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Journal articles on the topic 'Etiology'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Pratama, Bangkit Ary, and Wahyuni. "LITERATURE REVIEW: IDENTIFIKASI PENYEBAB HEPATITIS AKUT TANPA ETIOLOGI PADA ANAK." KOSALA : Jurnal Ilmu Kesehatan 10, no. 2 (2022): 63–75. http://dx.doi.org/10.37831/kjik.v10i2.233.

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Kasus hepatitis akut tanpa etiologi yang menyerang anak-anak telah dikaitkan dengan beberapa kemungkinan etiologi termasuk penyakit coronavirus 2019 (COVID-19), vaksinasi COVID-19, agen infeksi baru, racun, dan kemungkinan etiologi terkait makanan. Masih perlu banyak penelitian yang dilakukan untuk secara tepat mengidentifikasi agen penyebab hepatitis akut tanpa etiologi. Penelitian ini bertujuan untuk mengidentifikasi kemungkinan penyebab hepatitis akut tanpa etiologi pada anak. Penelitian ini menggunakan kajian literatur (literature review). Sumber data pada penelitian ini adalah jurnal ilmi

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2

White, Keith C. "Etiology." Clinical Obstetrics and Gynecology 31, no. 1 (1988): 141–52. http://dx.doi.org/10.1097/00003081-198803000-00017.

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3

Crowther, Mark, and Jeffrey Weitz. "ETIOLOGY." Evidence-based Cardiovascular Medicine 3, no. 2 (1999): 34–35. http://dx.doi.org/10.1054/ebcm.1999.0207.

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4

Field, Teresa. "ETIOLOGY." Evidence-based Oncology 1, no. 2 (2000): 44–45. http://dx.doi.org/10.1054/ebon.2000.0021.

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5

White, John W. "Etiology." Journal of Burn Care & Rehabilitation 7, no. 5 (1986): 446. http://dx.doi.org/10.1097/00004630-198609000-00037.

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6

Morgan, Rachel. "Etiology." Prairie Schooner 91, no. 2 (2017): 116. http://dx.doi.org/10.1353/psg.2017.0028.

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7

Alvarez, Vincent, M. Brandon Westover, Frank W. Drislane, et al. "Evaluation of a clinical tool for early etiology identification in status epilepticus." Epilepsia 55, no. 12 (2014): 2059–68. https://doi.org/10.1111/epi.12852.

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OBJECTIVES: Because early etiologic identification is critical to select appropriate specific status epilepticus (SE) management, we aim to validate a clinical tool we developed that uses history and readily available investigations to guide prompt etiologic assessment. METHODS: This prospective multicenter study included all adult patients treated for SE of all but anoxic causes from four academic centers. The proposed tool is designed as a checklist covering frequent precipitating factors for SE. The study team completed the checklist at the time the patient was identified by electroencephal

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8

Dr. Nupur Sah, Dr Nupur Sah, Dr Hemant Bhutani, and Dr Priyadarshini Shetty. "Halitosis – Etiology and Diagnosis." Indian Journal of Applied Research 3, no. 11 (2011): 352–54. http://dx.doi.org/10.15373/2249555x/nov2013/109.

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9

Vaish, Sunidhi, Sunil K Pollipalli, and Gurumurthy DM. "Genetic Etiology of Stillbirths." International Journal of Science and Research (IJSR) 13, no. 5 (2024): 617–22. http://dx.doi.org/10.21275/sr24508190240.

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10

Wahlgren, H., W. Mortensson, M. Eriksson, Y. Finkel, M. Forsgren, and M. Leinonen. "Radiological findings in children with acute pneumonia: age more important than infectious agent." Acta Radiologica 46, no. 4 (2005): 431–36. http://dx.doi.org/10.1080/02841850510021238.

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Purpose: To evaluate whether radiological findings and healing time in children with pneumonia are correlated to etiologic agent. Material and Methods: A total of 346 children with radiologically verified acute pneumonia, and with accomplished serological tests for bacteria and viruses, were included in the study. Five etiological groups were analysed: children with bacterial etiology only, with viral etiology only, with mixed bacterial and viral etiology, with Mycoplasma only, and children with no etiology. Results: The chest films of each etiological group were analysed and the findings were

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11

Nagai, Atsushi, and Kazutetsu Aoshiba. "2. Etiology." Nihon Naika Gakkai Zasshi 97, no. 6 (2008): 1177–83. http://dx.doi.org/10.2169/naika.97.1177.

