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Relevant bibliographies by topics / Excitatory neurotransmitter

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Academic literature on the topic 'Excitatory neurotransmitter'

Author: Grafiati

Published: 5 June 2025

Last updated: 16 July 2025

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Journal articles on the topic "Excitatory neurotransmitter"

1

Kim, Dongshin, and Jang-Sik Lee. "Emulating Excitatory and Inhibitory Functions in Artificial Synaptic Devices." ECS Meeting Abstracts MA2022-02, no. 33 (2022): 2585. http://dx.doi.org/10.1149/ma2022-02332585mtgabs.

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Synaptic signals are controlled by neurotransmitters. The synaptic signals can be excited or inhibited depending on the types of neurotransmitters. The demonstration of the balancing between excitatory and inhibitory signals has important implications for the complex and efficient computing of the nervous system. Emulating the excitatory-inhibitory balancing behaviors of the nervous system is one way to establish neuromorphic computing. In this study, we demonstrate artificial synapses using PEDOT:PSS channel and neurotransmitter solutions to emulate the excitatory-inhibitory balancing behavio

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Huang, Chun-Ping, Yi-Wen Lin, Der-Yen Lee, and Ching-Liang Hsieh. "Electroacupuncture Relieves CCI-Induced Neuropathic Pain Involving Excitatory and Inhibitory Neurotransmitters." Evidence-Based Complementary and Alternative Medicine 2019 (October 20, 2019): 1–9. http://dx.doi.org/10.1155/2019/6784735.

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Neuropathic pain caused by peripheral tissue injuries to the higher brain regions still has no satisfactory therapy. Disruption of the balance of excitatory and inhibitory neurotransmitters is one of the underlying mechanisms that results in chronic neuropathic pain. Targeting neurotransmitters and related receptors may constitute a novel approach for treating neuropathic pain. We investigated the effects of electroacupuncture (EA) on chronic constriction injury- (CCI-) induced neuropathic pain. The mechanical allodynia and thermal hyperalgesia pain behaviors were relieved by 15 Hz EA but not

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Stahl, Stephen M. "Glutamate: The Universal Excitatory Neurotransmitter." Psychiatric Annals 27, no. 3 (1997): 152–55. http://dx.doi.org/10.3928/0048-5713-19970301-03.

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Herring, B. E., K. Silm, R. H. Edwards, and R. A. Nicoll. "Is Aspartate an Excitatory Neurotransmitter?" Journal of Neuroscience 35, no. 28 (2015): 10168–71. http://dx.doi.org/10.1523/jneurosci.0524-15.2015.

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Korpi, Esa R., Riikka Mäkelä, and Mikko Uusi-Oukari. "Ethanol: Novel Actions on Nerve Cell Physiology Explain Impaired Functions." Physiology 13, no. 4 (1998): 164–70. http://dx.doi.org/10.1152/physiologyonline.1998.13.4.164.

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Molecular biological tools have revealed receptor proteins for excitatory and inhibitory neurotransmitters on cell membranes as targets of ethanol action. Behavioral and pharmacogenetic assays using rodent lines have supported this neurotransmitter theory of ethanol action and given a firm basis for future identification of the relevant genes and the central physiological processes vulnerable to ethanol.

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Li, Yi-Fan, Keshia L. Jackson, Javier E. Stern, Brandon Rabeler, and Kaushik P. Patel. "Interaction between glutamate and GABA systems in the integration of sympathetic outflow by the paraventricular nucleus of the hypothalamus." American Journal of Physiology-Heart and Circulatory Physiology 291, no. 6 (2006): H2847—H2856. http://dx.doi.org/10.1152/ajpheart.00625.2005.

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The paraventricular nucleus (PVN) of the hypothalamus is a central site known to modulate sympathetic outflow. Excitatory and inhibitory neurotransmitters within the PVN dictate final outflow. The goal of the present study was to examine the role of the interaction between the excitatory neurotransmitter glutamate and the inhibitory neurotransmitter GABA in the regulation of sympathetic activity. In α-chloralose- and urethane-anesthetized rats, microinjection of glutamate and N-methyl-d-aspartate (NMDA; 50, 100, and 200 pmol) into the PVN produced dose-dependent increases in renal sympathetic

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Patel, Piyush M., John C. Drummond, Daniel J. Cole, and Randall L. Goskowicz. "Isoflurane Reduces Ischemia-induced Glutamate Release in Rats Subjected to Forebrain Ischemia." Anesthesiology 82, no. 4 (1995): 996–1003. http://dx.doi.org/10.1097/00000542-199504000-00024.

