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Books on the topic 'Fat and carbohydrate metabolism'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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1

Jeukendrug, Asker E. Aspects of carbohydrate and fat metabolism during exercise. De Vrieseborch, 1997.

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2

Institute of Medicine (U.S.). Standing Committee on the Scientific Evaluation of Dietary Reference Intakes. Dietary reference intakes for energy, carbohydrate, fiber, fat, fatty acids, cholesterol, protein and amino acids. National Academy Press, 2002.

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3

Kenney, James J. The L.A. diet: The eating plan that raises your metabolism to lose fat forever. Contemporary Books, 1988.

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4

Kasprzyk, Anna. Rola fruktozo-2,6-bisfosforanu w metabolizmie węglowodanów ciała tłuszczowego owadów. Wydawn. Naukowe Uniwersytetu im. Adama Mickiewicza, 1998.

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5

Ezrin, Calvin. The Type II diabetes diet book: The insulin control diet : your fat can make you thin. Lowell House, 1995.

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6

Michael, Roden, ed. Carbohydrate metabolism. Baillière Tindall, 2003.

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7

Ezrin, Calvin. The type 2 diabetes diet book: The insulin control diet : your fat can make you thin. 3rd ed. Lowell House, 1999.

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8

1939-, Beitner Rivka, ed. Regulation of carbohydrate metabolism. CRC Press, 1985.

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9

Rivka, Beitner, ed. Regulation of carbohydrate metabolism. CRC Press, 1985.

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10

Jane, Stephenson. Carbohydrate, fat & calorie guide. 2nd ed. Appletree Press, 2004.

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11

E, Kowalski Robert, ed. The type 2 diabetes diet book: The insulin control diet : your fat can make you thin. 3rd ed. Lowell House, 1999.

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12

Morgan, Michael J., ed. Carbohydrate Metabolism in Cultured Cells. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-7679-8.

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13

J, Morgan Michael, ed. Carbohydrate metabolism in cultured cells. Plenum Press, 1986.

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14

1936-, Heller Richard F., ed. The carbohydrate addict's fat counter. Signet, 2000.

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15

1955-, Akoh Casimir C., and Swanson Barry Grant, eds. Carbohydrate polyesters as fat substitutes. M. Dekker, 1994.

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16

Arnis, Kuksis, ed. Fat absorption. CRC Press, 1986.

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17

Harkness, R. A., R. J. Pollitt, and G. M. Addison, eds. Carbohydrate and Glycoprotein Metabolism; Maternal Phenylketonuria. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-2175-7.

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18

1922-, Schwartz Robert, ed. Disorders of carbohydrate metabolism in infancy. 3rd ed. Blackwell Scientific Publications, 1991.

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19

Borushek, Allan. The CalorieKing calorie, fat, & carbohydrate counter. 2nd ed. Family Health, 2012.

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20

Lowe, Steph. Low Carb Healthy Fat Nutrition. Hachette Australia, 2019.

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21

Miller, Wayne C. Adaptations to a high fat diet which increase exercise endurance in male rats. 1985.

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22

Dismantling a myth: The role of fat and carbohydrates in our diet. C.C. Thomas, 1987.

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23

van Mil, Edgar G. A. H. Exercise, sport, and diabetes mellitus. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199232482.003.0036.

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Diabetes mellitus is a group of metabolic diseases characterized by chronic hyperglycaemia, resulting from defects in insulin secretion, insulin action, or both. The abnormalities in carbohydrate, fat, and protein metabolism that are found in diabetes are due to deficient action of insulin on target tissues (www.ispad.org)....
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24

Kenney, James J., and Diane Grabowski. The LA Diet: The Eating Plan That Raises Your Metabolism to Lose Fat Forever. Contemporary Books, 1988.

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25

Daniels, L. B. Ketogenic Diet : Fast Metabolism Diet: Your Guidebook about How to Lose Weight and Lose Fat Using a Low Carbohydrate Diet for Lasting Success! Independently Published, 2017.

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26

Soler, Ximena, and Lori A. Aronson. Diabetic Ketoacidosis During Appendicitis with Perforation. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199764495.003.0047.

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Type 1 diabetes mellitus (DM) is a chronic disease of carbohydrate, fat, and protein metabolism caused by a functional lack of insulin. Diabetic ketoacidosis (DKA) involves a combination of hyperglycemia, acidosis and ketosis, and is more often associated with type 1 DM. A child in DKA who presents for emergency surgery may be critically ill and requires expert management of his or her metabolic state to ensure a safe outcome.
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27

Bender, David A. 2. Energy nutrition. Oxford University Press, 2014. http://dx.doi.org/10.1093/actrade/9780199681921.003.0002.

