Relevant bibliographies by topics / Facial Pain; Trigeminal Neuralgia; Laser Therapy

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Academic literature on the topic 'Facial Pain; Trigeminal Neuralgia; Laser Therapy'

Author: Grafiati
Published: 5 June 2025
Last updated: 2 August 2025

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Journal articles on the topic "Facial Pain; Trigeminal Neuralgia; Laser Therapy"

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Guilherme, Alves Aguiar, Luiz Barbosa Pinheiro Antônio, and Cristina Teixeira Cangussu Maira. "Use Of Laser Phototherapy In The Treatment Of Trigeminal Neuralgia." INTERNATIONAL JOURNAL OF HEALTH & MEDICAL RESEARCH 03, no. 03 (2024): 121–26. https://doi.org/10.5281/zenodo.10819867.

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Responsible for  the sensory stimuli of the viscerocranium, the 5th pair of cranial nerves, called the trigeminal nerve due to its 3 branches, is a mixed-function nerve. The diagnosis of Trigeminal Neuralgia is directly related to the symptoms reported by the patient during the anamnesis. The right treatment depends on the correct diagnosis. The exact etiology of TN is still unknown. It affects around 4 to 13 individuals per 100,000 and although it seems to be a neuropathy with a low incidence, it is the most common among facial pain syndromes. A retrospective cross-sectional observationa
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Saraswathi Gopal.K, Mahesh Kumar.P, and Harini. B. K. "Low level laser therapy in the management of trigeminal neuralgia: A rare case report." International Journal of Science and Research Archive 12, no. 1 (2024): 2026–30. http://dx.doi.org/10.30574/ijsra.2024.12.1.0938.

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Trigeminal neuralgia (TN) is characterized by sudden, severe, brief, and stabbing recurrent episodes of facial pain in one or more branches of the trigeminal nerve. Pain attacks can occur spontaneously or can be triggered by non-noxious stimuli, such as talking, eating, washing the face, brushing teeth, shaving, a light touch or even a cool breeze. In addition to pain attacks, a proportion of the patients also experience persistent background pain, which along with autonomic signs and prolonged disease duration, represent predictors of worse treatment outcomes. Here, we present a classical cas
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Martinez, Diego M., Billy K. Huh, and Saba Javed. "Case report: use of high-intensity laser therapy for treatment of trigeminal neuralgia." Pain Management 13, no. 12 (2023): 709–16. http://dx.doi.org/10.2217/pmt-2023-0112.

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Trigeminal neuralgia represents a form of chronic facial pain that is characterized by its incapacitating nature. The current therapeutic approaches encompass pharmacological agents with carbamazepine or non-pharmacologic options including utilization of percutaneous rhizotomy, Gamma knife radiosurgery or microvascular decompression may be indicated in certain cases. While the interventions may be effective, medications have negative side effects and procedures are invasive which can pose challenges for patients with various comorbidities. High-intensity laser therapy (HILT) has demonstrated s
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Eckerdal, Arne, and Lehmann Bastian. "CAN LOW REACTIVE-LEVEL LASER THERAPY BE USED IN THE TREATMENT OF NEUROGENIC FACIAL PAIN? A DOUBLE-BLIND, PLACEBO CONTROLLED INVESTIGATION OF PATIENTS WITH TRIGEMINAL NEURALGIA." LASER THERAPY 8, no. 4 (1996): 247–51. http://dx.doi.org/10.5978/islsm.8.247.

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Walker, J. B., L. K. Akhanjee, M. M. Cooney, J. Goldstein, S. Tamzyoshi, and F. Segal-Gidan. "Laser Therapy for Pain of Trigeminal Neuralgia." Clinical Journal of Pain 3, no. 4 (1987): 183–88. http://dx.doi.org/10.1097/00002508-198712000-00001.

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Mitra, Sukanya, Prateek Upadhyay, Jasveer Singh, and Prashant Ahlawat. "Noninvasive pulsed radiofrequency for trigeminal neuralgia: Off the beaten path – Case series." Saudi Journal of Anaesthesia 18, no. 4 (2024): 569–72. http://dx.doi.org/10.4103/sja.sja_208_24.

