Academic literature on the topic 'ETV4'
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Journal articles on the topic "ETV4"
Eo, Jinwon, Kyuyong Han, Kenneth M Murphy, Haengseok Song, and Hyunjung Jade Lim. "Etv5, an ETS transcription factor, is expressed in granulosa and cumulus cells and serves as a transcriptional regulator of the cyclooxygenase-2." Journal of Endocrinology 198, no. 2 (May 20, 2008): 281–90. http://dx.doi.org/10.1677/joe-08-0142.
Full textNewton, Kim, Debra L. Dugger, Arundhati Sengupta-Ghosh, Ronald E. Ferrando, Felix Chu, Janet Tao, Wendy Lam, et al. "Ubiquitin ligase COP1 coordinates transcriptional programs that control cell type specification in the developing mouse brain." Proceedings of the National Academy of Sciences 115, no. 44 (October 15, 2018): 11244–49. http://dx.doi.org/10.1073/pnas.1805033115.
Full textDissanayake, Kumara, Rachel Toth, Jamie Blakey, Olof Olsson, David G. Campbell, Alan R. Prescott, and Carol MacKintosh. "ERK/p90RSK/14-3-3 signalling has an impact on expression of PEA3 Ets transcription factors via the transcriptional repressor capicúa." Biochemical Journal 433, no. 3 (January 14, 2011): 515–25. http://dx.doi.org/10.1042/bj20101562.
Full textSchmerr, Martin, Sune Kobberup, Ngai Woo, and Jan Jensen. "Role of Etv4 and Etv5 in pancreatic development." Developmental Biology 356, no. 1 (August 2011): 168. http://dx.doi.org/10.1016/j.ydbio.2011.05.622.
Full textBabal, Yigit Koray, Basak Kandemir, and Isil Aksan Kurnaz. "Gene Regulatory Network of ETS Domain Transcription Factors in Different Stages of Glioma." Journal of Personalized Medicine 11, no. 2 (February 17, 2021): 138. http://dx.doi.org/10.3390/jpm11020138.
Full textCurrie, Simon L., Desmond K. W. Lau, Jedediah J. Doane, Frank G. Whitby, Mark Okon, Lawrence P. McIntosh, and Barbara J. Graves. "Structured and disordered regions cooperatively mediate DNA-binding autoinhibition of ETS factors ETV1, ETV4 and ETV5." Nucleic Acids Research 45, no. 5 (February 6, 2017): 2223–41. http://dx.doi.org/10.1093/nar/gkx068.
Full textCooper, Christopher D. O., Joseph A. Newman, Hazel Aitkenhead, Charles K. Allerston, and Opher Gileadi. "Structures of the Ets Protein DNA-binding Domains of Transcription Factors Etv1, Etv4, Etv5, and Fev." Journal of Biological Chemistry 290, no. 22 (April 12, 2015): 13692–709. http://dx.doi.org/10.1074/jbc.m115.646737.
Full textShin, Ye Ji, Jae Won Yun, and Hong Sook Kim. "Portrait of Molecular Signaling and Putative Therapeutic Targets in Prostate Cancer with ETV4 Fusion." Biomedicines 10, no. 10 (October 20, 2022): 2650. http://dx.doi.org/10.3390/biomedicines10102650.
Full textZhang, Xueming, Lei Si, Zhanpeng Yue, Yang Liu, Bin Guo, Ziyi Li, and Dexue Li. "Expression of Ets Transcription Factors Etv4, Etv1 in Mouse Testis." Biology of Reproduction 83, Suppl_1 (November 1, 2010): 517. http://dx.doi.org/10.1093/biolreprod/83.s1.517.
Full textChen, Jing, Amelie T. Van der Ven, Joseph A. Newman, Asaf Vivante, Nina Mann, Hazel Aitkenhead, Shirlee Shril, et al. "ETV4 Mutation in a Patient with Congenital Anomalies of the Kidney and Urinary Tract." International Journal of Pediatrics and Child Health 4, no. 2 (September 4, 2016): 61–71. http://dx.doi.org/10.12974/2311-8687.2016.04.02.1.
Full textDissertations / Theses on the topic "ETV4"
Fonseca, Aline Simoneti. "Assinatura de mRNA entre adenoma e adenocarcinoma colorretal." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/17/17135/tde-18122014-095654/.
