Relevant bibliographies by topics / Faecalis

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  2. Dissertations / Theses
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Academic literature on the topic 'Faecalis'

Author: Grafiati
Published: 4 June 2021
Last updated: 24 January 2023

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Journal articles on the topic "Faecalis"

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Odeyemi, Adebowale, and Paul Omorovie. "Pathogenicity and antibiotic resistance of Enterococcus faecalis isolated from water used in health-care centers of Ekiti State University and environ." International Journal of Biological Research 4, no. 2 (September 26, 2016): 220. http://dx.doi.org/10.14419/ijbr.v4i2.6636.

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The quality of water samples obtained from the health-care center in the Ekiti State University and three other centers around the campus; Ekiti State University Teaching Hospital (EKSUTH), Iworoko-Ekiti health Centre (IHC) and the State Hospital, Ikole-Ekiti (SHI) were investigated by analyzing the total bacterial count using pour plate method; the incidence and antibiotic resistance of Enterococcus faecalis as water quality indicator was enumerated using selective isolation and disk diffusion method respectively. The mean TBC, TCC and TEC of all the water samples ranged from 9.1 x 102 to 17.4 x 103 CFU/ml, 4.1 x 102 to 5.5 x 103 CFU/ml and 0.4 x 102 to 0.4 x 103 CFU/ml respectively. A total of 70 (32.9%) Enterococcus faecalis were recovered from the water samples from Iworoko HC, which showed highest distribution in bore-hole and well water samples while least frequency of E. faecalis (15.7%) was recovered from EKSU HC. However, no incidence of E. faecalis in table water obtained from all the health-care facilities. Just 35% of 20 selected E. faecalis were caseinase producers while 80% of the isolates were biofilm producers. All the isolates were resistant to cefuroxime, cefixime, augmentin and ceftazidine while only 10% of them were resistant to ofloxacin. 58.6% of the isolates showed MAR to eight (8) antibiotics with three different resistotypes while only 1.4% of them showed MAR to four (4) antibiotics with just one resistotype (CRX-CXM-AUG-CAZ). Only E. faecalis15 among the selected isolates possessed two plasmids with molecular weight of 1.415bp and 13.535bp. However, consumption of contaminated water traceable to faecal sources and plasmid mediated of the causative microbes would be discussed.
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Sari, Ika Rhisty Cendana, Rini Devijanti Ridwan, and Diah Savitri Ernawati. "Inhibitory effects of siwak (Salvadora persica. L) extract on the growth of Enterococcus faecalis planktonics and biofilms by in vitro." Dental Journal (Majalah Kedokteran Gigi) 49, no. 3 (September 30, 2016): 158. http://dx.doi.org/10.20473/j.djmkg.v49.i3.p158-162.

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Background: Enterococcus faecalis (E. faecalis) is one of the most persistent gram positive bacteria in root canal, resulting in secondary infection after endodontic treatment. E. faecalis pathogenicity is caused by overgrowth of E. faecalis planktonics and biofilms. E. faecalis planktonics produce lipoteichoid acid (LTA) as a virulence factor that can defend their permeability cell. On the other hand, E. faecalis biofilms produce protease, such as Esp (enterococcal surface protein), GelE (gelatinase), and SprE (serin protease), that have quorum-sensing mechanism as an adhesion factor to form extracellular polysaccharide substance (EPS) and increase the growth of the biofilms themselves. Siwak (Salvadora persica L.) has active components, namely benzylisothio-cyanate, trimethylamine, and salvadorine that can inhibit the growth of E. faecalis planktonics and biofilms. Purpose: This study aimed to measure inhibitory effects of siwak extract on the growth of E. faecalis planktonics and biofilms. Method: This research was an antimicrobial research on the culture of E.faecalis incubated in a TSB medium. Siwak extract was diluted into different concentrations, namely 25%, 30%, 35%, 40%, 45%, 50%, 55%, 60%, 65%, 70%, 75%, and 100%. The extract then was placed into the E. faecalis’s colony and planted into Trypticase Soy Agar medium. After incubated for 24 hours at 37°C, the colony would be measured and compared with the control (+) and control (-). As an antibiofilm research, this research used biofilm microtitter assay method to form E. faecalis biofilms incubated in a well-plate medium containing TSB and 0.1 % glucose. Siwak extract then was diluted into the same range concentration as in first method, and placed into the colony of E. faecalis to form biofilms. The biofilms were measured and compared to the control (+) given siwak extract and the control (-) given 0.1% chlorhexidine. After the incubation, they were washed three times, and staining process was conducted using Chrystal violet. The optical density then was measured by ELISA Reader 595 nm. Result: Siwak extract could inhibit the growth of E. faecalis planktonics at the concentration of 35% as a minimum inhibitory concentration as well as the growth of E. faecalis biofilms at the concentration of 45% as a minimum biofilm inhibitory concentration. Conclusion: Siwak extract has an inhibitory effect, particularly at a concentration of 35% on the growth of E. faecalis planktonics and at the concentration of 45% on the growth of E. faecalis biofilms.
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Phillips, David M. "Enterococcus faecalis." New England Journal of Medicine 332, no. 1 (January 5, 1995): 26. http://dx.doi.org/10.1056/nejm199501053320105.

