (December 1996): 2098–103. http://dx.doi.org/10.1016/s0022-5347(01)65443-0.

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7

Aceitero, J., F. Gaytan, and F. B. Ranz. "Effects of neonatal estrogenization on rat bone development: A histomorphometric study." Calcified Tissue International 40, no. 4 (July 1987): 189–93. http://dx.doi.org/10.1007/bf02556620.

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8

MORALES-MONTOR, J., I. ARRIETA, L. I. DEL CASTILLO, M. RODRÍGUEZ-DORANTES, M. A. CERBÓN, and C. LARRALDE. "Remote sensing of intraperitoneal parasitism by the host's brain: regional changes of c-fos gene expression in the brain of feminized cysticercotic male mice." Parasitology 128, no. 3 (March 2004): 343–51. http://dx.doi.org/10.1017/s0031182003004529.

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Experimental intraperitoneal Taenia crassiceps cysticercosis in mice exhibits distinct genetical, immunological and endocrinological features possibly resulting from the complex interactive network of their physiological systems. Very notable is the tendency of parasites to grow faster in hosts of the female sex. It is also remarkable in the feminization process that the infection induces in chronically infected male mice, characterized by their estrogenization, deandrogenization and loss of sexual and aggressive patterns of behaviour. The proto-oncogene c-fos is a sex steroid-regulated transc

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9

Prins, Gail S. "Developmental estrogenization: Prostate gland reprogramming leads to increased disease risk with aging." Differentiation 118 (March 2021): 72–81. http://dx.doi.org/10.1016/j.diff.2020.12.001.

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10

Soto, Pedro, Hilda María Echevarría, Cristina Esther Monteavaro, and María del Carmen Catena. "Experimentally induced intravaginal Tritrichomonas foetus infection in a mouse model." Pesquisa Veterinária Brasileira 25, no. 4 (December 2005): 225–30. http://dx.doi.org/10.1590/s0100-736x2005000400007.

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The interest to develop research on the host-parasite relationship in bovine tritrichomonosis has accomplished the use of experimental models alternative to cattle. The BALB/c mouse became the most appropriate species susceptible to vaginal Tritrichomonas foetus infection requiring previous estrogenization. For the need of an experimental model without persistent estrogenization and with normal estrous cycles, the establishment and persistence of vaginal infection on BALB/c mouse with different concentrations of T. foetus in two experimental groups was evaluated. Group A was treated with 5mg o

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11

Lindau, Stacy Tessler, Annie Dude, Natalia Gavrilova, Joscelyn N. Hoffmann, L. Philip Schumm, and Martha K. McClintock. "Prevalence and correlates of vaginal estrogenization in postmenopausal women in the United States." Menopause 24, no. 5 (May 2017): 536–45. http://dx.doi.org/10.1097/gme.0000000000000787.

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12

Masui, Fujiko, Keiko Kurosaki, Takao Mori, and Manabu Matsuda. "Persistent trefoil factor 1 expression imprinted on mouse vaginal epithelium by neonatal estrogenization." Cell and Tissue Research 323, no. 1 (August 30, 2005): 167–75. http://dx.doi.org/10.1007/s00441-005-0049-4.

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13

Cabral, C., L. Correa, F. Sampaio, and L. Cardoso. "298 Compositional changes in vesical extracellular matrix in male rats after neonatal estrogenization with diethylstilbestrol." European Urology Supplements 3, no. 2 (February 2004): 77. http://dx.doi.org/10.1016/s1569-9056(04)90297-7.

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14

Masui, Fujiko, Manabu Matsuda, and Takao Mori. "Involvement of keratinocyte growth factor (KGF)-KGF receptor signaling in developmental estrogenization syndrome of mouse vagina." Cell and Tissue Research 318, no. 3 (October 5, 2004): 591–98. http://dx.doi.org/10.1007/s00441-004-0980-9.

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15

Tena-Sempere, M., J. Navarro, L. Pinilla, LC Gonzalez, I. Huhtaniemi, and E. Aguilar. "Neonatal exposure to estrogen differentially alters estrogen receptor alpha and beta mRNA expression in rat testis during postnatal development." Journal of Endocrinology 165, no. 2 (May 1, 2000): 345–57. http://dx.doi.org/10.1677/joe.0.1650345.