Full textColorectal cancer is among the main malignant neoplasia, it is the fourth leading cause of death in the world and the third in Brazil. Mutations in APC, DCC, KRAS and TP53 genes have been originally associated with the progression of sporadic colorectal cancer (CRC). However genome wide and exome studies have revealed other genes related to CRC. As a consequence of these mutations, a set of genes alters their expression modulating gene pathways in every stage of tumor progression. In this regard, there is great effort to define gene signatures that help to classify tumors in relation to patients diagnosis and prognosis. Therefore, the objective of this project was to analyze gene expression in genomic scale of colorectal adenoma and adenocarcinoma samples aiming to identify new genetic markers linked to adenoma- adenocarcinoma transition. For this purpose, ten paired adenoma and adenocarcinoma samples of the same patient were subjected to gene expression analysis by microarrays technique. Bioinformatics analyses revealed a signature of 689 genes differentially expressed (fold-change>2, p<0.05), which allowed the genetic classification between adenoma and adenocarcinoma. Eight genes (IL6, IL8, OSM, SFRP4, ETV4, ESM1, SIM2 and RETNLB) were chosen for validation based on their function and expression value in tumor tissue. In silico analysis of hyperexpressed genes, done in the program MetaCore (data analysis and gene pathways), highlighted diverse gene pathways linked to tumorigenesis, including the ones of cell adhesion and Epithelial-Mesenchymal Transition (EMT), important in the advanced phase of tumor progression. ETV4 gene was selected for functional assays due to its high expression levels in the ten samples of adenocarcinoma and due to its participation in cell proliferation mechanisms and in EMT. In vitro siRNA assays for ETV4 gene resulted in the decrease of cell proliferation and in the clonogenic potential of HT29 line. In addition, mutations in APC, KRAS and TP53 genes were investigated in paired samples of adenoma, adenocarcinoma, normal tissue, and peripheral blood from ten patients. All patients showed germline mutations in the three genes. However, only KRAS (40%) and TP53 (30%) genes showed somatic and pathogenic mutations, exclusively in adenocarcinomas. These results demonstrated that, in our cohort, mutations in TP53 and KRAS genes might be contributing to progression in a portion of sporadic-type colorectal cancer. In summary, the present study points out that ETV4 gene might contribute to activate cell proliferation mechanism in colorectal adenocarcinoma. Moreover, the study demonstrates the importance of combining the mutation analysis with expression profile in order to better understanding the molecular basis of colorectal cancer.
Dumortier, Mandy. "Étude du rôle des facteurs de transcription ETV4 et ETV1 de la famille ETS dans le processus tumoral de cancers hormono-dépendants : le cancer du sein, la progression métastatique du cancer de la prostate." Thesis, Lille 1, 2017. http://www.theses.fr/2017LIL10181.
Full textETV1, 4, 5 transcription factor are overexpress in various cancer. The first part is focus in MMP13 like target gene, potentially implicated in the mammary tumorigenesis induced by ETV4. Thus we show, the regulation of MMP13 expression by ETV4. Next we show that ETV4 promotes the mammary tumorigenesis and that MMP13 is a relay. In fact, the overexpression of MMP13 is implicate in pro-tumorigenesis effect and the repression of MMP13 in context of ETV4 overexpression decreases this effect. This approach was completed by the injection of cells in mice and show MMP13 mediate the pro-tomorigene effect of ETV4. We analysed expression of MMP13 and ETV4 in primary breast tumors and show the overexpression concomintant of ETV4 and MMP13 are associated with a poor prognosis. The second part of our study is about the ETV1 factor, in the progression of metastasis of prostate cancer (PCa) and the research of involved target genes. Gene fusion involving ERG and ETV1 and their overexpression are frequent in PCa and occur in 50% and 10% of cases respectively. To understand the role of ETV1, in comparison with available data on ERG, differents PCa cells overexpress or repress ETV1 were used. We have shown that the ETV1 factor enhance tumorigenesis capacities PCa cell lines and her truncated form has opposite effects. ETV1 full-lenght (FL) induces more bones metastasis formation than her truncated form. The expression of ETV1, and his truncated form was study in PCa samples. We confirmed the pro-tumorigenic statute of ETV1 factor in PCa and we defined differences beetwen FL and truncated length and complet the comprehension of the ETV1 function in the formation of bones metastasis of PCa
Cavazzola, Luciane Rostirola. "Análise da expressão do mRNA das enzimas timidina fosforilase e uridina fosforilase 1 e 2 e dos fatores de transcrição PDEF e ETV4 em tumores de próstata e de rim." Pontifícia Universidade Católica do Rio Grande do Sul, 2014. http://hdl.handle.net/10923/6884.
