Journal articles: 'FAMILIES (HUMAN)'

1

Gates, Hill, and Steven Harrell. "Human Families." Journal of the Royal Anthropological Institute 5, no. 3 (September 1999): 502. http://dx.doi.org/10.2307/2661327.

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Puszyk, Monika. "Families of non-heterosexual people with children - among old answers and new questions." Studia Humanistyczne AGH 15, no. 4 (2016): 7. http://dx.doi.org/10.7494/human.2016.15.4.7.

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Żak, Monika. "Instability or balance? Work– life dynamics in lives of policemen and their families." Studia Humanistyczne AGH 17, no. 4 (2018): 19. http://dx.doi.org/10.7494/human.2018.17.4.19.

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Charles, Nickie. "Post-Human Families? Dog-Human Relations in the Domestic Sphere." Sociological Research Online 21, no. 3 (August 2016): 83–94. http://dx.doi.org/10.5153/sro.3975.

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In this article I explore the ways in which dogs and other companion species become family members and engage with the argument that this indicates the emergence of post-human families. Using empirical data from responses to a Mass Observation directive on Animals and Humans and in-depth interviews with people who share their homes with companion animals, I explore the ways in which humans and dogs live with each other and the ‘daily practices of kinship’ which constitute them as kin. I argue that practices of kinship blur the species barrier but that human-dog relations take place in the context of unequal power relations which are an inevitable consequence of dogs’ incorporation into families as dependents. I conclude that while it may be possible to identify post-human practices in multi-species households, they exist alongside practices which reinforce the human-animal boundary and that, given the unequal relations of entanglement within which humans and animals interact, attempts to identify empirically a post-human family seem problematic. What can be said, however, is that a post-human approach to kinship practices highlights the porousness of the category human and alerts us both to the deep connections between humans and other animals and to the profoundly unequal ways in which animals are incorporated into social relations with humans.

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Niedbalski, Jakub. "IDENTITY AND THE SENSE OF SUBJECTIVITY IN PARENTS OF CHILDREN WITH INTELLECTUAL DISABILITIES IN THE CONTEXT OF FAMILY LIFE DESTABILIZATION." Studia Humanistyczne AGH 19, no. 3 (2020): 157–75. http://dx.doi.org/10.7494/human.2020.19.3.157.

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The lives of families who take care of people with disabilities are full of unexpected events, unwanted situations, and difficulties that accumulate at every step. Therefore, bearing in mind the purpose of this paper, I focused on determining those conditions which are a source of destabilization in the lives of families with intellectually disabled children. I attemptto reconstruct the sources of threats to the sense of subjectivity and the shaping of the identity of parents who have children with disabilities. The theoretical framework of the analysis is symbolic interactionism. The research material used in the presented article is composed of personal experiences of parents of disabled individuals, and unstructured interviews were performed with these people. Analysis of the research material was performed in accordance with the procedures of grounded theory methodology.

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Krzyżowski, Łukasz. "(Trans)national intergenerational care contract - attitudes and practises of transnational families towards elderly care." Studia Humanistyczne AGH 13, no. 2 (2014): 103. http://dx.doi.org/10.7494/human.2014.13.2.103.

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White, Ray, Jean-Marc Lalouel, Mark Leppert, Mark Lathrop, Yusuke Nakamura, and Peter O'Connell. "Linkage maps of human chromosomes." Genome 31, no. 2 (January 15, 1989): 1066–72. http://dx.doi.org/10.1139/g89-183.

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Finding the chromosomal location of human genes that heretofore have been defined solely by phenotypes, in particular clinical phenotypes that are transmitted in Mendelian fashion in families, is an early and often crucial step in the process of identifying the molecular basis of a disease. Recent progress in construction of chromosomal maps of genetically linked DNA markers has made almost the entire human genome accessible to linkage studies in families that are segregating genetic defects. Construction of linkage maps requires a panel of three-generation families for genotyping, a large number of polymorphic markers, and sophisticated computer programs for analysis of genotypic data. After a locus harboring a deleterious mutation has been identified by linkage to a mapped marker, a high-resolution map of the region can be constructed with new markers derived from cosmid libraries, to narrow the search for the gene in question. For example, this strategy has been pursued in the effort to characterize the gene responsible for familial adenomatous polyposis. When a target region has been narrowed to about 1 centiMorgan, corresponding to roughly a million base pairs in physical distance, other techniques of molecular biology can be brought to bear to isolate and clone the actual gene.Key words: genetic linkage, chromosome maps, DNA markers, chromosome 17, chromosome 10, genetic disease, familial adenomatous polyposis.

