Relevant bibliographies by topics / Experimental hypoxia

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Experimental hypoxia'

Author: Grafiati
Published: 28 May 2022
Last updated: 31 July 2025

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Journal articles on the topic "Experimental hypoxia"

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Khairova, V. R. "Changes of the activity of glutamine synthetase in an experimental model of hypoxic preconditioning." Azerbaijan Journal of Physiology, no. 2 (December 30, 2022): 47–52. http://dx.doi.org/10.59883/ajp.11.

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In the present work, the effect of hypoxic preconditioning on the activity of glutamine synthetase in the brain structures of old rats subjected to severe hypoxia was studied. The study of metabolic changes, in particular, some enzymes of glutamate metabolism, in the brain under conditions of oxygen deficiency and during hypoxic training is an actual problem, the solution of which makes it possible to determine possible ways to protect the nervous tissue from hypoxic damage and increase its stability. Hypoxia does not always induce cell death; under certain conditions, hypoxic exposure has a n
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Yang, Zhiyuan, Cristina Marcoci, Hatice Kübra Öztürk, et al. "Tissue Hypoxia and Associated Innate Immune Factors in Experimental Autoimmune Optic Neuritis." International Journal of Molecular Sciences 25, no. 5 (2024): 3077. http://dx.doi.org/10.3390/ijms25053077.

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Visual loss in acute optic neuritis is typically attributed to axonal conduction block due to inflammatory demyelination, but the mechanisms remain unclear. Recent research has highlighted tissue hypoxia as an important cause of neurological deficits and tissue damage in both multiple sclerosis (MS) and experimental autoimmune encephalomyelitis (EAE) and, here, we examine whether the optic nerves are hypoxic in experimental optic neuritis induced in Dark Agouti rats. At both the first and second peaks of disease expression, inflamed optic nerves labelled significantly for tissue hypoxia (namel
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Popovtseva, A. V., E. V. Suzopov, and Yu V. Korenovsky. "Acute hypoxic hypoxia increases lactate concentration in amniotic fluid of rabbits on 27-28th day of pregnancy." Biomeditsinskaya Khimiya 63, no. 1 (2017): 81–84. http://dx.doi.org/10.18097/pbmc20176301081.

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We evaluated the influence of hypoxic hypoxia on lactate, creatinine and urea concentrations in the amniotic fluid (AF) of rabbits on 27-28th day of pregnancy. Rabbits were randomly sudivided into two groups: experimental (n=9) and control (n=6). Rabbits of experimental groups were placed in a hypoxic chamber containing 10±2% oxygen and 90±2% nitrogen for 1 h and then were euthanized, AF was extracted from the amniotic sacs via disposable syringe. Acute hypoxic hypoxia had no effect on the AF volume, increased (1.4-fold) lactate, (1.3-fold) creatinine and (1.1-fold) urea concentrations in AF.
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Low, P. A., J. D. Schmelzer, K. K. Ward, and J. K. Yao. "Experimental chronic hypoxic neuropathy: relevance to diabetic neuropathy." American Journal of Physiology-Endocrinology and Metabolism 250, no. 1 (1986): E94—E99. http://dx.doi.org/10.1152/ajpendo.1986.250.1.e94.

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The cardinal electrophysiological abnormalities in experimental diabetic (EDN) and experimental galactose (EGN) neuropathy, models in which endoneurial hypoxia has been demonstrated, are a slowing in nerve conduction velocity (NCV) and a resistance to ischemic conduction block (RICB). These electrophysiological abnormalities are also present in human diabetic neuropathy, where microangiopathy has been demonstrated to be more severe than in EDN so that endoneurial hypoxia is also likely to be present. We examined the effects of endoneurial hypoxia per se on normal nerves. In rats subjected to c
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Tregub, Pavel P., Vladimir P. Kulikov, Irada Ibrahimli, et al. "Molecular Mechanisms of Neuroprotection after the Intermittent Exposures of Hypercapnic Hypoxia." International Journal of Molecular Sciences 25, no. 7 (2024): 3665. http://dx.doi.org/10.3390/ijms25073665.