Full textProstate cancer is the most important urological tumor and the second most common in men. The incidence and mortality of this multifactorial disease are varied and take into account behavioral factors and genetic predisposition. The main predisposing factors are age, race and family history. Among the various histological subtypes, the acinar adenocarcinoma is the most common. The classification of this tumor, from transrectal ultrasonography and biopsy needle, is made for degrees of Gleason score and TNM classification that provides for tumor staging. The prostatic carcinoma has a propensity to metastasize and is regulated by various molecular signaling pathways through genes, proteins, enzymes, receptors and transcription factors. The RCC is the third most common urological tumor. The wide variation in incidence correlates with the geographical area. Risk factors can be various as smoking, genetic diseases and genetic alterations. The vast majority of cases occur in adults and in the renal parenchyma, and comprise five histological subtypes, the clear and sporadic cells being the most common. The histological differentiation is made by Fuhrman grade and staging by TNM classification. As the clinical manifestations are varied, most patients are diagnosed incidentally and by ultrasonography. The enzymes thymidine phosphorylase and uridine phosphorylase 1 and 2 as well as the transcription factors PDEF and ETV4 are reported as components of pathways leading to tumorigenesis and / or metastasis. The objective of this work is to analyze these enzymes and transcription factors and their correlation with clinical and pathological variables in prostate and kidney cancers as well as in bening tissue, by RT-PCR in real time. In prostate cancer, the geometric mean of the relative expression of transcription factors PDEF and ETV were greater in the tumor than in the benign tissues These factors also had a significant correlation between them in tumor tissue and benign tissue. The geometric mean of relative expression of the enzyme thymidine phosphorylase and uridine phosphorylase 1 strongly and significantly correlated between them in benign and in malignant prostate samples. The geometric mean of relative expression of thymidine phosphorylase also correlated moderately and significantly with the ETV4 in benign tissues. The geometric mean of relative expression of uridine phosphorylase in tumor T3 was significantly lower than in T1 and T2 prostate tumors. Kidney carcinoma TNM Classification strongly and significantly correlated with the relative expression of the enzyme thymidine phosphorylase and negatively correlated very strong and significantly with the enzyme uridine phosphorylase. The relative expression of the transcription factor ETV4 correlated negatively and significantly stronger with the TNM classification, and correlated with strength and significance with the relative expression of uridine phosphorylase in tumor tissues of the kidney.
O câncer de próstata é o mais importante tumor urológico e o segundo mais frequente no homem. As taxas de incidência e de mortalidade desta doença multifatorial são muito variadas e leva em conta fatores comportamentais e prédisposição genética. Os principais fatores predisponentes são idade avançada, raça e história familiar. Dentre os vários subtipos histológicos, o adenocarcinoma acinar é o mais frequente. A classificação deste tumor, a partir da ultra-sonografia transretal com biópsia por agulha, é feita pelos graus de escore de Gleason e pela Classificação TNM que fornece o estadiamento do tumor. O carcinoma de próstata, além de ter grande propensão em formar metástases, é regulado por várias vias moleculares de sinalização por meio de genes, proteínas, enzimas, receptores e fatores de transcrição.O carcinoma de células renais é o terceiro mais comum tumor urológico. A grande variação na taxa de incidência se correlaciona com a área geográfica. Os fatores de risco podem ser vários como tabagismo, doenças genéticas e alterações gênicas. A grande maioria dos casos ocorre em adultos no parênquima renal e, além de compreender cinco subtipos histológicos, o de células claras e esporádico é o mais frequente. A diferenciação histológica é feita pelo grau de Fuhrman e o estadiamento pela Classificação TNM. Como as manifestações clínicas são variadas, a maioria dos pacientes são diagnosticados incidentalmente e por ultra-sonografia. As enzimas timidina fosforilase e uridina fosforilase 1 e 2, bem como os fatores de transcrição PDEF e ETV4, são relatados como componentes das vias que levam à tumorigênese e/ou a metastização. O objetivo deste trabalho é analizar estas enzimas e fatores de transcrição, correlacionando-as com variáveis clínicas e patológicas nos tumores de próstata