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The biological actions of estrogens on target cells are mediated by two nuclear receptors: the estrogen receptor (ER) alpha and the recently characterized ER beta. In the male rat, the physiological role of estrogens involves multiple actions, from masculinization of brain areas related to reproductive function and sexual behavior to regulation of testicular development and function. Paradoxically, however, administration of high doses of estrogen during the critical period of neonatal differentiation results in an array of defects in the reproductive axis that permanently disrupt male fertili

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16

Matsuda, Manabu, Fujiko Masui, and Takao Mori. "Neonatal estrogenization leads to increased expression of cellular retinol binding protein 2 in the mouse reproductive tract." Cell and Tissue Research 316, no. 1 (April 1, 2004): 131–39. http://dx.doi.org/10.1007/s00441-004-0852-3.

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17

Pinilla, L., M. Cocconi, S. Zoppi, and L. Martini. "Effect of neonatal estrogenization on testosterone metabolism in the prostate and in the epididymis of the rat." Journal of Steroid Biochemistry 32, no. 3 (March 1989): 459–65. http://dx.doi.org/10.1016/0022-4731(89)90222-7.

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18

Watanobe, Hajime, Shinsuke Sasaki, and Kazuo Takebe. "A comparative study of the effects of neonatal androgenization and estrogenization on prolactin secretion in adult female rats." Regulatory Peptides 34, no. 3 (July 1991): 149–58. http://dx.doi.org/10.1016/0167-0115(91)90174-f.

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19

Luo, Cheng, Leena Strauss, Ari Ristimäki, Tomi Streng, and Risto Santti. "Constant Expression of Cyclooxygenase-2 Gene in Prostate and the Lower Urinary Tract of Estrogen-Treated Male Rats." Zeitschrift für Naturforschung C 56, no. 5-6 (June 1, 2001): 455–63. http://dx.doi.org/10.1515/znc-2001-5-621.

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Expression of cyclooxygenase-2 (E. C. 1.14.99.1) in prostate and the lower urinary tract (LUT) of the neonatally estrogenized male rat has been studied by using a COX-2 ’s PCR fragment of 724 nt spanning 3 introns and a 478nt internal standard for quantitative RTPCR. The same fragment of 724 nt was used for RNA probe in Northern hybridization. Neonatal estrogenization (10 μg/day of diethylstilbestrol on days 1-5) had no effect on COX -2 expression in prostatic urethra, prostatic lobes, or bladder. Acute estrogen treatment of castrated animals did not induce COX-2 expression, either. In additio

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20

Velichko, N. F., N. O. Karpenko, E. M. Koreneva, E. E. Chistyakova, N. P. Smolenko, and V. О. Bondarenko. "THE EFFECT OF STRESS AND PHYTOESTROGENIZATION IN THE NEONATAL PERIOD ON THE FERTILITY OF ADULT MALE RATS." Fiziolohichnyĭ zhurnal 66, no. 4 (August 20, 2020): 37–45. http://dx.doi.org/10.15407/fz66.04.037.

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In the paper the significance of milk-feeding period for the formation of male reproductive health is experimentally shown. It has been found that the use of emotional stress and excessive phytoestrogenization, both separately and together, in male rats during breastfeeding leads to certain disorders of the reproductive system in adulthood. Modeled emotional stress or phytoestergenization on the principle of imprinting caused hyperostrogeny, androgen deficiency, changes in the ratio of androgenization/estrogenization in adulthood. The applied factors led to impaired spermatogenesis, inhibition

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21

Khan, Wahid Ali, Arshi Malik, and Mohd Wajid Ali Khan. "Estrogenization of insulin by catecholestrogen produced high affinity autoantibodies and altered the normal function of insulin in type 1 diabetes." Life Sciences 256 (September 2020): 117910. http://dx.doi.org/10.1016/j.lfs.2020.117910.