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8

Filsinger, Erik E., and Richard A. Fabes. "Odor Communication, Pheromones, and Human Families." Journal of Marriage and the Family 47, no. 2 (May 1985): 349. http://dx.doi.org/10.2307/352135.

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9

Shukul, Maneesha. "Investment in Human Capital by Families." Journal of Social Sciences 14, no. 2 (March 2007): 112–13. http://dx.doi.org/10.1080/09718923.2007.11978345.

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Shibahara, Toyohiro, Hiroshi Okamura, and Naoaki Yanagihara. "Human Leukocyte Antigens in Bell's Palsy." Annals of Otology, Rhinology & Laryngology 97, no. 6_suppl3 (November 1988): 11–13. http://dx.doi.org/10.1177/00034894880976s304.

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Ninety-three patients with Bell's palsy including 12 members of two families with familial Bell's palsy were typed for human leukocyte antigens (HLAs). Significant association between HLAs and Bell's palsy was found. The association of certain HLAs with the palsy may predict the specific clinical course and prognosis.

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Bekpen, Cemalettin, and Diethard Tautz. "Human core duplicon gene families: game changers or game players?" Briefings in Functional Genomics 18, no. 6 (September 16, 2019): 402–11. http://dx.doi.org/10.1093/bfgp/elz016.

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Abstract Illuminating the role of specific gene duplications within the human lineage can provide insights into human-specific adaptations. The so-called human core duplicon gene families have received particular attention in this respect, due to special features, such as expansion along single chromosomes, newly acquired protein domains and signatures of positive selection. Here, we summarize the data available for 10 such families and include some new analyses. A picture emerges that suggests broad functions for these protein families, possibly through modification of core cellular pathways. Still, more dedicated studies are required to elucidate the function of core-duplicons gene families and how they have shaped adaptations and evolution of humans.

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12

Arnett, Frank C. "Genetic Studies of Human Lupus in Families." International Reviews of Immunology 19, no. 4-5 (January 2000): 297–317. http://dx.doi.org/10.3109/08830180009055501.

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13

Krautwurst, Dietmar. "Human Olfactory Receptor Families and Their Odorants." Chemistry & Biodiversity 5, no. 6 (June 2008): 842–52. http://dx.doi.org/10.1002/cbdv.200890099.

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14

Polavarapu, Nalini, Nathan J. Bowen, and John F. McDonald. "Newly Identified Families of Human Endogenous Retroviruses." Journal of Virology 80, no. 9 (May 1, 2006): 4640–42. http://dx.doi.org/10.1128/jvi.80.9.4640-4642.2006.

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15

Hamity, Matthew, Carter Dillard, Sarah M. Bexell, and Catharina Graff-Hughey. "A Human Rights Approach to Planning Families." Social Change 49, no. 3 (September 2019): 469–92. http://dx.doi.org/10.1177/0049085719863894.

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We present reflections on the current normative model of family planning and provide a novel alternative model centred on children’s rights. Over the last half-century, environmentalists, ecological economists, and child psychologists have raised key issues facing child rights: the threat of climate change and environmental degradation, the critical importance of childhood development, and growing economic inequality. As these challenges have become more widely recognised, organisations and governments have responded by investing in renewable energy, preschools, and availability of birth control. However, human population is expected to reach an alarming 11 billion or more in 2100, endangering nearly all life on Earth. Family planning interventions are the most effective way to reduce population growth and improve human well-being while simultaneously preventing ecological collapse. However, comprehensive attempts at articulating a human rights approach to family planning are lacking: here is our attempt.