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The review introduces the stages of formation and experimental confirmation of the hypothesis regarding the mutual potentiation of neuroprotective effects of hypoxia and hypercapnia during their combined influence (hypercapnic hypoxia). The main focus is on the mechanisms and signaling pathways involved in the formation of ischemic tolerance in the brain during intermittent hypercapnic hypoxia. Importantly, the combined effect of hypoxia and hypercapnia exerts a more pronounced neuroprotective effect compared to their separate application. Some signaling systems are associated with the predomi
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Joyce, William, Karlina Ozolina, Florian Mauduit, Hélène Ollivier, Guy Claireaux, and Holly A. Shiels. "Individual variation in whole-animal hypoxia tolerance is associated with cardiac hypoxia tolerance in a marine teleost." Biology Letters 12, no. 1 (2016): 20150708. http://dx.doi.org/10.1098/rsbl.2015.0708.

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Hypoxia is a pervasive problem in coastal environments and is predicted to have enduring impacts on aquatic ecosystems. Intraspecific variation in hypoxia tolerance is well documented in fish; however, the factors underlying this variation remain unknown. Here, we investigate the role of the heart in individual hypoxia tolerance of the European sea bass ( Dicentrarchus labrax ). We found individual whole-animal hypoxia tolerance is a stable trait in sea bass for more than 18 months (duration of study). We next examined in vitro cardiac performance and found myocardial muscle from hypoxia-toler
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Dzhalilova, D. Sh, A. M. Kosyreva, I. S. Tsvetkov, and O. V. Makarova. "Phagocytic activity of peripheral blood monocytes under <i>in vivo</i> and <i>in vitro</i> hypoxia conditions in tolerant and susceptible to oxygen deficiency rats." Medical Immunology (Russia) 25, no. 3 (2023): 551–56. http://dx.doi.org/10.15789/1563-0625-pao-2779.

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It is known that there are individual differences in resistance to hypoxia, which can determine the predisposition to the development and severity of various diseases, including infectious, inflammatory and tumor. There are no standardized methods for assessing resistance to hypoxia in experimental animals and humans without hypoxic exposure. The search for molecular-biological markers, identifying people with different resistance to oxygen deficiency under normoxic conditions or under moderate hypoxic exposure is undoubtedly efficient. It is possible that the assessment of the basic resistanc
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Belik, Jaques, Anna Sienko, and R. Bruce Light. "The effect of repeated intermittent hypoxia on pulmonary vasoconstriction in the newborn." Canadian Journal of Physiology and Pharmacology 68, no. 3 (1990): 355–62. http://dx.doi.org/10.1139/y90-049.

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The effect of repeated intermittent hypoxia upon the basal pulmonary vascular tone in the newborn period is unknown. We therefore studied the central hemodynamic response to seven repeated intermittent hypoxic challenges in acutely prepared piglets under 2 weeks of age. Catheters were placed in the aorta, pulmonary artery, and atria, and an electromagnetic flow probe was positioned around the main pulmonary artery. Each hypoxic challenge (Fio2 = 0.14) lasted 5 min, and was separated by an equal duration of ventilation with air. Nine control animals were ventilated with air for 90 min, a period
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Ono, Yoko, and Hidemasa Bono. "Multi-Omic Meta-Analysis of Transcriptomes and the Bibliome Uncovers Novel Hypoxia-Inducible Genes." Biomedicines 9, no. 5 (2021): 582. http://dx.doi.org/10.3390/biomedicines9050582.

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Hypoxia is a condition in which cells, tissues, or organisms are deprived of sufficient oxygen supply. Aerobic organisms have a hypoxic response system, represented by hypoxia-inducible factor 1-α (HIF1A), to adapt to this condition. Due to publication bias, there has been little focus on genes other than well-known signature hypoxia-inducible genes. Therefore, in this study, we performed a meta-analysis to identify novel hypoxia-inducible genes. We searched publicly available transcriptome databases to obtain hypoxia-related experimental data, retrieved the metadata, and manually curated it.
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Ancel, Julien, Jeanne-Marie Perotin, Maxime Dewolf, et al. "Hypoxia in Lung Cancer Management: A Translational Approach." Cancers 13, no. 14 (2021): 3421. http://dx.doi.org/10.3390/cancers13143421.