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22

Ruhlen, Rachel L., Kembra L. Howdeshell, Jiude Mao, Julia A. Taylor, Franklin H. Bronson, Retha R. Newbold, Wade V. Welshons, and Frederick S. vom Saal. "Low Phytoestrogen Levels in Feed Increase Fetal Serum Estradiol Resulting in the “Fetal Estrogenization Syndrome” and Obesity in CD-1 Mice." Environmental Health Perspectives 116, no. 3 (March 2008): 322–28. http://dx.doi.org/10.1289/ehp.10448.

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23

Andel, R. A. Van, C. L. Franklin, M. C. St Claire, L. K. Riley, C. L. Besch-Williford, and R. R. Hook. "Lesions of Experimental Genital Tritrichomonas foetus Infections in Estrogenized BALB/c Mice." Veterinary Pathology 33, no. 4 (July 1996): 407–11. http://dx.doi.org/10.1177/030098589603300406.

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Ninety-seven BALB/c mice were inoculated intravaginally with 8.0 X 105 Tritrichomonas foetus organisms, using either isolate ATCC 30003 or field isolate MU Y22 2 days after estrogenization with 15 μg 17β-estradiol. Reproductive tracts were examined at several time points post-inoculation to determine gross and histologic responses to trichomonad infection as compared to estrogenized, uninfected control animals. The two isolates varied greatly in ability to maintain chronic infection; no ATCC 30003-inoculated animals remained culture-positive beyond 7 weeks post-inoculation, whereas MU Y22-inoc

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24

Asirvatham, Adlyne Reena, Karthik Balachandran, Packiamary Jerome, Vettriselvi Venkatesan, Teena Koshy та Shriraam Mahadevan. "Clinical, biochemical and genetic characteristics of children with congenital adrenal hyperplasia due to 17α-hydroxylase deficiency". Journal of Pediatric Endocrinology and Metabolism 33, № 8 (27 серпня 2020): 1051–56. http://dx.doi.org/10.1515/jpem-2020-0050.

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AbstractObjectivesCongenital adrenal hyperplasia (CAH) is an autosomal recessive disorder, that could rarely be due to 17 α-hydroxylase deficiency (17αOHD) and/or 17,20 lyase deficiency. Mutation of CYP17A1 gene causes deficiency of glucocorticoids and androgens but excess of mineralocorticoids. Lack of genital ambiguity in most children causes a delay in diagnosis even until puberty. Classical presentation with hypertension and hypokalemia is often not encountered. We intended to study the clinical, biochemical and genetic characteristics of children diagnosed with CAH due to 17αOHD.MethodsTh

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25

Collado, Dolores, and Enrique Aguilar. "Further evidence that prolactin secretion in adult female rats is differently modified after neonatal estrogenization or androgenization: Responses to methysergide, quipazine, and pizotifen." Physiology & Behavior 53, no. 1 (January 1993): 161–65. http://dx.doi.org/10.1016/0031-9384(93)90025-b.

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26

Batubara, Jose R. L., Adji Suranto, Sudigdo Sastroasmoro, Bambang Tridjaja, and Aman B. Pulungan. "Natural history of premature thelarche: review of 60 girls." Paediatrica Indonesiana 41, no. 6 (December 31, 2001): 279. http://dx.doi.org/10.14238/pi41.6.2001.279-83.

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In Indonesia report on the natural history of premature thelarche is very limited. Daily practice requires physicians to have some basic practical knowledge, among others the natural history of premature thelarche, in order to manage these patients properly. We reviewed data of 85 premature thelarche patients who visited our department from January 1989 until December 1998. Only 60 patients met the study criteria. The mean chronological age of the patients at diagnosis was 43.4 months. About half of these patients (31/60) were diagnosed before they were 2 years old. Half of the patients had bi

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27

Pisera, D., M. Candolfi, S. Navarra, J. Ferraris, V. Zaldivar, G. Jaita, M. G. Castro, and A. Seilicovich. "Estrogens sensitize anterior pituitary gland to apoptosis." American Journal of Physiology-Endocrinology and Metabolism 287, no. 4 (October 2004): E767—E771. http://dx.doi.org/10.1152/ajpendo.00052.2004.