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Background The release of excitatory neurotransmitters during ischemia is thought to contribute to ischemic neuronal injury. Volatile anesthetics have been shown to reduce excitatory neurotransmission in vitro, and it is conceivable that they reduce ischemia-induced neurotransmitter release. The current investigation was conducted to evaluate the effect of isoflurane and N2O-fentanyl anesthesia on ischemia-induced glutamate release in the rat and to compare it with that of mild hypothermia, an intervention known to reduce glutamate release significantly. Methods Microdialysis probes were impla

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Bickler, Philip E., Leslie T. Buck, and John R. Feiner. "Volatile and Intravenous Anesthetics Decrease Glutamate Release from Cortical Brain Slices during Anoxia." Anesthesiology 83, no. 6 (1995): 1233–40. http://dx.doi.org/10.1097/00000542-199512000-00014.

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Abstract Background Extracellular accumulation of the excitatory neurotransmitter L-glutamate during cerebral hypoxia or ischemia contributes to neuronal death. Anesthetics inhibit release of synaptic neurotransmitters but it is unknown if they alter net extrasynaptic glutamate release, which accounts for most of the glutamate released during hypoxia or ischemia. The purpose of this study was to determine if different types of anesthetics decrease hypoxia-induced glutamate release from rat brain slices.

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Vanden Berghe, Pieter, Sander Molhoek, Ludwig Missiaen, Jan Tack, and Jozef Janssens. "Differential Ca2+ signaling characteristics of inhibitory and excitatory myenteric motor neurons in culture." American Journal of Physiology-Gastrointestinal and Liver Physiology 279, no. 5 (2000): G1121—G1127. http://dx.doi.org/10.1152/ajpgi.2000.279.5.g1121.

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Physiological studies on functionally identified myenteric neurons are scarce because of technical limitations. We combined retrograde labeling, cell culturing, and fluorescent intracellular Ca2+ concentration ([Ca2+]i) signaling to study excitatory neurotransmitter responsiveness of myenteric motor neurons. 1,1-Didodecyl-3,3,3′,3′-tetramethyl indocarbocyanine (DiI) was used to label circular muscle motor neurons of the guinea pig ileum. DiI-labeled neurons were easily detectable in cultures prepared from these segments. The excitatory neurotransmitters (10−5 M) acetylcholine, substance P, and

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Puia, Giulia. "The inhibitory action of an excitatory neurotransmitter." Trends in Pharmacological Sciences 23, no. 2 (2002): 57. http://dx.doi.org/10.1016/s0165-6147(02)01976-4.

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Dissertations / Theses on the topic "Excitatory neurotransmitter"

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Agarwal, Shailesh Ramjilal. "Pharmacological modeling and regulation of excitatory amino acid transporters (EAATS)." CONNECT TO THIS TITLE ONLINE, 2007. http://etd.lib.umt.edu/theses/available/etd-09262007-111510/.

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Neville, Lewis Frederic. "Pre-synaptic modulation of cortical excitatory amino acid neurotransmitter release." Thesis, University of Southampton, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.328909.

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Magnuson, David Stuart Keith. "Analysis of excitatory amino acid receptors in the rat spinal cord in vivo and in vitro." Thesis, University of British Columbia, 1988. http://hdl.handle.net/2429/29017.

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Several endogenous amino acids including L-glutamate and L-aspartate have potent excitatory effects in the central nervous system. They are thought to act as synaptic transmitters in many neural pathways including those in the spinal cord. Three distinct receptors have been described through which these excitatory amino acids exert their effects. These are referred to as quisqualate, kainate and N-methyl-D-aspartate (NMDA) receptors, after the exogenous excitants most specific for each. In addition, sub-types of the NMDA receptor have been proposed to account for differences observed in the ac

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Vaccarella, Graziano. "Synthesis and activity of pyridazine analogues of the excitatory neurotransmitter, glutamic acid." Thesis, The University of Sydney, 1998. https://hdl.handle.net/2123/27573.

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The synthesis and pharmacological assessment of novel pyridazine analogues of the neurotransmitter glutamic acid was the focus of this investigation. The project aimed to prepare agents for selective activation of the AMPA receptor subtype. The proposition investigated was that the six-membered pyridazine ring with an acidic hydroxyl group would provide a versatile carboxylic acid bioisostere and these compounds would be amenable to further derivatisation.