e de rim, bem como nos tecidos adjacentes a estes tumores, pela técnica de RT-PCR em tempo real. No câncer de próstata, a média geométrica da expressão relativa dos fatores de transcrição PDEF e ETV4 entre os tecidos benignos e tumorais, apresentaram correlação significativa com uma maior expressão relativa no tumor quando comparado ao seu benigno. Estes fatores também apresentaram uma correlação significativa entre eles no tecido tumoral e no tecido benigno. As médias geométricas da expressão relativa das enzimas timidina fosforilase e uridina fosforilase 1 se correlacionaram fortemente e significativamente entre os tecidos benignos e tumorais da próstata. A média geométrica da expressão relativa da timidina fosforilase também se correlacionou de forma moderada e significativa com a do ETV4, entre os tecidos benignos. A média geométrica da expressão relativa da uridina fosforilase nos tumores T3 foi significativamente menor do que nos tumores T1 e T2 da próstata. No carcinoma de rim a Classificação TNM se correlacionou fortemente e significativamente com a expressão relativa da enzima timidina fosforilase, e se correlacionou de forma negativa, muito forte e significativamente com a enzima uridina fosforilase. A expressão relativa do fator de transcrição ETV4 se correlacionou de forma negativa, forte e significativamente com a Classificação TNM, e se correlacionou de forma forte e significativamente com a expressão relativa de uridina fosforilase nos tecidos tumorais de rim.
Cavazzola, Luciane Rostirola. "An?lise da express?o do mRNA das enzimas timidina fosforilase e uridina fosforilase 1 e 2 e dos fatores de transcri??o PDEF e ETV4 em tumores de pr?stata e de rim." Pontif?cia Universidade Cat?lica do Rio Grande do Sul, 2014. http://tede2.pucrs.br/tede2/handle/tede/1788.
Full textProstate cancer is the most important urological tumor and the second most common in men. The incidence and mortality of this multifactorial disease are varied and take into account behavioral factors and genetic predisposition. The main predisposing factors are age, race and family history. Among the various histological subtypes, the acinar adenocarcinoma is the most common. The classification of this tumor, from transrectal ultrasonography and biopsy needle, is made for degrees of Gleason score and TNM classification that provides for tumor staging. The prostatic carcinoma has a propensity to metastasize and is regulated by various molecular signaling pathways through genes, proteins, enzymes, receptors and transcription factors. The RCC is the third most common urological tumor. The wide variation in incidence correlates with the geographical area. Risk factors can be various as smoking, genetic diseases and genetic alterations. The vast majority of cases occur in adults and in the renal parenchyma, and comprise five histological subtypes, the clear and sporadic cells being the most common. The histological differentiation is made by Fuhrman grade and staging by TNM classification. As the clinical manifestations are varied, most patients are diagnosed incidentally and by ultrasonography. The enzymes thymidine phosphorylase and uridine phosphorylase 1 and 2 as well as the transcription factors PDEF and ETV4 are reported as components of pathways leading to tumorigenesis and / or metastasis. The objective of this work is to analyze these enzymes and transcription factors and their correlation with clinical and pathological variables in prostate and kidney cancers as well as in bening tissue, by RT-PCR in real time. In prostate cancer, the geometric mean of the relative expression of transcription factors PDEF and ETV were greater in the tumor than in the benign tissues These factors also had a significant correlation between them in tumor tissue and benign tissue. The geometric mean of relative expression of the enzyme thymidine phosphorylase and uridine phosphorylase 1 strongly and significantly correlated between them in benign and in malignant prostate samples. The geometric mean of relative expression of thymidine phosphorylase also correlated moderately and significantly with the ETV4 in benign tissues. The geometric mean of relative expression of uridine phosphorylase in tumor T3 was significantly lower than in T1 and T2 prostate tumors. Kidney carcinoma TNM Classification strongly and significantly correlated with the relative expression of the enzyme thymidine phosphorylase and negatively correlated very strong and significantly with the enzyme uridine phosphorylase. The relative expression of the transcription factor ETV4 correlated negatively and significantly stronger with the TNM classification, and correlated with strength and significance with the relative expression of uridine phosphorylase in tumor tissues of the kidney.