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16

PEELE, ROGER. "Homelessness, Health, and Human Needs; Homeless Families." American Journal of Psychiatry 147, no. 2 (February 1990): 252. http://dx.doi.org/10.1176/ajp.147.2.252.

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17

Miller, V. L., and A. M. Martin. "The Human Genome Project: Implications for Families." Health & Social Work 33, no. 1 (February 1, 2008): 73–76. http://dx.doi.org/10.1093/hsw/33.1.73.

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18

Nebert, Daniel W., Kjell Wikvall, and Walter L. Miller. "Human cytochromes P450 in health and disease." Philosophical Transactions of the Royal Society B: Biological Sciences 368, no. 1612 (February 19, 2013): 20120431. http://dx.doi.org/10.1098/rstb.2012.0431.

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There are 18 mammalian cytochrome P450 ( CYP ) families, which encode 57 genes in the human genome. CYP2 , CYP3 and CYP4 families contain far more genes than the other 15 families; these three families are also the ones that are dramatically larger in rodent genomes. Most (if not all) genes in the CYP1 , CYP2 , CYP3 and CYP4 families encode enzymes involved in eicosanoid metabolism and are inducible by various environmental stimuli (i.e. diet, chemical inducers, drugs, pheromones, etc.), whereas the other 14 gene families often have only a single member, and are rarely if ever inducible or redundant. Although the CYP2 and CYP3 families can be regarded as largely redundant and promiscuous, mutations or other defects in one or more genes of the remaining 16 gene families are primarily the ones responsible for P450-specific diseases—confirming these genes are not superfluous or promiscuous but rather are more directly involved in critical life functions. P450-mediated diseases comprise those caused by: aberrant steroidogenesis; defects in fatty acid, cholesterol and bile acid pathways; vitamin D dysregulation and retinoid (as well as putative eicosanoid) dysregulation during fertilization, implantation, embryogenesis, foetogenesis and neonatal development.

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19

Wills, Jenny. "Families, Children and Local Government." Children Australia 14, no. 3 (1989): 7–8. http://dx.doi.org/10.1017/s0312897000002290.

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Planner-coordinator, catalyst, facilitator, service provider and service funder now characterise Local Government's involvement in children's services and the human services generally.A 1987 report prepared for the Local Government Ministers, Community Development, Human Services and Local Government presents a national overview of the increased role of councils in human services clearly signalling that the debate of the 70s about whether Local Government should be involved has been replaced with questions about the basis of that involvement and implementation issues.

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20

Emanuel, PD, CJ Eaves, VC Broudy, T. Papayannopoulou, MR Moore, AD D'Andrea, JF Prchal, AC Eaves, and JT Prchal. "Familial and congenital polycythemia in three unrelated families." Blood 79, no. 11 (June 1, 1992): 3019–30. http://dx.doi.org/10.1182/blood.v79.11.3019.3019.

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Abstract Three families with polycythemia inherited through apparently different modes are described. Secondary causes of polycythemia were ruled out. Erythropoietin (EPO) levels were normal or low, even after phlebotomy. In vitro erythroid colony growth in standard assay cultures containing EPO was normal; however, in the absence of added EPO, a few progenitors from most of the affected individuals were able to generate recognizable colonies of mature erythroblasts, although these were smaller and proportionately less numerous than seen in polycythemia vera (PV). To search for EPO-receptor changes as a possible pathophysiologic mechanism, we examined, by Southern blot analysis, genomic DNA samples from affected and nonaffected family members, as well as three patients with PV. Two different probes, derived from the human EPO-receptor, were used. We found no evidence for chromosomal rearrangements or gene amplification in hereditary polycythemia or PV patients. Further, no nucleotide sequences were found that were homologous to the Friend spleen focus-forming virus glycoprotein gp55, which has been shown to bind to and activate the murine EPO-receptor. Functional studies examining number and binding affinity of the EPO- receptor on erythroid progenitors from three hereditary polycythemia patients demonstrated no abnormalities. We conclude that the mechanism(s) for the erythrocytosis in familial and congenital polycythemia and in PV may not involve the EPO-receptor and, therefore, may result from alterations of postreceptor responses.

