Relevant bibliographies by topics / Expressão de caspase 8 e caspase 9

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Academic literature on the topic 'Expressão de caspase 8 e caspase 9'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Expressão de caspase 8 e caspase 9"

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Demirci, Umut, Melek Yaman, and Umit E. Bagriacik. "Effects of acute doxorubicin exposure on caspase-mediated apoptosis in cardiomyocytes." Journal of Clinical Oncology 31, no. 15_suppl (2013): e12036-e12036. http://dx.doi.org/10.1200/jco.2013.31.15_suppl.e12036.

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e12036 Background: Doxorubicin binds to DNA-associated enzymes, intercalates the base pairs of the DNA and induces apoptosis in cancerous and healthy tissues especially in cardiomyocytes. Caspase mediated apoptosis in cardiomyocytes remains largely unknown. We investigated the role of doxorubicin via caspase system on apoptosis of cardiomyocytes. Methods: H9C2ratcardiomyocytes were incubated with doxorubicin a concentration of 10-6 Mfor 4 or 24 hours to perform gene expression and activity of caspases, respectively. Total RNA isolated and expression analysis were performed by real time PCR ass
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Ekonomopoulou, Maria Themistokli, Evaggelos Babas, Eleutheria Mioglou-Kalouptsi, Maria Malandri, and Zafiroula Iakovidou-Kritsi. "Changes in Activities of Caspase-8 and Caspase-9 in Human Cervical Malignancy." International Journal of Gynecologic Cancer 21, no. 3 (2011): 435–38. http://dx.doi.org/10.1097/igc.0b013e31820d3e42.

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IntroductionThe apoptotic process of programmed cell death and its dysfunctions in a variety of human diseases, including cervical cancer, has become the focus of extensive scientific research. Caspases are considered key factors in the execution of apoptosis, although there are many aspects of their role to be elucidated. It has been found that disturbance of initiator caspase-8 and caspase-9 expression or function may contribute to cancer formation/progression, and inactivation of them could promote resistance to current treatment approaches. In our research, the activities of caspase-8 and
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Janečková, Eva, Petra Bíliková, and Eva Matalová. "Osteogenic Potential of Caspases Related to Endochondral Ossification." Journal of Histochemistry & Cytochemistry 66, no. 1 (2017): 47–58. http://dx.doi.org/10.1369/0022155417739283.

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Caspases have functions particularly in apoptosis and inflammation. Increasing evidence indicates novel roles of these proteases in cell differentiation, including those involved in osteogenesis. This investigation provides a complex screening of osteogenic markers affected by pan caspase inhibition in micromass cultures derived from mouse forelimbs. PCR Array analysis showed significant alterations in expression of 49 osteogenic genes after 7 days of inhibition. The largest change was a decrease in CD36 expression, which was confirmed at organ level by caspase inhibition in cultured mouse uln
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Cao, Guodong, Yumin Luo, Tetsuya Nagayama, et al. "Cloning and Characterization of Rat Caspase-9: Implications for a Role in Mediating Caspase-3 Activation and Hippocampal Cell Death after Transient Cerebral Ischemia." Journal of Cerebral Blood Flow & Metabolism 22, no. 5 (2002): 534–46. http://dx.doi.org/10.1097/00004647-200205000-00005.

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Delayed hippocampal neurodegeneration after transient global ischemia is mediated, at least in part, through the activation of terminal caspases, particularly caspase-3, and the subsequent proteolytic degradation of critical cellular proteins. Caspase-3 may be activated by the membrane receptor-initiated caspase-8–dependent extrinsic pathway and the mitochondria-initiated caspase-9–dependent intrinsic pathway; however, the precise role of these deduced apoptosis-signaling pathways in activating caspase-3 in ischemic neurons remains elusive. The authors cloned the caspase-9 gene from the rat br
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Peluffo, Marina C., Richard L. Stouffer, and Marta Tesone. "Activity and expression of different members of the caspase family in the rat corpus luteum during pregnancy and postpartum." American Journal of Physiology-Endocrinology and Metabolism 293, no. 5 (2007): E1215—E1223. http://dx.doi.org/10.1152/ajpendo.00261.2007.

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Studies were designed to examine the expression and activity of four caspases that contribute to the initial (caspases-2, -8, and -9) and final (caspase-3) events in apoptosis in the rat corpus luteum (CL) during pregnancy ( days 7, 17, 19, and 21 of gestation), postpartum ( days 1 and 4), and after injection (0, 8, 16, 24, and 36 h) of the physiological luteolysin PGF2α. In addition, the temporal relationship of caspase expression/activity relative to steroid production and luteal regression was evaluated. During pregnancy, the activity of all four caspases was significantly greater on day 19
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Rathbun, R. Keaney, Tracy A. Christianson, Gregory R. Faulkner та ін. "Interferon-γ–induced apoptotic responses of Fanconi anemia group C hematopoietic progenitor cells involve caspase 8–dependent activation of caspase 3 family members". Blood 96, № 13 (2000): 4204–11. http://dx.doi.org/10.1182/blood.v96.13.4204.h8004204_4204_4211.