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Scheb-Wetzel, Matthias, Manfred Rohde, Alicia Bravo, and Oliver Goldmann. "New Insights into the Antimicrobial Effect of Mast Cells against Enterococcus faecalis." Infection and Immunity 82, no. 11 (August 11, 2014): 4496–507. http://dx.doi.org/10.1128/iai.02114-14.

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ABSTRACTEnterococcus faecalishas emerged as an important cause of life-threatening multidrug-resistant bacterial infections in the hospital setting. The pathogenesis of enterococcal infections has remained a relatively neglected field despite their obvious clinical relevance. The objective of this study was to characterize the interactions between mast cells (MCs), an innate immune cell population abundant in the intestinal lamina propria, andE. faecalis. This study was conducted with primary bone marrow-derived murine MCs. The results demonstrated that MCs exerted an antimicrobial effect againstE. faecalisthat was mediated both by degranulation, with the concomitant discharge of the antimicrobial effectors contained in the granules, and by the release of extracellular traps, in whichE. faecaliswas snared and killed. In particular, the cathelicidin LL-37 released by the MCs had potent antimicrobial effect againstE. faecalis. We also investigated the specific receptors involved in the recognition ofE. faecalisby MCs. We found that Toll-like receptors (TLRs) are critically involved in the MC recognition ofE. faecalis, since MCs deficient in the expression of MyD88, an adaptor molecule required for signaling by most TLRs, were significantly impaired in their capacity to degranulate, to reduceE. faecalisgrowth as well as to release tumor necrosis factor alpha (TNF-α) and interleukin 6 (IL-6) after encountering this pathogen. Furthermore, TLR2 was identified as the most prominent TLR involved in the recognition ofE. faecalisby MCs. The results of this study indicate that MCs may be important contributors to the host innate immune defenses againstE. faecalis.
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Bouanane-Darenfed, Amel, Wajdi Ben Hania, Jean-Luc Cayol, Bernard Ollivier, and Marie-Laure Fardeau. "Reclassification of Acetomicrobium faecale as Caldicoprobacter faecalis comb. nov." International Journal of Systematic and Evolutionary Microbiology 65, Pt_10 (October 1, 2015): 3286–88. http://dx.doi.org/10.1099/ijsem.0.000409.

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Taking into account its phenotypical and genetic characteristics, Acetomicrobium faecale was first recognized as a member of the genus Acetomicrobium, family Bacteroidaceae, order Bacteroidales, phylum Bacteroidetes, with Acetomicrobium flavidum the type species of the genus. However, it was found that A. faecale had 95.8 %, 97.6 % and 98.4 % similarity, respectively, with Caldicoprobacter guelmensis, Caldicoprobacter algeriensis and Caldicoprobacter oshimai and only 82 % similarity with A. flavidum. The DNA G+C content of A. faecale is 45 mol , which is of the same order as the DNA G+C content of the three strains of species of the genus Caldicoprobacter and its main fatty acid is C16 : 0, with its second most prominent fatty acid, iso-C17 : 0, also common to strains of species of the genus Caldicoprobacter. On the basis of further phylogenetic, genetic and chemotaxonomic studies, we propose that A. faecale (type strain DSM 20678T = JCM 30420T) be reclassified as Caldicoprobacter faecalis comb. nov.
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Endo, Marcos Sergio, Fernanda Graziela Corrêa Signoretti, Vivian Sayuri Kitayama, Ariane Cássia Salustiano Marinho, Frederico Canato Martinho, and Brenda Paula Figueiredo de Almeida Gomes. "Culture and molecular analysis of Enterococcus faecalis and antimicrobial susceptibility of clinical isolates from patients with failure endodontic treatment." Brazilian Dental Science 17, no. 3 (September 15, 2014): 83. http://dx.doi.org/10.14295/bds.2014.v17i3.1016.

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<p><strong>Objective: </strong>The aim of this study was to evaluate the minimal inhibitory concentrations (MIC) of different antibiotic agents against to the most prevalent microorganism found in root-filled canals by culture and molecular approaches. <strong>Material and </strong><strong>Methods: </strong>The microbial samples were taken either from thirty root-filled canals after removal of gutta-percha. Culture methods and 16s rDNA assay were used to identify the <em>E faecails</em> present in the samples. The antimicrobial susceptibilities of the isolates of <em>E faecalis </em>were determined by MIC values using the E test System and interpreted according to the Clinical and Laboratory Standards Institute guidelines. The following antibiotics were used: benzylpenicillin, amoxicillin, amoxicillin-clavulanic acid, erythromycin, azithromycin, vancomycin, chloramphenicol, tetracycline, doxycycline, ciprofloxacin, rifampicin and moxifloxacin. <strong>Results: </strong><em>E faecalis</em> were isolated (7/30) and detected (13/30) by culture and PCR assay, respectively. All tested <em>E faecalis</em> (<em>n</em>=12) were highly sensitive to amoxicillin, moxifloxacin, vancomycin, benzylpenicillin and amoxicillin-clavulanic acid. Some antibiotics were resistant against <em>E faecalis </em>strains such as rifampicin (4/12), tetracycline (2/12), doxycycline (1/12), erythromycin (3/12) and azythromycin (8/12). <strong>Conclusion</strong>: Amoxicillin, amoxicillin-clavulanic acid, benzylpenicillin, vancomycin and moxifloxacin were the most active antibiotics, in vitro, against <em>E faecalis</em> clinical strains, with all the isolates being susceptible. Azithromycin and erythromycin were least effective, with none percentage of isolates being susceptible, during laboratory testing. Moreover, <em>E faecalis </em>were identified more frequently by PCR assay than by culture technique.</p><p><strong>Keywords: </strong>Retreatment; Antibiotics, antimicrobial susceptibility; <em>Enterococcus faecalis;</em> Antibiotic resistance. </p>
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Hayakawa, Kayoko, Dror Marchaim, Mohan Palla, Uma Mahesh Gudur, Harish Pulluru, Pradeep Bathina, Khaled Alshabani, et al. "Epidemiology of Vancomycin-Resistant Enterococcus faecalis: a Case-Case-Control Study." Antimicrobial Agents and Chemotherapy 57, no. 1 (October 15, 2012): 49–55. http://dx.doi.org/10.1128/aac.01271-12.