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Tissue homeostasis results from a balance between cell proliferation and cell death by apoptosis. Estradiol affects proliferation as well as apoptosis in hormone-dependent tissues. In the present study, we investigated the apoptotic response of the anterior pituitary gland to lipopolysaccharide (LPS) in cycling female rats, and the influence of estradiol in this response in ovariectomized (OVX) rats. The OVX rats were chronically estrogenized with implanted Silastic capsules containing 1 mg of 17β-estradiol (E2). Cycling or OVX and E2-treated rats were injected with LPS (250 μg/rat ip). Apopto

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28

Watanobe, Hajime, and Kazuo Takebe. "A Comparative Study of the Effects of Neonatal Androgenization and Estrogenization on Vasoactive Intestinal Peptide Levels in the Anterior Pituitary and the Hypothalamus of Adult Female Rats." Neuroendocrinology 56, no. 5 (1992): 653–59. http://dx.doi.org/10.1159/000126289.

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29

Kopylova, I. V., V. Yu Sysoeva, T. M. Glybina, and M. A. Kareva. "Expression of estrogen and androgen receptors in tissues of external genitalia of girls with congenital adrenal hyperplasia." Problems of Endocrinology 60, no. 6 (December 15, 2014): 14–20. http://dx.doi.org/10.14341/probl201460614-20.

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The reason of post-operative introital stenosis in girls with CAH is a poor estrogenization of external genitalia before vaginoplasty. It is possible that the local sensitivity to estrogens can be reduced due to prenatal androgen excess in addition to inadequate compensation, which results to decrease of estrogens production by the ovaries. Objective. To determine the distribution of estrogen and androgen receptors in genital tissue, depending on the form of CAH, the degree of external virilization and serum levels of androgens. Material and methods. The waste surgical tissues obtained during

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30

Ruiz-Pino, F., V. M. Navarro, A. H. Bentsen, D. Garcia-Galiano, M. A. Sanchez-Garrido, P. Ciofi, R. A. Steiner, J. D. Mikkelsen, L. Pinilla, and M. Tena-Sempere. "Neurokinin B and the Control of the Gonadotropic Axis in the Rat: Developmental Changes, Sexual Dimorphism, and Regulation by Gonadal Steroids." Endocrinology 153, no. 10 (October 1, 2012): 4818–29. http://dx.doi.org/10.1210/en.2012-1287.

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Abstract Neurokinin B (NKB), encoded by Tac2 in rodents, and its receptor, NK3R, have recently emerged as important regulators of reproduction; NKB has been proposed to stimulate kisspeptin output onto GnRH neurons. Accordingly, NKB has been shown to induce gonadotropin release in several species; yet, null or even inhibitory effects of NKB have been also reported. The basis for these discrepant findings, as well as other key aspects of NKB function, remains unknown. We report here that in the rat, LH responses to the NK3R agonist, senktide, display a salient sexual dimorphism, with persistent

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31

Freyberger, A., Peter Andrews, Elke Hartmann, Rolf Eiben, Ingo Loof, U. Schmidt, M. Temerowski, et al. "Testing of endocrine active substances using an enhanced OECD test guideline 407: Experiences from studies on flutamide and ethinylestradiol." Pure and Applied Chemistry 75, no. 11-12 (January 1, 2003): 2483–89. http://dx.doi.org/10.1351/pac200375112483.

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Groups of five male and five female Wistar rats were treated by gavage with 0, 1, 10, and 100 mg/kg body weight (b.w.) flutamide (FLU) or 0.01, 0.05, and 0.2 mg/kg b.w. of ethinylestradiol (EE2) for at least 28 days according to an enhanced Organization for Economic Cooperation and Development (OECD) test guideline (TG) 407 to investigate which of the current and/or additional parameters would detect effects on the endocrine system and to provide data on intralaboratory variability. Two identical studies were performed in parallel on each compound. Common enhancements were determination of thy

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32

Vlasenko, S., O. Zhulinska, and O. Yeroshenko. "Clinical and laboratory prognostic indicators for fertility in sheep." Naukovij vìsnik veterinarnoï medicini, no. 1(149) (May 30, 2019): 6–14. http://dx.doi.org/10.33245/2310-4902-2019-149-1-6-14.