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Hematopoietic progenitor cells (HPC) from mice nullizygous at the Fanconi anemia (FA) group C locus and children with Fanconi anemia group C (FA-C) are hypersensitive to interferon-gamma (IFN-γ) and tumor necrosis factor-α. This hypersensitivity results, in part, from the capacity of these cytokines to prime the fas pathway. Because fas-mediated programmed cell death in many cells involves sequential activation of specific caspases, we tested the hypothesis that programmed cell death in FA HPC involves the ordered activation of specific caspase molecules. Lysates from lymphoblasts treated with
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Smith, Mitchell Reed, Fang Jin, and Indira Joshi. "Antibodies to TRAIL Receptors R1 and R2 Induce Apoptosis in Non-Hodgkin’s Lymphoma Cells." Blood 104, no. 11 (2004): 3290. http://dx.doi.org/10.1182/blood.v104.11.3290.3290.

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Abstract Monoclonal antibodies and chemotherapy can be effective, but not curative, therapy for non-Hodgkin s lymphoma (NHL). Rituximab acts by several mechanisms, including directly signaling apoptosis of CD20+ cells via the mitochondrial pathway involving caspase 9. Chemotherapy also activates this apoptotic pathway. TRAIL-R1 and -R2 signaling induces apoptosis via a pathway that activates caspase 8. Thus, we investigated the effects of agonistic monoclonal antibodies to TRAIL-R1 and -R2 on NHL cell lines (DoHH2, WSU-FSCCL and FC-TxFL2, each t(14;18)+, EBV-). TRAIL-R1 (HGS-ETR1) and -R2 (HGS
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Gaddy, Daniel F., and Douglas S. Lyles. "Vesicular Stomatitis Viruses Expressing Wild-Type or Mutant M Proteins Activate Apoptosis through Distinct Pathways." Journal of Virology 79, no. 7 (2005): 4170–79. http://dx.doi.org/10.1128/jvi.79.7.4170-4179.2005.

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ABSTRACT Vesicular stomatitis virus (VSV) induces apoptosis by at least two mechanisms. The viral matrix (M) protein induces apoptosis via the mitochondrial pathway due to the inhibition of host gene expression. However, in some cell types, the inhibition of host gene expression by VSV expressing wild-type (wt) M protein delays VSV-induced apoptosis, indicating that another mechanism is involved. In support of this, the recombinant M51R-M (rM51R-M) virus, expressing a mutant M protein that is defective in its ability to inhibit host gene expression, induces apoptosis much more rapidly in L929
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Rathbun, R. Keaney, Tracy A. Christianson, Gregory R. Faulkner та ін. "Interferon-γ–induced apoptotic responses of Fanconi anemia group C hematopoietic progenitor cells involve caspase 8–dependent activation of caspase 3 family members". Blood 96, № 13 (2000): 4204–11. http://dx.doi.org/10.1182/blood.v96.13.4204.

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Abstract Hematopoietic progenitor cells (HPC) from mice nullizygous at the Fanconi anemia (FA) group C locus and children with Fanconi anemia group C (FA-C) are hypersensitive to interferon-gamma (IFN-γ) and tumor necrosis factor-α. This hypersensitivity results, in part, from the capacity of these cytokines to prime the fas pathway. Because fas-mediated programmed cell death in many cells involves sequential activation of specific caspases, we tested the hypothesis that programmed cell death in FA HPC involves the ordered activation of specific caspase molecules. Lysates from lymphoblasts tre
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Papandile, Adrian, David Tyas, Donald M. O'Malley, and Carol M. Warner. "Analysis of caspase-3, caspase-8 and caspase-9 enzymatic activities in mouse oocytes and zygotes." Zygote 12, no. 1 (2004): 57–64. http://dx.doi.org/10.1017/s0967199404002588.

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The current consensus in the literature is that ovulated oocytes that are not fertilized die by apoptosis, but the details of the proteins involved in the apoptotic pathways have not been elucidated. In this paper we confirm that caspase-3, the executioner of apoptosis, is expressed in mouse oocytes, and show that two initiators of apoptosis, caspase-8 and caspase-9, are expressed in mouse oocytes. Comparisons were made of caspase-3, -8, and -9 activities in superovulated oocytes that were freshly collected or allowed to age in vivo or in vitro. We found that caspase-3 activity significantly i
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Dissertations / Theses on the topic "Expressão de caspase 8 e caspase 9"

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Cacere, Camila Rodrigues. "Aspectos moleculares envolvidos na apoptose de células mononucleares em pacientes com paracoccidioidomicose." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/42/42132/tde-19092008-095609/.