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ABSTRACTAlthough much is known about vancomycin-resistant (VR)Enterococcus faecium, little is known about the epidemiology of VREnterococcus faecalis. The predilection of VRE. faecalisto transfer the vancomycin resistance determinant toStaphylococcus aureusis much greater than that of VRE. faecium. The epidemiology of VRE. faecalishas important implications regarding the emergence of vancomycin-resistantS. aureus(VRSA); 8 of 13 reported VRSA cases have been from Michigan. A retrospective case-case-control study was conducted at the Detroit Medical Center, located in southeastern Michigan. Unique patients with VRE. faecalisinfection were matched to patients with strains of vancomycin-susceptible (VS)E. faecalisand to uninfected controls at a 1:1:1 ratio. Five hundred thirty-two VRE. faecaliscases were identified and were matched to 532 VSE. faecaliscases and 532 uninfected controls. The overall mean age of the study cohort (n= 1,596) was 63.0 ± 17.4 years, and 747 (46.8%) individuals were male. Independent predictors for the isolation of VRE. faecalis(but not VSE. faecalis) compared to uninfected controls were an age of ≥65 years, nonhome residence, diabetes mellitus, peripheral vascular disease, exposure to cephalosporins and fluoroquinolones in the prior 3 months, and immunosuppressive status. Invasive procedures and/or surgery, chronic skin ulcers, and indwelling devices were risk factors for both VRE. faecalisand VSE. faecalisisolation. Cephalosporin and fluoroquinolone exposures were unique, independent predictors for isolation of VRE. faecalis. A majority of case patients had VRE. faecalispresent at the time of admission. Control of VRE. faecalis, and ultimately VRSA, will likely require regional efforts focusing on infection prevention and antimicrobial stewardship.
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Hokama, Sanehiro, Yasuko Honma, Claudia Toma, and Yoshihide Ogawa. "Oxalate-DegradingEnterococcus faecalis." Microbiology and Immunology 44, no. 4 (April 2000): 235–40. http://dx.doi.org/10.1111/j.1348-0421.2000.tb02489.x.

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Fernández-Hidalgo, Nuria, and Laura Escolà-Vergé. "Enterococcus faecalis Bacteremia." Journal of the American College of Cardiology 74, no. 2 (July 2019): 202–4. http://dx.doi.org/10.1016/j.jacc.2019.03.526.

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Arsène, Stéphanie, and Roland Leclercq. "Role of a qnr-Like Gene in the Intrinsic Resistance of Enterococcus faecalis to Fluoroquinolones." Antimicrobial Agents and Chemotherapy 51, no. 9 (July 9, 2007): 3254–58. http://dx.doi.org/10.1128/aac.00274-07.

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ABSTRACT Fluoroquinolones are poorly active against enterococci. Recently, plasmid-borne resistance to fluoroquinolones due to the qnr gene was reported in members of the Enterobacteriaceae family. The gene encodes a pentapeptide repeat protein that protects DNA gyrase from inhibition by fluoroquinolones. We have identified in the genome of Enterococcus faecalis V583 a qnr-like gene, named E. faecalis qnr (qnr E. faecalis ), encoding a putative pentapeptide repeat protein that shares 25% identity with Qnr. To assess its potential role in the intrinsic resistance of E. faecalis to fluoroquinolones, qnr E. faecalis was inactivated in E. faecalis JH2-2 by insertion of the thermosensitive vector pG1KT. This strain was then complemented with qnr E. faecalis cloned in the multicopy plasmid pORI23. The effects of its overexpression were also studied. Inactivation of the qnr E. faecalis gene resulted in twofold decreases in the MICs of ofloxacin and ciprofloxacin. When the gene was complemented or overexpressed, MICs of fluoroquinolones increased four- to nine-fold, leading to MICs of ofloxacin and ciprofloxacin equal to 32 μg/ml and 8 μg/ml, respectively. The E. faecalis Qnr (Qnr E. faecalis ) protein was produced and purified. Qnr E. faecalis protein protected Escherichia coli DNA gyrase from inhibition by ofloxacin. The qnr E. faecalis gene was then introduced into E. coli DH10B, Staphylococcus aureus RN4220, and Lactococcus lactis IL-1419 to study its heterologous expression. MICs of the various fluoroquinolones tested increased 4- to 16-fold, showing that Qnr E. faecalis conferred resistance to fluoroquinolones in various bacterial backgrounds. Overexpression of qnr E. faecalis in enterococci or mobilization of the gene to other bacterial species may be anticipated as a possible new mechanism for fluoroquinolone resistance.
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Dissertations / Theses on the topic "Faecalis"