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Lung cancer represents the first cause of death by cancer worldwide and remains a challenging public health issue. Hypoxia, as a relevant biomarker, has raised high expectations for clinical practice. Here, we review clinical and pathological features related to hypoxic lung tumours. Secondly, we expound on the main current techniques to evaluate hypoxic status in NSCLC focusing on positive emission tomography. We present existing alternative experimental approaches such as the examination of circulating markers and highlight the interest in non-invasive markers. Finally, we evaluate the relev
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Dissertations / Theses on the topic "Experimental hypoxia"

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Kasti, M. "Experimental neuroinflammation : a study of hypoxia and protein translation." Thesis, University College London (University of London), 2013. http://discovery.ucl.ac.uk/1413014/.

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Multiple sclerosis is a chronic inflammatory disease of the CNS associated with widespread primary demyelination and axonal degeneration. The mechanisms underlying the expression of neurological deficits are incompletely understood. Recent histological findings are consistent with a view that active MS lesions may be hypoxic (i.e. suffer a low oxygen concentration). For example, inflammatory ‘Pattern III’ MS lesions express hypoxia inducible factor-1a (HIF-1a), a master regulator of genes whose function is to bias a cell for survival under hypoxic conditions. Hypoxia also results in a number o
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Тарасова, Ірина Віталіївна, Ирина Витальевна Тарасова, Iryna Vitaliivna Tarasova та ін. "Особливості забезпечення залізом тканин головного мозку щурів у разі експериментальної гіпоксії". Thesis, Видавництво СумДУ, 2012. http://essuir.sumdu.edu.ua/handle/123456789/27482.

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Тарасова, Ірина Віталіївна, Ирина Витальевна Тарасова, Iryna Vitaliivna Tarasova та ін. "Вплив експерементальної гіпоксії на вміст марганцю в органах новонароджених щурів". Thesis, Видавництво СумДУ, 2012. http://essuir.sumdu.edu.ua/handle/123456789/27481.

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Alfaus, Lafee Hussein, and Gharaibeh Mohammed. "Effects of experimental hypoxia on zinc balance in the brain tissue of rats." Thesis, Sumy State University, 2016. http://essuir.sumdu.edu.ua/handle/123456789/48131.

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Micronutrient disbalance is one of the mechanisms of damage of membranes. The role of microelements and especially ultramicroelements in metabolic adaptation of newborns on the background of hypoxia is staying unknown. Research purpose: to research zinc cerebral tissue supply in case of experimental hypoxia with different degrees of complexity. Microelement supply for zinc was studied as well as the lead level of cerebral tissue in experimental hypoxia conditions. Microelement supply was investigated on 44 laboratory rodents on their first and seventh days.
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Samee, Al Musa, and Sadid Laith. "Effects of experimental hypoxia on iron balance in the brain tissue of rats." Thesis, Sumy State University, 2016. http://essuir.sumdu.edu.ua/handle/123456789/48150.

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Hypoxic-ischemic lesion is one of important problems of neonatology, which is determined by their place in the structure of morbidity, perinatal mortality and a value in the disorders formation. Microelements provide course of important biological reactions and are catalysts of many of them. Research purpose: to research iron cerebral tissue supply in case of experimental hypoxia with different degrees of complexity.
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Griffin, Harry Sebastian. "Cardiovascular and respiratory effects of exposure to experimental models of obstructive sleep apnoea-related intermittent hypoxia." Thesis, University of Birmingham, 2013. http://etheses.bham.ac.uk//id/eprint/4039/.

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Obstructive sleep apnoea (OSA) is a common chronic condition characterised by repetitive nocturnal upper airway collapse that evokes intermittent hypoxia (IH). Although animal research has demonstrated a causal relationship between IH and cardiovascular disease there is a relative paucity of human research. Using a variety of different models of OSA we investigated the effects of IH on respiratory control and oxidative stress. In addition, we attempted to use Doppler ultrasound to investigate the effects of hypoxic airway occlusions on the pulmonary circulation but concluded that it is not a f
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Williams, K. J., M. R. Albertella, B. Fitzpatrick, et al. "In vivo activation of the hypoxia-targeted cytotoxin AQ4N in human tumor xenograft." AACR Publications, 2009. http://hdl.handle.net/10454/4561.