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Trigeminal neuralgia (TN) is a debilitating condition characterized by excruciating facial pain resulting from dysfunction in the trigeminal nerve. Treatment options are limited. Noninvasive pulsed radiofrequency (PRF) therapy is an emerging promising approach to managing TN. This therapy provides pain control without tissue damage or unpleasant side effects compared to invasive procedures. Here, we present five cases of trigeminal neuralgia successfully managed with noninvasive PRF therapy. Our case series contributes to the existing literature and supports the potential efficacy of noninvasi
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Mario Francesco, Fraioli, Lisciani Damiano, Pagano Andrea, and Fraioli Chiara. "Bilateral Trigeminal Neuralgia Refractory to Medical Therapy: Importance of A Multi-Therapeutic Approach." Archives of Case Reports 9, no. 1 (2025): 016–18. https://doi.org/10.29328/journal.acr.1001123.

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Bilateral trigeminal neuralgia refractory to medical therapy is a rare occurrence and it is mandatory to choose therapeutic procedures minimizing possible bilateral sensitive deficit due to the employment of bilateral mininvasive ablative techniques. A patient affected by bilateral trigeminal neuralgia refractory to medical therapy secondary to multiple sclerosis is presented. Multiple therapeutic tools were employed in this challenging pathology. The second and third left trigeminal divisions were involved by the neuralgia, while the third division was involved in the right facial side. Contr
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Kolakowski, Lukasz, Heiko Pohl, Lennart Stieglitz, et al. "Interdisciplinary strategies for diagnosis and treatment of trigeminal neuralgia." Swiss Medical Weekly 154, no. 7 (2024): 3460. http://dx.doi.org/10.57187/s.3460.

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Temporary, sudden, shooting and recurrent unilateral facial pain in the supply area of one or more trigeminal nerve branches characterises trigeminal neuralgia. Innocuous stimuli trigger the pain, e.g. chewing, speaking or brushing teeth. In some patients, paroxysms superimpose on continuous pain. In aetiological terms, idiopathic, classic (due to neurovascular compression) and secondary trigeminal neuralgia (e.g. due to multiple sclerosis, brainstem ischaemia and space-occupying lesions) are defined. Many drugs may be efficacious, with carbamazepine being first-choice therapy. However, non-ph
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Vásquez, Bélgica. "Trigeminal Neuralgy." International Journal of Medical and Surgical Sciences 1, no. 4 (2018): 353–58. http://dx.doi.org/10.32457/ijmss.2014.044.

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The objective of this review was to present information on the main causes, possible treatments and morpho-quantitative aspects of trigeminal neuralgia. Trigeminal neuralgia is a condition characterized by intense facial pain, severe throbbing or stabbing; it is usually unilateral and recurrent and is located in the facial area innervated by the trigeminal nerve. The causes of this disease are varied and include neurovascular compression stresses. Medical treatment of choice is carbamazepine, reserving surgical treatment for cases resistant to medical therapy or cases when side effects of drug
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Rulewska, Natalia Maria, Filip Grabowski, Dagmara Neska, et al. "Radiofrequency thermocoagulation of Gasserian ganglion in trigeminal neuralgia." Journal of Education, Health and Sport 77 (January 9, 2025): 56674. https://doi.org/10.12775/jehs.2025.77.56674.

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Introduction: Trigeminal neuralgia (TN) is a form of neuropathic pain that targets the fifth cranial nerve, leading to intense facial pain that can significantly impact one’s quality of life. This condition typically manifests as recurring episodes of sharp, unilateral pain along one or more branches of the trigeminal nerve.The exact mechanism of trigeminal neuralgia remains largely unknown. Most pain attacks are triggered by certain stimuli in the areas served by the trigeminal nerve. One possible treatment approach for trigeminal neuralgia is radiofrequency thermocoagulation. Aim: The aim of
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Book chapters on the topic "Facial Pain; Trigeminal Neuralgia; Laser Therapy"

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Grandhe, Radhika, Eli Johnson Harris, and Eugene Koshkin. "Trigeminal Neuralgia." In Neuropathic Pain, edited by Radhika Grandhe, Eli Johnson Harris, and Eugene Koshkin. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190298357.003.0030.