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Fritzenwanker, Moritz [Verfasser]. "Genomsequenz des probiotischen Enterococcus faecalis Symbioflor 1 (DSM 16431) und vergleichende Genomanalyse mit den Stämmen E. faecalis V583, E. faecalis OG1RF und E. faecalis 62 / Moritz Fritzenwanker." Gießen : Universitätsbibliothek, 2013. http://d-nb.info/1065320566/34.

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Roberts, Gretta. "Glycoprotein utilisation by enterococcus faecalis." Thesis, King's College London (University of London), 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.412446.

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Barros, Mariana Vilhena. "Infeções nosocomiais por enterococcus faecalis." Master's thesis, [s.n.], 2014. http://hdl.handle.net/10284/4512.

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Projeto de Pós-Graduação/Dissertação apresentado à Universidade Fernando Pessoa como parte dos requisitos para obtenção do grau de Mestre em Ciências Farmacêuticas
As infeções nosocomiais são consideradas um problema mundial de saúde pública. A sua disseminação tem contribuído para aumento das taxas de mortalidade e morbilidade, a maioria das vezes devido às limitadas ou mesmo inexistentes opções terapêuticas. Enterococcus faecalis é uma bactéria Gram-positiva, anaeróbia facultativa, presente na flora comensal do trato gastrintestinal de humanos e animais. Apesar da sua suposta inocuidade, nas últimas décadas E.faecalis tem-se revelado um patogénio oportunista, representando a segunda e a terceira maior causa de infeções hospitalares a nível mundial. Esta bactéria é apontada como uma das principais causas de endocardites, bacteremias, infeções do trato urinário, intra-abdominais e de feridas contraídas em hospitais. As suas caraterísticas fisiológicas permitem-lhe sobreviver a altas temperaturas, a elevados valores de pH e concentrações salinas. Esta bactéria resiste também em ambientes hostis, em situações de subnutrição, de stress oxidativo e às técnicas tradicionais de limpeza. Apresenta inúmeros fatores de virulência nomeadamente proteínas de superfície, enzimas hidrolíticas e capacidade de formação de biofilmes, o que auxilia esta bactéria a invadir, colonizar e infetar tecidos hospedeiros. Enterococcus faecalis exibe uma resistência intrínseca a algumas classes de antibióticos como β-lactâmicos, lincosamidas, trimetropim-sulfametoxazol, fluoroquinolonas e baixas concentrações de aminoglícosídeos. Devido à sua capacidade mutagénica e adaptativa, este microrganismo desenvolve e adquire novas resistências, através de mutações cromossomais ou por transferência de genes. Como é o caso do gene vanA e vanB, que lhe conferem resistência à vancomicina (VRE). E.faecalis tem-se revelado uma constante ameaça de vida em todo o mundo, por isso é urgente controlar a disseminação desta bactéria. É importante desenvolver novos métodos terapêuticos, tendo em conta a ineficácia dos atuais e aplicar estratégias, a nível hospitalar, que diminuam ao máximo a transmissão do microrganismo. The nosocomial infections are nowadays a worldwide issue in terms of health. Its dissemination has been the cause of the rising of mortality and disease rates, most of the time due to limited or even nonexistent therapeutic options. Enterococcus faecalis is a Gram-positive anaerobic facultative bacterium witch is found in the flora of the gastrointestinal tract of humans and animals. Despite its alleged harmlessness, lately E.faecalis has been proved to be an opportunistic pathogen, representing the second and the third leading cause of hospital-acquired infections worldwide. This bacterium is being pointed as a mainly cause of endocarditis, fast progressing bacteremia may present, urinary tract infections, intra-abdominal and wounds contracted in hospitals. Its physiological characteristics allows this bacterium to survive at high temperatures, high PH values and highly concentration salts. It can also resist in hostiles environments such as malnutrition, oxidative stress and traditional cleaning technics. It feature numerous virulence factors including surface proteins, hydrolytic enzymes and ability of biofilm formation, which helps the bacterium to invade, colonize and infect the host tissue. Enterococcus faecalis displays an intrinsic resistance to some classes of antibiotics such as β-lactam, lincosamides, trimetropim-sulfamethoxazole, fluoroquinolones and low concentration of aminoglycosides. Due to its mutagenic capacity and adaptive skills, this micro-organism develops and acquires new resistances, through chromosome mutations or by genetic transferences e.g. gene vanA e vanB, witch confers resistance to Vancomycin (VRE). E. faecalis has proved a constant threat of life throughout the world, so there is an urgent need to control the spread of this bacteria by developing new therapeutic methods, having regard to the ineffectiveness of current ones and looking for new strategies inside hospitals that will reduce as much as possible the transmission of the micro-organism.
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Kurenbach, Brigitta. "Konjugativer DNA-Transfer zwischen Gram-positiven und Gram-negativen Spezies: Transferkomponenten des Multiresistenzplasmids pIP501 aus Streptococcus agalactiae." [S.l.] : [s.n.], 2004. http://deposit.ddb.de/cgi-bin/dokserv?idn=971485305.