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Kühn, Claudia [Verfasser]. "Localization of excitatory and inhibitory neurotransmitter receptors in an identified motoneuron of the Drosophila Flight System / Claudia Kühn." Berlin : Freie Universität Berlin, 2012. http://d-nb.info/1029793298/34.

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Smith, Adam Luke. "Characterization of excitatory amino acid receptors using novel structural analogues." Thesis, University of Oxford, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.302953.

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Richmond, Saul Alexander. "Characterization of excitatory amino acid receptors in the mammalian central nervous system." Thesis, University of Oxford, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.308536.

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Richards, Dannette Shanon. "CHARACTERIZATION OF EXCITATORY AMINO ACID NEUROTRANSMITTERS AT MOTONEURON SYNAPSES CONTACTING RENSHAW CELLS." Wright State University / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=wright1260896604.

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Marx, Mari-Carmen. "The role of glutamine transporters in the maintenance of excitatory neurotransmission." Thesis, University of Cambridge, 2015. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.709012.

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De, Faoite Andrew. "Modulation of spontaneous activity in the rabbit urethra by excitatory and inhibitory neurotransmitters." Thesis, Queen's University Belfast, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.517278.

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Books on the topic "Excitatory neurotransmitter"

1

J, Roberts Peter, Storm-Mathisen Jon, and Bradford H. F, eds. Excitatory amino acids. Macmillan, 1986.

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D, Lodge David Ph, and Biological Council, eds. Excitatory amino acids in health and disease. Wiley, 1988.

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3

Ichiro, Kanazawa, and Kameyama Masakuni 1924-, eds. Neurotransmitters: Focus on excitatory amino acids : proceedings of the Fifth Workshop on Neurotransmitters and Diseases, Tokyo, June 18, 1988. Excerpta Medica, 1988.

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4

A, Cavalheiro Esper, Lehmann John 1952-, and Turski Lechoslaw, eds. Frontiers in excitatory amino acid research: Proceedings of an International Symposium "Excitatory Amino Acids '88," held in Manaus, Amazonas, Brazil, March 28-April 2, 1988. Liss, 1988.

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5

Philip, Hicks T., Lodge David Ph D, McLennan Hugh 1927-, and International Union of Physiological Sciences. Congress, eds. Excitatory amino acid transmission: Proceedings of a satellite symposium to the 30th Congress of the International Union of Physiological Sciences, held in Banff, Alberta, Canada, July 20-23, 1986. Liss, 1987.

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6

G, Avanzini, ed. Neurotransmitters in epilepsy. Elsevier, 1992.

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1941-, Lodge David, and Co-ordinating Committee for Symposia on Drug Action., eds. Excitatoryamino acids in health and disease. Wiley, 1988.

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8

Jerome, Engel, ed. Molecular neurobiology of epilepsy. Elsevier, 1992.

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9

Kollegger, Harald. Excitatory amino acids and brain damage. Facultas-Universitätsverlag, 1993.

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10

-T, Globus Mordecai Y., Dietrich W. Dalton, and Brain-91 (1991 : Miami, Fla.), eds. The Role of neurotransmitters in brain injury. Plenum Press, 1992.

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Book chapters on the topic "Excitatory neurotransmitter"

1

Mayer, Mark L., and Gary L. Westbrook. "Excitatory Amino Acids: Membrane Physiology." In Neurotransmitter Actions in the Vertebrate Nervous System. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-4961-7_5.

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Nusser, Z., and R. Shigemoto. "Input- and Output-Specific Segregation of Amino Acid Neurotransmitter Receptors on the Surface of Central Neurones." In Excitatory Amino Acids. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-662-03596-2_10.

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3

Etienne, P., and M. Baudry. "Role of excitatory amino acid neurotransmission in synaptic plasticity and pathology. An integrative hypothesis concerning the pathogenesis and evolutionary advantages of schizophrenia-related genes." In Neurotransmitter Actions and Interactions. Springer Vienna, 1990. http://dx.doi.org/10.1007/978-3-7091-9050-0_5.

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De Graan, P. N. E., L. H. Schrama, F. M. J. Heemskerk, L. V. Dekker, and W. H. Gispen. "The Role of Protein Kinase C Substrate B-50 (GAP-43) in Neurotransmitter Release and Long-Term Potentiation." In Excitatory Amino Acids and Neuronal Plasticity. Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-5769-8_38.

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5

Ellenbroek, Bart, Alfonso Abizaid, Shimon Amir, et al. "Excitatory Neurotransmitters." In Encyclopedia of Psychopharmacology. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_3250.