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Trigeminal neuralgia is a rare neuropathic pain condition but can be very disabling. The hallmark is brief episodes of intense, radiating pain within the territory of trigeminal nerve distribution. It is typically unilateral, often accompanied by facial spasms and can be triggered by facial movements in a majority of patients. Microvascular compression of trigeminal ganglion is the etiology for most patients with classical trigeminal neuralgia. Some patients can have continuous facial pain in addition to paroxysms of pain. Trigeminal neuralgia is a clinical diagnosis, but MRI is done to rule o
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Kim, Lily H., and Michael Bret Schneider. "Pain Psychiatry of Trigeminal Neuralgia." In Effective Techniques for Managing Trigeminal Neuralgia. IGI Global, 2018. http://dx.doi.org/10.4018/978-1-5225-5349-6.ch012.

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In addition to the repeated episodes of paroxysmal, electrical facial pain classically associated with the disease, many patients with trigeminal neuralgia (TN) suffer from severe emotional distress. At present, there is no universally agreed pathophysiological explanation for the high incidence of depression and anxiety within this patient population. Despite the unclear understanding, the psychiatric comorbidities should be addressed as a part of comprehensive, multi-modal approach. Anticonvulsants or serotonin and norepineprhine reuptake inhibitors are viable pharmacological options that ca
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Truini, Andrea, and Gianfranco de Stefano. "A Lightning in the Face." In Neuropathic Pain, edited by Nadine Attal and Didier Bouhassira. Oxford University PressNew York, 2023. http://dx.doi.org/10.1093/med/9780197616345.003.0019.

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Abstract This chapter presents the case of an old woman who suffers from facial pain characterized by intense attacks, limited to the right side of the face, involving the cheek, upper and lower lips, and the underlying gums, lasting fractions of a seconds, and sometimes occurring in bursts. Her clinical picture is typical of trigeminal neuralgia. Diagnosis is clinical, but a magnetic resonance imaging scan or complete trigeminal reflex testing is mandatory to exclude major neurological diseases. First-line therapy is medical and includes carbamazepine or oxcarbazepine. If the occurrence of si
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Costello, Kathleen, Rosalind Kalb, and Barbara S. Giesser. "Sensory Symptoms." In Navigating Life with Multiple Sclerosis, 2nd ed. Oxford University PressNew York, 2025. https://doi.org/10.1093/oso/9780197748695.003.0006.

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Abstract Changes in sensation, particularly touch sensation, and vision, can occur in multiple sclerosis (MS). Numbness and tingling, known as paresthesias, or painful sensations, known as dysethesias, can be disruptive to daily function. Various types of neuropathic pain, such as trigeminal neuralgia (facial pain), occipital neuralgia (scalp pain), and Lhermitte’s sign (pain down the back and legs with forward neck flexion), are possible in MS. Although other sensory changes can occur, including problems with hearing, taste, or smell, they are more likely to be caused by conditions other than
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Wong, Stanley Sau Ching, and Chi Wai Cheung. "Orofacial pain." In Oxford Textbook of Anaesthesia for Oral and Maxillofacial Surgery, Second Edition, 2nd ed. Oxford University PressOxford, 2022. http://dx.doi.org/10.1093/med/9780198790723.003.0019.

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Abstract Orofacial pain describes pain that is experienced in the face and/or oral cavity. Many of these patients suffer from chronic orofacial pain, which refers to pain lasting more than 3 months. Both acute and chronic orofacial pain are important clinical problems. In particular, chronic orofacial pain can be severe and result in long-term physical and psychosocial impairment. A detailed assessment through history, physical examination, and investigations is needed to delineate the underlying pain pathology, rule out red flag pathologies, and identify psychosocial barriers to pain relief.
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Nye, Barbara L., Thomas N. Ward, Naveen George, and Stewart J. Tepper. "“Biologic” Treatment for Primary Headaches." In Comprehensive Review of Headache Medicine, 2nd ed., edited by Morris Levin. Oxford University PressNew York, 2025. https://doi.org/10.1093/med/9780190888770.003.0026.

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Abstract Since the 1990s, physicians have suspected that botulinum toxins might be effective in the treatment of headache. Botulinum toxin prevents the release of neurotransmitters at cholinergic nerves (acetylcholine) and sensory nerves (glutamate, substance P, calcitonin gene-related peptide [CGRP], and others). OnabotulinumtoxinA has had U.S. Food and Drug Administration (FDA) approval for the treatment of chronic migraine since 2010. It also has FDA approval for treating cervical dystonia (torticollis), and it may not only improve the posture deformity associated with that condition but al
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