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Böttcher, Daiana Elisabeth. "Avaliação do efeito da presença do biofilme de Enterococcus faecalis no canal radicular sobre a manutenção da substantividade da clorexidina a 2% : estudo in vitro." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2014. http://hdl.handle.net/10183/110768.

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Introdução: O objetivo do presente estudo foi correlacionar o efeito antimicrobiano residual e a substantividade da solução de clorexidina a 2%, em dentina humana de dentes extraídos e contaminada com E. faecalis, por 48 horas, 7 e 30 dias. Metodologia: Cento e vinte e três dentes humanos extraídos foram utilizados para esse estudo. As amostras foram divididas em quatro grupos conforme a solução irrigadora utilizada (CHX 2% ou soro fisiológico) e na presença ou ausência do biofilme de Enterococcus faecalis. As amostras foram mantidas em contato com a respectiva solução durante 5 minutos. Cada grupo foi distribuído aleatoriamente em 3 subgrupos de acordo com o período de avaliação (n=10). A quantidade de CHX presente foi avaliada através de cromatografia líquida de alta eficiência (HPLC) e a viabilidade bacteriana foi analisada através de microscópio confocal a laser (CLSM). A análise estatistica foi feita através dos testes de Kruskal-Wallis e Mann-Whitney U (P<.05) e teste de correlação de Spearman (P<.01). Resultados: Houve uma correlação negativa entre o percentual de células viáveis e a quantidade de CHX remanescente (P = .000). A CHX reduziu significativamente o percentual de células viáveis em relação ao soro após 48 horas (P = .007). A diferença foi mantida no período de 7 dias (P = .001). Após 30 dias, o grupo CHX apresentou um aumento da viabilidade bacteriana tornando-se semelhante ao soro (P = .623). Simultaneamente, a quantidade de CHX reduziu significativamente após 30 dias (P = .000). Conclusão: Os resultados do presente estudo indicam que a CHX a 2% foi detectada nos periodos de 48 horas e 7 dias, mantendo reduzido o percentual de células viáveis. A presença de microrganismos na dentina humana não alterou a quantidade de CHX residual.
Introduction: The aim of this study was to correlate the bacterial viability and the presence of 2% chlorhexidine (CHX) on dentin by means of confocal laser scanning microscope (CLSM) and high-performance liquid chromatography (HPLC) for 48 hours, 7 and 30 days. Methods: One hundred twenty three extracted human teeth were used. Samples were divided into 4 groups according to the solution (CHX or saline) and the presence of Enterococus faecalis biofilm. Samples were kept in contact with 5mL of the solution for 5 minutes. Each group was divided into 3 subgroups according to the evaluation period (n = 10). Statistical analysis was performed by using the Kruskal- Wallis, Mann-Whitney U tests (P < .05) and Spearman’s Rank Correlation Coefficient (P < .01). Results: There was a negative correlation between the percentage of live cells and the amount of remaining CHX (P = .000). CHX significantly reduced the percentage of viable cells compared to saline after 48 hours (P = .007). Differences were maintained in the 7-day evaluation (P = .001). After 30 days, CHX group presented an increase of viable cells, thereby becoming similar to saline (P = .623). Simultaneously, remaining CHX significantly reduced in the 30-day specimens (P = .000). Conclusion: The results of this study indicate that 2% CHX solution was detected for 48 hours and 7 days, keeping a low percentage of viable cells. The presence of microorganisms on human dentin did not affect 2% chlorhexidine maintenance.
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Tse, Chee-choong Micheal, and 謝志聰. "Effect of ultrasonic agitation on enterococcus faecalis biofilm." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B45165993.

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Low, Yuen Li. "Metal regulation of the E. faecalis efaCBA operon." Thesis, University of Bath, 2002. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.760823.

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Abadía, Patiño Lorena. "Caractérisation de l'opéron vanE chez Enterococcus faecalis BM4405." Paris 7, 2003. http://www.theses.fr/2003PA077001.

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Solioz, Marc. "Studium des ATP-getriebenen Ionentransportes in Streptococcus faecalis /." [S.l.] : [s.n.], 1990. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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Diederich, Ann-Kristin [Verfasser], and Johannes [Akademischer Betreuer] Hübner. "Membrane lipids of Enterococcus faecalis as microbial pathogens." Freiburg : Universität, 2016. http://d-nb.info/1119452708/34.

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Books on the topic "Faecalis"

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Aitchison, Eileen Jane. The antigenic composition of Streptococcus faecalis associated with ineffective endocarditis. Birmingham: Aston University. Department of Pharmaceutical Sciences, 1987.

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Shorrock, Patricia Joan. Surface properties of enterococcus faecalis in relation to infective endocarditis. Birmingham: Aston University. Department of Pharmaceutical Sciences, 1990.

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Bao, Yinyin. Role of mprF1 and mprF2 in the pathogenicity of Enterococcus faecalis. Freiburg: Universität, 2012.

