Relevant bibliographies by topics / Factor XI Deficiency

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Factor XI Deficiency'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Factor XI Deficiency"

1

Seligsohn, Uri. "Factor XI Deficiency." Thrombosis and Haemostasis 70, no. 01 (1993): 068–71. http://dx.doi.org/10.1055/s-0038-1646162.

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O’Connell, Niamh M. "Factor XI deficiency." Seminars in Hematology 41 (January 2004): 76–81. http://dx.doi.org/10.1053/j.seminhematol.2003.11.015.

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Beilin, Yaakov, and Daniel J. Katz. "Factor XI Deficiency." Anesthesia & Analgesia 128, no. 1 (2019): e10-e9. http://dx.doi.org/10.1213/ane.0000000000003869.

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Santoro, Rita, Simona Prejanò, and Piergiorgio Iannaccaro. "Factor XI deficiency." Blood Coagulation & Fibrinolysis 22, no. 5 (2011): 431–35. http://dx.doi.org/10.1097/mbc.0b013e32834689e4.

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Duga, Stefano, and Ophira Salomon. "Factor XI Deficiency." Seminars in Thrombosis and Hemostasis 35, no. 04 (2009): 416–25. http://dx.doi.org/10.1055/s-0029-1225764.

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Brush, Pamela J. "Bovine factor XI deficiency." In Practice 13, no. 1 (1991): 26–29. http://dx.doi.org/10.1136/inpract.13.1.26.

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Bolton-Maggs, Paula H. B. "10 Factor XI deficiency." Baillière's Clinical Haematology 9, no. 2 (1996): 355–68. http://dx.doi.org/10.1016/s0950-3536(96)80068-0.

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8

Martin-Salces, Monica, Victor Jimenez-Yuste, Maria Teresa Alvarez, Manuel Quintana, and Fernando Hernandez-Navarro. "Review: Factor XI Deficiency: Review and Management in Pregnant Women." Clinical and Applied Thrombosis/Hemostasis 16, no. 2 (2008): 209–13. http://dx.doi.org/10.1177/1076029608327864.

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Factor XI deficiency is a rare disease found predominantly in Ashkenazi Jews. There is a poor correlation between factor XI level and bleeding in patients with factor XI deficiency. Individuals with severe factor XI deficiency are usually at risk of excessive bleeding after surgery and injury, particularly when trauma involves tissues rich in fibrinolytic activity. Women with partial or severe deficiency are at risk of excessive uterine bleeding during labor. The unpredictable nature of factor XI deficiency complicates management during pregnancy and delivery. This review gives an overview of
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9

Troxel, Mark T., Marjory B. Brooks, and Meredith L. Esterline. "Congenital Factor XI Deficiency in a Domestic Shorthair Cat." Journal of the American Animal Hospital Association 38, no. 6 (2002): 549–53. http://dx.doi.org/10.5326/0380549.

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A 6-month-old, female, domestic shorthair cat was examined after onychectomy and ovariohysterectomy because of bleeding from the paws. Prolonged activated partial thromboplastin time was discovered. Coagulation factor analyses revealed deficiency of factor XI coagulant activity. Plasma mixing studies indicated factor deficiency or dysfunction rather than factor inhibition. Feline factor XI deficiency in one adult cat has been previously reported but was attributed to factor XI inhibitors. The signalment, lack of primary disease, and the finding of persistent factor XI deficiency in the absence
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Rehan, Haider, Mehdi Asghar, Kumari Das Geetha, Ahmed Khanzada Zameer, and Zameer Khanzada Sambreen. "Factor XI deficiency and its management." i-manager's Journal on Life Sciences 3, no. 1 (2024): 11. http://dx.doi.org/10.26634/jls.3.1.20936.

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Factor XI deficiency, also known as hemophilia C, is an uncommon bleeding disorder caused by insufficient levels of coagulation factor XI, a critical protein in the intrinsic pathway of blood clotting. Unlike hemophilia A and B, which involve deficiencies in factors VIII and IX respectively, factor XI deficiency typically results in milder bleeding. Managing factor XI deficiency requires a comprehensive approach that considers the severity of bleeding episodes, patient responses, and potential risks. Treatment options include factor replacement therapy, desmopressin (DDAVP), and antifibrinolyt
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Dissertations / Theses on the topic "Factor XI Deficiency"

1

Bolton-Maggs, Paula H. B. "Phenotype and molecular genetics of factor XI deficiency in the United Kingdom." Thesis, University of Oxford, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.491905.