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no<br>AQ4N (banoxantrone) is a prodrug that, under hypoxic conditions, is enzymatically converted to a cytotoxic DNA-binding agent, AQ4. Incorporation of AQ4N into conventional chemoradiation protocols therefore targets both oxygenated and hypoxic regions of tumors, and potentially will increase the effectiveness of therapy. This current pharmacodynamic and efficacy study was designed to quantify tumor exposure to AQ4 following treatment with AQ4N, and to relate exposure to outcome of treatment. A single dose of 60 mg/kg AQ4N enhanced the response of RT112 (bladder) and Calu-6 (lung) xenograft
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Silveira, Eduardo Almeida da. "Modelo experimental de hipóxia normobárica intermitente em cavalos não treinados." Universidade Federal de Santa Maria, 2009. http://repositorio.ufsm.br/handle/1/10055.

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Coordenação de Aperfeiçoamento de Pessoal de Nível Superior<br>People living at high altitudes present compensatory adjustments to the hypoxic stimulation, such as physiological, morphological and functional adaptations. Humans have been submitted to intermittent hypoxic training (IHT) to achieve better athletic performance and to treat chronic diseases. The benefits related to IHT have been attributed to hematological, muscular, mitochondrial and plasmatic changes. An experimental model of altitude simulation was used on horses to assess the response of clinical, hematological and blood gas p
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Etheridge, Steven. "An acute experimental model of hearing loss using noise in combination with hypoxia and its use in the investigation of a putative cochleoprotectant." Thesis, University of Leicester, 2002. http://hdl.handle.net/2381/30762.

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The aim of this study was to develop a model of acute noise induced hearing loss that focused on excitotoxic damage, in the albino guinea pig. The model used moderate 12% hypoxia in combination with 100 dB SPL bandpass 5-20 kHz noise. The model employed gross electrophysiological measures as auditory endpoints, comprising the brainstem auditory evoked response (BAER) the cochlear compound action potential (CAP), and the ensemble spontaneous or driven cochlear nerve activity (ESAC/EDAC) derived from the Fast Fourier Transform of gross cochlear nerve (CN) activity. Preliminary model development
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Xavier, Marcelo Souza. "Efeito do azul de metileno como adjuvante no desfecho da parada cardíaca: estudo experimental em ratos." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/5/5152/tde-02072018-123155/.

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INTRODUÇAO: O uso da epinefrina na ressuscitação cardiopulmonar (RCP) tem sido questionado devido aos efeitos adversos como dano miocárdico e cerebral. Fármacos como azul de metileno têm sido estudados como adjuvantes, objetivando reduzir essas lesões. OBJETIVOS: Neste estudo objetivou-se avaliar o efeito da administração do azul de metileno em bôlus durante a RCP, na lesão miocárdica e cerebral. MÉTODO: Quarenta e nove ratos Wistar machos submetidos a parada cardíaca por fibrilação ventricular foram distribuídos aleatoriamente em quatro grupos com 11 animais: azul de metileno (GA, 2mg/kg), so
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Books on the topic "Experimental hypoxia"

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(Editor), Robert Roach, Peter D. Wagner (Editor), and Peter Hackett (Editor), eds. Hypoxia and Exercise (Advances in Experimental Medicine and Biology). Springer, 2006.

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(Editor), Robert C. Roach, Peter D. Wagner (Editor), and Peter H. Hackett (Editor), eds. Hypoxia: Through the Lifecycle (Advances in Experimental Medicine and Biology). Springer, 2003.

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Hypoxia: From Genes to the Bedside (Advances in Experimental Medicine and Biology). Springer, 2002.

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(Editor), Robert C. Roach, Peter D. Wagner (Editor), and Peter H. Hackett (Editor), eds. Hypoxia: Into the Next Millennium (ADVANCES IN EXPERIMENTAL MEDICINE AND BIOLOGY Volume 474). Springer, 1999.

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(Editor), Yoshiaki Hayashida, Constancio Gonzalez (Editor), and Hisatake Kondo (Editor), eds. The Arterial Chemoreceptors (Advances in Experimental Medicine and Biology). Springer, 2006.