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Peters, Tansy M. Studies on the activation of azo-dyes into direct-acting genotoxic agents by enterococcus faecalis. London: University of East London, 1995.

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Jeremy, Wilkinson, and Institute of Hydrology, eds. Modelling faecal coliform concentrations in streams. Wallingford: Institute of Hydrology, 1995.

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Kay, David, and C. Fricker, eds. Significance of Faecal Indicators in Water. Cambridge: Royal Society of Chemistry, 2012. http://dx.doi.org/10.1039/9781849735421.

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Heins, Terry. Beating sneaky poo: Ideas for faecal soiling. Canberra: ACT Health Authority, 1985.

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Radley, Simon. Biliary and faecal bile acids in colorectal cancer. Birmingham: University of Birmingham, 1995.

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Dunlop, Patrick S. M. The photocatalytic inactivation of faecal indicator organisms in water. [S.l: The Author], 2002.

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Faecal incontinence and related problems in the older adult. London: E. Arnold, 1993.

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Book chapters on the topic "Faecalis"

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Murray, Barbara E., Liangxia Jiang, Jianguo Xiao, Xiang Qin, Kavindra V. Singh, Aart de Kok, Al Claiborne, Patrice Courvalin, and George M. Weinstock. "Enterococcus faecalis." In Bacterial Genomes, 649–50. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-6369-3_59.

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Manson, Janet M., and Michael S. Gilmore. "Pathogenomics of Enterococcus faecalis." In Pathogenomics, 125–47. Weinheim, FRG: Wiley-VCH Verlag GmbH & Co. KGaA, 2006. http://dx.doi.org/10.1002/352760801x.ch7.

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Wirth, Reinhard. "The sex pheromone system of Enterococcus faecalis." In EJB Reviews 1994, 117–28. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-79502-2_9.

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You, C. B., W. Song, H. X. Wang, J. P. Li, M. Lin, and W. L. Hai. "Association of Alcaligenes faecalis with wetland rice." In Nitrogen Fixation, 195–99. Dordrecht: Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3486-6_34.

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Fujimoto, S., M. Bastos, K. Tanimoto, F. An, K. Wu, and D. B. Clewell. "The pAD1 Sex Pheromone Response in Enterococcus faecalis." In Streptococci and the Host, 1037–40. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1825-3_244.

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Stehle, T., G. E. Schulz, S. A. Ahmed, and A. Claiborne. "THE STRUCTURE OF NADH PEROXIDASE FROM STREPTOCOCCUS FAECALIS." In Flavins and Flavoproteins 1990, edited by B. Curti, S. Ronchi, and G. Zanetti, 651–54. Berlin, Boston: De Gruyter, 1991. http://dx.doi.org/10.1515/9783110855425-124.

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Clermont, Dominique, Gilda de Cespédès, Françoise Delbos, and Théa Horaud. "Genetic Analysis of Enterococcus faecalis Chromosome Carrying Mobile Elements." In Streptococci and the Host, 1023–27. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1825-3_241.

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Shankar, Viswanathan, and Michael S. Gilmore. "Characterization of the Enterococcus faecalis Alpha C Protein Homolog." In Streptococci and the Host, 1045–48. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1825-3_246.

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Nakayama, Jiro, Yuuichiro Takanami, and Akinori Suzuki. "Analysis of Pheromone Binding and Pheromone Reception by Enterococcus faecalis." In Streptococci and the Host, 1033–35. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1825-3_243.

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Pasquarella, Cesira, Donald Morrison, Angelo Savino, and Barry D. Cookson. "Dynamics of Enterococcus faecalis Colonization of Bone Marrow Transplant Patients." In Streptococci and the Host, 275–79. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1825-3_68.

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Conference papers on the topic "Faecalis"

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Keyhani, Jacqueline, and Ezzatollah Keyhani. "Increased resistance to detergent in Enterococcus faecalis." In Proceedings of the International Conference on Antimicrobial Research (ICAR2010). WORLD SCIENTIFIC, 2011. http://dx.doi.org/10.1142/9789814354868_0010.

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Santos, Pâmela Gomes, Rosane Nassar Meireles Guerra, Josivan Regis Farias, Simone Batista Muniz, and Danielle Cristine Gomes Franco. "AÇÃO ANTIMICROBIANA DAS FLORES DE ANACARDIUM OCCIDENTALE E DO ÁCIDO ELÁGICO PRESENTE NO EXTRATO." In I Congresso Brasileiro de Imunologia On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/965.