O c?ncer de pr?stata ? o mais importante tumor urol?gico e o segundo mais frequente no homem. As taxas de incid?ncia e de mortalidade desta doen?a multifatorial s?o muito variadas e leva em conta fatores comportamentais e pr?-disposi??o gen?tica. Os principais fatores predisponentes s?o idade avan?ada, ra?a e hist?ria familiar. Dentre os v?rios subtipos histol?gicos, o adenocarcinoma acinar ? o mais frequente. A classifica??o deste tumor, a partir da ultra-sonografia transretal com bi?psia por agulha, ? feita pelos graus de escore de Gleason e pela Classifica??o TNM que fornece o estadiamento do tumor. O carcinoma de pr?stata, al?m de ter grande propens?o em formar met?stases, ? regulado por v?rias vias moleculares de sinaliza??o por meio de genes, prote?nas, enzimas, receptores e fatores de transcri??o. O carcinoma de c?lulas renais ? o terceiro mais comum tumor urol?gico. A grande varia??o na taxa de incid?ncia se correlaciona com a ?rea geogr?fica. Os fatores de risco podem ser v?rios como tabagismo, doen?as gen?ticas e altera??es g?nicas. A grande maioria dos casos ocorre em adultos no par?nquima renal e, al?m de compreender cinco subtipos histol?gicos, o de c?lulas claras e espor?dico ? o mais frequente. A diferencia??o histol?gica ? feita pelo grau de Fuhrman e o estadiamento pela Classifica??o TNM. Como as manifesta??es cl?nicas s?o variadas, a maioria dos pacientes s?o diagnosticados incidentalmente e por ultra-sonografia. As enzimas timidina fosforilase e uridina fosforilase 1 e 2, bem como os fatores de transcri??o PDEF e ETV4, s?o relatados como componentes das vias que levam ? tumorig?nese e/ou a metastiza??o. O objetivo deste trabalho ? analizar estas enzimas e fatores de transcri??o, correlacionando-as com vari?veis cl?nicas e patol?gicas nos tumores de pr?stata e de rim, bem como nos tecidos adjacentes a estes tumores, pela t?cnica de RT-PCR em tempo real. No c?ncer de pr?stata, a m?dia geom?trica da express?o relativa dos fatores de transcri??o PDEF e ETV4 entre os tecidos benignos e tumorais, apresentaram correla??o significativa com uma maior express?o relativa no tumor quando comparado ao seu benigno. Estes fatores tamb?m apresentaram uma correla??o significativa entre eles no tecido tumoral e no tecido benigno. As m?dias geom?tricas da express?o relativa das enzimas timidina fosforilase e uridina fosforilase 1 se correlacionaram fortemente e significativamente entre os tecidos benignos e tumorais da pr?stata. A m?dia geom?trica da express?o relativa da timidina fosforilase tamb?m se correlacionou de forma moderada e significativa com a do ETV4, entre os tecidos benignos. A m?dia geom?trica da express?o relativa da uridina fosforilase nos tumores T3 foi significativamente menor do que nos tumores T1 e T2 da pr?stata. No carcinoma de rim a Classifica??o TNM se correlacionou fortemente e significativamente com a express?o relativa da enzima timidina fosforilase, e se correlacionou de forma negativa, muito forte e significativamente com a enzima uridina fosforilase. A express?o relativa do fator de transcri??o ETV4 se correlacionou de forma negativa, forte e significativamente com a Classifica??o TNM, e se correlacionou de forma forte e significativamente com a express?o relativa de uridina fosforilase nos tecidos tumorais de rim.
Huang-Hobbs, Helen. "Dissecting the mechanism of ETV6 polymerization." Thesis, University of British Columbia, 2013. http://hdl.handle.net/2429/45691.
Full textHart, Stephen Michael. "In vivo analysis of the ETV6-CBFA2 fusion gene." Thesis, University College London (University of London), 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.392499.
Full textJomrich, Gerd, Florian Maroske, Jasmin Stieger, Matthias Preusser, Aysegül Ilhan-Mutlu, Daniel Winkler, Ivan Kristo, Matthias Paireder, and Sebastian Friedrich Schoppmann. "MK2 and ETV1 Are Prognostic Factors in Esophageal Adenocarcinomas." IVyspring International Publisher, 2018. http://dx.doi.org/10.7150/jca.22310.