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A hiporreatividade das células T observada na resposta imune a antígenos de P. brasiliensis de pacientes com paracoccidioidomicose ativa deve contribuir para o não controle da doença, levando à disseminação do fungo. É, na maioria das vezes, reversível com tratamento antifúngico. Os mecanismos que levam a esta hiporreatividade não são bem conhecidos. No entanto, foram demonstrados em resultados prévios em nosso laboratório que células mononucleares de pacientes frente a gp43 apresentam níveis elevados de apoptose. Para tentarmos explicar esse mecanismo, nossa primeira hipótese foi de avaliar
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Chiang, Michelle. "Cardamonin induces apoptosis in human nasopharyngeal carcinoma cells via mitochondrial death pathway mediated by caspase-3 and caspase-8 activation, independent of caspase-9 signalling responses." Thesis, University of Nottingham, 2016. http://eprints.nottingham.ac.uk/32565/.

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Nasopharyngeal cancer lies in the upper part of throat behind the nose and near the base of the skull called the nasopharynx. It is more commonly diagnosed in parts of Asia, particularly in the southern China. Five local edible plants from different families; namely curry leaf (Murraya koenigii), temu kunci (Boesenbergia rotunda), spring onion leaf (Allium cepa), mushroom bean (Phaseolus vulgaris) and bunga kantan (Phaeomeria imperialis) were macerated to obtain methanol, ethyl acetate and hexane crude extracts. Each crude extract was tested against nasopharyngeal carcinoma (HK-1) and normal n
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Cagnol, Sébastien. "Contrôle de la mort cellulaire par la voie des MAPK1/3 (ERK2/1)." Phd thesis, Université de Nice Sophia-Antipolis, 2005. http://tel.archives-ouvertes.fr/tel-00104792.

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La mort cellulaire programmée ou apoptose est un mécanisme conservé chez les eucaryotes multicellulaires qui contribue au développement embryonnaire et à l'homéostasie cellulaire des organismes. Dans les cellules vivantes, l'activité des protéases qui exécutent le programme de mort cellulaire, les caspases, est contrôlée par des signaux de survie provenant de l'environnement cellulaire. Les caspases initiatrices de l'apoptose régulée par l'environnement, la caspase 9 et la caspase 8 sont activées respectivement par l'apoptosome et par les récepteurs de mort. Les signaux environnementaux, parmi
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Ishii, Priscila Lumi [UNESP]. "Efeito α-tomatina na proliferação celular, apoptose e expressão de RNAm dos genes APC, Ciclina A2, Catenina, CASP9, BAK, BAX e BCL-XL em células HT29". Universidade Estadual Paulista (UNESP), 2011. http://hdl.handle.net/11449/87688.

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Made available in DSpace on 2014-06-11T19:22:58Z (GMT). No. of bitstreams: 0 Previous issue date: 2011-02-17Bitstream added on 2014-06-13T18:08:59Z : No. of bitstreams: 1 ishii_pl_me_rcla.pdf: 341469 bytes, checksum: 30957c71a427ca667e0c86edb9d40228 (MD5)<br>Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)<br>A Nutrigenômica é definida como o efeito da dieta na expressão gênica, e a extensão pela qual as diferenças genéticas entre os indivíduos influenciam a resposta a um padrão específico de dieta, à ingestão de alimentos funcionais e à suplementação de micronutrientes, e
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Luzio, Ana Catarina Gonçalves. "Avaliação da expressão dos genes de caspase 8 e caspase 9 em Peixe-Zebra (Danio Rerio) expostos a cobre." Master's thesis, 2008. http://hdl.handle.net/10348/2153.

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Dissertação de Mestrado em Biologia Clínica Laboratorial<br>O cobre é um micronutriente essencial que está presente em condições naturais em águas não poluídas. Nas últimas décadas, os ecossistemas aquáticos têm sido contaminados por diversos poluentes resultantes da actividade mineira, da agricultura, da indústria, bem como dos efluentes domésticos. Em consequência, existe um grande interesse na determinação de biomarcadores de exposição ao cobre em peixes de água doce, com vista quer à monitorização dos ecossistemas aquáticos, quer como instrumento de análise da sua toxicidade. Com o objecti
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Santos, Stefanie Lopes dos. "Potencial apoptótico do cobre: estudo in vitro: expressão das caspases 3, 8 e 9, do AIF e da proteína p53." Master's thesis, 2014. http://hdl.handle.net/10348/6395.