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12

Davidson, Joseph. "Bacterial Etiology." Journal of the American Dental Association 122, no. 1 (1991): 28. http://dx.doi.org/10.14219/jada.archive.1991.0013.

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13

Gillison, M. "Viral etiology." Radiotherapy and Oncology 82 (February 2007): S4. http://dx.doi.org/10.1016/s0167-8140(07)80017-x.

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14

Chen, Cynthia, Neil Kaushal, David M. Scher, Shevaun M. Doyle, John S. Blanco, and Emily R. Dodwell. "Clubfoot Etiology." Journal of Pediatric Orthopaedics 38, no. 8 (2018): e462-e469. http://dx.doi.org/10.1097/bpo.0000000000001191.

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15

Kugay, Alexander. "Terrorism Etiology." Administrative Consulting 97, no. 3 (2017): 16–27. https://doi.org/10.5281/zenodo.14965170.

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Terrorist acts are preceded by processes of political radicalization, which, functionally, is an active preparation for the confrontation with the authorities. Descriptive, radicalization is the change of feelings, beliefs, and behaviors that justifies violence. The article discusses the radicalization of behavior that takes place on an individual and group level on the bases: personal grievances, ideological factors, adherence to the radical group on the basis of love, step of joining a radical group of like-minded group of extreme cohesion in conditions of isola

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16

Poedjiastoeti, Wiwiek. "Komunikasi oroantral: etiologi dan penatalaksanaannya Oroantral communication: the etiology and management." Journal of Dentomaxillofacial Science 10, no. 2 (2011): 116. http://dx.doi.org/10.15562/jdmfs.v10i2.267.

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Oroantral communication (OAC)may occur when upper posterior teeth are removed, and occasionally, as a resultof trauma. This sinus perforation formed particularly when a maxillary molar with widely divergent roots adjacentto edentulous spaces is extracted. In this instance the sinus is likely to be pneumatized into the edentulous alveolarprocessus surrounding the tooth, which weakens the entire alveolus and brings the tooth apices into a closerrelationship with the sinus cavity. In order to avoid OAC, preoperative radiograph is needed. When perforation, ifsinus opening is small and disease free

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17

De Araújo, Daniel Alencar, Denise Teixeira Santos, Larisse Yara De Carvalho, and Daniela Moura Parente. "Etiologia genética do aborto recorrente / Genetic etiology of recurrent abortion." Brazilian Journal of Health Review 4, no. 6 (2021): 25673–77. http://dx.doi.org/10.34119/bjhrv4n6-166.

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18

Uçan Tokuç, Firdevs Ezgi, Emine Görgülü, Fatma Genç, Meltem Korucuk, Abidin Erdal, and Yasemin Biçer Gömceli. "Etiology and Mortality of Nonconvulsive Status Epilepticus." Neurology International 17, no. 2 (2025): 29. https://doi.org/10.3390/neurolint17020029.

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Objectives: Nonconvulsive status epilepticus (NCSE) is a disease with a high mortality rate and a very diverse etiology. The disease prognosis is related to the etiology. We aimed to investigate the etiology, mortality rates, and factors affecting mortality in patients diagnosed with NCSE in a tertiary epilepsy center in Turkey. Methods: All electroencephalograms (EEGs) were taken in the EEG laboratory of the Department of Neurology, Antalya Training and Research Hospital, between June 2021 and February 2024. Patients who met the Salzburg Consensus Criteria (SCC) for NCSE were included. Demogr

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19

Lawren, Wirginia Pereira Dantas, and José Wyllon Batista Ricardo Hermínio. "PERFIL EPIDEMIOLÓGICO DE MENINGITE EM BARREIRAS-BAHIA ENTRE OS ANOS DE 2007 A 2021: MAIOR ACOMETIMENTO NA INFÂNCIA." Revistaft 27, no. 124 (2023): 31. https://doi.org/10.5281/zenodo.8115231.