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21

Emanuel, PD, CJ Eaves, VC Broudy, T. Papayannopoulou, MR Moore, AD D'Andrea, JF Prchal, AC Eaves, and JT Prchal. "Familial and congenital polycythemia in three unrelated families." Blood 79, no. 11 (June 1, 1992): 3019–30. http://dx.doi.org/10.1182/blood.v79.11.3019.bloodjournal79113019.

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Three families with polycythemia inherited through apparently different modes are described. Secondary causes of polycythemia were ruled out. Erythropoietin (EPO) levels were normal or low, even after phlebotomy. In vitro erythroid colony growth in standard assay cultures containing EPO was normal; however, in the absence of added EPO, a few progenitors from most of the affected individuals were able to generate recognizable colonies of mature erythroblasts, although these were smaller and proportionately less numerous than seen in polycythemia vera (PV). To search for EPO-receptor changes as a possible pathophysiologic mechanism, we examined, by Southern blot analysis, genomic DNA samples from affected and nonaffected family members, as well as three patients with PV. Two different probes, derived from the human EPO-receptor, were used. We found no evidence for chromosomal rearrangements or gene amplification in hereditary polycythemia or PV patients. Further, no nucleotide sequences were found that were homologous to the Friend spleen focus-forming virus glycoprotein gp55, which has been shown to bind to and activate the murine EPO-receptor. Functional studies examining number and binding affinity of the EPO- receptor on erythroid progenitors from three hereditary polycythemia patients demonstrated no abnormalities. We conclude that the mechanism(s) for the erythrocytosis in familial and congenital polycythemia and in PV may not involve the EPO-receptor and, therefore, may result from alterations of postreceptor responses.

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22

Khan, Vassilis-Javed, and Panos Markopoulos. "Busy families’ awareness needs." International Journal of Human-Computer Studies 67, no. 2 (February 2009): 139–53. http://dx.doi.org/10.1016/j.ijhcs.2008.09.006.

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23

Coulson, Andrew J. "Human Life, Human Organizations and Education." education policy analysis archives 2 (June 3, 1994): 9. http://dx.doi.org/10.14507/epaa.v2n9.1994.

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The social structures within which we live and work have a profound effect on the success of our pursuits. These effects are too often poorly understood by those who shape public policy, leading to organizations that are antagonistic to the very goals they are meant to achieve. Unfortunately, this has been the case with public education in the United States. Data are presented that illustrate the way in which the incentive structure of our public school system leads the goals of its employees to diverge from those of the families it is intended to serve. Arguments in support of government-run schooling are discussed and refuted. An alternative system of mutually beneficial cooperation within a competitive market is proposed, based on its proven success in the more liberal parts of our economy. It is demonstrated that such a market system would unite the goals of educators and families, encourage innovation, and discourage many of the inefficient and educationally irrelevant practices engendered by the public school system.

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24

Parcel, Toby L., and John P. Hoffmann. "Families and Crime." American Behavioral Scientist 62, no. 11 (July 10, 2018): 1455–62. http://dx.doi.org/10.1177/0002764218787023.

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This volume highlights the theoretical and empirical connections between family sociology and criminology. We review the historical interconnections between these two fields. We argue for greater intellectual conversation across the two areas, and then we identify several elements they hold in common. These include their use of social theory, their attention to human development, and their use and appreciation of longitudinal research. We conclude with brief overviews of the six articles that make up this special issue.

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25

Sang, Qing, Zhihua Zhang, Juanzi Shi, Xiaoxi Sun, Bin Li, Zheng Yan, Songguo Xue, et al. "A pannexin 1 channelopathy causes human oocyte death." Science Translational Medicine 11, no. 485 (March 27, 2019): eaav8731. http://dx.doi.org/10.1126/scitranslmed.aav8731.