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Dissertação de Mestrado em Biologia Clinica Laboratorial<br>A apoptose define-se como um processo de morte celular programada, controlada geneticamente, que possui um papel essencial no desenvolvimento e na manutenção da homeostasia nos organismos, sendo fundamental na eliminação de células ectópicas, danificadas, mutantes e infetadas. A ativação da apoptose pode ser desencadeada através de duas vias: a via extrínseca ou citoplasmática, onde os recetores de membrana funcionam como ponto de iniciação do processo apoptótico, e a intrínseca ou mitocondrial, na qual a mitocôndria tem o papel princ
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Book chapters on the topic "Expressão de caspase 8 e caspase 9"

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Widmann, Christian. "Caspase 8." In xPharm: The Comprehensive Pharmacology Reference. Elsevier, 2007. http://dx.doi.org/10.1016/b978-008055232-3.62945-9.

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van Raam, Bram J., and Guy S. Salvesen. "Caspase-9." In Handbook of Proteolytic Enzymes. Elsevier, 2013. http://dx.doi.org/10.1016/b978-0-12-382219-2.00506-8.

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Taber, Douglass F. "Stereoselective C-O Ring Construction: (+)-Pachastrissamine (Fujii/Ohno), Aspalathin (Minehan), (+)-Varitriol (Ghosh), Aspercyclide A (Spivey), Etnangien (Menche)." In Organic Synthesis. Oxford University Press, 2013. http://dx.doi.org/10.1093/oso/9780199965724.003.0051.

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(+)-Pachastrissamine 3, also known as jaspine B, induces apoptosis in melanoma cells by a caspase-dependent pathway. Nobutaka Fujii and Hiroaki Ohno of Kyoyo University developed (J. Org. Chem. 2010, 75, 3831) a practical route to 3 based on the Pd-mediated cyclization of 1 to 2. Thomas G. Minehan of California State University, Northridge, optimized (Organic Lett. 2010, 12, 1580) the condensation of 5 with the bis-pivalate 4. This opened a general route to C-aryl glycosides, including aspalathin 6. (+)-Varitriol 11 is vinylogously related to 7. A key step in the synthesis of 11 reported (J. Org. Chem. 2010, 75, 2107) by Subhash Ghosh of the Indian Institute of Chemical Technology was the intermolecular Heck coupling of 8 with 9. Aspercyclide A 14 and its more stable methyl ether are promising lead compounds for the treatment of asthma. In the course of a synthesis of 14, Alan C. Spivey of Imperial College developed (Chem. Commun. 2010, 1824) the intramolecular Heck cyclization of 12 to 13. In a bold synthesis of etnangien 17, Dirk Menche of Ruprecht-Karls-Universität Heidelberg showed (J. Org. Chem. 2010, 75, 2429) that the intramolecular Heck coupling of 15 to 16 proceeded efficiently. The substituents on 15 may be favoring conformations that lead to cyclization.
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Conference papers on the topic "Expressão de caspase 8 e caspase 9"

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Mcglorthan, Latoya, Ana Paucarmayta, Yovanni Casablanca, George L. Maxwell, and Viqar Syed. "Abstract 1929: Progesterone induces apoptosis by activation of caspase-8 and calcitriol via activation of caspase-9 pathways in ovarian and endometrial cancer cellsin vitro." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-1929.

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Hernandez, Lidia, Anne M. Noonan, Ethan Sagher та ін. "Abstract POSTER-TECH-1102: Caspase 8 cooperates with IKKβ to protect ovarian cancer cells from necroptosis". У Abstracts: 10th Biennial Ovarian Cancer Research Symposium; September 8-9, 2014; Seattle, WA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1557-3265.ovcasymp14-poster-tech-1102.

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Kapoor, Gurpreet Singh, Christopher A. Benetatos, Yasuhiro Mitsuuchi, et al. "Abstract 2278: The SMAC-mimetic birinapant regulates autocrine TNF production by caspase-8:RIPK1 complex via p38MAPK pathway." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-2278.

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Ji, Yu bin, Chen feng Ji, Lei Yue, and He Xu. "Study on HepG-2 Apoptosis Induced by Saponins Isolated from Asparagus and the Effects on the Activities of Caspase-3, 8, 9." In 2009 2nd International Conference on Biomedical Engineering and Informatics. IEEE, 2009. http://dx.doi.org/10.1109/bmei.2009.5305815.

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Mitsuuchi, Yasuhiro, Christopher A. Benetatos, Thomas Haimowitz, et al. "Abstract 1806: Birinapant, a bivalent SMAC-mimetic, promotes efficient cellular IAP E3 ligase activity and formation of a pro-apoptotic RIPK1:caspase-8 complex while monovalent IAP inhibitors are less efficient - implications for therapeutic utility." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-1806.

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