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A new bleeding disorder was described in 1953 in which the bleeding symptoms were present in both males and females. It was differentiated from haemophilia A and B by plasma mixing studies. Early clinical and family studies of this disorder, factor XI deficiency, were limited to a small number of families, some described before a reliable factor XI:C assay was developed. In 1985 a study was made of 24 families with factor XI deficiency in North London, . followed by a more extensive study of 30 families in North West England. These two studies confirmed the mode of inheritance (autosomal) but
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Mitchell, Michael John. "The molecular basis of factor XI deficiency : a study of structure/function relationships." Thesis, King's College London (University of London), 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.440457.

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O'Connell, N. M. "Factor XI deficiency : a study of clinical, laboratory and molecular modifiers of bleeding phenotype." Thesis, University College London (University of London), 2011. http://discovery.ucl.ac.uk/1324551/.

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Factor XI (FXI) is a plasma glycoprotein that participates in the consolidation phase of blood coagulation and is important in the creation of a stable fibrin clot. Deficiency of FXI leads to an injury-related bleeding diathesis, which is notable for the variability in the bleeding tendency and the lack of a clear relationship between bleeding and FXI coagulant activity (FXI:C). A comprehensive understanding of the factors which influence the bleeding tendency in factor XI deficiency would enable a more structured evaluation of bleeding risk and would focus the treatment choices for individual
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Pike, Gillian. "Investigation of the role of global haemostasis assays and bleeding scores in the assessment and management of patients with Factor XI deficiency." Thesis, University of Manchester, 2016. https://www.research.manchester.ac.uk/portal/en/theses/investigation-of-the-role-of-global-haemostasis-assays-and-bleeding-scores-in-the-assessment-and-management-of-patients-with-factor-xi-deficiency(0ff0defc-ba60-4f91-b283-86cb3fe0c9c5).html.

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The clinical management of Factor XI (FXI) deficiency is problematic due to the marked phenotypic heterogeneity between individuals with this disorder and the lack of a reliable test to predict bleeding risk. FXI-deficient individuals are currently at risk of being over- or under treated, with associated risks of transfusion-related complications or haemorrhage respectively. The improvement of care of FXI-deficient patients requires the development of measures that can predict bleeding phenotype and enable the identification of individuals who need treatment at times of haemostatic challenge.
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KOSOBUDOVÁ, Hana. "Kongenitální choroby skotu." Master's thesis, 2012. http://www.nusl.cz/ntk/nusl-137031.

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In the framework of this thesis was performed genotyping of 46 specimens of the breed Czech red cattle from the University farm in Czech Budejovice, which monitored the incidence of autosomal recessive genetic disorders, specifically bovine citrullinemia (BC) in exon 5 and deficiency of blood coagulation factor XI (FXI) in exon 9 and 12. Genotyping for BC was done using PCR/RFLP methods and for the disorder FXI in both exons genotypes were determined on the basis of different length of fragments using PCR technology and horizontal agarose electrophoresis. The presence of mutant allele was dete
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Ganopolsky, Jorge G. "Inflammation in protein C and factor XI deficient mice." 2004. http://etd.nd.edu/ETD-db/theses/available/etd-04162004-163619/.

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Thesis (Ph. D.)--University of Notre Dame, 2004.<br>Thesis directed by Francis J. Castellino for the Department of Chemistry and Biochemistry. "April 2004." Includes bibliographical references at end of each chapter.
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Book chapters on the topic "Factor XI Deficiency"

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Myers, Bethan, and Rezan A. Kadir. "Factor XI Deficiency." In Inherited Bleeding Disorders in Women 2e. John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119426080.ch7.

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Brown, James P. R., and Joanne Douglas. "Factor XI Deficiency." In Consults in Obstetric Anesthesiology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-59680-8_61.