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Apart from water, the body’s first requirement under all conditions is for an energy source to perform physical and chemical work. ‘Energy nutrition’ explains that the metabolic fuels to provide this energy are derived from fats, carbohydrates, protein, and alcohol in the diet. The constituents of a meal provide these fuels directly for a few hours. Simultaneously, reserves of fat and carbohydrate are laid down for use during fasting between meals. Only about one-third of the average person’s energy expenditure is for voluntary activity; two-thirds is required for maintenance of the body’s fun
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28

Murphy, Elaine, Yann Nadjar, and Christine Vianey-Saban. Fatty Acid Oxidation, Electron Transfer and Riboflavin Metabolism Defects. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0008.

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The fatty acid oxidation disorders are a group of autosomally recessively inherited disorders of energy metabolism that may present with life-threatening hypoketotic hypoglycemia, encephalopathy and hepatic dysfunction, muscle symptoms, and/or cardiomyopathy. Milder phenotypes may present in adulthood, causing exercise intolerance, episodic rhabdomyolysis, and neuropathy. Specific investigations include acylcarnitine profiling, urine organic acid analysis, fibroblast or leucocyte studies of fatty acid oxidation flux/enzyme activity, and genetic testing. Management varies depending on the condi
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29

Keto Chuffle: Ketogenic Chaffle Cookbook for Preparing Chaffles with 200 Low Carbohydrate Recipes to Burn Fat Quickly, Lose Weight and Increase Metabolism. Independently Published, 2020.

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30

Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng, and Anne Bardsley. Macronutrients and fibre requirements during pregnancy. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0004.

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In this chapter, the impact of varying intakes of protein, carbohydrate and lipids, which are the key nutrients that contribute to calorie intake, is examined. Fibre is also an important food component that needs to be considered. The maternal macronutrient profile can influence embryonic and fetal development. For instance, both low and excessively high protein intakes during pregnancy are associated with restricted growth, increased adiposity, and impaired glucose tolerance. High-fat maternal diets can significantly increase the susceptibility to diet-induced obesity and percentage total bod
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31

Daniels, L. B. Metabolism Diet : the Ketogenic Diet: A Keto Diet That Intends to Use Fat for Fuel, Burn Fat, and Teach You How to Lose Weight on a Low Carbohydrate Diet. Including Keto Diet Recipes and Fat Bombs! Independently Published, 2017.

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32

Stafstrom, Carl E. Dietary Therapy for Neurological Disorders. Edited by Jong M. Rho. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0018.

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Dietary and metabolic therapies such as the high-fat, low-carbohydrate ketogenic diet (KD) are best known for the treatment of intractable epilepsy. Yet, dietary and metabolic approaches have also found some efficacy in a wide variety of other neurological diseases, including autism spectrum disorder, brain trauma, Alzheimer’s disease, sleep disorders, brain tumors, pain, and multiple sclerosis, as discussed in other chapters of this volume. This chapter provides an overview of clinical and experimental studies using the KD in an array of other neurologic disorders: amyotrophic lateral scleros
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33

Klepper, Joerg, and Baerbel Leiendecker. Glut1 Deficiency. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0005.

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Glut1 Deficiency (Glut1D, OMIM #606777) is caused by impaired glucose transport into the brain. The resulting cerebral “energy crisis” causes intractable seizures, developmental delay, and a complex movement disorder. The diagnosis is based on clinical features, low CSF glucose and/or mutations in the SLC2A1 gene. Paroxysmal exertion-induced dystonia (PED) and hereditary cryohydrocytosis have been described as allelic variants. Adults are increasingly being recognized through family pedigrees. The condition is effectively treatable by mimicking the metabolic state of fasting. High-fat carbohyd
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34

Pot, Jason. Keto Lunch Cookbook: Collection of the Most Popular Ketogenic Recipes, Which Reactivate Your Metabolism and Allow You to Burn Fat Faster, with a Low Carbohydrate Content. Lulu.com, 2021.

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35

Pot, Jason. Keto Lunch Cookbook: Collection of the Most Popular Ketogenic Recipes, Which Reactivate Your Metabolism and Allow You to Burn Fat Faster, with a Low Carbohydrate Content. Lulu.com, 2021.

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36

Nicholson, Grainne, and George M. Hall. Neuroendocrine physiology in anaesthetic practice. Edited by Jonathan G. Hardman. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0008.