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Gallo, V., C. Giovannini, and G. Levi. "Regulation of Neurotransmitter Amino Acid Fluxes by Excitatory Amino Acid Receptors in Different Neural Cell Types in Culture." In Amino Acid Availability and Brain Function in Health and Disease. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-73175-4_20.

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Mori, Akitane, Isao Yokoi, Hideaki Kabuto, et al. "Control of Convulsions by Inhibitory and Excitatory Neurotransmitter Receptor Regulators in Epileptic El Mice and Neuromuscular Junction-Blocked Rats." In International Perspectives on Self-Regulation and Health. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4899-2596-1_7.

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Kanner, B. I., and R. Radian. "Mechanisms of Reuptake of Neurotransmitters from the Synaptic Cleft." In Excitatory Amino Acids. Palgrave Macmillan UK, 1986. http://dx.doi.org/10.1007/978-1-349-08479-1_11.

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Kruk, Zygmunt L., and Christopher J. Pycock. "Excitatory amino acids: L-glutamic acid and L-aspartic acid." In Neurotransmitters and Drugs. Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3132-2_8.

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Kruk, Zygmunt L., and Christopher J. Pycock. "Excitatory amino acids: L-glutamic acid and L-aspartic acid." In Neurotransmitters and Drugs. Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3134-6_8.

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Conference papers on the topic "Excitatory neurotransmitter"

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Schiller, Peter H. "ON and OFF channels of the visual system." In OSA Annual Meeting. Optica Publishing Group, 1990. http://dx.doi.org/10.1364/oam.1990.thf2.

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The receptors of the mammalian retina, which all hyperpolarize to light, make signconserving synapses with OFF bipolars and sign-inverting synapses with ON bipolars. Several hypotheses have been generated to explain this arrangement. To test them we applied the neurotransmitter agonist 2-amino-4-phosphonobutyrate (APB) to the retina, which selectively blocks the ON bipolars. Two kinds of experiments were carried out: In one, we examined the visual responses of single cells while reversibly inactivating the ON channel with APB. In the other, we assessed the visual capacities of monkeys before a

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Yuniati, Anis, and Retno Dwi Astuti. "Neural Network Synchronization of the Morris-Lecar Neuron Model Coupled with Short-Term Plasticity (STP)." In The 6th International Conference on Science and Engineering. Trans Tech Publications Ltd, 2024. http://dx.doi.org/10.4028/p-ymnn4n.

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This study used the Morris-Lecar (ML) neuron model coupled with Short-Term Plasticity (STP) to simulate neuronal connectivity and synaptic patterns. We analyze this neural network synchronization activity, examined the post-synaptic conductance patterns in the modelled neural network, investigated the dynamics of the neural network membrane potentials in the synchronous state, and analyze the Short-Term Plasticity (STP) synaptic transmission patterns by varying the inter-neuron connection probability for both inhibitory (pi) and excitatory (pe). This computational-based study was executed usin

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Yang, Chun-Lin, Nandan Shettigar, and C. Steve Suh. "A Proposition for Describing Real-World Network Dynamics." In ASME 2021 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2021. http://dx.doi.org/10.1115/imece2021-73360.

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Abstract This study presents a proposition for describing the dynamics of real-world networks under the general framework of complex networks. Outward behaviors of complex networks are the manifestation of the coupled dynamics at the macroscopic level and the individual dynamics at the microscopic level. At the macroscopic level a law of coupling governs the interactions of network constituents. At the microscopic level, the dynamics of individual constituent is defined by energy that follows normal distribution. Constituent dynamics are bounded by physical constraints. Consequently, network d

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Reports on the topic "Excitatory neurotransmitter"

1

Harris, Gregory, Brooke Hatchell, Davelin Woodard, and Dwayne Accardo. Intraoperative Dexmedetomidine for Reduction of Postoperative Delirium in the Elderly: A Scoping Review. University of Tennessee Health Science Center, 2021. http://dx.doi.org/10.21007/con.dnp.2021.0010.

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Background/Purpose: Post-operative delirium leads to significant morbidity in elderly patients, yet there is no regimen to prevent POD. Opioid use in the elderly surgical population is of the most significant risk factors for developing POD. The purpose of this scoping review is to recognize that Dexmedetomidine mitigates cognitive dysfunction secondary to acute pain and the use of narcotic analgesia by decreasing the amount of norepinephrine (an excitatory neurotransmitter) released during times of stress. This mechanism of action also provides analgesia through decreased perception and modul

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