Full textKaulfuß, Katja [Verfasser]. "Identifizierung von Zielgenen des chimären Transkriptionsfaktors ETV6/RUNX1 / Katja Kaulfuß." Berlin : Freie Universität Berlin, 2018. http://d-nb.info/1196805180/34.
Full textFavier, Marie. "Les thrombopénies héréditaires rares : implications des gènes ETV6, ITGA2B, ITGB3." Thesis, Aix-Marseille, 2017. http://www.theses.fr/2017AIXM0559.
Full textThe identification of the genes involved in thrombocytopenia provides important elements for understanding the pathways of regulation of the production and functions of platelets or even hematopoiesis. Our laboratory has developed a strategy for identifying genes causing thrombocytopenia without a priori hypothesis by sequencing exomes. This strategy has been applied to families with autosomal dominant thrombocytopenia and has demonstrated mutations in the genes etv6, itga2b and itgb3. Normal platelet thrombocytopenia are particularly important to detect because of the risk of developing onco-hematological pathology. The genetic origin of this category of thrombocytopenia has long been limited to mutations in the runx1 gene. More recently, mutations on the ankrd26 promoter have been reported. The work I did during my thesis helped to involve the etv6 gene in this group of thrombocytopenia. Concerning this gene six families have pathogenic mutations. All these mutations are the cause of a loss of the repressive activity of the gene and a high number of CD34+ cells circulating in the blood revealing the role of ETV6 in the onco-hematological predisposition. In addition, megakaryopoiesis has two main anomalies. They associate an increase in the number of megakaryocytic progenitor colonies with the formation of reduced proplatelets.Concerning the itga2b and itgb3 genes, 3 families were studied. These genes encode the αIIbβ3 integrin. Integrin αIIbβ3 is a platelet receptor for fibrinogen and Von Willebrand factor, and plays a crucial role in thrombosis and hemostasis
Craig, Michael P. "Transcriptional Regulation of Developmental and Tumor-Induced Angiogenesis by Etv2 and Fli1b." University of Cincinnati / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1427982504.
Full textBooks on the topic "ETV4"
Suman. Snēha: ETV dhārāvāhikaku skrīnplērūpaṃ. Haidarābād: Uṣōdayā Pablikēṣans, 1997.
Find full text(Greece), Hellēnikē Trapeza Viomēchanikēs Anaptyxeōs. Hē drastēriotēta tēs ETVA to etos 1985. [Athēna]: Hē Trapeza, 1985.
Find full textFrank, N. Magid Associates Inc. Executive Summary of Findings: Prepared for ETVC Limited(Survey Research). [Uk]: Frank N Magid Associates, 1991.
Find full text), National Risk Management Research Laboratory (U S. Environmental technology verification (ETV) program case studies: Demonstrating program outcomes. Cincinnati, OH: National Risk Management Research Laboratory, Office of Research and Development, U.S. Environmental Protection Agency, 2006.
Find full textDer Europäische Verbund territorialer Zusammenarbeit (ETVZ): Neue Chancen für die Europaregion Tirol-Südtirol-Trentino. Wien: Braumüller, 2011.
Find full text1945-, Kurfürst Ulrich, ed. Solid sample analysis: Direct and slurry sampling using GF-AAS and ETV-ICP. Berlin: Springer, 1998.
Find full textCurabay, Şensu. 20. kuruluş yılında Anadolu Üniversitesi açiköğretim sistemi ve Açıköğretim Fakültesi eğitim televizyonu, ETV. Eskişehir [Turkey]: Anadolu Üniversitesi, 2002.
Find full textBibliothèqye publique d'information. (23 et 24 octobre 2002). Les 25 ans de la BPI: Encyclopédisme, actualité, libre-accès : actes du colloque international organisé par la Bibliothèqye publique d'information, au Centre Pompidou, les 23 et24 octobre 2002. Paris: Editions de la Bibliothèque publique d'information, 2003.