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Introdução: As infecções bacterianas têm aumentado significativamente nas últimas décadas, sobretudo aquelas ocasionadas por microrganismos multirresistentes. Assim, o uso de produtos naturais com finalidades terapêuticas surge com alvo de bioprospecção na busca de novos compostos com ação antimicrobiana. Além disso, o uso de insetos, como o Tenebrio molitor como modelo experimental para avaliação in vivo tem sido muito frequente, pois exige menos material em relação aos testes com animais vertebrados. Objetivo: O presente trabalho investigou o efeito citotóxico e ação antimicrobiana do extrato hidroalcoólico das flores de Anacardium occidentale (EHAo) e do ácido elágico. Material e Métodos: Avaliamos a citoxicidade de ácido elágico e do EHAo nas concentrações (1; 5 e 50mg/kg) em Tenebrio molitor. A ação antimicrobiana para Enterococcus faecalis, Staphylococcus aureus, Streptococcus mutans, Escherichia coli, Pseudomonas aeruginosa e Klebsiella pneumoniae e a toxicidade do ácido elágico, foi avaliada por microdiluição, segundo a norma M7-A6 do manual da Clinical and Laboratory Standards Institute – CLSI. Foi determinada a Concentração Bactericida Mínima (CBM) e concentração inibitória mínima (CIM), em culturas de 24 horas, incubadas à 37ºC. Resultados: No ensaio de citotoxidade aguda se verificou que nenhuma das concentrações usados foram tóxicas, pois não ocorreram óbitos e nem nenhuma anormalia morfológica nas larvas de Tenebrio molitor. Os testes de concentração inibitória mínima (CIM) de concentração bactericida mínima (CBM) mostraram que o EHAo apresentou ação bactericida para Enterococcus faecalis em todas as concentrações testadas. Para Staphylococcus aureus os resultados mostraram ação bactericida para as maiores concentrações e bacteriostática para a menor diluição. O ácido elágico teve ação bactericida apenas para Enterococcus faecalis. Para as bactérias Streptococcus mutans, Escherichia coli, Pseudomonas aeruginosa e Klebsiella pneumoniae as concentrações de EHAo e ácido elágico testadas não foram inibitórias. Conclusões: Os resultados mostraram baixa toxicidade tanto para o EHAo como para o ácido elágico e ainda, que o extrato apresentou melhor efeito antimicrobiano do que o ácido elágico, para Enterococcus faecalis e Staphylococcus aureus.
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Wang, Lin, Yongfang Yang, and Ji Li. "Bioremediation of eutrophicated scenery water by Alcaligenes faecalis." In 2013 21st International Conference on Geoinformatics. IEEE, 2013. http://dx.doi.org/10.1109/geoinformatics.2013.6626082.

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Sarp, Ayşe S., and Murat Gülsoy. "Comparing irradiation parameters on disinfecting enterrecoccus faecalis in root canal disinfection." In SPIE BiOS, edited by Peter Rechmann and Daniel Fried. SPIE, 2016. http://dx.doi.org/10.1117/12.2217958.

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Tverezovska, Olesia, Viktoriia Holubnycha, Rafal Banasiuk, Yevheniia Husak, Savchenko Anton, and Viktoriia Korniienko. "The Effect of Silver Nanoparticles Against Formation of Enterococcus Faecalis Biofilms." In 2022 IEEE 12th International Conference Nanomaterials: Applications & Properties (NAP). IEEE, 2022. http://dx.doi.org/10.1109/nap55339.2022.9934155.

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Chin, Beek Yoke, Zhi Ping Yang, Lester Kobzik, and Leo E. Otterbein. "Carbon Monoxide Enhances The Bactericidal Activity Of Macrophages Infected With Enterococci Faecalis." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5640.

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LUIS BAPTISTA DE GUSMÃO, PEDRO, THAIS DA SILVA FEITOSA, Brenda Paula Figueiredo de Almeida Gomes, Eloá Cristina Bícego-Pereira, and MAICON RICARDO ZIEBERG PASSINI. "Isolamento e identificação de Enterococcus faecalis de dentes submetidos à reintervenção endodôntica." In XXIV Congresso de Iniciação Científica da UNICAMP - 2016. Campinas - SP, Brazil: Galoa, 2016. http://dx.doi.org/10.19146/pibic-2016-51716.

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Zhang, Hongtao, Xiaobei Zhan, Zhiyong Zheng, Jianrong Wu, and Dingqiang Chen. "New Strategy for Enhancement Curdlan Biosynthesis in Alcaligenes faecalis by Activating Gene Expression." In 2010 4th International Conference on Bioinformatics and Biomedical Engineering (iCBBE). IEEE, 2010. http://dx.doi.org/10.1109/icbbe.2010.5517254.

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Ke Sun, Jue Zhang, Jing Fang, Jing Wang, Jie Pan, and Weidong Zhu. "Cold plasma therapy for enterococcus faecalis biofilm infected tooth root canal in vitro." In 2012 IEEE 39th International Conference on Plasma Sciences (ICOPS). IEEE, 2012. http://dx.doi.org/10.1109/plasma.2012.6383868.

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Paula Figueiredo De Almeida Gomes, Brenda, Maikon Tadeu Ferrari Martinucho, Ela͍se Gabriele Martins, and Beatriz Leonardo Prudenciano. "Avaliação de substâncias químicas auxiliares, utilizadas em endodontia, na redução de Enterococcus faecalis." In XXIII Congresso de Iniciação Científica da Unicamp. Campinas - SP, Brazil: Galoá, 2015. http://dx.doi.org/10.19146/pibic-2015-38003.

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Reports on the topic "Faecalis"

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Muniz, I., L. Jimenez, G. A. Toranzos, and T. C. Hazen. Survival and activity of Streptococcus faecalis and Escherichia coli in tropical freshwater. Office of Scientific and Technical Information (OSTI), December 1988. http://dx.doi.org/10.2172/666262.