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This chapter describes the hormonal, metabolic, and inflammatory response to surgery—commonly known as the surgical stress response. The changes in protein, carbohydrate, and fat metabolism to provide fuel for oxidation are outlined as well as changes in salt and water metabolism. Psychological sequelae of fatigue and malaise are also common in patients undergoing surgery. Attenuating the metabolic and endocrine changes associated with surgery may reduce postoperative morbidity and expedite recovery; the choice of anaesthetic drugs and techniques (regional vs general anaesthesia) and the incre
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37

Levy, David. Insulin treatment and pancreas transplantation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198766452.003.0004.

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The physiology of insulin replacement therapy and the consequences of non-physiological treatment on carbohydrate and fat metabolism are reviewed. Historical aspects of long-acting and short-acting insulins are outlined, and current and future insulin preparations are described, including inhaled insulin. Implementation and refinement of the standard injected multiple dose insulin (MDI) regimen are described in detail. The current status of islet-cell and whole pancreas transplantation, their indications and impact on diabetes complications is reviewed.
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38

Daniels, L. B. Ketogenic Diet: Your Guide for Low Carbohydrate Ketogenic Success! Lose Weight and Keep It off with This Fast Metabolism Diet! Independently Published, 2017.

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39

Hyde, Parker, Vincent J. Miller, and Jeff S. Volek. Keto-Adaptation in Health and Fitness. Edited by Dominic P. D’Agostino. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0038.

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When dietary carbohydrate is restricted and protein consumed in moderation, the evolutionarily-conserved ketogenic metabolic machinery awakens. After just a few days circulating ketones increase by an order of magnitude, and over several weeks there is a profound shift away from glucose as the primary energy substrate to the preferred use of fatty acids and ketones. This metabolic process is known as keto-adaptation. The deemphasis on insulin-dependent glucose uptake into cells and concomitant increase in fat oxidation has important implications in management of insulin resistance and its seco
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40

Carbohydrate metabolism. Baillière Tindall, 2003.

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41

Pot, Jason. Keto Dinner Cookbook: Collection of the Most Popular Ketogenic Recipes for Dinner That Allow You to Burn Fat Faster and at the Same Time, Given the Low Carbohydrate Content, Reactivate Your Metabolism. Lulu.com, 2021.

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42

Pot, Jason. Keto Dinner Cookbook: Collection of the Most Popular Ketogenic Recipes for Dinner That Allow You to Burn Fat Faster and at the Same Time, Given the Low Carbohydrate Content, Reactivate Your Metabolism. Lulu.com, 2021.

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43

Hartman, Adam L. Amino Acids in the Treatment of Neurological Disorders. Edited by Dominic P. D’Agostino. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0035.

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Studies of metabolism- and diet-based therapies in epilepsy and neuroprotection have focused primarily on the quality and quantity of fat supplementation or carbohydrate restriction. However, protein is another key dietary component that has not been as thoroughly studied. A number of amino acids have been shown to stop, terminate, or prevent seizures. In addition, some have been shown to exert neuroprotective effects in other neurological disorders. Amino acids (and their metabolites) may exert their effects by acting at membrane or cytoplasmic receptors, serving as substrates for membrane tr
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44

Beitner, Rivka. Regulation of Carbohydrate Metabolism (Regulation of Carbohydrate Metabolism). Volume II. Reader's Digest Young Families, 1985.

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45

Cheng, Ning, Susan A. Masino, and Jong M. Rho. Metabolic Therapy for Autism Spectrum Disorder and Comorbidities. Edited by Jong M. Rho. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0014.

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Autism spectrum disorder (ASD) is a heretogenous developmental disorder characterized by deficits in sociability and communication and by repetitive and/or restrictive behaviors. Currently, only comorbid manifestations can be alleviated (such as seizures and sleep disturbance) not core behavioral symptoms. Recent studies have increasingly implicated mitochondrial dysfunction as a cause of ASD. Mitochondria play an integral role in many cellular functions and are susceptible to many pathophysiological insults. Derangements in mitochondrial structure and function provide a scientific rationale f
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46

Beitner. Regulation Of Carbohydrate Metabolism. CRC Press, 2018. http://dx.doi.org/10.1201/9781351076272.

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47

Carbohydrate metabolism and aging. 1990.

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48

Duffus, C. F. Carbohydrate Metabolism in Plants. Wiley & Sons, Incorporated, John, 1986.

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49

Carbohydrate metabolism and aging. 1987.

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50

Carbohydrate metabolism and aging. 1990.

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