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Connexins and pannexins are two protein families that play an important role in cellular communication. Pannexin 1 (PANX1), one of the members of pannexin family, is a channel protein. It is glycosylated and forms three species, GLY0, GLY1, and GLY2. Here, we describe four independent families in which mutations in PANX1 cause familial or sporadic female infertility via a phenotype that we term “oocyte death.” The mutations, which are associated with oocyte death, alter the PANX1 glycosylation pattern, influence the subcellular localization of PANX1 in cultured cells, and result in aberrant PANX1 channel activity, ATP release in oocytes, and mutant PANX1 GLY1. Overexpression of a patient-derived mutation in mice causes infertility, recapitulating the human oocyte death phenotype. Our findings demonstrate the critical role of PANX1 in human oocyte development, provide a genetic explanation for a subtype of infertility, and suggest a potential target for therapeutic intervention for this disease.

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26

Maeda, N., and O. Smithies. "The Evolution of Multigene Families: Human Haptoglobin Genes." Annual Review of Genetics 20, no. 1 (December 1986): 81–108. http://dx.doi.org/10.1146/annurev.ge.20.120186.000501.

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27

Bannert, Norbert, and Reinhard Kurth. "The Evolutionary Dynamics of Human Endogenous Retroviral Families." Annual Review of Genomics and Human Genetics 7, no. 1 (September 2006): 149–73. http://dx.doi.org/10.1146/annurev.genom.7.080505.115700.

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Du, Zhi-Qiang, Cai-Xia Yang, Max F. Rothschild, and Jason W. Ross. "Novel microRNA families expanded in the human genome." BMC Genomics 14, no. 1 (2013): 98. http://dx.doi.org/10.1186/1471-2164-14-98.

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29

Beeley, Josie A. "Fascinating families of proteins: electrophoresis of human saliva." Biochemical Society Transactions 21, no. 1 (February 1, 1993): 133–38. http://dx.doi.org/10.1042/bst0210133.

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30

Waters, Mary C. "Human Rights for Undocumented Students and Their Families." Harvard Educational Review 85, no. 3 (September 1, 2015): 305–9. http://dx.doi.org/10.17763/0017-8055.85.3.305.

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31

Cody, William K. "Parse’s Human Becoming School of Thought and Families." Nursing Science Quarterly 13, no. 4 (October 2000): 281–84. http://dx.doi.org/10.1177/08943180022107951.

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32

Cook, Jeanne F., Keith A. Alford, and Pat Conway. "Introduction to Rural Families and Reshaping Human Services." Journal of Family Social Work 15, no. 5 (October 2012): 351–58. http://dx.doi.org/10.1080/10522158.2012.721122.

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33

Bardelli, Alberto, and Victor E. Velculescu. "Mutational analysis of gene families in human cancer." Current Opinion in Genetics & Development 15, no. 1 (February 2005): 5–12. http://dx.doi.org/10.1016/j.gde.2004.12.009.

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34

Olsson, Per G., Lennart Hammarström, and C. I. Edvard Smith. "Strategies for codon usage in human VH families." Immunology Today 9, no. 2 (January 1988): 35–36. http://dx.doi.org/10.1016/0167-5699(88)91254-6.

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35

Tamura, Robert. "Fertility, human capital and the wealth of families." Economic Theory 4, no. 4 (July 1994): 593–603. http://dx.doi.org/10.1007/bf01213626.

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36

Roach, Jared C., Gustavo Glusman, Robert Hubley, Stephen Z. Montsaroff, Alisha K. Holloway, Denise E. Mauldin, Deepak Srivastava, et al. "Chromosomal Haplotypes by Genetic Phasing of Human Families." American Journal of Human Genetics 89, no. 3 (September 2011): 382–97. http://dx.doi.org/10.1016/j.ajhg.2011.07.023.

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37

Tristem, Michael. "Identification and Characterization of Novel Human Endogenous Retrovirus Families by Phylogenetic Screening of the Human Genome Mapping Project Database." Journal of Virology 74, no. 8 (April 15, 2000): 3715–30. http://dx.doi.org/10.1128/jvi.74.8.3715-3730.2000.