Find full textGeorge, Papandreou Andreas, Papanagiōtou Kōstas, Sallas Michalēs G, and Hellēnikē Trapeza Viomēchanikēs Anaptyxeōs (Greece), eds. Themeliōsē tou ergostasiou tēs Hellēnikēs Viomēchanias Alouminas stē Thisvē/Voiōtias stis 27 Oktōvriou 1987: Homilies tou Prōthypourgou Andrea Papandreou, tou Anaplērōtē-Hypourgou V.E.T. Kōsta Papanagiōtou, tou Dioikētē tēs ETVA Michalē G. Salla. Athēna: Hellēnikē Trapeza Viomēchanikēs Anaptyxeōs, 1987.
Find full textBook chapters on the topic "ETV4"
Bohlander, Stefan K. "ETV6." In Encyclopedia of Cancer, 1343–46. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_2035.
Full textSundaresh, Aishwarya, and Owen Williams. "Mechanism of ETV6-RUNX1 Leukemia." In Advances in Experimental Medicine and Biology, 201–16. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-3233-2_13.
Full textAramendía Marzo, Maite, Martín Resano, and Frank Vanhaecke. "ETV-ICPMS for Analysis of Polymers." In Mass Spectrometry Handbook, 1061–77. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2012. http://dx.doi.org/10.1002/9781118180730.ch44.
Full textFord, Anthony M., and Mel Greaves. "ETV6-RUNX1 + Acute Lymphoblastic Leukaemia in Identical Twins." In Advances in Experimental Medicine and Biology, 217–28. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-3233-2_14.
Full textDezena, Roberto Alexandre. "General Principles of Endoscopic Third Ventriculostomy (ETV)." In Endoscopic Third Ventriculostomy, 67–79. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-28657-6_5.
Full textDezena, Roberto Alexandre. "Surgical Technique of Endoscopic Third Ventriculostomy (ETV)." In Endoscopic Third Ventriculostomy, 81–91. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-28657-6_6.
Full textRuggeri, Tommaso, and Masaru Sugiyama. "Shock Wave in a Polyatomic Gas Analyzed by ET14." In Classical and Relativistic Rational Extended Thermodynamics of Gases, 389–407. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-59144-1_17.
Full textLo, William B., and Abhaya V. Kulkarni. "Endoscopic Third Ventriculostomy with Choroid Plexus Cauterization (ETV–CPC) Versus CSF Shunting." In Cerebrospinal Fluid Disorders, 317–29. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-97928-1_18.
Full textVerrept, Peter, Sylvie Boonen, Luc Moens, and Richard Dams. "Solid Sampling by Electrothermal Vaporization-Inductively Coupled Plasma-Atomic Emission and -Mass Spectrometry (ETV-ICP-AES/-MS)." In Solid Sample Analysis, 191–246. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-662-03716-4_4.
Full textSamandouras, George. "Shunts and endoscopic third ventriculostomy." In The Neurosurgeon's Handbook, 696–702. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780198570677.003.0572.
Full textConference papers on the topic "ETV4"
Kunju, Lakshmi P., Shannon Carskadon, Ritu Bhalla, Javed Siddiqui, Scott A. Tomlins, Arul M. Chinnaiyan, and Nallasivam Palanisamy. "Abstract 4216: Novel RNAIn situhybridization method for the detection of ETV1, ETV4 and ETV5 rearrangements in prostate cancer." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-4216.
Full textTyagi, Nikhil, Sanjeev K. Srivastava, Arun Bhardwaj, Sumit Arora, William E. Grizzle, Laurie B. Owen, Ajay P. Singh, and Seema Singh. "Abstract 2011: ETV4 promotes pancreatic cancer progression and metastasis." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-2011.
Full textKim, E. "PO-111 Capicua suppresses hepatocellular carcinoma progression by controlling ETV4-MMP1 axis." In Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.152.
Full textSrivastava, Sanjeev K., Nikhil Tyagi, Gurpreet Kaur, Sumit Arora, Arun Bhardwaj, William E. Grizzle, Ajay P. Singh, and Seema Singh. "Abstract 3120: Evidence supporting a role of ETV4 in pancreatic cancer pathogenesis." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-3120.
Full textButler, Miriam S., Michael Hsing, Mani Roshan Moniri, Desmond Lau, Paul Yen, Ari Kim, Scott Lien, et al. "Abstract 1648: Targeting ETS factor ETV4 as a novel therapeutic for the management of breast and prostate cancer." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-1648.