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Seligsohn, Uri, and Paula H. B. Bolton-Maggs. "Factor XI Deficiency." In Textbook of Hemophilia. Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9781444318555.ch53.

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Bolton-Maggs, Paula H. B., and Uri Seligsohn. "Factor XI Deficiency." In Textbook of Hemophilia. John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118398258.ch58.

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Desnick, Robert J., Orlando Guntinas-Lichius, George W. Padberg, et al. "Factor XI Deficiency." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_8707.

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Tabatabaei, Tahere, and Akbar Dorgalaleh. "Congenital Factor XI Deficiency." In Congenital Bleeding Disorders. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-76723-9_12.

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Bane, Charles E., Anne T. Neff, and David Gailani. "Factor XI Deficiency or Hemophilia C." In Hemostasis and Thrombosis. John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118833391.ch6.

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Davidson, Simon. "Congenital Factor XI Deficiency, Diagnosis and Management." In Congenital Bleeding Disorders. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-43156-2_13.

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Maak, B., F. Bergmann, L. Kochhan, and Ch Estel. "Factor XI Deficiency caused by a hitherto unknown Mutation in the Factor XI Gene." In 32nd Hemophilia Symposium Hamburg 2001. Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-18150-4_33.

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Lee, Christine A., and Rezan A. Kadir. "Inherited Bleeding Disorders in Pregnancy: von Willebrand Disease, Factor XI Deficiency, and Hemophilia A and B Carriers." In Disorders of Thrombosis and Hemostasis in Pregnancy. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-15120-5_11.

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Conference papers on the topic "Factor XI Deficiency"

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Halimeh, S., M. Siebert, and S. Alecsi. "Endemic Struma Nodosa in European Patients with Factor XI Deficiency." In GTH Congress 2024 – 68th Annual Meeting of the Society of Thrombosis and Haemostasis Research – Building Bridges in Coagulation. Georg Thieme Verlag, 2024. http://dx.doi.org/10.1055/s-0044-1779155.

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Khaerunnisa, Isyana, Sutikno, Ahmad Furqon, Fajrin Shidiq, and Cece Sumantri. "Identification of factor XI deficiency, complex vertebral malformation, and Bovine Leukocyte adhesion deficiency carriers in Indonesian Pasundan bull." In INTERNATIONAL CONFERENCE ON ORGANIC AND APPLIED CHEMISTRY (ICOAC) 2022. AIP Publishing, 2024. http://dx.doi.org/10.1063/5.0184735.

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Weiss, H. J., V. T. Turitto, and H. R. Baumgartner. "FACTORS INFLUENCING FIBRIN DEPOSITION ON SUBENDOTHELIUM." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642950.

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During the past several years, we have initiated studies to determine the role of plasma factors and platelets, and the properties of the blood vessel, which influence the activation of the coagulation mechanism on the subendothelium. Studies were performed by exposing everted segments of de-endothelialized rabbit aorta, mounted in a perfusion chamber, to non-anticoagulated human blood for 5 to 10 minutes under a range of flow conditions, and measuring fibrin and platelet deposition on the subendothelium, and fibrinopepstide A (FPA) levels in post-chamber blood. In normal subjects, platelet de
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McKenna, R., E. R. Cole, A. Yuk, and W. Whisler. "NORMAL APTT IN A PATIENT WITH A PERSISTENT ACQUIRED FACTOR XI INHIBITOR." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643303.

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A 55 year-old white male (MR) with documented normal hemostatic tests at our institution for 14 years, was admitted for a laminectomy. His pre-operative APTT and prothrombin time activity (Q) were again normal. A prolonged APTT of 58 seconds (21-31 N) and Q of 18% (&gt; . 70% N) was noted on the 14th post-operative day; patient had received I unit PRBC during surgery.Mixtures of pooled normal plasma (NPP) incubated with MR’ s plasma (301, 37° C) yielded normal APTTs with as little as 20% NPP in these mixtures, suggesting a plasma factor(s) deficiency. Factor XI level was undetectable with norm
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Harutyunyan, Hayk, Sevak Badalyan, Ruben Fanarjyan, Gurgen Melkumyan, and Ruzan Nazinyan. "P108/262 Surgical management of spontaneous intracerebral hematoma in a patient with hemophilia C(XI factor deficiency)." In 15TH Congress of the European Society of Minimally Invasive Neurological Therapy 2023 Meeting Abstracts. BMJ Publishing Group Ltd., 2023. http://dx.doi.org/10.1136/jnis-2023-esmint.138.