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ABSTRACT Human endogenous retroviruses (HERVs) were first identified almost 20 years ago, and since then numerous families have been described. It has, however, been difficult to obtain a good estimate of both the total number of independently derived families and their relationship to each other as well as to other members of the familyRetroviridae. In this study, I used sequence data derived from over 150 novel HERVs, obtained from the Human Genome Mapping Project database, and a variety of recently identified nonhuman retroviruses to classify the HERVs into 22 independently acquired families. Of these, 17 families were loosely assigned to the class I HERVs, 3 to the class II HERVs and 2 to the class III HERVs. Many of these families have been identified previously, but six are described here for the first time and another four, for which only partial sequence information was previously available, were further characterized. Members of each of the 10 families are defective, and calculation of their integration dates suggested that most of them are likely to have been present within the human lineage since it diverged from the Old World monkeys more than 25 million years ago.

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Cook, Rachelle D., Tim A. Hodgson, Alastair C. W. Waugh, Elizabeth M. Molyneux, Eric Borgstein, A. Sherry, Chong Gee Teo, and Stephen R. Porter. "Mixed patterns of transmission of human herpesvirus-8 (Kaposi’s sarcoma-associated herpesvirus) in Malawian families." Journal of General Virology 83, no. 7 (July 1, 2002): 1613–19. http://dx.doi.org/10.1099/0022-1317-83-7-1613.

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To study transmission patterns of human herpesvirus-8 (HHV-8) (Kaposi’s sarcoma-associated herpesvirus) in families in Malawi, nucleotide sequences derived from two hypervariable loci of the HHV-8 genome, the V1 and V2 regions of open reading frame K1 (K1/V1 and K1/V2, respectively), were amplified from blood and mouth rinse samples of 22 patients with treated and untreated Kaposi’s sarcoma (KS) and their first-degree relatives (n=67). In patients with KS, vincristine therapy was significantly associated with non-detectability of circulating, but not oral, K1/V1 DNA. Intra-familial K1/V1 phylogenetic comparisons of eight families were possible. Both identical and non-identical sequences were observed between family members, suggesting transmission of HHV-8 along both intra- and extra-familial transmission routes.

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Pekin, Jim. "The Human Face of the Rural Downturn." Children Australia 16, no. 04 (1991): 17–18. http://dx.doi.org/10.1017/s1035077200012517.

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The economic effects of the current rural downturn are relatively easy to monitor. This paper looks at the social costs and the impact on farm families. It also discusses the discrimination against farm families within Australia’s welfare system.

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Sinacore, Ada L., and F. Özge Akçali. "Men in Families." Journal of Career Development 27, no. 1 (September 2000): 1–13. http://dx.doi.org/10.1177/089484530002700101.

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Kim, H., A. Monk, G. Wood, M. Blythe, J. Wallace, and P. Olivier. "TimelyPresent: Connecting families across continents." International Journal of Human-Computer Studies 71, no. 10 (October 2013): 1003–11. http://dx.doi.org/10.1016/j.ijhcs.2013.05.001.

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Heath, Sebastian E., and Max Champion. "Human Health Concerns from Pet Ownership After a Tornado." Prehospital and Disaster Medicine 11, no. 1 (March 1996): 67–70. http://dx.doi.org/10.1017/s1049023x00042382.

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AbstractIntroduction:Although 50% to 60% of North American households own pets and many of these pets are considered family members, there is little information on the impact pet ownership on pet-owning families affected by disasters.Methods:This case report describes some of the effects of a tornado on 17 families whose dwellings were destroyed. The setting was a typical urban trailer park.Results:After a tornado at the Sagamore Village Trailer Park in north central Indiana, 104 families were evacuated. Seventeen (16.3%) of these families owned pets. For 14 families (13.5%), pet ownership had an important impact on the families' recovery from the tornado. Public- and mental-health concerns that arose from pet ownership included failure to evacuate a dangerous site, attempts to re-enter a dangerous site, separation anxiety leading to psychosomatic disturbances, and the need for additional animal care.Conclusions:In urban disasters, the behaviors of families with a human-animal bond are likely to pose a significant risk to their own and others' health and safety in urban disasters. In this small study of families affected by a tornado, the most prominent public-health concerns were failure to evacuate because of a pet and attempts of re-entry to save a pet; the most common mental-health concerns resulted from separation anxiety from a pet and refusal to accept medical treatment until a pet's well-being can be assured. These are thought to be typical issues that will arise out of the human-animal bond in urban disaster situations and differ considerably from traditional public-health concerns over dog bites, spread of zoonotic diseases, and human food contamination. Medical disaster preparedness planning should consider the substantial effects that the human-animal bond is likely to have on human recovery from large-scale urban disasters.