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Santo Domingo, J. W., F. A. Fuentes, and T. C. Hazen. Survival and activity of Streptococcus faecalis and Escherichia coli in petroleum-contaminated tropical marine waters. Office of Scientific and Technical Information (OSTI), December 1987. http://dx.doi.org/10.2172/666217.

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A. Komnos, Georgios, Antonios Papadopoulos, Efstratios Athanaselis, Theofilos Karachalios, and Sokratis E. Varitimidis. Migrating Periprosthetic Infection from a Total Hip Replacement to a Contralateral Non-Operated Osteoarthritic Knee Joint. Science Repository, January 2023. http://dx.doi.org/10.31487/j.ijscr.2022.03.02.

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Introduction: There is a paucity of published data on whether a treated infected arthroplasty is a risk factor for infection in another, non-operated joint. Contamination of a primary, arthritic, non-operated joint from an infected arthroplasty is a relatively rare entity. Case: We report a case of migration of a pathogen (Enterococcus faecalis) from an infected prosthetic joint (hip) to the contralateral native joint (knee). Identification of the pathogen was made with PCR, by obtaining cultures during the implantation of the primary knee prosthesis. Conclusion: Contamination of a primary, arthritic, non-operated joint from an infected arthroplasty has not been widely reported. Management of such cases is extremely challenging and without clear and established guidelines. Our experience shows that tissue samples should be taken intraoperatively and sent for cultures, so as to exclude contamination in those cases.
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Bezerra, Alexandre Sacchetti, Flavia Altheman Loureiro, Carla Maria Pasquareli Vazquez, Afonso Cesar Polimanti, and Rafi Felicio Bauab Dauar. Empiric Treatment of Foot Infection in Patients with Severe Diabetes. Science Repository, December 2021. http://dx.doi.org/10.31487/j.jicoa.2021.04.04.

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Background: Despite being treated with antibiotics of broad spectrum recommended by International Consensus, severe diabetic patients with lower limb infection do not present a positive clinical evolution during empirical treatment. This study’s bacterial profile was analysed and compared with other worldwide hospital centers. Objective: To confirm the need of an individualized empirical treatment for severe diabetic patients with foot infection. Methods: Retrospective analysis of cultures and antibiograms of severe diabetic patients admitted by foot infection. Results: The results were consistent with the socioeconomic realities of developing countries. Gram-negative bacteria (52,11%) were present in most bone cultures. Results presented a high incidence of Enterococcus faecalis in both gram-positive (21,2%) and polymicrobial (34,7%) samples. Bacterial resistance with the use of ordinary antibiotics in the statistical analysis was high. Conclusion: The community infections should undergo broad spectrum empirical therapy combining amikacin (80,43%) or meropenem (72,00%) with gram-negative and vancomycin (100%) or teicoplanin (90,00%) or linezolid (74,19%) with gram-positive.
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Veldkamp, T., J. W. van Riel, R. A. Dekker, S. Khalaji, V. Khaksar, H. Hashemipour, M. M. van Krimpen, and M. C. Blok. Estimating requirement values for apparent faecal digestible and standardised ileal digestible threonine in broilers by a meta-analysis approach. Wageningen: Wageningen UR Livestock Research, 2016. http://dx.doi.org/10.18174/388688.

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Veldkamp, T., J. W. van Riel, R. A. Dekker, S. Khalaji, V. Khaksar, H. Hashemipour, M. M. van Krimpen, and M. C. Blok. Estimating requirement values for apparent faecal digestible and standardised ileal digestible methionine+cysteine in broilers by a metaanalysis approach. Wageningen: Wageningen UR Livestock Research, 2016. http://dx.doi.org/10.18174/388689.

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Veldkamp, T., J. W. van Riel, R. A. Dekker, S. Khalaji, V. Khaksar, H. Hashemipour, M. M. van Krimpen, and M. C. Blok. Estimating requirement values for apparent faecal digestible and standardised ileal digestible lysine in broilers by a meta-analysis approach. Wageningen: Wageningen UR Livestock Research, 2016. http://dx.doi.org/10.18174/388690.

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Veldkamp, T., J. W. van Riel, R. A. Dekker, S. Khalaji, V. Khaksar, H. Hashemipour, M. M. van Krimpen, and M. C. Blok. Estimating requirement values for apparent faecal digestible and standardised ileal digestible tryptophan in broilers by a meta-analysis approach. Wageningen: Wageningen UR Livestock Research, 2016. http://dx.doi.org/10.18174/388749.

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Veldkamp, T., J. W. van Riel, R. A. Dekker, S. Khalaji, V. Khaksar, H. Hashemipour, M. M. van Krimpen, and M. C. Blok. Estimating requirement values for apparent faecal digestible and standardised ileal digestible arginine in broilers by a meta-analysis approach. Wageningen: Wageningen UR Livestock Research, 2016. http://dx.doi.org/10.18174/388750.

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Veldkamp, T., J. W. van Riel, R. A. Dekker, S. Khalaji, V. Khaksar, H. Hashemipour, M. M. van Krimpen, and M. C. Blok. Estimating requirement values for apparent faecal digestible and standardised ileal digestible valine in broilers by a meta-analysis approach. Wageningen: Wageningen UR Livestock Research, 2016. http://dx.doi.org/10.18174/388751.

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