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(Editor), Jean-Marc Pequignot, Constancio Gonzalez (Editor), Colin A. Nurse (Editor), Nanduri R. Prabhakar (Editor), and Yvette Dalmaz (Editor), eds. Chemoreception: From Cellular Signalling to Functional Plasticity (Advances in Experimental Medicine and Biology). Springer, 2003.

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Oxygen Transport to Tissue XXV (Advances in Experimental Medicine and Biology). Springer, 2003.

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Stocchetti, Nino, and Marco Carbonara. Pharmacologic Neuroprotection. Edited by David L. Reich, Stephan Mayer, and Suzan Uysal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190280253.003.0002.

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Acute cerebral injury sets into motion a cascade of deleterious biochemical events that cause further neuronal damage and amplify deleterious effects. This cascade develops over time and potentially may be attenuated or limited by pharmacologic manipulation. The neuroprotective properties of several molecules have been clearly demonstrated in experimental models of various pathologies. Based on these findings, many promising compounds have been tested in clinical trials. Large randomized controlled trials, however, have repeatedly failed to provide evidence of clinical efficacy. The authors pr
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Book chapters on the topic "Experimental hypoxia"

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Chang, Joan, and Janine Erler. "Hypoxia-Mediated Metastasis." In Advances in Experimental Medicine and Biology. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-5915-6_3.

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Krzykawska-Serda, Martyna, Richard C. Miller, Martyna Elas, et al. "Correlation Between Hypoxia Proteins and EPR-Detected Hypoxia in Tumors." In Advances in Experimental Medicine and Biology. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-55231-6_42.

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Engin, Atilla. "Adipose Tissue Hypoxia in Obesity: Clinical Reappraisal of Hypoxia Hypothesis." In Advances in Experimental Medicine and Biology. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-63657-8_11.

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Höpfl, Gisele, Omolara Ogunshola, and Max Gassmann. "Hypoxia and High Altitude." In Advances in Experimental Medicine and Biology. Springer US, 2003. http://dx.doi.org/10.1007/978-1-4419-8997-0_7.

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Marina, Nephtali, Vitaliy Kasymov, Gareth L. Ackland, Sergey Kasparov, and Alexander V. Gourine. "Astrocytes and Brain Hypoxia." In Advances in Experimental Medicine and Biology. Springer US, 2016. http://dx.doi.org/10.1007/978-1-4899-7678-9_14.

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Belikova, Maria V., Evgenia E. Kolesnikova, and Tatiana V. Serebrovskaya. "Intermittent Hypoxia and Experimental Parkinson’s Disease." In Intermittent Hypoxia and Human Diseases. Springer London, 2012. http://dx.doi.org/10.1007/978-1-4471-2906-6_12.

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Mayer, H. M., E. Fritschka, and J. Cervós-Navarro. "Regional Cerebral Blood Flow in Experimental Chronic Hypoxic Hypoxia." In Cerebral Blood Flow and Metabolism Measurement. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70054-5_74.

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Baer, Robert W. "The Influence of Hypoxic Hypoxia on Coronary Blood Flow Heterogeneity." In Advances in Experimental Medicine and Biology. Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-1875-4_44.

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Mankovskaya, I. N., and K. G. Lyabakh. "Oxygen Transport to Muscular Tissue under Adaptation to Hypoxic Hypoxia." In Advances in Experimental Medicine and Biology. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4717-4_36.

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Evans, S. M., M. Bergeron, D. M. Ferriero, et al. "Imaging Hypoxia in Diseased Tissues." In Advances in Experimental Medicine and Biology. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5399-1_84.

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Conference papers on the topic "Experimental hypoxia"

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Silva, Alejandro, Miguel Arocena, Ariel Fernández, and Julia R. Alonso. "Quantitative Phase Microscopy of hypoxic epithelial cells using the Transport of Intensity Equation." In Digital Holography and Three-Dimensional Imaging. Optica Publishing Group, 2024. http://dx.doi.org/10.1364/dh.2024.m3a.3.