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With the use of technology of artificial insemination in sheep farms are not yet defined criteria for assessing the full value of the preparation of females for insemination, and hence – the possibility of prediction and correction of their fertilization, which prevents the rational use of cryopreserved semen and ensuring the maximum reception of the offspring. We have proved the prognostic importance of fertilization of the morphofunctional state of the vulva and the vagina and the quality of mucus in sheep breeds during estrus. The material of the study was 327 sheep of ascanian breeding, wh

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33

Prins, Gail S., Lynn Birch, Helga Habermann, William Y. Chang, Christopher Tebeau, Oliver Putz, and Charles Bieberich. "Influence of neonatal estrogens on rat prostate development." Reproduction, Fertility and Development 13, no. 4 (2001): 241. http://dx.doi.org/10.1071/rd00107.

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Brief exposure of rodents to estrogens during early development alters prostate branching morphogenesis and cellular differentiation in a dose-dependant manner. If estrogenic exposures are high, these disturbances lead to permanent imprints of the prostate, which include reduced growth, differentiation defects of the epithelial cells, altered secretory function and reduced responsiveness to androgens in adulthood. This process, referred to as neonatal imprinting or developmental estrogenization, is associated with an increased incidence of prostatic lesions with aging, which include hyperplasi

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34

Prins, Gail S., William Y. Chang, Yan Wang, and Richard B. van Breemen. "Retinoic Acid Receptors and Retinoids Are Up-Regulated in the Developing and Adult Rat Prostate by Neonatal Estrogen Exposure." Endocrinology 143, no. 9 (September 1, 2002): 3628–40. http://dx.doi.org/10.1210/en.2002-220184.

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Abstract Exposure to estrogens during the neonatal period interrupts rat prostatic development by reducing branching morphogenesis and by blocking epithelial cells from entering a normal differentiation pathway. Upon aging, ventral prostates exhibit extensive hyperplasia, dysplasia, and massive lymphocytic infiltrate, suggesting that neonatal estrogens may predispose the prostate gland to precancerous lesions. Vitamin A (retinol) and their derivatives (retinoic acids) are known key developmental regulators that bind and activate retinoic acid receptors (RARs). To evaluate whether neonatal estr

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35

LONGHURST, PENELOPE A. "In Vitro Rat Bladder Function After Neonatal Estrogenization." Journal of Urology, December 2002, 2695–99. http://dx.doi.org/10.1097/00005392-200212000-00104.

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36

Lehtimaki, Jyrki, Sari Makela, Jaakko Viljamaa, Ahmed Yagi, Jorma Paranko, and Risto Santti. "Neonatal Estrogenization of the Male Mouse Results in Urethral Dysfunction." Journal of Urology, December 1996, 2098–103. http://dx.doi.org/10.1097/00005392-199612000-00057.

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37

Prins, Gail S. "Developmental estrogenization: Prostate gland reprogramming leads to increased disease risk with aging." Differentiation, January 2021. http://dx.doi.org/10.1016/j.diff.2020.12.001.

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38

Athonvarangkul, Diana, Hillary Wyeth Hosier, Brian Wojeck, and Silvio E. Inzucchi. "SAT-258 Surprising Transformation of a Microprolactinoma to a Macroprolactinoma." Journal of the Endocrine Society 4, Supplement_1 (April 2020). http://dx.doi.org/10.1210/jendso/bvaa046.1057.

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Abstract Background: Microprolactinomas are typically benign tumors that rarely become macroprolactinomas. We present a rare case of a microprolactinoma that, after discontinuation of dopamine agonist (DA) therapy, transformed into a macroprolactinoma over a period of 6 years.Clinical Case: A 16-year-old woman initially presented for evaluation of galactorrhea without menstrual irregularities and was found to have elevated prolactin (68 ng/ml, normal range: 0-20), and a 4 mm pituitary microadenoma on MRI imaging. The patient was otherwise asymptomatic and other pituitary hormone levels were no

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