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<strong>Objetivo: </strong>Definir o perfil epidemiológico dos pacientes internados com meningite na  cidade de Barreiras-Bahia entre os anos 2007 e 2021. <strong>Métodos: </strong>Trata-se de um  estudo epidemiológico, não experimental, de caráter descritivo-analítico, tipo coorte  transversal, com método quantitativo em tempo retrospectivo em dados que compõem  o Sistema de Informação de Agravos de Notificação (SINAN). <strong>Resultados: </strong>Em  Barreira

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20

Comer, James A., William L. Nicholson, John W. Sumner, James G. Olson, and James E. Childs. "Diagnosis of Human Ehrlichiosis by PCR Assay of Acute-Phase Serum." Journal of Clinical Microbiology 37, no. 1 (1999): 31–34. http://dx.doi.org/10.1128/jcm.37.1.31-34.1999.

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A PCR assay of 43 acute-phase serum samples was evaluated as a method for early detection of human granulocytic ehrlichiosis (HGE) and determination of etiology when serologic testing is inconclusive. Sequence-confirmed products of the HGE agent were amplified from three individuals residing or having exposure history in Minnesota or Wisconsin, and similarly confirmed products from Ehrlichia chaffeensis were amplified from three individuals from Florida or Maryland. Etiology, as determined by PCR and serology, was the same whenever there was a fourfold difference between the maximum titers of

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21

Berdysh, D. S., V. I. Shaposhnikov, and M. A. Maksimenko. "ETIOLOGY DEVELOPMENT VARICOTSEL." EurasianUnionScientists 4, no. 63 (2019): 6–8. http://dx.doi.org/10.31618/esu.2413-9335.2019.4.63.207.

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22

Petersen, Hermod, and Mikko Salaspuro. "Etiology and pathophysiology." Scandinavian Journal of Gastroenterology 20, sup109 (1985): 49. http://dx.doi.org/10.3109/00365528509103936.

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23

Ohyama, M. "Etiology and Pathology." Nihon Kikan Shokudoka Gakkai Kaiho 48, no. 2 (1997): 138–39. http://dx.doi.org/10.2468/jbes.48.138.

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24

Rubin, Harry. "Etiology of AIDS." Science 240, no. 4858 (1988): 1389–90. http://dx.doi.org/10.1126/science.240.4858.1389.d.

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25

Nazish, Zahra, Muhammad Inayatullah, and Muhammad Younus Khan. "ETIOLOGY OF DYSPHAGIA." Professional Medical Journal 23, no. 09 (2016): 1039–44. http://dx.doi.org/10.29309/tpmj/2016.23.09.1692.

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Objectives: To determine the etiology of dysphagia based on upper GI endoscopyin Nishtar Hospital Multan. Study design: Retrospective study. Place and Duration of study:This study was conducted at gastroenterology unit of Nishtar Hospital Multan from Feb 2013 toAugust 2014. Patients and methods: Three hundred and twenty three patients, ≥ 13 years old,who presented with history of dysphagia to the gastroenterology unit of Nishtar Hospital Multan.Results: Out of 323 patients, 43.7% were males and 56.3% were females. Mean age of patientswas 44.37±17.395 years. Most common finding was benign stric

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26

Metzger, Deborah A., and A. F. Haney. "Etiology of Endometriosis." Obstetrics and Gynecology Clinics of North America 16, no. 1 (1989): 1–14. http://dx.doi.org/10.1016/s0889-8545(21)00134-0.

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27

Isbit, J. "Etiology of Diverticulosis." North Carolina Medical Journal 77, no. 4 (2016): 303. http://dx.doi.org/10.18043/ncm.77.4.303.

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28

Rubin, H. "Etiology of AIDS." Science 240, no. 4858 (1988): 1389–90. http://dx.doi.org/10.1126/science.240.4858.1389-c.

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29

Ahmetagic, Aldina, Fatima Numanovic, Sead Ahmetagic, Lejla RakovacTupkovic, and Humera PorobicJahic. "Etiology of Peritonitis." Medical Archives 67, no. 4 (2013): 278. http://dx.doi.org/10.5455/medarh.2013.67.278-281.

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30

Sullivan, L. P., J. C. Stears, and S. P. Ringel. "Etiology of syringomyelia." Neurology 39, no. 2 (1989): 308. http://dx.doi.org/10.1212/wnl.39.2.308.

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31

Milstein, J. M., and D. M. Olson. "Etiology of syringomyelia." Neurology 39, no. 2 (1989): 308. http://dx.doi.org/10.1212/wnl.39.2.308-a.