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Kuder, H., S. K. Dikeson, D. Gailani, et al. "A missense variant in F11 gene causes coagulation factor XI deficiency in Maine Coon cats with increased bleeding tendency." In 30. Jahrestagung der FG „Innere Medizin und klinische Labordiagnostik“ der DVG (InnLab) – Teil 1: Vorträge. Georg Thieme Verlag, 2022. http://dx.doi.org/10.1055/s-0041-1741156.

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McKenna, R., E. R. Cole, and A. DOOLAS. "SUCCESSFUL SURGICAL MANAGEMENT OF A PATIENT WITH COMBINED FACTOR V AND VIII DEFICIENCY WITH DDAVP + FFP." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644131.

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A 59 year-old white male with a life long history of severe bleeding following trauma or surgical procedures was documented to have a combined factor V and factor Vlll-C deficiency. His baseline factor V ranged between 16% - 30% and factor VIII-C was between 20% - 30%. His APTT ranged between 50 - 56 seconds (21-31 N) with a prothrombin time activity between 33% - 40% of normal ( ≥ 70% N). Factor II, Vll/X, X, IX, XI, XII, template bleeding time and platelet function studies were normal. There was no severe factor XIII deficiency. Since the response of these patients to DDAVP is unknown and th
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Mannhalter, C., and E. Deutsch. "IMMUN0BL0TTING STUDIES OF THE INTERACTION OF PLASMA-FACT0R XI WITH KAOLIN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644806.

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An immunoblotting technique with a commercially available, polyclonal Factor XI antibody was developed for Factor XI in plasma samples.Aliquots containing equivalents of 6 ul plasma were treated with appropriate amounts of SDS, electrophoresed on 7.5 % SDS-PAGE and immunoblotted onto nitrocellulose membranes. The blots were quenched with low fat milk and incubated with 2 % Factor Xl-antiserum, which was preadsorbed with Factor XI deficient plasma. The bound Factor XI antibody was identified by incubation with 125I-labelled Factor XI followed by autoradiography. Using this method, the interacti
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Berrettini, M., M. J. Heeb, and J. H. Griffin. "ISOLATION AND FUNCTIONAL PROPERTIES OF MONOMERIC BLOOD COAGULATION FACTOR XI." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642803.

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To evaluate the significance of the normal dimeric structure (160,000 MW) of blood coagulation Factor XI (F.XI), a monomeric form (80,000 MW) was produced by mild reduction and alkylation of native F.XI. Since initial efforts to reduce and alkylate F.XI in solution inactivated the molecule, F.XI was bound to high MW kininogen (HMWK) to stabilize the native structure. Purified F.XI was bound to HMWK-Sepharose, and the column was washed for 2 h with 40 μM dithiothreitol in 4mM acetate buffer, 2mM EDTA, 1mM benzamidine, pH 7.8, and then for 2 h with 50 μM iodoacetamide in the same buffer. Elution
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Furmaniak-Kazmierczak, E., J. Jagielski, and T. Wilusz. "THE EFECT OF CMTI-I INHIBITOR ON HUMAN BLOOD CLOTTING SYSTEM." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644327.

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Polipeptyde inhibitors for different serine proteases have been isolated from a variety of plants. Among them there are the inhibitors from squash seeds of molecular weight about 3 300 /1/. The experiments were carried out to determine the effect of one of the squash inhibitors /CMTI-I/ on human blood clotting system. The 0.1 ml of inhibitor /O,8-100 ug/ was added to 0,1 ml of normal intact plasma and incubated 0,5, 15, 30 and 60 minutes at 37°C. It was found that partial tromboplastin time /PTT/ and activated partial thromboplastin time /APTT/ were prolonged. CMTI-I did not show a progressive
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