Full textShah, Amip, Chandrakant D. Patel, and Cullen Bash. "Designing Environmentally Sustainable Computer Systems Using Networks of Exergo-Thermo-Volume Building Blocks." In ASME 2009 InterPACK Conference collocated with the ASME 2009 Summer Heat Transfer Conference and the ASME 2009 3rd International Conference on Energy Sustainability. ASMEDC, 2009. http://dx.doi.org/10.1115/interpack2009-89037.
Full textChica-Parrado, Maria Rosario, Julio Montes-Torres, Cynthia Robles-Podadera, Martina Alvarez, Jose Jerez, Luis Vicioso, Lidia Pérez-Villa, et al. "Abstract P1-10-26: Gene expression levels of DTX3, CACNA1G, IL11, ETV4 and TSPAN7 selected by LASSO penalty regression could predict pCR after neoadjuvant chemotherapy in breast cancer tumors." In Abstracts: 2019 San Antonio Breast Cancer Symposium; December 10-14, 2019; San Antonio, Texas. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.sabcs19-p1-10-26.
Full textTerada, Minoru. "ETV." In the 10th annual SIGCSE conference. New York, New York, USA: ACM Press, 2005. http://dx.doi.org/10.1145/1067445.1067480.
Full textXagoraris, Zafiris, and George Soulis. "The new geographic information system in ETVA VI.PE." In Fourth International Conference on Remote Sensing and Geoinformation of the Environment, edited by Kyriacos Themistocleous, Diofantos G. Hadjimitsis, Silas Michaelides, and Giorgos Papadavid. SPIE, 2016. http://dx.doi.org/10.1117/12.2240856.
Full textUemura, Suguru, Daiichiro Hasegawa, Akemi Shono, Khin Kyae Mon Thwin, Nanako Nino, Satoru Takafuji, Takeshi Mori, et al. "Abstract A11: A pediatric ETV6-ABL1-positive acute lymphoblastic leukemia case with ETV6-ABL1-independent resistance to tyrosine kinase inhibitor." In Abstracts: AACR Special Conference: Pediatric Cancer Research: From Basic Science to the Clinic; December 3-6, 2017; Atlanta, Georgia. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.pedca17-a11.
Full textReports on the topic "ETV4"
Morrissey, Colm. Small Molecule Inhibitors of ERG and ETV1 in Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, September 2014. http://dx.doi.org/10.21236/ada613409.
Full textMorrissey, Colm. Small Molecule Inhibitors of ERG and ETV1 in Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, September 2013. http://dx.doi.org/10.21236/ada593129.
Full textOrkin, Stuart H. Exploring the Mechanisms of Pathogenesis in Prostate Cancer Involving TMPRSS2-ERG (Or ETV1) Gene Rearrangement. Fort Belvoir, VA: Defense Technical Information Center, January 2008. http://dx.doi.org/10.21236/ada491425.
Full textOrkin, Stuart H. Exploring the Mechanisms of Pathogenesis in Prostate Cancer Involving TMPRSS2-ERG (Or ETV1) Gene Rearrangement. Fort Belvoir, VA: Defense Technical Information Center, January 2009. http://dx.doi.org/10.21236/ada501163.
Full textDetcheva, Albena K., Jürgen Hassler, Lidia Ivanova, Caroline Hommel, and Thomas Vogt. ETV-ICP-OES Determination of Essential, Non-essential and Toxic Elements in Seven Bulgarian Herbs. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, April 2019. http://dx.doi.org/10.7546/crabs.2019.04.05.
Full textFisher, Robert J. Environmental Technology Verification Coatings and Coating Equipment Program (ETV CCEP). High Transfer Efficiency Spray Equipment - Generic Verification Protocol (Revision 0). Fort Belvoir, VA: Defense Technical Information Center, September 2006. http://dx.doi.org/10.21236/ada470549.
Full textFisher, Robert J. Environmental Technology Verification Coatings and Coating Equipment Program (ETV CCEP). Final Technology Applications Group TAGNITE - Testing and Quality Assurance Plan (T/QAP). Fort Belvoir, VA: Defense Technical Information Center, July 2006. http://dx.doi.org/10.21236/ada459899.
Full text[Environmental Hazards Assessment Program annual report, June 1992--June 1993]. South Carolina ETV Socratic Dialog II. Office of Scientific and Technical Information (OSTI), June 1993. http://dx.doi.org/10.2172/10109121.
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