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32

Pitts, Susan L. "Etiology of Blindness." Public Library Quarterly 18, no. 3-4 (2000): 81–93. http://dx.doi.org/10.1300/j118v18n03_11.

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33

Grobler, Leon J., Peter A. Robertson, John E. Novotny, and Malcolm H. Pope. "Etiology of Spondylolisthesis." Spine 18, no. 1 (1993): 80–91. http://dx.doi.org/10.1097/00007632-199301000-00013.

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34

Nanay, Bence. "Teleosemantics without Etiology." Philosophy of Science 81, no. 5 (2014): 798–810. http://dx.doi.org/10.1086/677684.

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35

&NA;. "Etiology and pathogenesis." Current Opinion in Rheumatology 3, no. 1 (1991): 32–35. http://dx.doi.org/10.1097/00002281-199102000-00007.

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36

DETORAKIS, ET. "Anatomy & Etiology." Acta Ophthalmologica 89, s248 (2011): 0. http://dx.doi.org/10.1111/j.1755-3768.2011.1311.x.

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37

Rubin, H. "Etiology of AIDS." Science 240, no. 4858 (1988): 1389–90. http://dx.doi.org/10.1126/science.3163849.

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38

Thomas, Rhys, and Philip Smith. "Etiology of Epilepsy." Seminars in Neurology 35, no. 03 (2015): 191–92. http://dx.doi.org/10.1055/s-0035-1552626.

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39

Fuchs, Bruno, and Douglas J. Pritchard. "Etiology of Osteosarcoma." Clinical Orthopaedics and Related Research 397 (April 2002): 40–52. http://dx.doi.org/10.1097/00003086-200204000-00007.

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40

Bhatia, M. S., N. K. Dhar, P. K. Singhal, V. R. Nigam, S. C. Malik, and D. N. Mullick. "Prevalence and Etiology." Clinical Pediatrics 29, no. 6 (1990): 311–15. http://dx.doi.org/10.1177/000992289002900603.

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41

Cammisa, Kathryne M., and Stephen H. Hobbs. "Etiology of Autism." Occupational Therapy in Mental Health 12, no. 2 (1993): 39–67. http://dx.doi.org/10.1300/j004v12n02_03.

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42

Panel, J. P. Mohr, Gregory W. Albers, et al. "Etiology of Stroke." Stroke 28, no. 7 (1997): 1501–6. http://dx.doi.org/10.1161/01.str.28.7.1501.

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43

Tomatsu, Shunji, Shaukat Khan, Hira Peracha, Alfred Wiesbauer, and Tadao Orii. "Etiology of mucopolysaccharidoses." Molecular Genetics and Metabolism 120, no. 1-2 (2017): S132. http://dx.doi.org/10.1016/j.ymgme.2016.11.347.

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44

Sharma, Om, and Darshana Kadakia. "Etiology of Sarcoidosis." Seminars in Respiratory and Critical Care Medicine 8, no. 01 (1986): 95–102. http://dx.doi.org/10.1055/s-2007-1012644.

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45

Meares, E. "Etiology of Prostatitis." Journal of Urology 133, no. 6 (1985): 1126. http://dx.doi.org/10.1016/s0022-5347(17)49394-3.

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46

Edwards, I. Keith. "Endometriosis Etiology Questioned." AORN Journal 49, no. 2 (1989): 478. http://dx.doi.org/10.1016/s0001-2092(07)66666-6.

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47

Redwine, David B. "Endometriosis Etiology Questioned." AORN Journal 49, no. 2 (1989): 478. http://dx.doi.org/10.1016/s0001-2092(07)66667-8.

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48

Kelly, Ellen M. "Modeling stuttering etiology." Journal of Fluency Disorders 25, no. 4 (2000): 359–68. http://dx.doi.org/10.1016/s0094-730x(00)00085-1.

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49

Sanders, Richard J., and Sharon L. Hammond. "Etiology and pathology." Hand Clinics 20, no. 1 (2004): 23–26. http://dx.doi.org/10.1016/s0749-0712(03)00079-9.

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50

Neidner, Kenneth H. "Etiology and pathogenesis." Clinics in Dermatology 6, no. 1 (1988): 123–35. http://dx.doi.org/10.1016/0738-081x(88)90018-1.

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