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Choi, Saeeun, Hyunjung Joo, and Gisun Lee. "Delphi Study on Human Rights Education Framework for Families." Family and Environment Research 58, no. 3 (August 20, 2020): 315–31. http://dx.doi.org/10.6115/fer.2020.023.

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This study clarifies the definition of and to provides guidelines on educational objectives, core concepts, and content in developing a Human Rights Education Program for families. The Delphi survey method was used to develop a Human Rights Education Program for families. As a result, a Human Rights Education Program for families was defined as education that would ensure all members of the family enjoy universal human rights without discrimination. In addition, that the prejudice and discrimination against socially marginalized would not be created within the family by learning values and attitudes that respects human rights and freedom. The objectives were to learn the merit of respecting individuals, relationships, and community. Core concepts of the program were the rights for myself and others, communicating and responsibilities and a sense of citizenship. Content included human rights, respect of oneself and others, empathetic understanding, acceptance, communication in an intimate relationship, conflict management, sharing the role of caring the family, no discrimination against the socially marginalized, and creating an inclusive community culture. This study can be used as a guideline for family human rights education based on family human rights, which is the core of family democracy.

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44

Becker, Gary S., and Nigel Tomes. "Human Capital and the Rise and Fall of Families." Journal of Labor Economics 4, no. 3, Part 2 (July 1986): S1—S39. http://dx.doi.org/10.1086/298118.

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45

Kaczkowski, B., E. Torarinsson, K. Reiche, J. H. Havgaard, P. F. Stadler, and J. Gorodkin. "Structural profiles of human miRNA families from pairwise clustering." Bioinformatics 25, no. 3 (December 4, 2008): 291–94. http://dx.doi.org/10.1093/bioinformatics/btn628.

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46

Mbulaiteye, Sam M., Ruth M. Pfeiffer, Denise Whitby, Glen R. Brubaker, John Shao, and Robert J. Biggar. "Human Herpesvirus 8 Infection within Families in Rural Tanzania." Journal of Infectious Diseases 187, no. 11 (June 2003): 1780–85. http://dx.doi.org/10.1086/374973.

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COHEN, FELISSA L. "Research on Families and Pediatric Human Immunodeficiency Virus Disease." Journal of Developmental & Behavioral Pediatrics 15, Supplement June (June 1994): S43. http://dx.doi.org/10.1097/00004703-199406001-00007.

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Esposito, S., S. Bosis, H. G. M. Niesters, E. Tremolati, C. Sabatini, A. Porta, E. Fossali, A. D. M. E. Osterhaus, and N. Principi. "Impact of Human Bocavirus on Children and Their Families." Journal of Clinical Microbiology 46, no. 4 (February 20, 2008): 1337–42. http://dx.doi.org/10.1128/jcm.02160-07.

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Almaghrabi, Reem S., Batool Ali, Sahar Al-Thawadi, Magid Abdel Halim, and Abdulrahman A. Alrajhi. "Human immunodeficiency virus type 2 in two Saudi families." Annals of Saudi Medicine 31, no. 4 (July 2011): 417–20. http://dx.doi.org/10.4103/0256-4947.76408.

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Mukai, Tetsu, Takeshi Yamamoto, Toshio Kondo, Kazuhiro Kondo, Toshiomi Okuno, Haruhiko Kosuge, and Koichi Yamanishi. "Molecular epidemiological studies of human herpesvirus 6 in families." Journal of Medical Virology 42, no. 3 (March 1994): 224–27. http://dx.doi.org/10.1002/jmv.1890420303.

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Journal articles on the topic 'FAMILIES (HUMAN)'

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