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Quantitative Phase Imaging of epithelial cells under different levels of hypoxia can be achieved by acquiring a multifocus stack with a 3D printed microscope and using the Transport of Intensity Equation. Experimental results are provided.
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Scripnic, Vitalie, and Valeriu Cobet. "Beneficial effects of hypoxia on vascular reactivity and peculiarities of aging (clinical and experimental data)." In Universitas Europaea: Towards a Knowledge Based Society Through Europeanisation and Globalisation. Free International University of Moldova, 2025. https://doi.org/10.54481/uekbs2024.v2.38.

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This clinic-experimental study aimed the estimation of functional effects of adaptation to hypobaric hypoxia exerted on cardiovascular system. During the 14 days 28 volunteers have been exposed on mountain altitude of 2000m, considering as an optimal level of adaptation to hypoxia. The dynamics (2nd, 13th days of altitude stay and 1 month of basal conditions) of the heart rate, rhythm variability, activity of sympatho-/parasympathetic systems, rhythmogram, scatterogram, cardiac output, systolic and diastolic blood pressure as well as the total peripheral resistance was analyzed. Experimental s
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Hassan, Md Sazzad, Marisa Lenga, Lucia Petrova, and Urs von Holzen. "Abstract 1230: Therapeutic targeting of hypoxia in experimental esophageal adenocarcinoma." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-1230.

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Hassan, Md Sazzad, Marisa Lenga, Lucia Petrova, and Urs von Holzen. "Abstract 1230: Therapeutic targeting of hypoxia in experimental esophageal adenocarcinoma." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-1230.

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Peters, Dorothea M., Christina Klöpping, Karsten Krüger, et al. "Prevention Of Experimental Hypoxia-Induced Pulmonary Hypertension By Moderate Exercise Training In Mice." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4967.

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Palatova, T. V., G. N. Maslyakova, A. B. Bucharskaya, E. S. Voronina, A. V. Medvedeva, and S. S. Pakhomiy. "The effect of chronic intrauterine hypoxia on the reproductive system fetus (experimental study)." In Scientific dialogue: Medical issues. TsNK MOAN, 2019. http://dx.doi.org/10.18411/sciencepublic-15-07-2019-03.

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Palatova, Tatyana V., Galina N. Maslyakova, Marina L. Chekhonatskaya, Alla B. Bucharskaya, Elina A. Genina, and Alexey N. Bashkatov. "Functional and morphological changes in the mother-placenta-fetus system during chronic hypoxia (experimental study)." In Saratov Fall Meeting 2018: Optical and Nano-Technologies for Biology and Medicine, edited by Valery V. Tuchin and Elina A. Genina. SPIE, 2019. http://dx.doi.org/10.1117/12.2523634.

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Donina, Zhanna. "Nonsteroid anti-inflammatory agents depress inflammation-related respiratory disoders and hypoxia-induced mortality. Experimental model." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.2758.

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Palatova, Tatyana, Galina Maslyakova, Alla Bucharskaya, Elina Genina, and Alexey Bashkatov. "Functional and morphological changes in the testicular tissue of rat newborns during chronic hypoxia: experimental study." In Saratov Fall Meeting 2019: Optical and Nano-Technologies for Biology and Medicine, edited by Valery V. Tuchin and Elina A. Genina. SPIE, 2020. http://dx.doi.org/10.1117/12.2563415.

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Makarov, A. F., and V. Yu Tkachuk. "ARTIFICIAL HYPOBIOSIS AS METHOD OF ACUTE ALTITUDE ILLNESS NEGATIVE IMPACT REDUCTION." In The 4th «OCCUPATION and HEALTH» International Youth Forum (OHIYF-2022). FSBSI «IRIOH», 2022. http://dx.doi.org/10.31089/978-5-6042929-6-9-2022-1-152-155.

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Introduction: While reaching ever new heights mankind met an altitude illness. A critical stage of altitude sickness, manifesting by loss of consciousness, convulsions, apnea and subsequent death, develops at above 7 km altitudes. Pilots, alpinists and extreme sportsmen are the main risk group. Acute oxygen starvation of body while significant hypobaric hypoxia is the main mechanism of altitude illness. It is proposed to reduce the level of metabolism (artificial hypobiosis) to prevent the negative impact of acute hypobaric hypoxia. The study goal to assess the efficiency of organism negative
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